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Journal articles on the topic 'Genetic influences'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

RUTTER, MICHAEL. "Pathways of genetic influences on psychopathology." European Review 12, no. 1 (2004): 19–33. http://dx.doi.org/10.1017/s1062798704000031.

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Quantitative genetics, using data from twin and adoptee studies, has shown substantial genetic influences on all forms of psychiatric disorder; however, with just a few exceptions, the evidence indicates that the disorders are multifactorial, with influences that are both genetic and environmental. In recent years, molecular genetics has begun to identify individual susceptibility genes; examples are given for schizophrenia, attention deficit/hyperactivity disorder, and Alzheimer's disease. Both quantitative and molecular genetics have shown the importance of gene-environment interplay with re

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Lau, Jennifer Y. F., and Thalia C. Eley. "Changes in genetic and environmental influences on depressive symptoms across adolescence and young adulthood." British Journal of Psychiatry 189, no. 5 (2006): 422–27. http://dx.doi.org/10.1192/bjp.bp.105.018721.

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BackgroundDepression rises markedly in adolescence, a time when increased and new genetic influences have been reported.AimsTo examine ‘new’ and ‘stable’ genetic and environmental factors on depressive symptoms in adolescence and young adulthood.MethodA questionnaire survey investigated a sample of twin and sibling pairs at three time points over an approximately 3-year period. Over 1800 twin and sibling pairs reported depressive symptoms at the three time points. Data were analysed using multivariate genetic models.ResultsDepressive symptoms at all time points were moderately heritable with s

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RODGERS, JOSEPH LEE, DAVID C. ROWE, and MAURY BUSTER. "NATURE, NURTURE AND FIRST SEXUAL INTERCOURSE IN THE USA: FITTING BEHAVIOURAL GENETIC MODELS TO NLSY KINSHIP DATA." Journal of Biosocial Science 31, no. 1 (1999): 29–41. http://dx.doi.org/10.1017/s0021932099000292.

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Fisher (1930) presented both theoretical and empirical results concerning genetic influences on fertility. Since then, only sparse research has been done on the genetics of fertility, although more sophisticated methodogy and data now exist than were available to Fisher. This paper presents a behavioural genetic analysis of age at first intercourse, accounting for genetic, shared environmental, and selected non-shared environmental influences. The data came from the nationally representative National Longitudinal Survey of Youth (NLSY). A newly developed kinship linking procedure was used that

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Matison, Annabel, Anbupalam Thalamuthu, Simone Reppermund, et al. "Nature Versus Nurture – Studying the Relationships Between Diet and Depression in Older Adults." Current Developments in Nutrition 6, Supplement_1 (2022): 1118. http://dx.doi.org/10.1093/cdn/nzac078.012.

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Abstract Objectives Prior work suggests that higher fruit and vegetable consumption may protect against depression in older adults. By better understanding the influence of genetic and environmental factors on fruit and vegetable intakes, more effective interventions could be designed to increase intakes in older adults to reduce and/or assist with the treatment of depression in older adults. The aim of this heritability study is to estimate the genetic and environmental influences on the consumption of fruit and vegetables in older adults and to investigate if there are shared influences betw

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Savage, Jeanne E., Elizabeth C. Long, Sally I.-Chun Kuo, et al. "Alcohol Misuse Across the Life Span: Insights From Developmental Studies in Behavior Genetics." Policy Insights from the Behavioral and Brain Sciences 4, no. 2 (2017): 186–93. http://dx.doi.org/10.1177/2372732217720222.

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Alcohol misuse, one of today’s greatest public health challenges, is a developmentally dynamic, complex behavior at the intersection of genetic and environmental influences. This review examines such influences from a behavior genetics perspective and discusses implications for public policy. Alcohol misuse is moderately heritable with genetic influences accounting for around 50% of its variance, but to date, few specific genes have been identified. However, numerous environmental and social factors moderate genetic risk, including parents, peers, romantic partners, family dynamics, employment

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Schachar, Russell J., Nadine Forget-Dubois, Ginette Dionne, Michel Boivin, and Philippe Robaey. "Heritability of Response Inhibition in Children." Journal of the International Neuropsychological Society 17, no. 2 (2010): 238–47. http://dx.doi.org/10.1017/s1355617710001463.

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AbstractWe report the heritability of response inhibition, latency, and variability, which are potential markers of genetic risk in neuropsychiatric conditions. Genetic and environmental influences on cancellation and restraint, response latency, and variability measured in a novel variant of the stop signal task were studied in 139 eight-year-old twin pairs from a birth cohort. Cancellation (50%), restraint (27%), and response latency (41%) showed significant heritability, the balance being non-shared environmental influences and/or error. Response variability was not heritable, with 23% of t

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Millichap, J. Gordon. "Genetic Influences in ADHD." Pediatric Neurology Briefs 11, no. 7 (1997): 49. http://dx.doi.org/10.15844/pedneurbriefs-11-7-1.

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Bolton, Patrick, and Michael Rutter. "Genetic Influences in Autism." International Review of Psychiatry 2, no. 1 (1990): 67–80. http://dx.doi.org/10.3109/09540269009028273.

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Gaston, Hill. "Genetic Influences in Rheumatology." Medicine 30, no. 8 (2002): 25–26. http://dx.doi.org/10.1383/medc.30.8.25.28222.

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10

Smalley, Susan L. "Genetic Influences in Autism." Psychiatric Clinics of North America 14, no. 1 (1991): 125–39. http://dx.doi.org/10.1016/s0193-953x(18)30329-0.

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Rutter, M. "Genetic influences on autism." European Psychiatry 17 (May 2002): 30. http://dx.doi.org/10.1016/s0924-9338(02)80134-1.

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12

Johnson, T. "Genetic influences on aging." Experimental Gerontology 32, no. 1-2 (1997): 11–22. http://dx.doi.org/10.1016/s0531-5565(96)00079-4.

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Spector, T. "Genetic Influences on Osteoarthritis." ACOG Clinical Review 1, no. 5 (1996): 12. http://dx.doi.org/10.1016/1085-6862(96)85141-7.

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14

Faraone, Stephen. "Genetic Influences on ADHD." Child and Adolescent Psychopharmacology News 6, no. 5 (2001): 7–11. http://dx.doi.org/10.1521/capn.6.5.7.22464.

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Ishihara, Mami, and Shigeaki Ohno. "Genetic influences on sarcoidosis." Eye 11, no. 2 (1997): 155–61. http://dx.doi.org/10.1038/eye.1997.44.

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Koudsi, Nael Al, and Rachel F. Tyndale. "Genetic Influences on Smoking." Therapeutic Drug Monitoring 27, no. 6 (2005): 704–9. http://dx.doi.org/10.1097/01.ftd.0000179842.63515.c6.

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Bamshad, Mike. "Genetic Influences on Health." JAMA 294, no. 8 (2005): 937. http://dx.doi.org/10.1001/jama.294.8.937.

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18

Rutter, Michael. "Nature, nurture, and psychopathology: A new look at an old topic." Development and Psychopathology 3, no. 2 (1991): 125–36. http://dx.doi.org/10.1017/s0954579400000031.

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AbstractThe important implications for developmental psychopathology that derive from advances in psychiatric and behavioral genetics are discussed in relation to a series of mistaken stereotypes: that strong effects mean that environmental influences must be unimportant; that genes provide a limit to potential; that genetic strategies are of no value for studying environmental influences; that nature and nurture are separate; that genes for serious diseases are necessarily bad; that diseases have nothing to do with normal variation; that genetic findings will not help identify diseases; that

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Rutter, Michael. "Nature, nurture, and psychopathology: A new look at an old topic." Development and Psychopathology 3, no. 2 (1991): 125–36. http://dx.doi.org/10.1017/s0954579400005198.

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AbstractThe important implications for developmental psychopathology that derive from advances in psychiatric and behavioral genetics are discussed in relation to a series of mistaken stereotypes: that strong effects mean that environmental influences must be unimportant; that genes provide a limit to potential; that genetic strategies are of no value for studying environmental influences; that nature and nurture are separate; that genes for serious diseases are necessarily bad; that diseases have nothing to do with normal variation; that genetic findings will not help identify diseases; that

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Nes, Ragnhild B., Espen Røysamb, Jennifer R. Harris, Nikolai Czajkowski, and Kristian Tambs. "Mates and Marriage Matter: Genetic and Environmental Influences on Subjective Wellbeing Across Marital Status." Twin Research and Human Genetics 13, no. 4 (2010): 312–21. http://dx.doi.org/10.1375/twin.13.4.312.

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AbstractSpecific environments and social relationships may alter the impact of genes. Previous studies have shown marriage to moderate heritability for depressive symptoms in females, suggesting that marriage provides protection or compensation against genetic risks. Similar mechanisms may be relevant for subjective wellbeing (SWB), which is considerably influenced by genes and almost universally associated with marital status. Questionnaire data on SWB from a population-based sample of 1250 monozygotic (MZ) and 981 dizygotic (DZ) male and female twin pairs (n= 4462) were analyzed using struct

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Chandra Sekar, Praveen Kumar, and Ramakrishnan Veerabathiran. "A Genetic Perspective on Ischemic Stroke: Recent Advances and Future Directions." Annals of Clinical and Experimental Neurology 18, no. 4 (2025): 55–67. https://doi.org/10.17816/acen.1064.

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Objective. This narrative review aimed to explore the multifaceted nature of ischemic stroke (IS) and its underlying genetic factors, emphasize the role of genetics in early detection and prevention, and acknowledge the complex influences on stroke prevalence across various countries. Methods. An extensive overview of the causes, mechanisms, and genetics of IS was conducted by reviewing several studies and recent findings. The role of specific genes in monogenic stroke disorders, implications of polygenic influences, recent advances in genetic evaluation, and methods for early IS detection wer

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ELEY, THALIA C., PAUL LICHTENSTEIN, and TERRIE E. MOFFITT. "A longitudinal behavioral genetic analysis of the etiology of aggressive and nonaggressive antisocial behavior." Development and Psychopathology 15, no. 2 (2003): 383–402. http://dx.doi.org/10.1017/s095457940300021x.

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Developmental studies of antisocial behavior (ASB) have found two subgroups of behaviors, roughly described as aggressive and nonaggressive ASB. Theoretical accounts predict that aggressive ASB, which shows greater stability, should have high heritability. In contrast, nonaggressive ASB is very common in adolescence, shows less continuity, and should be influenced both by genes and shared environment. This study explored the genetic and environmental influences on aggressive and nonaggressive ASB in over 1,000 twin pairs aged 8–9 years and again at 13–14 years. Threshold models were fit to the

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Mosing, Miriam A., Karin J. H. Verweij, David Z. Hambrick, Nancy L. Pedersen, and Fredrik Ullén. "Testing the Deliberate Practice Theory: Does Practice Reduce the Heritability of Musical Expertise?" Journal of Intelligence 12, no. 9 (2024): 87. http://dx.doi.org/10.3390/jintelligence12090087.

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The deliberate practice (DP) theory claims that expertise essentially reflects the accumulated amount of deliberate practice, and that with sufficient practice, genetic influences on expertise will be bypassed. Thus, a core prediction of the DP theory is that genetic effects on performance decrease as a function of practice. Here, we test this prediction using music as a model domain. Musical expertise (measured with a musical auditory discrimination test) and lifetime practice hours were determined in 6471 twins including 1302 complete twin pairs. We fitted a bivariate Cholesky decomposition

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Plomin, Robert, and Essi Colledge. "Genetics and Psychology: Beyond Heritability." European Psychologist 6, no. 4 (2001): 229–40. http://dx.doi.org/10.1027//1016-9040.6.4.229.

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The questions whether and how much genetic factors affect psychological dimensions and disorders represent important first steps in understanding the origins of individual differences. Because it is now widely accepted that genetic influences contribute importantly to individual differences throughout psychology, genetic research is moving beyond merely estimating heritability to asking questions about how genetic mechanisms work. We focus on two examples of ways in which genetic research is going beyond heritability. The first is to use genetically sensitive designs to identify specific envir

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Chen, J., J. Yu, J. Zhang, X. Li, and M. McGue. "Investigating genetic and environmental contributions to adolescent externalizing behavior in a collectivistic culture: a multi-informant twin study." Psychological Medicine 45, no. 9 (2015): 1989–97. http://dx.doi.org/10.1017/s0033291714003109.

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BackgroundLittle is known about the etiology of adolescents’ externalizing behavior (Ext) in collectivistic cultures. We aimed to fill this gap by investigating the genetic and environmental influences on Ext in Chinese adolescents. The etiological heterogeneity of aggression (AGG) and rule breaking (RB) was also examined.MethodThe study sample included 908 pairs of same-sex twins aged from 10 to 18 years (mean = 13.53 years, s.d. = 2.26). Adolescents’ Ext were assessed with the Achenbach System of Empirically Based Assessment including Child Behavior Checklist, Teacher Report Form, and Youth

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Grant, J. D., M. T. Lynskey, P. A. F. Madden, et al. "The role of conduct disorder in the relationship between alcohol, nicotine and cannabis use disorders." Psychological Medicine 45, no. 16 (2015): 3505–15. http://dx.doi.org/10.1017/s0033291715001518.

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Background.Genetic influences contribute significantly to co-morbidity between conduct disorder and substance use disorders. Estimating the extent of overlap can assist in the development of phenotypes for genomic analyses.Method.Multivariate quantitative genetic analyses were conducted using data from 9577 individuals, including 3982 complete twin pairs and 1613 individuals whose co-twin was not interviewed (aged 24–37 years) from two Australian twin samples. Analyses examined the genetic correlation between alcohol dependence, nicotine dependence and cannabis abuse/dependence and the extent

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Eilertsen, Espen Moen, Line C. Gjerde, Kenneth S. Kendler, et al. "Development of ADHD symptoms in preschool children: Genetic and environmental contributions." Development and Psychopathology 31, no. 04 (2018): 1299–305. http://dx.doi.org/10.1017/s0954579418000731.

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AbstractWe examined genetic and environmental contributions to the development of symptoms of attention-deficit/hyperactivity disorder (ADHD) in preschool children. ADHD symptoms in siblings at 1.5, 3, and 5 years of age were investigated in a population-based sample from the prospective Norwegian Mother and Child Cohort Study. The longitudinal contributions of additive genetic, shared, twin-specific, and unique environmental influences were estimated using biometric structural equation models. Heritability of ADHD symptoms ranged from 54% to 70%. There was evidence of partially new genetic in

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Narusyte, Jurgita, Annina Ropponen, Kristina Alexanderson, and Pia Svedberg. "Genetic and Environmental Influences on Disability Pension Due To Mental Diagnoses: Limited Importance of Major Depression, Generalized Anxiety, and Chronic Fatigue." Twin Research and Human Genetics 19, no. 1 (2015): 10–16. http://dx.doi.org/10.1017/thg.2015.86.

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Background:Previous research indicates that liability to disability pension (DP) due to mental diagnoses is moderately influenced by genetic factors. This study investigates whether genetic contributions to the liability to DP due to mood and neurotic diagnoses overlap with the genetic influences on major depression (MD), generalized anxiety disorder (GAD), or chronic fatigue (CF).Method:A prospective cohort study including 9,985 female twins born in Sweden 1933–1958. The presence of MD, GAD, and CF was assessed by computer-assisted telephone interviews conducted in 1998–2002. Data on DP due t

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Sambo, Danielle, and David Goldman. "Genetic Influences on Fetal Alcohol Spectrum Disorder." Genes 14, no. 1 (2023): 195. http://dx.doi.org/10.3390/genes14010195.

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Fetal alcohol spectrum disorder (FASD) encompasses the range of deleterious outcomes of prenatal alcohol exposure (PAE) in the affected offspring, including developmental delay, intellectual disability, attention deficits, and conduct disorders. Several factors contribute to the risk for and severity of FASD, including the timing, dose, and duration of PAE and maternal factors such as age and nutrition. Although poorly understood, genetic factors also contribute to the expression of FASD, with studies in both humans and animal models revealing genetic influences on susceptibility. In this arti

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Krebs, G., M. A. Waszczuk, H. M. S. Zavos, D. Bolton, and T. C. Eley. "Genetic and environmental influences on obsessive–compulsive behaviour across development: a longitudinal twin study." Psychological Medicine 45, no. 7 (2014): 1539–49. http://dx.doi.org/10.1017/s0033291714002761.

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BackgroundLittle is known about the factors influencing the stability of obsessive–compulsive behaviour (OCB) from childhood to adolescence. The current study aimed to investigate: (1) the stability of paediatric OCB over a 12-year period; (2) the extent to which genetic and environmental factors influence stability; and (3) the extent to which these influences are stable or dynamic across development.MethodThe sample included 14 743 twins from a population-based study. Parental ratings of severity of OCB were collected at ages 4, 7, 9 and 16 years.ResultsOCB was found to be moderately stable

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Fu, Qiang, Andrew C. Heath, Kathleen K. Bucholz, et al. "Common Genetic Risk of Major Depression and Nicotine Dependence: The Contribution of Antisocial Traits in a United States Veteran Male Twin Cohort." Twin Research and Human Genetics 10, no. 3 (2007): 470–78. http://dx.doi.org/10.1375/twin.10.3.470.

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AbstractMany studies that found associations between depression and nicotine dependence have ignored possible shared genetic influences associated with antisocial traits. The present study examined the contribution of genetic and environmental effects associated with conduct disorder (CD) and antisocial personality disorder (ASPD) to the comorbidity of major depression (MD) and nicotine dependence (ND). A telephone diagnostic interview, the Diagnostic Interview Schedule-III-R, was administered to eligible twins from the Vietnam Era Twin (VET) Registry in 1992. Multivariate genetic models were

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Whitfield, John B., Susan A. Treloar, Gu Zhu, and Nicholas G. Martin. "Genetic and Non-Genetic Factors Affecting Birth-Weight and Adult Body Mass Index." Twin Research 4, no. 5 (2001): 365–70. http://dx.doi.org/10.1375/twin.4.5.365.

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AbstractBirthweight affects neonatal mortality and morbidity and has been used as a marker of foetal undernutrition in studies of prenatal effects on adult characteristics. It is potentially influenced by genetic and environmental influences on the mother, and effects of foetal genotype, which is partially derived from the maternal genotype. Interpretations of variation in birthweight and associated characteristics as being due to prenatal environment ignore other possible modes of materno-foetal transmission. Subjects were adult twins recruited through the Australian Twin Registry, aged 17 to

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Vilor-Tejedor, Natalia, Tavia E. Evans, Hieab H. Adams, et al. "Genetic Influences on Hippocampal Subfields." Neurology Genetics 7, no. 3 (2021): e591. http://dx.doi.org/10.1212/nxg.0000000000000591.

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There is clear evidence that hippocampal subfield volumes have partly distinct genetic determinants associated with specific biological processes. The identification of genetic correlates of hippocampal subfield volumes may help to elucidate the mechanisms of neurologic diseases, as well as aging and neurodegenerative processes. However, despite the emerging interest in this area of research, the current knowledge of the genetic architecture of hippocampal subfields has not yet been consolidated. We aimed to provide a review of the current evidence from genetic studies of hippocampal subfields

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Semkovych, Yaroslav, and Dmytro Dmytriiev. "GENETIC INFLUENCES ON PAIN MECHANISMS." Wiadomości Lekarskie 75, no. 7 (2022): 1776–80. http://dx.doi.org/10.36740/wlek202207130.

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The aim: To review the available results for genetic influences on pain syndrome development. Materials and methods: In the period from 2009 to 2020, a total of 45 research papers describing the key points of genetic influences on pain mechanisms in both adults and children were published in Ukrainian and English and they are now included in the PubMed, EMBASE, Cochrane, and Google Scholar research databases. Conclusions: Pain is a comprehensive characteristic of a person; therefore, it is inevitable that several genes with little individual effect interact with each other and environmental fa

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Gross, W. B., and P. B. Siegel. "Environment-Genetic Influences on Immunocompetence." Journal of Animal Science 66, no. 8 (1988): 2091. http://dx.doi.org/10.2527/jas1988.6682091x.

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Folstein, Susan E., and Joseph Piven. "Etiology of Autism: Genetic Influences." Pediatrics 87, no. 5 (1991): 767–73. http://dx.doi.org/10.1542/peds.87.5.767.

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Infantile autism was first described by Kanner in 1943.1 Based on the observation that symptoms often began shortly after birth and always by two or three years of age, Kanner believed that autism was caused by an unknown, inborn defect. Because this syndrome was so severe and peculiar, it seemed unlikely to be an understandable outcome of a child's life experiences. In the years since Kanner first described this disorder, research findings have supported his initial interpretations. It is now generally accepted that autism has a biologic cause, and considerable research has been carried out w

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Giarratano, Gloria. "Genetic Influences on Preterm Birth." MCN, The American Journal of Maternal/Child Nursing 31, no. 3 (2006): 169–75. http://dx.doi.org/10.1097/00005721-200605000-00008.

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Peripato and Cheverud. "Genetic Influences on Maternal Care." American Naturalist 160, no. 6 (2002): S173. http://dx.doi.org/10.2307/3078955.

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DeFranco, Emily, Kari Teramo, and Louis Muglia. "Genetic Influences on Preterm Birth." Seminars in Reproductive Medicine 25, no. 1 (2007): 040–51. http://dx.doi.org/10.1055/s-2006-956774.

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Vinkhuyzen, A. A. E., S. van der Sluis, E. J. C. de Geus, D. I. Boomsma, and D. Posthuma. "Genetic influences on ‘environmental’ factors." Genes, Brain and Behavior 9, no. 3 (2010): 276–87. http://dx.doi.org/10.1111/j.1601-183x.2009.00554.x.

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Rende, Richard. "The Limits of Genetic Influences." Psychological Inquiry 6, no. 2 (1995): 157–61. http://dx.doi.org/10.1207/s15327965pli0602_12.

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Thompson, Paul M., Tyrone D. Cannon, Katherine L. Narr, et al. "Genetic influences on brain structure." Nature Neuroscience 4, no. 12 (2001): 1253–58. http://dx.doi.org/10.1038/nn758.

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Trang, Nguyen Minh, Jennifer Tran Cao Van, and Do Hoang Phuong Trang. "Genetic Influences on Mental Health." European Journal of Medical and Health Research 2, no. 5 (2024): 247–55. http://dx.doi.org/10.59324/ejmhr.2024.2(5).25.

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Mental and physical health are two most crucial factor that deciding the happiness and development of each individual in the world. Physical health problem is quite obvious and easy to be recognized through advanced and modern technology. However, mental health sometimes invisible and hard to be revealed by the machines and the patients themselves. It is believed that genetic factors play a significant role in the development of various mental health conditions. Based on extensive research, scientists categorize major mental illnesses into several types, including schizophrenia, bipolar disord

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Swallow, Dallas M. "Genetic influences on carbohydrate digestion." Nutrition Research Reviews 16, no. 01 (2003): 37. http://dx.doi.org/10.1079/nrr200253.

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Roy, Alec. "Genetic influences on suicide risk." Clinical Neuroscience Research 1, no. 5 (2001): 324–30. http://dx.doi.org/10.1016/s1566-2772(01)00033-0.

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Rutherford, Peter A. "Genetic influences in human hypertension." Journal of Hypertension 21, no. 1 (2003): 19–22. http://dx.doi.org/10.1097/00004872-200301000-00005.

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Stelzner, Thomas, and John Weil. "Genetic Influences on Ventilatory Control." Seminars in Respiratory and Critical Care Medicine 7, no. 04 (1986): 333–42. http://dx.doi.org/10.1055/s-2007-1012629.

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Heydemann, Ahlke, Jill M. Huber, Alexis Demonbreun, Michele Hadhazy, and Elizabeth M. McNally. "Genetic background influences muscular dystrophy." Neuromuscular Disorders 15, no. 9-10 (2005): 601–9. http://dx.doi.org/10.1016/j.nmd.2005.05.004.

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Grimshaw, Gina, and M. P. Bryden. "Genetic influences on the environment." Behavioral and Brain Sciences 17, no. 4 (1994): 750–51. http://dx.doi.org/10.1017/s0140525x00036967.

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McLaren, David G., and Mark Bovey. "Genetic Influences on Reproductive Performance." Veterinary Clinics of North America: Food Animal Practice 8, no. 3 (1992): 435–59. http://dx.doi.org/10.1016/s0749-0720(15)30697-6.

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