Relevant bibliographies by topics / Endocrine resistance

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Endocrine resistance'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Endocrine resistance"

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Pinto, Ana Catarina, and Martine J. Piccart-Gebhart. "IN5 ADVANCES IN ENDOCRINE THERAPY AND ENDOCRINE RESISTANCE." Breast 22 (November 2013): S19—S20. http://dx.doi.org/10.1016/s0960-9776(13)70020-1.

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Arneth, Borros. "Insulin Resistance and Glucose Metabolism during Infection." Endocrines 4, no. 4 (2023): 685–95. http://dx.doi.org/10.3390/endocrines4040049.

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Specific critical functions of endocrine and immune cells ensure that an individual remains healthy and free from infection. This study aimed to explore immune–endocrine associations involved in disease. Methods: The PsycINFO, PubMed, Web of Science, and CINAHL databases were searched for relevant articles using the following search terms and phrases: “hormones”, “hormonal responses”, “immune system”, “endocrine system”, “infection”, “immune cells”, “endocrine cells”, “infection”, “immune”, “endocrine”, and “interactions”. The search was limited to articles published between 2009 and 2023. Res
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Zheng, L. H., Y. H. Zhao, H. L. Feng, and Y. J. Liu. "Endocrine resistance in breast cancer." Climacteric 17, no. 5 (2013): 522–28. http://dx.doi.org/10.3109/13697137.2013.864268.

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Dixon, J. M. "Endocrine Resistance in Breast Cancer." New Journal of Science 2014 (September 17, 2014): 1–27. http://dx.doi.org/10.1155/2014/390618.

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Around 70% of all breast cancers are estrogen receptor alpha positive and hence their development is highly dependent on estradiol. While the invention of endocrine therapies has revolusioned the treatment of the disease, resistance to therapy eventually occurs in a large number of patients. This paper seeks to illustrate and discuss the complexity and heterogeneity of the mechanisms which underlie resistance and the approaches proposed to combat them. It will also focus on the use and development of methods for predicting which patients are likely to develop resistance.
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KRAEMER, WILLIAM J. "Endocrine responses to resistance exercise." Medicine & Science in Sports & Exercise 20, Sup 1 (1988): S152—S157. http://dx.doi.org/10.1249/00005768-198810001-00011.

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Nicholson, Robert I., Iain R. Hutcheson, Janice M. Knowlden, et al. "Nonendocrine Pathways and Endocrine Resistance." Clinical Cancer Research 10, no. 1 (2004): 346s—354s. http://dx.doi.org/10.1158/1078-0432.ccr-031206.

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Johnston, Stephen R. D. "Molecular insights into endocrine resistance." European Journal of Cancer Supplements 3, no. 3 (2005): 225–36. http://dx.doi.org/10.1016/s1359-6349(05)80279-4.

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Lei, Jonathan T., Meenakshi Anurag, Svasti Haricharan, Xuxu Gou, and Matthew J. Ellis. "Endocrine therapy resistance: new insights." Breast 48 (November 2019): S26—S30. http://dx.doi.org/10.1016/s0960-9776(19)31118-x.

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Carey, Lisa A. "IN23 RE-DEFINING ENDOCRINE RESISTANCE." Breast 71 (October 2023): S22. http://dx.doi.org/10.1016/s0960-9776(23)00598-2.

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Hartkopf, Andreas D., Eva-Maria Grischke, and Sara Y. Brucker. "Endocrine-Resistant Breast Cancer: Mechanisms and Treatment." Breast Care 15, no. 4 (2020): 347–54. http://dx.doi.org/10.1159/000508675.

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Background: Endocrine treatment is one of the most effective therapies for estrogen receptor-positive breast cancer. However, most tumors will develop resistance to endocrine therapy as the cancer progresses. This review focuses on the mechanisms and markers of endocrine-resistant breast cancer. In addition, current and future strategies to overcome endocrine resistance are discussed. Summary: Several molecular mechanisms of endocrine resistance have been identified, including alterations in the ESR1 gene or in the PIK3CA/mTOR pathway. Meanwhile, CDK4/6, mTOR, and PI3K inhibition have shown to
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Dissertations / Theses on the topic "Endocrine resistance"

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Tewari, Nilanjana. "Mechanisms underlying obesity-related insulin resistance." Thesis, University of Nottingham, 2016. http://eprints.nottingham.ac.uk/34081/.

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This thesis investigates the effect of body composition on insulin resistance and the impact of supplementation with nutritional support or carbohydrate treatment. Insulin resistance occurs as a response to a number of stressors, including surgery. However, the mechanism underlying the development of insulin resistance is as yet unclear. Adipose tissue distribution appears to play a role in the development of insulin resistance and obesity-related complications. In obese and non-obese patients undergoing open abdominal surgery who received preoperative carbohydrate or placebo, there was a sign
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Ertefai, Benyamin. "Resistance mechanisms during endocrine treatment in breast cancer." Thesis, Cardiff University, 2016. http://orca.cf.ac.uk/95393/.

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Prolonged endocrine therapy is the mainstay of treatment for ER+ breast cancer patients. However, resistance develops in many patients which leads to more aggressive disease. Understanding the mechanisms of acquired resistance that emerge as a consequence of prolonged endocrine treatment remains critical. This study aimed to use gene expression profiling to discover induced mechanisms shared by a panel of MCF7-derived acquired resistant cells that underpin endocrine resistant growth. The in vitro panel represents resistance to oestrogen deprivation, tamoxifen or fulvestrant and includes long-t
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McNeil, Catriona Mairi Garvan Institute of Medical Research Faculty of Medicine UNSW. "Downstream targets of the oestrogen receptor and endocrine resistance." Publisher:University of New South Wales. Garvan Institute of Medical Research, 2008. http://handle.unsw.edu.au/1959.4/41025.

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The transcription factor c-Myc is an early downstream target of oestrogen action in breast cancer cells in culture and it has been speculated that aberrant c-Myc expression may mediate antioestrogen resistance. However, studies of c-Myc protein expression as either a prognostic or predictive marker in human breast cancer have been limited and contradictory, as have been studies of c-Myc expression during breast cancer evolution. In order to assess the relationship between c-Myc protein expression and outcome from breast cancer, a representative cohort of 292 women with invasive ductal carcinom
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Moore, Kate. "Collateral resistance to oestrogen and erbB receptor activated growth in endocrine resistant breast cancer." Thesis, University of Edinburgh, 2007. http://hdl.handle.net/1842/24993.

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A panel of breast cancer cell lines (MIII, LCC1, LCC2, LCC9, LY2) derived from the MCF-7 breast cancer cell line with varying oestrogen and anti-oestrogen insensitivity (termed ‘resistant’) was used as a model to determine signalling pathways which may contribute to the development of this insensitivity. 17β-oestradiol (E<sub>2</sub>) only significantly stimulated growth in MCF-7, MIII and LCC1 cell lines. Resistant cells were insensitive to growth factors, while MCF-7 cells remained responsive. MIII and LCC1 cells retained some tamoxifen sensitivity, while the remaining cell lines were unaffe
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Burmi, Rajpal Singh. "Identification of genes associated with endocrine resistance in breast cancer." Thesis, Cardiff University, 2006. http://orca.cf.ac.uk/55621/.

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Resistance to tamoxifen, Faslodex and oestrogen-deprivation represents a major hurdle in breast cancer management, and determining the underlying factors driving resistant growth may improve treatment and prognosis. Expression microarrays (Atlas Plastic Human 12K Microarrays GeneSifter software) were used to identify genes altered in breast cancer models with acquired resistance to tamoxifen (TamR) or Faslodex (FasR) versus their parental MCF-7 cell line through cluster analysis, t-testing and ontological examination. Selected genes were verified by PCR, Western blotting and immunocytochemistr
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Wallace, I. R. "The influence of dietary and endocrine factors on insulin resistance." Thesis, Queen's University Belfast, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.677846.

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Insulin resistance is a characteristic feature of type 2 diabetes mellitus (DM ) and is associated with increased cardiovascular disease. This thesis presents one randomised controlled trial and two cross-sectional analyses, examining the influence of fruit and vegetable consumption and endocrine factors (se~ hormone binding globulin and vitamin D) on insulin resistance. After a 4 week diet of 1-2 portions fruit and vegetables per day, 105 overweight (8MI 27 - 35kg/m2), nondiabetic subjects at elevated cardiovascular disease risk (>20% 1 O-year CVD risk), were randomised to follow a diet of 1-
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Chen, Chun. "Systems Biology Study of Breast Cancer Endocrine Response and Resistance." Diss., Virginia Tech, 2013. http://hdl.handle.net/10919/51965.

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As a robust system, cells can wisely choose and switch between different signaling programs according to their differentiation stages and external environments. Cancer cells can hijack this plasticity to develop drug resistance. For example, breast cancers that are initially responsive to endocrine therapy often develop resistance robustly. This process is dynamically controlled by interactions of genes, proteins, RNAs and environmental factors at multiple scales. The complexity of this network cannot be understood by studying individual components in the cell. Systems biology focuses on the i
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Atefi, Mohammad Sadegh. "Estrogen receptor signaling and mechanism of resistance to endocrine therapy." Diss., Restricted to subscribing institutions, 2008. http://proquest.umi.com/pqdweb?did=1581647181&sid=19&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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Bokhari, Ali, Sathvika Gaddam, and Deepika 7471363 Nallala. "A Case of Compensated Thyroid Hormone Resistance." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/asrf/2019/schedule/50.

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INTRODUCTION Impaired sensitivity to thyroid hormone is described as any process that interferes with the effectiveness of thyroid hormone and includes defects in thyroid hormone action, transport, or metabolism. Here we present a case of a 60-year-old man with resistance to thyroid hormone (RTH), the most common form of impaired sensitivity. CASE A 60-year-old male presented to the endocrinology clinic with complaints of fatigue, decreased concentration, and impaired memory. He denied neck swelling, neck pain, peripheral edema, or any significant changes in weight, temperature sensitivity, bo
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Sadler, Amanda J. "Identification of novel genes associated with endocrine resistance in breast cancer." Thesis, University of Reading, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.485503.

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The overall aims of this project were to identify rnRNAs overexpressed or underexpressed as MCF7 human breast cancer cells progress to growth pathways independent of oestrogen and resistant to the antioestrogen, fulvestrant. Growth of oestrogen maintained and long-term oestrogen deprived MCF7,cells with or without la-8M l7p-oestradiol for 7 days enabled the comparison of expression profiles to identify a number of oestrogen regulated genes, in addition to a number of genes differentially expressed in long-term oestrogen deprived cells compared to cells which had been deprived of oestrogen for
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Books on the topic "Endocrine resistance"

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Cohen, Margo Panush, and Piero P. Foà, eds. Hormone Resistance and Other Endocrine Paradoxes. Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4612-4758-6.

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P, Cohen Margo, and Foà Piero P. 1911-, eds. Hormone resistance and other endocrine paradoxes. Springer-Verlag, 1987.

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Galland, Leo. The fat resistance diet: Reprogram your body to stay thin forever. Rodale, 2008.

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Evanthia, Diamanti-Kandarkis, ed. Insulin resistance and polycystic ovarian syndrome: Pathogenesis, evaluation, and treatment. Humana Press, 2007.

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Berns, P. M. J. J., Romijn J. C, and Schröder F. H, eds. Mechanisms of progression to hormone-independent growth of breast and prostatic cancer. Parthenon Pub. Group, 1991.

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Banyameen, Hene. Coping with pollution: Alteration in metabolic gene expression among sensitive, resistant, and hybrid Fundulus heterolitus embryos during organogenesis. National University, 2015.

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Cohen, Margo P., and Piero P. Foa. Hormone Resistance and Other Endocrine Paradoxes. Springer, 2012.

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Cohen, Margo P., and Piero P. Foa. Hormone Resistance and Other Endocrine Paradoxes. Springer, 2011.

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Hormone Resistance and Other Endocrine Paradoxes. Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1987.

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Cohen, Margo P., and Piero P. Foa. Hormone Resistance and Other Endocrine Paradoxes. Island Press, 1987.

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Book chapters on the topic "Endocrine resistance"

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Shankar, Sudha S., and Helmut O. Steinberg. "Insulin Resistance and Hypertension." In Endocrine Hypertension. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-548-4_12.

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Saez, S. "Drug Resistance." In Endocrine Therapy of Breast Cancer III. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74504-1_3.

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Fu, Xiaoyong, Carmine De Angelis, Jamunarani Veeraraghavan, C. Kent Osborne, and Rachel Schiff. "Molecular Mechanisms of Endocrine Resistance." In Estrogen Receptor and Breast Cancer. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99350-8_11.

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Charmandari, Evangelia, Tomoshige Kino, and George P. Chrousos. "Primary Generalized Familial and Sporadic Glucocorticoid Resistance (Chrousos Syndrome) and Hypersensitivity." In Endocrine Hypertension. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-548-4_4.

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Hiscox, Stephen, Julia Gee, and Robert I. Nicholson. "Endocrine resistance and breast cancer invasion." In Metastasis of Breast Cancer. Springer Netherlands, 2007. http://dx.doi.org/10.1007/978-1-4020-5867-7_7.

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Demetriou, Demetra, Richard Khanyile, Zukile Mbita, and Zodwa Dlamini. "Resistance to Endocrine Therapy in Breast Cancer." In Overcoming Breast Cancer Therapy Resistance. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-52860-6_5.

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McLeod, James F., and John G. Haddad. "Syndromes of Vitamin D Resistance." In Hormone Resistance and Other Endocrine Paradoxes. Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4612-4758-6_5.

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Jordan, V. Craig. "Drug Resistance to Antioestrogen Therapy." In Endocrine Therapy of Breast Cancer VI. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78814-7_7.

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Foà, Piero P. "Introduction: The Journey of the Endocrine Signal: A Paradigm of Murphy’s Law." In Hormone Resistance and Other Endocrine Paradoxes. Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4612-4758-6_1.

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Tager, Howard S. "Insulin Gene Mutations and Abnormal Products of the Human Insulin Gene." In Hormone Resistance and Other Endocrine Paradoxes. Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4612-4758-6_2.

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Conference papers on the topic "Endocrine resistance"

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Chen, S., S. Masri, H. Chan, et al. "Molecular Characterization of Endocrine Resistance." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-407.

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Rody, A., T. Karn, C. Solbach, et al. "The Luminal B Marker NHERF1 Predicts Endocrine Resistance." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-3164.

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Yin, W., G. Liu, G. Di, Z. Shen, and Z. Shao. "Intermittent Hypoxia: Potential Factor of Resistance to Endocrine Therapy." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-5148.

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Lichtenfels, Martina, Gilberto Schwartsmann та Stefan Wiemann. "ESTROGEN RECEPTOR β AND AS A POSSIBLE BIOMARKER OF TAMOXIFEN RESISTANCE". У Scientifc papers of XXIII Brazilian Breast Congress - 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s1048.

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Introduction: Approximately two-thirds of all breast cancer patients overexpress hormonal receptors and are treated with endocrine therapy, being tamoxifen (TAM) the standard treatment. However, many of the initial responders to TAM as first-line experience relapse. Several mechanisms have been proposed to explain the occurrence of acquired TAM resistance. Previous studies showed that estrogen receptor β (ERβ) expression is associated with better response to tamoxifen treatment, as the co-expression of ERα and ERβ is associated with TAM antiproliferative effects. Moreover, there is a growing i
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Anderson, H., H. Anderson, R. A'Hern, et al. "Early Acquired Resistance to Endocrine Therapy: Extending the Neoadjuvant Model." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-2005.

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Chabot, Catherine, and Mark Basik. "Abstract 3564: ARID1A promotes endocrine resistance in ER+ breast cancer cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3564.

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Liu, Zexuan, Junhao Liu, Weiwei Tang, et al. "Abstract 733: Significance of PELP1/SETDB1 axis in endocrine therapy resistance." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-733.

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Gokmen-Polar, Yesim, Yaseswini Nellamraju, Xiaoping Gu, Sarath C. Janga, and Sunil S. Badve. "Abstract 4187: Splicing factorsESRP1/ESRP2as regulators of endocrine resistance in breast cancer." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4187.

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Rudolph, Mark A., Stephen Sizemore, Yuanzhi Lu, et al. "Abstract 3463: Targeting Hedgehog pathway and EGFR signaling to overcome endocrine resistance." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3463.

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Liu, Junhao, Suryavathi Viswanadhapalli, Mengxing Li, et al. "Abstract 4371: PELP1-TFAP2C crosstalk promotes endocrine resistance in breast cancer cells." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-4371.

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Reports on the topic "Endocrine resistance"

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Bergamaschi, Anna. Targeted Approaches to Overcoming Endocrine Resistance in Breast Cancer. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada541957.

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Bergamaschi, Anna. Targeted Approaches to Overcoming Endocrine Resistance in Breast Cancer. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada567133.

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Bergamaschi, Anna. Targeted Approaches to Overcoming Endocrine Resistance in Breast Cancer. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada552549.

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Lyerly, H. K. Developing a HER3 Vaccine to Prevent Resistance to Endocrine Therapy. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada612924.

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Clarke, Robert, Milton Brown, Ayesha N. Shajahan, and Jacqueline Smith. Targeting the UPR to Circumvent Endocrine Resistance in Breast Cancer. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613724.

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Lyerly, H. K. Developing a HER3 Vaccine to Prevent Resistance to Endocrine Therapy. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada597890.

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Lippman, Marc, and Liang Xu. Acquired Tamoxifen Resistance and Overexpression of Anti-Apoptotic Molecules: A Potential Strategy for Overcoming Endocrine Resistance. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada434491.

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Klapperich, Catherine, and Jennifer Rosen. Barriers to Therapy: A Novel 3-D Model to Study the Effect of Tumor Interstitial Pressure on Endocrine-Resistant Breast Cancer. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada475124.

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