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Dissertations / Theses on the topic 'Fetal Fibroblast'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Maker, Garth Lucas. "Regulation of surfactant production by fetal type II pneumocytes and characterization of fibroblast-pneumocyte factor /." Access via Murdoch University Digital Theses Project, 2007. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080430.141113.

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2

Franke, Jana, Vanessa Abs, Claudia Zizzadoro, and Getu Abraham. "Comparative study of the effects of fetal bovine serum versus horse serum on growth and differentiation of primary equine bronchial fibroblasts." Universitätsbibliothek Leipzig, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-144893.

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Background: Airway fibroblasts have become a critical addition to all facets of structural lung tissue changes such as in human asthma and chronic obstructive pulmonary disease, but little is known about their role in the equine recurrent airway obstruction, a disease that resembles to the human asthma. Since the equine bronchial fibroblasts (EBF) have not been isolated and characterized yet, the use of defined medium was investigated. Results: Primary EBF were cultured on non-collagen coated flasks without serum or in the presence of feta bovine serum (FBS) or horse serum (HS) or in serum dep

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3

au, G. Maker@murdoch edu, and Garth Lucas Maker. "Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor." Murdoch University, 2008. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080430.141113.

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The fetal lung undergoes extensive physiological and biochemical maturation prior to birth in preparation for its postnatal function as an organ for gas exchange. Pulmonary surfactant, a substance that reduces surface tension and prevents alveolar collapse, is produced by type II pneumocytes within the lung. Reduced ability to produce surfactant leads to neonatal respiratory distress syndrome. Synthesis of the phospholipid component of surfactant, phosphatidylcholine (PC), is stimulated by fibroblast-pneumocyte factor (FPF), a protein expressed by fibroblast cells within the fetal lung. Althou

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4

Maker, Garth Lucas. "Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor." Thesis, Maker, Garth Lucas ORCID: 0000-0003-1666-9377 (2008) Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor. PhD thesis, Murdoch University, 2008. https://researchrepository.murdoch.edu.au/id/eprint/486/.

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The fetal lung undergoes extensive physiological and biochemical maturation prior to birth in preparation for its postnatal function as an organ for gas exchange. Pulmonary surfactant, a substance that reduces surface tension and prevents alveolar collapse, is produced by type II pneumocytes within the lung. Reduced ability to produce surfactant leads to neonatal respiratory distress syndrome. Synthesis of the phospholipid component of surfactant, phosphatidylcholine (PC), is stimulated by fibroblast-pneumocyte factor (FPF), a protein expressed by fibroblast cells within the fetal lung. Althou

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5

Maker, Garth Lucas. "Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor." Maker, Garth Lucas (2008) Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor. PhD thesis, Murdoch University, 2008. http://researchrepository.murdoch.edu.au/486/.

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The fetal lung undergoes extensive physiological and biochemical maturation prior to birth in preparation for its postnatal function as an organ for gas exchange. Pulmonary surfactant, a substance that reduces surface tension and prevents alveolar collapse, is produced by type II pneumocytes within the lung. Reduced ability to produce surfactant leads to neonatal respiratory distress syndrome. Synthesis of the phospholipid component of surfactant, phosphatidylcholine (PC), is stimulated by fibroblast-pneumocyte factor (FPF), a protein expressed by fibroblast cells within the fetal lung. Althou

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6

Schmidt, Joachim. "A novel binding protein for fibroblast growth factors (FGF-BP2) cloning, expression profile, tumorigenic activity and regulation of gene expression by fetal bovine serum and retinoic acid /." [S.l.] : [s.n.], 2003. http://archiv.ub.uni-marburg.de/diss/z2003/0713/.

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7

Mello, Marco Roberto Bourg de. ""Clonagem em bovinos: uso de fibroblastos fetal e adulto como fonte doadora de núcleo"." Universidade de São Paulo, 2003. http://www.teses.usp.br/teses/disponiveis/10/10131/tde-29062005-161626/.

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O objetivo deste estudo foi avaliar a viabilidade in vitro e in vivo de embriões bovinos reconstruídos com oócitos enucleados em Metáfase II e núcleos de células somáticas (fibroblastos) fetais e adultas. Para tanto, oócitos de ovários colhidos em matadouro foram maturados in vitro por 17 horas e enucleados pela remoção do primeiro corpúsculo polar (CP) e da região do oolema contendo a placa metafásica. Como núcleo doador, foram utilizados fibroblastos de orelha de vaca da raça Nelore e de feto colhido em abatedouro. Para a reconstrução dos embriões, cada célula doadora de núcleo, após indução

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8

Zaunbrecher, Gretchen Marie. "Comparison of several protocols for the increase in homologous recombination in normal porcine fetal fibroblasts and the application to an actual locus." Diss., Texas A&M University, 2003. http://hdl.handle.net/1969.1/76.

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Together with the advancements in animal cloning, the ability to efficiently target specific genes in somatic cells would greatly enhance several areas of research. While it has been possible for quite some time to target specific genes in the germ cells of mice, the advancements in somatic cell gene targeting has been slowed for two main reasons. First, the finite lifespan of somatic cells, due mainly to the inability of the somatic cells to regenerate or maintain their telomeres, poses a major problem given the lengthy selection process needed to identify a targeting event. The second pro

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9

Montenegro, Luciana Ribeiro. "Estudo in vitro da sensibilidade ao IGF-1 de fibroblastos de crianças nascidas pequenas para a idade gestacional sem recuperação estatural pós-natal." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-08092009-132008/.

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Introdução: Crianças nascidas pequenas para a idade gestacional (PIG) apresentam maior risco de permanecerem com baixa estatura na vida adulta. Os fatores de crescimento insulino-símile tipo 1 e 2 (IGF-1 e IGF-2) são os principais fatores endócrinos determinantes do crescimento fetal. A maioria das ações conhecidas do IGF-1 e 2 é mediada via um receptor tirosina quinase, conhecido como IGF-1R. Recentemente, a insensibilidade ao IGF-1 foi identificada como uma das causas de retardo de crescimento em crianças nascidas PIG que não apresentaram recuperação espontânea do crescimento na vida pós-nat

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10

Cavallin, Mara. "Physiopathologie moléculaire et cellulaire des anomalies du développement du cortex cérébral : le syndrome d'Aicardi WDR81 mutations cause extreme microcephaly and impair mitotic progression in human fibroblasts and Drosophila neural stem cells TLE1, a key player in neurogenesis, a new candidate gene for autosomal recessive postnatal microcephaly Mutations in TBR1 gene leads to cortical malformations and intellectual disability Aicardi syndrome: Exome, genome and RNA-sequencing of a large cohort of 19 patients failed to detect the genetic cause Recurrent RTTN mutation leading to severe microcephaly, polymicrogyria and growth restriction Recurrent KIF2A mutations are responsible for classic lissencephaly Recurrent KIF5C mutation leading to frontal pachygyria without microcephaly Rare ACTG1 variants in fetal microlissencephaly De novo TUBB2B mutation causes fetal akinesia deformation sequence with microlissencephaly: An unusual presentation of tubulinopathy A novel recurrent LIS1 splice site mutation in classic lissencephaly Further refinement of COL4A1 and COL4A2 related cortical malformations Prenatal and postnatal presentations of corpus callosum agenesis with polymicrogyria caused By EGP5 mutation Delineating FOXG1 syndrome from congenital microcephaly to hyperkinetic encephalopathy Delineating FOXG1 syndrome: From congenital microcephaly to hyperkinetic encephalopathy." Thesis, Sorbonne Paris Cité, 2019. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2213&f=18201.

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Les malformations du cortex cérébral (MDC) représentent une cause importante de handicap et d'épilepsie pharmaco-résistante. Le séquençage à haut débit a permis une amélioration considérable de l'identification des bases moléculaires des MDC non syndromiques. Toutefois, certaines formes, notamment les MDC complexes, demeurent inexpliquées. Mon projet de thèse a pour objectif de progresser dans la compréhension des MDC complexes en utilisant deux modèles : les microlissencéphalies (MLIS) et le syndrome d'Aicardi (AIC), une forme syndromique particulière associant des malformations de l'oeil et

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11

Lin, Yi, and 林毅. "Study Of Fetal Mice Dental Stem Cells Cultured With Fibroblast Growth Factor -9." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/45430088466739617567.

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碩士<br>國立臺灣大學<br>口腔生物科學研究所<br>97<br>The human dentition is indispensable for nutrition, communication and physiology. Caries, pulpitis, apical periodontitis and trauma may lead to loss of tooth, causing the problem of pronunciation, mastication, and appearance difficulities. Abiological tooth substitute that could replace lost teeth would provide a vital alternative to currently avalible clinical treatment. For years research, people have obtained much more knowledge about this field. By animal model of embryonic tooth germ, we can understand how tooth germ cells separation of embryo layer, cul

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12

Ahmed, Asra. "Apoptosis and caspase-3 activity in isolated fetal rat lung cells, human A549 cells and rat periodontal ligament fibroblasts following exposure to cigarette smoke extract." 2012. http://hdl.handle.net/1993/5205.

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Exposure cigarette smoke (CS) during prenatal life is the leading cause of preventable premature death. In this study, we explored the hypothesis that in vitro exposure of fetal lung cells to cigarette smoke extract (CSE) may result in the alteration of apoptosis through activation of caspase-3. Alongside we compared the responses of fetal lung cells with A549 cells and rat periodontal ligament (PDL) fibroblasts exposed to CSE in a dose dependent manner. Caspase-3 activity and inhibition was measured using a fluorometric assay. Cell viability in smoke exposed cells was measured using MTT forma

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13

Schmidt, Joachim [Verfasser]. "A novel binding protein for fibroblast growth factors (FGF-BP2) : cloning, expression profile, tumorigenic activity and regulation of gene expression by fetal bovine serum and retinoic acid / vorgelegt von Joachim Schmidt." 2003. http://d-nb.info/972868488/34.

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14

Verdánová, Martina. "Vliv uhlíkových nanostruktur na chování lidských buněk a význam fetálního bovinního séra během buněčné adheze." Doctoral thesis, 2016. http://www.nusl.cz/ntk/nusl-353470.

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Graphene (G) and nanocrystalline diamond (NCD) are carbon allotropes and promising nanomaterials with an excellent combination of their properties, such as high mechanical strength, electrical and thermal conductivity, possibility of functionalization and very high surface area to volume ratio. For these reasons, G and NCD are employed next to electronics in biomedical applications, including implant coating, drug and gene delivery and biosensing. For a fundamental characterization of cell behavior on G and NCD, we studied osteoblast adhesion and proliferation on differently treated G and NCD.

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15

Salame, Joumana. "Rat periodontal ligament fibroblasts and fetal rat pulmonary cells altered gelatinase expression upon exposure to cigarette smoke extract and lipolysaccharide." 2008. http://hdl.handle.net/1993/21247.

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