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Books on the topic 'Drug-receptor interaction'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

G, Cannon Joseph, ed. Advances in CNS Drug-receptor interactions: A research annual. JAIPress, 1991.

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2

Malmberg, Åsa. Dopamine D2 and D3 receptor-ligand interactions: Molecular pharmacology and medicinal chemistry. Acta Universitatis Upsaliensis, 1996.

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3

Kjell, Fuxe, and Agnati Luigi Francesca, eds. Receptor-receptor interactions: A new intramembrane integrative mechanism : proceedings of an international symposium held at the Wenner-Gren Center, Stockholm, October 9th-11th, 1986. Plenum Press, 1987.

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4

Robert, Rein, and Golombek Amram, eds. Computer-assisted modeling of receptor-ligand interactions: Theoretical aspects and applications to drug design : proceedings of the 1988 OHOLO Conference held in Eilat, Israel, April 24-28, 1988. Liss, 1989.

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5

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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6

Molecular foundations of drug-receptor interaction. Cambridge University Press, 1987.

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7

Pharmacologic analysis of drug-receptor interaction. 2nd ed. Raven, 1993.

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8

Pharmacologic analysis of drug-receptor interaction. Raven Press, 1987.

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9

Pharmacologic analysis of drug-receptor interaction. 3rd ed. Lippincott-Raven Publishers, 1997.

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10

Serotonin-dopamine interaction: Experimental evidence and therapeutic relevance. Elsevier, 2008.

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11

Lambert, David G. Mechanisms and determinants of anaesthetic drug action. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0013.

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This chapter is broken into two main sections: a general description of the principles of ligand receptor interaction and a discussion of the main groups of ‘targets’; and explanation of some common pharmacological interactions in anaesthesia, critical care, and pain management. Agonists bind to and activate receptors while antagonists bind to receptors and block the effects of agonists. Antagonists can be competitive (most common) or non-competitive/irreversible. The main classes of drug target are enzymes, carriers, ion channels, and receptors with examples of anaesthetic relevance interacti

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12

Windshügel, Bjö, and Antti Poso. Drug-Receptor Interactions. Wiley & Sons, Incorporated, John, 2021.

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13

Windshügel, Bjö, and Antti Poso. Drug-Receptor Interactions. Wiley & Sons, Incorporated, John, 2017.

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14

Windshügel, Bjö, and Antti Poso. Drug-Receptor Interactions. Wiley & Sons, Incorporated, John, 2021.

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15

Windshügel, Bjö, and Antti Poso. Drug-Receptor Interactions. Wiley & Sons, Incorporated, John, 2021.

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16

Cannon, Joseph G. Advances in Cns Drug Receptor Interactions (Advances in CNS Drug-receptor Interactions). Jai Pr, 1996.

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17

Cannon, J. G. Advances in CNS Drug-Receptor Interactions, Volume 1. Jai Pr, 1991.

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18

Affinity and Efficacy: The Components of Drug-Receptor Interactions. World Scientific Publishing Co Pte Ltd, 2014.

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19

ReceptorReceptor Interactions. Springer, 1987.

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20

Drews, J. Drug Receptor Interactions in Antimicrobial Chemotherapy: "Symposium, Vienna, September 4-6, 1974". Drews J, 2012.

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21

Drug Receptor Interactions in Antimicrobial Chemotherapy: Symposium, Vienna, September 4-6 1974. Springer, 2012.

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22

Insights into Receptor Function and New Drug Development Targets (Research and Perspectives in Endocrine Interactions). Springer, 2006.

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23

(Editor), Michael Conn, C. Kordon (Editor), and Y. Christen (Editor), eds. Insights into Receptor Function and New Drug Development Targets (Research and Perspectives in Endocrine Interactions). Springer, 2006.

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24

Peppin, John, Joseph V. Pergolizzi, Robert B. Raffa, and Steven L. Wright, eds. The Benzodiazepines Crisis. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780197517277.001.0001.

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When properly prescribed, benzodiazepines and related “Z” drugs, are usually safe and effective. However, some patients experience lack of efficacy, severe adverse effects, and/or protracted withdrawal symptoms. Unfortunately, there is no reliable way to predict outcome prior to treatment. Use has dramatically expanded, to the point where some experts suggest a disconnect with actual medical need. With increased and longer prescribing there has been a corresponding increase in the “down-side” of these drugs. Benzodiazepines, as all drugs, produce some degree of normal physiologic tolerance and

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25

Cavacuiti, Christopher. The Pharmacology of Opioids (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190265366.003.0008.

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This chapter focuses on the attributes of and component medications within the class of opioids, emphasizing kinetics, dynamics, and therapeutic and adverse effects. To help patients make informed decisions about opioid use, the clinicians prescribing these medications must be able to explain when opioids are likely to help and when they are likely to do harm. Subclasses of opioids include phenanthrenes, benzomorphans, phenylpiperidines, and diphenylheptanes; examples are given of each, with respective utilities and limitations. A discussion then follows of pharmacodynamics, pharmacokinetics,

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26

Computer-assisted modeling of receptor-ligand interactions: Theoretical aspects and applications to drug design : Proceedings of the 1988 OHOLO Conference ... in clinical and biological research). Liss, 1989.

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27

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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