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Journal articles on the topic 'Humoral and cellular immunity'

Author: Grafiati

Published: 12 June 2021

Last updated: 3 February 2022

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1

Colombo, Daniela, Alessandra Fritsch, Karen Gomes Ordovas, Allesandra Spode, and Maria Lucia Scroferneker. "Playing with cellular and humoral immunity." Biochemical Education 26, no. 1 (1998): 20–21. http://dx.doi.org/10.1016/s0307-4412(97)00140-4.

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2

Veien, N. K., F. Hardt, G. Bendixen, et al. "Humoral and Cellular Immunity in Sarcoidosis." Acta Medica Scandinavica 203, no. 1-6 (2009): 321–26. http://dx.doi.org/10.1111/j.0954-6820.1978.tb14881.x.

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3

Kiilstra, Aize. "Ocular humoral immunity." Experimental Eye Research 55 (September 1992): 29. http://dx.doi.org/10.1016/0014-4835(92)90311-f.

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4

Maria Angeles Gomez Morales and Edoardo Pozio. "Humoral and Cellular Immunity Against Cryptosporidium Infection." Current Drug Target - Immune, Endocrine & Metabolic disorders 2, no. 3 (2002): 291–301. http://dx.doi.org/10.2174/1568005310202030291.

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Angeles, M., G. Morales, and E. Pozio. "Humoral and Cellular Immunity Against Cryptosporidium Infection." Current Drug Targets-Immune, Endocrine & Metabolic Disorders 2, no. 3 (2002): 291–301. http://dx.doi.org/10.2174/1568008023340505.

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6

Skovmann-Sørensen, O., H. Schrøder, Anné Møller-larsen, and S. Haahr. "Cellular and Humoral Immunity in Hodgkin's Disease." Scandinavian Journal of Haematology 27, no. 3 (2009): 171–80. http://dx.doi.org/10.1111/j.1600-0609.1981.tb00469.x.

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7

NAKANISHI, Yoshinobu. "Humoral and Cellular Responses in Innate Immunity." YAKUGAKU ZASSHI 126, no. 12 (2006): 1207–12. http://dx.doi.org/10.1248/yakushi.126.1207.

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8

Seledtsov, V. I., L. S. Litvinova, I. A. Seledtsova, E. V. Kirienkova, and V. V. Shupletsova. "HUMORAL AND CELLULAR IMMUNITY FACTORS IN MYOCARDIAL INFARCTION." Medical Immunology (Russia) 12, no. 6 (2014): 477. http://dx.doi.org/10.15789/1563-0625-2010-6-477-484.

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9

Steelman, Samantha M., Daisy Johnson, Bettina Wagner, AshleyM Stokes, and Bhanu P. Chowdhary. "Cellular and humoral immunity in chronic equine laminitis." Veterinary Immunology and Immunopathology 153, no. 3-4 (2013): 217–26. http://dx.doi.org/10.1016/j.vetimm.2013.03.001.

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10

Dola, O. L. "The state of the immunity system in women with latent Papillomavirus infection of the cervix." HEALTH OF WOMAN, no. 7(123) (September 30, 2017): 135–38. http://dx.doi.org/10.15574/hw.2017.123.135.

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The article presents modern aspects of the state of immunity in women with latent papillomavirus infection (PVI) of the cervix. The objective: the study of the indicators characterizing the state of immunity in women with latent PVI of the cervix, often associated with urogenital infections (UGI), at the beginning of the survey and after 6 months of observation. Patients and methods. The study of cellular and humoral immunity was performed in 210 women with latent papillomavirus infection (PVI) and 15 healthy women at the beginning of the examination and 6 months later. 84 women were diagnosed with a monoinfection, 126 had a combined PVI and a urogenital infection (UGI). In 140 women transient PVI is established, in 70 – persistent. Results. In women with latent cervical pylori cervix, disorders of cellular immunity were observed (a slight decrease in the relative amount of CD3+, CD4+ lymphocytes and a simultaneous increase in the number of CD8+ lymphocytes, natural killers and B-lymphocytes) against the background of activation of the humoral immunity unit. Nonspecific protection of the female body with PVI was characterized by inhibition of the phagocytic reaction of neutrophils (Nf) and monocytes (Mg) against the background of an increase in oxygen-dependent metabolism, primarily HF, and a decrease in the functional reserve for both NF and MZ. The most pronounced disorders were found for patients with combined PVI and UGI. Conclusion. In women with transient PVI normalization of cellular and humoral immunity was observed, and the further persistence of the human papillomavirus in the genitals led to a more significant inhibition of phagocytic and HCT activity of phagocytes, as well as to a decrease in some parameters of the cellular and humoral immunity units. Key words: papillomavirus infection, cervix, cellular and humoral immunity.

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Sarkar, Amrita, and Khadija Rafiq. "Humoral Immunity in Heart Failure." Cardiovascular & Hematological Disorders-Drug Targets 19, no. 1 (2019): 14–18. http://dx.doi.org/10.2174/1871529x18666180518101527.

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Cardiovascular Disease (CVD) is a class of diseases that involve disorders of heart and blood vessels, including hypertension, coronary heart disease, cerebrovascular disease, peripheral vascular disease, which finally lead to Heart Failure (HF). There are several treatments available all over the world, but still, CVD and heart failure became the number one problem causing death every year worldwide. Both experimental and clinical studies have shown a role for inflammation in the pathogenesis of heart failure. This seems related to an imbalance between pro-inflammatory and anti-inflammatory cytokines. Cardiac inflammation is a major pathophysiological mechanism operating in the failing heart, regardless of HF aetiology. Disturbances of the cellular and humoral immune system are frequently observed in heart failure. This review describes how B-cells play a specific role in the heart failure states. There is an urgent need to identify novel therapeutic targets and develop advanced therapeutic strategies to combat the syndrome of HF. Understanding and describing the elements of the humoral immunity function are essential and may suggest potential new treatment strategies.

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12

Lendvai, Nikoletta, Sacha Gnjatic, Erika Ritter, et al. "Host Immune Responses Against CT Antigens in Multiple Myeloma Patients." Blood 108, no. 11 (2006): 3492. http://dx.doi.org/10.1182/blood.v108.11.3492.3492.

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Abstract The type I Melanoma Antigen GEne (MAGE) proteins belong to the Cancer-Testis (CT) family of tumor-associated antigens and are widely expressed in solid and hematologic malignancies. They are immunogenic and frequently elicit spontaneous immune responses in patients with CT antigen-expressing tumors, particularly in malignant melanoma. In melanoma patients, there is high concordance between humoral and cellular immunity. Based on these findings, CT antigens are widely investigated as potential antigenic targets for tumor-specific therapeutic vaccines. We previously showed that the type I MAGE proteins CT7 (MAGE-C1), CT10 (MAGE-C2) and MAGE-A3 were commonly detected in primary myeloma specimens, and expression of CT7 and MAGE-A3 was correlated with abnormally elevated plasma cell proliferation. These findings suggest that type I MAGE may be rational targets for vaccine therapy in multiple myeloma. Therefore, it is important to determine if type I MAGE elicit cellular or humoral immune responses in myeloma patients. To investigate this hypothesis, we assessed cellular immunity against CT7 and humoral immunity against a broad panel of CT antigens. To quantify CT7-specific cellular immunity, expanded, polyclonal pools of T cells from the bone marrow, the tumor microenvironment, were co-cultured in interferon gamma (IFNγ) ELISpot assays with autologous antigen-presenting cells (APC) transduced with in vitro transcribed mRNA coding for CT7 or control antigens. CT antigen-specific humoral immunity was examined by ELISA assay using patient serum or plasma and recombinant CT antigens. This analysis demonstrated that 2/9 patients exhibited specific T cell immunity against CT7 in their bone marrow lymphocytes as measured by IFNγ secretion. These same two patients had positive titers for other CT antigens; one for MAGE-A1 (another type I MAGE), the other for SSX-1 (a structurally distinct CT antigen). Interestingly, neither patient had positive serology for CT7. Serum from 16 other myeloma patients did not have detectible antibody titers for a broad panel of CT antigens. These results show that CT antigens are immunogenic in myeloma patients, with cellular responses against CT7 and humoral responses against MAGE-A1 and SSX-1. However, unlike other types of cancer, there appears to be discordance between humoral and cellular immunity against CT7 in multiple myeloma. This may be due in part to the significant derangements of humoral immunity in this disease. These results support further investigation of immunologic therapies targeting type I MAGE in myeloma, especially therapeutic vaccine strategies.

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Stogov, Maksim V., and Natalya D. Nenenko. "Features of cellular and humoral immunity athletes Khanty-Ugra." Yugra State University Bulletin 12, no. 1 (2016): 201–3. http://dx.doi.org/10.17816/byusu2016121201-203.

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The study found some features of the immune status of women with different levels of physical activity, living in climatic conditions of Khanty-Ugra, regarding the reference values. Marked changes in cellular and humoral immunity can be regarded as a manifestation of the adaptive immune response to ecological conditions of the region. Sport activities help to maintain and improve the adaptability of the female body to the climate-geographical conditions of Khanty-Ugra.

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14

KUZMINOV, Yu. "Cellular and humoral immunity in infants with acute bronchitis." Experimental and Clinical Physiology and Biochemistry 2015, no. 2 (2015): 84–88. http://dx.doi.org/10.25040/ecpb2015.02.084.

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15

Shearer, Gene M., and Mario Clerici. "Vaccine strategies: Selective elicitation of cellular or humoral immunity?" Trends in Biotechnology 15, no. 3 (1997): 106–9. http://dx.doi.org/10.1016/s0167-7799(97)01011-1.

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16

Nielsen, Johan Lanng, and Sven Haahr. "Interferon Production, Cellular and Humoral Immunity in Splenectomized Patients." Scandinavian Journal of Haematology 29, no. 2 (2009): 168–74. http://dx.doi.org/10.1111/j.1600-0609.1982.tb00579.x.

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17

Berezniakova, AI, VF Cheremisina, and OD Zhemela. "Cellular and humoral immunity in rats with atopic dermatitis." Fiziolohichnyĭ zhurnal 60, no. 4 (2014): 50–55. http://dx.doi.org/10.15407/fz60.04.050.

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18

Lindesmith, Lisa, Christine Moe, Jacques LePendu, Jeffrey A. Frelinger, John Treanor, and Ralph S. Baric. "Cellular and Humoral Immunity following Snow Mountain Virus Challenge." Journal of Virology 79, no. 5 (2005): 2900–2909. http://dx.doi.org/10.1128/jvi.79.5.2900-2909.2005.

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ABSTRACT Little is known about the immune response to noroviruses. To elucidate the immunobiology of norovirus infection in humans, 15 volunteers were challenged with Snow Mountain virus (SMV), a genogroup 2 norovirus. We assessed the cellular and humoral immune response and infection by analyzing stool, serum, saliva, and peripheral blood mononuclear cell (PBMC) responses pre- and postchallenge. In contrast to Norwalk virus (NV), SMV infection was not dependent upon blood group secretor status. Nine of 15 volunteers were infected and showed a ≥4-fold increase over the prechallenge anti-SMV serum immunoglobulin G (IgG) titer, mostly subclass IgG1. Although serum IgG elicited by SMV infection was cross-reactive with Hawaii virus (HV), another genogroup 2 norovirus, salivary IgA was less cross-reactive. Neither SMV-elicited serum IgG nor salivary IgA cross-reacted with NV, a genogroup 1 norovirus. Significant increases in serum gamma interferon (IFN-γ) and IL-2, but not IL-6 or IL-10, were noted on day 2 postchallenge. For the majority of volunteers, both infected and uninfected, PBMCs stimulated with norovirus virus-like particles secreted IFN-γ and other Th1 cytokines, suggesting previous norovirus exposure in most volunteers. Like the IgG antibodies, the SMV-activated T cells were cross-reactive with HV but not NV. IFN-γ production was dependent upon CD4+ cells, consistent with a predominant, but not exclusive, Th1 response. To our knowledge, this is the first report characterizing T-cell and cytokine responses following live norovirus challenge.

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19

Dallago, B. S. L., C. M. McManus, D. F. Caldeira, et al. "Humoral and Cellular Immunity in Chromium Picolinate-Supplemented Lambs." Biological Trace Element Research 154, no. 2 (2013): 196–201. http://dx.doi.org/10.1007/s12011-013-9731-7.

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20

Plommet, M. "Brucellosis and immunity: Humoral and cellular components in mice." Annales de l'Institut Pasteur / Microbiologie 138, no. 1 (1987): 105–10. http://dx.doi.org/10.1016/0769-2609(87)90086-x.

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21

Salmon, Henri, Mustapha Berri, Volker Gerdts, and François Meurens. "Humoral and cellular factors of maternal immunity in swine." Developmental & Comparative Immunology 33, no. 3 (2009): 384–93. http://dx.doi.org/10.1016/j.dci.2008.07.007.

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22

Zavadova, Eva, Jan Spacek, Michal Vocka, Bohuslav Konopasek, and Lubos Petruzelka. "Decreased cellular and humoral immunity in patients with breast cancer." Journal of Clinical Oncology 36, no. 5_suppl (2018): 4. http://dx.doi.org/10.1200/jco.2018.36.5_suppl.4.

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4 Background: A growing body of evidence over the past few years suggests that the presence of immune elements within the tumor or in the tumor stroma has prognostic and predictive value in breast cancer. Immunotherapy is successfully used in many types of cancer, and modulation of immune system became standard part of the cancer patients treatment Responses in breast cancer have been more recently reported. However it has yet to be determined whether predictable biomarkers of response can be identified. Therefore in recent years, research focused on a precise description of the status and function of the immune system.The purpose of the study was to monitor immune responses in patients with breast cancer, particularly the examination of cellular (CD4, CD8, B cells) as well as humoral immunity (IgG, IgG1, IgG2, IgG3, IgG4. It appears that a factor contributing to the immunosupression may be a transforming factor-beta (TGF-beta).It is highly immunosuppressive factor that inhibits the natural and specific immunity against tumors. Methods: 50 patients included in the research project were implemented routine cancer treatment. Basic parameters (histological type and grade, the degree of expression of ER and PR, HER2, and the proliferative marker) were established. Patients were evaluated by a cancer clinical immunologist to exclude immune disorders, allergic or autoimmune origin. Anti-tumor cellular immunity (CD4, CD8, CD19) was measured by flow citometry, humoral immunity (IgG, IgG1, IgG2, IgG3, IgG4) was measured and TGF beta and VEGF production was monitored by ELISA. Results: In breast cancer patients mainly depression in cellular immunity was found. Immunglobuline plasma level was decreased as well (mainly IgG4 subtype). TGF beta as well as VEGF plasma level were increased. Most patients have shown clinical symptoms of immunodeficiency (frequent infections of respiratory or urinary tract, herpetic infections).Those patient could benefit from immunomodulation. Conclusions: The state of anticancer immunity could contribute to the selection of targeted immune therapy in breast cancer patients and to help to find optimal combination of immunotherapy. This project was supported by governmental grant AZV CR 15-28188A.

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23

Potyomkina, Ye Ye, D. S. Rafibekov, Ye Ye Fomina, N. V. Pesheva, and A. P. Kalinin. "Humoral and cellular immune factors in autoimmune thyroiditis." Problems of Endocrinology 41, no. 1 (1995): 9–12. http://dx.doi.org/10.14341/probl11324.

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Special immunological methods of investigation were used in the examinations of 102 patients with autoimmune thyroiditis. Group 1 were 43 patients with euthyrosis, group 2 18 subjects with compensated, and group 3 41 patients with decompensated hypothyrosis. Humoral, cell-mediated, and phagocytic factors of the immunity were assessed by 16 parameters, and the findings interpreted in correlation with the findings of similar examinations of 35 donors. Marked changes in the T-cellular and humoral components of the immunity system were revealed, which depended on the function of the thyroid. These changes were the most expressed in decompensated hypothyrosis. Individual analysis showed that euthyrosis and compensated hypothyrosis are more often associated with increased immunologic reaction. The absence of E- receptor hyperreactivity in compensated hypothyrosis and a sharp reduction of T-gamma lymphocyte subpopulation may be explained by the development of adaptation mechanisms. The authors necessitate simultaneous use of the cytotoxic test and E-RFC test for the assesstment of T-cell populations. They emphasize the importance of immunologic monitoring over the course of treatment of patients with autoimmune thyroiditis.

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24

Wang, Zhi-Biao, and Jing Xu. "Better Adjuvants for Better Vaccines: Progress in Adjuvant Delivery Systems, Modifications, and Adjuvant–Antigen Codelivery." Vaccines 8, no. 1 (2020): 128. http://dx.doi.org/10.3390/vaccines8010128.

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Traditional aluminum adjuvants can trigger strong humoral immunity but weak cellular immunity, limiting their application in some vaccines. Currently, various immunomodulators and delivery carriers are used as adjuvants, and the mechanisms of action of some of these adjuvants are clear. However, customizing targets of adjuvant action (cellular or humoral immunity) and action intensity (enhancement or inhibition) according to different antigens selected is time-consuming. Here, we review the adjuvant effects of some delivery systems and immune stimulants. In addition, to improve the safety, effectiveness, and accessibility of adjuvants, new trends in adjuvant development and their modification strategies are discussed.

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25

Мирсаева, Фания, Faniya Mirsaeva, Тимур Ханов, and Timur Hanov. "CONDITION OF CELLULAR AND HUMORAL RELATIONSHIPS OF IMMUNITY IN CANDIDA ASSOCIATED PARODONTITIS WITH MEDIUM AND HIGH DEGREE OF QUANTITATIVE REPLACEMENT OF PARADONTAL POCKETS." Actual problems in dentistry 15, no. 2 (2019): 122–27. http://dx.doi.org/10.18481/2077-7566-2019-15-2-122-127.

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According to many researchers, the change in the course of chronic generalized periodontitis is associated with changes in the patient's immune status. As a result, colonization resistance decreases, the etiological role of opportunistic pathogens, fungal associations, which act synergistically with periodontal pathogenic bacteria and contribute to the development of candida-associated periodontitis, increases. Due to the fact that this disease has little specific symptoms, relapsing course and does not respond to standard therapy, characteristic disorders in the immune system can serve as a diagnostic criterion as well as a criterion for evaluating the effectiveness of the treatment. However, to date, this issue has not been studied, taking into account the degree of quantitative contamination of periodontal pockets with fungi of the genus Candida, which was the subject of this study.  The aim is to study the state of cellular and humoral immunity during the exacerbation of candida - associated periodontitis with an average and high degree of dissemination of periodontal pockets by Candida fungi.  Methodology. A survey of cellular and humoral immunity units of 25 people with intact periodontal disease and 60 patients with exacerbation of candida-associated periodontitis, taking into account the degree of dissemination of periodontal pockets by Candida fungi. Modern and informative immunological methods of research have been used, such as T and B lymphocytes and their subpopulations, phagocytic unit, humoral factors of systemic and local immunity.  Results. An immunological study revealed that the exacerbation of candida-associated periodontitis is accompanied by changes in cellular and humoral immunity. The severity and nature of the changes depend on the degree of dissemination of periodontal pockets by Candida fungi. Conclusions. The obtained data on changes in the indices of cellular and humoral immunity can serve as a diagnostic criterion and a criterion for evaluating the effectiveness of the treatment being carried out.

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26

Xu, Rong, Aaron J. Johnson, Denny Liggitt, and Michael J. Bevan. "Cellular and Humoral Immunity against Vaccinia Virus Infection of Mice." Journal of Immunology 172, no. 10 (2004): 6265–71. http://dx.doi.org/10.4049/jimmunol.172.10.6265.

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27

Romanchuk, N. "Healthy Microbiota and Natural Functional Nutrition: Humoral and Cellular Immunity." Bulletin of Science and Practice 6, no. 9 (2020): 127–66. http://dx.doi.org/10.33619/2414-2948/58/14.

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The human immune system and microbiota jointly evolve, and their balanced systemic interaction occurs throughout life. This close association of both overall composition and microbiota richness plays an important role in modulating host immunity and can influence the immune response in vaccination. The availability of innovative technologies, such as next-generation sequencing and correlated bioinformatics tools, allows deeper investigation of the cross-network relationships between the microbiota and human immune responses. A new managed healthy biomicrobiota and personalized functional and balanced “brain and microbiota” nutrition is a patient’s long-term medical program that allows the combined use of nutritional epigenetics and pharmacepigenetics, and most importantly, the prevention of polypharmacy. A functional food product using biomarkers and artificial intelligence technologies is a targeted nutrient medium for both the body as a whole and biomicrobiota in particular. Lifestyle and environmental factors leave epigenetic traces on our DNA that affect gene expression, some have protective effects and others are harmful. Genetic and epigenetic factors that ensure longevity and super-longevity require a reasonable new interaction with nature and society, and responsibility for future healthy generations. In the studies of P. I. Romanchuk, it was shown that an increase in the average life expectancy of a person and neuroendocrine changes in physiological and pathological aging, on the one hand, epigenetic factors and electromagnetic information load/overload, on the other hand, made a significant contribution to the circadian nature of the neural network interaction of the human brain with artificial intelligence. The microbiota is a key element potentially capable of affecting antigen functions to elicit a protective immune response and the ability of the immune system to adequately respond to antigenic stimulation (vaccine efficacy) by acting as an immunological modulator as well as a natural vaccine adjuvant. The mechanisms underlying the crosstalk between the gut microbiota and the immune system play a crucial role, especially at an early age (early gut microbiota forms immunological functions). New interactions, along with other genetic and environmental factors, lead to a certain composition and richness of the microbiota, which can diversify the individual response to vaccinations. Variations in microbial communities may partly explain the geographical heterogeneity in vaccination success, and a deep understanding of this dynamics may be a tool for improving immunization strategies.

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KITAO, HIROYO, MITSUO MATSUDA, ICHIRO KONO, SHUKOU HAGA, and HIDEO FUKUSHIMA. "EFFECTS OF HABITUAL EXERCISE TRANING ON CELLULAR AND HUMORAL IMMUNITY." Japanese Journal of Physical Fitness and Sports Medicine 36, no. 4 (1987): 217–20. http://dx.doi.org/10.7600/jspfsm1949.36.217.

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29

Bansal, AmolakS, PhilipB Wilson, RichardS H. Pumphrey, et al. "Cellular and humoral immunity in patients with insulin-dependent diabetes." Lancet 342, no. 8865 (1993): 246. http://dx.doi.org/10.1016/0140-6736(93)92340-y.

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Şatir, T., T. Oral, A. Verimli, M. Arasli, and G. Deniz. "The cellular and humoral immunity in drug free affective patients." Biological Psychiatry 42, no. 1 (1997): 251S. http://dx.doi.org/10.1016/s0006-3223(97)87958-9.

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31

Paik, In-ho, Kyu-young Toh, Chul Lee, Jung-jin Kim, and Soo-jung Lee. "Psychological Stress May Induce Increased Humoral and Decreased Cellular Immunity." Behavioral Medicine 26, no. 3 (2000): 139–41. http://dx.doi.org/10.1080/08964280009595761.

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32

Torres, P., F. Guerra, A. Arenas, et al. "Humoral and Cellular Immunity in Different Phases of Contact Dermatitis." Dermatitis 2, no. 3 (1991): 187–91. http://dx.doi.org/10.1097/01206501-199109000-00007.

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Torres, P., F. Guerra, A. Arenas, et al. "Humoral and Cellular Immunity in Different Phases of Contact Dermatitis." American Journal of Contact Dermatitis 2, no. 3 (1991): 187–91. http://dx.doi.org/10.1097/01634989-199109000-00007.

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34

Zittoun, J., M. Tulliez, B. Estournet, and M. Goulon. "Humoral and Cellular Immunity to Intrinsic Factor in Myasthenia Gravis." Scandinavian Journal of Haematology 23, no. 5 (2009): 442–48. http://dx.doi.org/10.1111/j.1600-0609.1979.tb02747.x.

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Arvin, Ann M. "Humoral and Cellular Immunity to Varicella‐Zoster Virus: An Overview." Journal of Infectious Diseases 197, s2 (2008): S58—S60. http://dx.doi.org/10.1086/522123.

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36

Smith, Sylvia L. "Cellular and humoral aspects of innate immunity in the shark." Fish & Shellfish Immunology 10, no. 3 (2000): 287. http://dx.doi.org/10.1006/fsim.1999.0257.

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Terada, Kihei, Kimiko Hagihara, Tomohiro Oishi, et al. "Cellular and humoral immunity after vaccination or natural mumps infection." Pediatrics International 59, no. 8 (2017): 885–90. http://dx.doi.org/10.1111/ped.13306.

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38

Ramsay, Alistair J., Stephen J. Kent, Richard A. Strugnell, et al. "Genetic vaccination strategies for enhanced cellular, humoral and mucosal immunity." Immunological Reviews 171, no. 1 (1999): 27–44. http://dx.doi.org/10.1111/j.1600-065x.1999.tb01341.x.

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39

Fang, Hao, Ming Tan, Ming Xia, Leyi Wang, and Xi Jiang. "Norovirus P Particle Efficiently Elicits Innate, Humoral and Cellular Immunity." PLoS ONE 8, no. 4 (2013): e63269. http://dx.doi.org/10.1371/journal.pone.0063269.

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McMillen, G. L., D. J. Briggs, D. S. McVey, R. M. Phillips, and F. R. Jordan. "Vaccination of racing greyhounds: effects on humoral and cellular immunity." Veterinary Immunology and Immunopathology 49, no. 1-2 (1995): 101–13. http://dx.doi.org/10.1016/0165-2427(95)05446-d.

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Shlobin, O. A., N. Lechtzin, J. B. Orens, B. B. Detrick, and W. M. Baldwin. "Humoral immunity during acute cellular rejection in lung transplant patients." Journal of Heart and Lung Transplantation 24, no. 2 (2005): S53. http://dx.doi.org/10.1016/j.healun.2004.11.059.

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Rykova, M. P., E. N. Antropova, I. M. Larina, and B. V. Morukov. "Humoral and cellular immunity in cosmonauts after the ISS missions." Acta Astronautica 63, no. 7-10 (2008): 697–705. http://dx.doi.org/10.1016/j.actaastro.2008.03.016.

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Alsayb, May A., Ali Dakhilallah D. Alsamiri, Hatem Q. Makhdoom, Turki Alwasaidi, Haitham Mohammed Osman, and Waleed H. Mahallawi. "Prolonged humoral and cellular immunity in COVID-19-recovered patients." Saudi Journal of Biological Sciences 28, no. 7 (2021): 4010–15. http://dx.doi.org/10.1016/j.sjbs.2021.04.008.

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Chen, Zhiqing, Jie Xiao, Han Liu та ін. "Astaxanthin attenuates oxidative stress and immune impairment in d-galactose-induced aging in rats by activating the Nrf2/Keap1 pathway and suppressing the NF-κB pathway". Food & Function 11, № 9 (2020): 8099–111. http://dx.doi.org/10.1039/d0fo01663b.

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Parent, Michelle A., Kiera N. Berggren, Lawrence W. Kummer, et al. "Cell-Mediated Protection against Pulmonary Yersinia pestis Infection." Infection and Immunity 73, no. 11 (2005): 7304–10. http://dx.doi.org/10.1128/iai.73.11.7304-7310.2005.

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ABSTRACT Pulmonary infection with the bacterium Yersinia pestis causes pneumonic plague, an often-fatal disease for which no vaccine is presently available. Antibody-mediated humoral immunity can protect mice against pulmonary Y. pestis infection, an experimental model of pneumonic plague. Little is known about the protective efficacy of cellular immunity. We investigated the cellular immune response to Y. pestis in B-cell-deficient μMT mice, which lack the capacity to generate antibody responses. To effectively prime pulmonary cellular immunity, we intranasally vaccinated μMT mice with live replicating Y. pestis. Vaccination dramatically increased survival of μMT mice challenged intranasally with a lethal Y. pestis dose and significantly reduced bacterial growth in pulmonary, splenic, and hepatic tissues. Vaccination also increased numbers of pulmonary T cells, and administration of T-cell-depleting monoclonal antibodies at the time of challenge abrogated vaccine-induced survival. Moreover, the transfer of Y. pestis-primed T cells to naive μMT mice protected against lethal intranasal challenge. These findings establish that vaccine-primed cellular immunity can protect against pulmonary Y. pestis infection and suggest that vaccines promoting both humoral and cellular immunity will most effectively combat pneumonic plague.

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Uribe, C., H. Folch, R. Enriquez, and G. Moran. " Innate and adaptive immunity in teleost fish: a review." Veterinární Medicína 56, No. 10 (2011): 486–503. http://dx.doi.org/10.17221/3294-vetmed.

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&nbsp; The immune system of fish is very similar to vertebrates, although there are some important differences. Fish are free-living organisms from the embryonic stage of life in their aquatic environment. They have mechanisms to protect themselves from a wide variety of microorganisms. Consequently, fish rely on their innate immune system for an extended period of time, beginning at the early stages of embryogenesis. The components of the innate immune response are divided into physical, cellular and humoral factors and include humoral and cellular receptor molecules that are soluble in plasma and other body fluids. The lymphoid organs found in fish include the thymus, spleen and kidney. Immunoglobulins are the principal components of the immune response against pathogenic organisms. Immunomodulatory products, including nucleotides, glucans and probiotics, are increasingly used in aquaculture production. The use of these products reduces the need for therapeutic treatments, enhances the effects of vaccines and, in turn, improves the indicators of production. The aim of this review is to provide a review of the immune system in fish, including the ontogeny, mechanisms of unspecific and acquired immunity and the action of some immunomodulators.

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Ogunjimi, Benson, Evelien Smits, Steven Heynderickx, et al. "Influence of Frequent Infectious Exposures on General and Varicella-Zoster Virus-Specific Immune Responses in Pediatricians." Clinical and Vaccine Immunology 21, no. 3 (2014): 417–26. http://dx.doi.org/10.1128/cvi.00818-13.

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ABSTRACTReexposure to viruses is assumed to strengthen humoral and cellular immunity via the secondary immune response. We studied the effects of frequent exposure to viral infectious challenges on immunity. Furthermore, we assessed whether repetitive exposures to varicella-zoster virus (VZV) elicited persistently high immune responses. Blood samples from 11 pediatricians and matched controls were assessed at 3 time points and 1 time point, respectively. Besides the assessment of general immunity by means of measuring T-cell subset percentages, antibody titers and gamma interferon (IFN-γ)/interleukin 2 (IL-2)-producing T-cell percentages against adenovirus type 5 (AdV-5), cytomegalovirus (CMV), tetanus toxin (TT), and VZV were determined. Pediatricians had lower levels of circulating CD4+-naive T cells and showed boosting of CD8+effector memory T cells. Although no effect on humoral immunity was seen, repetitive exposures to VZV induced persistently higher percentages of IFN-γ-positive T cells against all VZV antigens tested (VZV glycoprotein E [gE], VZV intermediate-early protein 62 [IE62], and VZV IE63) than in controls. T cells directed against latency-associated VZV IE63 benefitted the most from natural exogenous boosting. Although no differences in cellular or humoral immunity were found between the pediatricians and controls for AdV-5 or TT, we did find larger immune responses against CMV antigens in pediatricians. Despite the high infectious burden, we detected a robust and diverse immune system in pediatricians. Repetitive exposures to VZV have been shown to induce a stable increased level of VZV-specific cellular but not humoral immunity. Based on our observations, VZV IE63 can be considered a candidate for a zoster vaccine.

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Shymanska, Ya V., V. K. Likhachov, L. M. Dobrovolska, and O. O. Taranovska. "PECULIARITIES OF CELLULAR AND HUMORAL IMMUNITY IN WOMEN WHO BECOME PREGNANT THROUGH IN VITRO FERTILIZATION AND AT RISK OF PRETERM DELIVERY." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 21, no. 1 (2021): 60–64. http://dx.doi.org/10.31718/2077-1096.21.1.60.

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Introduction. In the structure of the causes of preterm delivery among patients who become pregnant through in vitro fertilization (IVF), immunological problems rank one of the leading places. Purpose: To identify alterations in cellular and humoral immunity in women with a history of infertility, who became pregnant through IVF, and who was at risk of preterm delivery. Materials and methods. The test group consisted of 37 women who became pregnant through IVF and manifested prognostic signs indicating a high risk of preterm delivery in the future. These women refused taking preventive therapy to reduce the risks of preterm birth. The control group (healthy pregnant women) included 20 women with physiological course of pregnancy. The indicators characterizing cellular and humoral components of immunity were investigated. Results and discussion. The women of the test group demonstrated changes in both cellular (decreased blood level of total T-lymphocytes, decreased level of T-helpers, fall in the of B-lymphocyte content) and humoral (decreased IgA content, decreased immunoregulatory index and tendency toward the lowering of immunoglobulin index, decreased concentration of circulating immune complexes) immunity. Conclusions: The changes in cellular and humoral immunity in pregnant women who undergoing IVF and were at risk of preterm delivery but refused preventive therapy have been manifested by decreased blood level of total T-lymphocytes, decreased level of T-helpers, considerable lowering in B-lymphocytes, and fall in Ig and IgG content). The results point out the progression of immunosuppression characteristic of the pregnancy course in general.

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Bezrodnova, S. M., I. V. Demurcheva, and O. O. Kravchenko. "Study of postvaccinal immunity to whooping cough in children with connective tissue dyslasia syndrome." CHILDREN INFECTIONS 20, no. 1 (2021): 28–33. http://dx.doi.org/10.22627/2072-8107-2021-20-1-28-33.

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Purpose: to evaluate the effectiveness of post-vaccination immunity against whooping cough in children aged 1 to 17 years with connective tissue dysplasia syndrome in Stavropol.The principal component analysis was used to optimize the interpretation of immunograms in connective tissue dysplasia syndromedepending on protective titers for whooping cough. Analysis by the method of principal components showed that the links of cellular immunity predominate in children with connective tissue dysplasia syndrome with low titers of antibodies to whooping cough.The general variant of the response of the immune system was observed in children with protective titers of antibodies to whooping cough (1: 160—1: 320). In children with connective tissue dysplasia with titres of 1: 640or more antibodies to whooping cough, humoral and cellular immunities are closely interrelated, it is the presence of humoral and cellular immunity that makesit possible to develop a stable immunity to preventable infections.Thus, the method of main components effectively provides accessible information on key immune processes and is promising.

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Liu, Xiao-Fei, Jun Zhu, Shao-Yang Ge, et al. "Orally Administered Dendrobium Officinale and its Polysaccharides Enhance Immune Functions in BALB/c Mice." Natural Product Communications 6, no. 6 (2011): 1934578X1100600. http://dx.doi.org/10.1177/1934578x1100600627.

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The immunoactivity was evaluated of Dendrobium officinale Kimura & Migo, a Chinese herbal plant, and its crude polysaccharides. Different dosages of D. officinale and its polysaccharides were orally administered to healthy BALB/c mice. The control group was given distilled water. After 4 weeks, immune parameters, including cellular immunity (delayed-type hypersensitivity and natural killer cell activity), humoral immunity (serum hemolytic complement activity), nonspecific immunity (peritoneal macrophage phagocytosis) and interferon-γ production by splenocytes were measured. The results showed that D. officinale and its polysaccharides can significantly enhance cellular immunity and nonspecific immunity in mice. Humoral immunity was also enhanced after oral administration of D. officinale, but the polysaccharides had no influence. Both D. officinale and its polysaccharides markedly increased IFN-γ production by murine splenocytes. Six fractions were isolated from the polysaccharides; the molecular weight of the major fraction was 533,700 Da, and composed of mannose, glucose and rhamnose in a molar ratio of 7.3:1.3:1.0.

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