Academic literature on the topic 'Haemodynamic conditions'

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Journal articles on the topic "Haemodynamic conditions"

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Kuboki, Takuo, Kenji Maekawa, and Glenn Clark. "Intramuscular haemodynamics using near infra-red spectroscopy as a research strategy to understand chronic muscle pain pathophysiology." Spectroscopy 19, no. 1 (2005): 27–36. http://dx.doi.org/10.1155/2005/148586.

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Recently, localized intramuscular haemodynamic disturbance has been recognized as one of the cardinal features of a chronically painful muscle. To test this hypothesis, we have carried out several studies that have assessed intramuscular haemodynamics to understand more about haemodynamic alteration under conditions of an experimentally induced vasoreactive challenges in healthy controls and in patients with chronic muscle pain using near infra?red (NIR) spectroscopy. In addition, we have validated the NIR based haemodynamic signals by comparing against a water signal intensity change taken fr
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Midgett, Madeline, Venkat Keshav Chivukula, Calder Dorn, Samantha Wallace, and Sandra Rugonyi. "Blood flow through the embryonic heart outflow tract during cardiac looping in HH13–HH18 chicken embryos." Journal of The Royal Society Interface 12, no. 111 (2015): 20150652. http://dx.doi.org/10.1098/rsif.2015.0652.

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Blood flow is inherently linked to embryonic cardiac development, as haemodynamic forces exerted by flow stimulate mechanotransduction mechanisms that modulate cardiac growth and remodelling. This study evaluated blood flow in the embryonic heart outflow tract (OFT) during normal development at each stage between HH13 and HH18 in chicken embryos, in order to characterize changes in haemodynamic conditions during critical cardiac looping transformations. Two-dimensional optical coherence tomography was used to simultaneously acquire both structural and Doppler flow images, in order to extract b
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Scully, M. E., L. Collins, and C. M. Cooney. "HAEMODYNAMIC STABILITY AND INTUBATING CONDITIONS USING PROPOFOL-SEVOFLURANE." Anesthesia & Analgesia 88, Supplement (1999): 305S. http://dx.doi.org/10.1097/00000539-199902001-00302.

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Kovacs, Gabor, Philippe Herve, Joan Albert Barbera, et al. "An official European Respiratory Society statement: pulmonary haemodynamics during exercise." European Respiratory Journal 50, no. 5 (2017): 1700578. http://dx.doi.org/10.1183/13993003.00578-2017.

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There is growing recognition of the clinical importance of pulmonary haemodynamics during exercise, but several questions remain to be elucidated. The goal of this statement is to assess the scientific evidence in this field in order to provide a basis for future recommendations.Right heart catheterisation is the gold standard method to assess pulmonary haemodynamics at rest and during exercise. Exercise echocardiography and cardiopulmonary exercise testing represent non-invasive tools with evolving clinical applications. The term “exercise pulmonary hypertension” may be the most adequate to d
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Johny, Cherian, S. M. Abdul Khader, B. Raghuvir Pai, M. Zuber, K. A. Ahmed, and Zanuldin Ahmad. "Haemodynamic study of idealistic renal artery under different flow conditions." Journal of Computational Methods in Sciences and Engineering 19, no. 2 (2019): 541–52. http://dx.doi.org/10.3233/jcm-181059.

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Kara, H., T. Aydogdu, and V. Ulusan. "A comparison of intubation conditions and haemodynamic effects of magnesium." European Journal of Anaesthesiology 17, Supplement 19 (2000): 133–34. http://dx.doi.org/10.1097/00003643-200000002-00434.

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Kobr, J., V. Treska, J. Molacek, V. Kuntscher, V. Liska, and J. Koppl. "Different cardiac loading conditions and haemodynamic monitoring in animal models." Bratislava Medical Journal 113, no. 03 (2012): 131–34. http://dx.doi.org/10.4149/bll_2012_032.

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Kahn, J. F., B. Kapitaniak, F. Huart, and H. Monod. "Physiological modifications of local haemodynamic conditions during bilateral isometric contractions." European Journal of Applied Physiology and Occupational Physiology 54, no. 6 (1986): 624–31. http://dx.doi.org/10.1007/bf00943351.

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Ströhn, A., H. M. Häfner, and M. Jünger. "Biophysical characteristics of medical compression stockings." Phlebologie 36, no. 04 (2007): 197–204. http://dx.doi.org/10.1055/s-0037-1622183.

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Summary Aim: Haemodynamic effectivity of 13 compression stockings in correlation with its physical characteristics. Patients, methods: In a prospective study, 42 patients in clinical stage C1–4 were examined with dynamic mercury strain gauge plethysmography to determine the effects of 13 different compression stockings in compression classes 2 and 3 (CEN) on venous haemodynamics. At the same time that venous function measurement was monitored, the pressure exerted by the compression stockings was measured under resting conditions and during standardized exercises by the patients. Results: Rest
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Chenzbraun, Adrian. "Non-ischaemic cardiac conditions: role of stress echocardiography." Echo Research and Practice 1, no. 1 (2014): R1—R7. http://dx.doi.org/10.1530/erp-14-0030.

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Stress echocardiography (SE) has a unique ability for simultaneous assessment of both functional class and exercise-related haemodynamic changes and as such is increasingly recognised for the evaluation of non-coronary artery disease pathologies. Some indications such as valvular heart disease or hypertrophic cardiomyopathy have been well established already, while others such as diastolic exercise testing are emerging of late. This paper addresses the main and best established indications for SE in non-ischaemic conditions, providing a practical perspective correlated with updated guidelines.
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Dissertations / Theses on the topic "Haemodynamic conditions"

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Goodson, Robert Andrew Hawksley. "Analysis of growth and rupture of fusiform abdominal aortic aneurysms." Thesis, Nottingham Trent University, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341287.

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McElroy, Michael. "Boundary condition assessment and geometrical accuracy enhancement for computational haemodynamics." Thesis, Manchester Metropolitan University, 2017. http://e-space.mmu.ac.uk/619019/.

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Cardiovascular diseases cause over 47 % of all deaths in Europe each year. Computational fluid dynamics provides the research community with a unique opportunity to investigate cardiovascular diseases with the intent of enabling optimised, patient-specific medical therapies. Incorporating physiologically accurate geometries and boundary conditions into computational fluid dynamics simulations can be difficult tasks and are a concern for researchers. This thesis analyses the impact various inlet and outlet boundary conditions can have on the outcome of a simulation. It also presents a novel, se
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Books on the topic "Haemodynamic conditions"

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Galiè, Nazzareno, Alessandra Manes, and Massimiliano Palazzini. Pulmonary hypertension. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0065.

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Pulmonary hypertension is a haemodynamic and pathophysiological condition defined as an increase in the mean pulmonary arterial pressure of ≥25 mmHg at rest, as assessed by right heart catheterization. In fact, while transthoracic echocardiography may provide clues on the presence of pulmonary hypertension, the haemodynamic evaluation offers a more precise and comprehensive assessment. Pulmonary hypertension is heterogeneous from a pathophysiological point of view, and the diversity is reflected in the haemodynamic definitions. The different haemodynamic forms of pulmonary hypertension can be
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Lancellotti, Patrizio, and Bernard Cosyns. Systemic Disease and Other Conditions. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0017.

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This chapter describes the effect of various activities on the heart and associated disorders. It details the echocardiographic findings of athlete’s heart and differential diagnosis. It considers pregnancy which induces several haemodynamic changes: increase in heart rate, stroke volume, cardiac output, and decrease in systemic vascular resistance. Several echocardiographic changes may also present in normal pregnancy and these must be recognized. Echocardiography should be performed in each pregnant woman with cardiac signs or symptoms to search for new cardiac disease occurring during pregn
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Pasquet, Agnes, Marcia Barbosa, and Jo-Nan Liao. Systemic disease and other conditions: the heart during pregnancy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0056.

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Normal pregnancy represents a haemodynamic challenge for the heart. The main changes are increases in blood (plasma) volume and cardiac output, and a decrease in systemic vascular resistance. These change start early during pregnancy and will be maximal around the 24th week of gestation. This translates into echocardiographic changes such as increase in ventricular volume, stroke volume, and changes in geometry. Peripartum cardiomyopathy is a left ventricular dysfunction without any underlying cause, arising near the end of the gestation or in the early postpartum period. Echocardiography is t
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D’Andrea, Antonello, André La Gerche, and Christine Selton-Suty. Systemic disease and other conditions: athlete’s heart. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0055.

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The term ‘athlete’s heart’ refers to the structural, functional, and electrical adaptations that occur as a result of habitual exercise training. It is characterized by an increase of the internal chamber dimensions and wall thickness of both atria and ventricles. The athlete’s right ventricle also undergoes structural, functional, and electrical remodelling as a result of intense exercise training. Some research suggests that the haemodynamic stress of intense exercise is greater for the right heart and, as a result, right heart remodelling is slightly more profound when compared with the lef
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Vieillard-Baron, Antoine. Right ventricular function in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0135.

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Under normal conditions, the right ventricle (RV) virtually acts as a passive conduit. In critically-ill patients many situations induce uncoupling between the right ventricle and pulmonary circulation, leading to RV systolic dysfunction, then failure. Mechanical ventilation has a major impact by decreasing RV preload, but also significantly increasing RV afterload. RV function should thus always be interpreted and re-evaluated in the light of respiratory mechanics and ventilator settings. RV systolic function is key to the patient’s haemodynamic profile and must be monitored to achieve optima
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Leach, Dr Richard, and Professor Kevin Moore. Practical procedures and monitoring. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.00019.

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Chapter 19 covers practical procedures and monitoring of patients presenting with vascular and haemodynamic, respiratory, gastrointestinal, and other conditions, as well as additional various practical procedures.
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Schairer, John R., and Steven J. Keteyian. Pathophysiology and causes of pericardial tamponade. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0166.

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Pericardial disease leading to pericardial effusion (PEF) is a common clinical disorder. The most common causes are viral infections, metastatic cancer, renal disease, and bleeding disorders. PEF that accumulates slowly can become quite large before haemodynamic embarrassment occurs, while PEF that accumulates rapidly from trauma or aortic dissection can be small,yet cause haemodynamic embarrassment. As the PEF increases in size, the pressure in the pericardial space increases, leading to a decrease in atrial and ventricular chamber sizes, and limiting filling of the chambers. Ultimately, card
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Paul, Richard, and Susanna Price. Imaging the cardiovascular system in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0143.

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Cardiac imaging in the critically ill can be challenging. Interpretation requires a broad knowledge of cardiovascular pathophysiology, the range of available investigations, and their sensitivity and specificity in diagnosing individual conditions. Applying first principles and interpreting findings in the clinical context are mandatory. Useful non-invasive investigations include simple chest X-ray, thoracic ultrasound, and computed tomography (CT) to detect pulmonary and extrapulmonary pathology, whilst CT coronary angiography can evaluate stent and graft patency, and identify extramural plaq
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Orenbuch-Harroch, Efrat, and Charles L. Sprung. Pulmonary artery catheterization in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0133.

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Haemodynamic monitoring is a significant component in the management of critically-ill patients. Flow-directed pulmonary artery catheters (PAC) are a simple and rapid technique for measuring several continuous or intermittent circulatory variables. The PAC is helpful in diagnosis, guidance of therapy, and monitoring therapeutic interventions in various clinical conditions, including myocardial infarction and its complications, non-cardiogenic pulmonary oedema and severely ill patients.The catheter is inserted through a large vein. The PAC is advanced, after ballooninflation with 1.5 mL of air,
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Badano, Luigi P., and Denisa Muraru. Assessment of right heart function and haemodynamics. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0011.

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Assessment of right ventricular (RV) size, function, and haemodynamics has been challenging because of its unique cavity geometry. Conventional two-dimensional assessment of RV function is often qualitative. Doppler methods involving tricuspid inflow and pulmonary artery flow velocities, which are influenced by changes in pre- and afterload conditions, may not provide robust prognostic information for clinical decision making. Recent advances in echocardiographic assessment of the RV include tissue Doppler imaging, speckle-tracking imaging, and volumetric three-dimensional imaging, but they ne
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Book chapters on the topic "Haemodynamic conditions"

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Lawson, Charlotte, Marlene Rose, and Sabine Wolf. "Leucocyte Adhesion Under Haemodynamic Flow Conditions." In Methods in Molecular Biology. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-461-6_3.

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Revell, W. J., and M. Brookes. "Arteriolar Blockade Revisited: Comparisons Between the Use of Resin Particles and Microspheres for Bone Haemodynamic Studies." In Bone Circulation and Vascularization in Normal and Pathological Conditions. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2838-8_10.

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Sauberer, Andrea, Thomas Publig, and Ingrid Schindler. "Intubating Conditions and Haemodynamic Effects of Rapid Sequence Induction with Fentanyl, Propofol and Rocuronium." In Muscle Relaxants. Springer Japan, 1995. http://dx.doi.org/10.1007/978-4-431-66896-1_113.

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Brueton, R. N., W. J. Revell, and M. Brookes. "Haemodynamics of Bone Healing in a Model Stable Fracture." In Bone Circulation and Vascularization in Normal and Pathological Conditions. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2838-8_15.

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Formaggia, Luca, and Christian Vergara. "Defective Boundary Conditions for PDEs with Applications in Haemodynamics." In Numerical Methods for PDEs. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-94676-4_10.

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Przywara-Chowaniec, Brygida, Lech Poloński, Maciej Gawlikowski, and Tadeusz Pustelny. "Research into the Possibility to Use Impedance Rheocardiography in a Non-invasive Assessment of Haemodynamic Condition of Patients with Heart Diseases." In Information Technologies in Biomedicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-31196-3_18.

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Evans, T. J. "The Role of Nitric Oxide in Septic Shock and Other Inflammatory Conditions." In The Haemodynamic Effects of Nitric Oxide. PUBLISHED BY IMPERIAL COLLEGE PRESS AND DISTRIBUTED BY WORLD SCIENTIFIC PUBLISHING CO., 1999. http://dx.doi.org/10.1142/9781848160781_0022.

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Palazzini, Massimiliano, Nazzareno Galiè, and Alessandra Manes. "Pulmonary hypertension." In The ESC Textbook of Intensive and Acute Cardiovascular Care, edited by Marco Tubaro, Pascal Vranckx, Eric Bonnefoy-Cudraz, Susanna Price, and Christiaan Vrints. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198849346.003.0063.

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Pulmonary hypertension is a haemodynamic and pathophysiological condition defined as an increase in the mean pulmonary arterial pressure of ?25 mmHg at rest, as assessed by right heart catheterization. Recently, a new definition has been proposed as mean pulmonary arterial pressure >20 mmHg combined with pulmonary vascular resistance ? 3 Wood units. While transthoracic echocardiography may provide clues on the presence of pulmonary hypertension, the haemodynamic evaluation offers a more precise and comprehensive assessment. Pulmonary hypertension is heterogeneous from a pathophysiological point of view, and the diversity is reflected in the haemodynamic definitions. The different haemodynamic and clinical forms of pulmonary hypertension can be found in multiple clinical conditions which have been classified into five main groups and at least twenty-six subgroups. Each main clinical group shows specific pathological changes in the lung distal arteries, capillaries, and small veins. If we combine the haemodynamic and clinical heterogeneity, we understand the complexity of an accurate diagnosis in the individual patient which is crucial for the prognostic assessment and treatment strategy. In addition, the concomitant presence of different haemodynamic and clinical mechanisms cannot be excluded in individual cases (e.g. in patients with congestive heart failure and associated lung diseases). The presence of pulmonary hypertension, as defined above, is always an ominous prognostic sign, even if the severity may differ according to the haemodynamic changes and underlying clinical condition. The therapeutic approach also is markedly different, according to the clinical group, and symptomatic and haemodynamic severity. For these reasons, the four more frequent clinical groups are discussed individually, while the classifications are described in the Introduction section.
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Oleas, Francisca A. Gavilanes, Caio J. C. Fernandes, and Rogério Souza. "Pulmonary hypertension: clinical classification." In ESC CardioMed, edited by Marc Humbert. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0581.

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Pulmonary hypertension is a clinical and haemodynamic condition associated with several baseline clinical conditions or existing as a pure pulmonary vascular disease. The current classification groups aetiologies of pulmonary hypertension that share similarities in terms of clinical presentation, pathological findings, and haemodynamic profiles, in an attempt to guide management strategies in these distinct conditions. As a consequence, pulmonary hypertension is classified into five different groups: pulmonary arterial hypertension, pulmonary hypertension due to left heart disease, pulmonary hypertension due to lung disease and/or hypoxia, chronic thromboembolic pulmonary hypertension, and pulmonary hypertension with unclear multifactorial mechanisms. This chapter reviews the rational supporting the current classification system alluding also to the existing evidence for specific therapies in each one of the groups.
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Frise, Charlotte, and Sally Collins. "Cardiology." In Obstetric Medicine. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198821540.003.0002.

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Pregnancy is associated with global haemodynamic changes, which occur early and gradually return to normal after delivery. This chapter covers the changes in physiological cardiac changes, before covering over 40 different cardiac conditions and syndromes, including inherited defects, problems arising from prosthetic heart valves, aortic pathologies, cardiomyopathies, as well as cardiac problems specific to the pregnant patient.
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Conference papers on the topic "Haemodynamic conditions"

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Albu, Gergely, Cédric Sottas, Magali Walesa, Ferenc Peták, Walid Habre, and Mirko Dolci. "Temporal changes in lung function following haemodilution under stable haemodynamic conditions in pigs." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2267.

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Boutsianis, Evangelos, Thomas Frauenfelder, Hitendu Dave, et al. "Cardiovascular Haemodynamic Simulations of Anatomically Accurate Coronaries." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-42728.

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The present study is devoted to the investigation of the pulsatile blood flow within the first few vessels of the Left Coronary Artery (LCA) vasculature of an anatomically accurate porcine coronary tree. Transient computational fluid dynamics simulations were performed under realistic pulsatile volume inflow boundary conditions. The numerical results have provided a comprehensive collection of information regarding the haemodynamics within the LCA and its major branches, namely the Left Anterior Descending (LAD) and the Left Circumflex (LCX) arteries. The underlying principle of developing com
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Artmann, G., R. Grebe, H. Wolff, R. Degenhardt, and H. Schmid-SchÖnbein. "NOVEL TECHIQUES FOR QUANTIFICATION OF RBC-SHAPE (RS) AND SHEAR INDUCED RBC ELONGATION (SIRE): APPLICATION FOR ANALYSIS OF DRUG INDUCED ALTERATIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644217.

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In the past, red cell resting shape could only be assessed by subjective scaling, red cell deformability by a variety of rheological tests that are extremelydifficult to standardize and which all subject the RBC to high deforming forces. None of the latter have been accepted as reference in haematology, haemorheologyor pharmacology. A recent development from our group now allows objective, numerical analysis of red cell membrane curvature (i.e. the echinocytic or stomatocytic deviation from the discocytic resting shape) by a tangent count procedure in optical sections through freely suspended,
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Johnston, Lauren, Ruth Allen, Pauline Hall-Barrientos, Avril Mason, and Asimina Kazakidi. "P13 Computational haemodynamics in turner syndrome patient-specific aortae with PC-MRI obtained boundary conditions." In Scottish Cardiovascular Forum – 23rd annual meeting, University of Strathclyde, Saturday 1st February 2020. BMJ Publishing Group Ltd and British Cardiovascular Society, 2020. http://dx.doi.org/10.1136/heartjnl-2020-scf.23.

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Lowe, O. DG. "RHEOLOGY AND VENOUS THROMBOEMBOLISM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643990.

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Changes in the composition of the blood, venous stasis, and interaction of the blood with the vessel wall (Virchow's triad) all have rheological aspects which may promote venous thrombogenesis.Blood composition and rheology. Increasing levels of venous haematocrit and fibrinogen increase bulk blood viscosity, especially at low shear rates such as are encountered in veins, when red cell aggregation occurs. Static blood requires a minimum shear stress for flow (yield stress), which is also strongly dependent on haematocrit and fibrinogen levels. Increases in haematocrit and fibrinogen also promo
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Morbiducci, Umberto, Raffaele Ponzini, Matteo Nobili, et al. "Prediction of Shear Induced Platelet Activation in Prosthetic Heart Valves by Integrating Fluid–Structure Interaction Approach and Lagrangian-Based Blood Damage Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206162.

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Altered haemodynamics are implicated in the blood cells damage that leads to thromboembolic complications in presence of prosthetic cardiovascular devices, with platelet activation being the underlying mechanism for cardioemboli formation in blood flow past mechanical heart valves (MHVs). Platelet activation can be initiated and maintained by flow patterns arising from blood flowing through the MHV, and can lead to an enhancement in the aggregation of platelets, increasing the risk for thromboemboli formation. Hellums and colleagues compiled numerous experimental results to depict a locus of i
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Gallo, Diego, Raffaele Ponzini, Filippo Consolo, et al. "A Numerical Multiscale Study of the Haemodynamics in an Image-Based Model of Human Carotid Artery Bifurcation." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206159.

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The initiation and progression of vessel wall pathologies have been linked to disturbances of blood flow and altered wall shear stress. The development of computational techniques in fluid dynamics, together with the increasing performances of hardware and software allow to routinely solve problems on a virtual environment, helping to understand the role of biomechanics factors in the healthy and diseased cardiovascular system and to reveal the interplay of biology and local fluid dynamics nearly intractable in the past, opening to detailed investigation of parameters affecting disease progres
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Le Gouez, J. M. "Numerical Simulation of Non Newtonian Hemodynamics in Compliant Vessels." In ASME 2006 Pressure Vessels and Piping/ICPVT-11 Conference. ASMEDC, 2006. http://dx.doi.org/10.1115/pvp2006-icpvt-11-93801.

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The numerical simulation of hemodynamics is increasingly recognized as a valuable analysis tool for the bioengineering laboratories who design implantable vascular grafts, and it is thought to become in a not so far future a complement to the physician’s analysis for the choice of interventional methods to restore a proper irrigation in diseased arteries [1]. The detailed numerical results obtained from 3d unsteady simulations permit to verify the hypotheses formulated by physiologists concerning the evolution of arterial disease and to quantify the risks associated to medical intervention. Th
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