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Journal articles on the topic 'Influenza B virus'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

McCullers, Jonathan A., Sergio Facchini, P. Joan Chesney, and Robert G. Webster. "Influenza B Virus Encephalitis." Clinical Infectious Diseases 28, no. 4 (1999): 898–900. http://dx.doi.org/10.1086/515214.

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2

FRANK, ARTHUR L., LARRY H. TABER, and CHERYL M. PORTER. "INFLUENZA B VIRUS REINFECTION." American Journal of Epidemiology 125, no. 4 (1987): 576–86. http://dx.doi.org/10.1093/oxfordjournals.aje.a114571.

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3

Osterhaus, A. D. "Influenza B Virus in Seals." Science 288, no. 5468 (2000): 1051–53. http://dx.doi.org/10.1126/science.288.5468.1051.

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4

Mäkelä, Sanna M., Pamela Österlund, Veera Westenius, et al. "RIG-I Signaling Is Essential for Influenza B Virus-Induced Rapid Interferon Gene Expression." Journal of Virology 89, no. 23 (2015): 12014–25. http://dx.doi.org/10.1128/jvi.01576-15.

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ABSTRACTInfluenza B virus causes annual epidemics and, along with influenza A virus, accounts for substantial disease and economic burden throughout the world. Influenza B virus infects only humans and some marine mammals and is not responsible for pandemics, possibly due to a very low frequency of reassortment and a lower evolutionary rate than that of influenza A virus. Influenza B virus has been less studied than influenza A virus, and thus, a comparison of influenza A and B virus infection mechanisms may provide new insight into virus-host interactions. Here we analyzed the early events in

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5

Voeten, J. T. M., J. Groen, D. van Alphen, et al. "Use of Recombinant Nucleoproteins in Enzyme-Linked Immunosorbent Assays for Detection of Virus-Specific Immunoglobulin A (IgA) and IgG Antibodies in Influenza Virus A- or B-Infected Patients." Journal of Clinical Microbiology 36, no. 12 (1998): 3527–31. http://dx.doi.org/10.1128/jcm.36.12.3527-3531.1998.

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The nucleoprotein genes of influenza virus A/Netherlands/018/94 (H3N2) and influenza virus B/Harbin/7/94 were cloned into the bacterial expression vector pMalC to yield highly purified recombinant influenza virus A and B nucleoproteins. With these recombinant influenza nucleoproteins, enzyme-linked immunosorbent assays (ELISAs) were developed for the detection of influenza virus A- and B-specific immunoglobulin A (IgA) and IgG serum antibodies. Serum samples were collected at consecutive time points after the onset of clinical symptoms from patients with confirmed influenza virus A or B infect

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6

Flandorfer, Astrid, Adolfo García-Sastre, Christopher F. Basler, and Peter Palese. "Chimeric Influenza A Viruses with a Functional Influenza B Virus Neuraminidase or Hemagglutinin." Journal of Virology 77, no. 17 (2003): 9116–23. http://dx.doi.org/10.1128/jvi.77.17.9116-9123.2003.

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ABSTRACT Reassortment of influenza A and B viruses has never been observed in vivo or in vitro. Using reverse genetics techniques, we generated recombinant influenza A/WSN/33 (WSN) viruses carrying the neuraminidase (NA) of influenza B virus. Chimeric viruses expressing the full-length influenza B/Yamagata/16/88 virus NA grew to titers similar to that of wild-type influenza WSN virus. Recombinant viruses in which the cytoplasmic tail or the cytoplasmic tail and the transmembrane domain of the type B NA were replaced with those of the type A NA were impaired in tissue culture. This finding corr

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7

Nakajima, S., F. Nishikawa, K. Nakamura, H. Nakao, and K. Nakajima. "Reinfection with influenza B virus in children: analysis of the reinfection influenza B viruses." Epidemiology and Infection 113, no. 1 (1994): 103–12. http://dx.doi.org/10.1017/s0950268800051517.

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SUMMARYInfluenza B virus reinfection in Japanese children was studied epidemi-ologically during 1979–91 and virologically during 1985–91. During this investigation, there were four epidemics caused by influenza B viruses, each of which accompanied antigenic drift. Between the epidemics in 1987/88 and 1989/90, the viruses changed drastically, both genetically and antigenically. The minimum rate of reinfection with influenza B virus during the whole period was 3–25% depending on the influenza seasons. The antigens of primary and reinfection strains of influenza B virus isolated from 18 children

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8

Ñamendys-Silva, Silvio A., María O. González-Herrera, Julia Texcocano-Becerra, and Angel Herrera-Gómez. "Acute Respiratory Distress Syndrome Caused by Influenza B Virus Infection in a Patient with Diffuse Large B-Cell Lymphoma." Case Reports in Medicine 2011 (2011): 1–4. http://dx.doi.org/10.1155/2011/647528.

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Influenza B virus infections are less common than infections caused by influenza A virus in critically ill patients, but similar mortality rates have been observed for both influenza types. Pneumonia caused by influenza B virus is uncommon and has been reported in pediatric patients and previously healthy adults. Critically ill patients with pneumonia caused by influenza virus may develop acute respiratory distress syndrome. We describe the clinical course of a critically ill patient with diffuse large B-cell lymphoma nongerminal center B-cell phenotype who developed acute respiratory distress

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9

Mao, Steve. "Stressed out by influenza virus." Science 362, no. 6412 (2018): 301.2–301. http://dx.doi.org/10.1126/science.362.6412.301-b.

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10

Wahyutomo, Ridha, V. Rizke Ciptaningtyas, and Purnomo Hadi. "Prevalence of Influenza Viruses (Influenza Like Illness) In Regional Laboratory Avian Influenza Semarang." Sains Medika : Jurnal Kedokteran dan Kesehatan 3, no. 2 (2011): 157. http://dx.doi.org/10.30659/sainsmed.v3i2.398.

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Background: Influenza is the major health threat worldwide causing illness and death every year. However, data on the epidemiology of influenza in tropical countries, including Indonesia, are still limited. Up dated data for its prevalence is needed to monitor its spreading and to evaluate its outbreak. Therefore a working regional laboratory in surveillance of ILI (Influenza Like Illness) was formed. This research was conducted to provide updated data on prevalence of ILI in regional laboratorium avian influenza Semarang.Design and Method: Data from patients examined in the regional laborator

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11

Ping, Jihui, Tiago J. S. Lopes, Gabriele Neumann, and Yoshihiro Kawaoka. "Development of high-yield influenza B virus vaccine viruses." Proceedings of the National Academy of Sciences 113, no. 51 (2016): E8296—E8305. http://dx.doi.org/10.1073/pnas.1616530113.

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The burden of human infections with influenza A and B viruses is substantial, and the impact of influenza B virus infections can exceed that of influenza A virus infections in some seasons. Over the past few decades, viruses of two influenza B virus lineages (Victoria and Yamagata) have circulated in humans, and both lineages are now represented in influenza vaccines, as recommended by the World Health Organization. Influenza B virus vaccines for humans have been available for more than half a century, yet no systematic efforts have been undertaken to develop high-yield candidates. Therefore,

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12

Nolte, Frederick S., Lori Gauld, and Susan B. Barrett. "Direct Comparison of Alere i and cobas Liat Influenza A and B Tests for Rapid Detection of Influenza Virus Infection." Journal of Clinical Microbiology 54, no. 11 (2016): 2763–66. http://dx.doi.org/10.1128/jcm.01586-16.

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We compared two rapid, point-of care nucleic acid amplification tests for detection of influenza A and B viruses (Alere i [Alere] and cobas Liat [Roche Diagnostics]) with the influenza A and B virus test components of the FilmArray respiratory panel (BioFire Diagnostics) using 129 respiratory specimens collected in universal viral transport medium (80 influenza A virus and 16 influenza B virus positive) from both adult and pediatric patients. The sensitivities of the Alere test were 71.3% for influenza A virus and 93.3% for influenza B virus, with specificities of 100% for both viruses. The se

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Yasui, Hisako, Junko Kiyoshima, Tetuji Hori, and Kan Shida. "Protection against Influenza Virus Infection of Mice Fed Bifidobacterium breve YIT4064." Clinical Diagnostic Laboratory Immunology 6, no. 2 (1999): 186–92. http://dx.doi.org/10.1128/cdli.6.2.186-192.1999.

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ABSTRACT Mice fed Bifidobacterium breve YIT4064 and immunized orally with influenza virus were more strongly protected against influenza virus infection of the lower respiratory tract than ones immunized with influenza virus only. The number of mice with enhanced anti-influenza virus immunoglobulin G (IgG) in serum upon oral administration of B. breve YIT4064 and oral immunization with influenza virus was significantly greater than that upon oral immunization with influenza virus only. These findings demonstrated that the oral administration of B. breve YIT4064 increased anti-influenza virus I

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14

Rott, O., J. Charreire, K. Mignon-Godefroy, and E. Cash. "B cell superstimulatory influenza virus activates peritoneal B cells." Journal of Immunology 155, no. 1 (1995): 134–42. http://dx.doi.org/10.4049/jimmunol.155.1.134.

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Abstract We evaluated the potential of B cell "superstimulatory" influenza viruses to activate peritoneal B cells (PBC) from BALB/c mice containing both CD5+ and CD5- "sister" cells. Like conventional B cells, PBCs responded to influenza viruses in a hemagglutinin glycoprotein (HA) subtype-specific manner with proliferation and vigorous Ig synthesis. However, a number of HA subtypes that are highly stimulatory for conventional B cells failed to induce significant responses of PBC. Isotype-determination revealed a high predominance of IgM and only very low production of IgA and IgG. HA-activate

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15

Ghebremedhin, B., I. Engelmann, W. König, and B. König. "Comparison of the performance of the rapid antigen detection actim Influenza A&B test and RT-PCR in different respiratory specimens." Journal of Medical Microbiology 58, no. 3 (2009): 365–70. http://dx.doi.org/10.1099/jmm.0.004358-0.

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Nowadays, influenza antigen detection test kits are used most frequently to detect influenza A or B virus to establish the diagnosis of influenza rapidly and initiate appropriate therapy. This study was conducted to evaluate the performance of the actim Influenza A&B test (Medix Biochemica). Overall, 473 respiratory specimens were analysed in the actim Influenza A&B test and the results were compared with those from an RT-PCR assay; 461 of these samples originated from paediatric patients aged 7 weeks to 6.5 years either with influenza-related symptoms or from the intensive care unit,

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16

Wu, Yuet, Wenwei Tu, Kwok-Tai Lam, et al. "Lethal Coinfection of Influenza Virus and Streptococcus pneumoniae Lowers Antibody Response to Influenza Virus in Lung and Reduces Numbers of Germinal Center B Cells, T Follicular Helper Cells, and Plasma Cells in Mediastinal Lymph Node." Journal of Virology 89, no. 4 (2014): 2013–23. http://dx.doi.org/10.1128/jvi.02455-14.

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ABSTRACTSecondaryStreptococcus pneumoniaeinfection after influenza is a significant clinical complication resulting in morbidity and sometimes mortality. Prior influenza virus infection has been demonstrated to impair the macrophage and neutrophil response to the subsequent pneumococcal infection. In contrast, how a secondary pneumococcal infection after influenza can affect the adaptive immune response to the initial influenza virus infection is less well understood. Therefore, this study focuses on how secondary pneumococcal infection after influenza may impact the humoral immune response to

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17

Jiang, Jingwen, Jing Li, Wenhui Fan, et al. "Robust Lys63-Linked Ubiquitination of RIG-I Promotes Cytokine Eruption in Early Influenza B Virus Infection." Journal of Virology 90, no. 14 (2016): 6263–75. http://dx.doi.org/10.1128/jvi.00549-16.

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ABSTRACTInfluenza A and B virus infections both cause a host innate immunity response. Here, we report that the robust production of type I and III interferons (IFNs), IFN-stimulated genes, and proinflammatory factors can be induced by influenza B virus rather than influenza A virus infection in alveolar epithelial (A549) cells during early infection. This response is mainly dependent on the retinoic acid-inducible gene I (RIG-I)-mediated signaling pathway. Infection by influenza B virus promotes intense Lys63-linked ubiquitination of RIG-I, resulting in cytokine eruption. It is known that the

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18

Wanitchang, Asawin, Jaraspim Narkpuk, Peera Jaru-ampornpan, Juggagarn Jengarn, and Anan Jongkaewwattana. "Inhibition of influenza A virus replication by influenza B virus nucleoprotein: An insight into interference between influenza A and B viruses." Virology 432, no. 1 (2012): 194–203. http://dx.doi.org/10.1016/j.virol.2012.06.016.

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19

To, Janet, and Jaume Torres. "Viroporins in the Influenza Virus." Cells 8, no. 7 (2019): 654. http://dx.doi.org/10.3390/cells8070654.

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Influenza is a highly contagious virus that causes seasonal epidemics and unpredictable pandemics. Four influenza virus types have been identified to date: A, B, C, and D, where only A–C are known to infect humans. Influenza A (IAV) and B (IBV) viruses are responsible for seasonal influenza epidemics in humans and are responsible for up to a billion flu infections annually. The M2 protein is present in all influenza types and belongs to the class of viroporins (i.e., small proteins that form ion channels that increase membrane permeability in virus-infected cells). In influenza A and B, AM2 an

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20

Kang, Hae-Ji, Ki-Back Chu, Keon-Woong Yoon, Gi-Deok Eom, Jie Mao, and Fu-Shi Quan. "Cross-Protection Induced by Virus-like Particles Derived from the Influenza B Virus." Biomedicines 10, no. 7 (2022): 1618. http://dx.doi.org/10.3390/biomedicines10071618.

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The mismatch between the circulating influenza B virus (IBV) and the vaccine strain contributes to the rapid emergence of IBV infection cases throughout the globe, which necessitates the development of effective vaccines conferring broad protection. Here, we generated influenza B virus-like particle (VLP) vaccines expressing hemagglutinin, neuraminidase, or both antigens derived from the influenza B virus (B/Washington/02/2019 (B/Victoria lineage)-like virus, B/Phuket/3073/2013 (B/Yamagata lineage)-like virus. We found that irrespective of the derived antigen lineage, immunizing mice with the

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21

Anna Rozaliyani, Asiyah Taqiyya Fakhrur Razi, Ammar Abdurrahman Hasyim, et al. "<b>Perkembangan Terkini Penyakit Influenza dan Peran Favipiravir dalam Tata Laksana Penyakit</b>." MEDICINUS 38, no. 6 (2025): 8–19. https://doi.org/10.56951/3trygg29.

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Influenza virus infection remains a major global health challenge, particularly for vulnerable groups with compromised immune systems. Influenza is primarily caused by infection of influenza A and B viruses, with a varying clinical spectrum, ranging from asymptomatic cases to life-threatening conditions. Influenza viruses tend to undergo rapid genetic mutations, limiting the long-term effectiveness of vaccine-induced immune responses. Diagnosing influenza remains challenging due to the nonspecific signs and symptoms of the disease. The gold standard for influenza diagnosis is reverse transcrip

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22

Petro-Turnquist, Erika, Brigette Corder Kampfe, Amber Gadeken, Matthew J. Pekarek, and Eric A. Weaver. "Multivalent Epigraph Hemagglutinin Vaccine Protects against Influenza B Virus in Mice." Pathogens 13, no. 2 (2024): 97. http://dx.doi.org/10.3390/pathogens13020097.

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Influenza B virus is a respiratory pathogen that contributes to seasonal epidemics, accounts for approximately 25% of global influenza infections, and can induce severe disease in young children. While vaccination is the most commonly used method of preventing influenza infections, current vaccines only induce strain-specific responses and have suboptimal efficacy when mismatched from circulating strains. Further, two influenza B virus lineages have been described, B/Yamagata-like and B/Victoria-like, and the limited cross-reactivity between the two lineages provides an additional barrier in d

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23

Gao, Qinshan, Edward W. A. Brydon, and Peter Palese. "A Seven-Segmented Influenza A Virus Expressing the Influenza C Virus Glycoprotein HEF." Journal of Virology 82, no. 13 (2008): 6419–26. http://dx.doi.org/10.1128/jvi.00514-08.

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ABSTRACT Influenza viruses are classified into three types: A, B, and C. The genomes of A- and B-type influenza viruses consist of eight RNA segments, whereas influenza C viruses only have seven RNAs. Both A and B influenza viruses contain two major surface glycoproteins: the hemagglutinin (HA) and the neuraminidase (NA). Influenza C viruses have only one major surface glycoprotein, HEF (hemagglutinin-esterase fusion). By using reverse genetics, we generated two seven-segmented chimeric influenza viruses. Each possesses six RNA segments from influenza virus A/Puerto Rico/8/34 (PB2, PB1, PA, NP

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Bodewes, Rogier, Danny Morick, Gerrie de Mutsert, et al. "Recurring Influenza B Virus Infections in Seals." Emerging Infectious Diseases 19, no. 3 (2013): 511–12. http://dx.doi.org/10.3201/eid1903.120965.

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25

Nogales, Aitor, Irene Rodríguez-Sánchez, Kristen Monte, Deborah J. Lenschow, Daniel R. Perez, and Luis Martínez-Sobrido. "Replication-competent fluorescent-expressing influenza B virus." Virus Research 213 (February 2016): 69–81. http://dx.doi.org/10.1016/j.virusres.2015.11.014.

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26

&NA;. "Sialidase inhibitors: influenza B virus resistance emerges." Inpharma Weekly &NA;, no. 1582-1583 (2007): 14. http://dx.doi.org/10.2165/00128413-200715820-00038.

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27

Kugelberg, Elisabeth. "Promoting B cell responses to influenza virus." Nature Reviews Immunology 14, no. 5 (2014): 283. http://dx.doi.org/10.1038/nri3662.

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28

Stepanova, Ekaterina, Elena Krutikova, Pei-Fong Wong, et al. "Safety, Immunogenicity, and Protective Efficacy of a Chimeric A/B Live Attenuated Influenza Vaccine in a Mouse Model." Microorganisms 9, no. 2 (2021): 259. http://dx.doi.org/10.3390/microorganisms9020259.

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Influenza A and B viruses cause significant morbidity and mortality worldwide. Current influenza vaccines are composed of three or four strains: A/H1N1, A/H3N2, and B (Victoria and Yamagata lineages). It is of great interest if immunization against both type A and B influenza viruses can be combined in a single vaccine strain, thus reducing the cost of vaccine production and the possibility of strain interference within the multicomponent vaccine. In the current study, we developed an experimental live cold-adapted influenza intertype reassortant (influenza A and B) vaccine on the live attenua

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Sam, I.-Ching, Yvonne C. F. Su, Yoke Fun Chan, et al. "Evolution of Influenza B Virus in Kuala Lumpur, Malaysia, between 1995 and 2008." Journal of Virology 89, no. 18 (2015): 9689–92. http://dx.doi.org/10.1128/jvi.00708-15.

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Influenza B virus causes significant disease but remains understudied in tropical regions. We sequenced 72 influenza B viruses collected in Kuala Lumpur, Malaysia, from 1995 to 2008. The predominant circulating lineage (Victoria or Yamagata) changed every 1 to 3 years, and these shifts were associated with increased incidence of influenza B. We also found poor lineage matches with recommended influenza virus vaccine strains. While most influenza B virus lineages in Malaysia were short-lived, one circulated for 3 to 4 years.

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Tan, Jessica, Guha Asthagiri Arunkumar, and Florian Krammer. "Universal influenza virus vaccines and therapeutics: where do we stand with influenza B virus?" Current Opinion in Immunology 53 (August 2018): 45–50. http://dx.doi.org/10.1016/j.coi.2018.04.002.

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31

Rotteveel, F. T. M., E. Braakman, B. Robbe, and C. J. Lucas. "Recognition of influenza virus-infected B-cell lines by human influenza virus-specific CTL." Cellular Immunology 111, no. 2 (1988): 473–81. http://dx.doi.org/10.1016/0008-8749(88)90110-4.

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32

Laursen, Nick S., Robert H. E. Friesen, Xueyong Zhu, et al. "Universal protection against influenza infection by a multidomain antibody to influenza hemagglutinin." Science 362, no. 6414 (2018): 598–602. http://dx.doi.org/10.1126/science.aaq0620.

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Broadly neutralizing antibodies against highly variable pathogens have stimulated the design of vaccines and therapeutics. We report the use of diverse camelid single-domain antibodies to influenza virus hemagglutinin to generate multidomain antibodies with impressive breadth and potency. Multidomain antibody MD3606 protects mice against influenza A and B infection when administered intravenously or expressed locally from a recombinant adeno-associated virus vector. Crystal and single-particle electron microscopy structures of these antibodies with hemagglutinins from influenza A and B viruses

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Wang, Qinghua, Feng Cheng, Mingyang Lu, Xia Tian, and Jianpeng Ma. "Crystal Structure of Unliganded Influenza B Virus Hemagglutinin." Journal of Virology 82, no. 6 (2008): 3011–20. http://dx.doi.org/10.1128/jvi.02477-07.

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ABSTRACT Here we report the crystal structure of hemagglutinin (HA) from influenza B/Hong Kong/8/73 (B/HK) virus determined to 2.8 Å. At a sequence identity of ∼25% to influenza A virus HAs, B/HK HA shares a similar overall structure and domain organization. More than two dozen amino acid substitutions on influenza B virus HAs have been identified to cause antigenicity alteration in site-specific mutants, monoclonal antibody escape mutants, or field isolates. Mapping these substitutions on the structure of B/HK HA reveals four major epitopes, the 120 loop, the 150 loop, the 160 loop, and the

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Bianchi, Elisabetta, Xiaoping Liang, Paolo Ingallinella, et al. "Universal Influenza B Vaccine Based on the Maturational Cleavage Site of the Hemagglutinin Precursor." Journal of Virology 79, no. 12 (2005): 7380–88. http://dx.doi.org/10.1128/jvi.79.12.7380-7388.2005.

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ABSTRACT Conventional influenza vaccines can prevent infection, but their efficacy depends on the degree of antigenic “match” between the strains used for vaccine preparation and those circulating in the population. A universal influenza vaccine based on invariant regions of the virus, able to provide broadly cross-reactive protection, without requiring continuous manufacturing update, would solve a major medical need. Since the temporal and geographical dominance of the influenza virus type and/or subtype (A/H3, A/H1, or B) cannot yet be predicted, a universal vaccine, like the vaccines curre

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Glebova, T. I., N. G. Klivleyeva, A. M. Baimukhametova, et al. "Circulation of influenza viruses in the epidemic season of 2018–2019 among people residing in Northern and Western Kazakhstan." Infekcionnye bolezni 19, no. 2 (2021): 70–75. http://dx.doi.org/10.20953/1729-9225-2021-2-70-75.

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Objective. Detection of influenza viruses among the population on the territory of the Northern and Western Kazakhstan during the 2018–2019 epidemic season. Patients and methods. The study involved 835 patients with ARVI symptoms. Biological samples were screened in real-time polymerase chain reaction (RT-PCR), hemagglutination inhibition (HAI) assay, and enzyme-linked immunosorbent assay (ELISA). Hemagglutinating agents were isolated in 9-10-day-old developing chicken embryos. Identification of isolates was carried out in RT-PCR and HAI assay. Results. 936 clinical samples (835 nasopharyngeal

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Dharmayanti, N. L. P. I., R. Damayanti, R. Indriani, A. Wiyono, and R. M. A. Adjid. "Molecular characterization of Indonesia avian influenza virus." Jurnal Ilmu Ternak dan Veteriner 10, no. 2 (2012): 127–33. https://doi.org/10.14334/jitv.v10i2.465.

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Avian influenza outbreaks in poultry have been reported in Java island since August 2003. A total of 14 isolates of avian influenza virus has been isolated from October 2003 to October 2004. The viruses have been identified as HPAI H5N1 subtype. All of them were characterized further at genetic level and also for their pathogenicity. Phylogenetic analysis showed all of the avian influenza virus isolates were closely related to avian influenza virus from China (A/Duck/China/E319-2/03(H5N1). Molecular basis of pathogenicity in HA cleavage site indicated that the isolates of avian influenza virus

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McElhaney, Janet E., Stefan Gravenstein, Peggy Krause, Jonathan W. Hooton, Craig M. Upshaw, and Paul Drinka. "Assessment of Markers of the Cell-Mediated Immune Response after Influenza Virus Infection in Frail Older Adults." Clinical Diagnostic Laboratory Immunology 5, no. 6 (1998): 840–44. http://dx.doi.org/10.1128/cdli.5.6.840-844.1998.

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ABSTRACT The purpose of this study was to determine whether measures of the cell-mediated immune response to influenza virus could be used as markers of influenza virus infection. We studied 23 subjects who developed upper respiratory, lower respiratory, or systemic symptoms during a small outbreak of influenza in a nursing home population. Influenza virus culture from nasopharyngeal swabs yielded influenza virus isolates from 7 of the 23 subjects. Only three of the subjects had a fourfold rise in antibody titer to the influenza virus antigen positivity after the infection. Granzyme B and cyto

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Kiseleva, Irina, Elena Krutikova, Ekaterina Stepanova, et al. "Cross-Protective Efficacy of Monovalent Live Influenza B Vaccines against Genetically Different Lineages of B/Victoria and B/Yamagata in Ferrets." BioMed Research International 2018 (August 30, 2018): 1–11. http://dx.doi.org/10.1155/2018/9695628.

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Background.Currently, two genetic lineages of influenza B virus, B/Victoria and B/Yamagata, are cocirculating in humans in various countries. This situation has raised a question regarding the possibility of cross-protection between B components of live attenuated influenza vaccine (LAIV) belonging to different lineages. This study aimed to assess in naïve ferrets the potential protective activity of monovalent B-LAIVs against challenge with homologous and heterologous wild-type (WT) influenza B viruses.Methods.Groups of seronegative female ferrets 5-6 months of age were given one dose of mono

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Zhou, Bin, Yi-Mo Deng, John R. Barnes, et al. "Multiplex Reverse Transcription-PCR for Simultaneous Surveillance of Influenza A and B Viruses." Journal of Clinical Microbiology 55, no. 12 (2017): 3492–501. http://dx.doi.org/10.1128/jcm.00957-17.

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ABSTRACTInfluenza A and B viruses are the causative agents of annual influenza epidemics that can be severe, and influenza A viruses intermittently cause pandemics. Sequence information from influenza virus genomes is instrumental in determining mechanisms underpinning antigenic evolution and antiviral resistance. However, due to sequence diversity and the dynamics of influenza virus evolution, rapid and high-throughput sequencing of influenza viruses remains a challenge. We developed a single-reaction influenza A/B virus (FluA/B) multiplex reverse transcription-PCR (RT-PCR) method that amplif

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Shamenova, Mira Galautdinovna, Tatyana Ivanovna Glebova, Nailya Galiveyevna Klivleyeva, et al. "Serological studies of influenza infection among population in southern region of Kazakhstan during the 2018-2021 epidemic season." Journal of the Pakistan Medical Association 73, no. 4 (2023): 804–7. http://dx.doi.org/10.47391/jpma.6348.

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Objective: To conduct serological studies of influenza infection rate during an epidemic. Methods: The retrospective study was conducted at the Research and Production Centre for Microbiology and Virology, Almaty, Kazakhstan, and comprised data, including blood samples, from patients with symptoms of acute respiratory viral infection, bronchitis and pneumonia during 2018-21 from various healthcare institutions in the Almaty region. Serological tests on blood serums were carried out sing haem agglutination inhibition assay and enzyme-linked immunosorbent assay. Data was analysed using Graph Pad

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Anokhi, Goswami, Vinzuda Mital, Trivedi Toral, Rajput Anil, Rathod Sanjay, and Kadam Mina. "Study of Influenza Virus Infection at Tertiary Care Center, Ahmedabad." International Journal of Current Pharmaceutical Review and Research 16, no. 2 (2024): 27–31. https://doi.org/10.5281/zenodo.10969824.

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Background: Influenza viruses belong to Orthomyxoviridae family of viruses. Orthomyxoviridae familyviruses are negative-sense ssRNA viruses. Four genera are described; Influenza A, B, C & D. Among this,Influenza type A infects humans and is responsible for all flu pandemics. In 2009, an Influenza pandemic causedby a novel strain of H1N1 resulted in millions of infections in more than 214 countries. Since their introductionin 1968, H3N2 Influenza viruses have undergone extensive genetic and antigenic evolution leading to numerousseasonal epidemics. In the present study, patients categorized

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Tung, Chang-Shung, Joshua L. Goodman, Henry Lu, and Catherine A. Macken. "Homology model of the structure of influenza B virus HA1." Journal of General Virology 85, no. 11 (2004): 3249–59. http://dx.doi.org/10.1099/vir.0.80021-0.

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Influenza B virus is one of two types of influenza virus that cause substantial morbidity and mortality in humans, the other being influenza A virus. The inability to provide lasting protection to humans against influenza B virus infection is due, in part, to antigenic drift of the viral surface glycoprotein, haemagglutinin (HA). Studies of the antigenicity of the HA of influenza B virus have been hampered by lack of knowledge of its structure. To address this gap, two possible models have been inferred for this structure, based on two known structures of the homologous HA of the influenza A v

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Wanitchang, Asawin, Phonphimon Wongthida, and Anan Jongkaewwattana. "Influenza B virus M2 protein can functionally replace its influenza A virus counterpart in promoting virus replication." Virology 498 (November 2016): 99–108. http://dx.doi.org/10.1016/j.virol.2016.08.016.

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Baranov, Konstantin V., Pei-Fong Wong, Ekaterina A. Stepanova, et al. "Construction of the vaccine strain of the influenza B virus with chimeric hemagglutinin to induce a cross-protective immune response." Medical academic journal 21, no. 3 (2021): 91–96. http://dx.doi.org/10.17816/maj77556.

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BACKGROUND: Influenza viruses cause worldwide epidemics, and the most effective method to prevent influenza disease is regular vaccinations. The development of new generation vaccines is aimed primarily at the formation of an immune response against a wide range of influenza viruses. One of the promising approaches is sequential vaccination with chimeric influenza viruses with identical stem domains of the hemagglutinin surface protein. AIM: The development of an experimental vaccine strain of influenza B virus with chimeric hemagglutinin consisting of head and stem domains of influenza

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Narong, Nattapol, Siriwat Manajit, Sirikarn Athipanyasil, et al. "Prevalence of Influenza Virus Type and Subtype at Siriraj Hospital, Bangkok, Thailand During 2013 - 2017." Ramathibodi Medical Journal 43, no. 3 (2020): 1–7. http://dx.doi.org/10.33165/rmj.2020.43.3.241918.

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Background: Influenza A (pandemic and seasonal H1/H3) and influenza B viruses were the predominant circulating seasonal influenza strains. Following its massive outbreak in 2009 globally, including Thailand, influenza A (H1N1) pdm09 viruses have replaced the previous seasonal H1 strain and become one of the circulating strains ever since. Both influenza A and B viruses are highly contagious and potentially cause respiratory illness ranging from mild to severe. Objective: To determine the prevalence of types and subtypes of circulating influenza virus strains in Bangkok, Thailand during 2

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Grudinin, Mikhail P., Maria M. Pisareva, Andrey B. Komissarov, et al. "Changes in the antigenic and genetic structure of influenza viruses: analysis of surveillance data of influenza A and B in Russia in 2006-2013." Microbiology Independent Research Journal 2, no. 1 (2015): 28–38. https://doi.org/10.18527/2500-2236-2015-2-1-28-38.

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   The goal of this research project was to study the natural variability of human influenza A and B viruses based on the analysis of the population structure of influenza viruses, circulating in Russia in 2006-2013, in order to determine the direction of their genetic and antigenic drift by comparison to the WHO reference strains. Our results proved that during that period significant changes occurred in the genetic structure of influenza viruses, their phylogenetic affiliation, as well as their sensitivity to antiviral drugs. According to the surveillance data, the percentage of in

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Egorov, Andrej Y. "The challenges of creating a universal influenza vaccine." Microbiology Independent Research Journal 3, no. 1 (2016): 31–41. https://doi.org/10.18527/2500-2236-2016-3-1-31-41.

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   The lack of population immunity to the periodically emerging pandemic influenza strains makes influenza infection especially dangerous. The fragmented nature of the influenza virus genome contributes to the formation of influenza virus reassortants containing genomic fragments from different strains. This mechanism is the main reason for the natural influenza virus antigenic diversity as well as for the occurrence of influenza pandemics. Vaccination is the best measure to prevent the spread of influenza infection, but the efficacy of existing vaccines is not sufficient, especially

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Wilson, Jo L., Elgin Akin, Ruifeng Zhou, et al. "The Influenza B Virus Victoria and Yamagata Lineages Display Distinct Cell Tropism and Infection-Induced Host Gene Expression in Human Nasal Epithelial Cell Cultures." Viruses 15, no. 9 (2023): 1956. http://dx.doi.org/10.3390/v15091956.

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Understanding Influenza B virus infections is of critical importance in our efforts to control severe influenza and influenza-related diseases. Until 2020, two genetic lineages of influenza B virus—Yamagata and Victoria—circulated in the population. These lineages are antigenically distinct, but the differences in virus replication or the induction of host cell responses after infection have not been carefully studied. Recent IBV clinical isolates of both lineages were obtained from influenza surveillance efforts of the Johns Hopkins Center of Excellence in Influenza Research and Response and

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Gao, Yongzhen, Lilian Sun, Jie Dong, et al. "Rapid Identification of Small Interfering Rna that Can Effectively Inhibit the Replication of Multiple Influenza B Virus Strains." Antiviral Therapy 11, no. 4 (2005): 431–38. http://dx.doi.org/10.1177/135965350601100411.

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Influenza B virus is a cause of substantial morbidity and mortality in humans and current vaccination strategies and antiviral drugs only provide limited protection. Here, we report the evaluation of small interfering RNA (siRNA) for repression of viral replication in cultured cells as well as in chicken embryos. Several siRNAs targeting conserved regions of the virus (in chemically synthesized or plasmid-encoded forms) were found to effectively block the replication of the influenza B virus. The siRNAs were found to offer broad protection over several strains of influenza B virus (B/Beijing/7

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Meijer, A., T. J. Meerhoff, L. E. Meuwissen, J. Van Der Velden, W. J. Paget, and European Influenza Surveillance Scheme (EISS). "Epidemiological and virological assessment of influenza activity in Europe during the winter 2005-2006." Eurosurveillance 12, no. 9 (2007): 11–12. http://dx.doi.org/10.2807/esm.12.09.00733-en.

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Influenza activity in Europe during the winter 2005-2006 started late January - early February 2006 and first occurred in the Netherlands, France, Greece and England. Subsequently, countries were affected in a random pattern across Europe and the period of influenza activity lasted till the end of April. In contrast to the winter seasons in the period 2001-2005, no west-east pattern was detected. In 12 out of 23 countries, the consultation rates for influenza-like illness or acute respiratory infection in the winter 2005-2006 were similar or higher than in the winter 2004-2005, despite a domin

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