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Dissertations / Theses on the topic 'Glomerular basement membrane'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Wootton, Andrew. "The glomerular basement membrane and nephritis /." Title page, contents and abstract only, 1985. http://web4.library.adelaide.edu.au/theses/09PH/09phw918.pdf.

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2

Walton, H. A. "The effect of structural modifications on the permeation properties of renal basement membrane." Thesis, University of Oxford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382711.

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3

Falcone, Sara. "Nephrotic syndrome and glomerular basement membrane : genetic defect of the laminin α5 chain". Thesis, Open University, 2018. http://oro.open.ac.uk/56060/.

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Nephrotic syndrome is a heterogeneous group of disorders characterised by renal and extra-renal manifestations. Classic symptoms of nephrotic syndrome include severe proteinuria, hypoalbumiaemia, oedema and hyperlipidaemia. Genetic studies of hereditary forms of nephrotic syndrome have led to the identification of proteins playing a crucial role in slit diaphragm signalling, regulation of actin cytoskeleton dynamics and cell-matrix interactions. The laminin α5 chain is a 404 kDa protein essential for embryonic development and, in association with laminin β2 and laminin γ1, it is a major compon

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4

Leow, Chon Kar. "The long-term metabolic function of pancreatic islet grafts and the effect on glomerular basement membrane thickness." Thesis, University of Bristol, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.358194.

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5

Cox, Melissa Luanne. "Identification of a mutation in COL4A5 causative for X-linked Alport syndrome in the domestic dog and analysis of gene expression in the kidneys of affected and nonaffected siblings." Diss., Texas A&M University, 2003. http://hdl.handle.net/1969.1/244.

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The domestic dog, Canis lupus familiaris, plays many roles in the lives of humans. Additionally, the dog is recognized for its potential as a model for many human hereditary diseases. Thus, the genetics and genomics of the dog are being studied extensively in order to facilitate its use as a model, as well as to help the dog for its own sake. As part of this research effort, our laboratory has added type I markers (i.e., the acidic and basic keratins, c-kit, type I and IV collagens, and the gene encoding uromodulin) to the emerging map of the canine genome. The mapping of genes, particularly t

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6

Toda, Naohiro. "Crucial Role of Mesangial Cell-derived Connective Tissue Growth Factor in a Mouse Model of Anti-Glomerular Basement Membrane Glomerulonephritis." Kyoto University, 2018. http://hdl.handle.net/2433/232131.

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7

Dessapt, Cecile. "Regulation of alpha3beta1 integrin expression in podocytes by mechanical stretch, TGFbeta1 and glucose : implication for podocyte detachment from the glomerular basement membrane." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.437750.

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8

Kawamura, Takahide. "Ultrastructural localization of dominantly increased fibronectin in the markedly thickend glomerular basement membrane in selectively mated murine high IgA strain(HIGA mice)." Kyoto University, 2001. http://hdl.handle.net/2433/150201.

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9

Cornet, Sylvie. "Evolution de la lame basale glomerulaire au cours de la nephrogenese et de la senescence, chez le rat." Paris 6, 1988. http://www.theses.fr/1988PA066166.

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10

Bolton, Glen R. (Glen Reed) 1970. "Permeation of ficoll and ficoll sulfate through glomeruler basement membrane : effects of molecular size and charge." Thesis, Massachusetts Institute of Technology, 1998. http://hdl.handle.net/1721.1/50390.

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11

Moilanen, J. (Jyri). "Functional analysis of collagen XVII in epithelial cancers and a mouse model." Doctoral thesis, Oulun yliopisto, 2016. http://urn.fi/urn:isbn:9789526211695.

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Abstract Basement membranes (BM) underlie epithelia and endothelia and surround many tissues. In cutaneous BM epithelial cells are attached to the stroma via multiprotein complexes called hemidesmosomes (HD). Collagen XVII and integrin α6β4 are components of HD and they bind to laminin 332, a component of anchoring filaments, extracellularly. The main interest of this study is the function of collagen XVII and its interactions with these proteins. What is known about the function of collagen XVII is mostly derived from its role as an adhesive component in cutaneous HD. Here we demonstrate for

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12

Bakala, Hilaire. "Contribution a l'etude de la membrane basale glomerulaire (mbg) : analyse structurale, fonctionnelle et biochimique au cours du developpement foetal, lors de la senescence et en conditions experimentales, chez le rat." Paris 7, 1987. http://www.theses.fr/1987PA077089.

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La mise en place de la membrane glomerulaire (mbg) au cours de la nephrogenese chez le rat a ete etudie par des techniques biochimiques, cytoimmunologiques et structurales. Toutes les macromolecules de la mbg (collagene iv, laminine, fibronectine, pgags) sont detectees par immunofluorescence des le stade precoce de la nephrogenese. La filtration renale ne commenceq u'au 18**(e) jour de la gestation alors que la nibg n'est encore constituee que par une lame base (epitheliale). Les proprietes d'ultrafiltration ne deviennent effectives qu'a partir du jour 20 lorsque la mbg acquiert sa structure t

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13

Wootton, Andrew. "The glomerular basement membrane and nephritis." 1986. http://web4.library.adelaide.edu.au/theses/09PH/09phw918.pdf.

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14

Naidoo, Anban. "The measurement of glomerular basement membrane components and glycated albumin as improved markers of incipient diabetic nephropathy." Thesis, 2010. http://hdl.handle.net/10413/5375.

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Diabetes causes early structural changes to the glomerular basement membrane (GBM), which alters its function and leads to loss of protein in urine. Formation of advanced glycation endproducts (AGEs) is one mechanism proposed to be responsible for the structural changes to the GBM. AGEs are thought to affect blood flow i.e. glomerular filtration rate (GFR) and vascular permeability which over time manifests as overt proteinuria. The gradual loss of minute amounts of protein (albumin) is referred to as microalbuminuria (MA). Microalbuminuria is a dynamic process, with patients regressing to nor

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15

Grönemeyer, Lisa-Lena. "Die Interaktion zwischen der glomerulären Basalmembran und der Schlitzmembran im col43+/-/nphs2+/R140Q-Tiermodell." Doctoral thesis, 2011. http://hdl.handle.net/11858/00-1735-0000-000D-F1F2-1.

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16

Prinz, Carolin Susanne. "Nidogen-2 in der Pathogenese Kollagen IV-assoziierter Nephropathien bei zusätzlicher Podocin-Mutation." Doctoral thesis, 2016. http://hdl.handle.net/11858/00-1735-0000-002B-7CB5-B.

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Kollagen IV assoziierte Nephropathien sind hereditäre Erkrankungen, die die glomeruläre Basalmembran betreffen. Homozygote Aberrationen des COL4A3- oder des COL4A4-Gens zeigen wie X-chromosomal dominant vererbte Mutationen des COL4A5-Gens das klinische Bild des Alport-Syndroms mit frühzeitigem terminalem Nierenversagen. Heterozygote COL4A3-Mutationen sind ursächlich für die benigne familiäre Hämaturie. Ein zusätzlicher Polymorphismus in Nphs2, welches das Schlitzmembranprotein Podocin kodiert, könnte hierbei zu einem aggravierten Krankheitsverlauf führen. Um diese These zu überprüfen, ist eine

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17

Spieker, Christine. "Die Bedeutung der glomerulären Basalmembrankomponente Nidogen-1 bei podozytären Erkrankungen der Niere." Doctoral thesis, 2017. http://hdl.handle.net/11858/00-1735-0000-0023-3E54-4.

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18

Martin, Maria. "Die Bedeutung des Kollagenrezeptors α2β1- Integrin bei der Pathogenese und Prävention der Nierenfibrose in hereditären Typ IV- Kollagen- Erkrankungen". Doctoral thesis, 2011. http://hdl.handle.net/11858/00-1735-0000-0006-B1D1-8.

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