Relevant bibliographies by topics / Growth plate Diseases

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Growth plate Diseases'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Growth plate Diseases"

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Xie, Yangli, Siru Zhou, Hangang Chen, Xiaolan Du, and Lin Chen. "RECENT RESEARCH ON THE GROWTH PLATE: Advances in fibroblast growth factor signaling in growth plate development and disorders." Journal of Molecular Endocrinology 53, no. 1 (August 2014): T11—T34. http://dx.doi.org/10.1530/jme-14-0012.

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Skeletons are formed through two distinct developmental actions, intramembranous ossification and endochondral ossification. During embryonic development, most bone is formed by endochondral ossification. The growth plate is the developmental center for endochondral ossification. Multiple signaling pathways participate in the regulation of endochondral ossification. Fibroblast growth factor (FGF)/FGF receptor (FGFR) signaling has been found to play a vital role in the development and maintenance of growth plates. Missense mutations inFGFsandFGFRscan cause multiple genetic skeletal diseases wit
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Holzer, Tatjana, Kristina Probst, Julia Etich, Markus Auler, Veronika S. Georgieva, Björn Bluhm, Christian Frie, et al. "Respiratory chain inactivation links cartilage-mediated growth retardation to mitochondrial diseases." Journal of Cell Biology 218, no. 6 (May 13, 2019): 1853–70. http://dx.doi.org/10.1083/jcb.201809056.

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In childhood, skeletal growth is driven by transient expansion of cartilage in the growth plate. The common belief is that energy production in this hypoxic tissue mainly relies on anaerobic glycolysis and not on mitochondrial respiratory chain (RC) activity. However, children with mitochondrial diseases causing RC dysfunction often present with short stature, which indicates that RC activity may be essential for cartilage-mediated skeletal growth. To elucidate the role of the mitochondrial RC in cartilage growth and pathology, we generated mice with impaired RC function in cartilage. These mi
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Gersing, Alexandra S., Klaus Woertler, Pia M. Jungmann, Christine Bollwein, and Benedikt J. Schwaiger. "Vertebrae, Vertebral End Plates, and Disks: Concepts and Specific Pathologies." Seminars in Musculoskeletal Radiology 23, no. 05 (September 25, 2019): 489–96. http://dx.doi.org/10.1055/s-0039-1693976.

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AbstractVertebral end plates cover the osseous vertebral body. The integrity of the cartilaginous end plates is of great importance for the entire vertebral segment because the vascularized end plate provides the nutrition for the avascular disk. Yet several pathologies may occur at these end plates at the embryonic stage, in childhood to adolescence (e.g., ossification and segmentation disorders of the spine, persistent notochord, slippage of the growth plate), as well as in the mature spine of an adult (degenerative disk disease), that may impact the integrity of the cartilaginous end plate
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Sederquist, Bettina, Paola Fernandez-Vojvodich, Farasat Zaman, and Lars Sävendahl. "RECENT RESEARCH ON THE GROWTH PLATE: Impact of inflammatory cytokines on longitudinal bone growth." Journal of Molecular Endocrinology 53, no. 1 (April 7, 2014): T35—T44. http://dx.doi.org/10.1530/jme-14-0006.

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Children with inflammatory diseases usually display abnormal growth patterns as well as delayed puberty. This is a result of several factors related to the disease itself, such as malnutrition, hypercortisolism, and elevated levels of pro-inflammatory cytokines. These factors in combination with glucocorticoid treatment contribute to growth retardation during chronic inflammation by systemically affecting the major regulator of growth, the GH/IGF1 axis. However, recent studies have also shown evidence of a direct effect of these factors at the growth plate level. In conditions of chronic infla
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Guevara-Morales, Johana M., Michael Frohbergh, Hector Castro-Abril, Juan J. Vaca-González, Luis A. Barrera, Diego A. Garzón-Alvarado, Edward Schuchman, and Calogera Simonaro. "Growth Plate Pathology in the Mucopolysaccharidosis Type VI Rat Model—An Experimental and Computational Approach." Diagnostics 10, no. 6 (May 31, 2020): 360. http://dx.doi.org/10.3390/diagnostics10060360.

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Background: Mucopolysaccharidoses (MPS) are a group of inherited metabolic diseases caused by impaired function or absence of lysosomal enzymes involved in degradation of glycosaminoglycans. Clinically, MPS are skeletal dysplasias, characterized by cartilage abnormalities and disturbances in the process of endochondral ossification. Histologic abnormalities of growth cartilage have been reported at advanced stages of the disease, but information regarding growth plate pathology progression either in humans or in animal models, as well as its pathophysiology, is limited. Methods: Histological a
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Kosowska-Shick, Klaudia, Lois M. Ednie, Pamela McGhee, Kathy Smith, Cynthia D. Todd, Amanda Wehler, and Peter C. Appelbaum. "Incidence and Characteristics of Vancomycin Nonsusceptible Strains of Methicillin-Resistant Staphylococcus aureus at Hershey Medical Center." Antimicrobial Agents and Chemotherapy 52, no. 12 (October 6, 2008): 4510–13. http://dx.doi.org/10.1128/aac.01073-08.

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ABSTRACT All 982 methicillin-resistant Staphylococcus aureus strains collected from August 2006 to December 2007 were tested for vancomycin susceptibility by using 3-μg/ml vancomycin brain heart infusion screening plates, a vancomycin Etest, and a vancomycin/teicoplanin macro Etest. Three vancomycin-intermediate Staphylococcus aureus (VISA) (0.3%) and two heterogeneous VISA (0.2%) isolates were identified. The screening method yielded 895 cases of ≤1 colony and 87 positive results (with growth of >1 colony after 48 h); further Etests showed 82/87 isolates with growth on screening plates to
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Evans, K. D., L. E. Sheppard, D. I. Grossman, S. H. Rao, R. B. Martin, and A. M. Oberbauer. "Long Term Cyclic Pamidronate Reduces Bone Growth by Inhibiting Osteoclast Mediated Cartilage-to-Bone Turnover in the Mouse." Open Orthopaedics Journal 2, no. 1 (July 14, 2008): 121–25. http://dx.doi.org/10.2174/1874325000802010121.

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Bisphosphonates, used to treat diseases exhibiting increased osteoclast activity, reduce longitudinal bone growth through an as yet undefined mechanism. Pamidronate, an aminobisphosphonate, was given weekly to mice at 0, 1.25, or 2.50 mg/kg/wk beginning at 4 weeks of age. At 12 weeks of age, humeral length, growth plate area, regional chondrocyte cell numbers, chondrocyte apoptosis, TRAP stained osteoclast number, and osteoclast function assessed by cathepsin K immunohistochemistry were quantified. Humeral length was decreased in pamidronate treated mice compared to vehicle control mice, and c
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Jeremic, Svetlana, Vladimir Radosavljevic, and Dobrila Jakic-Dimic. "Current bacterial diseases of fresh water fishes." Biotehnologija u stocarstvu 21, no. 3-4 (2005): 141–51. http://dx.doi.org/10.2298/bah0504141j.

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From the beginning of fish cultivation, diseases have appeared as a serious problem in this branch of agriculture. In recent years, by the intensifying of production, fish diseases have become even more significant and complex field. Bacterial diseases are constant threat for fish farming, and because of rapid course and severity of clinical manifestations the represent significant part of fish pathology, and also have great economical importance. Harmful effects of bacterial diseases on fishes are: increased morbidity and mortality rate, decreased feed conversion efficiency, decreased growth
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Martsyniak, S. M., and S. S. Strafun. "Surgical treatment of multidimensional deformities of lower limbs before closure of the growth plate in children with rachitic diseases." TRAUMA 21, no. 2 (March 1, 2020): 17–23. http://dx.doi.org/10.22141/1608-1706.2.21.2020.202229.

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Haseeb, Abdul, Ranjan Kc, Marco Angelozzi, Charles de Charleroy, Danielle Rux, Robert J. Tower, Lutian Yao, et al. "SOX9 keeps growth plates and articular cartilage healthy by inhibiting chondrocyte dedifferentiation/osteoblastic redifferentiation." Proceedings of the National Academy of Sciences 118, no. 8 (February 17, 2021): e2019152118. http://dx.doi.org/10.1073/pnas.2019152118.

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Cartilage is essential throughout vertebrate life. It starts developing in embryos when osteochondroprogenitor cells commit to chondrogenesis, activate a pancartilaginous program to form cartilaginous skeletal primordia, and also embrace a growth-plate program to drive skeletal growth or an articular program to build permanent joint cartilage. Various forms of cartilage malformation and degeneration diseases afflict humans, but underlying mechanisms are still incompletely understood and treatment options suboptimal. The transcription factor SOX9 is required for embryonic chondrogenesis, but it
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Dissertations / Theses on the topic "Growth plate Diseases"

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Foster, Bruce Kristian. "Epiphyseal plate repair using fat interposition to reverse physeal deformity : an experimental study." Title page, contents and summary only, 1989. http://web4.library.adelaide.edu.au/theses/09MD/09mdf754.pdf.

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Bibliography: leaves 169-197. Hypothesises that the physis has an internal mechanism of repair to restore physeal function. Aims to establish a defined degree of deformity by partial growth plate excision, then to examine different methods of reversal of such deformity to observe the process of growth plate repair. A secondary aim was to define the percentage of physis that could be resected yet still enable reversal of deformity.
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Rutt, Julianne Eileen. "Molecular Analysis Of The Epiphyseal Growth Plate In Rachitic Broilers: Evidence For The Etilogy Of The Condition." Columbus, Ohio : Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1223319403.

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Taïeb, Mahdia. "Investigation des mécanismes moléculaires impliqués dans les anomalies du développement ostéoarticulaire chez la souris invalidée pour le gène de la Xylosyltransférase I." Thesis, Université de Lorraine, 2019. http://www.theses.fr/2019LORR0032/document.

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Les protéoglycanes (PGs) jouent un rôle essentiel dans plusieurs processus physiologiques majeurs tels que la signalisation cellulaire, la prolifération et la migration ; ceci grâce aux interactions entre leurs chaînes de glycosaminoglycanes (GAGs) avec des médiateurs solubles et leurs récepteurs. L'initiation de la synthèse des chaînes de GAGs des PGs est catalysée par la xylosyltransferase I (XT-I). Récemment plusieurs études ont montré différentes mutations au niveau du gène de la XT-I associées au syndrome Desbuquois de type II, caractérisée des anomalies ostéoarticulaires. Afin d’étudier
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Bercetche, Joëlle. "Effets rhizogenes exerces par a. Rhizogenes chez le pois : aspects morphogenetiques, cellulaires et hormonaux." Paris 6, 1987. http://www.theses.fr/1987PA066145.

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La sequence ontogenetique de la rhizogenese transformee et les effets cellulaires induits par trois souches d'a. Rhizogenes (1855, 2659 et 8196) chez l'epicotyle de pois sont recherches. Apres inoculation par la souche 1855, on enregistre une large gamme de symptomes comparables a ceux produits par des auxines exogenes. Il s'agit de symptomes comparables a ceux produits par des auxines exogenes. Il s'agit de symptomes cytophysiologiques (hyperhydrie, synthese d'adn, fragmentations nucleaires), histologiques (cambiogenese, tracheogenese) et morphogenetiques (rhizogenese transformee aux points d
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LE, COZ SERGE. "La rhizomanie de la betterave sucriere : multiplication du virus et aspects agronomiques de la maladie." Paris 6, 1986. http://www.theses.fr/1986PA066644.

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Les chloroplastes des feuilles de betteraves rhizomaniees sont appauvries en pigments, en proteines, en liquides polaires et leur activite photosynthetique est reduite. Dans les cellules virosees, l'etude ultrastructure montre une association des amas de virus avec le reticulum endoplasmique granuleux. Un protocole pour la preparation de suspensions enrichies en cytosores isoles de polymyxa betae est propose. Le champignon est retrouve a tous les niveaux du sol de quatre parcelles rhizomaniees ou saines de la region de pithiviers. Une terre rhizomaniee reste infectieuse aprese un an et demi de
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Foster, Bruce K. "Epiphyseal plate repair using fat interposition to reverse physeal deformity : an experimental study / thesis submitted in March 1989 for the degree of Doctor of Medicine in the University of Adelaide by Bruce Kristian Foster." 1989. http://hdl.handle.net/2440/38304.

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Bibliography: leaves 169-197.<br>xiv, 197 leaves :<br>Title page, contents and abstract only. The complete thesis in print form is available from the University Library.<br>Hypothesises that the physis has an internal mechanism of repair to restore physeal function. Aims to establish a defined degree of deformity by partial growth plate excision, then to examine different methods of reversal of such deformity to observe the process of growth plate repair. A secondary aim was to define the percentage of physis that could be resected yet still enable reversal of deformity.<br>Thesis (Ph.D.)--Uni
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Lapointe, Marie. "Les facteurs écologiques influençant la dynamique d'une espèce exotique envahissante, Acer platanoides, et d'un congénère indigène, A. saccharum, dans une forêt urbaine du sud du Québec." Thèse, 2009. http://hdl.handle.net/1866/8136.

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Books on the topic "Growth plate Diseases"

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M, Shapiro Irving, Boyan Barbara, and Anderson H. Clarke, eds. The growth plate. Amsterdam: IOS Press, 2002.

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Organisation, International Labour, ed. HIV and AIDS in the informal workplaces: Pilot project research report : creating an enabling environment in the market place for business growath and sustainability. Lusaka: International Labour Organisation, 2007.

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1925-, Uhthoff Hans K., Wiley James J, University of Ottawa. Division of Orthopedic Surgery., and Pediatric Orthopaedic Society of North America., eds. Behavior of the growth plate. New York: Raven Press, 1988.

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(Editor), Irving M. Shapiro, Barbara Boyan (Editor), and H. Clarke Anderson (Editor), eds. The Growth Plate (Biomedical and Health Research, 54). Ios Pr Inc, 2002.

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Joshi, Mahesh K., and J. R. Klein. Australia—The Hidden Jewel. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198827481.003.0012.

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The twenty-first century is being touted as the Asian century. With its stable economy, good governance, education system, and above all the abundant natural resources, will Australia to take its place in the global economy by becoming more entrepreneurial and accelerating its rate of growth, or will it get infected with the so-called Dutch disease? It has been successful in managing trade ties with fast-developing economies like China and India as well as developed countries like the United States. It has participated in the growth of China by providing iron ore and coal. Because it is a low-
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Medforth, Janet, Linda Ball, Angela Walker, Sue Battersby, and Sarah Stables. Antenatal care. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198754787.003.0004.

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This chapter comprises confirming the pregnancy, the signs and symptoms of pregnancy, pregnancy testing and how to go about it, pregnancy adaptation, how the body changes in response to the pregnancy, the booking interview, and where, when, how, and why it takes place. Pregnancy screening and risk assessment, based on the medical, social, and obstetric history, is outlined. Blood group and the rhesus factor, their importance in terms of preventing haemolytic disease in the newborn, and anti-D prophylaxis are explained. Down’s syndrome risk screening protocols, gaining consent, and the testing
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Fox, Dov. Birth Rights and Wrongs. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780190675721.001.0001.

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Today, tens of millions of Americans rely on reproductive advances to help them carry out decisions more personal and far-reaching than almost any other they will ever make: They use birth control or abortion to delay or avoid having children; surrogacy or tissue donation to start or grow a family; and genetic diagnosis or embryo selection to have offspring who survive and flourish. This is no less than the medicine of miracles: It fills empty cradles; frees families from debilitating disease; and empowers them to plan a life that doesn’t include parenthood. But accidents happen: Embryologists
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Book chapters on the topic "Growth plate Diseases"

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Meßler, H., W. Rüther, J. Kühr, and K. J. Münzenberg. "The Influence of Magnesium and Calcium Antagonists on the Epiphyseal Growth Plate — A Comparison." In Generalized Bone Diseases, 313–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-73346-8_28.

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Karbowski, A., J. Herwig, H. H. Matthiaß, and E. Buddecke. "Effects of Mechanical Factors on Structure and Function of the Growth Plate of Long Bones." In Generalized Bone Diseases, 337–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-73346-8_31.

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Quint, P., J. Althoff, I. Harmeyer, K. D. Richter, and H. J. Höhling. "Concentration Profiles of Zinc and Lead Along the Epiphyseal Growth Plate of Normal and Rachitic Piglets as Related to Activities of Esterases." In Generalized Bone Diseases, 181–89. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-73346-8_18.

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Srinivas, Vickram, and Irving M. Shapiro. "The Epiphyseal Growth Plate: The Engine That Drives Bone Elongation." In Handbook of Growth and Growth Monitoring in Health and Disease, 1331–49. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-1795-9_80.

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Salvatore, Seminara, Stagi Stefano, and Nanni Laura. "Thyroid Function During Catch-Up Growth: A Focus on the Growth Plate." In Handbook of Growth and Growth Monitoring in Health and Disease, 905–16. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-1795-9_54.

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Gat-Yablonski, Galia, and Moshe Phillip. "Nutritional-Induced Longitudinal Catch-Up Growth: A Focus on the Growth Plate, Growth-Related Genes, Autophagy, mTOR, and microRNAs." In Handbook of Growth and Growth Monitoring in Health and Disease, 1029–43. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-1795-9_61.

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Lui, Julian C., and Jeffrey Baron. "Effects of Glucocorticoids on the Growth Plate." In Pediatric Adrenal Diseases, 187–93. S. Karger AG, 2010. http://dx.doi.org/10.1159/000321244.

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Da Rin de Lorenzo, Ferdinando. "Specific Bacterial Immunotherapy in Treating Chronic Osteomyelitis." In Infectious Diseases and Sepsis [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.98751.

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The immunological experience is treating osteomyelitis chronic forms at the Istituto Putti in Cortina starts in 1963 by introducing immunotherapy, applied by the progressive administration in growing doses of a staphylococci pool, that had been collected from some patients with bone infections by the same germ and then inactivated in an aqueous solution suspension. This therapy is coadjutant of antibiotics, surgical and hyperbaric therapy and not substitutive of these. This study ascertained indeed a reduction of the phagocytic activity as a whole, and especially the opsonisation activity It has been thought therefore that in immunotherapy more factors are involved; their principal property is to reduce the allergising effect and therefore to desensitise vs. the germ proteins and to increase the phagocytic activity. This condition, neither whose entity nor its lasting may be defined, does not appear to be unlimited. Obviously this desensitisation can be obtained also by the right antibiotic choice that, as already said mainly in acute forms, may develop their bactericidal properties and sterilise the focus. In the chronic forms it is possible to provoke this mechanism by carrying out a surgical toilette that restores the vascularization and stimulation conditions needed for a correct antibiotic action. Checks upon immuno-stimulation treatment termination clearly showed corresponding results between laboratory deficit corrected and clinical conditions bettering. The casuistry is based on 50 patients with hematogenic osteomyelitis, all under the age of 16, age at which the growth plate is still active, and 117 post-traumatic septic non-union, where this term was adopted for cases that showed a lack of non-solidification at 6 months after trauma. We have expressly made a distinction between hematogenic and post-traumatic forms, since the relationships between bacterial counts vs. host response do differ.
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"Growth of tissues and organs." In Oxford Assess and Progress: Medical Sciences, edited by Jade Chow, John Patterson, Kathy Boursicot, and David Sales. Oxford University Press, 2012. http://dx.doi.org/10.1093/oso/9780199605071.003.0017.

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Many of the basic pathological processes that take place in cells, tissues, and organs are common, regardless of tissue types. These are due to aetiologies and pathogenetic mechanisms that lead to a common fate. Understanding these allows students to develop a logical approach to classifying diseases according to the underlying mechanism. In so doing, students should not have to memorize lists, and will be able to deal with new or unfamiliar clinical scenarios. An understanding of the underlying disease processes, including having precise definitions of these, allows doctors to describe the diseases accurately and to communicate more clearly. Unfortunately, many of the terms sound like each other, but fortunately, there are not too many terms. In recent years, the molecular mechanisms underlying many disease processes that may have been known for decades have been uncovered. It is important to know these as they may have implications for management, for example genetic counselling, or may lead to development of new therapies.
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Semple-Hess, Janet. "Pediatric Orthopedic Emergencies." In Pediatric Emergencies, 410–42. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190073879.003.0037.

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Children’s bones, due to their continual maturity during childhood and into late adolescence and their unique features such as growth plates, can present a challenge to the emergency physician. Interpretation of radiographs when looking for fractures in growing children, as well as deciding on treatment and determining whether to employ surgical versus nonsurgical management, requires knowledge of the patterns of injury in children. This chapter discusses these injuries as well as fractures related to child abuse, overuse injuries, back pain in children, radial head subluxation, slipped capital femoral epiphyses, avulsion fractures of the pelvis, Perthes disease, and infectious diseases (osteomyelitis and septic arthritis).
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Conference papers on the topic "Growth plate Diseases"

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Trachet, Bram, Daniel Devos, Julie De Backer, Anne De Paepe, Bart L. Loeys, and Patrick Segers. "Patient-Specific Modelling of Aortic Arch Wall Shear Stress Patterns in Patients With Marfan Syndrome." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206340.

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Marfan syndrome (MFS) is a genetic connective tissue disorder with a high prevalence of aortic aneurysm formation (a pathological dilatation of the aorta), typically at the aortic root. The disorder is caused by mutations in the gene encoding fibrillin-1 [1]. Recently, it has been shown in mouse models that selected manifestations of MFS, such as aortic aneurysm formation, can be explained by excessive signaling by the transforming growth factor–beta (TGF-beta) family of cytokines [2]. Although the footprint of the disease is clearly genetic, there is still a role for (computational) biomechan
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McClain, Jacob, Sara L. Tuell, and Susan N. Thomas. "Tumors Change the Elastic and Viscoelastic Properties of Draining Lymph Node Tissues." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14419.

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Changes in tissue mechanical properties are often the first indication of malignant disease, with the detection of a stiff lump by a patient. These changes include growth-induced solid stresses, increased matrix stiffness, high fluid pressure, and increased interstitial flow, which in turn enhance fluid flux away from the tumor to downstream lymph nodes (LNs). But in addition to changing the way a tumor feels to a patient, altered tumor tissue mechanics promote cancer cell invasion into lymphatic vessels, allowing their metastatic dissemination to draining LNs. LN swelling and stiffening is an
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Zhang, Bolun, Daniel Farley, Heidi-Lynn Ploeg, and Michael Zinn. "Validation of Feedback Control Approach for an Implantable Limb Lengthening Device." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3456.

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Lower limb length discrepancy (LLD), defined by unequal length of paired lower limbs, contributes to lower back pain, osteoarthritis of the hip, and stress fractures [1–3]. The Center for Disease Control and Prevention estimated that there were approximately 700 children born with LLD each year in US [4]. Patients may receive distraction osteogenesis treatment, in which an osteotomy is performed on the shorter limb, and mechanical force is applied to gradually distract the two halves of the bone during the healing process. This stretches the bone callus during healing to achieve desired limb l
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Ochie, Samuel, and Karen Ochie. "Design and Construction of an Air Quality Monitoring System to Mitigate Virus Spread." In SPE Nigeria Annual International Conference and Exhibition. SPE, 2021. http://dx.doi.org/10.2118/208263-ms.

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Abstract Air pollution is one of the most dangerous problems we face in the world today. It causes many illnesses and diseases that affect the immune system of humans and non-human animals and is also a means of propagating the novel COVID-19 or corona virus. Temperature, humidity, and the level of carbon dioxide characterize the air quality in an environment. In addition to abnormal temperatures and humidity causing direct problems to humans like headaches, heatstroke, hypothermia and hyperthermia, death and so on, it could also cause complicated problems like the acceleration of the growth a
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Mendygarin, Yertay, Luis R. Rojas-Solórzano, Nurassyl Kussaiyn, Rakhim Supiyev, and Mansur Zhussupbekov. "Eulerian-Eulerian Multiphase Modeling of Blood Cells Segregation in Flow Through Microtubes." In ASME 2017 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/imece2017-70850.

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Cardiovascular Diseases, the common name for various Heart Diseases, are responsible for nearly 17.3 million deaths annually and remain the leading global cause of death in the world. It is estimated that this number will grow to more than 23.6 million by 2030, with almost 80% of all cases taking place in low and middle income countries. Surgical treatment of these diseases involves the use of blood-wetted devices, whose relatively recent development has given rise to numerous possibilities for design improvements. However, blood can be damaged when flowing through these devices due to the lac
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