Academic literature on the topic 'Guillain-Barré'

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Journal articles on the topic "Guillain-Barré"

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Cibulčík, František. "Guillain-Barré polyradiculoneuritis." Neurologie pro praxi 17, no. 1 (2016): 11–15. http://dx.doi.org/10.36290/neu.2016.003.

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Manfroi, Bárbara, and Letícia Aranega Dalari. "Síndrome de Guillain-Barré / Guillain-Barré Syndrome." Brazilian Journal of Health Review 5, no. 1 (2022): 2565–72. http://dx.doi.org/10.34119/bjhrv5n1-229.

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Sharma, Kamal, Supatida Tengsupakul, Omar Sanchez, Rozaleen Phaltas, and Paul Maertens. "Guillain–Barré syndrome with unilateral peripheral facial and bulbar palsy in a child: A case report." SAGE Open Medical Case Reports 7 (January 2019): 2050313X1983875. http://dx.doi.org/10.1177/2050313x19838750.

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Guillain–Barré syndrome is characterized by progressive motor weakness, sensory changes, dysautonomia, and areflexia. Cranial nerve palsies are frequent in Guillain–Barré syndrome. Among cranial nerve palsies in Guillain–Barré syndrome, facial nerve palsy is the most common affecting around half of the cases. Facial palsy in Guillain–Barré syndrome is usually bilateral. We describe a pediatric Guillain–Barré syndrome variant presenting with unilateral peripheral facial palsy and dysphagia. A 5-year-old boy had progressive lower extremity weakness and pain 3 days prior to onset of unilateral pe
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Bondi, Steven, Elizabeth Carroll, and Jaydeep Bhatt. "Taking a Strohl Through History: Putting Strohl Back in Guillain-Barré-Strohl Syndrome." Journal of Neuromuscular Diseases 7, no. 4 (2020): 521–22. http://dx.doi.org/10.3233/jnd-200545.

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Guillain-Barré Syndrome is a popular eponym that comes from a 1916 paper by Drs. Guillain, Barré, and Strohl. These physicians described two soldiers in the French Sixth Army during World War I who developed acute progressive motor weakness. Although Drs. Guillain and Barré have continued to be included in the syndrome’s eponym, Dr. Strohl has been forgotten despite having strongly contributed to the original paper. The reasons previously mentioned for Dr. Strohl’s absence appear trivial in contemporary practice and thus, his name deserves to be reintroduced to Guillain-Barré-Strohl Syndrome.
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Shahrizaila, Nortina, and Nobuhiro Yuki. "Guillain-Barré Syndrome Animal Model: The First Proof of Molecular Mimicry in Human Autoimmune Disorder." Journal of Biomedicine and Biotechnology 2011 (2011): 1–5. http://dx.doi.org/10.1155/2011/829129.

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Molecular mimicry between self and microbial components has been proposed as the pathogenic mechanism of autoimmune diseases, and this hypothesis is proven in Guillain-Barré syndrome. Guillain-Barré syndrome, the most frequent cause of acute neuromuscular paralysis, sometimes occurs afterCampylobacter jejunienteritis. Gangliosides are predominantly cell-surface glycolipids highly expressed in nervous tissue, whilst lipo-oligosaccharides are major components of the Gram-negative bacteriumC. jejuniouter membrane. IgG autoantibodies to GM1 ganglioside were found in the sera from patients with Gui
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Morshed, Md Mahabub, AKM Ferdous Rahman, Syed Tariq Reza, Muhammad Asaduzzaman, Mohammad Selim, and Mohammad Asrafuzzaman. "A case of a recurrent GBS." Bangladesh Critical Care Journal 5, no. 2 (2017): 135–38. http://dx.doi.org/10.3329/bccj.v5i2.34397.

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Background: Guillain-Barré syndrome is an acquired polyradiculo-neuropathy, often preceded by an antecedent event. It is a monophasic disease but a recurrence rate of 1–6 % is documented in a subset group of patients.Case presentation: Thirty-five-years-old female with past history of near complete recovery following Guillain-Barré syndrome 17 years back presented with acute, ascending symmetrical flaccid quadriparasis extending to bulbar muscles and respiratory compromise needing mechanical ventilation. Nerve conduction study revealed AMAN variant of Guillain-Barré syndrome. Cerebrospinal flu
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Surbakti, Stefanie Meitha Putri Sejahtera, and Aida Fitri. "Mortality of Guillain Barre Syndrome and Myasthenic Crisis in Haji Adam Malik Central General Hospital Medan January 2021- December 2022." Asian Australasian Neuro and Health Science Journal (AANHS-J) 6, no. 2 (2024): 35–41. http://dx.doi.org/10.32734/aanhsj.v6i2.17730.

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Introduction: Neuromuscular disorders are disorders of the neuro system that impact the muscular system. Among them are Guillain-Barré Syndrome and Myasthenic Crisis. Guillain-Barré syndrome is an autoimmune disorder of the nervous system that is polyradiculoneuropathic, characterized by weakness that radiates from the legs to the arms, torso, and face. Myasthenic crisis is a state of acute respiratory failure due to the exacerbation of Myasthenia Gravis. These two diseases are the most common types of neurodiseases found in hospital ICU patients. Objective: Determining the mortality rate of G
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Shahrizaila, Nortina, Helmar C. Lehmann, and Satoshi Kuwabara. "Guillain-Barré syndrome." Lancet 397, no. 10280 (2021): 1214–28. http://dx.doi.org/10.1016/s0140-6736(21)00517-1.

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Mirian, Ario, Michael W. Nicolle, and Adrian Budhram. "Guillain–Barré syndrome." Canadian Medical Association Journal 193, no. 11 (2021): E378. http://dx.doi.org/10.1503/cmaj.202710.

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Chung, Aimee, and Melissa Deimling. "Guillain-Barré Syndrome." Pediatrics in Review 39, no. 1 (2018): 53–54. http://dx.doi.org/10.1542/pir.2017-0189.

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Dissertations / Theses on the topic "Guillain-Barré"

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Phongsisay, Vongsavanh, and vongsavang@yahoo com au. "Campylobacter jejuni and the Guillain-Barré syndrome." RMIT University. Applied Sciences, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20061221.100446.

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Campylobacter jejuni is an enteric bacterium that causes human gastroenteritis worldwide. Some C. jejuni strains exhibiting human ganglioside-like lipooligosaccharide (LOS) structures, such as GM1 ganglioside, can induce an autoimmune neuropathy of the peripheral nervous system known as the Guillain-Barré syndrome (GBS). This GBS-inducible determinant is encoded by a gene cluster, which shows a high degree of variation among C. jejuni strains. The experiments presented in this thesis were conducted to give a better insight into the LOS synthesis genes in relation to the patho
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Mori, Izumi. "Syndrome de Guillain-Barré et perturbations immunitaires." Paris 6, 2008. http://www.theses.fr/2008PA066343.

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Le syndrome de Guillain–Barré (SGB) consiste en une atteinte auto–immune aiguë du système nerveux périphérique. L’infection déclenchant l’atteinte neurologique est souvent détectée, et la date du début de la maladie est connue. Notre hypothèse était que le profil de la réponse immunitaire pourrait influencer non seulement l’évolution naturelle de la maladie, mais également la sensibilité au traitement par immunoglobulines intraveineuses (IgIv). Nous proposons ici le premier biomarqueur de l’efficacité du traitement par IgIv au cours du SGB. Une forte proportion de cellules B matures circulante
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Sampaio, Pedro Henrique Marte de Arruda. "Assimetrias no exame neurológico de crianças com síndrome de Guillain-Barré." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17161/tde-25042018-150422/.

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A Síndrome de Guillain-Barré (SGB) é uma neuropatia periférica inflamatória aguda que tem sido definida pelo achado ou história de tetraparesia flácida arreflexa ascendente. Apresentações atípicas podem ser mais frequentes do que tem sido referido na literatura, particularmente na faixa etária infantil. Objetivo: Avaliar dados epidemiológicos e a prevalência de assimetria no exame neurológico em crianças com SGB. Métodos: Foram revisados 40 prontuários de crianças de 0 a 15 anos de idade com o diagnóstico de SGB, atendidas entre janeiro de 2000 e agosto de 2016. Avaliouse a presença de assimet
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Perez, Simone. "Fator neurotrófico ciliar e interleucina-6 na síndrome de Guillain-Barré." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2011. http://hdl.handle.net/10183/30980.

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A Síndrome de Guillain-Barré é uma polirradiculoneuropatia aguda imuno-mediada, clinicamente apresentando-se com envolvimento sensitivo e motor e curso progressivo, em muitos casos determinando grande incapacidade e morbimortalidade nos pacientes que a desenvolvem. Este projeto de pesquisa teve como objetivo o seguimento de uma coorte de 22 indivíduos portadores da Síndrome de Guillain-Barré admitidos no Hospital de Clínicas de Porto Alegre durante o período de Janeiro de 2008 a Dezembro de 2009 para a correlação dos achados clínicos com os níveis liquóricos de interleucina-6 (IL-6) e do fator
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Fok, Nga-yin Angel, and 霍雅妍. "Influenza vaccination and its association with Guillain-barréSyndrome." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45172043.

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Maire, Olivier. "Avenir fonctionnel et rééducation du syndrome de Guillain et Barré d'évolution prolongée." Montpellier 1, 1991. http://www.theses.fr/1991MON11176.

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Pontes, Tainá Madeira Barros. "Identificação etiológica de dengue em indivíduos com suspeita clínica da síndrome de Guillain Barré." Universidade de Fortaleza, 2017. http://dspace.unifor.br/handle/tede/108656.

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Made available in DSpace on 2019-03-30T00:18:21Z (GMT). No. of bitstreams: 0 Previous issue date: 2017-09-27<br>Guillain-Barré Syndrome is a polyneuropathy characterized by weakness, often started in an acute way, which bilaterally affects the limbs in a symmetrical manner, although there are some cases with asymmetry. In two-thirds of the patients, the start of the symptoms is preceded by an infectious disease of the upper respiratory or gastrointestinal tract. In literature, there are reports showing that the dengue virus is the causal agent of this syndrome; that way, this research was to
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Torres, Vitor Félix. "Níveis séricos e liquóricos da proteína S100B, enolase específica do neurônio e neurotrofinas na síndrome de Guillain-Barré." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2010. http://hdl.handle.net/10183/26920.

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A Síndrome de Guillain-Barré é caracterizada por uma polirradiculoneuropatia de instalação aguda, usualmente imuno-mediada, com características clínicas associado a um envolvimento sensitivo e motor progressivo, muitas vezes determinando grande incapacidade e morbimortalidade nos indivíduos acometidos pela doença. Este projeto de pesquisa teve como objetivo o seguimento de uma coorte de indivíduos portadores da Síndrome de Guillain-Barré admitidos no Hospital de Clínicas de Porto Alegre durante o período de Janeiro de 2008 à Dezembro de 2009 para a correlação dos achados clínicos com os níveis
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Gries, Manuela [Verfasser], and Tobias [Akademischer Betreuer] Hartmann. "Die Bedeutung angeborener Immunrezeptoren beim experimentellen Guillain-Barré-Syndrom / Manuela Gries. Betreuer: Tobias Hartmann." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2013. http://d-nb.info/1052904904/34.

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Greenshields, Kay. "Pathogenic potential of anti-ganglioside antibodies in a murine model of axonal Guillain-Barré syndrome." Thesis, University of Glasgow, 2007. http://theses.gla.ac.uk/3760/.

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Guillian-Barré Syndrome (GBS) is the world’s leading cause of neuromuscular paralysis occurring in serologically and pathogenically distinct forms. GBS is believed to have an autoimmune basis, where antibodies raised during antecedent infections (eg Campylobacter jejuni) cross-react with self antigens, exemplifying the process of molecular mimicry. These self-antigens are gangliosides, which are glycolipid structures enriched in peripheral nerve in specific membrane compartments termed lipid rafts. To date, successful murine models of anti-GD1a and anti-Gq1b ganglioside mediated neuropathy exi
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Books on the topic "Guillain-Barré"

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Hughes, Richard A. C. Guillain-Barré Syndrome. Springer London, 1990. http://dx.doi.org/10.1007/978-1-4471-3175-5.

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National Institute of Neurological and Communicative Disorders and Stroke. Office of Communications and Public Liaison and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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National Institute of Neurological and Communicative Disorders and Stroke. Office of Communications and Public Liaison. and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. El síndrome de Guillain-Barré. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. El síndrome de Guillain-Barré. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. El síndrome de Guillain-Barré. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. El síndrome de Guillain-Barré. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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Book chapters on the topic "Guillain-Barré"

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Öztürk, Şerefnur. "Guillain–Barré Syndrome." In Neurological Disorders in Clinical Practice. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23168-6_7.

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Bowes, Denise. "Guillain-Barré Syndrome." In When Doctors Get Sick. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4899-2001-0_7.

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Fary, Khan, and Louisa Ng. "Guillain-Barré Syndrome." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_554.

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Kaida, Kenichi. "Guillain–Barré Syndrome." In Advances in Experimental Medicine and Biology. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9636-7_20.

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Khadilkar, Satish V., Rakhil S. Yadav, and Bhagyadhan A. Patel. "Guillain–Barré Syndrome." In Neuromuscular Disorders. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5361-0_36.

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Jann, S., D. Facchetti, and I. Costi. "Guillain-Barré Syndrome." In Emergency Management in Neurology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56654-2_3.

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Fary, Khan, and Louisa Ng. "Guillain-Barré Syndrome." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_554-3.

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Benatar, Michael. "Guillain-Barré Syndrome." In Neuromuscular Disease. Humana Press, 2006. http://dx.doi.org/10.1007/978-1-59745-106-2_11.

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Rodríguez, Yhojan, Christopher Chang, Diana C. González-Bravo, M. Eric Gershwin, and Juan-Manuel Anaya. "Guillain-Barré Syndrome." In Neuroimmune Diseases. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-19515-1_24.

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Der, Christopher K., and Jie Zhou. "Guillain-Barré Syndrome." In Consults in Obstetric Anesthesiology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-59680-8_70.

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Conference papers on the topic "Guillain-Barré"

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Iqbal, Sameera, Muhammad Abrar-ul-Haq, and Abdulwahab Janjua. "Integrating Guillain-Barré Syndrome Awareness with Sustainable Islamic Business and Finance: A Path Towards Enhanced Health and Well-being." In 2024 International Conference on Sustainable Islamic Business and Finance (SIBF). IEEE, 2024. https://doi.org/10.1109/sibf63788.2024.10883843.

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Coppetti, Liara Eickhoff, Lana Caroline Palaver Dall’Ago Dall’Ago, and Andressa Wierzbicki. "Guillain-Barre related to Covid-19." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.525.

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Background: COVID-19 is a disease that affects the nervous system as proven by the presence of symptoms such as headache, nausea, dizziness, myalgia, anosmia and ageusia - and can cause numerous neurological complications. These complications include Guillain-Barré Syndrome, an autoimmune polyneuropathy usually caused by a previous infectious process. Its main clinical manifestation is asthenia in the limbs, in an ascending form. Objectives: Review studies that suggest relation between COVID-19 infection and Guillain-Barré Syndrome. Methods: A systematic review was carried out in April of 2021
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Filho, José Augusto de Medeiros, Lillian Monizy Mesquita Carlos, Milena Fontes de Azevedo, and Victor Hugo Rodrigues Morais. "NEUROPATOLOGIA DA SÍNDROME DE GUILLAIN-BARRÉ." In I Congresso Brasileiro de Estudos Patológicos On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbesp/57.

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Introdução: Síndrome de Guillain-Barré (SGB) é uma doença autoimune que age atacando a bainha de mielina na parte proximal dos nervos periféricos. O quadro clínico da doença se dá através de uma dormência e fraqueza inicialmente nos membros inferiores podendo evoluir até os membros superiores. O diagnóstico é clínico e o tratamento farmacológico deve ser feito com Imunoglobulina humana na fase aguda, para evitar progressão da sintomatologia. Objetivos: Este trabalho objetiva analisar à neuropatologia da SGB, mediante o relato de caso de um paciente. Desta forma contribuir para o esclarecimento
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Aamir, N. "Post COVID-19 Guillain-Barré Syndrome." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a2486.

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Cases, Eugenia, María del Rosario Zuloaga, Javier Aranda, Patricia Zorrilla, Irene Luquín, and Álvaro Pérez. "Síndrome de Guillain Barré en urgencias." In 31 Congreso Nacional de Medicina General y de Familia. Grupo Pacífico, 2025. https://doi.org/10.48158/semg25-634.

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Rodriguez, W., and S. M. Alegre-Boschetti. "One in a Million: Guillain-Barré Syndrome." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1775.

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de Quadros Martins, Vanessa, Jean Paulo Veronese de Souza, Guilherme Stüker, et al. "EOSINOPHILIC GRANULOMATOSIS WITH POLYANGIITIS MIMICKING GUILLAIN-BARRÉ SYNDROME." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17201.

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Lee, Annelise Akemi Higa, Felipe Teijeiro Cabral, Francisco Tomaz Meneses de Oliveira, and Rubens José Gagliardi. "Guillain Barré syndrome in late lupus: case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.557.

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Context: In the literature, there are few cases of Guillain Barré syndrome (GBS) in association with systemic lupus erythematosus (SLE). There is description of 19 neuropsychiatric syndromes associated with SLE, 12 of the central nervous system (CNS) and 7 of the peripheral nervous system (PNS). 10% of the patients with SLE and neurological manifestations have PNS complications. SLE is considered to have a late onset when it starts above 50 years of age and it consists in, on average, 26% of the total of individuals with SLE, however, in contrast to juvenile lupus, the number of studies is sca
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Barros, Gabriel Santaterra, Ana Paula Ramires Chiminazzo, Maria Luiza Ricarte Ruggeri, Maria Luisa Pelaes Stipp, and Helen Maia Tavares de Andrade. "Guillain-Barré Syndrome and Hyperreflexia: a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.588.

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Context: areflexia and hyporeflexia are mandatory clinical criteria for the diagnosis of Guillain-Barré Syndrome (GBS). However there are case reports in literature of GBS that exhibit hyperreflexia. Case report: Male patient, 38 years old, after weakness and pain in lower limbs, after exercise, for 4 days, without alteration of balance and sphincter. A week earlier, he had diarrhea and fever. Previous bariatric surgery. On examination: For both upper limbs, the grade of power was 5/5 in the proximal muscle group and 4/5 in the distal muscle group. For the lower limbs, the grade of power was 4
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Alves, Letícia Furtado, NATHALIA ALVES DO AMARAL, GLÓRIA EDENI DIAS PEREIRA AMORIM, LORENA ALCEBÍADES BORGES, and THIAGO CAVALCANTE RIBEIRO. "POLIRRADICULOPATIA AGUDA INFLAMATÓRIA IMUNOMEDIADA: SÍNDROME DE GUILLAIN-BARRÉ." In II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/6048.

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Introdução: A Síndrome de Guillain-Barré (SGB) é considerada uma polirradiculopatia aguda inflamatória imunomediada que pode acometer a bainha de mielina, o axônio do neurônio ou o nervo oculomotor. Por conseguinte, o primeiro caso define a polineuropatia aguda desmielinizante. Em contrapartida, a segunda situação determina a manifestação axonal da doença, a qual pode ser sensitivo motora ou motora pura. Por fim, tem-se a variante Miller fisher que os anticorpos afetam as fibras nervosas do oculomotor. A justificativa para a realização deste trabalho é que o conhecimento médico sobre o mecanis
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Reports on the topic "Guillain-Barré"

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Khalfaoui, S. Guillain Barré syndrome complicated by hip osteoarthropathy. Science Repository OU, 2019. http://dx.doi.org/10.31487/j.ssg.2018.01.02.

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