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Journal articles on the topic 'Helicobacter pylori. Helicobacter pylori infections'

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Published: 4 June 2021

Last updated: 7 February 2022

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1

Haesebrouck, Freddy, Frank Pasmans, Bram Flahou, et al. "Gastric Helicobacters in Domestic Animals and Nonhuman Primates and Their Significance for Human Health." Clinical Microbiology Reviews 22, no. 2 (2009): 202–23. http://dx.doi.org/10.1128/cmr.00041-08.

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SUMMARY Helicobacters other than Helicobacter pylori have been associated with gastritis, gastric ulcers, and gastric mucosa-associated lymphoid tissue lymphoma in humans. These very fastidious microorganisms with a typical large spiral-shaped morphology were provisionally designated “H. heilmannii,” but in fact they comprise at least five different Helicobacter species, all of which are known to colonize the gastric mucosa of animals. H. suis, which has been isolated from the stomachs of pigs, is the most prevalent gastric non-H. pylori Helicobacter species in humans. Other gastric non-H. pyl

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FAISAL, NABIHA, MUHAMMAD MANSOOR UL HAQ, HAFEEZULLAH SHAIKH, Pervez Ashraf, and Jamila H. Esmail. "HELICOBACTER PYLORI INFECTION;." Professional Medical Journal 19, no. 02 (2012): 202–7. http://dx.doi.org/10.29309/tpmj/2012.19.02.2011.

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Objective: To determine the frequency of H. pylori infection in dyspeptic patients undergoing endoscopy at a tertiary care centerin Karachi. Data source: Patients undergoing endoscopy at Liaquat National Hospital, Karachi. Design of study: Cross sectional descriptivestudy. Setting: Department of Gastroenterology, Liaquat National Hospital, Karachi. Period: May 2008–October 2008. Material andmethods: All adult patients with symptoms of dyspepsia for more than 1 month duration were included. Patients with upper gastrointestinalbleed, anemia or weight loss were excluded. Upper gastrointestinal en

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Suwarni, Sri, Cilmiaty Risya, Dono Indarto, and Suradi Pulmonology. "Helicobacter Pylori is associated with decrease serum level of the thyroid hormonal in healthy elderly population." Bangladesh Journal of Medical Science 16, no. 4 (2017): 515–20. http://dx.doi.org/10.3329/bjms.v16i4.33604.

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Background: Helicobacter pylori infection is the most prevalence infectious disease as it affects more than half of the world population and causes chronic cellular inflammatory response in the gastric mucosa. Helicobacter pylori infection has been epidemiologically proven to be linked to extra-digestive conditions and disease. It has been speculated that H.pylori infection may be responsible for various endocrine disorders. The thyroid may be one of the targets of Helicobacter pylori chronic inflammation. Here we sought too investigate whether H.pylori infections were associated with decrease

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4

Chiba, N., A. Matisko, P. Sinclair, and ABR Thomson. "Helicobacter pylori: From Bench to Bedside." Canadian Journal of Gastroenterology 11, no. 7 (1997): 589–96. http://dx.doi.org/10.1155/1997/975469.

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With the exponential increase in research in the field ofHelicobacter pyloria paradigm shift has occurred. It is now recognized thatH pyloriis a chronic infection of the stomach causing inflammation. Some patients remain asymptomatic, while others may develop dyspepsia, duodenal or gastric ulcer, gastric cancer or a mucosa-associated lymphoid tissue lymphoma. However, the role ofH pyloriin contributing to nonulcer dyspepsia or nonsteroidal anti-inflammatory drug gastropathy remains controversial. An effective vaccine againstH pyloriis years away. Major interest has focused on the questions "wh

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Jurnalis, Yusri Dianne, Yorva Sayoet, and Sari Dewi. "HELICOBACTER PYLORI INFECTION IN CHILDREN." Majalah Kedokteran Andalas 35, no. 1 (2011): 43. http://dx.doi.org/10.22338/mka.v35.i1.p43-49.2011.

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AbstrakFaktor risiko infeksi Helicobacter pylori adalah tinggal di negara berkembang, kondisi sosial ekonomi yang rendah, jumlah anggota keluarga yang banyak, etnik dan genetik. Tatalaksana dan diagnosis Helicobacter pylori belum memuaskan karena adanya resistensi antibiotik pada pasien Helicobacter pylori. Kami melaporkan seorang pasien perempuan usia 8 tahun 6 bulan yang terinfeksi Helicobacter pylori. Diagnosis ditegakkan berdasarkan anamnesis, pemeriksaan fisik, dan hasil laboratorium. Pasien diduga terinfeksi Helicobacter pylori karena mengalami nyeri perut berulang. Dari laboratorium did

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Bertholom, Chantal. "Infections à Helicobacter pylori." Option/Bio 26, no. 521 (2015): 13–15. http://dx.doi.org/10.1016/s0992-5945(15)30025-8.

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7

Paradowski, Leszek. "Helicobacter pylori – a still ongoing problem." Medycyna Faktów 13, no. 4 (2020): 491–95. http://dx.doi.org/10.24292/01.mf.0420.17.

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The diagnosis and treatment of Helicobacter pylori infection are up-to-date, which is due to the frequency of human infections and the consequences of the chronic once, in most cases Helicobacter pylori infection is asymptomatic. If there are symptoms, the most common are dyspeptic complaints. In some patients Helicobacter pylori causes severe disease, mainly in stomach. This pathogen has been recognized as the main cause of the peptic ulcer, gastric cancer and MALT lymphoma. It is also known to play an important role in some other disease such essential thrombocytopenia, some types of deficie

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8

Dunn, B. E., H. Cohen, and M. J. Blaser. "Helicobacter pylori." Clinical Microbiology Reviews 10, no. 4 (1997): 720–41. http://dx.doi.org/10.1128/cmr.10.4.720.

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Helicobacter pylori is a gram-negative bacterium which causes chronic gastritis and plays important roles in peptic ulcer disease, gastric carcinoma, and gastric lymphoma. H. pylori has been found in the stomachs of humans in all parts of the world. In developing countries, 70 to 90% of the population carries H. pylori. In developed countries, the prevalence of infection is lower. There appears to be no substantial reservoir of H. pylori aside from the human stomach. Transmission can occur by iatrogenic, fecal-oral, and oral-oral routes. H. pylori is able to colonize and persist in a unique bi

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Ferrero, Richard L., Patrick Avé, Delphine Ndiaye та ін. "NF-κB Activation during Acute Helicobacter pylori Infection in Mice". Infection and Immunity 76, № 2 (2007): 551–61. http://dx.doi.org/10.1128/iai.01107-07.

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ABSTRACT Nuclear factor κB (NF-κB) plays a key regulatory role in host cell responses to Helicobacter pylori infection in humans. Although mice are routinely used as a model to study H. pylori pathogenesis, the role of NF-κB in murine cell responses to helicobacters has not been studied in detail. We thus investigated the abilities of different Helicobacter isolates to induce NF-κB-dependent responses in murine gastric epithelial cells (GECs) and in transgenic mice harboring an NF-κB-responsive lacZ reporter gene. H. pylori and Helicobacter felis strains up-regulated the synthesis in mouse GEC

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10

Tabassum, Tahira, Ayesha Imtiaz, Aamir Sharif, and Muhammad Akram. "HELICOBACTER PYLORI INFECTION;." Professional Medical Journal 24, no. 09 (2017): 1425–30. http://dx.doi.org/10.29309/tpmj/2017.24.09.884.

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Objectives: To estimate the prevalence of Helicobacter pylori infection inpopulation of Sargodha and determination of possible risk factors. Design: Descriptive study.Place and Duration: University medical complex & Research Center, Sargodha Medical College,Sargodha from 01.01.2017 to 31.03.2017. Material and Methods: The study was conductedamong 486 clinically suspected individuals of Sargodha, Punjab, Pakistan. Data was obtainedby questionnaire and H. pylori antibodies were detected by H. pylori ‘One Step Test Device’based on immune-chromatographic technique. Results: A total of 486 indi

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Kuntoji, Shrikanth B., Ankita A. Naik, and Suhas D. S. "A prospective study of proportion of Helicobacter pylori infection in patients presenting with dyspeptic symptoms in S. Nijalingappa Medical College and H.S.K. Hospital, Bagalkot." International Surgery Journal 7, no. 8 (2020): 2685. http://dx.doi.org/10.18203/2349-2902.isj20203255.

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Background: More than half of the world’s population is infected with Helicobacter pylori, the primary cause of chronic gastritis. Chronic gastritis is associated with peptic ulcer and in advanced stages with an increased risk of developing gastric adenocarcinoma. The aim of the study was to determine the prevalence of Helicobacter pylori infection in dyspeptic patients and its relationship with gastroduodenal pathologies using gastric biopsy histology.Methods: A cross sectional prospective study was conducted from 1st January 2018 to 30th June 2019. It included out-patients and in-patients, p

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12

Rengaswamy, Ramapriya, and Thayumanavan I. "Incidence and profile of Helicobacter pylori infection among HIV positive patients." International Journal of Advances in Medicine 5, no. 5 (2018): 1299. http://dx.doi.org/10.18203/2349-3933.ijam20183912.

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Background: People having HIV infection present with a number of symptoms related to gastrointestinal tract like dyspepsia. HIV itself as well as opportunistic infections is responsible for such symptoms. Notably is Helicobacter pylori infection causes variety of such symptoms. The objective of the present research was to study incidence and profile of Helicobacter pylori among HIV positive patients.Methods: A hospital based cross sectional study was carried out over a period of two years among 101 HIV positive patients in the Department of General Medicine, Madurai Medical College, Madurai in

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13

Lauritano, Moreo, Carinci, et al. "Helicobacter Pylory and Oral Diseases." Proceedings 35, no. 1 (2019): 16. http://dx.doi.org/10.3390/proceedings2019035016.

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Helicobacter pylori (H. pylori) gastric infection is considered one of the most common human infections. It occurs in half of the world’s population is the most common cause of adenocarcinoma of the distal stomach [1]. [...]

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14

Parsonnet, Julie. "HELICOBACTER PYLORI." Infectious Disease Clinics of North America 12, no. 1 (1998): 185–97. http://dx.doi.org/10.1016/s0891-5520(05)70417-7.

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15

Moss, Steven F., and Shivani Sood. "Helicobacter pylori." Current Opinion in Infectious Diseases 16, no. 5 (2003): 445–51. http://dx.doi.org/10.1097/00001432-200310000-00011.

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16

Syngal, Sapna, Michael M. Wolfe, Lawrence S. Friedman, and Jacques Van Dam. "Helicobacter pylori." Infectious Diseases in Clinical Practice 6, no. 1 (1997): 17–27. http://dx.doi.org/10.1097/00019048-199701000-00005.

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17

Syngal, Sapna, Michael M. Wolfe, Lawrence S. Friedman, and Jacques Van Dam. "Helicobacter pylori." Infectious Diseases in Clinical Practice 6, no. 1 (1997): 17–27. http://dx.doi.org/10.1097/00019048-199706010-00005.

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18

IMANZADEH, FARID, AMIR IMANZADEH, ALI AKBAR SAYYARI, Mehrnosh Yeganeh, Hazhir Javaherizadeh, and Bizhan Hatamian. "HELICOBACTER PYLORI INFECTION." Professional Medical Journal 17, no. 04 (2010): 543–45. http://dx.doi.org/10.29309/tpmj/2010.17.04.2955.

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Introduction: In most individuals H. Pylori is acquired early in the life (before 5 years). H. Pylori infection is more common in the third world countries, where about 90% of adults may be infected. Helicobacter pylori is one of the suspected causes of halitosis in children. Objectives: To evaluate the relationship of helicobacter pylori and halitosis. Patients and Material: 33 patients with chief complaint of halitosis included in our study. Halitosis was evaluated as a subjective symptom in this study. Careful history was obtained. All patients underwent physical examination in order to rul

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19

Manyakina, O. M., I. S. Akkuratova-Maksimova, T. G. Pukhova, and A. S. Shitova. "Role of genetic structure of Helicobacter Pylori in formation of chronic inflammatory process in gastric mucosa." Perm Medical Journal 38, no. 1 (2021): 87–99. http://dx.doi.org/10.17816/pmj38187-99.

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The literature review highlights the questions of the interaction of Helicobacter pylori and the human body. Modern data on the structure of the pathogenicity island in the Helicobacter pylori genome are presented. There is given a detailed description of both well-known virulence and pathogenicity factors of the infection (genes encoding the formation of urease subunits, in particular urel, cytotoxin associated gene A, vacuolating cytotoxin gen A, blood group associated binding adhesion, induced by contact with epithelium) and less studied ones (sialic acid-binding adhesion, adhesion-associat

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20

SHAIKH, MUHAMMED KHALID, JAVED AKHTAR SAMO, SAMINA SHAIKH, and Syed Zulfiquar Ali Shah. "HELICOBACTER PYLORI INFECTION." Professional Medical Journal 20, no. 05 (2013): 667–71. http://dx.doi.org/10.29309/tpmj/2013.20.05.1210.

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Objective: To determine the frequency of raised C-reactive protein in patients with Helicobacter pylori infection. Design:Cross sectional descriptive study. Setting: Department of Medicine at Liaquat University Hospital (tertiary care teaching hospitals).Period: April 2012 to September 2012. Patients and methods: All patients above 12 years of age, of either gender with history of nausea,vomiting, recurrent abdominal pain, dyspepsia or abdominal discomfort, heartburn, bloating and halitosis through casualty outdoordepartment (COD) or admitted in medical unit were evaluated and enrolled in the

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21

Ansari, Shuaib, Irfan Murtaza Shahwani, Aqeel Ahmed Channa, Syed Zulfiquar Ali Shah, and Tarachand Devrajani. "HELICOBACTER PYLORI." Professional Medical Journal 21, no. 04 (2018): 679–83. http://dx.doi.org/10.29309/tpmj/2014.21.04.2232.

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Objective: To determine the frequency of H. pylori in patients with functionaldyspepsia. Patients and methods: This six months multidisciplinary study was conducted attertiary care teaching hospital as well as at private hospital Hyderabad from February 2012 to July2012. All patients presented with history of dyspepsia were admitted and evaluated for functionaldyspepsia by performing endoscopy. After confirmation of functional dyspepsia the subjectswere further evaluated for H. pylori infection by taking the biopsy specimen, label it and sent tolaboratory for histopathological examination. The

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22

Alziyadi, Faisal, Nouf Alshammeri, Ali Alamer, et al. "The relation between Helicobacter pylori and gastroesophageal reflux disease in KSA." International Journal Of Community Medicine And Public Health 5, no. 9 (2018): 3691. http://dx.doi.org/10.18203/2394-6040.ijcmph20183436.

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Estimates say that Helicobacter pylorus affects more than half of the general population over the world. Its prevalence varies significantly among different areas. Studies done in 1990 reported a prevalence of 70% of people older than 20 years who live in Saudi Arabia, and 40% of people aged 5-10 years. The first line regimen of helicobacter pylori infections in Saudi Arabia is 7 days of triple therapy. However, in areas of high antibiotic resistance, or in cases of failure of triple therapy, a ‘rescue’ quadruple therapy (that contains bismuth) is indicated. In this study we aimed to look at t

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LASA, Juan, Ignacio ZUBIAURRE, Guillermo DIMA, Daniel PERALTA, and Luis SOIFER. "HELICOBACTER PYLORI PREVALENCE IN PATIENTS WITH CELIAC DISEASE: results from a cross-sectional study." Arquivos de Gastroenterologia 52, no. 2 (2015): 139–42. http://dx.doi.org/10.1590/s0004-28032015000200012.

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Background Some previously published studies have suggested an inverse relationship between celiac disease and Helicobacter pylori, raising the possibility of the protective role Helicobacter pylori could have against celiac disease development. Nevertheless, this association is inconclusive. Objectives To determine the prevalence of Helicobacter pylori infection in celiac subjects. Methods Between January 2013 and June 2014, patients over 18 years old undergoing upper endoscopy who required both gastric and duodenal biopsies were included for analysis. Enrolled subjects were divided in two gr

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Blaser, M. J. "HELICOBACTER PYLORI." Pediatric Infectious Disease Journal 12, no. 5 (1993): 422. http://dx.doi.org/10.1097/00006454-199305000-00027.

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25

Lilian Okwubenata, Okonkwo, Musa Bolanle Olufunke Priscilla, Hali Bello, and Mustapha Shettima Kagu. "Sero-Prevalence and Risk Factors for Helicobacter pylori Infection Among Individuals with and those Without Dyspepsia in Zaria, Northwestern Nigeria." Annals of Clinical and Experimental Medicine 1, no. 1 (2020): 73–76. http://dx.doi.org/10.47838/acem.26011977.11162020.asmeda.12.0.

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Background Helicobacter pylori infection is prevalent in African region especially in areas with low socioeconomic status. This study aimed to determine the prevalence and risk factors for Helicobacter pylori Infection among individuals with and those without dyspepsia Materials and Methods The study was cross sectional in which individuals with and those without dyspepsia were enrolled. The prevalence for Helicobacter pylori infection was determined by the screening of Helicobacter pylori immunoglobulin G and this was compared across variables of interest. IBM SPSS was used for the data analy

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Turbabina, N. A., M. S. Maksimova, E. V. Stepanova, et al. "HELICOBACTER PYLORI DIAGNOSTIC METHODS." Medical Parasitology and Parasitic Diseases, no. 3 (October 2020): 53–58. http://dx.doi.org/10.33092/0025-8326mp2020.3.53-58.

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Helicobacteriosis is one of the most urgent problems of gastroenterology, and due to the fact that the frequency of H. pylori infection is progressively increasing, this disease is increasingly being detected in people of young working age. The development of algorithms for early and accurate diagnosis of helicobacteriosis can improve the quality of treatment and follow-up of this category of patients. To date, in medical practice, H. pylori is detected using bacteriological, histological (immunohistological), molecular genetic, biochemical and immunological methods (enzyme-linked immunosorben

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27

Buckley, Martin, Paul Martin, and Colm OʼMorain. "Helicobacter pylori Infection." Drugs & Aging 5, no. 1 (1994): 1–7. http://dx.doi.org/10.2165/00002512-199405010-00001.

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28

Vandenplas, Y. "Helicobacter pylori infection." Clinical Microbiology and Infection 5, no. 1 (1999): 1–11. http://dx.doi.org/10.1111/j.1469-0691.1999.tb00091.x.

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29

Crowe, Sheila E. "Helicobacter pylori Infection." New England Journal of Medicine 380, no. 12 (2019): 1158–65. http://dx.doi.org/10.1056/nejmcp1710945.

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30

Wu, Deng-Chyang, Ping-I. Hsu, Hui-Hwa Tseng, et al. "Helicobacter pylori Infection." Medicine 90, no. 3 (2011): 180–85. http://dx.doi.org/10.1097/md.0b013e31821c9d1c.

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31

Belhoussine-Idrissi, Lila, and Edgar C. Boedeker. "Helicobacter pylori infection." Current Opinion in Internal Medicine 1, no. 2 (2002): 113–20. http://dx.doi.org/10.1097/00132980-200201020-00002.

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32

Anand, J. K. "Helicobacter Pylori Infection." Journal of the Royal Society of Medicine 90, no. 3 (1997): 180. http://dx.doi.org/10.1177/014107689709000331.

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Goodwin, C. Stewart, Michael M. Mendall, and Timothy C. Northfield. "Helicobacter pylori infection." Lancet 349, no. 9047 (1997): 265–69. http://dx.doi.org/10.1016/s0140-6736(96)07023-7.

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34

Polyzos, Stergios A., and Jannis Kountouras. "Helicobacter pylori Infection." Journal of Clinical Gastroenterology 53, no. 8 (2019): 624–26. http://dx.doi.org/10.1097/mcg.0000000000001218.

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35

Axon, A. T. R. "Helicobacter pylori infection." Journal of Antimicrobial Chemotherapy 32, suppl A (1993): 61–68. http://dx.doi.org/10.1093/jac/32.suppl_a.61.

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36

EBRAHIM, G. J. "Helicobacter pylori infection." Journal of Tropical Pediatrics 39, no. 6 (1993): 326–27. http://dx.doi.org/10.1093/tropej/39.6.326.

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Larsen, John Teilmann, Erik Skjelbo, and Lars Freng Gram. "Helicobacter pylori infection." Lancet 349, no. 9055 (1997): 878–79. http://dx.doi.org/10.1016/s0140-6736(05)61783-7.

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Numans, Mattijs E., and A. Otto Quartero. "Helicobacter pylori infection." Lancet 349, no. 9055 (1997): 879. http://dx.doi.org/10.1016/s0140-6736(05)61784-9.

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Harris, Adam, John Danesh, and David Forman. "Helicobacter pylori infection." Lancet 349, no. 9055 (1997): 879–80. http://dx.doi.org/10.1016/s0140-6736(05)61785-0.

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40

Ruggiero, Paolo. "Helicobacter pylori infection." Current Opinion in Infectious Diseases 25, no. 3 (2012): 337–44. http://dx.doi.org/10.1097/qco.0b013e3283531f7c.

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Malfertheiner, Peter, Marino Venerito, and Michael Selgrad. "Helicobacter pylori infection." Current Opinion in Gastroenterology 29, no. 6 (2013): 669–75. http://dx.doi.org/10.1097/mog.0b013e328365d443.

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Hughes, Walter T. "Helicobacter pylori Infection." Pediatric Annals 25, no. 9 (1996): 491–93. http://dx.doi.org/10.3928/0090-4481-19960901-05.

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Tovey, F. I. "Helicobacter Pylori Infection." Tropical Doctor 25, no. 2 (1995): 49. http://dx.doi.org/10.1177/004947559502500201.

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Bateson, M. C. "Helicobacter pylori infection." Gut 35, no. 5 (1994): 716–17. http://dx.doi.org/10.1136/gut.35.5.716.

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Hanafi, Manal I., and Aida M. Mohamed. "Helicobacter pylori infection." Journal of the Egyptian Public Health Association 88, no. 1 (2013): 40–45. http://dx.doi.org/10.1097/01.epx.0000427043.99834.a4.

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46

Anderson, Monte L. "Helicobacter pylori infection." Postgraduate Medicine 96, no. 6 (1994): 40–50. http://dx.doi.org/10.1080/00325481.1994.11945920.

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Metz, David C., H. Christian Weber, Murray Orbuch, Doris B. Strader, Irina A. Lubensky, and Robert T. Jensen. "Helicobacter pylori infection." Digestive Diseases and Sciences 40, no. 1 (1995): 153–59. http://dx.doi.org/10.1007/bf02063959.

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48

Qadir, Adnan, Irfan Younis, Shahid Raza Khalid, Zamir Butt, and Shahid Sarwar. "HELICOBACTER PYLORI INFECTION." Professional Medical Journal 23, no. 10 (2016): 1243–46. http://dx.doi.org/10.29309/tpmj/2016.23.10.1730.

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Objectives: To determine diagnostic accuracy of H.pylori Stool Antigen test fordiagnosing Helicobactor pylori infection, keeping histopathology as gold standard. Studydesign: Cross sectional Validation Study. Place and duration: Study was conducted indepartment of Gastroenterology Military Hospital, Rawalpindi for six months from 1st November2014 to 30th April, 2015. Patients and methods: Serial patients presenting with dyspepsiafulfilling the inclusion criteria were entered in study program. Endoscopy was performed to takeantral biopsies for histopathology and stool was taken to test H.pylori

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Cho, Jaehoon, Akriti Prashar, Nicola L. Jones, and Steven F. Moss. "Helicobacter pylori Infection." Gastroenterology Clinics of North America 50, no. 2 (2021): 261–82. http://dx.doi.org/10.1016/j.gtc.2021.02.001.

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50

Lemke, Laura B., Zhongming Ge, Mark T. Whary, et al. "Concurrent Helicobacter bilis Infection in C57BL/6 Mice Attenuates Proinflammatory H. pylori-Induced Gastric Pathology." Infection and Immunity 77, no. 5 (2009): 2147–58. http://dx.doi.org/10.1128/iai.01395-08.

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ABSTRACT Because coinfections can alter helicobacter gastritis, we investigated whether enterohepatic Helicobacter bilis modulates Helicobacter pylori gastritis in C57BL/6 mice. Thirty mice per group were sham dosed, H. bilis or H. pylori infected, or H. bilis infected followed in 2 weeks by H. pylori and then evaluated at 6 and 11 months postinfection (mpi) for gastritis and premalignant lesions. Compared to H. pylori-infected mice, H. bilis/H. pylori-infected mice at 6 and 11 mpi had less severe gastritis, atrophy, mucous metaplasia and hyperplasia (P < 0.01) and, additionally, at 11 mpi,

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