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Relevant bibliographies by topics / Cystathionine-gamma-lyase (CSE)

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Academic literature on the topic 'Cystathionine-gamma-lyase (CSE)'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Cystathionine-gamma-lyase (CSE)"

1

Roglans, Núria, Elena Fauste, Roger Bentanachs, et al. "Bempedoic Acid Restores Liver H2S Production in a Female Sprague-Dawley Rat Dietary Model of Non-Alcoholic Fatty Liver." International Journal of Molecular Sciences 24, no. 1 (2022): 473. http://dx.doi.org/10.3390/ijms24010473.

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We previously demonstrated that treatment with BemA (bempedoic acid), an inhibitor of ATP citrate lyase, significantly reduces fatty liver in a model of liver steatosis (HFHFr—female Sprague-Dawley rat fed a high-fat high-fructose diet). Since the hepatic production of the gasotransmitter H2S is impaired in liver disorders, we were interested in determining if the production of H2S was altered in our HFHFr model and whether the administration of BemA reversed these changes. We used stored liver samples from a previous study to determine the total and enzymatic H2S production, as well as the ex

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2

Zhao, Shuang, Céline Deslarzes-Dubuis, Severine Urfer, Martine Lambelet, Sébastien Déglise, and Florent Allagnat. "Cystathionine Gamma Lyase Is Regulated by Flow and Controls Smooth Muscle Migration in Human Saphenous Vein." Antioxidants 12, no. 9 (2023): 1731. http://dx.doi.org/10.3390/antiox12091731.

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The saphenous vein is the conduit of choice for bypass grafting. Unfortunately, the hemodynamic stress associated with the arterial environment of the bypass vein graft leads to the development of intimal hyperplasia (IH), an excessive cellular growth and collagen deposition that results in restenosis and secondary graft occlusion. Hydrogen sulfide (H2S) is a ubiquitous redox-modifying gasotransmitter that inhibits IH. H2S is produced via the reverse trans-sulfuration pathway by three enzymes: cystathionine γ-lyase (CSE), cystathionine β-synthase (CBS) and 3-mercaptopyruvate sulfurtransferase

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3

Arroyo Almenas, Francisco, Gábor Törő, Peter Szaniszlo та ін. "Cystathionine Gamma-Lyase Regulates TNF-α-Mediated Injury Response in Human Colonic Epithelial Cells and Colonoids". Antioxidants 13, № 9 (2024): 1067. http://dx.doi.org/10.3390/antiox13091067.

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Cystathionine gamma-lyase (CSE) and TNF-α are now recognized as key regulators of intestinal homeostasis, inflammation, and wound healing. In colonic epithelial cells, both molecules have been shown to influence a variety of biological processes, but the specific interactions between intracellular signaling pathways regulated by CSE and TNF-α are poorly understood. In the present study, we investigated these interactions in normal colonocytes and an organoid model of the healthy human colon using CSE-specific pharmacological inhibitors and siRNA-mediated transient gene silencing in analytical

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4

Toro, Gabor, Peter Szaniszlo, Francisco Arroyo Almenas, et al. "THE FUNCTION OF THE CYSTATHIONINE-GAMMA-LYASE/HYDROGEN SULFIDE AXIS IN THE PATHOGENESIS OF ULCERATIVE COLITIS." Inflammatory Bowel Diseases 30, Supplement_1 (2024): S62. http://dx.doi.org/10.1093/ibd/izae020.129.

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Abstract INTRODUCTION Current combination therapies in ulcerative colitis (UC) target different inflammatory cascades, including tumor necrosis factor-alpha (TNF-alpha) signaling. However, most patients do not achieve long-term remission, suggesting that the molecular pathophysiology of UC is still poorly understood. Endogenously produced hydrogen sulfide (H2S) has emerged as a critical physiological mediator of gastrointestinal homeostasis, limiting mucosal inflammation and promoting tissue healing in response to epithelial injury. Inhibition of cystathionine-gamma-lyase (CSE)-dependent H2S p

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5

Jamaluddin, Mohammad, Aline Haas de Mello, Nisha Tapryal, Tapas K. Hazra, Roberto P. Garofalo, and Antonella Casola. "NRF2 Regulates Cystathionine Gamma-Lyase Expression and Activity in Primary Airway Epithelial Cells Infected with Respiratory Syncytial Virus." Antioxidants 11, no. 8 (2022): 1582. http://dx.doi.org/10.3390/antiox11081582.

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Cystathionine-y-lyase (CSE) is a critical enzyme for hydrogen sulfide (H2S) biosynthesis and plays a key role in respiratory syncytial virus (RSV) pathogenesis. The transcription factor NRF2 is the master regulator of cytoprotective and antioxidant gene expression, and is degraded during RSV infection. While some evidence supports the role of NRF2 in CSE gene transcription, its role in CSE expression in airway epithelial cells is not known. Here, we show that RSV infection decreased CSE expression and activity in primary small airway epithelial (SAE) cells, while treatment with tert-butylhydro

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6

Ilieva, Bilyana, Elena Marinova, Hristo Gagov та Rossitza Konakchieva. "Basal and PGF2α-stimulated secretion of pro-inflammatory cytokines from 3T3-L1 adipocyte-like cells". BioDiscovery 20 (1 серпня 2017): e17757. https://doi.org/10.3897/biodiscovery.20.e17757.

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Adipocytes were recently identified as an important source of endocrine and paracrine mediators, regulating the metabolism and activity of various cell types and body functions. 3T3-L1 preadipocytes are useful model for physiological, pharmacological and cell signaling studies. Differentiation of 3T3-L1 murine fibroblasts into adipocyte-like cells was conducted in presence of IBMX, dexamethasone and insulin and demonstrated by Oil Red O staining of accumulated lipid droplets. Using Inflammatory Multi- Analyte Cytokines ELISArray Kit we investigated the release of cytokines under basal conditio

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7

Zhong, Xin, Yuwen Wang, Jichao Wu, et al. "Calcium Sensing Receptor Regulating Smooth Muscle Cells Proliferation Through Initiating Cystathionine-Gamma-Lyase/Hydrogen Sulfide Pathway in Diabetic Rat." Cellular Physiology and Biochemistry 35, no. 4 (2015): 1582–98. http://dx.doi.org/10.1159/000373973.

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Aims: Hydrogen sulfide (H2S) inhibits the proliferation of vascular smooth muscle cells (VSMCs). However, how cystathionine-gamma-lyase (CSE), a major enzyme that produces H2S, is regulated remains unknown. Whether calcium-sensing receptor (CaSR) inhibits the proliferation of VSMCs by regulating the endogenous CSE/H2S pathway in diabetic rat has not been previously investigated. Methods and Results: The morphological and ultrastructure alterations were tested by transmission electron microscopy, changes in the H2S concentration and the relaxation of the mesenteric secondary artery loop of diab

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8

Cheung, Sau Ha, Wai Kei Kwok, Ka Fai To, and James Yun Wong Lau. "Anti-Atherogenic Effect of Hydrogen Sulfide by Over-Expression of Cystathionine Gamma-Lyase (CSE) Gene." PLoS ONE 9, no. 11 (2014): e113038. http://dx.doi.org/10.1371/journal.pone.0113038.

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9

Thanki, Ketan K., Michael E. Nicholls, Romain Villeger, et al. "Tu1766 - Macrophage Interleukin-10 (IL-10) Production is Dependent on Cystathionine-Gamma-Lyase (CSE) Expression." Gastroenterology 154, no. 6 (2018): S—1013—S—1014. http://dx.doi.org/10.1016/s0016-5085(18)33392-4.

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10

Gaddam, Ravinder, Stephen Chambers, Robin Fraser, et al. "Cystathionine-Gamma-Lyase-Derived Hydrogen Sulfide-Regulated Substance P Modulates Liver Sieve Fenestrations in Caecal Ligation and Puncture-Induced Sepsis." International Journal of Molecular Sciences 20, no. 13 (2019): 3191. http://dx.doi.org/10.3390/ijms20133191.

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Cystathionine-γ-lyase (CSE) is a hydrogen sulfide (H2S)-synthesizing enzyme that promotes inflammation by upregulating H2S in sepsis. Liver sinusoidal endothelial cells (LSECs) are fenestrated endothelial cells (liver sieve) that undergo alteration during sepsis and H2S plays a role in this process. Substance P (SP) is encoded by the preprotachykinin A (PPTA) gene, and promotes inflammation in sepsis; however, its regulation by H2S is poorly understood. Furthermore, the interaction between H2S and SP in modulating LSEC fenestrations following sepsis remains unclear. This study aimed to investi

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