Relevant bibliographies by topics / Endothelin Axi

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Endothelin Axi'

Author: Grafiati
Published: 9 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "Endothelin Axi"

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Tyagi, Suresh C., Lane M. Smiley, and Vibhas S. Mujumdar. "Homocyst(e)ine impairs endocardial endothelial function." Canadian Journal of Physiology and Pharmacology 77, no. 12 (1999): 950–57. http://dx.doi.org/10.1139/y99-102.

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Homocyst(e)ine injured vascular endothelium and modulated endothelial-dependent vascular function. Endothelium plays an analogous role in both the vessel and the endocardium. Therefore, we hypothesized that homocyst(e)ine modulated endocardial endothelium (EE) dependent cardiac function. The ex vivo cardiac rings from normal male Wistar-Kyoto rats were prepared. The contractile responses of left and right ventricular rings were measured in an isometric myobath, using different concentrations of CaCl2. The response was higher in the left ventricle than right ventricle and was elevated in endoca
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O. R. Luchko. "A relation of the indices of systemic inflammation and endothelial dysfunction in patients with chronic pyelonephritis and arterial hypertension." Bukovinian Medical Herald 17, no. 1 (65) (2013): 59–63. http://dx.doi.org/10.24061/2413-0737.xvii.1.65.2013.15.

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The dynamics of the indicators of systemic inflammation (C-reactive protein - CRP, tumor necrosis factoralpha – TNF-α, IL-1 – IL-1, IL-6 – IL-6, soluble cell-cell adhesion molecule – sICAM-1) and endothelial dysfunction (pulse blood pressure – PBP, aortic stiffness index – ASI, the thickness of intima-media complex – TIMC, the velocity of the pulse wave propagation – VPWP, CAVI, endothelium dependent vasodilatation – EDVD and endothelium independent vasodilatation – EIVD, endothelin-1 – ET-1) in 105 patients with chronic pyelonephritis (CPN) and arterial hypertension (AH), depending on the glo
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Hao, Ying, Zhuang Wang, Francis Frimpong, and Xingjuan Chen. "Calcium–Permeable Channels and Endothelial Dysfunction in Acute Lung Injury." Current Issues in Molecular Biology 44, no. 5 (2022): 2217–29. http://dx.doi.org/10.3390/cimb44050150.

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The increased permeability of the lung microvascular endothelium is one critical initiation of acute lung injury (ALI). The disruption of vascular-endothelium integrity results in leakiness of the endothelial barrier and accumulation of protein-rich fluid in the alveoli. During ALI, increased endothelial-cell (EC) permeability is always companied by high frequency and amplitude of cytosolic Ca2+ oscillations. Mechanistically, cytosolic calcium oscillations include calcium release from internal stores and calcium entry via channels located in the cell membrane. Recently, numerous publications h
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Lam, Chen-Fuh, Yen-Chin Liu, Jen-Kuo Hsu, et al. "Autologous Transplantation of Endothelial Progenitor Cells Attenuates Acute Lung Injury in Rabbits." Anesthesiology 108, no. 3 (2008): 392–401. http://dx.doi.org/10.1097/aln.0b013e318164ca64.

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Background Acute lung injury (ALI) and end-stage acute respiratory distress syndrome (ARDS) are among the most common causes of death in intensive care units. Activation and damage of pulmonary endothelium is the hallmark of ALI/ARDS. Recent studies have demonstrated the importance of circulating endothelial progenitor cells (EPCs) in maintaining normal endothelial function as well as endothelial repairing after vascular injury. Here, the authors present the first study demonstrating the therapeutic potential of EPCs in a rabbit model of ALI/ARDS. Methods Circulating EPCs were obtained from ra
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Vorob’eva, Nadezda A., Alena I. Vorob’eva, and Anastasiya A. Marusiy. "Risk of Endothelial Dysfunction and Total Antioxidant Capacity in Seafarers During an Arctic Voyage." Journal of Medical and Biological Research, no. 2 (May 9, 2021): 192–200. http://dx.doi.org/10.37482/2687-1491-z057.

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Among other areas of research, clinical medicine studies the adaptation mechanisms of the vascular endothelium to the extreme conditions of the Arctic. This paper dwells on the possible relationship of the development of endothelial dysfunction and the antioxidant system with modifiable risk factors during a short translatitudinal voyage in the Arctic. The study involved 32 crew members of the research vessel Mikhail Somov during the TransArctic–2019 research expedition. Venous blood sampling was carried out before the voyage at the zero point (Arkhangelsk, 64°33’N 40°32’E) and at the highest
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Mejía-Salgado, Germán, Paula Tatiana Muñoz-Vargas, Carlos Cifuentes-González, et al. "Quantitative changes in the corneal endothelium and central corneal thickness during anterior chamber inflammation: A systematic review and meta-analysis." PLOS ONE 19, no. 1 (2024): e0296784. http://dx.doi.org/10.1371/journal.pone.0296784.

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Purpose To establish the effects of anterior chamber inflammation (ACI) on the corneal endothelium parameters and central corneal thickness (CCT). Methods We conducted a comprehensive literature review using medical databases (PubMed, EMBASE, VHL, and medRxiv) on March 8, 2023, for studies that included patients with ACI who had undergone specular microscopy or pachymetry. Case series with >10 patients, cross-sectional, case-control, and cohort studies were included. The risk of bias was assessed using CLARITY tools and validated scales such as those by Hassan Murad et al. and Hoy et al. A
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Zaikina, T. S., P. G. Kravchun, D. V. Minukhina, D. V. Minukhin, D. O. Yevtushenko, and I. O. Kudrevych. "COMPREHENSIVE ASSESSMENT OF ENDOTHELIUM-DEPENDENT MEDIATORS IN PATIENTS WITH ACUTE MYOCARDIAL INFARCTION AND DIABETES MELLITUS TYPE 2." Problems of Endocrine Pathology 76, no. 2 (2021): 14–18. http://dx.doi.org/10.21856/j-pep.2021.2.02.

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The aim of study is to evaluate the levels of endothelium-dependent mediators: endothelial nitric-oxide synthase (NOS), plasminogen activator inhibitor-1 (PAI-1) and circulating soluble CD40 ligand (sCD40L) in patients with acute myocardial infarction (AMI) and concomitant type 2 diabetes mellitus (DM). The study included 255 patients with AMI, who were divided into two groups depending on the presence of concomitant type 2 DM: 1 group — 143 patients with concomitant type 2 DM; 2 group — 112 patients without concomitant disturbances of carbohydrate metabolism. Studied endothelial-dependent ind
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Wang, Fei, Hui Zhang, Kotaro Uchida, et al. "The Evaluation Value of Non-Invasive Indices of Arterial Stiffness in the Early Stage of Coronary Artery Disease: Preliminary Results from an Exploratory Study." Journal of Vascular Diseases 3, no. 3 (2024): 278–89. http://dx.doi.org/10.3390/jvd3030022.

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Background: Recently, the arterial velocity pulse index (AVI) and arterial pressure volume index (API) have been used to evaluate arterial stiffness and endothelial function. As arterial stiffness and endothelial injury are risk factors for coronary artery disease (CAD), these two indexes are therefore expected to predict and evaluate the future risk of CAD and cardiovascular events before clinical manifestations. Methods: A total of 90 consecutive patients with coronary angiography (CAG) were enrolled. After excluding normal patients and acute coronary syndrome patients, forty-seven patients
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Kolosova, Irina A., Tamara Mirzapoiazova, Liliana Moreno-Vinasco, Saad Sammani, Joe G. N. Garcia та Alexander D. Verin. "Protective effect of purinergic agonist ATPγS against acute lung injury". American Journal of Physiology-Lung Cellular and Molecular Physiology 294, № 2 (2008): L319—L324. http://dx.doi.org/10.1152/ajplung.00283.2007.

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Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) are major causes of acute respiratory failure associated with high morbidity and mortality. Although ALI/ARDS pathogenesis is only partly understood, pulmonary endothelium plays a major role by regulating lung fluid balance and pulmonary edema formation. Consequently, endothelium-targeted therapies may have beneficial effects in ALI/ARDS. Recently, attention has been given to the therapeutic potential of purinergic agonists and antagonists for the treatment of cardiovascular and pulmonary diseases. Extracellular purines (ad
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Dube, Shataakshi, Tejasvi Matam, Jessica Yen, et al. "Endothelial STAT3 Modulates Protective Mechanisms in a Mouse Ischemia-Reperfusion Model of Acute Kidney Injury." Journal of Immunology Research 2017 (2017): 1–9. http://dx.doi.org/10.1155/2017/4609502.

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STAT3 is a transcriptional regulator that plays an important role in coordinating inflammation and immunity. In addition, there is a growing appreciation of the role STAT3 signaling plays in response to organ injury following diverse insults. Acute kidney injury (AKI) from ischemia-reperfusion injury is a common clinical entity with devastating consequences, and the recognition that endothelial alterations contribute to kidney dysfunction in this setting is of growing interest. Consequently, we used a mouse with a genetic deletion of Stat3 restricted to the endothelium to examine the role of S
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Dissertations / Theses on the topic "Endothelin Axi"

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SPINA, CECILIA. "Emerging Evidences for a Protumorigenic Role of the Endothelin Axis Uncover New Therapeutic Targets in Multiple Myeloma." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2017. http://hdl.handle.net/10281/144189.

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Multiple Myeloma (MM) is a monoclonal tumor of bone marrow (BM) plasma cell (PC), usually associated with a number of disease manifestations, including skeletal damage, anemia and immunosuppression. Despite recent therapeutic advances and an increased patient life expectancy, MM still remains an incurable malignancy. MM PC is characterized by a strong BM niche dependence, acquiring an autonomous capacity of growth only in the latter stages of disease. The endothelin (ET) axis is mainly composed of Endothelin-1 (ET-1), and 2 G-protein coupled receptors, the ET receptor A (ETAR) and ET recepto
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Adner, Mikael. "Altered expression of contractile endothelin receptors in the vascular bed." Lund : Dept. of Internal Medicine, University of Lund, 1998. http://catalog.hathitrust.org/api/volumes/oclc/39103326.html.

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Irani, Soussan. "The Endothelin Axis and Angiogenesis in Papillary Thyroid Carcinoma." Thesis, Griffith University, 2014. http://hdl.handle.net/10072/366833.

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Papillary Thyroid Cancer (PTC) is the most common thyroid cancer accounting for 80% of all cases. The prognosis is good, with 5-year survival rates of 95%, but in some cases the tumour behaves in an aggressive manner characterized by local recurrence and/or metastasis, processes contributed to by angiogenesis. Angiogenesis is an essential physiologic activity involved in normal tissue biology and several pathologic conditions such as cardiac failure, and cancer. During carcinogenesis, tumour cells secrete pro-angiogenic factors to initiate angiogenesis. Angiogenesis also causes the migration
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Rabura, Sebastian. "Bedeutung der Endothelin-Plasmakonzentration für die Effektivität von inhalativem Stickstoffmonoxid im Modell des akuten Lungenversagens." Doctoral thesis, Universitätsbibliothek Leipzig, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-137313.

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Das akute Lungenversagen ist gekennzeichnet durch eine schwere Störung des Gasaustausches mit ausgeprägter arterieller Hypoxämie. Die inhalative Gabe von Stickstoffmonoxid (iNO) erfolgt in der Therapie zur Verbesserung der arteriellen Oxygenierung, der Effekt ist jedoch variabel. Bisher existieren nur wenige Studien zur Identifikation von Faktoren, die die Effektivität von iNO bestimmen. Die positive Wirkung von iNO auf den Gasaustausch lässt eine Vasokonstriktion in beatmeten Lungenarealen vermuten. Wir untersuchten eine mögliche Wechselwirkung zwischen dem endogenen Vasokonstriktor Endotheli
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Borrull, Aurélie. "Obtention et caractérisation d’anticorps monoclonaux dirigés contre les récepteurs des endothélines, ETAR et ETBR, surexprimés dans de nombreux cancers et impliqués dans la progression tumorale." Thesis, Paris 11, 2015. http://www.theses.fr/2015PA114820/document.

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Il est admis que l’axe endothéline (endothélines ET-1, -2 et -3 et leurs RCPG ETAR et ETBR), participe à la progression tumorale. Alors qu’ETAR est par exemple surexprimé dans le cancer de l’ovaire, ETBR l’est dans le mélanome. Cette surexpression, ainsi que l’implication d’ETA/BR dans la carcinogenèse, font de ces RCPG une cible tumorale pertinente. En raison de leurs forte spécificité, actions cytotoxiques variées, possibilités de couplage, les anticorps monoclonaux (AcM) sont des outils de choix en diagnostic et thérapie anti-cancéreuse. Cependant, on déplore actuellement l’absence d’AcM ci
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Allard, Bertrand. "Production et caractérisation d’anticorps polyclonaux et monoclonaux ciblant les récepteurs des endothélines en vue d’une immunothérapie des cancers." Thesis, Paris 11, 2012. http://www.theses.fr/2012PA114803/document.

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Le développement des anticorps monoclonaux thérapeutiques est en plein essor notamment à cause de leur bénéfice important pour le traitement des cancers. Cependant, à l’heure actuelle, aucun anticorps monoclonal sur le marché ou en phase III ne cible de RCPGs, en dépit de l’implication grandissante de ces récepteurs dans la carcinogenèse. Parmi les RCPGs les plus pertinents pour l’oncologie, souvent cités dans la littérature et dont certains inhibiteurs chimiques sont en phase clinique avancée, on trouve les deux sous-types de récepteurs des endothélines ETAR et ETBR. Dans ce contexte, mon pro
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Miyoshi, Takashi. "The role of endothelial interleukin-8/NADPH oxidase 1 axis in sepsis." Kyoto University, 2011. http://hdl.handle.net/2433/142065.

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Sax, Michael John. "The CCL5-CCR5 Axis in Breast Cancer." Thesis, Griffith University, 2015. http://hdl.handle.net/10072/365646.

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Cancer is one of the leading underlying causes of death worldwide. Despite decades of research, cancer is predicted to be the leading cause of death by the year 2020. Anti angiogenic therapies that target the key signalling molecule, vascular endothelial growth factor (VEGF) have shown promise in the treatment of some solid tumours, resulting in increased progression-free survival times in patients. However, there are several significant problems with current anti-angiogenic therapies, such as the phenomenon of resistance. Tumours can be divided into those that are intrinsically nonresponsive
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Fang, Jennifer S., Brian G. Coon, Noelle Gillis, et al. "Shear-induced Notch-Cx37-p27 axis arrests endothelial cell cycle to enable arterial specification." NATURE PUBLISHING GROUP, 2017. http://hdl.handle.net/10150/626459.

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Establishment of a functional vascular network is rate-limiting in embryonic development, tissue repair and engineering. During blood vessel formation, newly generated endothelial cells rapidly expand into primitive plexi that undergo vascular remodeling into circulatory networks, requiring coordinated growth inhibition and arterial-venous specification. Whether the mechanisms controlling endothelial cell cycle arrest and acquisition of specialized phenotypes are interdependent is unknown. Here we demonstrate that fluid shear stress, at arterial flow magnitudes, maximally activates NOTCH signa
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Lindberg, Lars. "Endothelial function during ischemia-reperfusion and effects of inhalation of nitric oxide." Lund : Dept. of Anesthesiology and Intensive Care, University of Lund, 1996. http://catalog.hathitrust.org/api/volumes/oclc/38164585.html.

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Books on the topic "Endothelin Axi"

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McAuley, Danny F., and Thelma Rose Craig. Measurement of extravascular lung water in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0140.

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The accumulation of fluid in the interstitium and alveolar space is known as extravascular lung water (EVLW). EVLW is associated with increased morbidity and mortality in critically ill patients and is elevated in patients with cardiogenic pulmonary oedema, acute lung injury (ALI), and the acute respiratory distress syndrome (ARDS). Pulmonary oedema is a consequence of increased pulmonary capillary hydrostatic pressure and/or an increased capillary permeability. The quantity of pulmonary oedema fluid is dependent on the balance of fluid formation and clearance, and this contributes to the over
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Goligorsky, Michael S., Julien Maizel, Radovan Vasko, May M. Rabadi, and Brian B. Ratliff. Pathophysiology of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0221.

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In the intricate maze of proposed mechanisms, modifiers, modulators, and sensitizers for acute kidney injury (AKI) and diverse causes inducing it, this chapter focuses on several common and undisputable strands which do exist.Structurally, the loss of the brush border, desquamation of tubular epithelial cells, and obstruction of the tubular lumen are commonly observed, albeit to various degrees. These morphologic hallmarks of AKI are accompanied by functional defects, most consistently reflected in the decreased glomerular filtration rate and variable degree of reduction in renal blood flow, a
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Book chapters on the topic "Endothelin Axi"

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Gordon, John. "1992 NATO ASI Conference on “Vascular Endothelium: Physiological Basis of Clinical Problems II”." In Vascular Endothelium. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2437-3_14.

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Becker, Richard C., and Frederick A. Spencer. "Vascular Biology, Thromboresistance, and Inflammation." In Fibrinolytic and Antithrombotic Therapy. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195155648.003.0006.

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The delivery of vital substrate to metabolically active tissues and vital organs is achieved and maintained by the cardiovascular system including the heart, macrovasculature, and microvasculature. This life-sustaining process requires a normally functioning vascular endothelium—a multifunctional organ system composed of physiologically responsive cells responsible for vasomotion (vascular tone), thromboresistance, and inflammoresistance. Simply by virtue of its anatomic location, the vascular endothelium is functionally complex. It defines the intra- and extravascular components, serves as a
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Setyawati, Titik, Ricky Aditya, and Tinni Trihartini Maskoen. "Sepsis Associated Acute Kidney Injury." In Infectious Diseases and Sepsis [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97609.

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AKI is a syndrome consisting of several clinical conditions, due to sudden kidney dysfunction. Sepsis and septic shock are the causes of AKI and are known as Sepsis-Associated AKI (SA-AKI) and accounted for more than 50% of cases of AKI in the ICU, with poor prognosis. Acute Kidney Injury (AKI) is characterized by a sudden decline in kidney function for several hours/day, which results in the accumulation of creatinine, urea and other waste products. The most recent definition was formulated in the Kidney Disease consensus: Improving Global Outcome (KDIGO), published in 2012, where the AKI was
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Moreira, Roberto de Souza, Maria Claudia Costa Irigoyen, José Manuel Condor Capcha, et al. "Protective effect of apolipoprotein AI mimetic peptide 4F on renal and cardiac injury and endothelial dysfunction induced by acute myocardial infarction in hypercholesterolemic rats receiving iodinated contrast." In Medicine: an exploration of the anatomy of the human body. Seven Editora, 2024. http://dx.doi.org/10.56238/sevened2024.005-019.

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The use of contrast after angiography in infarcted animals induces acute kidney injury, being associated with worsening prognosis and increased mortality. Hypercholesterolemia is an aggravating factor of endothelial injury in acute myocardial infarction (AMI) and the use of contrast in diagnosis and treatment can cause acute kidney injury. Treatment with the apolipoprotein AI mimetic peptide 4F can reverse endothelial injury by reducing LDL levels and preventing its oxidation. OBJECTIVES: To analyze the effect of Apo A-I (using mimetic peptide 4F) on cardiac and renal injury induced by acute m
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Piña, Ileana L., and Magdy Abdelhamid. "Angiotensin receptor blockers in heart failure." In The ESC Textbook of Heart Failure, edited by Petar M. Seferović, Andrew J. S. Coats, Gerasimos Filippatos, Stefan D. Anker, Johann Bauersachs, and Giuseppe Rosano. Oxford University PressOxford, 2023. http://dx.doi.org/10.1093/med/9780198891628.003.0041.

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Abstract Excess activation of the renin–angiotensin system (RAS) contributes to the pathophysiology of heart failure. Activation of the RAS results in increased production of angiotensin I (AI), which is converted to angiotensin II (AII) by angiotensin-converting enzyme (ACE). AII is a potent vasoconstrictor and also stimulates aldosterone secretion, which increases sodium and water retention. AII and aldosterone are also implicated in other potentially deleterious effects on the cardiovascular system, including endothelial damage, sympathetic activation, collagen formation, and decreased nitr
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Ghosh, Samit. "Kidney Injuries in Sickle Cell Disease." In Sickle Cell Disease [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.102839.

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Sickle cell disease (SCD), characterized by the presence of unstable sickle hemoglobin in the homozygous state (HbSS), results in progressive organ damage and early mortality with the median age of death in the 40s. The kidney is one of the most severely affected organs in SCD. Kidney diseases gradually develop in individuals with SCD. Microalbuminuria is evident in childhood, progressing to apparent proteinuria, deteriorating glomerular filtration rate (GFR) in early adulthood. While CKD becomes prevalent in adults. Moreover, among SCD patients, exacerbation of anemia is an independent risk f
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B. Oien, Derek, Jeremy Chien, Julian Molina, and Viji Shridhar. "Emerging Drug Therapies for Mesothelioma." In Mesothelioma. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.91752.

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The systemic chemotherapy combination of cisplatin and pemetrexed has been the mesothelioma standard of care for well over a decade. This regimen has only achieved a disappointing overall median survival of about 1 year. Improved survival has been reported when systemic chemotherapy is combined with surgery and radiotherapy, and for using localized chemotherapy in some cases. The choice of mesothelioma treatment often depends on the anatomical location, histologic subtype, and disease progression. Several experimental drugs have also been investigated in mesothelioma, often with limited positi
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Sharma, Yogita, Neeraj Kumar, and Devyani Thakur. "Interleukin 6 in Patients with Rheumatoid Arthritis." In Interleukins - The Immune and Non-Immune Systems’ Related Cytokines. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96887.

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Rheumatoid Arthritis is a widespread disease causing varying degrees of disability. It is characterised by flares and remissions and since ancient times, every culture has tried to get the better of it. Even now, research is aimed at finding novel serum biomarkers as surrogates for disease activity and newer targets to sharpen therapy. One such target is IL-6.It mediates neutrophil migration, osteoclast maturation and pannus formation through vascular endothelial growth factor (VEGF) stimulation causing synovitis and joint destruction.IL-6 leads to various systemic manifestations like hepcidin
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Li, Sinian, Yiming Shao, Kanglan Li, et al. "Vascular Cognitive Impairment and the Gut Microbiota." In Advances in Alzheimer’s Disease. IOS Press, 2022. http://dx.doi.org/10.3233/aiad220026.

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Vascular cognitive impairment (VCI), the second most common cause of dementia in elderly people, is a term that refers to all forms of cognitive disorders that can be attributed to cerebrovascular disease such as manifestations of discrete infarctions, brain hemorrhages, and white matter lesions. The gut microbiota (GM) has emerged recently as an essential player in the development of VCI. The GM may affect the brain’s physiological, behavioral, and cognitive functions through the brain-gut axis via neural, immune, endocrine, and metabolic pathways. Therefore, microbiota dysbiosis may mediate
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Vicinanza, Roberto, Giuseppe Coppotelli, Carolina Malacrino, et al. "Role of LDL Oxidation on Non-Genomic Thyroid Hormone Action in Human Endothelial Cells." In BASIC - Hypothalamic-Pituitary-Thyroid Axis: Thyroid Hormone Metabolism, Cellular Uptake & Action. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p16.p1-664.

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Conference papers on the topic "Endothelin Axi"

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Kim, Mark S., Hyun Jin Choi, Ho-Jeong Lee, et al. "Abstract 5463: Role of the endothelin axis in astrocyte and endothelial cell mediated chemoprotection of cancer cells." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-5463.

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Said, Neveen A., and Dan Theodorescu. "Abstract 2280: Endothelin axis in bladder cancer metastasis." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2280.

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Gupta, Suprit, Satyanarayana Rachagani, Sushil Kumar, Kavita Mallya, Surinder Kumar Batra, and Maneesh Jain. "Abstract 4191: Alterations in endothelin axis during pancreatic acinar to ductal metaplasia." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4191.

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Bhagwani, A. R., M. Ali, E. A. Goncharova, and L. Farkas. "Endothelial Loss of P53 Promotes Endothelial Dysfunction via Impaired TLR3-BMP Axis in Pulmonary Arterial Hypertension." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3652.

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Rizzi, Simone C., Charlotte Mermier, Sophie Crettaz, Valeria Blumer-Nesca, and Céline Gandar. "Abstract 5098: In vitro designer microenvironments for modeling the endothelin axis in brain cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-5098.

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Yu, Hyun Kyung, Ho Jeong Lee, Fahao Zhang, Qiuyu Wu, Isaiah J. Fidler, and Sun Jin Kim. "Abstract 2943: Hypoxia induced resistance to chemotherapy is regulated by the endothelin receptor axis." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-2943.

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Hirani, D. V. B., V. Wagde, J. Selle, et al. "Targeting IL-6 and Endothelin Axis Enables Alveolar Growth in Mechanically Ventilated Newborn Mice." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a6360.

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Gupta, Suprit, Satyanarayana Rachagani, Sushil Kumar, Surinder Kumar Batra, and Maneesh Jain. "Abstract 2691: Cigarette smoke induced upregulation of endothelin axis in the initiation of pancreatic cancer." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2691.

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Kim, Seung Wook, Sun Jin Kim, Hyun Jin Choi, et al. "Abstract 4951: The role of endothelin axis signaling pathway in astrocyte mediated chemoprotection of tumor cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4951.

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Mimmler, M., O. Dremova, A. Mann, et al. "Small intestinal villus vascularization is driven via an epithelial-to-endothelial PAR1 signaling axis." In GTH Congress 2024 – 68th Annual Meeting of the Society of Thrombosis and Haemostasis Research – Building Bridges in Coagulation. Georg Thieme Verlag, 2024. http://dx.doi.org/10.1055/s-0044-1779238.

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Reports on the topic "Endothelin Axi"

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Wolfenson, David, William W. Thatcher, and James E. Kinder. Regulation of LH Secretion in the Periovulatory Period as a Strategy to Enhance Ovarian Function and Fertility in Dairy and Beef Cows. United States Department of Agriculture, 2003. http://dx.doi.org/10.32747/2003.7586458.bard.

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Abstract:
The general research objective was to increase herd pregnancy rates by enhancing corpus luteum (CL) function and optimizing follicle development, in order to increase conception rate and embryo survival. The specific objectives were: to determine the effect of the duration of the preovulatory LH surge on CL function; to determine the function of LH during the postovulatory period on CL development; to optimize CL differentiation and follicle development by means of a biodegradable GnRH implant; to test whether optimization of CL development and follicle dynamics in timed- insemination protocol
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