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Books on the topic 'Demenza frontotemporale'

Author: Grafiati

Published: 9 March 2023

Last updated: 31 July 2025

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1

R, Hodges John, ed. Frontotemporal dementia syndromes. Cambridge University Press, 2007.

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R, Hodges John, ed. Frontotemporal dementia syndromes. Cambridge University Press, 2007.

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3

F, Lebert, and Pasquier F, eds. Frontotemporal dementia. ICG Publications, 1996.

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4

Dickerson, Bradford C., ed. Hodges' Frontotemporal Dementia. Cambridge University Press, 2016. http://dx.doi.org/10.1017/cbo9781316091586.

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Hodges, John R., ed. Frontotemporal Dementia Syndromes. Cambridge University Press, 2007. http://dx.doi.org/10.1017/cbo9781316135457.

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Roberson, Erik D., ed. Alzheimer's Disease and Frontotemporal Dementia. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-744-0.

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7

W, Mates Andrea, Mikesell Lisa, and Smith Michael Sean, eds. Language, interaction and frontotemporal dementia: Reverse engineering the social mind. Equinox Pub. Ltd., 2010.

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8

Griffiths, Helen L. Communication disorder in dementia due to frontotemporal cerebral atrophy: A linguistic and cognitive analysis : a comparative study with Alzheimer's disease. University of Manchester, 1996.

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9

Hodges, John R. Frontotemporal Dementia Syndromes. University of Cambridge ESOL Examinations, 2016.

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10

Hodges, John R. Frontotemporal Dementia Syndromes. Cambridge University Press, 2011.

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11

Frontotemporal Dementia Syndromes. Cambridge University Press, 2007.

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12

Deramecourt, Vincent, Florence Lebert, and Florence Pasquier. Frontotemporal dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0036.

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Frontotemporal dementia (FTD) is the second most common form of dementia in persons younger than 65 years after Alzheimer’s disease. The FTD spectrum is characterized by clinical, molecular and genetic heterogeneity. Core features of FTD are behavioural and language manifestations and the clinical spectrum of FTD currently includes a behavioural variant, progressive nonfluent aphasia and semantic dementia. The most common behavioural features are disinhibition, apathy, loss of empathy, hyperorality and perseveration. Neuroimaging usually demonstrates focal atrophy and hypometabolism in the ant

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13

Miller, Bruce L. Frontotemporal Dementia. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780195380491.001.0001.

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Frontotemporal Dementia provides an in-depth look at the history, various types, genetics, neuropathology and psychosocial aspects of one of the most common but least understood causes of dementia, frontotemporal lobar degeneration, from one of the world's leading centers for the study of dementia. Aided by the latest research in diagnosis, mechanism and treatment, this resource captures the rich and quickly changing landscape of a devastating neurodegenerative disease, and offers up-to-date clinical advice for patient care. Frontotemporal dementia, in particular, raises psychological and phil

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14

Frontotemporal Dementia. Oxford University Press, Incorporated, 2014.

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15

Frontotemporal Dementia. Oxford University Press, Incorporated, 2014.

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16

Evans, Lauren, Ellen Steinbart, NetCE, and CE Resource. Frontotemporal Dementia. CE Resource, Incorporated, 2021.

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17

Hodges' Frontotemporal Dementia. Cambridge University Press, 2016.

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18

Dickerson, Bradford C. Hodges' Frontotemporal Dementia. Cambridge University Press, 2016.

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19

Dickerson, Bradford C. Hodges' Frontotemporal Dementia. Cambridge University Press, 2015.

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20

Brookes, Alpha. Dementia Signs : Frontotemporal Dementia: Dementia Meaning. Independently Published, 2021.

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21

Beller, Jerry, and Beller Health. BvFTD Behavioral Variant Dementia: Frontotemporal Dementia. Independently Published, 2019.

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22

Graff-Radford, Jonathan, and Keith A. Josephs. Frontotemporal Lobar Degeneration. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0018.

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Frontotemporal dementia (FTD) is an uncommon but important form of degenerative disease characterized by clinical syndromes that result from degeneration of the frontal and temporal lobes. FTD is divided based on clinical presentation into behavioral variant FTD (bvFTD), semantic dementia, and progressive nonfluent/agrammatic aphasia. Several recent studies have advanced our knowledge of the genetics of FTD, with the three most common FTD genes being microtubule-associated protein tau (MAPT) and progranulin (GRN), and a noncoding repeat expansion in C9ORF72. Tau and TDP-43 are the most common

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23

Roberson, Erik D. Alzheimer's Disease and Frontotemporal Dementia: Methods and Protocols. Humana Press, 2016.

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24

Briggs, John, Jerry Beller, Beller Health, and Brain Research. Frontotemporal Related Dementias: Behavioral Variant Frontotemporal Dementia and Progressive Supranuclear Palsy. Independently Published, 2020.

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25

Beller, Jerry, and Beller Health. 2019 Primary Progressive Aphasia: Frontotemporal Dementia. Independently Published, 2019.

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26

Kaplan, Tamara, and Tracey Milligan. Dementia 1: Defining Dementia, Alzheimer’s Disease and Frontotemporal Dementia (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190650261.003.0009.

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The video in this chapter explores dementia, and focuses on definitions of dementia, Alzheimer’s disease and Frontotemporal Dementia. Dementia is defined as a cognitive decline in one or more cognitive domains including memory, language, attention, visuospatial processing and social behavior. Two hallmark pathologic features of Alzheimer’s disease (AD) are plaques, which are formed from amyloid and neurofibrillary tangles, which involve tau, whereas symptoms of Frontotemporal Dementia (FTD) may include behavioral changes, apathy and disinhibition and ritualistic or repetitive behaviors. Langua

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27

Beller, Jerry, and Beller Health. FTD Dementia : Frontotemporal Dementia: Behavioral Variant Primary Progressive Aphasia. Independently Published, 2019.

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28

Jr, Presz Walter. Engineering Approach Improves Care for Frontotemporal Dementia. BookBaby, 2023.

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29

Frontotemporal disorders: Information for patients, families, and caregivers. National Institutes of Health, National Institute on Aging, 2010.

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30

Alzheimer's disease and frontotemporal dementia: Methods and protocols. Humana Press, 2011.

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31

Houlden, Henry, Alan Edward Renton, and Francesca Luisa Conforti, eds. Multifaceted Genes in Amyotrophic Lateral Sclerosis-Frontotemporal Dementia. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-904-2.

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32

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Dementia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0011.

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This chapter discusses the clinical features and evidence-based pharmacological management of dementia disorders (Alzheimer’s disease (AD), vascular dementia, dementia with Lewy bodies (DLB), Parkinson’s disease dementia (PDD), frontotemporal dementia (FTD), mixed dementia, and mild cognitive impairment (MCI)).

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33

Beller, Jerry, and Beller Health. 14 Dementia Types: Alzheimers Lewy Body Dementia Frontotemporal Dementia Vascular Dementia Huntington's Disease Other Dementias. Independently Published, 2019.

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34

Beller, Jerry, and Beller Health. 2019 Dementia Overview: Alzheimers , Lewy Body Dementia , Frontotemporal Dementia , Vascular Dementia , Huntington's Disease , Other Dementias. Independently Published, 2019.

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35

When Love Meets Dementia: Frontotemporal Degeneration and the Family. McFarland & Company, Incorporated Publishers, 2019.

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36

Strong, Michael J. Amyotrophic Lateral Sclerosis and the Frontotemporal Dementias. Oxford University Press, 2012.

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37

Language, Interaction, and Frontotemporal Dementia: Reverse Engineering the Social Mind. Equinox Publishing (Indonesia), 2013.

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38

Jr, Moncivais Manuel Manny. Just Squeeze My Hand: A Caregiver's Experience with Frontotemporal Dementia. Christian Faith Publishing, 2021.

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39

Jr, Moncivais Manuel Manny. Just Squeeze My Hand: A Caregiver's Experience with Frontotemporal Dementia. Christian Faith Publishing, 2021.

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40

Language, interaction and frontotemporal dementia: Reverse engineering the social mind. Equinox Pub., 2010.

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41

Dementia: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/dementia.

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Dementia is a condition that affects memory, thinking, behavior, and the ability to manage everyday life. This article offers a complete guide to understanding dementia, aimed at the general public, patients, and caregivers. It begins by explaining what dementia is and outlines the different types, including Alzheimer’s disease, vascular dementia, Lewy body dementia, frontotemporal dementia, and mixed dementia. The article then explores risk factors such as age, genetics, heart health, and lifestyle habits. It continues with a description of common symptoms like memory loss and confusion, foll

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42

Hilsabeck, Robin C., and Gayle Y. Ayers, eds. Dementia. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/med/9780197690024.001.0001.

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Abstract Adults aged 65 and older are the fastest-growing segment of the United States population. This increase in older adults will result in a greater number of individuals with age-related neurocognitive disorders such as Alzheimer’s, vascular disease, and frontotemporal lobular degeneration. The purpose of this book is to provide trainees and early career professionals, particularly in psychiatry, psychology, neurology, geriatrics, family medicine, and internal medicine, with the information necessary to care for the often complex clinical presentations of older adults with mild cognitive

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43

Utianski, Rene L. Primary Progressive Aphasia and Other Frontotemporal Dementias: Diagnosis and Treatment of Associated Communication Disorders. Plural Publishing, Incorporated, 2019.

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44

Burrell, James R., and John R. Hodges. Dementia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199658602.003.0010.

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Cognitive neurology has exploded over the last century, and especially over the last 20 years. From the distinction of dementia as a pathological entity, rather than just ‘normal’ ageing, to more sophisticated sub-classification of dementia syndromes, much has been learned, though great challenges remain. From an incredible array of worthy research studies, ten landmark papers in the field of dementia are presented in this chapter. With regard to Alzheimer’s disease, the following are discussed: the initial description of the disease, both clinically and pathologically; the development of mean

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45

I Can't Hear the Music Anymore: One Family's Journey with Frontotemporal Dementia. 2007.

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46

Hodges, John R. Delirium and Dementia. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198749189.003.0002.

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Delirium and dementia affect one or more cognitive domains with a distributed neural basis—attention, memory, and executive function—in some instances accompanied by more focal cognitive deficits. Patients with one, or both, of these conditions constitute the commonest presentation in behavioural neurology and in geriatric psychiatry. This chapter first describes the core characteristics and causes of delirium. This is followed by a description of the major causes of dementia notably, Alzheimer’s disease, frontotemporal dementia, Huntington’s disease, dementia with Lewy bodies, and progressive

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47

Zahn, Roland, and Alistair Burns. Dementia disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198779803.003.0001.

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This chapter provides a brief overview of the different forms of dementia syndromes and provides a simple algorithm for initial differential diagnosis. Rapidly progressive dementias have to be excluded which require specific investigations to detect Creutzfeldt–Jakob as well as inflammatory and autoimmune diseases. A lead symptom-based approach in patients with slowly progressive cognitive and behavioural impairments without neurological symptoms is applied: progressive and primary impairments in recent memory are characteristic of typical Alzheimer’s dementia, primary behavioural changes poin

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48

Gan, Li. Cellular Mechanisms of Dementia. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0054.

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Neurodegenerative dementias, including Alzheimer’s disease (AD), Parkinson’s disease (PD) and Frontotemporal dementia (FTD), pose enormous challenges for our aging society. Genetic and mechanistic studies have revealed common molecular and cellular pathways, including imbalanced proteostasis and aberrant innate immune responses. Key pathogens in AD, PD, and FTD accumulate and spread from one brain region to another, resulting in network dysfunction and cognitive decline. These diseases are multifactorial, caused by interactions among multiple genetic, epigenetic, and environmental factors and

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49

Briggs, John, Jerry Beller, Beller Health, and Brain Research. BvFTD Behavioral Variant Frontotemporal Dementia: Guide for Doctors, Nurses, Patients, Families, and Caregivers. Independently Published, 2020.

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50

Piggott, Margaret Ann. Neurochemical pathology of dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0007.

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This chapter considers the neurodegenerative disorders Alzheimer’s disease (AD), Lewy body dementias (dementia with Lewy bodies (DLB) and Parkinson’s disease dementia(PDD)), frontotemporal dementia (FTD); and also vascular dementia (VaD) which results from cerebrovascular disease. These different conditions, which give rise to dementia syndromes, each have distinct neurochemical pathologies, with important implications for treatment. As increased age is the common risk factor generally associated with dementing illnesses, neurochemical changes are set in the context of the changes which occur

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