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1

1946-, Barnes Peter J., Page C. P, and Henson P. M. 1940-, eds. Platelet activating factor and human disease. Blackwell Scientific, 1989.

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2

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119375913.

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3

Scott, N. J. Investigations on human factor VIII and platelet function. North East London Polytechnic, 1985.

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4

Baker, Jennifer Mary. The effect of extracellular pH on human platelet metabolism. University of Birmingham, 2000.

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5

European Thrombosis Research Organisation. Workshop. Monoclonal antibodies and human blood platelets: Proceedings of the European Thrombosis Research Organisation Workshop, under the patronage of the Institut national de la santé et de la recherche médicale, held in Lyon (France), 26-27 September 1985. Edited by McGregor John Louis and Institut national de la santé et de la recherche médicale (France). Elsevier Science Publishers, 1986.

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6

Stott, Andrew James. Studies on human blood platelets. typescript, 1990.

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7

C, Somogyi J., Renaud Serge, and Astier-Dumas M, eds. Emerging problems in human nutrition. Karger, 1987.

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8

L, Gordon J., ed. Vascular endothelium: Interactions with circulating cells. Elsevier, 1991.

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9

Bjorn, Neu, and Meiselman Herbert J, eds. Red blood cell aggregation. CRC Press, 2012.

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10

1946-, Barnes P. J., Page Clive P, and Henson P. M. 1940-, eds. Plateletactivating factor and human disease. Blackwell Scientific, 1989.

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11

(Editor), C. P. Page, P. Barnes (Editor), and P. Henson (Editor), eds. Platelet Activating Factor & Human Disease, Frontiers in Pharmacology & Therapeutics. Blackwell Science, 1989.

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12

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function: Applications in Cardiovascular Health. Wiley & Sons, Incorporated, John, 2018.

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13

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function: Applications in Cardiovascular Health. Wiley & Sons, Limited, John, 2018.

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14

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function: Applications in Cardiovascular Health. Wiley & Sons, Incorporated, John, 2018.

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15

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function: Applications in Cardiovascular Health. Wiley & Sons, Limited, John, 2018.

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16

O'Flaherty, Joseph T., and Peter W. Ramwell. Paf Antagonists: New Developments for Clinical Application (Advances in Applied Biotechnology Series, Vol 9). Portfolio Pub Co, 1991.

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17

Goziotis, Ann. A longitudinal study of intact platelet u︠3r︠H-imipramine binding in normal human subjects. 1986.

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18

Geet, C. Van. Signal Transduction in Human Endothelial Cells and Endothelial Cell and Platelet Activation in Vivo in Man. Leuven University Press, 1990.

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19

Gawaz, Meinrad, Andreas Zirlik, and Christoph Bode. Platelets, Haemostasis and Inflammation. Springer, 2019.

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20

Gawaz, Meinrad, Andreas Zirlik, and Christoph Bode. Platelets, Haemostasis and Inflammation. Springer, 2018.

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21

Page, Clive, and Joseph M. Immunopharmacology of Platelets. Elsevier Science & Technology Books, 1995.

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22

Immunopharmacology of Platelets (Handbook of Immunopharmacology). Academic Press, 1995.

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23

Macdougall, Iain C. Clinical aspects and overview of renal anaemia. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0123.

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Anaemia is an almost ubiquitous complication of chronic kidney disease, which has a number of implications for the patient. It is associated with adverse outcomes, an increased rate of red cell transfusions, poor quality of life, and reduced physical capacity. Severe anaemia also impacts on cardiac function, as well as on platelet function, the latter contributing to the bleeding diathesis of uraemia. Renal anaemia occurs mainly in the later stages of chronic kidney disease (stages 3B, 4, and 5), and up to 95% of patients on dialysis suffer from this condition. It is caused largely by inapprop
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24

PlateletVessel Wall Interactions in Hemostasis and Thrombosis. Morgan & Claypool, 2010.

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25

Alikhan, Raza. Normal haemostatic function. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0283.

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Humans have evolved an intricate system that maintains blood in a fluid state. This relies on an intact vascular endothelium modulating vascular tone and forming a barrier between blood components and reactive subendothelial components. It also involves the production of inhibitors of both blood coagulation and platelet aggregation. In addition, haemostatic systems are primed to convert blood from its fluid state to a solid state, to allow the formation of a haemostatic plug, following vessel injury, to stem the flow of blood from or within a blood vessel. This chapter reviews the physiology o
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26

Lutgens, Esther, Marie-Luce Bochaton-Piallat, and Christian Weber. Atherosclerosis: cellular mechanisms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0013.

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Atherosclerosis is a lipid-driven, chronic inflammatory disease of the large and middle-sized arteries that affects every human being and slowly progresses with age. The disease is characterized by the presence of atherosclerotic plaques consisting of lipids, (immune) cells, and debris that form in the arterial intima. Plaques develop at predisposed regions characterized by disturbed blood flow dynamics, such as curvatures and branch points. In the past decades, experimental and patient studies have revealed the role of the different cell-types of the innate and adaptive immune system, and of
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27

El Kenz, Hanane, and Philippe Van der Linden. The physiology of blood in anaesthetic practice. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0011.

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Following the discovery of the ABO blood groups by Landsteiner in 1901, Albert Hustin described the first transfusion of a whole blood unit in 1914. The modern transfusion era really begins in 1916 with the discovery of sodium citrate as an anticoagulant by the same physician, allowing blood conservation in dedicated packs. Since that time, many advances have been made especially over the past two decades in the storage, the conservation, and the laboratory testing of blood components and in transfusion medicine practice. Transfusion of whole blood has been replaced by blood component therapy,
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