Relevant bibliographies by topics / Fetal intrauterine infection

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Fetal intrauterine infection'

Author: Grafiati
Published: 28 May 2022
Last updated: 29 May 2022

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Journal articles on the topic "Fetal intrauterine infection"

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Silingardi, Enrico, Anna Laura Santunione, Francesco Rivasi, Bernard Gasser, Silvia Zago, and Lorella Garagnani. "Unexpected Intrauterine Fetal Death in Parvovirus B19 Fetal Infection." American Journal of Forensic Medicine and Pathology 30, no. 4 (December 2009): 394–97. http://dx.doi.org/10.1097/paf.0b013e3181c17b2e.

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Jawor, Paulina, John F. Mee, and Tadeusz Stefaniak. "Role of Infection and Immunity in Bovine Perinatal Mortality: Part 2. Fetomaternal Response to Infection and Novel Diagnostic Perspectives." Animals 11, no. 7 (July 15, 2021): 2102. http://dx.doi.org/10.3390/ani11072102.

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Bovine perinatal mortality due to infection may result either from the direct effects of intrauterine infection and/or the fetal response to such infection, leading to the fetal inflammatory response syndrome (FIRS). Both intrauterine infection and FIRS, which causes multi-organ damage and involution of immune organs, compromise fetal survivability, sometimes fatally. Organ injury associated with FIRS may, in addition to causing fetal mortality, irreversibly compromise extrauterine adaptation of the neonate, a recognized problem in human fetuses. Diagnosis of intrauterine infection and of FIRS requires related, but independent analytical approaches. In addition to detection of pathogens, the immune and inflammatory responses of the bovine fetus may be utilized to diagnose intrauterine infection. This can be done by detection of specific changes in internal organs and the measurement of antibodies and/or elements of the acute phase reaction. Currently our ability to diagnose FIRS in bovine fetuses and neonates is limited to research studies. This review focuses on both the fetomaternal response to infection and diagnostic methods which rely on the response of the fetus to infection and inflammatory changes, as well other methods which may improve diagnosis of intrauterine infection in cases of bovine perinatal mortality.
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Watson, William J., Sami Awadallah, and Mary Jo Jaqua. "Intrauterine Infection With Coxsackievirus: Is it a Cause of Congenital Cardiac Malformations?" Infectious Diseases in Obstetrics and Gynecology 3, no. 2 (1995): 79–81. http://dx.doi.org/10.1155/s1064744995000366.

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Background: Although maternal infections with coxsackievirus during pregnancy are relatively common, fetal infections are quite rare. Coxsackievirus infection in utero has been associated with myocarditis, but has not been proven a teratogen.Case: A patient whose fetus had structural cardiac anomalies and hydrops was found to have an intrauterine infection with Coxsackie B-1 virus, proven by virus isolation from the amniotic fluid. This infection led to increasing intrauterine hydrops and subsequent neonatal death. Conclusion: This interesting association of intrauterine infection with Coxsackie B virus and structural cardiac anomalies in the fetus warrants further investigation.
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Válková, Andrea, and Petr Hubka. "Covid-19 as a possible risk factor of intrauterine fetal death." Česká gynekologie 86, no. 6 (December 21, 2021): 410–13. http://dx.doi.org/10.48095/cccg2021410.

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Objective: To summarize information about possible effects of covid-19 on intrauterine fetal death and present three cases of intrauterine fetal death in women with recent covid-19 infection. Methods: Review of available information about pregnancy with covid-19 and comparison with own observation of cases during spring 2021. Conclusion: Covid-19 influences risk of intrauterine fetal death, preeclampsia/eclampsia or HELLP syndrome. Coagulation changes and drop of platelets is considered as one of the causes of intrauterine fetaldeath due to fetal vascular malperfusion. Key words: covid-19 – intrauterine fetal death – obstetrics
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Schwartz, David A., and Amareen Dhaliwal. "Infections in Pregnancy With COVID-19 and Other Respiratory RNA Virus Diseases Are Rarely, If Ever, Transmitted to the Fetus: Experiences With Coronaviruses, Parainfluenza, Metapneumovirus Respiratory Syncytial Virus, and Influenza." Archives of Pathology & Laboratory Medicine 144, no. 8 (April 27, 2020): 920–28. http://dx.doi.org/10.5858/arpa.2020-0211-sa.

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Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the agent of coronavirus disease 2019 (COVID-19), is similar to 2 other coronaviruses, SARS-CoV and Middle East respiratory syndrome coronavirus (MERS-CoV), in causing life-threatening maternal respiratory infections and systemic complications. Because of global concern for potential intrauterine transmission of SARS-CoV-2 from pregnant women to their infants, this report analyzes the effects on pregnancy of infections caused by SARS-CoV-2 and other respiratory RNA viruses, and examines the frequency of maternal-fetal transmission with SARS-CoV-2, severe acute respiratory syndrome (SARS), Middle East respiratory syndrome (MERS), influenza, respiratory syncytial virus (RSV), parainfluenza (HPIV), and metapneumovirus (hMPV). There have been no confirmed cases of intrauterine transmission reported with SARS-CoV-2 or any other coronaviruses—SARS and MERS. Influenza virus, despite causing approximately 1 billion annual infections globally, has only a few cases of confirmed or suspected intrauterine fetal infections reported. Respiratory syncytial virus is an unusual cause of illness among pregnant women, and with the exception of 1 premature infant with congenital pneumonia, no other cases of maternal-fetal infection are described. Parainfluenza virus and hMPV can produce symptomatic maternal infections but do not cause intrauterine fetal infection. In summary, it appears that the absence thus far of maternal-fetal transmission of the SARS-CoV-2 virus during the COVID-19 pandemic is similar to other coronaviruses, and is also consistent with the extreme rarity of suggested or confirmed cases of intrauterine transmission of other respiratory RNA viruses. This observation has important consequences for pregnant women because it appears that if intrauterine transmission of SARS-CoV-2 does eventually occur, it will be a rare event. Potential mechanisms of fetal protection from maternal viral infections are also discussed.
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BRADY, WILLIAM KIM, and ALFRED PURDON. "Intrauterine Fetal Demise Associated With Enterovirus Infection." Southern Medical Journal 79, no. 6 (June 1986): 770–72. http://dx.doi.org/10.1097/00007611-198606000-00032.

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Schild, R. L., R. Bald, H. Plath, A. M. Eis-Hübinger, G. Enders, and M. Hansmann. "Intrauterine management of fetal parvovirus B19 infection." Ultrasound in Obstetrics and Gynecology 13, no. 3 (March 1999): 161–66. http://dx.doi.org/10.1046/j.1469-0705.1999.13030161.x.

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Hichijo, Atsuko, and Mikio Morine. "A Case of Fetal Parvovirus B19 Myocarditis That Caused Terminal Heart Failure." Case Reports in Obstetrics and Gynecology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/463571.

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Parvovirus B19 is a well-established cause of fetal anemia and nonimmune fetal hydrops in pregnancy. Fetal parvovirus infection can cause severe destruction of erythroid progenitor cells, resulting in fetal anemia, hydrops, and intrauterine death. However, viral myocarditis with subsequent heart failure is another possible mechanism for hydrops formation as viral infection of fetal myocardial cells has been reported in postmortem examinations. We herein report a case of fetal cardiomegaly and massive pericardial effusion secondary to myocarditis as a result of parvovirus B19 infection. The case developed hydrops as consequence of severe anemia and experienced terminal heart failure, which led to the fetus dying an intrauterine death at 22 weeks of gestation. This case demonstrates that there may be an association between myocarditis caused by intrauterine parvovirus B19 infection and a poor outcome. The presence of viral myocarditis may be the determining prognostic factor in that situation.
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Agra, Isabela, Antonio Amorim Filho, Lawrence Lin, Sckarlet Biancolin, Rossana Francisco, and Maria Brizot. "Parameters Associated with Adverse Fetal Outcomes in Parvovirus B19 Congenital Infection." Revista Brasileira de Ginecologia e Obstetrícia / RBGO Gynecology and Obstetrics 39, no. 11 (September 25, 2017): 596–601. http://dx.doi.org/10.1055/s-0037-1606859.

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Objective To investigate the clinical and sonographic parameters associated with adverse fetal outcomes in patients with congenital parvovirus B19 infection managed by intrauterine transfusion. Methods This was a single-center retrospective study conducted from January 2005 to December 2016 that assessed patients with singleton pregnancies with fetal parvovirus infection confirmed by a polymerase chain reaction of the amniotic fluid or fetal blood samples who underwent at least one intrauterine transfusion. The maternal characteristics, sonographic findings and parameters related to intrauterine transfusion were compared between the two groups (recovery/non-recovery), who were categorized based on fetal response after in-utero transfusions. Progression to fetal death or delivery without fetal recovery after the transfusions was considered non-recovery and categorized as an adverse outcome. Results The final analysis included ten singleton pregnancies: seven of which were categorized into the recovery group and three of which into the non-recovery group. The baseline characteristics were similar between the groups. All fetuses were hydropic at the time of diagnosis. No significant differences related to sonographic or intrauterine transfusion parameters were identified between the groups; however, the non-recovery group tended to have an increased number of sonographic markers and lower fetal hemoglobin and platelet levels before the transfusion. Conclusion We were unable to firmly establish the clinical or sonographic parameters associated with adverse fetal outcomes in patients with parvovirus infection managed with intrauterine transfusions; however, edema, placental thickening and oligohydramnios may indicate greater fetal compromise and, subsequently, adverse outcomes. However, further studies are necessary, mainly due to the small number of cases analyzed in the present study.
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Torabi, Rozbeh, Svetlana Charnova, Rosanna G. Abellar, Halit Pinar, and Monique E. De Paepe. "Intrauterine Infection with Klebsiella Pneumoniae: Report of a Case and Literature Review." Pediatric and Developmental Pathology 11, no. 2 (March 2008): 152–55. http://dx.doi.org/10.2350/07-09-0337.1.

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We report a case of intrauterine Klebsiella pneumoniae infection that resulted in premature rupture of membranes and fetal demise at 20 weeks' gestation in a pregnancy achieved by in vitro fertilization. Postmortem findings included massive panlobar pneumonia, the presence of abundant gram-negative, rod-shaped bacteria within the pulmonary air spaces and the lumen of the gastrointestinal tract, and fetal lung and blood cultures positive for Klebsiella pneumoniae. The placenta showed severe acute chorioamnionitis associated with a brisk fetal inflammatory response (umbilical cord and chorionic plate vasculitis). Marked pancreatic fibrosis was noted, indicative of a preceding necrotizing pancreatitis. In spite of this fulminant histopathologic evidence of intrauterine infection, the infection was clinically silent. This represents, to our knowledge, the 1st reported case of fatal intrauterine Klebsiella pneumoniae infection fully supported by conclusive fetal and placental histopathological evidence.
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Dissertations / Theses on the topic "Fetal intrauterine infection"

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Patrick, Lindsay Alexandra Laurentia. "Investigation of the effect of intrauterine inflammation and infection on fetal brain injury using human and animal models." Thesis, Kingston, Ont. : [s.n.], 2008. http://hdl.handle.net/1974/1055.

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Коваленко, Ю. О., Світлана Анатоліївна Сміян, Светлана Анатольевна Смиян, and Svitlana Anatoliivna Smiian. "Значення змін мінерального обміну при внутрішньоутробному інфікуванні плоду." Thesis, Видавництво СумДУ, 2009. http://essuir.sumdu.edu.ua/handle/123456789/4616.

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Books on the topic "Fetal intrauterine infection"

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Purandare, Amol, and Barbara A. Jantausch. Parvovirus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0012.

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Parvovirus B19 is a common infection in humans that occurs worldwide. Parvovirus B19 is transmitted through exposure to respiratory droplets, blood, and blood products, and through mother-to-child transmission (MTCT) in utero. Intrauterine parvovirus B19 infection is a rare occurrence during pregnancy but can result in significant morbidity and mortality for the fetus, including severe fetal anemia and nonimmune fetal hydrops (NIFH). Intrauterine transfusion can be successful in treating fetal anemia. Neurodevelopmental impairment has been reported in infants with congenital infection who have received intrauterine transfusion (IUT). Future research on the development of antiviral agents for the treatment of parvovirus B19 infection in pregnant women is needed, along with the development of a parvovirus B19 vaccine. Longitudinal studies to evaluate neurodevelopmental outcome of infants with a history of congenital parvovirus B19 infection are needed in order to facilitate the optimal evaluation and management of these infants.
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Olson-Chen, Courtney. Neurologic Infections in Pregnancy. Edited by Emma Ciafaloni, Cheryl Bushnell, and Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0011.

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Despite advances in prevention, diagnosis, and treatment, infectious diseases continue to be a major cause of maternal, fetal, and neonatal morbidity and mortality. Immunologic changes in pregnancy can increase both susceptibility to certain infections and the severity of infection. Infectious diseases in pregnancy contribute to the development of congenital fetal syndromes in addition to adverse outcomes including preterm birth, stillbirth, and intrauterine growth restriction. While infections of the maternal central nervous system, or CNS, are rare during pregnancy, the potential impact can be critical.1 This chapter will cover both the types of infections within the CNS and the potential organisms that cause these infections. The chapter will also provide general management recommendations for pregnancy in order to both prevent and maintain awareness about CNS infections.
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McLeod, Rima, Kelsey Wheeler, Pauline Levigne, and Kenneth Boyer. Toxoplasma gondii. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0017.

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Mother-to-child transmission (MTCT) of the parasite Toxoplasma gondii can result in congenital toxoplasmosis. Untreated congenital toxoplasmosis presents considerable potential risks to patients and costs for society, with manifestations recurring throughout life. Infection with T. gondii, acquired at any time during pregnancy can damage the fetus, but especially during early gestation. Fetal infection with T. gondii can cause fetal loss, intrauterine growth retardation, and damage to organs (especially the brain and eyes). Treatment with pyrimethamine and sulfadiazine improves manifestations of active infection in the fetus, congenital infection in infants, and recurrent disease when manifested later in life in those congenitally infected. Key components of the prevention and treatment of congenital toxoplasmosis include prompt, correct diagnosis and treatment with effective anti–T. gondii medications. Several countries have gestational screening programs to detect newly acquired T. gondii infections. In the future, development of new medications, including those for chronic infection, and vaccines for prevention will be important.
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Belcher, Harolyn M. E., and Samantha Hutchison. Neurobiology of Intrauterine Opiate and Cocaine Exposure. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0182.

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Drug abuse results from a complex interplay among the drug, the individual and the socio-cultural environment. Current pain reliever, heroin, and OxyContin® use is reported in 0.9% (n = 19,000), 0.2% (n = 4,000), and 0.1% (n = 3,000), respectively, of pregnant women in the United States each year. Cocaine use was reported in 0.2% of pregnant women. Intrauterine drug exposure to opiates is associated with risk for narcotic abstinence syndrome, prenatal infections, and sudden infant death. Illicit drug use during pregnancy also places the mother-to-be at risk for reduced prenatal care, obstetric complications, including abruption placentae, prolonged rupture of membranes, and fetal distress. This chapter focuses on the neurobiological effects of intrauterine opiate and cocaine exposure on the developing fetus.
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Book chapters on the topic "Fetal intrauterine infection"

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"Intrauterine Infection." In Embryo and Fetal Pathology, 601–21. Cambridge University Press, 2001. http://dx.doi.org/10.1017/cbo9780511547423.024.

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"Scalp Infection Following Intrauterine Fetal Monitoring." In Pediatric Anaerobic Infections, 149–54. CRC Press, 2001. http://dx.doi.org/10.3109/9780203904022-17.

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Singh, Sarman. "Laboratory Diagnosis of Congenital Fetal/Neonatal TORCH Infection." In Congenital Intrauterine TORCH Infections, 182. Jaypee Brothers Medical Publishers (P) Ltd., 2004. http://dx.doi.org/10.5005/jp/books/10172_13.

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Romero, Roberto, Tinnakorn Chaiworapongsa, and Maria-Teresa Gervasi. "Fetal and Maternal Responses to Intrauterine Infection." In Fetal and Neonatal Physiology, 131–42. Elsevier, 2004. http://dx.doi.org/10.1016/b978-0-7216-9654-6.50018-7.

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Romero, Roberto, Francesca Gotsch, Offer Erez, Edi Vaisbuch, and Juan Pedro Kusanovic. "Fetal and Maternal Responses to Intrauterine Infection." In Fetal and Neonatal Physiology, 155–72. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4160-3479-7.10016-3.

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Geva, Ronny. "Intrauterine Growth Restriction." In Cognitive and Behavioral Abnormalities of Pediatric Diseases. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195342680.003.0049.

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Recent data shows that 30 million low-birth-weight (LBW) infants are born annually worldwide (23.8% of all births). Although the global prevalence of such births is gradually decreasing, rates are still as high as 30% in many developing countries (World Health Organization 2008). Low birth weight is due to intrauterine growth restriction (IUGR), rather than or in addition to prematurity, in approximately one-third of these cases. This staggering number of affected children underscores the importance of understanding the short- and long-term cognitive and behavioral complications of IUGR. Intrauterine growth restriction conveys short- and long-term neurodevelopmental risks and thus requires costly long-term investment of medical, cognitive emotional, educational, and economical resources. Nevertheless, if treated aggressively, IUGR more often than not bears a fairly optimistic outlook, once the infant overcomes the initial life-threatening issues (Geva et al. 2006a). Intrauterine growth restriction is frequently detected in a pregnancy with a less-than-expected third trimester weight gain (100–200 g [3.5–7 oz] per week) or as an incidental finding on ultrasound examination when fetal measurements are less than expected for gestational age (GA; Geva et al. 2005). An estimated fetal weight under the 10th percentile, as determined by serial ultrasound examination, strongly correlates with growth restriction (Bernstein and Gabbe 1996; McCormick 1985). The etiologies of IUGR are typically thought of according to three interdependent categories: fetal factors, placental factors, and maternal factors (Kay 2008). Fetal factors include chromosomal events, such as trisomy 18 and 13 and sex chromosome abnormalities, which account for 5%–15% of all IUGR cases. Further exploration of genetic factors is currently under way, with mixed results (Kotzot et al. 2001). Other fetal factors linked to IUGR include congenital anomalies, mostly cardiovascular malformations, gastroschisis and omphalocele; infection, often related to rubella, cytomegalovirus, and toxoplasmosis (see Chapter 25); and multiple gestations, in which uteroplacental blood flow variations and/or twin–twin transfusion develops (Miller et al. 2008). Fetal villus circulation abnormalities are placental factors related to IUGR (Roberts and Post 2008).
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Clark, Robin D., and Cynthia J. Curry. "Intrauterine Growth Restriction." In Genetic Consultations in the Newborn, edited by Robin D. Clark and Cynthia J. Curry, 11–16. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780199990993.003.0002.

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This chapter reviews isolated and syndromic intrauterine growth restriction (IUGR) or small for gestational age infants. The differential diagnosis of intrauterine growth restriction includes placental, maternal, and fetal causes. Maternal causes of IUGR include exposure to teratogens, various maternal illnesses, and multiple gestation. Infant causes include congenital infection, chromosomal aneuploidy, and multiple syndromes including primordial dwarfism. Other causes include genomic imprinting errors (Russell Silver syndrome and IMAGe syndrome) and endocrine and metabolic causes, the lipodystrophies, and skeletal dysplasias including SHOX deficiency. The evaluation of IUGR usually includes a SNP microarray and often targeted or gene panel testing. A clinical case presentation features an infant with Majewski (microcephalic) osteodysplastic primordial dwarfism (MOPD II) .
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Gotsch, Francesca, Roberto Romero, and Juan Pedro Kusanovic. "Intrauterine Infection, Preterm Parturition, and the Fetal Inflammatory Response Syndrome." In High Risk Pregnancy, 457–68. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4160-5908-0.00026-0.

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Baldwin, Andrew, Nina Hjelde, Charlotte Goumalatsou, and Gil Myers. "Obstetrics." In Oxford Handbook of Clinical Specialties, 1–97. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198719021.003.0001.

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This chapter explores obstetrics, including obstetric histories, abdominal examination, physiological changes in pregnancy, pre-pregnancy counselling, the placenta, plasma chemistry in pregnancy, antenatal care, structural abnormalities and ultrasound, screening and diagnosis of aneuploidy, minor symptoms of pregnancy, hyperemesis gravidarum, sickle cell disease in pregnancy, cardiac disease in pregnancy, drugs used in psychiatry and epilepsy, anaemia, HIV in pregnancy and labour, diabetes mellitus in pregnancy, thyroid disease in pregnancy, jaundice in pregnancy, malaria, renal disease in pregnancy, epilepsy, respiratory disease in pregnancy, connective tissue diseases in pregnancy, hypertension in pregnancy, thromboprophylaxis, thrombophilia in pregnancy, venous thromboembolism, infection, group B streptococcus (GBS), abdominal pain in pregnancy, sepsis in pregnancy and the puerperium, fetal monitoring in labour, pre-eclampsia, prematurity, small for gestational age (SGA), postmaturity (prolonged pregnancy), maternal collapse, antepartum haemorrhage, prelabour rupture of membranes at term, normal labour, induction of labour, management of delay in labour, home birth, pain relief in labour, multiple pregnancy, breech presentation and other malpresentations/malpositions, cord prolapse, shoulder dystocia, meconium-stained liquor, operative vaginal delivery, caesarean section (CS), uterine rupture, mendelson’s syndrome, stillbirth (intrauterine fetal death, IUD), postpartum haemorrhage (PPH), retained placenta, uterine inversion, placenta praevia, accreta and increta, DIC and coagulation defects, amniotic fluid embolism, birth injuries, episiotomy and tears, the puerperium, maternal and perinatal mortality.
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Gupta, Sangeeta, and Amar Bhide. "Parvovirus B19—Fetal Implications and Therapy." In Congenital Intrauterine Infections, 128. Jaypee Brothers Medical Publishers (P) Ltd., 2009. http://dx.doi.org/10.5005/jp/books/10171_12.

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Conference papers on the topic "Fetal intrauterine infection"

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Oliveira, Sayd Douglas Rolim Carneiro, Carlos Jorge Maciel Uchoa Gadelha, Dara da Silva Mesquita, and Tereza Cristina Ribeiro Brito. "SARS-CoV-2 infection during pregnancy and risk of neurodevelopmental disorders in neonatals: a literature review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.031.

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Introduction: With the emergence of SARS-CoV-2 and its rapid spread, a concern with the pregnant women have increased, since viruses have a wide range of obstetric and neonatal issues. Recent findings indicate that the gestational period and the postpartum period make mothers and their offspring more susceptible to COVID-19 and the rapid progression to the critical stage of the disease. Objectives: To carry out a bibliographic study on SARS-CoV- 2 during pregnancy and the potential risk of neurodevelopmental disorders in neonates. Methods: A review, developed from articles selected on the following bases: PubMed, Web of Science and Scopus. In the search, articles indexed until March 2021 and published in English, using the descriptors: “COVID-19”; “Pregnancy”; “Offspring”; “Neonatal”; “Neurodevelopment”; “Anomalies” and “Complications”. Exclusion criteria: duplicates and articles outside the scope of the study. Results: The initial search resulted in 533 articles, 498 from PubMed, 2 from Web of Science and 33 from Scopus. After reading the title and abstract, the application of the inclusion and exclusion criteria, the sample of 48 documents were included. In the studies, 89.0% of all patients had cesarean delivery (n = 201), 33.3% had gestational complications, 35.3% had premature delivery and about 2.5% were stillborn or had neonatal death. Among those tested, 6.45% of neonates diagnosed positive for COVID-19. In another study, the newborn showed neurological issues similar to the adult patients and transient neurological complications due to cerebral vasculitis. Conclusions: The results demonstrate that further investigations are needed to determine the potential for vertical intrauterine transmission in pregnant women with COVID-19 and possibles fetal and neonatal consequences.
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Balestra, Amanda Fernandes de Sousa Oliveira, Flávia Pascoal Teles, and Karine Felipe Martins. "Fetal surgery in the context of myelomeningocele: repercussions and prognosis." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.055.

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Background: Myelomeningocele (MMC) is a congenital malformation of neural tube closure. The clinical picture comprises sensory and motor deficits at the point of spinal cord injury and below, in addition to ventriculomegaly, which requires ventriculo-peritoneal drains (DVP). Exposure of nervous tissue to amniotic fluid and trauma to the uterine wall, generates secondary damage. Intrauterine correction is the gold standard for MMC and aims to reduce organic and functional sequelae, improving the patient’s neurological prognosis. Objectives: The objective of this work is to identify the impact of fetal surgery against MMC. Methods: An integrative literature review was carried out based on articles selected from the Google Scholar and Scientific Eletronic Library Online databases. Results: The benefits of intrauterine neurosurgery outweigh the harm, based on maternal complications. Such maternal risks are: oligohydramnios, spontaneous rupture of the membrane, uterine dehiscence, premature birth, infections, blood transfusion, acute lung edema and contraindication for vaginal delivery due to uterine scarring. For the child, all the studies analyzed showed the same gains, extremely significant when compared to postnatal surgery: better cognitive development, greater probability of walking without using orthoses, less need for DVP. The gains from the fetal surgery technique go beyond the postnatal intervention. Conclusions: Therefore, the importance of early intrauterine treatment, in a properly equipped place and by qualified professionals, is reiterated, offering comprehensive care to pregnant women, preventing potential impasses and aiming at a better prognosis and quality of life for the child.
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