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Journal articles on the topic 'Host-parasite'

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1

O??CONNER, G. RICHARD. "Host-Parasite Interaction." International Ophthalmology Clinics 25, no. 2 (1985): 63–70. http://dx.doi.org/10.1097/00004397-198502520-00009.

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2

Nutman, Thomas B. "Host-parasite interactions." Trends in Microbiology 5, no. 6 (1997): 250. http://dx.doi.org/10.1016/s0966-842x(97)89537-6.

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3

May, R. M., and R. M. Anderson. "Parasite—host coevolution." Parasitology 100, S1 (1990): S89—S101. http://dx.doi.org/10.1017/s0031182000073042.

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In this paper we wish to develop three themes, each having to do with evolutionary aspects of associations between hosts and parasites (with parasite defined broadly, to include viruses, bacteria and protozoans, along with the more conventionally defined helminth and arthropod parasites). The three themes are: the evolution of virulence; the population dynamics and population genetics of host–parasite associations; and invasions by, or ‘emergence’ of, new parasites.
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4

VRIJENHOEK, ROBERT C. "Host-Parasite Coevolution." Science 232, no. 4746 (1986): 112.1–112. http://dx.doi.org/10.1126/science.232.4746.112.

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5

Cox, F. E. G. "Parasite-host environment." Parasitology Today 8, no. 1 (1992): 35. http://dx.doi.org/10.1016/0169-4758(92)90311-o.

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6

Butcher, G. A. "Host-parasite diplomacy." Parasitology Today 9, no. 8 (1993): 275–76. http://dx.doi.org/10.1016/0169-4758(93)90115-v.

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7

Toft, Catherine A., and Andrew J. Karter. "Parasite-host coevolution." Trends in Ecology & Evolution 5, no. 10 (1990): 326–29. http://dx.doi.org/10.1016/0169-5347(90)90179-h.

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8

Clayton, D. H., S. E. Bush, B. M. Goates, and K. P. Johnson. "Host defense reinforces host-parasite cospeciation." Proceedings of the National Academy of Sciences 100, no. 26 (2003): 15694–99. http://dx.doi.org/10.1073/pnas.2533751100.

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9

Sweet, Andrew D., Sarah E. Bush, Daniel R. Gustafsson, et al. "Host and parasite morphology influence congruence between host and parasite phylogenies." International Journal for Parasitology 48, no. 8 (2018): 641–48. http://dx.doi.org/10.1016/j.ijpara.2018.01.007.

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10

Izhar, Rony, Jarkko Routtu, and Frida Ben-Ami. "Host age modulates within-host parasite competition." Biology Letters 11, no. 5 (2015): 20150131. http://dx.doi.org/10.1098/rsbl.2015.0131.

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In many host populations, one of the most striking differences among hosts is their age. While parasite prevalence differences in relation to host age are well known, little is known on how host age impacts ecological and evolutionary dynamics of diseases. Using two clones of the water flea Daphnia magna and two clones of its bacterial parasite Pasteuria ramosa , we examined how host age at exposure influences within-host parasite competition and virulence. We found that multiply-exposed hosts were more susceptible to infection and suffered higher mortality than singly-exposed hosts. Hosts old
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11

Dallas, Tad, Shan Huang, Charles Nunn, Andrew W. Park, and John M. Drake. "Estimating parasite host range." Proceedings of the Royal Society B: Biological Sciences 284, no. 1861 (2017): 20171250. http://dx.doi.org/10.1098/rspb.2017.1250.

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Estimating the number of host species that a parasite can infect (i.e. host range) provides key insights into the evolution of host specialism and is a central concept in disease ecology. Host range is rarely estimated in real systems, however, because variation in species relative abundance and the detection of rare species makes it challenging to confidently estimate host range. We applied a non-parametric richness indicator to estimate host range in simulated and empirical data, allowing us to assess the influence of sampling heterogeneity and data completeness. After validating our method
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12

McPartland, John M., and Karl W. Hillig. "Host-Parasite Relationships inCannabis." Journal of Industrial Hemp 10, no. 2 (2006): 85–104. http://dx.doi.org/10.1300/j237v10n02_08.

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13

GUZMÁN-CORNEJO, CARMEN, RICHARD G. ROBBINS, and TILA M. PÉREZ. "The Ixodes (Acari: Ixodidae) of Mexico: parasite-host and host-parasite checklists." Zootaxa 1553, no. 1 (2007): 47–58. http://dx.doi.org/10.11646/zootaxa.1553.1.2.

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Parasite-host and host-parasite checklists are provided for all species of Ixodes known from Mexico; host and locality data are from specimens housed in the Colección Nacional de Ácaros, Instituto de Biología, Universidad Nacional Autónoma de México, and from literature. Six Ixodes species (I. brunneus, I. conepati, I. dentatus, I. eadsi, I. guatemalensis, I. texanus) are newly recorded from Mexico; in addition, 17 new locality records are presented for eight species (I. affinis, I. boliviensis, I. luciae, I. rubidus, I. scapularis, I. spinipalpis, I. tancitarius, I. woodi), and eight new host
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14

Guzmán-Cornejo, Carmen, Richard G. Robbins, and Tila M. Pérez. "The Ixodes (Acari: Ixodidae) of Mexico: parasite-host and host-parasite checklists." Zootaxa 1553, no. 1 (2007): 47–58. https://doi.org/10.11646/zootaxa.1553.1.2.

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Guzmán-Cornejo, Carmen, Robbins, Richard G., Pérez, Tila M. (2007): The Ixodes (Acari: Ixodidae) of Mexico: parasite-host and host-parasite checklists. Zootaxa 1553 (1): 47-58, DOI: 10.11646/zootaxa.1553.1.2, URL: https://biotaxa.org/Zootaxa/article/view/zootaxa.1553.1.2
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15

Garnick, Eric. "Parasite Virulence and Parasite-Host Coevolution: A Reappraisal." Journal of Parasitology 78, no. 2 (1992): 381. http://dx.doi.org/10.2307/3283496.

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16

Kaitala, V. "Host-parasite dynamics and the evolution of host immunity and parasite fecundity strategies." Bulletin of Mathematical Biology 59, no. 3 (1997): 427–50. http://dx.doi.org/10.1016/s0092-8240(96)00090-0.

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17

Kaitala, Veijo, Mikko Heino, and Wayne M. Getz. "Host-parasite dynamics and the evolution of host immunity and parasite fecundity strategies." Bulletin of Mathematical Biology 59, no. 3 (1997): 427–50. http://dx.doi.org/10.1007/bf02459459.

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18

Buschinger, Alfred, and Regina G. Kleespies. "Host Range and Host Specificity of an Ant-Pathogenic Gregarine Parasite, Mattesia geminata (Neogregarinida: Lipotrophidae)." Entomologia Generalis 24, no. 2 (1999): 93–104. http://dx.doi.org/10.1127/entom.gen/24/1999/93.

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19

de ROODE, J. C., L. R. GOLD, and S. ALTIZER. "Virulence determinants in a natural butterfly-parasite system." Parasitology 134, no. 5 (2006): 657–68. http://dx.doi.org/10.1017/s0031182006002009.

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SUMMARYMuch evolutionary theory assumes that parasite virulence (i.e. parasite-induced host mortality) is determined by within-host parasite reproduction and by the specific parasite genotypes causing infection. However, many other factors could influence the level of virulence experienced by hosts. We studied the protozoan parasite Ophryocystis elektroscirrha in its host, the monarch butterfly, Danaus plexippus. We exposed monarch larvae to wild-isolated parasites and assessed the effects of within-host replication and parasite genotype on host fitness measures, including pre-adult developmen
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20

White, P. Signe, Angela Choi, Rishika Pandey, et al. "Host heterogeneity mitigates virulence evolution." Biology Letters 16, no. 1 (2020): 20190744. http://dx.doi.org/10.1098/rsbl.2019.0744.

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Parasites often infect genetically diverse host populations, and the evolutionary trajectories of parasite populations may be shaped by levels of host heterogeneity. Mixed genotype host populations, compared to homogeneous host populations, can reduce parasite prevalence and potentially reduce rates of parasite adaptation due to trade-offs associated with adapting to specific host genotypes. Here, we used experimental evolution to select for increased virulence in populations of the bacterial parasite Serratia marcescens exposed to either heterogeneous or homogeneous populations of Caenorhabdi
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21

Hall, Matthew D., and Dieter Ebert. "Disentangling the influence of parasite genotype, host genotype and maternal environment on different stages of bacterial infection in Daphnia magna." Proceedings of the Royal Society B: Biological Sciences 279, no. 1741 (2012): 3176–83. http://dx.doi.org/10.1098/rspb.2012.0509.

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Individuals naturally vary in the severity of infectious disease when exposed to a parasite. Dissecting this variation into genetic and environmental components can reveal whether or not this variation depends on the host genotype, parasite genotype or a range of environmental conditions. Complicating this task, however, is that the symptoms of disease result from the combined effect of a series of events, from the initial encounter between a host and parasite, through to the activation of the host immune system and the exploitation of host resources. Here, we use the crustacean Daphnia magna
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22

DALLAS, TAD, ANDREW W. PARK, and JOHN M. DRAKE. "Predictability of helminth parasite host range using information on geography, host traits and parasite community structure." Parasitology 144, no. 2 (2016): 200–205. http://dx.doi.org/10.1017/s0031182016001608.

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SUMMARYHost–parasite associations are complex interactions dependent on aspects of hosts (e.g. traits, phylogeny or coevolutionary history), parasites (e.g. traits and parasite interactions) and geography (e.g. latitude). Predicting the permissive host set or the subset of the host community that a parasite can infect is a central goal of parasite ecology. Here we develop models that accurately predict the permissive host set of 562 helminth parasites in five different parasite taxonomic groups. We developed predictive models using host traits, host taxonomy, geographic covariates, and parasit
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23

Benesh, Daniel P. "Tapeworm manipulation of copepod behaviour: parasite genotype has a larger effect than host genotype." Biology Letters 15, no. 9 (2019): 20190495. http://dx.doi.org/10.1098/rsbl.2019.0495.

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Compared with uninfected individuals, infected animals can exhibit altered phenotypes. The changes often appear beneficial to parasites, leading to the notion that modified host phenotypes are extended parasite phenotypes, shaped by parasite genes. However, the phenotype of a parasitized individual may reflect parasitic manipulation, host responses to infection or both, and disentangling the contribution of parasite genes versus host genes to these altered phenotypes is challenging. Using a tapeworm ( Schistocephalus solidus ) infecting its copepod first intermediate host, I performed a full-f
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24

Westby, Katie M., Brenden M. Sweetman, Solny A. Adalsteinsson, Elizabeth G. Biro, and Kim A. Medley. "Host food quality and quantity differentially affect Ascogregarina barretti parasite burden, development and within-host competition in the mosquito Aedes triseriatus." Parasitology 146, no. 13 (2019): 1665–72. http://dx.doi.org/10.1017/s0031182019000994.

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AbstractHost condition depends in large part on the quality and quantity of available food and heavily influences the outcome of parasite infection. Although parasite fitness traits such as growth rate and size may depend on host condition, whether host food quality or quantity is more important to parasite fitness and within-host interactions is poorly understood. We provided individual mosquito hosts with a standard dose of a gregarine parasite and reared mosquitoes on two food types of different quality and two quantities. We measured host size, total parasite count and area, and average si
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25

Elliott, David A., and Douglas P. Clark. "Cryptosporidium parvum Induces Host Cell Actin Accumulation at the Host-Parasite Interface." Infection and Immunity 68, no. 4 (2000): 2315–22. http://dx.doi.org/10.1128/iai.68.4.2315-2322.2000.

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ABSTRACT Cryptosporidium parvum is an intracellular protozoan parasite that causes a severe diarrheal illness in humans and animals. Previous ultrastructural studies have shown thatCryptosporidium resides in a unique intracellular compartment in the apical region of the host cell. The mechanisms by which Cryptosporidium invades host intestinal epithelial cells and establishes this compartment are poorly understood. The parasite is separated from the host cell by a unique electron-dense structure of unknown composition. We have used indirect immunofluorescence microscopy and confocal laser scan
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26

Pfenning-Butterworth, Alaina C., T. Jonathan Davies, and Clayton E. Cressler. "Identifying co-phylogenetic hotspots for zoonotic disease." Philosophical Transactions of the Royal Society B: Biological Sciences 376, no. 1837 (2021): 20200363. http://dx.doi.org/10.1098/rstb.2020.0363.

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The incidence of zoonotic diseases is increasing worldwide, which makes identifying parasites likely to become zoonotic and hosts likely to harbour zoonotic parasites a critical concern. Prior work indicates that there is a higher risk of zoonotic spillover accruing from closely related hosts and from hosts that are infected with a high phylogenetic diversity of parasites. This suggests that host and parasite evolutionary history may be important drivers of spillover, but identifying whether host–parasite associations are more strongly structured by the host, parasite or both requires co-phylo
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27

Dallas, Tad A., Anna-Liisa Laine, and Otso Ovaskainen. "Detecting parasite associations within multi-species host and parasite communities." Proceedings of the Royal Society B: Biological Sciences 286, no. 1912 (2019): 20191109. http://dx.doi.org/10.1098/rspb.2019.1109.

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Understanding the role of biotic interactions in shaping natural communities is a long-standing challenge in ecology. It is particularly pertinent to parasite communities sharing the same host communities and individuals, as the interactions among parasites—both competition and facilitation—may have far-reaching implications for parasite transmission and evolution. Aggregated parasite burdens may suggest that infected host individuals are either more prone to infection, or that infection by a parasite species facilitates another, leading to a positive parasite–parasite interaction. However, pa
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28

Halliday, Fletcher W., James Umbanhowar, and Charles E. Mitchell. "A host immune hormone modifies parasite species interactions and epidemics: insights from a field manipulation." Proceedings of the Royal Society B: Biological Sciences 285, no. 1890 (2018): 20182075. http://dx.doi.org/10.1098/rspb.2018.2075.

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Parasite epidemics can depend on priority effects, and parasite priority effects can result from the host immune response to prior infection. Yet we lack experimental evidence that such immune-mediated priority effects influence epidemics. To address this research gap, we manipulated key host immune hormones, then measured the consequences for within-host parasite interactions, and ultimately parasite epidemics in the field. Specifically, we applied plant immune-signalling hormones to sentinel plants, embedded into a wild host population, and tracked foliar infections caused by two common fung
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29

Penley, McKenna J., and Levi T. Morran. "Host mating system and coevolutionary dynamics shape the evolution of parasite avoidance in Caenorhabditis elegans host populations." Parasitology 145, no. 6 (2017): 724–30. http://dx.doi.org/10.1017/s0031182017000804.

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AbstractHosts exhibit a variety of defence mechanisms against parasites, including avoidance. Both host–parasite coevolutionary dynamics and the host mating system can alter the evolutionary trajectories of populations. Does the nature of host–parasite interactions and the host mating system affect the mechanisms that evolve to confer host defence? In a previous experimental evolution study, mixed mating and obligately outcrossing Caenorhabditis elegans host populations adapted to either coevolving or static Serratia marcescens parasite populations. Here, we assessed parasite avoidance as a me
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30

Boots, Michael, and Akira Sasaki. "Parasite‐Driven Extinction in Spatially Explicit Host‐Parasite Systems." American Naturalist 159, no. 6 (2002): 706–13. http://dx.doi.org/10.1086/339996.

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31

Ambroise-Thomas, Pierre. "Emerging parasite zoonoses: the role of host–parasite relationship." International Journal for Parasitology 30, no. 12-13 (2000): 1361–67. http://dx.doi.org/10.1016/s0020-7519(00)00131-4.

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32

Gupta, Sunetra. "Parasite immune escape: new views into host–parasite interactions." Current Opinion in Microbiology 8, no. 4 (2005): 428–33. http://dx.doi.org/10.1016/j.mib.2005.06.011.

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33

SMITH, GARY. "Characteristics of Host-Parasite Interactions That Promote Parasite Persistence." Annals of the New York Academy of Sciences 740, no. 1 Disease in Ev (1994): 242–48. http://dx.doi.org/10.1111/j.1749-6632.1994.tb19874.x.

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34

CAMPIÃO, K. M., A. RIBAS, and L. E. R. TAVARES. "Diversity and patterns of interaction of an anuran–parasite network in a neotropical wetland." Parasitology 142, no. 14 (2015): 1751–57. http://dx.doi.org/10.1017/s0031182015001262.

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SUMMARYWe describe the diversity and structure of a host–parasite network of 11 anuran species and their helminth parasites in the Pantanal wetland, Brazil. Specifically, we investigate how the heterogeneous use of space by hosts changes parasite community diversity, and how the local pool of parasites exploits sympatric host species of different habits. We examined 229 anuran specimens, interacting with 32 helminth parasite taxa. Mixed effect models indicated the influence of anuran body size, but not habit, as a determinant of parasite species richness. Variation in parasite taxonomic divers
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35

Puustinen, Susanna, and Pia Mutikainen. "Host-Parasite-Herbivore Interactions: Implications of Host Cyanogenesis." Ecology 82, no. 7 (2001): 2059. http://dx.doi.org/10.2307/2680069.

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36

Puustinen, Susanna, and Pia Mutikainen. "HOST–PARASITE–HERBIVORE INTERACTIONS: IMPLICATIONS OF HOST CYANOGENESIS." Ecology 82, no. 7 (2001): 2059–71. http://dx.doi.org/10.1890/0012-9658(2001)082[2059:hphiio]2.0.co;2.

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37

McCallum, H. I. "Population effects of parasite survival of host death: experimental studies of the interaction of Ichthyophthirius multifiliis and its fish host." Parasitology 90, no. 3 (1985): 529–47. http://dx.doi.org/10.1017/s0031182000055529.

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Current models of the regulation of host populations by parasite infection assume that death of a host results in death of all the parasites contained within or on the host. This assumption acts as a density-dependent constraint on parasite population growth and contributes to the stability of the interaction between host and parasite populations. The protozoan Ichthyophthirius multifiliis, a parasite of freshwater fish, is one of a number of pathogens in which the reproductive capacity of an individual is not entirely destroyed by the death of its host. The effect of host death on the reprodu
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38

Frank, Steven A. "Evolution of Host-Parasite Diversity." Evolution 47, no. 6 (1993): 1721. http://dx.doi.org/10.2307/2410216.

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39

Andersen, Sandra, and David A. Hughes. "Host specificity of parasite manipulation." Communicative & Integrative Biology 5, no. 2 (2012): 163–65. http://dx.doi.org/10.4161/cib.18712.

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40

Montenegro, Mario R. "Host Parasite Relationship in Paracoccidioidomycosis." Nippon Ishinkin Gakkai Zasshi 36, no. 3 (1995): 209–13. http://dx.doi.org/10.3314/jjmm.36.209.

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41

Lively, Curtis M. "Host-Parasite Coevolution and Sex." BioScience 46, no. 2 (1996): 107–14. http://dx.doi.org/10.2307/1312813.

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42

Speer, Kelly A. "Microbiomes mediate host−parasite interactions." Molecular Ecology 31, no. 7 (2022): 1925–27. http://dx.doi.org/10.1111/mec.16381.

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43

Beaman, B. L., and L. Beaman. "Nocardia species: host-parasite relationships." Clinical Microbiology Reviews 7, no. 2 (1994): 213–64. http://dx.doi.org/10.1128/cmr.7.2.213.

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The nocardiae are bacteria belonging to the aerobic actinomycetes. They are an important part of the normal soil microflora worldwide. The type species, Nocardia asteroides, and N. brasiliensis, N. farcinica, N. otitidiscaviarum, N. nova, and N. transvalensis cause a variety of diseases in both normal and immunocompromised humans and animals. The mechanisms of pathogenesis are complex, not fully understood, and include the capacity to evade or neutralize the myriad microbicidal activities of the host. The relative virulence of N. asteroides correlates with the ability to inhibit phagosome-lyso
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Beaman, B. L., and L. Beaman. "Nocardia species: host-parasite relationships." Clinical Microbiology Reviews 7, no. 2 (1994): 213–64. http://dx.doi.org/10.1128/cmr.7.2.213-264.1994.

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45

Calderone, R. A. "Host-Parasite Relationships in Candidosis." Mycoses 32 (April 24, 2009): 12–17. http://dx.doi.org/10.1111/j.1439-0507.1989.tb02303.x.

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46

Shea-Donohue, Terez, and Joseph F. Urban,. "Gastrointestinal parasite and host interactions." Current Opinion in Gastroenterology 20, no. 1 (2004): 3–9. http://dx.doi.org/10.1097/00001574-200401000-00003.

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47

Stanley, Samuel L., and Sharon L. Reed. "VI.Entamoeba histolytica: parasite-host interactions." American Journal of Physiology-Gastrointestinal and Liver Physiology 280, no. 6 (2001): G1049—G1054. http://dx.doi.org/10.1152/ajpgi.2001.280.6.g1049.

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The protozoan intestinal parasite Entamoeba histolytica remains a significant cause of morbidity and mortality worldwide. E. histolytica causes two major clinical syndromes, amebic colitis and amebic liver abscess. Recent advances in the development of in vitro and in vivo models of disease, new genetic approaches, the identification of key E. histolytica virulence factors, and the recognition of crucial elements of the host response to infection have led to significant insights into the pathogenesis of amebic infection. E. histolytica virulence factors include 1) a surface galactose binding l
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48

Sjöstedt, A., A. Tärnvik, and G. Sandström. "Francisella tularensis: Host—parasite interaction." FEMS Immunology & Medical Microbiology 13, no. 3 (1996): 181–84. http://dx.doi.org/10.1111/j.1574-695x.1996.tb00233.x.

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49

Franco, M. "Host-parasite relationships in paracoccidioidomycosis." Medical Mycology 25, no. 1 (1987): 5–18. http://dx.doi.org/10.1080/02681218780000021.

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50

Blair, John E. "HOST-PARASITE RELATIONSHIPS: A SUMMATION." Annals of the New York Academy of Sciences 128, no. 1 (2006): 451–56. http://dx.doi.org/10.1111/j.1749-6632.1965.tb11654.x.

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