Relevant bibliographies by topics / CTCFL

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Academic literature on the topic 'CTCFL'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "CTCFL"

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Tong, Xin, Yang Gao, and Zhongjing Su. "Interaction of CTCF and CTCFL in genome regulation through chromatin architecture during the spermatogenesis and carcinogenesis." PeerJ 12 (October 15, 2024): e18240. http://dx.doi.org/10.7717/peerj.18240.

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The zinc finger protein CTCF is ubiquitously expressed and is integral to the regulation of chromatin architecture through its interaction with cohesin. Conversely, CTCFL expression is predominantly restricted to the adult male testis but is aberrantly expressed in certain cancers. Despite their distinct expression patterns, the cooperative and competitive mechanisms by which CTCF and CTCFL regulate target gene expression in spermatocytes and cancer cells remain inadequately understood. In this review, we comprehensively examine the literature on the divergent amino acid sequences, target site
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Yao, Haibo, Qinshu Shao, and Yanfei Shao. "Transcription Factor CTCFL Promotes Cell Proliferation, Migration, and Invasion in Gastric Cancer via Activating DPPA2." Computational and Mathematical Methods in Medicine 2021 (October 19, 2021): 1–11. http://dx.doi.org/10.1155/2021/9097931.

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Objective. To explore the relationship between CTCFL and DPPA2 and validate the positive role of CTCFL/DPPA2 in cell malignant behaviors in gastric cancer. Methods. We predicted gastric cancer-related transcription factors and corresponding target mRNAs through bioinformatics. Levels of CTCFL and DPPA2 were assessed via qRT-PCR and western blot. In vitro experiments were utilized to assay the cell biological behaviors. CHIP was utilized for the assessment of the targeted relationship between CTCFL and DPPA2. Results. CTCFL and DPPA2 were both highly expressed in gastric cancer cells, and high
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Debaugny, Roxanne E., and Jane A. Skok. "CTCF and CTCFL in cancer." Current Opinion in Genetics & Development 61 (April 2020): 44–52. http://dx.doi.org/10.1016/j.gde.2020.02.021.

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Voutsadakis, Ioannis. "Molecular Lesions of Insulator CTCF and Its Paralogue CTCFL (BORIS) in Cancer: An Analysis from Published Genomic Studies." High-Throughput 7, no. 4 (2018): 30. http://dx.doi.org/10.3390/ht7040030.

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CTCF (CCCTC-binding factor) is a transcription regulator with hundreds of binding sites in the human genome. It has a main function as an insulator protein, defining together with cohesins the boundaries of areas of the genome called topologically associating domains (TADs). TADs contain regulatory elements such as enhancers which function as regulators of the transcription of genes inside the boundaries of the TAD while they are restricted from regulating genes outside these boundaries. This paper will examine the most common genetic lesions of CTCF as well as its related protein CTCFL (CTCF-
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DEL CAMPO, EDUARDO PORTILLO, JOSÉ JORGE TALAMÁS MÁRQUEZ, FRANCIANELLA REYES-VARGAS та ін. "CTCF and CTCFL mRNA expression in 17β-estradiol-treated MCF7 cells". Biomedical Reports 2, № 1 (2013): 101–4. http://dx.doi.org/10.3892/br.2013.200.

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Hore, Timothy A., Janine E. Deakin, and Jennifer A. Marshall Graves. "The Evolution of Epigenetic Regulators CTCF and BORIS/CTCFL in Amniotes." PLoS Genetics 4, no. 8 (2008): e1000169. http://dx.doi.org/10.1371/journal.pgen.1000169.

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Campbell, Amy E., Selena R. Martinez, and JJ L. Miranda. "Molecular architecture of CTCFL." Biochemical and Biophysical Research Communications 396, no. 3 (2010): 648–50. http://dx.doi.org/10.1016/j.bbrc.2010.04.146.

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Pugacheva, Elena M., Naoki Kubo, Dmitri Loukinov, et al. "CTCF mediates chromatin looping via N-terminal domain-dependent cohesin retention." Proceedings of the National Academy of Sciences 117, no. 4 (2020): 2020–31. http://dx.doi.org/10.1073/pnas.1911708117.

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The DNA-binding protein CCCTC-binding factor (CTCF) and the cohesin complex function together to shape chromatin architecture in mammalian cells, but the molecular details of this process remain unclear. Here, we demonstrate that a 79-aa region within the CTCF N terminus is essential for cohesin positioning at CTCF binding sites and chromatin loop formation. However, the N terminus of CTCF fused to artificial zinc fingers was not sufficient to redirect cohesin to non-CTCF binding sites, indicating a lack of an autonomously functioning domain in CTCF responsible for cohesin positioning. BORIS (
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Hernandez-Gonzalez, Ignacio, Jair Tenorio-Castano, Nuria Ochoa-Parra, et al. "Novel Genetic and Molecular Pathways in Pulmonary Arterial Hypertension Associated with Connective Tissue Disease." Cells 10, no. 6 (2021): 1488. http://dx.doi.org/10.3390/cells10061488.

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Pulmonary Arterial Hypertension (PAH) is a severe complication of Connective Tissue Disease (CTD), with remarkable morbidity and mortality. However, the molecular and genetic basis of CTD-PAH remains incompletely understood. This study aimed to screen for genetic defects in a cohort of patients with CTD-PAH, using a PAH-specific panel of 35 genes. During recruitment, 79 patients were studied, including 59 Systemic Sclerosis patients (SSc) and 69 females. Disease-associated variants were observed in nine patients: 4 pathogenic/likely pathogenic variants in 4 different genes (TBX4, ABCC8, KCNA5
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Soto Reyes Solis, Ernesto, Daniela Morales-Espinosa, David Cantu, et al. "Characterization of DNA methylation of the promoters CTCF and BORIS (CTCFL) in breast and ovarian cancer." Journal of Clinical Oncology 31, no. 15_suppl (2013): e22151-e22151. http://dx.doi.org/10.1200/jco.2013.31.15_suppl.e22151.

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e22151 Background: Genetic and epigenetic alterations may promote the initiation or development of cancer. Global DNA hypomethylation and local hypermethylation have been observed, particularly in cell cycle control-associated genes, such as tumor suppressor genes like CTCF. The dissociation of CTCF is associated with hypermethylation of several promoters; its paralogue gene (BORIS) is normally expressed in testicular tissue during spermatogenesis. BORIS over-expression has been identified in multiple neoplasms such as melanoma, gynecological cancer, glioblastoma and – recently – breast cancer
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Dissertations / Theses on the topic "CTCFL"

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Dienstbach, Sven [Verfasser]. "Identifizierung der CTCFL-Bindestellen in NIH3T3-Zellen und Einfluss der CTCFL-Expression auf die globale Genexpression / Sven Dienstbach." Gießen : Universitätsbibliothek, 2013. http://d-nb.info/1064992293/34.

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Bergmaier, Philipp Franz Hermann [Verfasser]. "Analyse des genom-weiten Bindeverhaltens des Faktors CTCFL / Philipp Franz Hermann Bergmaier." Gießen : Universitätsbibliothek, 2016. http://d-nb.info/1116894564/34.

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Bergmaier, Philipp [Verfasser]. "Analyse des genom-weiten Bindeverhaltens des Faktors CTCFL / Philipp Franz Hermann Bergmaier." Gießen : Universitätsbibliothek, 2016. http://d-nb.info/1116894564/34.

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Alkhatib, Shaza. "Investigation of the immunological differences between granulocytes from healthy donors and breast cancer patients, with respect to the cancer testis antigen, CTCFL." Thesis, University of Essex, 2015. http://repository.essex.ac.uk/16454/.

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Granulocytes or Polymorphonuclear neutrophils (PMNs) are key players in the non-specific immune system against microbial infection as they express surface receptors for the recognition of general antigenic patterns found on pathogens. In addition PMN’s also act as a mediator for the antigen-specific adaptive immunity and T-cells function. However, an anti-tumoral role of the PMN’s was suggested over the recent years beside their well-studied innate function, opening the door for new studies in this area. CTCFL or BORIS (Brother Of the Regulator of the Imprinting Site) is a paralogue protein to
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Rai, Sushma Debi. "Tumour specific CTCF point mutations abrogate CTCF function." Thesis, University of Essex, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.442523.

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Kung, Johnny Tsun-Yi. "Genome-wide Analysis of Ctcf-RNA Interactions." Thesis, Harvard University, 2014. http://dissertations.umi.com/gsas.harvard:11618.

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Ctcf is a "master regulator" of the genome that plays a role in a variety of gene regulatory functions as well as in genome architecture. Evidence from studying the epigenetic process of X-chromosome inactivation suggests that, in certain cases, Ctcf might carry out its functions through interacting with RNA. Using mouse embryonic stem (ES) cells and a modified protocol for UV-crosslinking and immunoprecipitation followed by high-throughput sequencing (CLIP-seq), Ctcf is found to interact with a multitude of transcripts genome-wide, both protein-coding mRNA (or noncoding transcripts therein) a
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Roberts, Julian Charles. "Identification and characterisation of novel DNA targets of the transcription factor CTCF and analysis of selected CTCF-DNA interactions." Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709829.

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Kita, Georgie-Xanthi. "Characterization of the CTCF isoforms and BORIS, the CTCF paralogue, in normal and cancer breast tissues and investigation of their role in breast tumourigenesis." Thesis, University of Essex, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.537934.

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Mukhopadhyay, Rituparna. "Chromatin Insulators and CTCF: Architects of Epigenetic States during Development." Doctoral thesis, Uppsala University, Department of Animal Development and Genetics, 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4241.

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<p>A controlled and efficient coordination of gene expression is the key for normal development of an organism. In mammals, a subset of autosomal genes is expressed monoallelically depending on the sex of the transmitting parent, a phenomenon known as genomic imprinting.</p><p>The imprinted state of the <i>H19</i> and <i>Igf2</i> genes is controlled by a short stretch of sequences upstream of <i>H19</i> known as the imprinting control region (ICR). This region is differentially methylated and is responsible for the repression of the maternally inherited <i>Igf2</i> allele. It harbors hypersens
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Fischer, Sabine. "Inducible systems for the characterization of insulating and repressing motifs." kostenfrei, 2009. http://d-nb.info/999863568/34.

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Books on the topic "CTCFL"

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Kemp, Joanne. CTCL 1996 survey on library internet services. Canadian Association of College and University Libraries, 1997.

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Amuthavalli, K. Regulation of Apoptosis in CTCL. Quadry, Fatima, 2023.

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Száraz, Tibor. Istqb Ctfl Practice Tests Level 2018 : ISTQB CTFL Practice Tests Book: Your Chance to Pass. Independently Published, 2019.

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Kung, Johnny Tsun-Yi. Genome-wide Analysis of Ctcf-RNA Interactions. 2014.

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Schlich, M. Softwaretesten Nach ISTQB CTFL 4. 0 Für Dummies. Wiley & Sons, Limited, John, 2024.

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Softwaretesten Nach ISTQB CTFL 4. 0 Für Dummies. Wiley & Sons, Incorporated, John, 2024.

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Monahan, Kevin Gunther. CTCF and Cohesin are Required for Protocadherin alpha Gene Expression. 2011.

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CERTIFIED TESTER FOUNDATION LEVEL GUIDE ISQI CTFL EXAM QUESTIONS and ANSWERS. Independently Published, 2020.

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Dosaj, Chhavi. Self-Study Guide for the ISTQB Agile Foundation Exam (CTFL-At). Independently Published, 2019.

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ISTQB Foundation Sample Exam Questions Certified Tester Foundation Level (CTFL) 2018 Syllabus. Independently Published, 2019.

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Book chapters on the topic "CTCFL"

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Nicolay, Jan P., and Claus-Detlev Klemke. "Personalized Treatment in Cutaneous T-Cell Lymphoma (CTCL)." In Personalized Treatment Options in Dermatology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-45840-2_4.

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Linnemann, Thomas, Carsten Brock, Katrin Sparbier, et al. "Identification of Epitopes for CTCL-Specific Cytotoxic T Lymphocytes." In Advances in Experimental Medicine and Biology. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5357-1_36.

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Kohl, O., R. Dummer, J. Gillessen, and G. Burg. "Interleukin-4 (IL-4) und kutane T-Zell-Lymphome (CTCL)." In Onkologische Dermatologie. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77690-8_10.

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Kuang, Shuzhen, and Liangjiang Wang. "Deep Learning of CTCF-Mediated Chromatin Loops in 3D Genome Organization." In Computational Advances in Bio and Medical Sciences. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-46165-2_7.

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Logie, Colin. "The Role of CTCF-Mediated Chromatin Looping in Enhancer-Promoter Communication." In Epigenetics in Biological Communication. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-59286-7_16.

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González-Buendía, Edgar, Rosario Pérez-Molina, Erandi Ayala-Ortega, Georgina Guerrero, and Félix Recillas-Targa. "Experimental Strategies to Manipulate the Cellular Levels of the Multifunctional Factor CTCF." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0856-1_5.

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Farrar, Dawn, Igor Chernukhin, and Elena Klenova. "Generation of Poly(ADP-ribosyl)ation Deficient Mutants of the Transcription Factor, CTCF." In Methods in Molecular Biology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-270-0_18.

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Salih, Hala, and Abuelgasim Sabah Elsaid Mohammed. "Evaluating the Certificate of Teaching English as a Foreign Language (CTEFL): A Way to Quality." In Second Language Learning and Teaching. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-43234-2_14.

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Chang, Tzu-Pei, Myra Kim, and Ivana Vancurova. "Analysis of TGFβ1 and IL-10 Transcriptional Regulation in CTCL Cells by Chromatin Immunoprecipitation." In Cytokine Bioassays. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0928-5_30.

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Halder, Anup Kumar, Abhishek Agarwal, Sevastianos Korsak, Karolina Jodkowska, and Dariusz Plewczynski. "ccLoopER: Deep Prediction of CTCF and cohesin Mediated Chromatin looping Using DNA Transformer Model." In Lecture Notes in Computer Science. Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-45170-6_91.

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Conference papers on the topic "CTCFL"

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Tian, Ye, Ruonan Luo, and Xiyin Zhang. "Music recommendation methods using the MR-CTCFI algorithm." In Second International Conference on Big Data, Computational Intelligence and Applications (BDCIA 2024), edited by Sos S. Agaian. SPIE, 2025. https://doi.org/10.1117/12.3059874.

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Mazumdar, Indrajit, and Jayanta Mukhopadhyay. "CTCF-UNet: 3D Concurrent Transformer-CNN Fusion U-Net for Brain Tumor Segmentation." In 2024 IEEE 21st India Council International Conference (INDICON). IEEE, 2024. https://doi.org/10.1109/indicon63790.2024.10958365.

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Benhattar, Jean, and Loredana Alberti. "Abstract 3383: Expression and role of BORIS/CTCFL in human cancer stem cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3383.

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Kingsley, Cameron, and Mohammad Poursina. "Computed Torque Control of Articulated Multibody Systems in the Generalized Divide and Conquer Algorithm Framework." In ASME 2015 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/detc2015-46853.

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An extension to the Generalized-Divide-and-Conquer Algorithm (GDCA) is presented in this paper in conjunction with the Computed-Torque-Control-Law (CTCL) to model and control fully actuated multibody systems. CTCL uses the inverse dynamics to provide control inputs to the system while, the dynamics of the system must be formed and solved in each iteration. Herein, the GDCA is extended to form and solve the inverse dynamics to find control torques. Further, this method is also extended to efficiently solve the equations of motion of the controlled system. This significantly reduces the complexi
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"CTCF and the evolution of genome spatial organization." In Биоинформатика регуляции и структуры геномов / системная биология. ИЦиГ СО РАН, 2024. http://dx.doi.org/10.18699/bgrs2024-1.3-13.

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Wang, Atom, Youqi Han, Peikun Chen, Nanyang Jia, and Mark D. Minden. "Abstract 2991: Cytoplasmic mislocalization of CTCF by NPM1c in acute myeloid leukemia resulting in inhibited CTCF regulatory functions generating aberrant genetic and epigenetic profiles." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-2991.

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Fang, Celestia, Zhenjia Wang, Carlos A. Martinez, Panagiotis Ntziachristos, and Chongzhi Zang. "Abstract 5181: Global alteration of CTCF binding in the cancer genome." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-5181.

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Fang, Celestia, Zhenjia Wang, Carlos A. Martinez, Panagiotis Ntziachristos, and Chongzhi Zang. "Abstract 5181: Global alteration of CTCF binding in the cancer genome." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-5181.

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Hilmi, Khalid, Chenxi Zhang, Zhenbao Yu, et al. "Abstract A12: CTCF facilitates DNA double-strand break repair by homologous recombination." In Abstracts: AACR Special Conference on DNA Repair: Tumor Development and Therapeutic Response; November 2-5, 2016; Montreal, QC, Canada. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1557-3125.dnarepair16-a12.

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Peng, Wan-Xin, and Yin-Yuan Mo. "Abstract 1821: LINC00346 regulates c-Myc transcriptional activity through CTCF in pancreatic cancer." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1821.

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