Relevant bibliographies by topics / Intravascular activity

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Intravascular activity'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Intravascular activity"

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Schmedes, Clare M., Steven P. Grover, Yohei M. Hisada, et al. "Circulating Extracellular Vesicle Tissue Factor Activity During Orthohantavirus Infection Is Associated With Intravascular Coagulation." Journal of Infectious Diseases 222, no. 8 (2019): 1392–99. http://dx.doi.org/10.1093/infdis/jiz597.

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Abstract Background Puumala orthohantavirus (PUUV) causes hemorrhagic fever with renal syndrome (HFRS). Patients with HFRS have an activated coagulation system with increased risk of disseminated intravascular coagulation (DIC) and venous thromboembolism (VTE). The aim of the study was to determine whether circulating extracellular vesicle tissue factor (EVTF) activity levels associates with DIC and VTE (grouped as intravascular coagulation) in HFRS patients. Methods Longitudinal samples were collected from 88 HFRS patients. Patients were stratified into groups of those with intravascular coag
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McDonald, Braedon, Rachelle Davis, and Craig N. Jenne. "Neutrophil extracellular traps (NETs) promote disseminated intravascular coagulation in sepsis." Journal of Immunology 196, no. 1_Supplement (2016): 60.8. http://dx.doi.org/10.4049/jimmunol.196.supp.60.8.

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Abstract Neutrophil extracellular traps (NETs; webs of DNA coated in anti-microbial proteins) are released into the vasculature during sepsis and contribute to organ damage. Various components of NETs have been shown to modulate the coagulation cascade in vitro. We hypothesized that NETs released into the vasculature during sepsis activate intravascular coagulation, leading to microvascular hypoperfusion and end-organ damage. Multi-color confocal intravital microscopy was used in mouse models of sepsis (endotoxemia and E. coli peritonitis) to visualize and quantify neutrophil and platelet traf
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Yusupova, A. N., and I. A. Andrushko. "Blood 5´-nucleotidase activity in gestosis complicated pregnancy." Kazan medical journal 68, no. 3 (1987): 202–5. http://dx.doi.org/10.17816/kazmj96060.

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The data on the role of disseminated intravascular coagulation in the occurrence of ischemic lesions in severe forms of this pathology are attracting increasing attention in the study of the pathogenesis of pregnant gestosis. V. P. Skipetrov suggests that coagulopathy developing in pregnant gestosis is the result of increased thromboplastic activity of the blood in response to damaging agents. Direct evidence has been obtained that severe forms of pregnancy gestosis are accompanied by a chronic form of disseminated intravascular coagulation syndrome (DIC). Thromboplastinemia acts as one of the
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TURNER, T. T., K. J. BROWN, and C. L. SPANN. "Testicular Intravascular Volume and Microvessel Mitotic Activity: Effect of Experimental Varicocele." Journal of Andrology 14, no. 3 (1993): 180–86. http://dx.doi.org/10.1002/j.1939-4640.1993.tb00377.x.

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ABSTRACT: Testicular blood flow increases bilaterally in animals with experimental left varicocele (ELV). The present experiments were undertaken to determine if increases in testicular intravascular volume and capillary growth were potential contributors to this increase in flow. Testicular intravascular volume (μl/g) was estimated by determining testicular vascular 125I‐albumin space. Microvessel mitotic activity was estimated by examining 3 H‐thymidine incorporation into endothelial nuclei, as assayed by silver grain localization over microvessel endothelial nuclei after exposure of testes
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Andrushko, I. A. "Thromboplastinemia — an initiator of continuous hemocoagulation in organism and disseminated interavascular coagulation syndrome." Kazan medical journal 75, no. 3 (1994): 185–87. http://dx.doi.org/10.17816/kazmj89831.

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The concept of thromboplastinemia as an initial factor of phisiologic and pathologic intravascular activation of hemostasis system is fully validated. It is stated that tissue thromboplastin comprises the fragments of cell membranes where 5'-nucleotidase is the marker enzyme. The increase of this enzyme activity is indirect evidence of the presence of tissue thromboplastin in vascular bed, that is bears witness to thromboplastinemia. Experimentally and in clinic the direct correlation is established between the activity increase of 5'-nucleotidase and the extent of hypercoagu- lemia, proving t
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Cécilia, Roustit, Vallé Baptiste, Clouzeau Benjamin, et al. "Acute Hemolysis in the Emergency Department: Think aboutClostridium perfringens!" Case Reports in Emergency Medicine 2013 (2013): 1–2. http://dx.doi.org/10.1155/2013/948071.

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Clostridium perfringens(CP) gives several clinical settings, from an asymptomatic to a massive intravascular hemolysis. We report a case of fatal intravascular hemolysis due to CP septicemia having a hepatic supposed starting point in the emergency department. Like in many cases, the diagnosis was made when patient had already gone into shock and died. The CP septicemia often complicated the course of the digestive or genital pathologies. The alpha toxin can damage the structural integrity of the red cell membrane by means of a phospholipase activity. Nevertheless, a massive intravascular hemo
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Wenner, Megan M., William C. Rose, Erin P. Delaney, Michael E. Stillabower, and William B. Farquhar. "Influence of plasma osmolality on baroreflex control of sympathetic activity." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 4 (2007): H2313—H2319. http://dx.doi.org/10.1152/ajpheart.01383.2006.

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The purpose of this study was to determine if plasma osmolality alters baroreflex control of sympathetic activity when controlling for a change in intravascular volume; we hypothesized that baroreflex control of sympathetic activity would be greater during a hyperosmotic stimulus compared with an isoosmotic stimulus when intravascular volume expansion was matched. Seven healthy subjects (25 ± 2 yr) completed two intravenous infusions: a hypertonic saline infusion (HSI; 3% NaCl) and, on a separate occasion, an isotonic saline infusion (ISO; 0.9% NaCl), both at a rate of 0.15 ml·kg−1·min−1. To i
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Stief, Thomas W. "Coagulation Activation by Lipopolysaccharides." Clinical and Applied Thrombosis/Hemostasis 15, no. 2 (2007): 209–19. http://dx.doi.org/10.1177/1076029607309256.

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Lipopolysaccharides at approximate plasma reactivities >3 ng/mL or β-glucans at >0.5-1 μg/mL are toxic for human blood; lipopolysaccharide interacts with membrane components of susceptible cells (eg, monocytes) activating phospholipase A2 that destroys the cell membrane. Cell fragments (microparticles or DNA) possess polynegative niches that activate intrinsic hemostasis. Pathologic disseminated intravascular coagulation arises. Blood vessels are obstructed by disseminated thrombi, and vital organ areas become ischemic. Multiorgan failure threatens life of the patient. Diagnosis and ther
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Tseng, Kuo-Yao, Yu-Tsung Huang, Yu-Ting Huang, et al. "Regulation of candidalysin underlies Candida albicans persistence in intravascular catheters by modulating NETosis." PLOS Pathogens 20, no. 6 (2024): e1012319. http://dx.doi.org/10.1371/journal.ppat.1012319.

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Candida albicans is a leading cause of intravascular catheter-related infections. The capacity for biofilm formation has been proposed to contribute to the persistence of this fungal pathogen on catheter surfaces. While efforts have been devoted to identifying microbial factors that modulate C. albicans biofilm formation in vitro, our understanding of the host factors that may shape C. albicans persistence in intravascular catheters is lacking. Here, we used multiphoton microscopy to characterize biofilms in intravascular catheters removed from candidiasis patients. We demonstrated that, NETos
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Lasson, Å., and K. Ohlsson. "Disseminated Intravascular Coagulation and Antiprotease Activity in Acute Human Pancreatitis." Scandinavian Journal of Gastroenterology 21, sup126 (1986): 35–39. http://dx.doi.org/10.3109/00365528609091890.

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Dissertations / Theses on the topic "Intravascular activity"

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Horobin, Jarod. "The effects of shear stress on blood." Thesis, Griffith University, 2018. http://hdl.handle.net/10072/380568.

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The scientist’s advancement in human biological and physiological understanding, and the engineer’s increasing capabilities has allowed for development of mechanical circulatory support (MCS) devices with improved biocompatibility. Indeed, investigations into the mechanical responses of red blood cells (RBC) to supra-physiological shear stress and the underlying biochemical processes have provided an outline of the RBC mechanical sensitivity (MS) and allowed for identification of the functional tolerance of RBC (i.e., the ability of the RBC to deform) to shear stress. Nevertheless, the adaptat
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Books on the topic "Intravascular activity"

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Grom, Alexei A., and Athimalaipet V. Ramanan. Macrophage activation syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0168.

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Macrophage activation syndrome (MAS) is a life-threatening condition caused by excessive activation and proliferation of T lymphocytes and haemophagocytic macrophages. Although MAS has been reported in association with almost any rheumatic disease, it is by far most common in systemic juvenile idiopathic arthritis. Flares of the underlying disease or infection are most common triggers of MAS. The pathognomonic feature of MAS is typically found in bone marrow: numerous, well-differentiated macrophagic histiocytes phagocytosing normal haematopoietic elements. The expansion of these histiocytes l
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Book chapters on the topic "Intravascular activity"

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Osborn, Eric A., and Farouc A. Jaffer. "Intravascular Molecular Imaging of Proteolytic Activity." In Cardiovascular Imaging. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09268-3_4.

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Serrato, Lluìs Albert Matas, Alessandro Bilella, and Simon Blanchoud. "Noninvasive Intravascular Microtransfusion in Colonial Tunicates." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2172-1_21.

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AbstractTunicates are a diverse group of worldwide marine filter-feeders that are vertebrates’ closest invertebrate relatives. Colonial tunicates are the only know chordates that have been shown to undergo whole-body regeneration (WBR). Botrylloides in particular can regenerate one fully functional adult from a minute fragment of their vascular system in as little as 10 days. This regenerative process relies on the proliferation of circulating stem cells, likely supported by the activity of some of the 11 identified types of hemocytes. To study and challenge WBR, it is thus important to have t
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Gastounioti, A., S. Golemati, P. Mermigkas, M. Prevenios, and K. S. Nikita. "Implications of the Kinematic Activity of the Atherosclerotic Plaque." In Computing and Visualization for Intravascular Imaging and Computer-Assisted Stenting. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-811018-8.00005-9.

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Abarbanel, David N., and Ivan D. Carabenciov. "Night Sweats and Paraparesis." In Mayo Clinic Cases in Neuroimmunology, edited by Andrew McKeon, B. Mark Keegan, and W. Oliver Tobin. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197583425.003.0064.

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A 78-year-old man sought care for saddle anesthesia, left lower extremity numbness, and bilateral lower extremity weakness. The sensory loss occurred suddenly, starting initially in the left perianal region and over the course of 3 hours extending down to involve the entirety of the left lower extremity. Symptoms were stable until 3 weeks later, when he had a few episodes of urinary incontinence. Diffuse, severe, bilateral, lower extremity weakness developed. The patient reported 6 months of intermittent night sweats. Serum studies were notable for pancytopenia and increased erythrocyte sedime
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N. Magnani, Harry. "Danaparoid Sodium: A Review of Its Use in Hepatic Thrombotic Disorders." In Anticoagulation - Current Perspectives [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.103851.

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Danaparoid sodium is an antithrombotic isolated from porcine mucosa. Its main constituent is a mixture of heparan sulphates that inhibits thrombin generation and also possesses anti-inflammatory and immune-modulatory activity. It has shown safety and efficacy in its main indications of deep venous thrombosis prophylaxis, heparin-induced thrombocytopenia treatment and disseminated intravascular thrombosis treatment. In addition, there are reports of its off-label use for the treatment of portal vein thrombosis in adults and for prevention of the hepatic thrombotic microangiopathies haematogenou
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MD, MsSc, Martin Flores-Aguilar. "Optical Coherence Tomography vs Optical Coherence Tomography Angiography in the Differential Diagnosis of Choroidal and Vitreoretinal Diseases." In Optical Coherence Tomography Angiography for Choroidal and Vitreoretinal Disorders - Part 1. BENTHAM SCIENCE PUBLISHERS, 2023. http://dx.doi.org/10.2174/9789815124095123010006.

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Optical coherence tomography (OCT) is a practical and common imaging method for the study of diseases of the retina, choroid, and vitreoretinal interface. Software and technological advances have allowed us to observe changes in the retinal at resolutions less than 5 µm; the development of angiography with OCT (OCTA) allows us to three-dimensionally evaluate the existing perfusion in the analyzed retina and choroid non-invasively and without a specific dye, such as fluorescein or indocyanine green angiography. We can detect important clinical differences between OCT and OCTA, although these ap
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Turner A.P.F. "CHEMICAL SENSORS FOR IN VIVO MONITORING." In Studies in Health Technology and Informatics. IOS Press, 1993. https://doi.org/10.3233/978-1-60750-851-9-99.

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The Concerted Action analysed the clinical problems, identified suitable analytes, and considered sites for continuous in vivo monitoring. As a result its activity focused onto subcutaneous glucose monitoring for diabetes, and intravascular oxygen and ion monitoring for critical care. Common standards for the evaluation and use of continuous invasive glucose sensors were agree. Device strategies were formulated, defining the necessary characteristics and matching them with available technologies. The need for new technology was identified and the development of these systems was promoted, with
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Schoenhagen, Paul, Richard D. White, and Steven E. Nissen. "Imaging of High-Risk Atherosclerotic Plaque by Intravascular Ultrasound: Focal Assessment of Morphology and Vulnerability or Systemic Assessment of Disease Burden and Activity?" In High-Risk Atherosclerotic Plaques. CRC Press, 2004. http://dx.doi.org/10.1201/9781420037883-5.

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Becker, Richard C., and Frederick A. Spencer. "Fibrinolytic Agents." In Fibrinolytic and Antithrombotic Therapy. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195155648.003.0011.

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The fibrinolytic system plays a vital role in maintaining vital organ homeostasis. Fibrinolysis, defined as the dissolution of fibrin (the major scaffold for intravascular thrombus), is the process that regulates thrombus growth after hemostasis has been achieved, thus preserving tissue perfusion. An understanding of fibrinolysis has led to the development of pharmacologic agents that can be used in the treatment of arterial and venous thrombotic disorders, including acute myocardial infarction, acute ischemic stroke, and pulmonary embolism. Fibrinolytic therapy makes use of the vascular syste
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Grom, Alexei A., and Athimalaipet V. Ramanan. "Macrophage activation syndrome." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0168_update_001.

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Macrophage activation syndrome (MAS) is a life-threatening condition caused by excessive activation and proliferation of T lymphocytes and haemophagocytic macrophages. Although MAS has been reported in association with almost any rheumatic disease, it is by far most common in systemic juvenile idiopathic arthritis. Flares of the underlying disease or infection are most common triggers of MAS. The pathognomonic feature of MAS is typically found in bone marrow: numerous, well-differentiated macrophagic histiocytes phagocytosing normal haematopoietic elements. The expansion of these histiocytes l
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Conference papers on the topic "Intravascular activity"

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Páramo, J. A., B. Cuesta, M. Hernrnández, J. Fernrnández, M. J. Paloma, and E. Rocha. "PLASMINOGEN ACTIVATOR INHIBITOR ACTIVITY IN SEPTICAEMIA." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644694.

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In vitro and in vivo studies have shown that endotoxin induces a marked increase in plasma plasminogen activator inhibitor (PAI) activity. Plasma PAI and endotoxin concentration (limulus lysate chromo-genic peptide substrate) were determined in 61 patients with sepsis: temperature greater than 38° and either positive blood cultures (n= 32) or negative blood cultures in neutropenic patients. Thirty age-matched healthy subjects served as control group.There was a marked increase in PAI in patients (7.1±10.5 U/ml) as compared to controls (0.9±0.8 U/ml) with statistical differences (p<0.002). M
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Falang, A., G. M. Alessio, M. Donati, and T. A. Barbui. "DISSEMINATED INTRAVASCULAR COAGULATION (DIC) AND ACUTE LEUKEMIA:IDENTIFICATION OF A NEW CELLULAR PROCOAGULANT." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643661.

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There is an enhanced incidence (>50%) of severe coagulopathy in association with several types of acute leukemias. Cell associated procoagulants are considered important in this context. So far only a Tissue Factor (TF)-type procoagulant has been described in leukemic cells. We have set up here the experimentalconditions to identify other possible cellular procoagulants in leukemia. We have tested blast cell extracts from 21 patients with 5 different cytological subtypes (from Ml to M5 of acute non lymphoid leukemia (ANLL), according to theFAB classification, in order to assay whether they
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Engebretsen, L. F., P. Kierulf, T. Lyberg, A. B. Andersen, M. Gulla, and G. B. Joo. "HUMAN PERIPHERAL MONOCYTES STIMULATED WITH ENDOTOXIN IN VITRO SHOWED A FAST PROCOAGULANT ACTIVITY (PCA) ELEVATION AND A LATE ANTIFIBRINOLYTIC RESPONSE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644253.

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Septicemia causes severe disseminated intravascular coagulation (DIC). Monocytes may contribute to the DIC process through endotoxin stimulated synthesis of PCA. This study was initiated to evaluate the role of the monocytes in DIC further. Blood was taken from healthy persons. Mononuclear leucocytes (30?o monocytes) in microtiter wells (25.000 monocytes/well) were added 1 ug/ml endotoxin, and kept in culture from 0 to 160 hours. The cells were harvested, and the PCA (thromboplastin), the fibrinolytic activity (urokinase-PA), and the antifibrinolytic response (plasminogen activator inhibitor-P
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Sultan, Y., A. Harris, G. Strauch, D. De Lauture, and A. Venot. "RATIONAL FOR A DYNAMIC TEST TO INVESTIGATE POTENTIAL VASCULAR FIBRINOLYSIS IN PATIENTS AT RISK OF PERSISTENT THROMBOSIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643112.

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Defense mechanism against intravascular clot formation is dependant upon the release of free tissue plasminogen activator (t-PA) from the vascular wall. To investigate this function a dynamic test is necessary to differenciate patients at risk of persistant thrombosis from patients who possess potential capacity to dissolve intravascular clots. In the present study, three different routes of administration of DDAVP and venous occlusion (VO) were applied to 9 healthy young volunteers in order to determine the best stimulus and the best test to assess individual capacity of releasing free t-PA i
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Andersson, T. R., H. Bell, P. M. Sandset, O. R. Ødegaard, and L.-M. Aamodt. "“NEW” COAGULATION INHIBITORS LEVELS IN PNEUMONIA DISSEMINATED INTRAVASCULAR COAGULATION AND LIVER DISEASES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643914.

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The activity levels of the “new” coagulation inhibitors, heparin cofactor II (HC II) and extrinsic pathway inhibitor (EPI),have been determined with chromogenic substrates assays, in patients with pneumonia (n=8), disseminated intravascular coagulation (DIC) (n=8) and various liver diseases (n=19). For comparison antithrombin (AT) and Protein C (PC) were also measured. In cases with DIC low values (<50%) for HC II,AT and PC were found, while EPI showed a much greater variation (60-190%). Persistent low values heralds a poor prognosis.In survivors is rapidly normalized. In pneumonia , initia
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Takahashi, Hoyu, Wataru Tatewaki, Tadao Nakamura, Masaharu Hanano, Ken Wada, and Akira Shibata. "COAGULATION STUDIES IN THROMBOTIC THROMBOCYTOPENIC PURPURA, WITH SPECIAL REFERENCE TO VON WILLEBRAND FACTOR ABNORMALITIES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644586.

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The profile of blood coagulation and fibrinolysis was studied in detail in 8 patients with thrombotic thrombocytopenic purpura (TTP), who had most of the characteristic findings such as fluctuating neurologic signs, schistocytic hemolytic anemia, marked thrombocytopenia, renal abnormalities and fever. Fibrinogen (2.2-3.6 g/L) and factor XIII levels were normal in all patients, while FDP values were slightly elevated (from below 5 to 4-0 mg/L). Anti thrombin III, alpha 2-plasmin inhibitor and plasminogen were normal in all patients except one with elevated FDP. Alpha 2-macroglobulin was normal
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Bajaj, M. S., S. V. Rana, R. B. Wysolmerski, and S. P. Bajaj. "INHIBITOR OF THE FACTOR VIIA-TISSUE FACTOR COMPLEX IS REDUCED IN PATIENTS WITH DISSEMINATED INTRAVASCULAR COAGULATION BUT NOT IN PATIENTS WITH SEVERE HEPATOCELLULAR DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643916.

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Recently, inhibition of factor VIIa-tissue factor activity by a plasma component(s) which requires factor Xa has been described. In this communication, we have developed a specific radiometric assay (which utilizes 3H-factor IX and is sensitive to <1% of plasma level) for this inhibitor and have measured its activity in various disease states. Strikingly, the levels of this inhibitor were found to be normal in patients with advanced chronic hepatocellular disease but low in patients with disseminated intravascular coagulation (DIC). When endotoxin was used to induce DIC in rabbits, the leve
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Grand, B., A. Blanco, A. Kempfer, G. Elgue, D. Riveros, and M. Lazzari. "ADENOCARClNOMPrOF COLON AND THROMBOSIS: INCREASED THROMBIN,PLASMIN OR NEUTRAL PROTEASES ACTIVITY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644805.

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Thrombotic and hemorrhagic complications are common in cancer patients and are an important cause of morbidity andmortality in these subjects.Hemostatic abnormalities, with or without these complications are found in nearly 95% of these patients .These abnormalities havebeen atributed to the presence of intravascular coagulation produced by an increased fibrin gene rat ion. We report apatient with adenocarcinoma of colon and multiple venous thrombosis, whose laboratory findings were consistent with anincreased proteolytic activity different from thrombin or plasmin.A 36 year old man with deep
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Takamatsu, J., H. Saito, T. Kamiya, Y. Muranaka, Y. F. Minami, and H. T. Fan. "A NEW MUCOPOLYSACCHARIDE FROM STICHPUS JAPONICUS(SEA CUCUMBER) AND ITS ANTICOAGULANT PRORETIES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644185.

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A new mucopolysaccharide(FGAG) has been isolated from the cell wall of Stichopus japonicua. The molecular weight of FGAG is about 50,000. The component sugar of the FGAG are identified as galactosamine, glucuronic acid, fucose and sulfate with the molar ratioof 1:0.94:0.84:3.60,respectively. The anticoagulant effects of FGAG were studied. At low concentration(lμg/ml), FGAG completely inhibited the rabbit plateletaggregation induced by thrombin and prolonged thrombin time of not only human plasma but purified fibrinogen solution to a similar extent, suggesting that action of FGAG is not depend
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Egbring, R., R. Seitz, M. Wolf, L. Lerch, and T. Menges. "PROTEINASE-INHIBITOR COMPLEXES (PIC) IN SEPTIC AND NON-SEPTIC SHOCK. COAGULATION; LEUKOCYTE AND BACTERIAL PROTEASE INHIBITION BY MEANS OF PLASMA-INHIBITOR REPLACEMENT." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644244.

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In septic or cardiac shock antithrombin III-thrombin (AT III-Thr) and a1antitrypsin-elastase(a1AT-ELP) as well as a2antiplas-min-plasmin (a2AP-Pl) are found to be elevated to different extents. In cardiac shock AT III-Thr is predominantly increased, while in septic disorders a2AT-ELP as indicator of leukocyte stimulation is additionally found to be elevated. Stimuli for leukocyte activation are bacterial endotoxins, immune complexes, factor Xlla and others. The possible action of bacterial proteases during septic infections is only known in animal models. To stop hemorrhagic complications in d
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