Relevant bibliographies by topics / Hormone immune networks

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Reports

Academic literature on the topic 'Hormone immune networks'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Hormone immune networks"

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Meik, Kunz, Ahmed Nazeer, Dandekar Thomas, and Naseem Muhammad. "Hormone Signaling Networks Open Multiple Routes for Immunity and Disease in Plants." Biohelikon 1, no. 2 (2013): 1–3. https://doi.org/10.5281/zenodo.816323.

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Plant hormones are small signaling molecules that regulate almost every aspect of plant life cycle. Plant pathogens alter hormonal balance of the host to cause disease. In response, host redefines hormone signaling networks to cope with the invading pathogen. Biotrophic pathogens are generally sensitive to salicylic acid mediated defense responses, whereas necrotrophic pathogens are deterred through jasmonate/ethylene pathways. Interaction between these pathways is regarded as central backbone of plant immunity. Classic hormones such as auxin, gibberellin, and cytokinin either promote salicyli
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Keith, Scott A. "Steroid hormone regulation of innate immunity in Drosophila melanogaster." PLOS Genetics 19, no. 6 (2023): e1010782. http://dx.doi.org/10.1371/journal.pgen.1010782.

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Endocrine signaling networks control diverse biological processes and life history traits across metazoans. In both invertebrate and vertebrate taxa, steroid hormones regulate immune system function in response to intrinsic and environmental stimuli, such as microbial infection. The mechanisms of this endocrine-immune regulation are complex and constitute an ongoing research endeavor facilitated by genetically tractable animal models. The 20-hydroxyecdysone (20E) is the major steroid hormone in arthropods, primarily studied for its essential role in mediating developmental transitions and meta
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Dan, Song, and Shi Yichao. "Immune system modifications and feto-maternal immune tolerance." Chinese Medical Journal 127, no. 17 (2014): 3171–80. http://dx.doi.org/10.3760/cma.j.issn.0366-6999.20133072.

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Objective This review aimed at understanding pregnancy-induced changes in the maternal immune response and mechanisms for the establishment of feto-maternal tolerance. Data sources Articles cited in this review were obtained from PubMed in English from 2000 to 2014, and the search string included keywords such as feto-maternal tolerance, dendritic cells, macrophage, T regulatory cells, natural killer cells, cytokines and hormone. Study selection Articles regarding altered maternal immune response, including the proliferation and differentiation of the altered cells, and the production of cytok
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Naseem, Muhammad, Meik Kunz, and Thomas Dandekar. "Probing the Unknowns in Cytokinin-Mediated Immune Defense in Arabidopsis with Systems Biology Approaches." Bioinformatics and Biology Insights 8 (January 2014): BBI.S13462. http://dx.doi.org/10.4137/bbi.s13462.

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Plant hormones involving salicylic acid (SA), jasmonic acid (JA), ethylene (Et), and auxin, gibberellins, and abscisic acid (ABA) are known to regulate host immune responses. However, plant hormone cytokinin has the potential to modulate defense signaling including SA and JA. It promotes plant pathogen and herbivore resistance; underlying mechanisms are still unknown. Using systems biology approaches, we unravel hub points of immune interaction mediated by cytokinin signaling in Arabidopsis. High-confidence Arabidopsis protein—protein interactions (PPI) are coupled to changes in cytokinin-medi
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Wang, Hongtao, and Zhiming Xie. "Cullin-Conciliated Regulation of Plant Immune Responses: Implications for Sustainable Crop Protection." Plants 13, no. 21 (2024): 2997. http://dx.doi.org/10.3390/plants13212997.

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Cullins are crucial components of the ubiquitin–proteasome system, playing pivotal roles in the regulation of protein metabolism. This review provides insight into the wide-ranging functions of cullins, particularly focusing on their impact on plant growth, development, and environmental stress responses. By modulating cullin-mediated protein mechanisms, researchers can fine-tune hormone-signaling networks to improve various agronomic traits, including plant architecture, flowering time, fruit development, and nutrient uptake. Furthermore, the targeted manipulation of cullins that are involved
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Bright, John J., Saravanan Kanakasabai, Wanida Chearwae, and Sharmistha Chakraborty. "PPAR Regulation of Inflammatory Signaling in CNS Diseases." PPAR Research 2008 (2008): 1–12. http://dx.doi.org/10.1155/2008/658520.

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Central nervous system (CNS) is an immune privileged site, nevertheless inflammation associates with many CNS diseases. Peroxisome proliferator-activated receptors (PPARs) are a family of nuclear hormone receptors that regulate immune and inflammatory responses. Specific ligands for PPAR, , and isoforms have proven effective in the animal models of multiple sclerosis (MS), Alzheimer's disease, Parkinson's disease, and trauma/stroke, suggesting their use in the treatment of neuroinflammatory diseases. The activation of NF-B and Jak-Stat signaling pathways and secretion of inflammatory cytokines
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Nagy, Laszlo, Attila Szanto, Istvan Szatmari, and Lajos Széles. "Nuclear Hormone Receptors Enable Macrophages and Dendritic Cells to Sense Their Lipid Environment and Shape Their Immune Response." Physiological Reviews 92, no. 2 (2012): 739–89. http://dx.doi.org/10.1152/physrev.00004.2011.

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A key issue in the immune system is to generate specific cell types, often with opposing activities. The mechanisms of differentiation and subtype specification of immune cells such as macrophages and dendritic cells are critical to understand the regulatory principles and logic of the immune system. In addition to cytokines and pathogens, it is increasingly appreciated that lipid signaling also has a key role in differentiation and subtype specification. In this review we explore how intracellular lipid signaling via a set of transcription factors regulates cellular differentiation, subtype s
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Ribarac-Stepic, Nevena, Snezana Djurica, Zorica Zakula, Goran Koricanac, and Dragoslav Milosevic. "Molecular basis of glucocorticoid action." Srpski arhiv za celokupno lekarstvo 133, Suppl. 1 (2005): 61–66. http://dx.doi.org/10.2298/sarh05s1061r.

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Glucocorticoid hormones are involved in regulation of cell processes and coordinate physiological response to diverse signals. These hormones, through interaction with specific intracellular receptors, coordinate components of physiological repertoires by activating the expression of gene networks. Thus hormone-receptor complexes may function as key constituent in regulation of specific cell functions as well as in provoking differentiation in already determined cells. Analysis of steroid receptors are important for understanding of molecular details of transcriptional control as well as provi
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McKinney, Lawrence P., Rajesh Singh, I. King Jordan, Sooryanarayana Varambally, Eric B. Dammer, and James W. Lillard. "Transcriptome Analysis Identifies Tumor Immune Microenvironment Signaling Networks Supporting Metastatic Castration-Resistant Prostate Cancer." Onco 3, no. 2 (2023): 81–95. http://dx.doi.org/10.3390/onco3020007.

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Prostate cancer (PCa) is the second most common cause of cancer death in American men. Metastatic castration-resistant prostate cancer (mCRPC) is the most lethal form of PCa and preferentially metastasizes to the bones through incompletely understood molecular mechanisms. Herein, we processed RNA sequencing data from patients with mCRPC (n = 60) and identified 14 gene clusters (modules) highly correlated with mCRPC bone metastasis. We used a novel combination of weighted gene co-expression network analysis (WGCNA) and upstream regulator and gene ontology analyses of clinically annotated transc
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Zhu, Wenyong, Hao Huang, Zixi Hu, et al. "Comprehensive Transcriptome Analysis Expands lncRNA Functional Profiles in Breast Cancer." International Journal of Molecular Sciences 25, no. 15 (2024): 8456. http://dx.doi.org/10.3390/ijms25158456.

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Breast cancer is a heterogeneous disease that arises as a multi-stage process involving multiple cell types. Patients diagnosed with the same clinical stage and pathological classification may have different prognoses and therapeutic responses due to alterations in molecular genetics. As an essential marker for the molecular subtyping of breast cancer, long non-coding RNAs (lncRNAs) play a crucial role in gene expression regulation, cell differentiation, and the maintenance of genomic stability. Here, we developed a modular framework for lncRNA identification and applied it to a breast cancer
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Dissertations / Theses on the topic "Hormone immune networks"

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Hamy, Anne-Sophie. "Identification of Factors Predicting Sensitivity or Resistance to Neoadjuvant Chemotherapy in Breast Cancer Neoadjuvant treatment : the future of patients with breast cancer Neoadjuvant treatment for intermediate/high-risk HER2-positive and triple-negative breast cancers: no longer an “option” but an ethical obligation Long-term outcome of the REMAGUS 02 trial, a multicenter randomised phase II trial in locally advanced breast cancer patients treated with neoadjuvant chemotherapy with or without celecoxib or trastuzumab according to HER2 status BIRC5 (survivin) : a pejorative prognostic marker in stage II/III breast cancer with no response to neoadjuvant chemotherapy Beyond Axillary Lymph Node Metastasis, BMI and Menopausal Status Are Prognostic Determinants for Triple-Negative Breast Cancer Treated by Neoadjuvant Chemotherapy Pathological complete response and prognosis after neoadjuvant chemotherapy for HER2-positive breast cancers before and after trastuzumab era: results from a real-life cohort The presence of an in situ component on pre-treatment biopsy is not associated with response to neoadjuvant chemotherapy for breast cancer Chemosensitivity, tumor infiltrating lymphocytes (TILs), and survival of postpartum PABC patients treated by neoadjuvant chemotherapy Lymphovascular invasion after neoadjuvant chemotherapy is strongly associated with poor prognosis in breast carcinoma New insight for pharmacogenomics studies from the transcriptional analysis of two large-scale cancer cell line panels Biological network-driven gene selection identifies a stromal immune module as a key determinant of triple-negative breast carcinoma prognosis A Stromal Immune Module Correlated with the Response to Neoadjuvant Chemotherapy, Prognosis and Lymphocyte Infiltration in HER2-Positive Breast Carcinoma Is Inversely Correlated with Hormonal Pathways Stromal lymphocyte infiltration after neoadjuvant chemotherapy is associated with aggressive residual disease and lower disease-free survival in HER2-positive breast cancer Interaction between molecular subtypes, stromal immune infiltration before and after treatment in breast cancer patients treated with neoadjuvant chemotherapy COX2/PTGS2 Expression Is Predictive of Response to Neoadjuvant Celecoxib in HER2-negative Breast Cancer Patients Celecoxib With Neoadjuvant Chemotherapy for Breast Cancer Might Worsen Outcomes Differentially by COX-2 Expression and ER Status: Exploratory Analysis of the REMAGUS02 Trial Comedications influence immune infiltration and pathological response to neoadjuvant chemotherapy in breast cancer." Thesis, Université Paris-Saclay (ComUE), 2019. http://www.theses.fr/2019SACLS129.

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La chimiothérapie néoadjuvante (CNA) est utilisée dans les cancers du sein agressifs ou localement avancés (CS). Au delà des bénéfices cliniques, elle représente une opportunité pour monitorer in vivo la sensibilité d’une tumeur à un traitement.A partir de l’analyse de sets de données de patients traités par CNA, nous souhaitons identifier des mécanismes associes à la résistance ou sensibilité au traitement. Dans la première partie, nous avons évalué des paramètres, cliniques, anatomopathologiques et transcriptomiques. Nous avons démontré que des éléments non explorés comme la présence d’embol
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Book chapters on the topic "Hormone immune networks"

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Besedovsky, H. O., and A. del Rey. "Introduction: Immune-Neuroendocrine Network." In Frontiers of Hormone Research. KARGER, 2001. http://dx.doi.org/10.1159/000061055.

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Sullivan, David A. "Hormonal Influence on the Secretory Immune System of the Eye." In The Neuroendocrine-Immune Network. CRC Press, 2018. http://dx.doi.org/10.1201/9781351077194-15.

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Plas, Eugen, Stephan Madersbacher, and Peter Berger. "Hormonal Changes in Ageing Men." In The Neuroendocrine Immune Network in Ageing. Elsevier, 2004. http://dx.doi.org/10.1016/s1567-7443(04)80012-7.

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Bartke, A., M. Heiman, D. Turyn, F. Dominici, and J. J. Kopchick. "The Role of Growth Hormone Signaling in the Control of Ageing." In The Neuroendocrine Immune Network in Ageing. Elsevier, 2004. http://dx.doi.org/10.1016/s1567-7443(04)80010-3.

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Hugo Del Río-Araiza, Victor, Romel Hernandéz-Bello, and Jorge Morales-Montor. "New Uses for Old Drugs and Their Application in Helminthology." In Parasitic Helminths and Zoonoses - From Basic to Applied Research [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.106176.

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Parasitic infection research, performed on both humans and domestic animals, has been mostly focused on vaccines, diagnostic methods, epidemiology, and the evolutionary origins of parasites, thanks to the emergence of genomics and proteomics. However, the basic biology of the host-parasite interactions of several medical or veterinary important parasites has not been fully studied. Limited information has been obtained on the intricate neuroimmunoendocrine effects of host-parasite interplay in particular; therefore, the consequences of these interactions, and their possible therapeutic applica
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K. Kanu, Kingsley, and Vincent C. Kanu. "Cultivating Resilience: Exploring Biotic Stressors and Plant Immune Responses." In Physiology. IntechOpen, 2025. https://doi.org/10.5772/intechopen.1006319.

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Plants encounter various biotic stressors, including pathogens, pests, herbivores, and parasites, which significantly threaten their health and productivity. To combat these challenges, plants have evolved sophisticated immune systems that rely on pattern recognition receptors (PRRs) to detect microbe-associated molecular patterns (MAMPs) or damage-associated molecular patterns (DAMPs), initiating pattern-triggered immunity (PTI). A more robust defense mechanism, effector-triggered immunity (ETI), is activated when intracellular nucleotide-binding leucine-rich repeat receptors (NLRs) recognize
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Rozengurt, E. "Neuropeptide growth factors: signalling pathways and role in cancer." In Cell Proliferation in Cancer. Oxford University PressOxford, 1995. http://dx.doi.org/10.1093/oso/9780198547914.003.0009.

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Abstract Multicellular organisms have developed highly efficient regulatory networks to control cell proliferation. These involve cellular interactions with positive and negative diffusible modulators as well as with the extracellular matrix proteins. In fully mature organisms, the cells of many tissues and organs are maintained in a non-proliferating state (the G0/G1 phase of the cell cycle), but can be stimulated to resume DNA synthesis and cell division in response to external stimuli such as hormones, antigens or growth factors. In this manner, the growth of individual cells is regulated a
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Reports on the topic "Hormone immune networks"

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Li, Zifeng, Yuling Fan, Yunhong Lei, Xiaoqiang Hou, and Caiyun Chang. A protocol for systematic review and network meta-analysis Efficacy and Safety of Kunxian Capsule in Treating Patients with Lupus Nephritis：A network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.12.0005.

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Review question / Objective: To study on the efficacy and safety of Kunxian Capsule in treating patients with lupus nephritis( LN). Condition being studied: The study showed that Kunxian capsule significantly improved immune status and renal function and suppressed IL-10 and TNF in serum of lupus mice- α Of secretion, works better in combination with and reduces hormone dosage. At present, there are few studies in which Kunxian capsules are used in the clinic, and the conclusions of the studies are inconsistent, so this study was conducted in order to comprehensively evaluate the efficacy and
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