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Books on the topic 'Inhibitors compounds'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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1

Kato, T., W. Krämer, K. H. Kuck, D. M. Norris, and H. Scheinpflug, eds. Sterol Biosynthesis Inhibitors and Anti-Feeding Compounds. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-69790-6.

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2

Kuznetsov, Yurii I. Organic inhibitors of corrosion of metals. Edited by Thomas J. G. N. Plenum Press, 1996.

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3

Cunningham, Bernadette Deirdre Mary. Flavones and related compounds as inhibitors of protein tyrosine kinases. Aston University. Department of Pharmaceutical Sciences, 1987.

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4

Handbook of compounds with anti-inflammatory and anti-platelet aggregation activities isolated from plants. Nova Science Publishers, 2008.

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5

1946-, Kato T., ed. Sterol biosynthesis inhibitors and anti-feeding compounds. Springer-Verlag, 1986.

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6

E, Stütz Arnold, ed. Iminosugars as glycosidase inhibitors: Nojirimycin and beyond. Wiley-VCH, 1999.

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7

Stu, Arnold E. Iminosugars As Glycosidase Inhibitors: Nojirimycin and Beyond. John Wiley & Sons, 1999.

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8

Ramesh C. Gupta, PhD, DABT, FACT, FATS. Toxicology of Organophosphate & Carbamate Compounds. Academic Press, 2005.

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9

Toxicology of Organophosphate & Carbamate Compounds. Academic Press, 2005.

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10

Böldicke, Thomas. Protein Targeting Compounds: Prediction, Selection and Activity of Specific Inhibitors. Springer, 2018.

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11

Böldicke, Thomas. Protein Targeting Compounds: Prediction, Selection and Activity of Specific Inhibitors. Springer, 2016.

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12

Bryan, Ballantyne, and Marrs Timothy C, eds. Clinical and experimental toxicology of organophosphates and carbamates. Butterworth Heinemann, 1992.

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13

Word for Windows Made Simple (Computing Made Simple). Made Simple, 1994.

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14

(Editor), Janice E. Chambers, and Patricia E. Levi (Editor), eds. Organophosphates: Chemistry, Fate, and Effects. Academic Press, 1992.

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15

(Editor), Janice E. Chambers, and Patricia E. Levi (Editor), eds. Organophosphates: Chemistry, Fate, and Effects. Academic Press, 1992.

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16

E, Chambers Janice, and Levi Patricia E, eds. Organophosphates: Chemistry, fate, and effects. Academic Press, 1992.

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17

United States. National Aeronautics and Space Administration., ed. Phospha-s-triazines and related compositions of improved hydrolytic and thermal stability. National Aeronautics and Space Administration, 1996.

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18

United States. National Aeronautics and Space Administration., ed. Phospha-s-triazines and related compositions of improved hydrolytic and thermal stability. National Aeronautics and Space Administration, 1996.

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19

United States. National Aeronautics and Space Administration., ed. Phospha-s-triazines and related compositions of improved hydrolytic and thermal stability. National Aeronautics and Space Administration, 1996.

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20

Porst, Hartmut. Erectile dysfunction. Edited by David John Ralph. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0103.

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Erectile dysfunction (ED) can be improved by changing certain lifestyle factors such as sedentary lifestyle, unhealthy food, nicotine and alcohol abuse, or optimal management of risk factors/concomitant diseases causing or aggravating ED such as dyslipidaemia, hypertension, diabetes mellitus, depression, BPH/LUTS, or hypogonadism.First choice in the medical therapy of ED are PDE-5 inhibitors such as sildenafil, vardenafil, and tadalafil used p.r.n, or on a daily low-dose regimen regarding tadalafil, especially in patients suffering from ED and BPH/LUTS. Yohimbine and L-arginine may be consider

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21

Simon, Lee S., and Marc C. Hochberg. Non-steroidal anti-inflammatory drugs. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0030.

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Non-steroidal anti-inflammatory drugs (NSAIDs) are a chemically diverse group of compounds that share three cardinal characteristics: they are anti-inflammatory, analgesic, and antipyretic. They are approved by regulatory authorities for the treatment of patients with osteoarthritis, rheumatoid arthritis, ankylosing spondylitis, acute gout, and some forms of juvenile idiopathic arthritis. There are at least 20 chemically different NSAIDs currently available in Europe and the United States. These include not only the ‘traditional’ non-selective cyclooxygenase (COX) inhibitors that inhibit both

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22

Woywodt, Alexander, and Diana Chiu. Drug-induced and toxic glomerulopathies. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0082.

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Glomerulopathies induced by particular exogenous compounds or molecules include those attributable to toxicity, and those caused by inducing an immune or autoimmune response. Tubules are more commonly the target of toxicity as they absorb and concentrate components of filtrate. Damage to endothelial cells may account for thrombotic microangiopathy in response to calcineurin inhibitors. Endothelial cells are also likely to be the target in drug-induced small vessel vasculitis. Toxicity to podocytes accounts for focal segmental glomerulosclerosis caused by pamidronate and other agents. Chloroqui

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23

Brown, T., M. Lee, Moses Lee, et al. Heterocyclic Antitumor Antibiotics. Springer, 2010.

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24

(Contributor), D. P. Arya, T. Brown (Contributor), M. Daneshtalab (Contributor), et al., eds. Heterocyclic Antitumor Antibiotics (Topics in Heterocyclic Chemistry). Springer, 2006.

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25

Dougherty, Darin D., Scott L. Rauch, and Michael A. Jenike. Pharmacological Treatments for Obsessive Compulsive Disorder. Edited by Gail Steketee. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195376210.013.0061.

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Progress in treating OCD has accelerated in recent years. Effective first-line treatments include behavior therapy and medications, with overwhelming evidence supporting the efficacy of serotonergic reuptake inhibitors (SRIs). Second-line medication treatments for OCD include augmentation of SRIs with neuroleptics, clonazepam, or buspirone, with limited support for other strategies at present. Alternative monotherapies (e.g., buspirone, clonazepam, phenelzine) have more limited supporting data and require further study. Behavior therapy, and perhaps cognitive therapy, is as effective as medica

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26

Lancaster, Jr Jack. Nitric Oxide: Principles and Actions. Academic Press, 1996.

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27

Nitric Oxide: Principles and Actions. Academic Press, 1996.

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28

Geracioti, Thomas D., Jeffrey R. Strawn, and Matthew D. Wortman. Mechanisms of Action in the Pharmacology of PTSD. Edited by Israel Liberzon and Kerry J. Ressler. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190215422.003.0020.

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This chapter reviews medications currently available for PTSD in the context of their mechanisms of action, pathophysiological relevance, and clinical efficacy data. It systematically reviews aminergic mechanisms in PTSD pharmacology, including commonly used serotonin and norepinephrine agents, selective reuptake inhibitors and receptors drugs, as well as dopaminergic agents and psychostimulants. It also discusses the use of anticonvusants and antianxiety agents that modulate GABAergic and glutamatergic signaling, such as carbamazepine, VPA, benzodiazepines, gabapentine, and others. It also re

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29

Novel Developments In Stem Cell Mobilization Focus On Cxcr4. Springer, 2012.

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30

Behrens, Frank, Michaela Koehm, and Michael J. Parnham. Synthetic DMARDs. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0028.

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Synthetic disease modifying anti-rheumatic drugs (sDMARDs) are first line systemic treatment options for management of active psoriatic arthritis (PsA). Most of the compounds are used based on evidence from clinical trials in rheumatoid arthritis and from experience in routine care. Methotrexate is often recommended as the first choice within among sDMARDs, despite controversial or missing evidence of efficacy, according to experience over many years. Leflunomide, sulfasalazine, and cyclosporin are additional established options to treat PsA. The recently approved PDE4 inhibitor, apremilast, h

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