Relevant bibliographies by topics / Diffuse glioma

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Diffuse glioma'

Author: Grafiati
Published: 25 January 2025
Last updated: 31 July 2025

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Journal articles on the topic "Diffuse glioma"

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Wong, Shu Chyi, Muhamad Noor Alfarizal Kamarudin, and Rakesh Naidu. "Anticancer Mechanism of Flavonoids on High-Grade Adult-Type Diffuse Gliomas." Nutrients 15, no. 4 (2023): 797. http://dx.doi.org/10.3390/nu15040797.

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High-grade adult-type diffuse gliomas are the most common and deadliest malignant adult tumors of the central nervous system. Despite the advancements in the multimodality treatment of high-grade adult-type diffuse gliomas, the five-year survival rates still remain poor. The biggest challenge in treating high-grade adult-type diffuse gliomas is the intra-tumor heterogeneity feature of the glioma tumors. Introducing dietary flavonoids to the current high-grade adult-type diffuse glioma treatment strategies is crucial to overcome this challenge, as flavonoids can target several molecular targets
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Rao, Aparna, Xiaoran Zhang, Christopher Deibert, Paola Sette, Paola Grandi, and Nduka Amankulor. "IDH Mutant Gliomas Escape Natural Killer Cell Immune Surveillance by Downregulation of NKG2D Ligand Expression." Journal of Immunology 196, no. 1_Supplement (2016): 142.11. http://dx.doi.org/10.4049/jimmunol.196.supp.142.11.

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Abstract Background Diffuse gliomas are fatal primary brain tumors that are poorly immunogenic. The basis for insufficient anti-tumor immunity in diffuse gliomas is not understood. Mutations in isocitrate dehydrogenases (IDH1 and IDH2) promote diffuse glioma formation through epigenetic reprogramming of a number of genes, including immune-related genes. Here, we identify epigenetic dysregulation of natural killer (NK) cell ligand genes as significant contributors to immune escape in glioma. Methods We analyzed the TCGA database for immune gene expression patterns in IDH mutant or wild-type gli
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Barron, Tara, Belgin Yalçın, Aaron Mochizuki, et al. "CNSC-01. GABAERGIC NEURON-TO-GLIOMA SYNAPSES IN DIFFUSE MIDLINE GLIOMAS." Neuro-Oncology 25, Supplement_1 (2023): i11. http://dx.doi.org/10.1093/neuonc/noad073.044.

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Abstract High-grade gliomas include clinically and molecularly distinct subtypes that stratify by anatomical location into diffuse midline gliomas (DMG) such as diffuse intrinsic pontine glioma (DIPG) and hemispheric high-grade gliomas. Neuronal activity drives high-grade glioma progression both through paracrine signaling and direct neuron-to-glioma synapses. Glutamatergic, AMPA receptor-dependent synapses between neurons and malignant glioma cells have been demonstrated in both pediatric and adult high-grade gliomas, but neuron-to-glioma synapses mediated by other neurotransmitters remain la
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Nguyen, Anthony V., Jose M. Soto, Sarah-Marie Gonzalez, et al. "H3G34-Mutant Gliomas—A Review of Molecular Pathogenesis and Therapeutic Options." Biomedicines 11, no. 7 (2023): 2002. http://dx.doi.org/10.3390/biomedicines11072002.

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The 2021 World Health Organization Classification of Tumors of the Central Nervous System reflected advances in understanding of the roles of oncohistones in gliomagenesis with the introduction of the H3.3-G34R/V mutant glioma to the already recognized H3-K27M altered glioma, which represent the diagnoses of pediatric-type diffuse hemispheric glioma and diffuse midline glioma, respectively. Despite advances in research regarding these disease entities, the prognosis remains poor. While many studies and clinical trials focus on H3-K27M-altered-glioma patients, those with H3.3-G34R/V mutant glio
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Nuechterlein, Nicholas, and Patrick Cimino. "PATH-24. COPY NUMBER ALTERATIONS IN NOTCH PATHWAY GENES ARE PROGNOSTIC IN A SUBSET OF DIFFUSE ASTROCYTIC GLIOMAS." Neuro-Oncology 23, Supplement_6 (2021): vi120. http://dx.doi.org/10.1093/neuonc/noab196.476.

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Abstract Inactivating mutations in NOTCH1 occur in many cancer types and are frequently observed in IDH-mutant, 1p/19q-codeleted oligodendroglioma. Although the role of NOTCH1 as a tumor suppressor in diffuse glioma has become appreciated in human tissue and small animal models, the spectrum of inactivating mutations in Notch pathway genes in diffuse astrocytic gliomas has not been well described. To address this, we queried the TCGA lower-grade glioma and glioblastoma datasets to establish the extent of inactivation of Notch pathway genes, specifically by cataloging single nucleotide variants
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Campos Paiva, Aline, João Luiz Vitorino Araujo, Guilherme Brasileiro de Aguiar, Gabriel Rezende Batistella, Marcos Maldaum, and José Esteves veiga. "SURG-11. NEOADJUVANT CHEMOTHERAPY FOR DIFFUSE GLIOMAS: A FEASIBLE OPTION?" Neuro-Oncology 22, Supplement_2 (2020): ii205. http://dx.doi.org/10.1093/neuonc/noaa215.858.

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Abstract BACKGROUND Diffuse gliomas are slow-growing tumors with predilection for deep and eloquent structures such as supplementary motor area and insula. They can reach huge dimensions which is a surgical challenging. OBJECTIVE Describe indications and limitations of neoadjuvant chemotherapy for diffuse gliomas. METHODS Systematic review was performed in November/2019 using Pubmed, Bireme and Cochrane databases. The following combinations were used: “Chemotherapy” AND “neoadjuvant” AND “diffuse glioma”, “Chemotherapy” AND “neoadjuvant” AND “low grade glioma”, “Chemotherapy” AND “preoperative
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Soni, Vaishali Walke, Deepti Joshi, Tanya Sharma, Adesh Shrivastava, and Amit Agrawal. "The spectrum of microvascular patterns in adult diffuse glioma and their correlation with tumor grade." Journal of Pathology and Translational Medicine 58, no. 3 (2024): 127–33. http://dx.doi.org/10.4132/jptm.2024.03.11.

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Background: Primary brain tumors constitute the leading cause of cancer-related mortality. Among them, adult diffuse gliomas are the most common type, affecting the cerebral hemispheres and displaying a diffuse infiltrative pattern of growth in the surrounding neuropil that accounts for about 80% of all primary intracranial tumors. The hallmark feature of gliomas is blood vessel proliferation, which plays an important role in tumor growth, tumor biological behavior, and disease outcome. High-grade gliomas exhibit increased vascularity, the worst prognosis, and lower survival rates. Several ang
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Sarangi, Sasmit, and Eric Wong. "HOUT-05. COMPARING OUTCOMES OF ADULT DIFFUSE MIDLINE GLIOMAS TO GLIOBLASTOMA (GBM) IN YOUNG PATIENTS." Neuro-Oncology 21, Supplement_6 (2019): vi112—vi113. http://dx.doi.org/10.1093/neuonc/noz175.470.

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Abstract Diffuse midline gliomas are a diagnostic entity with astrocytic tumors in a midline location and in the pediatric age group they have been associated with poor outcomes. There is ongoing debate if adults with diffuse midline gliomas also have a poor prognosis particularly as complete surgical resection is often not achievable in these patients. One of the limitations of looking at case-series based outcomes with these patients is that the incidence of these tumors has a skew towards younger patients and recent case-series with observed better than expected outcomes, might be subject t
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Paracha, Awais, Mohamed S. Abdelbaki, and Pournima Navalkele. "DIPG-63. Therapies for diffuse gliomas in pediatric patients: a systematic review and meta-analysis." Neuro-Oncology 24, Supplement_1 (2022): i33. http://dx.doi.org/10.1093/neuonc/noac079.120.

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Abstract Diffuse gliomas are one of the most challenging pediatric brain tumors of the current era. The new WHO classification has brought in a paradigm shift in the diagnosis and management of diffuse gliomas. Although these tumors are not surgically resectable, an integrated molecular analysis could make them more amenable to targeted agents. We conducted a thorough systematic review and meta-analysis of therapies for diffuse gliomas in pediatric patients. Methods: Using PRISMA guidelines, PubMed, (Medline), Cochrane, and Google Scholar database searches are being conducted using search term
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Dellaretti, Marcos, Nicolas Reyns, Gustavo Touzet, et al. "Diffuse brainstem glioma: prognostic factors." Journal of Neurosurgery 117, no. 5 (2012): 810–14. http://dx.doi.org/10.3171/2012.7.jns111992.

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Object Brainstem gliomas were regarded as a single entity prior to the advent of MRI; however, several studies investigating MRI have recognized that these lesions are a heterogeneous group, and certain subgroups have a better prognosis for long-term survival. The aim of this study was to conduct a retrospective analysis of prognostic factors of patients with brainstem gliomas confirmed by histopathological diagnosis, particularly regarding assessment of whether histological grade, age, and MRI findings are prognostic factors for patient survival. Methods The study evaluated 100 patients diagn
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Dissertations / Theses on the topic "Diffuse glioma"

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Konnully, Augustus Meera Bessy. "Characterization of cellular heterogeneity in Diffuse Low Grade Glioma." Thesis, Montpellier, 2020. http://www.theses.fr/2020MONTT038.

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Les gliomes diffus de bas grade (DLGG) sont des tumeurs gliales de grade II qui affectent principalement les jeunes adultes. Elles sont caractérisées par une croissance lente et une activité mitotique réduite. Cependant, ces tumeurs diffusent et envahissent le cerveau sain via les vaisseaux sanguins et les fibres nerveuses. Après plusieurs années de croissance lente, ces tumeurs peuvent évoluer vers des glioblastomes, des tumeurs cérébrales très agressives dont la survie médiane moyenne est alors de 12 à 15 mois après le diagnostic. La caractérisation cellulaire des DLGG est encore limitée ce
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Furnish, Robin. "Evaluating Immune Modulatory Therapeutic Strategies for Diffuse Intrinsic Pontine Glioma." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1595849080346532.

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Trisolini, Elena. "Targeted molecular characterization of adult midline and circumscribed gliomas for the identification of new potential targets for personalized therapy." Doctoral thesis, Università del Piemonte Orientale, 2020. http://hdl.handle.net/11579/114872.

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Diffuse midline gliomas (MLG) are primary brain tumours arising from thalamus, hypothalamus, brainstem, cerebellum or spinal cord, mainly occurring in children. In adults, less than 10% of diffuse gliomas arises in midline structures and recent works suggested that this subset of tumours may present with phenotypic and molecular characteristics differing from both pediatric MLG and adult supratentorial gliomas. Circumscribed gliomas (CG) are low-grade tumours but may progress to anaplasia. They have lower genetic complexity than diffuse gliomas and could be better candidate for targeted ther
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Ben, Abdallah Mériem. "Un modèle de l'évolution des gliomes diffus de bas grade sous chimiothérapie." Thesis, Université de Lorraine, 2016. http://www.theses.fr/2016LORR0215/document.

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Les gliomes diffus de bas grade sont des tumeurs cérébrales des jeunes adultes. Dans cette thèse, nous nous intéressons à la segmentation et à la modélisation de ces tumeurs. Dans la première partie du manuscrit, nous étudions la segmentation des gliomes diffus de bas grade à base de différentes méthodes manuelles et semi-automatiques. La délimitation de ces tumeurs peut être problématique en raison de leur caractère très infiltrant et inhomogène. En pratique clinique, le suivi des gliomes diffus de bas grade repose sur l'estimation du volume tumoral, soit par une segmentation suivie d'une rec
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Fromm, Jan. "Investigating the expression and role of chloride ion channels in diffuse intrinsic pontine glioma." Thesis, Fromm, Jan (2021) Investigating the expression and role of chloride ion channels in diffuse intrinsic pontine glioma. Honours thesis, Murdoch University, 2021. https://researchrepository.murdoch.edu.au/id/eprint/63626/.

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Diffuse intrinsic pontine glioma (DIPG) is a highly aggressive type of glial brain tumour found in the pons region of the brainstem. DIPG accounts for about 10% of childhood central nervous system tumours and the prognosis for these children is poor. Resistance to radiation, the only current available therapy for DIPG, is one of the biggest challenges. This resistance could be due to the plasticity of DIPG cells, allowing them to rapidly adapt in response to different conditions. Preliminary RNA sequencing analyses of patient tumours identified the expression of ion channel genes including the
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Andreiuolo, Felipe. "Target in context : molecular pathology of pediatric ependymoma and high grade glioma." Phd thesis, Université Paris Sud - Paris XI, 2012. http://tel.archives-ouvertes.fr/tel-00913042.

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Biomarkers for the classification, clinical management and prognosis of pediatric brain tumors (ependymoma and high grade glioma, (HGG)) are lacking. To address this, biomarkers were developed and explored in view of classification, prognostication, target identification and prediction of the efficacy of treatment for patients with such tumors.We show that overexpression of neuronal markers distinguishes supratentorial from infratentorial ependymoma, and among the former higher immunoexpression of neurofilament 70 (NEFL) is correlated with better progression free survival (PFS). Tenascin-C (TN
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Ben, Abdallah Mériem. "Un modèle de l'évolution des gliomes diffus de bas grade sous chimiothérapie." Electronic Thesis or Diss., Université de Lorraine, 2016. http://www.theses.fr/2016LORR0215.

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Les gliomes diffus de bas grade sont des tumeurs cérébrales des jeunes adultes. Dans cette thèse, nous nous intéressons à la segmentation et à la modélisation de ces tumeurs. Dans la première partie du manuscrit, nous étudions la segmentation des gliomes diffus de bas grade à base de différentes méthodes manuelles et semi-automatiques. La délimitation de ces tumeurs peut être problématique en raison de leur caractère très infiltrant et inhomogène. En pratique clinique, le suivi des gliomes diffus de bas grade repose sur l'estimation du volume tumoral, soit par une segmentation suivie d'une rec
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Khalid, Fahad. "Magnetic Resonance Imaging and Genomic Mutation in Diffuse Intrinsic Pontine Glioma : Machine Learning Approaches for a Comprehensive Analysis." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPAST006.

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Le diagnostic du gliome infiltrant du tronc cérébral (GITC) chez les enfants est l'un des plus éprouvants en oncologie pédiatrique. Malgré de nombreux essais cliniques explorant divers traitements, le pronostic reste sombre, la plupart des patients succombant entre 9 et 11 mois après le diagnostic. Les mutations génétiques clé associées au GITC incluent H3K27M, ACVR1 et TP53. Chaque mutation a des caractéristiques distinctes, poussant les médecins à suggérer des thérapies personnalisées, soulignant l'importance d'une détection précise des mutations pour guider le traitement. Situées dans la ré
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Laurenge-Leprince, Alice. "Impact of D-2HG on the Tumor Microenvironment of IDH-mutated Gliomas." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASL096.

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Contexte : Les gliomes diffus sont les tumeurs primitives malignes du système nerveux central les plus fréquentes. Certains présentent une mutation du gène codant pour l'isocitrate déshydrogénase 1 ou 2 (IDH) qui leur confère la néo-activité de transformer l'alpha-cétoglutarate (α-KG) produit par l'enzyme non mutée en D-2hydroxyglutarate (D-2HG). L'accumulation de cet oncométabolite entraîne l'inhibition compétitive des dioxygénases dépendantes de l'α-KG, incluant la famille TET (Ten Eleven Translocase) des ADN hydroxylases, et les JmjC/KMDs histone déméthylases, qui conduit elle-même à l'hype
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Herbet, Guillaume. "Vers un modèle à double voie dynamique et hodotopique de l'organisation anatomo-fonctionnelle de la mentalisation : étude par cartographie cérébrale multimodale chez les patients porteurs d'un gliome diffus de bas-grade." Thesis, Montpellier 1, 2014. http://www.theses.fr/2014MON1T004/document.

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Comprendre comment le cerveau humain engendre les formes les plus élaborées de comportements est profondément lié à nos connaissances générales sur son organisation anatomique et fonctionnelle. Jusqu'à récemment encore, on pensait que les fonctions cognitives n'étaient rien d'autre que le sous-produit de l'activité neurale de régions corticales discrètes et hyper-fonctionnalisées. Les découvertes majeures obtenues ces dix dernières années dans le champ de la neuro-imagerie, et plus particulièrement de la connectomique, invitent cependant à complexifier nos représentations sur les liens qu'entr
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Books on the topic "Diffuse glioma"

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Duffau, Hugues, ed. Diffuse Low-Grade Gliomas in Adults. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-55466-2.

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Duffau, Hugues, ed. Diffuse Low-Grade Gliomas in Adults. Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-2213-5.

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Weller, Michael, Michael Brada, Tai-Tong Wong, and Michael A. Vogelbaum. Astrocytic tumours: diffuse astrocytoma, anaplastic astrocytoma, glioblastoma, and gliomatosis cerebri. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0003.

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Astrocytic gliomas are primary brain tumours thought to originate from neural stem or progenitor cells. They are assigned grades II, III, or IV by the World Health Organization according to degree of malignancy as defined by histology. The following molecular markers are increasingly used for diagnostic subclassification or clinical decision-making: 1p/19q co-deletion status, O6-methylguanine-DNA methyltransferase (MGMT) promoter methylation status, and isocitrate dehydrogenase 1 and 2 mutation status. Extent of resection is a favourable prognostic factor, but surgery is never curative. Radiot
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Kleihues, Paul, Elisabeth Rushing, and Hiroko Ohgaki. The 2016 revision of the WHO classification of tumours of the central nervous system. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0001.

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The revised fourth edition of the WHO classification of Tumours of the Central Nervous System, published in 2016, comprises several newly recognized tumour entities, and a significant restructuring of the classification, mainly based on genetic profiling. Glioblastomas are now classified into two major types. Isocitrate dehydrogenase (IDH)-wildtype glioblastoma (primary glioblastoma IDH-wildtype) develops rapidly de novo without a recognizable precursor lesion. IDH-mutant glioblastoma (secondary glioblastoma IDH-mutant) develops more slowly through malignant progression from diffuse or anaplas
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Duffau, Hugues. Diffuse Low-Grade Gliomas in Adults. Springer, 2018.

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Duffau, Hugues. Diffuse Low-Grade Gliomas in Adults. Springer, 2017.

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Schroeder, Kristin, and Oren Becher. Pontine Gliomas. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0138.

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Pontine gliomas-also known as diffuse intrinsic pontine gliomas (DIPG)-primarily occur in children and typically present subacutely with a combination of cranial nerve palsies associated with long track signs including hyper-reflexia in the legs, positive Babinski responses, and cerebellar signs. On imaging they typically appear as intrinsic mass lesions within the pons. Treatment with radiation can prolong the course of from months to years but the tumors are rarely curable. Chemotherapy combined with radiation therapy targeted at specific signaling pathways has shown only modest impact on su
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Diffuse Lowgrade Gliomas In Adults Natural History Interaction With The Brain And New Individualized Therapeutic Strategies. Springer London Ltd, 2013.

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A new diffusely infiltrating glioma mouse model reveals neuronal alterations in the brain tumor microenvironment. [publisher not identified], 2018.

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Duffau, Hugues. Diffuse Low-Grade Gliomas in Adults: Natural History, Interaction with the Brain, and New Individualized Therapeutic Strategies. Springer, 2013.

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Book chapters on the topic "Diffuse glioma"

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Noureldine, Mohammad Hassan A., Nir Shimony, and George I. Jallo. "Diffuse Midline Glioma – Diffuse Intrinsic Pontine Glioma." In Brainstem Tumors. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-38774-7_8.

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Liu, Dongyou. "Chordoid Glioma, Angiocentric Glioma, and Diffuse Midline Glioma." In Tumors and Cancers. CRC Press, 2017. http://dx.doi.org/10.1201/9781315120522-6.

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Vanan, Magimairajan Issai, Vivek Mehta, and David D. Eisenstat. "Diffuse Intrinsic Pontine Glioma." In Pediatric Neuro-oncology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1541-5_11.

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Vanan, Magimairajan Issai, Craig Erker, Vivek Mehta, Cynthia Hawkins, and David D. Eisenstat. "Diffuse Midline Glioma-Pons." In Pediatric Neuro-oncology. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-62017-1_11.

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Morrison, Melanie A., and Adam D. Waldman. "Imaging Markers of Lower-Grade Diffuse Glioma." In Glioma Imaging. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-27359-0_9.

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Karajannis, Matthias A., Matija Snuderl, Brian K. Yeh, et al. "High-Grade Glioma, Including Diffuse Intrinsic Pontine Glioma." In Brain Tumors in Children. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-43205-2_9.

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McNeill, Katharine, Kenneth Aldape, and Howard A. Fine. "Adult High-Grade (Diffuse) Glioma." In Molecular Pathology Library. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1830-0_6.

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De Witt Hamer, Philip C., Emmanuel Mandonnet, and Hugues Duffau. "Resection Probability Maps of Glioma." In Diffuse Low-Grade Gliomas in Adults. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-55466-2_32.

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Cesselli, Daniela, Antonio Paolo Beltrami, Anja Pucer, et al. "Human Low-Grade Glioma Cultures." In Diffuse Low-Grade Gliomas in Adults. Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-2213-5_10.

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Pope, Whitney B., and Kevin Spitler. "Molecular Imaging of Diffuse Low Grade Glioma." In Diffuse Low-Grade Gliomas in Adults. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-55466-2_10.

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Conference papers on the topic "Diffuse glioma"

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Noble Anbunesan, Silvia N., Alba Alfonso García, Mohamed A. Hassan, et al. "In vivo characterization of FLIm signatures on the diffuse glioma in the brain cortical surface (Conference Presentation)." In Advanced Biomedical and Clinical Diagnostic and Surgical Guidance Systems XXIII, edited by Caroline Boudoux and James W. Tunnell. SPIE, 2025. https://doi.org/10.1117/12.3043927.

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Giannoni, Luca, Camilla Bonaudo, Marta Marradi, Alessandro Della Puppa, and Francesco S. Pavone. "Optical characterisation and study of ex vivo glioma tissue for hyperspectral imaging during neurosurgery." In Diffuse Optical Spectroscopy and Imaging, edited by Davide Contini, Yoko Hoshi, and Thomas D. O'Sullivan. SPIE, 2023. http://dx.doi.org/10.1117/12.2670854.

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Ryall, Scott T., Robert Siddaway, Arun Ramani, Andrei Turinsky, Michael Brudno, and Cynthia Hawkins. "Abstract 1184: Clonal evolution of diffuse intrinsic pontine glioma." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-1184.

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Tolson, Hannah. "Abstract 466: Epigenetic drug profiling in diffuse intrinsic pontine glioma." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-466.

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Ben Abdallah, Meriem, Marie Blonski, Sophie Wantz-Mezieres, Yann Gaudeau, Luc Taillandier, and Jean-Marie Moureaux. "Predictive models for diffuse low-grade glioma patients under chemotherapy." In 2016 38th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2016. http://dx.doi.org/10.1109/embc.2016.7591692.

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Becher, Oren Josh. "Abstract IA22: Developing improved diffuse intrinsic pontine glioma mouse models." In Abstracts: AACR Special Conference: Advances in Brain Cancer Research; May 27-30, 2015; Washington, DC. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.brain15-ia22.

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Diaz, Alexander K., Gang Wu, Barbara S. Paugh, et al. "Abstract PR03: The genomic landscape of diffuse intrinsic pontine glioma and pediatric non-brainstem high-grade glioma." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-pr03.

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Ferris, Sarah F., Rachel K. Surowiec, Carlos Espinoza, and Stefanie Galban. "Abstract 6152: Characterizing cancer stem cells in diffuse intrinsic pontine glioma." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-6152.

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Yadavilli, Sridevi, Madhuri Kambhampati, Oren J. Becher, et al. "Abstract 5004: NG2 upregulation and its defective asymmetric distribution in pediatric brainstem glioma and diffuse intrinsic pontine glioma." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5004.

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Grasso, Catherine S. "Abstract LB-B06: Functionally defined therapeutic targets in diffuse intrinsic pontine glioma." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics; November 5-9, 2015; Boston, MA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1535-7163.targ-15-lb-b06.

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Reports on the topic "Diffuse glioma"

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Becher, Oren, and Alex Chung. Genetically Engineered Mouse Model of Diffuse Intrinsic Pontine Glioma as a Preclinical Tool. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada569511.

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Becher, Oren. Genetically Engineered Mouse Model of Diffuse Intrinsic Pontine Glioma as a Preclinical Tool. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada620002.

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