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Dissertations / Theses on the topic 'Helicobacter pylori. Helicobacter pylori infections'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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1

Kivi, Mårten. "Aspects of Helicobacter pylori transmission /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-422-8/.

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2

Garrett, June Kazumi. "Inhibition of Helicobacter pylori by Wild Blueberry Phenolics." Fogler Library, University of Maine, 2009. http://www.library.umaine.edu/theses/pdf/GarrettJK2009.pdf.

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3

Houben, Martinus Henricus Maria Gerardus. "Clinical aspects in Helicobacter pylori infections." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2000. http://dare.uva.nl/document/81155.

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4

Rader, Bethany Anne. "Autoinducer-2 regulation of motility in Helicobacter pylori /." view abstract or download file of text, 2006. http://proquest.umi.com/pqdweb?did=1251819321&sid=6&Fmt=2&clientId=11238&RQT=309&VName=PQD.

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Thesis (Ph. D.)--University of Oregon, 2006.<br>Typescript. Includes vita and abstract. Includes bibliographical references (leaves 80 - 90). Also available for download via the World Wide Web; free to University of Oregon users.

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5

Wanken, Amy Elizabeth. "Helicobacter pylori colonization of the mouse gastric mucosa the Entner-Doudoroff pathway and development of a promoter-trapping system /." Columbus, Ohio : Ohio State University, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1059079727.

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Thesis (Ph. D.)--Ohio State University, 2003.<br>Title from first page of PDF file. Document formatted into pages; contains xiii, 145 p.; also includes graphics (some col.). Includes abstract and vita. Advisor: Kathryn Eaton, Dept. of Veterinary Biosciences. Includes bibliographical references (p. 130-145).

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6

Ben, Cheikh M'Hamed Laurence De Korwin Jean-Dominique. "Diagnostic en médecine générale." [S.l] : [s.n], 2003. http://www.scd.uhp-nancy.fr/docnum/SCDMED_T_2003_GONDA_BEN_CHEIKH_M_HAMED_LAURENCE.pdf.

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7

Unge, Peter. "Pharmacological therapy of Helicobacter pylori infection /." Linköping : Univ, 2002. http://www.bibl.liu.se/liupubl/disp/disp2002/med734s.pdf.

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8

Wong, Wai-man Raymond, and 王衛民. "The pathology, diagnosis and treatment of helicobacter pylori infection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B23331707.

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9

Olfat, Farzad O. "Helicobacter pylori - bacterial adhesion and host response /." Umeå : Univ, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-133.

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10

Boublil, Laurence Yae͏̈l. "Maladie ulcéreuse : physiopathologie, traitements ; rôle d'hélicobacter pylori." Paris 5, 1998. http://www.theses.fr/1998PA05P006.

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11

Chu, Kent-man. "Helicobacter pylori infection and gastroduodenal ulcer disease." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23636555.

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12

Thomas, Julian Edward. "Helicobacter pylori infection in childhood." Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.300216.

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13

Wong, Chun-yu Benjamin. "Helicobacter pylori : related diseases in the Chinese /." Hong Kong : University of Hong Kong, 2000. http://sunzi.lib.hku.hk/hkuto/record.jsp?B22226977.

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14

朱建民 and Kent-man Chu. "Helicobacter pylori infection and gastroduodenal ulcer disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B23685426.

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15

Wheeldon, Thục-Uyên. "Management of Helicobacter pylori infection in Vietnam /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-876-9/.

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16

Warren, John Robin. "The discovery and pathology of H pylori /." Title page and contents only, 1999. http://web4.library.adelaide.edu.au/theses/09MD/09mdw289.pdf.

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17

Rigot, Agnès. "Hélicobacter pylori et gastrites chez l'enfant : à propos de 14 observations." Bordeaux 2, 1991. http://www.theses.fr/1991BOR23113.

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18

Nilsson, Christina. "Genome-plasticity and adaptation in Helicobacter pylori /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-189-X/.

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19

Outlioua, Ahmed. "Exploration des cytokines pro-inflammatoires et de l’inflammasome NLRP3 dans les infections intracellulaires : cas de H. pylori et des virus à ARN Gastric IL-1β, IL-8, and IL-17A expression in Moroccan patients infected with Helicobacter pylori may be a predictive signature of severe pathological stages RNA viruses promote activation of the NLRP3 inflammasome through cytopathogenic effect-induced potassium efflux The heme-regulated inhibitor is a cytosolic sensor of protein misfolding that controls innate immune signaling The Role of Optineurin in Antiviral Type I Interferon Production Possible introduction of Leishmania tropica to urban areas determined by epidemiological and clinical profiles of patients with cutaneous leishmaniasis in Casablanca (Morocco)". Thesis, université Paris-Saclay, 2021. http://www.theses.fr/2021UPASL029.

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Helicobacter pylori (H. pylori) est une bactérie qui infecte l’estomac et induit une gastrite inflammatoire, qui peut être chronique et évoluer vers un cancer gastrique. La sévérité de l’infection et son évolution clinique sont associées aux différents facteurs notamment le statut immunitaire de l’hôte. La réponse inflammatoire initiale à l'infection à H. pylori entraîne la sécrétion d'un large panel de cytokines, notamment l'interleukine-1β (IL-1β), l'IL-8 et l'IL-17A. qui semblent jouer un rôle clé dans l'initiation et la progression du cancer gastrique. Parmi ces cytokines, l'IL-1β est une

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20

Milgrom, Sarah Allison. "Seroprevalence of Helicobacter pylori in Rural Ecuador." Yale University, 2009. http://ymtdl.med.yale.edu/theses/available/etd-03292009-172126/.

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Helicobacter pylori infection causes chronic type B gastritis, peptic ulcer disease, gastric adenocarcinoma, and gastric mucosa-associated lymphoid tumor. Infection with H. pylori is common in parts of the developing world. The Clínica Misional Nuestra Señora de Guadalupe serves indigenous people of the Ecuadorian Amazon. At the clinic, gastritis is a common diagnosis that is based solely on reported symptoms and is presumed to be due to H. pylori infection. Additionally, gastric cancer, a corollary of H. pylori infection, is the leading cause of cancer deaths in Ecuador and, thus, an importan

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21

Talbi, Patrice. "Macrolides et éradication de Hélicobacter pylori." Bordeaux 2, 1994. http://www.theses.fr/1994BOR23066.

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22

Massiere, Jessica. "La transition épithélio-mésenchymateuse dans les cellules épithéliales gastriques : rôle des microARN régulés par Helicobacter pylori." Thesis, Bordeaux 2, 2011. http://www.theses.fr/2011BOR21835/document.

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Les microARN sont de petits ARN non codant régulant post-transcriptionnellement l’expression de certains gènes. Du fait de leur fort potentiel régulateur, une modification de leur expression peut conduire à l’apparition de pathologies telles que le cancer ou l’inhibition des mécanismes de défense contre des pathogènes. Notre objectif est de caractériser le rôle de certains miARN dans la formation de cancer gastrique dû à Helicobacter pylori. En effet, cette bactérie peut conduire à l’apparition d’adénocarcinome gastrique et de lymphome du MALT. Sa virulence est essentiellement due à la protéin

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23

Daugule, Ilva. "Helicobacter pylori infection among children in Riga, Latvia /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7140-630-9/.

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24

Nguyen, Thi Viet Ha. "Diagnosis and treatment of Helicobacter pylori infection in Vietnamese children." Stockholm : Karolinska institutet, 2009. http://diss.kib.ki.se/2009/978-91-7409-717-7/.

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25

Khoder, Ghalia. "Biomarqueurs protéomiques du pathogène gastrique Helicobacter pylori." Poitiers, 2008. http://www.theses.fr/2008POIT1803.

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26

Palau, de Miguel Montserrat. "Detection of Helicobacter pylori Microevolution and Multiple Infection, and Genomic Analysis of Helicobacter pylori Strains." Doctoral thesis, Universitat de Barcelona, 2019. http://hdl.handle.net/10803/668148.

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In the past decades, Helicobacter pylori has received the attention of many researchers because of its known relation with gastric cancer. Although many studies have tried to decipher the exact relation between the bacteria and cancer state, and several virulence factors have been discovered, an exact answer has not been found yet. Further work should be made in order to study more accurately the genome of this bacterium and to understand its precise involvement. The bacterium is characterised for a highly genetic diversity, meaning it is continuously changing in order to adapt itself to its h

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27

Rapp, Nathalie. "Gastrites chroniques et formes folliculaires associées à hélicobacter pylori : aspects histologiques et endoscopiques : étude prospective de 445 observations." Bordeaux 2, 1993. http://www.theses.fr/1993BOR2M139.

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28

Wong, Chun-yu Benjamin, and 王振宇. "Helicobacter pylori: related diseases in the Chinese." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2000. http://hub.hku.hk/bib/B31981732.

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29

Wallace, Robyn A. "The biopsychosocial implications of helicobacter pylori infection in adults with intellectual disability /." St. Lucia, Qld, 2002. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe16438.pdf.

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30

Wen, Sicheng. "The mucosal immune response against Helicobacter pylori infection /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-033-6/.

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31

Ngoyi, Esther Nina. "Résistance de Helicobacter pylori aux antibiotiques et d’autres substances antimicrobiennes. : Aspects moléculaires des mécanismes de détection." Thesis, Bordeaux, 2016. http://www.theses.fr/2016BORD0442/document.

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Contexte : Amélioration de la prise en charge de l’infection à H. pylori. Matériels et Méthodes : Détection de H. pylori, la résistance à la clarithromycine, la tétracycline, la lévofloxacine, et la détermination des gènes de pathogénicité ont été réalisées par PCR en temps réel, du gène de l’ARNr 23 S, de l’ARNr 16S, PCR classique et séquençage. L’évaluation de la stabilité du mutant résistant par rapport à l’isolat sensible était obtenue par compétition en culture sur cellules gastriques AGS sur une longue période, suivie du séquençage du génome entier. L’évaluation de l’effet de l’extrait d

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32

Bourzac, Kevin. "A molecular and cellular investigation of Helicobacter pylori's cag pathogenicity island and interactions with host cells /." view abstract or download file of text, 2006. http://proquest.umi.com/pqdweb?index=0&did=1251819291&SrchMode=1&sid=1&Fmt=2&VInst=PROD&VType=PQD&RQT=309&VName=PQD&TS=1181341914&clientId=11238.

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Thesis (Ph. D.)--University of Oregon, 2006.<br>Typescript. Includes vita and abstract. Includes bibliographical references (leaves 80-91). Also available for download via the World Wide Web; free to University of Oregon users.

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33

Hoang, Thi Thu Ha. "Serodiagnosis and seroprevalence of Helicobacter pylori infection in Vietnam /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-918-1/.

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34

Tindberg, Ylva. "Helicobacter pylori infection among children in Sweden /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4629-9/.

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35

Khamri, Wafa. "Impact of collectins in 'Helicobacter pylori' infection." Thesis, Imperial College London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.409084.

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36

Stacey, Alison Rose. "Human immune responses to Helicobacter pylori infection." Thesis, Open University, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386268.

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37

Khandaker, Md Abul Kashem. "A study of gastric Helicobacter pylori infection." Thesis, University of Edinburgh, 1993. http://hdl.handle.net/1842/19899.

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<i>Helicobacter pylcri (H.pylori)</i> was studied in duodenal ulcer (DU) and gastric cancer patients. The relationship between dietary linoleic acid, antral <i>H.pylori</i> infection and DU was studied. A detailed dietary history was obtained from DU patients and matched non-ulcer dyspepsia controls. The fatty acid content of abdominal adipose tissue biopsies was measured in both groups. Levels of dietary and adipose tissue fatty acids were related in controls and non-smokers. The adipose fatty acids did not reflect dietary intake in DU patients or controls who smoked. DU is associated with re

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38

Paillé, Pascal. "Hélicobacter Pylori et gastrite chronique chez l'adulte jeune, essai d'utilisation du "Sydney system"." Bordeaux 2, 1991. http://www.theses.fr/1991BOR2M187.

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39

Olfat, Farzad. "Helicobacter pylori : bacterial adhesion and host response." Doctoral thesis, Umeå universitet, Odontologi, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-133.

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The gastric pathogen Helicobacter pylori infects more than half of the population worldwide. H. pylori manage to establish persistent infection, which would be life-long if not treated. In order to establish such an infection, this pathogen has to deal with the host immune system. H. pylori has certain characteristics which make the bacteria less announced to the host immune system. Additionally, for remaining in the harsh and acidic environment of the stomach with peristaltic movements and a high frequency of turnover of epithelial cells, H. pylori has developed different binding modes to str

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40

Jansson, Rehnberg Ann-Sofie. "Diagnostic methods in helicobacter pylori infection : development, evaluation and application /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-993-9/.

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41

Foynes, Susan. "Motility and chemotaxis studies in Helicobacter pylori." Thesis, University College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322447.

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42

Renou-Piton, Béatrice. "Prévalence de l'infection à Helicobacter pylori dans une population de 249 patients souffrant de dyspepsie fonctionnelle." Bordeaux 2, 1999. http://www.theses.fr/1999BOR23107.

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43

Thévenot, Sarah. "Variations génétiques chez Helicobacter pylori au cours d'une infection expérimentale murine : rôle de la polyphosphate kinase dans la colonisation." Poitiers, 2002. http://www.theses.fr/2002POIT1802.

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44

Audibert, Céline. "Polymorphisme des régions génomiques liées à la virulence chez Helicobacter pylori." Poitiers, 2000. http://www.theses.fr/2000POIT1805.

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45

Petersson, Christoffer. "Characterisation of surface traits of Helicobacter pylori and their role in the infectious process /." Linköping : Univ, 2003. http://www.bibl.liu.se/liupubl/disp/disp2003/med805s.pdf.

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46

Miendjé, Deyi Véronique Yvette. "Contribution au management de l'infection à Helicobacter pylori en Belgique." Doctoral thesis, Universite Libre de Bruxelles, 2011. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209925.

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Peu étudiée et méconnue, initialement décrite au début du XXe siècle, Helicobacter pylori fut redécouverte en 1982 par deux chercheurs australiens, JR Warren et BJ Marshall. Ils soutinrent que la plupart des ulcères gastro-duodénaux étaient causés par cette bactérie, et non par le stress ou la nourriture épicée, comme pensé auparavant. Cette découverte révolutionna le monde de la gastroentérologie et leur valut le prix Nobel de physiologie et de médecine 2005. Environ la moitié de la population mondiale est colonisée par H. pylori au niveau de l'estomac. Dans 10 à 20% des cas, l'infection peut

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47

Enroth, Helena. "Helicobacter pylori : bacterial diversity and human disease : microbiological and epidemiological studies with special reference to gastric cancer /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3673-0/.

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48

Neto, Cunha Gomes Joana. "Biological effects of Pteridium aquilinum and its toxin in gastric carcinogenesis : relationship with Helicobacter pylori infection." Thesis, Paris 11, 2012. http://www.theses.fr/2012PA114822.

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La cancérogenèse gastrique est un processus d’origine multifactorielle, incluant des facteurs génétiques de l’hôte, mais aussi d’origine bactérienne et de l’environnent. Les populations humaines peuvent être exposées directement ou indirectement à des composés toxiques/génotoxiques présents dans les plantes, comme la fougère Pteridium aquilinum. Cette plante comprend une toxine, le ptaquiloside associée à des maladies graves et le développement de cancer chez les animaux. Des études épidémiologiques ont démontré une association entre l'exposition à ces fougères et l’incidence du cancer gastriq

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49

Bamford, Kathleen Branigan. "An investigation of infection caused by Helicobacter pylori." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394465.

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50

Bartfeld, Sina. "NF-kappaB activation in infections with Helicobacter pylori or Legionella pneumophila." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2009. http://dx.doi.org/10.18452/15939.

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Bakterielle Infektionen aktivieren den Transkriptionsfaktor NF-κB. Dies trägt sowohl zur Immunantwort, als auch zum Überleben der Wirtszellen bei. In Infektionen mit dem Bakterium Helicobacter pylori stellt diese doppelte Funktion einen mechanistischen Link zwischen der chronischen Entzündung und der Krebsentstehung dar. Für das intrazelluläre Bakterium Legionella pneumophila ist das Überleben der Wirtszelle notwendig für die Vermehrung. Bisher sind jedoch bei beiden Infektionen die Signalwege, die zur NF-κB-Aktivierung führen, nicht ausreichend untersucht. Um NF-κB analysieren zu können, wurd

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