Relevant bibliographies by topics / Hypoxia-Inducible

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Hypoxia-Inducible'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Hypoxia-Inducible"

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Aldecoa Bedoya, Franklin. "Factor inducible por hipoxia en cáncer." Horizonte Médico (Lima) 23, no. 4 (2023): e2584. http://dx.doi.org/10.24265/horizmed.2023.v23n4.11.

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Liu, Ting, Le Zhao, Huilian Hou, Lu Ding, Wei Chen, and Xu Li. "Ginsenoside 20(S)-Rg3 suppresses ovarian cancer migration via hypoxia-inducible factor 1 alpha and nuclear factor-kappa B signals." Tumor Biology 39, no. 5 (2017): 101042831769222. http://dx.doi.org/10.1177/1010428317692225.

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Hypoxia-inducible factor 1 is believed to play a prominent role in the survival and developing progress of cancers. As a result, inhibiting α subunit of hypoxia-inducible factor 1 represents an attractive strategy against tumor. Although hypoxia-inducible factor 1α is a hypoxia-regulated subunit, increasing evidence indicates that hypoxia-inducible factor 1α could stable expression under normoxic conditions, regulated by non-hypoxia-mediated mechanisms. However, there are few strategies to target hypoxia-inducible factor 1α under normoxic conditions. Here, we report that ginsenoside 20(S)-Rg3,
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Sang, Xiguang, Zhiyong Wang, Tao Qin та Yonggang Li. "Elevated concentrations of hypoxia-inducible factor-1α in patients with fracture and concomitant traumatic brain injury". Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 54, № 5 (2016): 584–92. http://dx.doi.org/10.1177/0004563216673087.

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Background Compelling evidence indicate that traumatic brain injury is highly related to accelerated bone fracture repair, but the underlying mechanism still remains elusive. Fracture repair process relies greatly on the formation of new blood vessels in fracture site, and angiogenic factors have been confirmed to be essential for the initiation and maintenance of the fracture healing. Hypoxia-inducible factor-1α was demonstrated to be a critical regulator of angiogenic–osteogenic coupling during bone development and regeneration. The aim of the present study was to investigate the local and c
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kun, Zhang, Yang yuling, Wang dongchun, Xie bingbing, Li xiaoli та Xu bin. "HIF-1α Inhibition Sensitized Pituitary Adenoma Cells to Temozolomide by Regulating Presenilin 1 Expression and Autophagy". Technology in Cancer Research & Treatment 15, № 6 (2016): NP95—NP104. http://dx.doi.org/10.1177/1533034615618834.

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Pituitary adenomas usually develop temozolomide resistance, which could compromise the anticancer effects of temozolomide. Suppression of hypoxia-inducible factor 1α has been shown to sensitize glioblastoma cells to temozolomide treatment according to previous reports. However, whether and how the suppression of hypoxia-inducible factor 1α could sensitize pituitary adenomas to temozolomide treatment are still poorly understood. In the present study, using hypoxia-inducible factor 1α knockdown strategy, we demonstrated for the first time that hypoxia-inducible factor 1α knockdown could inhibit
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Walmsley, Sarah R., Edwin R. Chilvers, and Moira KB Whyte. "Hypoxia. Hypoxia, hypoxia inducible factor and myeloid cell function." Arthritis Research & Therapy 11, no. 2 (2009): 219. http://dx.doi.org/10.1186/ar2632.

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Pan, Fan, Joseph Barbi, and Drew M. Pardoll. "Hypoxia-inducible factor 1." OncoImmunology 1, no. 4 (2012): 510–15. http://dx.doi.org/10.4161/onci.19457.

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Semenza, Gregg L., Faton Agani, Narayan Iyer, et al. "Hypoxia-Inducible Factor 1." Chest 114, no. 1 (1998): 40S—45S. http://dx.doi.org/10.1378/chest.114.1_supplement.40s.

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Tsuchiya, T. "Hypoxia-inducible transcriptional cofactor." Neuroscience Research 38 (2000): S40. http://dx.doi.org/10.1016/s0168-0102(00)81086-x.

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Fandrey, Joachim. "Hypoxia-inducible gene expression." Respiration Physiology 101, no. 1 (1995): 1–10. http://dx.doi.org/10.1016/0034-5687(95)00013-4.

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Lee, Kyoung Eun, and M. Celeste Simon. "SnapShot: Hypoxia-Inducible Factors." Cell 163, no. 5 (2015): 1288–1288. http://dx.doi.org/10.1016/j.cell.2015.11.011.

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Dissertations / Theses on the topic "Hypoxia-Inducible"

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Lau, Yue-huen Thomas. "Nuclear transcription factors and hypoxia-inducible genes in chronic liver hypoxia." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31939302.

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Lau, Yue-huen Thomas, and 劉汝這. "Nuclear transcription factors and hypoxia-inducible genes in chronic liver hypoxia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B31939302.

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Laitala, A. (Anu). "Hypoxia-inducible factor prolyl 4-hydroxylases regulating erythropoiesis, and hypoxia-inducible lysyl oxidase regulating skeletal muscle development during embryogenesis." Doctoral thesis, Oulun yliopisto, 2014. http://urn.fi/urn:isbn:9789526206943.

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Abstract Erythropoiesis is the process of red blood cell production. The main regulator is the erythropoietin (EPO) hormone, which is strongly upregulated in low oxygen concentration (hypoxia) in cells via the hypoxia-inducible transcription factor HIF. The stability of HIF is regulated in an oxygen-dependent manner by three HIF prolyl 4-hydroxylases, all of which are known to participate in the regulation of erythropoiesis. A role in erythropoiesis of a fourth prolyl 4-hydroxylase, P4H-TM, which possesses a transmembrane domain, is not known, but it is able to hydroxylate HIF at least in vitr
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Lidgren, Anders. "Hypoxia inducible factor-1α in renal cell carcinoma". Doctoral thesis, Umeå universitet, Kirurgisk och perioperativ vetenskap, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1462.

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Hypoxia Inducible Factor-1α in Renal Cell Carcinoma Departments of Surgical and Perioperative Sciences, Urology and Andrology; Radiation Sciences, Oncology; Medical Biosciences, Pathology; and Medical Biosciences, Clinical Chemistry, Umeå University, Umeå, Sweden Background: Renal cell carcinoma (RCC) accounts for approximately 2-3% of all human cancers. A distinguished feature of RCC is vascularisation and among the three dominating RCC types conventional RCC (cRCC) generally is more vascularised than papillary RCC (pRCC) and chromophobe RCC (chRCC). Angiogenesis is a critical step in tumour
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Wilson, William J. "Hypoxia inducible factor 1a : molecular mechanisms of regulation /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-223-X.

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Miles, Anna Louise. "V-ATPase regulation of Hypoxia Inducible transcription Factors." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/283217.

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Metazoans have evolved conserved mechanisms to promote cell survival under low oxygen tensions by initiating a transcriptional cascade centered on the action of Hypoxia Inducible transcription Factors (HIFs). In aerobic conditions, HIFs are inactivated by ubiquitin-proteasome-mediated degradation of their a subunit, which is dependent on prolyl hydroxylation by 2-oxoglutarate (2-OG) and Fe(II)-dependent prolyl hydroxylases (PHDs). In hypoxia, HIF-$\alpha$ is no longer hydroxylated and is therefore stabilised, activating a global transcriptional response to ensure cell survival. Interestingly,
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Chan, Mun Chiang. "Inhibition studies on hypoxia-inducible factor (HIF) hydroxylases." Thesis, University of Oxford, 2014. https://ora.ox.ac.uk/objects/uuid:5c8a24bd-3acc-4271-a24b-8bd601d7a807.

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The hypoxia-inducible factor (HIF) is a key regulator of transcriptional responses to hypoxia in animals. As part of the cellular response to decreased oxygen concentrations, the transcriptional activity of a heterodimeric complex consisting of HIFα and HIFβ activates the expression of hundreds of target genes, including those involved in cellular growth, apoptosis, energy metabolism and angiogenesis. HIFα prolyl hydroxylation, as catalysed by the HIF prolyl hydroxylases (PHDs), leads to subsequent HIFα polyubiquitination and proteasomal degradation. HIFα asparaginyl hy
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Lau, Chi-keung, та 劉智強. "Blockade of hypoxia inducible factor-1α sensitizes hepatocellular carcinoma to hypoxia and chemotherapy". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B39634243.

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Lau, Chi-keung. "Blockade of hypoxia inducible factor-1[alpha] sensitizes hepatocellular carcinoma to hypoxia and chemotherapy." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B39634243.

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Hägg, Maria. "Hypoxia-inducible factors (HIFs) and biological responses in hypoxia, inflammation and embryonic vascular development /." Göteborg: Department of Medical Biochemistry and Cell Biology, Institute of Biomedicine, the Sahlgrenska Academy at University of Gothenburg, 2008. http://hdl.handle.net/2077/9991.

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Books on the topic "Hypoxia-Inducible"

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Connor, Thomas, and Patrick H. Maxwell. Hypoxia-inducible factor and renal disorders. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0331.

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Hypoxia-inducible factors (HIFs) are transcription factors that control the cellular response to changes in oxygen levels. This response is common to all cells in the body and is highly conserved in evolution. The kidney exhibits steep gradients in oxygenation which are important in the homeostatic response to anaemia. The cellular response to low levels of oxygen (hypoxia) also plays a role in such diverse processes as acute kidney injury, the progression of chronic kidney disease, and kidney cancer. There is now considerable interest in using drugs to manipulate the HIF response to treat the
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Hypoxia-Inducible Factors: Regulation and Therapeutic Potential. MDPI, 2022. http://dx.doi.org/10.3390/books978-3-0365-2913-4.

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Raju, Raghavan, and Irshad H. Chaudry. The host response to hypoxia in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0305.

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The hypoxic response of the host is complex. While the oxygen-sensing intracellular machinery attempts to restore cellular homeostasis by augmenting respiration and blood flow, events such as severe haemorrhage lead to whole body hypoxia and decreased mitochondrial function. Immunological perturbations following severe haemorrhage may result in multiple organ dysfunction and sepsis, while impaired perfusion may lead to microvascular injury and local hypoxia. Trauma-haemorrhage or hypoxic exposure in animals causes a systemic inflammatory response, decreased antigen presentation by peritoneal m
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McLaren, Anya T. Increased expression of hypoxia inducible factor-1alpha (HIF-1alpha) and neuronal nitric oxide synthase (nNOS) in the cerebral cortex of anemic rats. 2006.

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Kanno, Hiroshi, and Joachim P. Steinbach. Familial tumour syndromes: von Hippel–Lindau disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0016.

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Von Hippel–Lindau (VHL) disease, an autosomal dominant familial tumour syndrome, is often associated with haemangioblastoma of the central nervous system. In the presence of oxygen, VHL protein serves to prevent the accumulation of hypoxia-inducible factor (HIF) protein by targeting it to the proteasomal pathway, while biallelic inactivation of the VHL gene blocks degradation of HIF and leads to constitutive activation of the HIF pathway although oxygen is present. HIF-target genes are involved in angiogenesis, proliferation, and metabolism enabling tumour growth. Haemangioblastoma is a highly
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Macdougall, Iain C. Erythropoiesis-stimulating agents in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0124.

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The advent of recombinant human erythropoietin (epoetin) in the late 1980s transformed the management of renal anaemia, liberating many dialysis patients from lifelong regular blood transfusions, in turn causing severe iron overload and human leucocyte antigen sensitization. Epoetin can be administered either intravenously or subcutaneously, but the half-life of the drug is fairly short at around 6–8 hours, necessitating frequent injections. To circumvent this problem, two manipulations to the erythropoietin molecule were engineered. The first of these was to attach an extra two carbohydrate c
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Book chapters on the topic "Hypoxia-Inducible"

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Sainburg, Robert L., Andrew L. Clark, George E. Billman, et al. "Hypoxia-Inducible Factors." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2524.

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Zhang, Tong, Zhaoji Liu, Zhaoyong Li, and Huafeng Zhang. "Hypoxia-Inducible Factor-1." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101697.

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Semenza, Gregg L. "Hypoxia-Inducible Factor 1." In Brain Hypoxia and Ischemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-579-8_14.

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Shim, Hyunsuk. "Hypoxia-Inducible Factor-1." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_2928-2.

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Zhang, Tong, Zhaoji Liu, Zhaoyong Li, and Huafeng Zhang. "Hypoxia-Inducible Factor-1." In Encyclopedia of Signaling Molecules. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101697-1.

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Moses, Marsha A. "Hypoxia-inducible Factor-1." In Encyclopedia of Systems Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_1547.

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Shim, Hyunsuk. "Hypoxia-Inducible Factor-1." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_2928.

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Shim, Hyunsuk. "Hypoxia-Inducible Factor 1." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_2928.

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Watt, Suzanne M., Grigorios Tsaknakis, Sinead P. Forde, and Lee Carpenter. "Stem Cells, Hypoxia and Hypoxia-Inducible Factors." In Regulatory Networks in Stem Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-227-8_18.

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Sharp, Frank R., Marcelle Bergeron, and Myriam Bernaudin. "Hypoxia-inducible factor in brain." In Advances in Experimental Medicine and Biology. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4757-3401-0_18.

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Conference papers on the topic "Hypoxia-Inducible"

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Wang, Xintong. "Research and application of hypoxia-inducible factor." In 4TH INTERNATIONAL CONFERENCE ON FRONTIERS OF BIOLOGICAL SCIENCES AND ENGINEERING (FBSE 2021). AIP Publishing, 2022. http://dx.doi.org/10.1063/5.0095073.

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Saygıdeğer, Yasemin, Demet Kahraman, Sedat İlhan, et al. "Generation of small molecules targeting hypoxia inducible factors." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2850.

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Lin, Qun, Xiangyu Cong та Zhong Yun. "Abstract 2053: Differential regulation of hypoxia-inducible factors 1α and 2α by hypoxia". У Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2053.

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Yasuo, Masanori, Shiro Mizuno, Donatas Kraskauskas, et al. "Hypoxia Inducible Factor-1± In Human Emphysema Lung Tissue." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2919.

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Vadivel, Arul, Rajesh Alphonse, Nicholas Etches, Tim van Haaften, Farah Eaton, and Bernard Thebaud. "Hypoxia Inducible Factor (HIF) Promotes Alveolar Growth And Repair." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3936.

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Li, J., S. Bevans-Fonti, LA Shimoda, GL Semenza, and VY Polotsky. "Hypoxia Up-Regulates Genes of Lipid Biosynthesis Via Hypoxia Inducible Factor 1 (HIF-1)." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5333.

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Cassavaugh, Jessica, Sarah Hale, Cheung Wong та Karen M. Lounsbury. "Abstract 3092: FBW7 targets hypoxia inducible factor-1α (HIF-1α) for proteasomal degradation during hypoxia". У Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3092.

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Farha, Samar, Kewal Asosingh, Weiling Xu, et al. "Hypoxia Inducible Factors And Myeloid Abnormalities In Pulmonary Arterial Hypertension." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1041.

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Barnes, E. A., X. Che, R. Ito, S. Reddy, and D. N. Cornfield. "Overexpression of Hypoxia Inducible Factors in Pulmonary Artery Smooth Muscle Cells Mitigates Hypoxia-Induced Pulmonary Hypertension." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5288.

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To, Masako, Catherine E. Charron, Kosuke Haruki, Peter J. Barnes, and Kazuhiro Ito. "Molecular Mechanism Of Poor Hypoxia Inducible Factor 1± Response To Hypoxia In Chronic Obstructive Pulmonary Disease." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4560.

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Reports on the topic "Hypoxia-Inducible"

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Bedi, Atul. Inhibition of Breast Cancer by Repression of Angiogenic Hypoxia-Inducible Transcription Factors. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada495898.

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Seagroves, Tiffany N. The Contribution of Hypoxia-Inducible Factor (HIF) -1 Alpha to Normal Mammary Gland Development and Mammary Tumorigenesis. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada422423.

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Dougherty, Shona. Isolation and Functional Characterization of Prostate Tumor-Specific Hypoxia-Inducible Promoter/Enhancer Elements for Use in Gene Therapy. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada413596.

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Kazi, Armina A. Investigating the Regulation and Potential Role of Nonhypoxic Hypoxia-Inducible Factor 1 (HIF-1) in Aromatase Inhibitor Resistant Breast Cancer. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada568100.

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Kazi, Armina A. Investigation the Regulation and Potential Role of Nonhypoic Hypoxia-Inducible Factor 1 (HIF-1) in Aromatase Inhibitor-Resistant Breast Cancer. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada625486.

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Fukuta, Hidekatsu, Hiromi Hagiwara, and Takeshi Kamiya. Hypoxia-inducible factor prolyl hydroxylase inhibitors for anemia in heart failure patients: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.3.0103.

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Fukuta, Hidekatsu, Toshihiko Goto, and Takeshi Kamiya. Effects of hypoxia-inducible factor prolyl hydroxylase inhibitors in anemic heart failure patients: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.10.0062.

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