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Journal articles on the topic 'Diabetes Mellitus, Beta Cells, ACE2, SARS-CoV-2'

Author: Grafiati

Published: 14 March 2023

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1

Nimje, Prajakta D., Vidya Wasnik (Thatere), and Sumeeta Jain. "MANAGEMENT THROUGH AYURVEDIC MEDICATIONS AND LIFESTYLE MODIFICATION (PATHYASEVANA AND YOGASANA) IN A CASE OF POST-COVID COMPLICATION DIABETES MELLITUS (TYPE 2 DM): A SINGLE CASE STUDY." International Ayurvedic Medical Journal 9, no. 10 (2021): 2619–28. http://dx.doi.org/10.46607/iamj5409102021.

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Diabetes is an asymptomatic disease in most people, so there could be a good number of people who may not be aware of their diabetes before they caught COVID-19. Some studies state that in poorly resourced countries, as many as 50% of people with chronic illnesses, such as diabetes, are undiagnosed. Theoretically, COVID-19 can also cause diabetes as the pancreas have ACE2 receptors, which can enable SARS COV2 to enter the pancreatic beta cells, resulting in structural and functional damage. The present study is carried out to know the traditional Ayurvedic treatment for Diabetes Mellitus. For

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Junhai, Gazi Rayeeda, Hasi Rani Saha, and Bidhan Chandra Sarkar. "Prevalence of COVID-19 Among Older People with Type 2 Diabetes Mellitus: A Systematic Review." International Journal of Science and Healthcare Research 7, no. 3 (2022): 273–88. http://dx.doi.org/10.52403/ijshr.20220738.

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Background: On March 11, 2020, the WHO proclaimed the Coronavirus Disease 2019 (COVID-19) pandemic owing to coronavirus-2 (SARS-CoV-2). SARS-CoV-2 is a zoonotic virus that may be spread from bats to humans through airborne droplets and aerosols. SARS-CoV-2 spike protein has a high binding affinity for ACE2 receptors, widely expressed throughout the respiratory system, notably in epithelial lung cells. ACE2 receptors are found in intestinal mucosal, endothelial, heart, renal epithelial as well as cerebral neuronal cells, explaining COVID-19 extrapulmonary symptoms like diarrhea, nausea, vomitin

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Kadir, Nursin Abdul, and Ida Parwati. "COVID-19 (Symptomatic Non-Respiratory) with Type 2 Diabetes Mellitus." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 29, no. 1 (2023): 101–6. http://dx.doi.org/10.24293/ijcpml.v29i1.1863.

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COVID-19 is a respiratory infection caused by a new strain of Coronavirus, Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2), which is highly contagious, primarily through respiratory droplets and contact. Typical symptoms include fever, cough, and shortness of breath. Weakness, nausea, and vomiting are often accompanied by respiratory symptoms but are sometimes confusing when these symptoms occur without respiratory symptoms. COVID-19 can affect any age group, are more common in adults and males and increase in patients with comorbidities. One of the most common comorbidities is Di

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Casillas Santana, Miguel Angel, Juan Antonio Arreguín Cano, Alejandro Dib Kanán, et al. "Should We Be Concerned about the Association of Diabetes Mellitus and Periodontal Disease in the Risk of Infection by SARS-CoV-2? A Systematic Review and Hypothesis." Medicina 57, no. 5 (2021): 493. http://dx.doi.org/10.3390/medicina57050493.

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The objective of this article was to conduct a systematic review of the literature to contrast the existing evidence regarding the relationship between periodontal disease (PD) and diabetes mellitus (DM) with the possibly increased risk of SARS-CoV-2 infection, as well as to establish a hypothesis that explains the ways in which this interaction could take place. A literature search up from 1 January 2020 to 21 March 2021 was conducted in three electronic databases, namely, PubMed, Web of Science, and Scopus, in order to identify studies on periodontal disease alone or in conjunction with diab

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Beliard, Kara A., Mabel Yau, Meredith Wilkes, Christopher Joseph Romero, Elizabeth Wallach, and Robert Rapaport. "SARS-CoV-2 Infection Related Diabetes Mellitus." Journal of the Endocrine Society 5, Supplement_1 (2021): A397. http://dx.doi.org/10.1210/jendso/bvab048.808.

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Abstract Introduction: SARS-Cov-2 (severe acute respiratory distress syndrome- coronavirus 2) viral infection has a predilection for pancreatic beta cells causing insulin deficiency. Studies from the SARS-CoV outbreak in 2003 highlighted the relationship between SARS-CoV and ACE-2 (angiotensin-converting enzyme 2) receptors in pancreatic islet cells. We describe a pediatric patient who developed Diabetes Mellitus after exposure to the Sars-CoV-2 virus. Case Report: A previously healthy 13-year-old female of Mexican descent was found to be hyperglycemic at her annual visit. The patient endorsed

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Pramanik, Parthiba, and Purushottam Pramanik. "Diabetes mellitus augments the complications of patients with COVID-19: a review." International Journal of Research in Medical Sciences 8, no. 7 (2020): 2716. http://dx.doi.org/10.18203/2320-6012.ijrms20202925.

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Corona virus disease 2019 (COVID-19) is current pandemic infection caused by RNA virus named severe acute respiratory syndrome coronavirus-2 (SARS Cov-2). The lungs are the organs most affected by COVID-19 and people were died due to severe acute respiratory s syndrome, pneumonia and multi-organs failure. Fatality rate was more, those who suffer in chronic diseases including diabetes mellitus (DM). As COVID-19 pandemic is accelerating , it is important to understand the molecular mechanism through which DM increases the severity related to COVID-19 to able to design more appropriate therapy. T

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Wicik, Zofia, Ceren Eyileten, Daniel Jakubik, et al. "ACE2 Interaction Networks in COVID-19: A Physiological Framework for Prediction of Outcome in Patients with Cardiovascular Risk Factors." Journal of Clinical Medicine 9, no. 11 (2020): 3743. http://dx.doi.org/10.3390/jcm9113743.

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Background: Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection (coronavirus disease 2019; COVID-19) is associated with adverse outcomes in patients with cardiovascular disease (CVD). The aim of the study was to characterize the interaction between SARS-CoV-2 and Angiotensin-Converting Enzyme 2 (ACE2) functional networks with a focus on CVD. Methods: Using the network medicine approach and publicly available datasets, we investigated ACE2 tissue expression and described ACE2 interaction networks that could be affected by SARS-CoV-2 infection in the heart, lungs and nervous s

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Pelle, Maria Chiara, Isabella Zaffina, Stefania Lucà, et al. "Endothelial Dysfunction in COVID-19: Potential Mechanisms and Possible Therapeutic Options." Life 12, no. 10 (2022): 1605. http://dx.doi.org/10.3390/life12101605.

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SARS-CoV-2, a novel coronavirus found in Wuhan (China) at the end of 2019, is the etiological agent of the current pandemic that is a heterogeneous disease, named coronavirus disease 2019 (COVID-19). SARS-CoV-2 affects primarily the lungs, but it can induce multi-organ involvement such as acute myocardial injury, myocarditis, thromboembolic eventsandrenal failure. Hypertension, chronic kidney disease, diabetes mellitus and obesity increase the risk of severe complications of COVID-19. There is no certain explanation for this systemic COVID-19 involvement, but it could be related to endothelial

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Parrey, Ashaq, Abir aijaz, Mohd Ismail, et al. "New Onset Diabetes and Its Incidence in Severe COVID 19 Disease A Single Centre Study From Kashmir." Journal of Endocrinology and Diabetes 8, no. 2 (2012): 1–4. http://dx.doi.org/10.15226/2374-6890/8/2/001152.

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Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the novel coronavirus that causes coronavirus disease 2019 (COVID-19), was first reported in Wuhan, China, in December 2019 and has spread worldwide. SARS-CoV-2 is a positivestranded RNA virus that is enclosed by a protein containing lipid bilayer with a single-stranded RNA genome; SARS-CoV-2 has 82% homology with human SARS-CoV, which causes severe acute respiratory syndrome.SARS-CoV-2, virus binds to angiotensinconverting enzyme 2 (ACE2) receptors, which are expressed in key metabolic organs and tissues, including pancreatic beta

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Blîndu, Emanuel, Renata Gerculy, Diana Opincariu, Daniel Cernica, and Imre Benedek. "Cessation of Renin-Angiotensin System Antagonists During the SARS-CoV-2 Pandemic – Do We Have the Evidence?" Journal of Interdisciplinary Medicine 5, no. 3 (2020): 105–9. http://dx.doi.org/10.2478/jim-2020-0022.

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AbstractThe aim of this review is to provide a short update on whether treatment with angiotensin-converting enzyme inhibitors (ACEIs) or angiotensin receptor blockers (ARBs) has beneficial or harmful effects in patients infected with SARS-CoV-2. Epidemiological studies have shown that SARS-CoV-2 infects all age groups, presenting a higher incidence in elderly patients with various comorbidities such as hypertension, diabetes mellitus, and cardiovascular diseases. A large proportion of these patients are treated with ACEIs and ARBs. Since it has been demonstrated that SARS-CoV-2 uses angiotens

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Ivantsova, N. L., A. G. Beliakova, M. I. Dmitrievskaya, A. N. Useinova, V. M. Melnikova, and K. I. Abramova. "PECULIAR PROPERTIES OF USAGE OF DRUGS THAT AFFECT THE RENIN-ANGIOTENSIN SYSTEM IN PATIENTS WITH COVID-19." Crimea Journal of Experimental and Clinical Medicine 11, no. 2 (2022): 75–81. http://dx.doi.org/10.37279/2224-6444-2021-11-2-75-81.

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Currently, the main task of public health authorities is a developing of coordinated global measures to prepare health systems to deal with the spread of coronavirus infection, an accompanying concern has been identified that is of particular interest to clinicians and researchers about hypertension. Hypertension, coronary artery disease and diabetes mellitus, especially in the elderly, increase susceptibility to SARS-CoV-2 infection. Antihypertensive drugs decrease blood pressure due to dilating blood vessels and decreasing cardiac output. One of the groups of these drugs includes inhibitors

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Perlman, Jordan E., and Justin B. Echouffo-Tcheugui. "A Case of Possible SARS-COV-2 Induced Beta-Cell Failure." Journal of the Endocrine Society 5, Supplement_1 (2021): A359—A360. http://dx.doi.org/10.1210/jendso/bvab048.732.

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Abstract Background: There are several known viral triggers of ketosis-prone diabetes, including SARS-1 and HHV-81,2. SARS-COV-2 can bind ACE-2 receptors on the beta-cell causing destruction and acute impairment of insulin secretion3. There is accruing evidence to suggest that COVID-19 infection can worsen preexisting diabetes or induce new disease4. Clinical Case: A 40-year-old Hispanic male presented to the ER complaining of fatigue, polyuria, and polydipsia. A screening COVID-19 PCR was positive but he denied URI symptoms. His admission labs were notable for hyperglycemia (434 mg/dL; n 71–9

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Rivera Nazario, Ivan Augusto, Arnaldo Nieves Ortiz, Jose Ayala Rivera, et al. "An Atypical Presentation of Hyperosmolar Hyperglycemic State Induced by SARS CoV 2." Journal of the Endocrine Society 5, Supplement_1 (2021): A366—A367. http://dx.doi.org/10.1210/jendso/bvab048.746.

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Abstract Hyperglycemic emergencies such as Diabetic Ketoacidosis (DKA) or Hyperosmolar Hyperglycemic State (HHS) are commonly precipitated by infectious processes. Severe Acute Respiratory Syndrome-Coronavirus 2 (SARS-CoV-2) is a novel infectious process prompting hyperglycemic crisis. SARS-CoV-2 at the level of the lungs affects ACE2 functioning which in turns decrease the B cells proliferation at the pancreas and hinders insulin secretion. Advanced age and comorbidities such as hypertension, cardiovascular disease and diabetes mellitus are considered to be a risk factors for severe illness a

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Zayas, Francisco Jose, Marianne Hernandez-Negron, and Michelle Marie Mangual Garcia. "Development of Diabetes Type 3 After SARS-COV-2 Pancreatic B Cell Injury." Journal of the Endocrine Society 5, Supplement_1 (2021): A373. http://dx.doi.org/10.1210/jendso/bvab048.760.

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Abstract SARS-COV-2 has caused millions of deaths in less than one year, yet little is known about the long-term consequences survivors may suffer. The novel coronavirus uses the ACE2 receptor to infect human cells, allowing it to target organ systems with such receptors including the respiratory, cardiovascular, gastrointestinal and endocrine system. The purpose of this case report is to describe the long-term implications COVID-19 may cause in the endocrine system. A 46-year-old woman was referred to our clinic due to abrupt uncontrolled blood glucose levels ranging from 200-550mg/dL after b

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Cavalcante, Letícia Carla de Lima, Larissa Cristina de Lima Cavalcante, Maria Vitória Menezes Coutinho, Rebeca Montenegro De Lacerda, Rodrigo de Oliveira Arakaki, and Cristiane Monteiro Da Cruz. "Analysis of the infectional profile of diabetic patients infected by COVID 19 in the period of 2020 – 2021 Análise do perfil de infecção dos pacientes diabéticos infectados pela COVID 19 no período de 2020-2021." Brazilian Journal of Health Review 4, no. 5 (2021): 19930–40. http://dx.doi.org/10.34119/bjhrv4n5-119.

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The COVID-19 disease has as its etiological agent the SARS-CoV-2, a RNA virus that causes severe respiratory syndrome. Diabetes mellitus is one of the significant risk factors for COVID-19 due to the weakened immune response of these patients and widely distribution of ACE2 in diferent organs. ACE2 is a receptor that antagonizes the RAS system, and is related to the clinical symptoms of COVID 19 .Its expression in the islet cells and exocrine pancreas suggest the susceptibility for SARS-CoV 2 infection in diabetic patients. This stydy analyses the infeccional profile of diabetic patients infec

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Cavalcante, Letícia Carla de Lima, Larissa Cristina de Lima Cavalcante, Maria Vitória Menezes Coutinho, Rebeca Montenegro De Lacerda, Rodrigo de Oliveira Arakaki, and Cristiane Monteiro Da Cruz. "Analysis of the infectional profile of diabetic patients infected by COVID 19 in the period of 2020 – 2021 Análise do perfil de infecção dos pacientes diabéticos infectados pela COVID 19 no período de 2020-2021." Brazilian Journal of Health Review 4, no. 5 (2021): 19930–40. http://dx.doi.org/10.34119/bjhrv4n5-119.

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The COVID-19 disease has as its etiological agent the SARS-CoV-2, a RNA virus that causes severe respiratory syndrome. Diabetes mellitus is one of the significant risk factors for COVID-19 due to the weakened immune response of these patients and widely distribution of ACE2 in diferent organs. ACE2 is a receptor that antagonizes the RAS system, and is related to the clinical symptoms of COVID 19 .Its expression in the islet cells and exocrine pancreas suggest the susceptibility for SARS-CoV 2 infection in diabetic patients. This stydy analyses the infeccional profile of diabetic patients infec

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Komine, Mayumi, Tuba Mussarat Ansary, Md Razib Hossain, Koji Kamiya, and Mamitaro Ohtsuki. "Inflammation Causes Exacerbation of COVID-19: How about Skin Inflammation?" International Journal of Molecular Sciences 23, no. 20 (2022): 12260. http://dx.doi.org/10.3390/ijms232012260.

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COVID-19 is a recently emerged viral infection worldwide. SARS-CoV-2, the causative virus, is believed to have emerged from bat coronaviruses, probably through host conversion. The bat coronavirus which has the highest gene homology to SARS-CoV-2 specifically infects deep forest bats in China whose habitat extends through the Middle East to Southern Europe. Host conversion might have occurred due to the deforestation by humans exposing wild bats to the environment they had never encountered before. SARS-CoV-2 infects cells through two mechanisms: through its receptor ACE2 with the help of enzy

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Vikulova, O. K., Zamira Zuraeva, L. V. Nikankina, and M. V. Shestakova. "The role of renin-angiotensin system and angiotensin-converting enzyme 2 (ACE2) in the development and course of viral infection COVID-19 in patients with diabetes mellitus." Diabetes mellitus 23, no. 3 (2020): 242–49. http://dx.doi.org/10.14341/dm12501.

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The role of renin-angiotensin system (RAS) in general and angiotensin-converting enzyme 2 (ACE2) in particular in the pathogenesis and course of viral infection caused by SARS-CoV-2 (COVID-19) is of particular interest. This is due not only to the fact that ACE2 is a receptor for the virus the target cells. RAS hyperactivation in patients with arterial hypertension, cardiovascular disease and diabetes mellitus, is considered one of the most important factors for a more severe infection in persons with concomitant pathology. In addition, the effects of PAS blockage with angiotensin converting e

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Rida, Rima, Paola Rios, and Alex Manzano. "A Rare Cause of Diabetes: COVID-19." Journal of the Endocrine Society 5, Supplement_1 (2021): A362. http://dx.doi.org/10.1210/jendso/bvab048.737.

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Abstract A Rare Cause of Diabetes: COVID-19 Introduction: Environmental factors like viruses have been described in the mechanism of pancreatic beta cell destruction. Various viruses can trigger autoimmunity in individuals genetically predisposed to diabetes. Severe acute respiratory syndrome coronavirus 2 (SARS CoV-2) has been shown to bind to angiotensin-converting enzyme 2 (ACE 2) which is expressed throughout the body, including pancreatic cells, leading to direct injury of the endocrine pancreas. We report a case of coronavirus infection disease 2019 (COVID-19) causing a new onset of diab

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Vinciguerra, Mattia, Silvia Romiti, Khalil Fattouch, Antonio De Bellis, and Ernesto Greco. "Atherosclerosis as Pathogenetic Substrate for Sars-Cov2 Cytokine Storm." Journal of Clinical Medicine 9, no. 7 (2020): 2095. http://dx.doi.org/10.3390/jcm9072095.

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The severe acute respiratory syndrome coronavirus 2 (Sars-CoV-2) outbreak is a public health emergency affecting different regions around the world. The lungs are often damaged due to the presence of Sars-CoV-2 binding receptor ACE2 on epithelial alveolar cells. Severity of infection varies from complete absence of symptomatology to more aggressive symptoms, characterized by sudden acute respiratory distress syndrome (ARDS), multiorgan failure, and sepsis, requiring treatment in intensive care unit (ICU). It is not still clear why the immune system is not able to efficiently suppress viral rep

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Kusuma, Victor Perdana, and Deasy Ardiany. "The Pathophysiology and Outcomes of Diabetic Patients with Coronavirus Disease 2019 (COVID-19)." Biomolecular and Health Science Journal 4, no. 2 (2021): 124. http://dx.doi.org/10.20473/bhsj.v4i2.30030.

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Introduction: Coronavirus Disease 2019 (COVID-19) is a respiratory tract infection caused by the SARS-CoV-2 virus, which was announced a pandemic by the World Health Organization (WHO) on March 11, 2020. On March 2, 2020, two confirmed cases of COVID-19 were initially reported in Indonesia. COVID-19 has been reported in 96.2 million people around the world. COVID-19 has already stolen the lives of almost 2 million individuals. Diabetes mellitus patients face an additional challenge with this disease (DM). Several studies have found a link between diabetes mellitus and COVID-19, as well as a ba

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Sardu, Celestino, Raffaele Marfella, Francesco Prattichizzo, Rosalba La Grotta, Giuseppe Paolisso, and Antonio Ceriello. "Effect of Hyperglycemia on COVID-19 Outcomes: Vaccination Efficacy, Disease Severity, and Molecular Mechanisms." Journal of Clinical Medicine 11, no. 6 (2022): 1564. http://dx.doi.org/10.3390/jcm11061564.

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Background/Aims: The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a positive-stranded single-stranded RNA virus, a member of the subgenus Sarbecovirus (beta-CoV lineage B) and responsible for the coronavirus disease 2019 (COVID-19). COVID-19 encompasses a large range of disease severity, from mild symptoms to severe forms with Intensive Care Unit admission and eventually death. The severe forms of COVID-19 are usually observed in high-risk patients, such as those with type two diabetes mellitus. Here, we review the available evidence linking acute and chronic hyperglycemia t

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Khaydarova, F. A., N. U. Alimova, A. V. Alieva, A. S. Sadykova, and M. D. Aripova. "Impact of COVID-19 infection on the development of type 1 diabetes mellitus in children and adolescents." Diabetes mellitus 25, no. 1 (2022): 21–26. http://dx.doi.org/10.14341/dm12785.

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Children did not escape the COVID-19 pandemic. Although, in general, the course of viral infection in children is mild, the question of the long-term effects of COVID-19 on a child and adolescent, in particular, on pancreatic beta cells, remains unclear.Аim: Тo study the characteristics of children with diabetes mellitus identified after COVID-19 infection.Materials and methods: This article presents the preliminary results obtained from children and adolescents hospitalized at the RSSPMCE clinic with the newly diagnosed diabetes mellitus after COVID infection, as well as a systematic review o

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McCord, Joe M., Brooks M. Hybertson, Adela Cota-Gomez, Kara P. Geraci, and Bifeng Gao. "Nrf2 Activator PB125® as a Potential Therapeutic Agent against COVID-19." Antioxidants 9, no. 6 (2020): 518. http://dx.doi.org/10.3390/antiox9060518.

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Nrf2 is a transcription factor that regulates cellular redox balance and the expression of a wide array of genes involved in immunity and inflammation, including antiviral actions. Nrf2 activity declines with age, making the elderly more susceptible to oxidative stress-mediated diseases, which include type 2 diabetes, chronic inflammation, and viral infections. Published evidence suggests that Nrf2 activity may regulate important mechanisms affecting viral susceptibility and replication. We examined gene expression levels by GeneChip microarray and by RNA-seq assays. We found that the potent N

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JC, Ekezie, and Haddad D. "New Onset Type-1 Diabetes Mellitus in a Toddler with Sars-Cov-2 Infection Presenting In Diabetic Ketoacidosis: A Case Report." New Medical Innovations and Research 2, no. 5 (2021): 01–03. http://dx.doi.org/10.31579/2767-7370/022.

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Introduction: Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), though mostly sparing the lungs in children, has been found to affect other organs including the endocrine pancreas. Type 1 diabetes mellitus (T1DM) may occur through direct negative effects of the virus on beta-cell function leading to diminished insulin production. Diabetic ketoacidosis (DKA) is a known and life-threatening complication of T1DM. Case presentation: This is a case of a 3-year-old previously healthy male who presented with 4 days history of fever, with malaise and hyperpnea for one day. Review of system

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Evi Awwaliyah, Hotimah, and Michio Shimabukuro. "Clinical characteristics and mortality associated with COVID-19 in islamic hospital of Jemursari, Surabaya, Indonesia: A hospital-based retrospective cohort study." Bali Medical Journal 11, no. 3 (2022): 1202–6. http://dx.doi.org/10.15562/bmj.v11i3.3541.

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Introduction: According to WHO, the COVID-19 epidemic a public health emergency came to international attention in March 2020, and the pandemic quickly spread around the world. This disease is caused by the novel coronavirus SARS-CoV-2, which may enter human target cells via angiotensin-converting enzyme 2 (ACE2). This study aimed to assess the risk factors associated with poor prognosis among COVID-19 patients in the Islamic Hospital of Jemursari, Surabaya, Indonesia. Methods: This study was a retrospective cohort study that used patients hospitalized with COVID-19 at the Islamic Hospital of

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Babenko, A. Yu, and M. Yu Laevskaya. "Diabetes mellitus and COVID-19. How are they connected? Current strategy of fight." "Arterial’naya Gipertenziya" ("Arterial Hypertension") 26, no. 3 (2020): 304–11. http://dx.doi.org/10.18705/1607-419x-2020-26-3-304-311.

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Infectious epidemic of COVID-19 caused by the new coronavirus is characterized by severe course in patients with diabetes mellitus, which presents another noninfectious pandemic accelerating for last decades. Today, according to the International Diabetic Federation data, there are 463 million patients with diabetes mellitus in the world. The burden of the COVID-19 epidemic is largely explained by a frequent combination of these two pathologies. From the previous flu epidemics and already available data of the current epidemic, diabetes mellitus and obesity are considered to be the predictors

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Kara, Mehmet, and Ilhami Celik. "Effect of clinical progress in antihypertensive medications among COVID-19 patients." Medical Science and Discovery 9, no. 10 (2022): 593–98. http://dx.doi.org/10.36472/msd.v9i10.829.

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Objective: Many chronic diseases, such as hypertension, diabetes, and coronary heart disease, paving the way for the disease to progress unfavorably in Covid-19. Angiotensin-converting enzyme inhibitors (ACEIs) and angiotensin-2 receptor blockers (ARBs) can upregulate ACE2 receptors (which SARS-COV-2 uses to enter the host cell) or protect against infection by limiting the effects of Angiotensin 2. This study aimed to reveal the impact of antihypertensive drugs on the hospital staying, and mortality in Covid-19 patients followed in the hospital. Methods and Results: One hundred patients were r

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Martinez, Alfonso Hoyos, Kelly Anne Hicks, Tracy Patel Moorjani, Jennifer Bell, and Yuezhen Lin. "A Case of Autoantibody Negative Pediatric Diabetes Mellitus With Marked Insulin Resistance Concomitant With COVID-19: A Novel Form of Disease?" Journal of the Endocrine Society 5, Supplement_1 (2021): A690—A691. http://dx.doi.org/10.1210/jendso/bvab048.1406.

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Abstract Background: SARS-CoV-2 uses the angiotensin-converting enzyme 2 (ACE2) receptor to enter human cells. This receptor is avidly expressed in the pancreatic islets, suggesting the virus may target β-cell function. 17% of adults with COVID-19 have evidence of pancreatic injury. There is a direct relation between insulin resistance and COVID-19 severity and mortality with patients with higher insulin resistance presenting with higher inflammatory response. Fulminant type 1 diabetes (fT1D) has abrupt onset of symptoms with insulinopenia without evidence of autoimmunity, usually preceded by

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Alqaisi, Sura, and Ali Rahman. "ODP649 COVID-19 Induced Diabetic Ketoacidosis Which Unmasked Latent Autoimmune Diabetes In Adult." Journal of the Endocrine Society 6, Supplement_1 (2022): A346—A347. http://dx.doi.org/10.1210/jendso/bvac150.721.

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Abstract Latent autoimmune diabetes in adults (LADA)" is a kind"of autoimmune diabetes" that progresses slowly. Numerous studies have termed LADA type 1.5 diabetes because it is often associated with antibodies against insulin-producing pancreatic cells. In contrast to type 1 diabetes, patients with LADA may go six months without insulin following diagnosis. Individuals with LADA are often well beyond the age of 30. Because they develop symptoms later than patients with type 1 diabetes and because their pancreases initially generate some insulin, persons with LADA are sometimes misdiagnosed as

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Wasilewska, Inga, Jakub Rezmer, and Łukasz Świątek. "Diabetes mellitus and COVID-19: current state of knowledge." Journal of Education, Health and Sport 12, no. 8 (2022): 1170–75. http://dx.doi.org/10.12775/jehs.2022.12.08.101.

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Introduction: Diabetes mellitus is a chronic metabolic disease characterized by disturbances in carbohydrate metabolism. Long-term high blood glucose levels can lead to a variety of serious complications and organ failure. Diabetic patients are particularly at risk of the severe course of COVID-19. Objective: The study aimed to investigate the relationship between diabetes and COVID-19. The literature available in the PubMed database was used to carry out a systematic review of the literature. The meta-analyzes from 2021 and 2022 were used for the analysis, with the inclusion of the phrases "D

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Yatskov, I. A., V. A. Beloglazov, and E. I. Ryapova. "Lipopolysaccharide and ARDS caused by new coronavirus infection: hypotheses and facts." Medical Immunology (Russia) 24, no. 1 (2022): 7–18. http://dx.doi.org/10.15789/1563-0625-laa-2229.

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This review presents data from the literature that provide insight into the role of the lipopolysaccharide (LPS) of the Gram-negative bacteria in pathogenesis of acute respiratory distress syndrome (ARDS) caused by the novel SARS-CoV-2 coronavirus infection. ARDS is a syndrome of severe respiratory failure, an acutely occurring diffuse inflammatory lesion of lung tissue that develops as a nonspecific reaction to various direct (aspiration, inhalation of toxic gases), and systemic (sepsis, polytrauma) damaging factors and leading to development of acute respiratory failure (ARF), due to impaire

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Schiaffini, Riccardo, Andrea Campana, Annalisa Deodati, Emanuela Peschiaroli, Maria Francesca Lanzillotta, and Alessandra Fierabracci. "SARS-CoV-2 infection as possible downstream disease precipitator in autoantibody-positive insulin-dependent diabetes mellitus: a case report." Italian Journal of Pediatrics 48, no. 1 (2022). http://dx.doi.org/10.1186/s13052-022-01226-5.

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Abstract Background SARS-CoV-2 causes lesions, in addition to lung, in endocrine organs such as the pancreas through ACE2 receptor. Recently the relationship between SARS-CoV-2 exposition and the incidence or evolution of clinical autoimmune diabetes has attracted the attention of diabetologists. Case presentation We report the analysis of the clinical history of a child diagnosed for insulin-dependent diabetes mellitus (Type 1 diabetes) at the time a paucisymptomatic COVID-19 infection occurred, followed by well-controlled metabolic status. As opposite to previous findings SARS-CoV2 did not c

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Akkuş, Gamze. "Newly-onset Autoimmune Diabetes Mellitus triggered by Covid 19 infection: A case based review." Endocrine, Metabolic & Immune Disorders - Drug Targets 22 (October 4, 2022). http://dx.doi.org/10.2174/2666145415666221004111511.

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Abstract: The devastating global pandemic Coronavirus disease 2019 (Covid 19) which was isolated in China in January 2020 is responsible for outbreak of pneumonia and other multisystemic complications. The clinical picture of the infection has an extreme variability: it goes from asymptomatic patients or mild forms with fever, cough, fatigue and loss of smell and taste, to severe cases ending up in the intensive care unit (ICU). This is due to possible cytokine storm that may lead to multi organ failure, septic shock, or thrombosis. Severe Acute Respiratory Syndrome coronavirus 2 (SARS-CoV -2)

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Steenblock, Charlotte, Stefanie Richter, Ilona Berger, et al. "Viral infiltration of pancreatic islets in patients with COVID-19." Nature Communications 12, no. 1 (2021). http://dx.doi.org/10.1038/s41467-021-23886-3.

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AbstractMetabolic diseases are associated with an increased risk of severe COVID-19 and conversely, new-onset hyperglycemia and complications of preexisting diabetes have been observed in COVID-19 patients. Here, we performed a comprehensive analysis of pancreatic autopsy tissue from COVID-19 patients using immunofluorescence, immunohistochemistry, RNA scope and electron microscopy and detected SARS-CoV-2 viral infiltration of beta-cells in all patients. Using SARS-CoV-2 pseudoviruses, we confirmed that isolated human islet cells are permissive to infection. In eleven COVID-19 patients, we exa

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Lazartigues, Eric, Mirza Muhammad Fahd Qadir, and Franck Mauvais-Jarvis. "Endocrine Significance of SARS-CoV-2’s Reliance on ACE2." Endocrinology 161, no. 9 (2020). http://dx.doi.org/10.1210/endocr/bqaa108.

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Abstract The current COVID-19 pandemic is the most disruptive event in the past 50 years, with a global impact on health care and world economies. It is caused by severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2), a coronavirus that uses angiotensin-converting enzyme 2 (ACE2) as an entry point to the cells. ACE2 is a transmembrane carboxypeptidase and member of the renin-angiotensin system. This mini-review summarizes the main findings regarding ACE2 expression and function in endocrine tissues. We discuss rapidly evolving knowledge on the potential role of ACE2 and SARS coronavirus

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Shao, Shiying, Qin Yang, Ruping Pan, Xuefeng Yu, and Yong Chen. "Interaction of Severe Acute Respiratory Syndrome Coronavirus 2 and Diabetes." Frontiers in Endocrinology 12 (October 6, 2021). http://dx.doi.org/10.3389/fendo.2021.731974.

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Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is causing a worldwide epidemic. It spreads very fast and hits people of all ages, especially patients with underlying diseases such as diabetes. In this review, we focus on the influences of diabetes on the outcome of SARS-CoV-2 infection and the involved mechanisms including lung dysfunction, immune disorder, abnormal expression of angiotensin-converting enzyme 2 (ACE2), overactivation of mechanistic target of rapamycin (mTOR) signaling pathway, and increased furin level. On the other hand, SARS-CoV-2 may trigger the development of

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Al-kuraishy, Hayder M., Ali I. Al-Gareeb, M. Alblihed, Susana G. Guerreiro, Natália Cruz-Martins, and Gaber El-Saber Batiha. "COVID-19 in Relation to Hyperglycemia and Diabetes Mellitus." Frontiers in Cardiovascular Medicine 8 (May 20, 2021). http://dx.doi.org/10.3389/fcvm.2021.644095.

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Coronavirus disease 2019 (COVID-19), triggered by the severe acute respiratory syndrome-coronavirus 2 (SARS-CoV-2), may lead to extrapulmonary manifestations like diabetes mellitus (DM) and hyperglycemia, both predicting a poor prognosis and an increased risk of death. SARS-CoV-2 infects the pancreas through angiotensin-converting enzyme 2 (ACE2), where it is highly expressed compared to other organs, leading to pancreatic damage with subsequent impairment of insulin secretion and development of hyperglycemia even in non-DM patients. Thus, this review aims to provide an overview of the potenti

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"COVID-19 y su asociación con los inhibidores de la enzima convertidora de angiotensina y los antagonistas de los receptores para angiotensina II." Revista de la Facultad de Medicina 63, no. 4 (2020): 30–34. http://dx.doi.org/10.22201/fm.24484865e.2020.63.4.05.

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Worldwide, over 7 million people have been infected due to the pandemic of COVID-19. The comorbidities associated to this disease are: hypertension, diabetes mellitus, obesity, obstructive pulmonary disease (COPD), cardiovascular disease, chronic renal failure, smoking, immunosuppression, and hypertension. Angiotensin-converting enzyme 2 (ACE2) is the functional receptor for SARS-CoV-2. This virus has an S protein that recognizes ACE2 as its receptor to enter the cell. ACE2 is a plasmatic protein expressed in alveolar cells type I, II, fibroblasts, endothelial cells and macrophages. Treatment

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Nouri-Keshtkar, Marjan, Sara Taghizadeh, Aisan Farhadi, et al. "Potential Impact of Diabetes and Obesity on Alveolar Type 2 (AT2)-Lipofibroblast (LIF) Interactions After COVID-19 Infection." Frontiers in Cell and Developmental Biology 9 (July 8, 2021). http://dx.doi.org/10.3389/fcell.2021.676150.

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Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), a new emerging respiratory virus, caused evolving pneumonia outbreak around the world. In SARS-Cov-2 infected patients, diabetes mellitus (DM) and obesity are two metabolic diseases associated with higher severity of SARS-CoV-2 related complications, characterized by acute lung injury requiring assisted ventilation as well as fibrosis development in surviving patients. Different factors are potentially responsible for this exacerbated response to SARS-CoV-2 infection. In patients with DM, base-line increase in inflammation and oxida

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Okoduwa, Stanley I. R., Daniel H. Mhya, Idongesit A. Enang, and Akinbobola O. Salawu. "Diabetes Mellitus and COVID-19: Possible Interactions and Mechanisms in Comorbid Patients." Journal of Health Science and Medical Research, October 27, 2022. http://dx.doi.org/10.31584/jhsmr.2022904.

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Beginning in December 2019 and still ongoing, coronavirus disease 2019 (COVID-19) infections have posed a public health challenge worldwide. There have been reports of diabetes mellitus (DM) as one of the most prevalent comorbidities in patients with COVID-19. Although the interactions and possible mechanisms of this association have not been fully established, the existence of DM is believed to aggravate the adverse effects of COVID-19 infection. Hence, the need for this paper. Findings from other studies have shown different possible mechanisms of how COVID-19 and DM aggravate the severity o

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Najafi, Reza, Nahid Mamizadeh, Seyed Hossein Hosseini, Sima Roushenas, and Leila Bazhdan. "A challenging case of COVID-19: a COVID-19 positive adolescent presented with severe diabetic ketoacidosis, resistant hypertension." BMC Endocrine Disorders 22, no. 1 (2022). http://dx.doi.org/10.1186/s12902-022-00979-8.

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Abstract Background Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a virus which causes COVID-19. It binds to the angiotensin-converting enzyme 2 (ACE2) receptors, expressed in key metabolic organs and tissues, including pancreatic beta cells, adipose tissue, the small intestine, and kidneys. This condition has been linked to a variety of additional symptoms, including acute encephalopathy, changes in consciousness, and even gastrointestinal bleeding. Case presentation In this study, we have reported a 13-year-old boy, 69 kg, with SARS-COV-2 infection. In this case, multiple s

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Alizadeh, Shaban, Reza Afrisham, Yasaman Jadidi, et al. "Gastrointestinal, liver, pancreas, oral and psychological long-term symptoms of COVID-19 after recovery; A review." Mini-Reviews in Medicinal Chemistry 23 (November 16, 2022). http://dx.doi.org/10.2174/1389557523666221116154907.

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Abstracts: Due to the importance of control and prevention of COVID-19-correlated long-term symptoms, the present review article has summarized what has been currently known regarding the molecular and cellular mechanisms linking COVID-19 to important long-term complications including psychological complications, liver and gastrointestinal manifestations, oral signs as well as even diabetes. COVID-19 can directly affect the body cells through their Angiotensin-converting enzyme 2 [ACE-2] to induce inflammatory responses and cytokine storm. The cytokines cause the release of reactive oxygen spe

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Schipani, Elena, Andrea Cozzi, Giuseppe Salvatore Murgida, et al. "New-onset type 1 diabetes mellitus triggered by SARS-CoV-2 infection in a patient with Hashimoto thyroiditis: a case report." Italian Journal of Medicine, January 25, 2021. http://dx.doi.org/10.4081/itjm.2021.1387.

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New onset type 1 diabetes mellitus is an uncommon but possible complication triggered by SARSCoV- 2 infection. Metabolic inflammation supported by cytokine storm leading to pancreatic beta cells destruction is the most probable link between COVID-19 and diabetes. Here, we describe the case of a 51-year-old female suffering from Hashimoto thyroiditis, who came to our attenction for new onset polyuria-polydipsia syndrome associated to hyperglycemia after a mild form of COVID- 19 recognized two months before and already recovered. Type 1 diabetes was diagnosed.

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Roeroe, Pomantow A. L., Bisuk P. Sedli, and Octavianus Umboh. "Faktor Risiko Terjadinya Coronavirus Disease 2019 (Covid-19) pada Penyandang Diabetes Melitus Tipe 2." e-CliniC 9, no. 1 (2021). http://dx.doi.org/10.35790/ecl.v9i1.32301.

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Abstract: Coronavirus Disease 2019 (COVID-19) is an infectious disease caused by SARS-CoV-2 and has been declared as a pandemic by WHO in March 11, 2020. This disease is an additional problem in people with type 2 diabetes mellitus (T2DM). Several studies have shown that diabetes is a risk factor for COVID-19. This study was aimed to determine the risk factors for the occurrence of Coronavirus Disease 2019 (COVID-19) in T2DM patients. This was a literature review study using several journal databases, namely Google Scholar, PubMed, and Science Direct. Literature searching was performed by usin

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Patel, Sanyamee, Prashilkumar Patel, Ayushi Mehta, Ishan Patel, Shubham Thakor, and Om Lumbhani. "incidence of myocarditis and pericarditis in post COVID-19 unvaccinated patients." International journal of health sciences, July 31, 2022, 11743–51. http://dx.doi.org/10.53730/ijhs.v6ns5.11049.

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Background and Aim: Viral infections have also been associated with the presence of autoimmune diseases such as systemic lupus disease, rheumatoid arthritis, and diabetes mellitus. SARS-CoV-2 gains entry into human cells by binding its spike protein to the membrane protein angiotensinconverting enzyme 2 (ACE2). It has recently been reported that the incidence of myocarditis and pericarditis is increased in COVID-19 patients during the acute illness. However; whether or not myocarditis and pericarditis after the recovery period are a part of the long COVID-19 syndrome is yet unknown. Hence, we

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Saheb Sharif-Askari, Fatemeh, Narjes Saheb Sharif-Askari, Shirin Hafezi, et al. "Upregulation of interleukin-19 in saliva of patients with COVID-19." Scientific Reports 12, no. 1 (2022). http://dx.doi.org/10.1038/s41598-022-20087-w.

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AbstractCytokines are major players in orchestrating inflammation, disease pathogenesis and severity during COVID-19 disease. However, the role of IL-19 in COVID-19 pathogenesis remains elusive. Herein, through the analysis of transcriptomic datasets of SARS-CoV-2 infected lung cells, nasopharyngeal swabs, and lung autopsies of COVID-19 patients, we report that expression levels of IL-19 and its receptor, IL-20R2, were upregulated following SARS-CoV-2 infection. Of 202 adult COVID-19 patients, IL-19 protein level was significantly higher in blood and saliva of asymptomatic patients compared to

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