Relevant bibliographies by topics / Dopaminergic mechanisms

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Dopaminergic mechanisms'

Author: Grafiati
Published: 4 June 2021
Last updated: 30 July 2024

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Journal articles on the topic "Dopaminergic mechanisms"

1

Millichap, J. Gordon, and John J. Millichap. "Dopaminergic Mechanisms in Adhd." Pediatric Neurology Briefs 28, no. 1 (January 1, 2014): 8. http://dx.doi.org/10.15844/pedneurbriefs-28-1-10.

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Razzolini, R., P. Ostan, A. Ramondo, P. Stritoni, R. Chioin, and S. Dalla-Volta. "Dopaminergic mechanisms in heart failure." Pharmacological Research 22 (September 1990): 422. http://dx.doi.org/10.1016/s1043-6618(09)80432-5.

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Barone, P., K. S. Bankiewicz, G. U. Corsini, I. J. Kopin, and T. N. Chase. "Dopaminergic mechanisms in hemiparkinsonian monkeys." Neurology 37, no. 10 (October 1, 1987): 1592. http://dx.doi.org/10.1212/wnl.37.10.1592.

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IMAM, SYED Z. "Molecular Mechanisms of Dopaminergic Neurodegeneration." Annals of the New York Academy of Sciences 993, no. 1 (May 2003): 377. http://dx.doi.org/10.1111/j.1749-6632.2003.tb07547.x.

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Chen, Jianping. "Dopaminergic mechanisms and brain reward." Seminars in Neuroscience 5, no. 5 (October 1993): 315–20. http://dx.doi.org/10.1016/s1044-5765(05)80038-7.

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Lu, Jing-Shan, Qi-Yu Chen, Xiang Chen, Xu-Hui Li, Zhaoxiang Zhou, Qin Liu, Yuwan Lin, Miaomiao Zhou, Ping-Yi Xu, and Min Zhuo. "Cellular and synaptic mechanisms for Parkinson’s disease-related chronic pain." Molecular Pain 17 (January 2021): 174480692199902. http://dx.doi.org/10.1177/1744806921999025.

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Parkinson’s disease is the second most common neurodegenerative disorder after Alzheimer’s disease. Chronic pain is experienced by the vast majority of patients living with Parkinson’s disease. The degeneration of dopaminergic neuron acts as the essential mechanism of Parkinson’s disease in the midbrain dopaminergic pathway. The impairment of dopaminergic neurons leads to dysfunctions of the nociceptive system. Key cortical areas, such as the anterior cingulate cortex (ACC) and insular cortex (IC) that receive the dopaminergic projections are involved in pain transmission. Dopamine changes syn
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Hughes, A., G. Martin, S. Thom, and P. Sever. "Dopaminergic Mechanisms in Human Peripheral Vasculature." Journal of Hypertension 3, no. 6 (December 1985): 664–65. http://dx.doi.org/10.1097/00004872-198512000-00024.

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Wood, Patrick B. "Mesolimbic dopaminergic mechanisms and pain control." Pain 120, no. 3 (February 2006): 230–34. http://dx.doi.org/10.1016/j.pain.2005.12.014.

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Riddle, Evan L., Annette E. Fleckenstein, and Glen R. Hanson. "Mechanisms of methamphetamine-induced dopaminergic neurotoxicity." AAPS Journal 8, no. 2 (June 2006): E413—E418. http://dx.doi.org/10.1007/bf02854914.

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Wickens, J. R., J. C. Horvitz, R. M. Costa, and S. Killcross. "Dopaminergic Mechanisms in Actions and Habits." Journal of Neuroscience 27, no. 31 (August 1, 2007): 8181–83. http://dx.doi.org/10.1523/jneurosci.1671-07.2007.

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Dissertations / Theses on the topic "Dopaminergic mechanisms"

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Bloomfield, Michael. "Dopaminergic mechanisms underlying psychosis." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/44332.

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Schizophrenia is a potentially devastating mental illness with a complex aetiology, in which the odds ratios for environmental risk factors for the disorder are greater than the odds ratios of any single gene hitherto identified. Within schizophrenia, striatal dopamine dysfunction has been proposed to underlie the development of psychosis. The Aberrant Salience hypothesis provides an explanatory model based on empirical findings to explain how psychotic symptoms may arise from striatal hyperdopaminergia, whereby multiple risk factors converge to elevate striatal dopamine synthesis capacity as
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Szostak, Carolyn Margaret. "Dopaminergic mechanisms in conditioned circling." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/29438.

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After unilateral lesions of the mesotelencephalic dopamine (DA) system, the administration of DA receptor agonists results in circling. This response is believed to reflect an asymmetry in mesotelencephalic DA activity. Moreover, drug-induced circling is thought to be directed away from the projection of higher dopaminergic activity. Recently, it has been reported that circling can be established and maintained using operant procedures in surgically intact and drug naive rats. The phenomenon of conditioned circling has been associated with an asymmetrical change in DA metabolism within the str
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Stopper, Colin Michael. "Dopaminergic mechanisms guiding probabilistic choice." Thesis, University of British Columbia, 2014. http://hdl.handle.net/2429/46395.

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Racz, Karoly. "Peripheral dopaminergic mechanisms in experimental hypertension." Thesis, McGill University, 1985. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=72776.

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Pöltl, Dominik [Verfasser]. "Degeneration mechanisms in human dopaminergic neurons / Dominik Pöltl." Konstanz : Bibliothek der Universität Konstanz, 2012. http://d-nb.info/1025226135/34.

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Killcross, Andrew Simon. "Dopaminergic mechanisms and latent inhibition : implications for schizophrenia." Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261541.

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Ferland, Jacqueline-Marie N. "Investigating the influence of risky decision making on dopaminergic reward mechanisms." Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/63131.

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Addiction is a chronic relapsing psychiatric disorder affecting millions worldwide. Despite years of research investigating the etiology and phenomenology of substance abuse, there is no cure. Determining factors which promote the addictive phenotype may help to discover new therapeutics. Several clinical studies have shown addicts demonstrate poor cost/benefit decision making as measured by validated tasks such as the Iowa Gambling Task (IGT), a cognitive deficit maintained during periods of abstinence and associated with relapse risk. However, it is unclear whether disadvantageous choice pre
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Narayanaswami, Vidya. "DIET-INDUCED OBESITY: DOPAMINERGIC AND BEHAVIORAL MECHANISMS AS OUTCOMES AND PREDICTORS." UKnowledge, 2013. http://uknowledge.uky.edu/pharmacy_etds/12.

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Obesity and drug abuse share common neural circuitries including the mesocoticolimbic and striatal dopamine reward system. In the current study, a rat model of diet-induced obesity (DIO) was used to determine striatal dopamine function, impulsivity and motivation as neurobehavioral outcomes and predictors of obesity. For the outcome study, rats were randomly assigned a high-fat (HF) or a low-fat (LF) diet for 8 wk. Following the 8-wk HF-diet exposure, rats were segregated into obesity-prone and obesity-resistant groups based on maximum and minimum body weight gain, respectively, and neurobehav
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Bradley, Curtis. "Examining the Associative Learning and Accumbal Dopaminergic Mechanisms of Caffeine Reinforcement." Digital Commons @ East Tennessee State University, 2018. https://dc.etsu.edu/etd/3457.

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Caffeine is the most consumed psychoactive substance in the world, and most caffeine consumption in coffee and energy drinks is intended to produce a psychoactive effect. However, caffeine is not a primary reinforcer in preclinical paradigms – non-human species do not reliably take the drug to produce a psychoactive effect. However, caffeine is a ‘reinforcement enhancer’ in preclinical models; the effects of caffeine increase the motivation to obtain other non-drug reinforcers. The overall goal of this project was to determine if these reinforcement enhancing effects of caffeine could promote
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Daniel, James St Vincent Clinical School UNSW. "Studies of neurotransmitter release mechanisms in dopamine neurons." Awarded by:University of New South Wales. St. Vincent Clinical School, 2007. http://handle.unsw.edu.au/1959.4/31934.

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Medications that treat diseases such as Parkinson???s disease work by regulating dopamine transmission at synapses. Surprisingly, little is known about the mechanisms regulating dopamine release at synapses. In this thesis, we study mechanisms that regulate vesicle recycling in axons and dendrites of dopamine neurons. Key questions we addressed were: (1) Are vesicles in axons and dendrites associated with the same regulatory proteins, and thus by implication the same regulatory mechanisms, as in excitatory neurons; (2) Do vesicles undergo recycling, and (3) if so, are they characterised by a d
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Books on the topic "Dopaminergic mechanisms"

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1937-, Bodis-Wollner Ivan, Piccolino Marco, and International Brain Research Organization. Congress, eds. Dopaminergic mechanisms in vision. New York: Liss, 1988.

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2

R, Ashby Charles, ed. The modulation of dopaminergic neurotransmission by other neurotransmitters. Boca Raton: CRC Press, 1996.

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3

Kujacic, Mirjana. Dopaminergic control of adrenomedullary function in the rat. Göteberg [Sweden]: Dept. of Pharmacology, University of Göteberg, 1994.

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4

P, Riederer, ed. The Role of brain dopamine. Berlin: Springer-Verlag, 1989.

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5

Paul, Willner, Scheel-Krüger Jørgen, and European Behavioral Pharmacology Society, eds. The mesolimbic dopamine system: From motivation to action. Chichester: Wiley, 1991.

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6

Miller, Robert, 1943 Aug. 29- and Wickens J, eds. Brain dynamics and the striatal complex. Amsterdam, Netherlands: Harwood Academic, 2000.

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7

Anton, Reiner, ed. Phylogeny and development of catecholamine systems in the CNS of vertebrates. Cambridge: University of Cambridge Press, 1994.

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8

Kjell, Fuxe, and Wenner-Grenska samfundet, eds. Trophic regulation of the basal ganglia: Focus on dopamine neurons. Oxford, OX, UK: Pergamon, 1994.

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9

W, Stone T., ed. CNS neurotransmitters and neuromodulators: Glutamate. Boca Raton: CRC Press, 1995.

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10

C, Baron J., and European Economic Community, eds. Brain dopaminergic systems: Imaging with positron tomography : proceedings of a workshop held in Caen, France, within the framework of the European Community medical and public health research. Dordrecht: Kluwer Academic Publishers, 1991.

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Book chapters on the topic "Dopaminergic mechanisms"

1

Memo, M., C. Missale, L. Castelletti, M. Pizzi, and P. F. Spano. "Transduction Mechanisms at Dopamine Receptors." In Dopaminergic Systems and their Regulation, 153–63. London: Palgrave Macmillan UK, 1986. http://dx.doi.org/10.1007/978-1-349-07431-0_10.

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Farsang, Csaba, and Alexander Alföldi. "Opioidergic-Dopaminergic Interaction in Hypertension." In Central Neural Mechanisms in Cardiovascular Regulation, 265–75. Boston, MA: Birkhäuser Boston, 1991. http://dx.doi.org/10.1007/978-1-4615-9834-3_19.

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Felder, Robin A., Robert M. Carey, and Pedro A. Jose. "The Renal Dopaminergic System, Hypertension, and Salt Sensitivity." In Hypertension and Hormone Mechanisms, 159–72. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59259-987-5_10.

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Albert, Paul R., Mohammad H. Ghahremani, and Stephen J. Morris. "Mechanisms of Dopaminergic Regulation of Prolactin Secretion." In The Dopamine Receptors, 359–81. Totowa, NJ: Humana Press, 1997. http://dx.doi.org/10.1007/978-1-4757-2635-0_12.

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Stone, Richard A., Ton Lin, P. Michael Luvone, and Alan M. Laties. "Postnatal Control of Ocular Growth: Dopaminergic Mechanisms." In Ciba Foundation Symposium 155 - Myopia and the Control of Eye Growth, 45–62. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514023.ch4.

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Raiteri, M., G. Bonanno, A. Caviglia, M. Rottigni, and M. Marchi. "Mechanisms Underlying the Modulation of Dopamine Release through Presynaptic Muscarinic Receptors." In Dopaminergic Systems and their Regulation, 35–43. London: Palgrave Macmillan UK, 1986. http://dx.doi.org/10.1007/978-1-349-07431-0_3.

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Huffman, Ronald D., Maharaj K. Ticku, and Joseph M. Frey. "Gabaergic Mechanisms after Manipulation of Striatal Dopaminergic Neurons." In Pharmacology and Functional Regulation of Dopaminergic Neurons, 219–25. London: Palgrave Macmillan UK, 1988. http://dx.doi.org/10.1007/978-1-349-10047-7_35.

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Clopton, J. K., and J. H. Gordon. "Separate Molecular Mechanisms for Estrogen-induced Up- and Down-regulation of Striatal Dopamine Receptors." In Dopaminergic Systems and their Regulation, 293–302. London: Palgrave Macmillan UK, 1986. http://dx.doi.org/10.1007/978-1-349-07431-0_20.

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Glowinski, J., Y. Torrens, and J. C. Beaujouan. "The Striatonigral Substance P Pathway and Dopaminergic Mechanisms." In Novartis Foundation Symposia, 281–95. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720738.ch16.

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Diana, M., Z. L. Rossetti, and G. L. Gessa. "Central Dopaminergic Mechanisms of Alcohol and Opiate Withdrawal Syndromes." In Drug Addiction and Related Clinical Problems, 19–26. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-6901-8_3.

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Conference papers on the topic "Dopaminergic mechanisms"

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Pitta, Marina Galdino da Rocha, Jordy Silva de Carvalho, Luzilene Pereira de Lima, and Ivan da Rocha Pitta. "iPSC therapies applied to rehabilitation in parkinson’s disease." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.022.

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Background: Parkinson’s disease (PD) is a neurological disorder that affects movement, mainly due to damage and degeneration of the nigrostriatal dopaminergic pathway. The diagnosis is made through a clinical neurological analysis where motor characteristics are considered. There is still no cure, and treatment strategies are focused on symptoms control. Cell replacement therapies emerge as an alternative. Objective: This review focused on current techniques of induced pluripotent stem cells (iPSCs). Methods: The search terms used were: “Parkinson’s Disease”, “Stem cells” and “iPSC”. Open arti
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Wong, S. T., J. G. Sivak, A. K. Bal, M. G. Callender, and A. J. Bakelaar. "Changes in Amacrine Cell Numbers and Morphology in Response To Induced Myopia and Hyperopia." In Vision Science and its Applications. Washington, D.C.: Optica Publishing Group, 1998. http://dx.doi.org/10.1364/vsia.1998.suc.2.

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Myopia and hyperopia have been artificially induced in animal models by various manipulations of their early visual environment. Ametropias have been produced using lid suture1, changing illumination levels (dark-rearing2, constant light3, dim lighting4), intra-ocular injections5, treatment with defocussing lenses6, and the application of translucent occluders7. Abnormal ocular growth appears to be a major factor that causes ametropia. Myopic eyes are enlarged, while hyperopic eyes are smaller compared to control eyes. Changes in the sclera8, choroid9, and orbital bone10 surrounding the affect
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Pinheiro, Amanda Pereira Sindeaux, Pedro Vitor Ferreira Rodrigues, Raoni de Oliveira da Silva Domingues, and Leonardo José Rodrigues Araújo Melo. "Gastrointestinal dysmotility associated with Parkinson’s disease’s mechanism." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.461.

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Introduction: Parkinson’s Disease (PD) is a condition of the brain that consiste of the death of dopaminergic neurons in the substantia nigra, therefore causing dyskinesias and dystonias. Besides the motor symptoms, the neurogastro motility is affected by the disease, since gastrointestinal dysfunction is a frequent and clinically relevant symptom of PD. Objectives: To link the neural pathways and neurotransmitters that involve the neuroenteric system control and the PD’s pathology. Methods: A systematic literature review was performed based on data extraction through the advanced research eng
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Chaprov, Kirill, Valeria Goloborshcheva, Ekaterina Teterina, Valeriayn Kucheryanu, Sergey Morozov, and Ruslan Ovchinnikov. "STUDIES OF THE ROLE OF SYNUCLEIN FAMILY PROTEINS IN THE MECHANISM OF TOXIN-INDUCED DAMAGE OF SUBSTANTIA NIGRA DOPAMINERGIC NEURONS." In XVI International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2020. http://dx.doi.org/10.29003/m1325.sudak.ns2020-16/505-506.

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