Relevant bibliographies by topics / Helicobacter pylori etiology

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Helicobacter pylori etiology'

Author: Grafiati
Published: 28 May 2022
Last updated: 31 July 2025

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Journal articles on the topic "Helicobacter pylori etiology"

1

Alempijevic, Tamara, Miodrag Krstic, Vesna Antonic, and Slobodan Krstic. "Frequency of Helicobacter pylori infection in patients with liver cirrhosis." Srpski arhiv za celokupno lekarstvo 135, no. 9-10 (2007): 536–40. http://dx.doi.org/10.2298/sarh0710536a.

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Introduction Ulcer disease occurrence is increased among patients with liver cirrhosis. It is widely accepted that Helicobacter pylori infection is important in etiology of ulcer disease. The role of Helicobacter pylori infection in pathogenesis of ulcer disease in patients with liver cirrhosis has not been satisfactorily explored yet. Objective The aim of this study was to determine frequencies of Helicobacter pylori infection in patients with liver cirrhosis in relation to the etiology of cirrhosis, clinical stage and concomitant ulcer disease. Method We studied 50 patients with liver cirrho
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Miljkovic-Selimovic, Biljana, Branislava Kocic, and Tatjana Babic. "Campylobacter and helicobacter in the etiology of gastrointestinal diseases." Archives of Biological Sciences 64, no. 4 (2012): 1389–404. http://dx.doi.org/10.2298/abs1204389s.

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The order Campylobacterales comprises two genera: Campylobacter and Helicobacter, with a widespread distribution in both humans and animals. They are Gram-negative, spiral, helical and microaerophilic bacteria, with an optimal growth temperature of 37?C for H. pylori and 42?C for C. jejuni strains. While Helicobacter pylori are restricted to humans, other helicobacter species can be found in different mammals and occasionally in humans. Several Campylobacter species are recognized as human pathogens, while distinct species are pathogenic only occasionally, in children, the elderly and immunoco
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Herszényi, László, Márk Juhász, Emese Mihály, and Zsolt Tulassay. "A fekélybetegség és a stressz." Orvosi Hetilap 156, no. 35 (2015): 1426–29. http://dx.doi.org/10.1556/650.2015.30249.

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The discovery that Helicobacter pylori infection is the major cause of peptic ulcer disease revolutionised our views on the etiology and treatment of the disease. This discovery has tempted many experts to conclude that psychological factors and, specifically, stress are unimportant. However, Helicobacter pylori infection alone does not explain fully the incidence and prevalence of peptic ulcer disease. It has been demonstrated that stress can cause peptic ulcer disease even in the absence of Helicobacter pylori infection, supporting a multicausal model of peptic ulcer etiology. Psychological
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Urazova, R. Z. "Treatment of inflammatory periodontal diseases in children infected with Helicobacter pylori." Kazan medical journal 82, no. 3 (2001): 208–9. http://dx.doi.org/10.17816/kazmj72140.

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Microorganisms, including Helicobacter pylori, play an important role in the etiology of inflammatory periodontal diseases. The aim of our study was to study the effectiveness of topical use of metronidazole on the mucous membranes of the oral cavity in combination with antihelicobacter therapy for Helicobacter pylori infection in childhood.
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KHADEMI, BIJAN, Negar Azar Pira, Mohammad Javad Ashraf, and Abdul Hameed Chohedri. "HELICOBACTER PYLORI IN NASAL POLYPOSIS." Professional Medical Journal 19, no. 04 (2012): 455–61. http://dx.doi.org/10.29309/tpmj/2012.19.04.2256.

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Background: Nasal polyposis is an inflammatory condition of unknown etiology. Recently concern regarding GER orHelicobacter pylori as a possible pathologic cause of nasal polyps has been increasing. The present study was planned to investigate thepresence of Helicobacter Pylor in Nassal polyps by PCR , rapid Urease test and serology. Design: Case control study. Setting: ENT ward ofShiraz, Khalilli Hospital, Iran. Period: April 2006 to March 2008. Materials and Methods: 37 patients with nasal polyps who had undergonenasal endoscopic sinus surgery and 38 control subjects who had undergone septop
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Harahap, Rini Syahrani, Delyuzar, and Jessy Chrestella. "The Presence of Helicobacter pylori in cases of Chronic Tonsillitis in Medan." Sumatera Medical Journal 3, no. 1 (2020): 1–6. http://dx.doi.org/10.32734/sumej.v3i1.2044.

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Background: Helicobacter pylori is a well known pathogen which is recognized as the most frequent infection of individuals. Helicobacter pylori plays important role in the etiology og gastritis, gastric and duodenal ulcers. We investigated the presence of Helicobacter pylori as an extragastric reservoir in the tonsilectomy specimens.
 Aim: To determine the presence of Helicobacter pylori in cases of chronic tonsillitis in tonsilectomy specimens.
 Methods: This is a descriptive study with cross sectional which the sample will be obtained using consecutive sampling method. We collect 4
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7

Subedi, Ram Chandra, Binit Upadhaya Regmi, Bishnu Deep Pathak, et al. "Prevalence of Helicobacter pylori infection in dyspeptic patients presenting to a tertiary care center of a developing country: a cross-sectional study." F1000Research 12 (September 25, 2023): 1195. http://dx.doi.org/10.12688/f1000research.137429.1.

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Background: Among the various causes for dyspepsia in patients presenting to the gastroenterology department, Helicobacter pylori is the most common etiological agents. Here, we explore the prevalence of Helicobacter pylori infection, particularly, in dyspeptic patients and different endoscopic findings in such patients. Methods: We conducted a simple observational study in the gastroenterology department of a tertiary care center of Nepal. The Helicobacter pylori was detected using rapid urease test and endoscopic findings were collected from gastroduodenoscopy unit. The obtained data was ana
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Ghelmici, T., Iu Lupasco, V.-T. Dumbrava, and Gh Harea. "OXIDATIVE STRESS IN PATIENTS WITH CHRONIC VIRAL HEPATITIS C WITH DUODENAL ZONE ALTERATION OF HELICOBACTER PYLORI ETIOLOGY." Biological Markers in Fundamental and Clinical Medicine (collection of abstracts) 3, no. 1 (2019): 40–42. http://dx.doi.org/10.29256/v.03.01.2019.escbm24.

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9

Hardikar, Winita, Colin Feekery, Arnold Smith, Frank Oberklaid, and Keith Grimwood. "Helicobacter pylori and Recurrent Abdominal Pain in Children." Journal of Pediatric Gastroenterology and Nutrition 22, no. 2 (1996): 148–52. http://dx.doi.org/10.1002/j.1536-4801.1996.tb00063.x.

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SummaryRecurrent abdominal pain is one of the most common presentations to pediatricians; yet an organic etiology can be found in only 10% of cases. Because infection with Helicobacter pylori in adults and children results in gastritis, a causative role for the organism has been postulated. To investigate this theory, we conducted a prospective case‐control study in children with recurrent abdominal pain using serum H. pylori IgG antibodies measured by an enzyme immunoabsorbent assay. Age, sex, ethnicity, and socioeconomic status were adjusted in the statistical model. Five subjects (5.1%) and
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Zelickson, Marc S., Cathy M. Bronder, Brent L. Johnson, et al. "Helicobacter Pylori Is Not the Predominant Etiology for Peptic Ulcers Requiring Operation." American Surgeon 77, no. 8 (2011): 1054–60. http://dx.doi.org/10.1177/000313481107700827.

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As the number of patients requiring operation for peptic ulcer disease (PUD) declines, presumed contemporary ulcer etiology has largely been derived from medically treated patients not subjected to surgery. The purpose of this study was to examine the specific causes of PUD in patients requiring surgery. Our Acute Care Surgical Service registry was reviewed for patients operated on for complications of PUD from 2004 to 2009. Emphasis was placed on individual etiologic factors for PUD. There were 128 patients (52% male, 81% white) who underwent emergency operation including: simple patch closur
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Dissertations / Theses on the topic "Helicobacter pylori etiology"

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Batista, Carlos Alexandre Gonçalves. ""Estudo clínico e endoscópico em pacientes com úlcera péptica gastroduodenal após 1 ano de erradicação do Helicobater pylori. Avaliação da relação entre o surgimento da esofagite erosiva e a cepa do Helicobacter pylori erradicado"." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/5/5147/tde-20092006-134022/.

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Atualmente, muitas são as diretrizes na literatura quanto à influência do Helicobacter pylori na Doença do Refluxo Gastroesofágico. Alguns autores acreditam que o H. pylori poderia ter um efeito protetor para o desenvolvimento na DRGE e outros até mesmo concluem que o agente possa ser um fator agravante na doença. Muitas publicações nos alertam para o desenvolvimento de sintomas da DRGE, ou mesmo da esofagite, em uma porcentagem razoável de pacientes erradicados pelo esquema tríplice para tratar o H. pylori, sendo que aproximadamente 10% teriam DRGE. Na verdade, por essas dúvidas, ainda não fo
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徐維勝 and Wei-sheng Xu. "Gastric lymphoma of MALT type: the etiologic factors and the molecular basis of pathogenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31239572.

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Орловський, Олександр Вікторович, Александр Викторович Орловский та Oleksandr Viktorovych Orlovskyi. "Апоптоз епітеліоцитів шлунка у хворих на виразкову хворобу дванадцятипалої кишки (ВХДПК) хелікобактерної етіології". Thesis, Видавництво СумДУ, 2003. http://essuir.sumdu.edu.ua/handle/123456789/9059.

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Pereira-Lima, Júlio Carlos. "Gastrite associada ao helicobacter pylori e dispepsia nao-ulcerosa : uma contribuicao ao seu estudo." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 1993. http://hdl.handle.net/10183/139224.

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Xu, Wei-sheng. "Gastric lymphoma of MALT type : the etiologic factors and the molecular basis of pathogenesis /." Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B20793133.

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Bonamigo, Renan Rangel. "Analise sorologica para helicobacter pylori em amostra de pacientes com rosacea : um estudo de casos e controles." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 1998. http://hdl.handle.net/10183/164179.

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Fundamentos: a rosácea é uma dermatose freqüente que possui multiplicidade de fatores envolvidos no seu desencadeamento. Na literatura recente, há autores que apontam o Helicobacter pylori como um possível agente etiológico da doença. Porém os poucos estudos publicados sobre o assunto que utilizaram pacientes controles não encontraram evidências de que esta associação seja importante. No Brasil, não há trabalhos publicados quanto à exposição ao Helicobacter pylori em pacientes com rosácea, e mesmo o perfil geral dos pacientes com rosácea, no país, é pouco conhecido. Objetivos: o principal obje
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"Role of helicobacter pylori catalysed N-nitrosation in gastric carcinogenesis." 2002. http://library.cuhk.edu.hk/record=b6073441.

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by Chan Chi Wai, Michael.<br>"July 2002."<br>Thesis (Ph.D.)--Chinese University of Hong Kong, 2002.<br>Includes bibliographical references (p. 238-272).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Electronic reproduction. Ann Arbor, MI : ProQuest Information and Learning Company, [200-] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Mode of access: World Wide Web.<br>Abstracts in English and Chinese.
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"The role of helicobacter pylori-related gastritis in pathogenesis of gastroesophageal reflux disease." 2000. http://library.cuhk.edu.hk/record=b6073292.

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by Wu Che-yuen Justin.<br>"September 2000 (amendment)."<br>Thesis (M.D.)--Chinese University of Hong Kong, 2000.<br>Includes bibliographical references (p. 237-267).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Mode of access: World Wide Web.
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Books on the topic "Helicobacter pylori etiology"

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IARC Working Group on the Evaluation of Carcinogenic Risks to Humans (1994 Lyon, France). Schistosomes, liver flukes and Helicobacter pylori. The Agency, 1994.

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Workshop of Gastroduodenal Pathology and Helicobacter pylori (5th 1992 Dublin, Ireland). Helicobacter pylori: Implications and practice : Vth Workshop of Gastroduodenal Pathology adn Helicobacter pylori, Trinity College, Dublin, Ireland. Normed Verlag, 1994.

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International Symposium on Helicobacter Pylori (2nd 1989 Bad Nauheim, Germany). Helicobacter pylori 1990: Proceedings of the Second International Symposium on Helicobacter Pylori, Bad Nauheim, August 25-26th, 1989. Springer-Verlag, 1991.

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1939-, Menge H., ed. Helicobacter pylori 1991: Proceedings of the Second International Symposium on Helicobacter pylori, Bad Nauheim, August 25-26th, 1989. Springer-Verlag, 1991.

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Falk Symposium (81st 1994 Berlin, Germany). Malignancy and chronic inflammation in the gastrointestinal tract: New concepts : proceedings of the 81st Falk Symposium, held in Berlin, Germany, November 3-5, 1994. Kluwer Academic Publishers, 1995.

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Helicobacter pylori: Implications and practice : Vth Workshop of Gastroduodenal Pathology adn Helicobacter pylori, Trinity College, Dublin, Ireland. Normed Verlag, 1994.

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Menge, H., Guido N. J. Tytgat, Gregor M, Barry J. Marshall, and C. A. M. McNulty. Helicobacter Pylori 1990: Proceedings of the Second International Symposium on Helicobacter Pylori Bad Nauheim, August 25 26th, 1989. Springer, 1991.

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(Editor), Francis Megraud, and Herve Lamouliatte (Editor), eds. Gastroduodenal Pathology and Campylobacter Pylori (International congress series). Elsevier, 1989.

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The great ulcer war: How an axis of an American university research group and a global pharmaceutical company attacked one of the great medical discoveries of the twentieth century. William S. Hughes, 2014.

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Thagard, Paul. How Scientists Explain Disease. Princeton University Press, 2000.

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Book chapters on the topic "Helicobacter pylori etiology"

1

Bello Kumo, Ahmad. "Gastric Cancer: Diet and Helicobacter Pylori as Major Modifiable Risk Factors." In Gastric Cancer - Progress and Challenges in the Era of Precision Medicine [Working Title]. IntechOpen, 2024. http://dx.doi.org/10.5772/intechopen.1004434.

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Gastric cancer is ranked as the sixth cancer worldwide and the fourth leading cause of cancer-related deaths. There exists marked geographic variation in the incidence of gastric cancer the world over, with the highest rates reported in Japan, Korea, and Eastern Europe. The gastric cancer highest incidence regions of the world are Eastern Asia, Europe, Central and South America, while North America, Australia, and Africa are considered low incidence areas. Sex differences exist in gastric cancer incidence, which is almost two-fold higher in males than females. There is a declining incidence an
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Ávila Godoy, Kelin Janeth, Luis José Ramírez Osorto, Fernando Javier Caceres Carranza, et al. "Ulcera Peptica." In Manual esencial de fisiopatología y farmacología. Tomo I Autores/as. Religación Press, 2024. http://dx.doi.org/10.46652/religacionpress.177.c218.

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La enfermedad ulcerosa péptica es un trastorno inflamatorio crónico de la mucosa gástrica o duodenal. Esta condición ocurre generalmente por un aumento en los mecanismos agresores o disminución de los mecanismos protectores. Helicobacter pylori es la responsable de hasta 80% de los casos, seguido por el consumo crónico de fármacos antiinflamatorios no esteroideos (AINES). La principal manifestación de una úlcera péptica es el dolor epigástrico de tipo urente. Otros síntomas asociados son: náuseas, vómitos, anorexia y modificaciones del peso corporal. La úlcera puede complicarse y generar hemor
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Conference papers on the topic "Helicobacter pylori etiology"

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RODRIGUES, F. A., T. S. LIMA, T. Z. BARRESE, and C. S. CIRQUEIRA. "A AVALIAÇÃO DE COLORAÇÕES HISTOQUÍMICAS NA PESQUISA HISTOPATOLÓGICA DE Helicobacter pylori." In III Mostra Dos Trabalhos De Conclusão De Curso Da Especialização Em Vigilância Laboratorial Em Saúde Pública. Agron Science, 2022. http://dx.doi.org/10.53934/101031-2.

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Estima-se que metade da população mundial apresente a mucosa gástrica colonizada por Helicobacter pylori, resultando em maiores riscos para o desenvolvimento de gastrite crônica, úlceras peptídicas, adenocarcinoma gástrico etc. A determinação histológica em lesões suspeitas constitui-se como uma importante ferramenta de diagnóstico de doenças crônicas gástricas de etiologia infecciosa. O objetivo do estudo consistiu em avaliar o desempenho das colorações histoquímicas Giemsa modificado e Warthin-Starry em biópsias gástricas, fixadas em formalina e incluídas em parafina, com detecção imuno-hist
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Pires, Ana Luíza Guedes, Débora Rodrigues Martins, and Rodrigo de Oliveira Peixoto. "Hiperplasia de glândulas de brunner: Relato de caso e revisão de literatura." In I SEVEN INTERNATIONAL EDUCATION CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/ieducationcongress-032.

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As neoplasias benignas do duodeno são raras, aproximadamente 0,01%. Dentre essas lesões, a Hiperplasia das Glândulas de Brunner e o Hamartoma das Glândulas de Brunner são importantes diagnósticos diferenciais no que diz respeito às massas encontradas no duodeno, correspondendo a cerca de 5% desses achados. As glândulas de Brunner são estruturas mucosas e submucosas encontradas, principalmente, no bulbo duodenal e têm ação digestiva ainda indefinida, com possível papel protetor contra acidez gástrica. As lesões culminam na proliferação excessiva dessas estruturas, cuja etiologi
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