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Dissertations / Theses on the topic 'Leukaemia Inhibitory Factor Receptor'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 February 2022

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1

Chobotova, Katya. "Ligand binding determinants of LIF receptor." Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244596.

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2

Hill, Eric J. "Polarised secretion of leukaemia inhibitory factor." Thesis, Aston University, 2004. http://publications.aston.ac.uk/11019/.

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Leukaemia inhibitory factor (LIF) is a cytokine that is active on a wide variety of cells. Multiple LIF transcripts have been described. The transcripts LIF-D and LIF-M encode different signal peptides, which in mouse have been associated with differential localisation of the mature protein. LIF-D is associated with a freely diffusible protein, whereas the LIF-M is associated with the extracellular matrix. The polarity of LIF secretion has yet to be described and could illuminate the mechanisms of LIF localisation. Here the polarised endogenous secretion of human LIF and IL-6 in Caco-2 cells w

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3

Grey, Laura M. "Structure and function of leukaemia inhibitory factor." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.359460.

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4

Sherwin, James Robert Alexander. "The role of leukaemia inhibitory factor in endometrial receptivity." Thesis, University of Cambridge, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.619897.

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5

Muthukumarana, Poorni Apsara de Silva. "Stem cell factors, axotrophin and leukaemia inhibitory factor in immune regulation." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.611884.

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6

Voyle, Roger Bruce. "Mechanisms of intracellular and extracellular cytokine production from the human leukaemia inhibitory factor gene." Title page, contents and summary only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phv975.pdf.

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Addendum attached to back facing leaves. Includes bibliographical references (leaves 172-199). The findings establish leukemia inhibitory factor, and possibly oncostatin M, as new members of a small but growing class of cytokines produced in an intracellularly active form and also suggest that the production of alternate transcripts and intercellularly-retained proteins may be a common and important feature of cytokines of the IL-6 and other families.

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7

Wiest, Stephanie. "Mutationen im Leukaemia-inhibitory-factor-(LIF)-Gen bei wiederholtem Implantationsversagen nach extrakorporaler Befruchtung." [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=975102621.

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8

Segrave, Alicia Maree. "An investigation of the pharmacokinetics and lymphatic transport of recombinant human leukaemia inhibitory factor." Monash University, Dept. of Pharmaceutics, 2004. http://arrow.monash.edu.au/hdl/1959.1/9389.

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9

Schiemann, William Paul. "Determination and characterization of leukemia inhibitory factor receptor signal transduction systems /." Thesis, Connect to this title online; UW restricted, 1996. http://hdl.handle.net/1773/6277.

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10

West, Peter William. "The regulation of Toll-like receptor signalling by macrophage migration inhibitory factor." Thesis, University of Sheffield, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.505343.

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Toll-like receptors (TLRs) fonn a vital part of the innate immune response to infection through the recognition of diverse molecular patterns leading to the generation of an inflammatory reaction. The resulting cytokines act on both tissue and immune cells to coordinate the response to infection. Cytokine networks also play an important role in the modulation of an increasing number of diseases which we now understand to have an inflammatory basis. TLR activation has been implicated in both chronic and acute diseases, and understanding and modulation of these responses may be central to th'e m

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11

Ng, Yu Pong. "Leukemia inhibitory factor receptor signaling in NGF-induced neuronal differentiation of PC12 cells /." View abstract or full-text, 2004. http://library.ust.hk/cgi/db/thesis.pl?BICH%202004%20NG.

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Thesis (Ph. D.)--Hong Kong University of Science and Technology, 2004.<br>Includes bibliographical references (leaves 134-172). Also available in electronic version. Access restricted to campus users.

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12

Cui, Shuliang. "Molecular cloning, characterisation and expression of the leukaemia inhibitory factor (LIF) gene from the marsupial Sminthopsis crassicaudata /." Title page, table of contents and summary only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phc9662.pdf.

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13

Jarrett, Nicholas Hywel. "Purification and characterisation of the 5-HT1A receptor inhibitory factor in alcoholic beverages." Thesis, Imperial College London, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298335.

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14

Port, Martha D. "Regulation of expression and function of neurokine receptors /." Thesis, Connect to this title online; UW restricted, 2008. http://hdl.handle.net/1773/6283.

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15

Taylor, Alan. "The role of leukaemia inhibitory factor and a leukaemic associated inhibitor in the control of the proliferation of haematopoietic stem cells." Thesis, University of St Andrews, 1996. http://hdl.handle.net/10023/14962.

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Activities associated with, or interacting with, leukaemic cell populations were assayed for the ability to influence in vitro haematopoiesis. The first of these, the glycoprotein leukaemia inhibitory factor (LIF), has a role in aspects of murine, non human primate and human haematopoiesis. It is thought to be particularly important in the development of megakaryocytes and is also known to induce the terminal differentiation of certain leukaemic cell lines. LIF was assayed both for direct and indirect effects on the proliferation of haematopoietic precursor cell populations in vitro. As a dire

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16

Fujita, Kazuyuki. "Administration of tymocytes derived from non-pregnant mice induces an endometrial receptive stage and leukaemia inhibitory factor expression in the uterus." Kyoto University, 2000. http://hdl.handle.net/2433/180877.

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17

Malki, Marwa. "Correlations between unexplained infertility and single nucleotide polymorphism in the genes of leukemia inhibitory factor receptor and gp130." Thesis, Uppsala University, Department of Medical Biochemistry and Microbiology, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-128921.

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<p>About 30 % of all infertile couples suffer from infertility of an unexplained cause. Leukemia inhibitory factor (LIF) is a glycoprotein produced by the endometrium and is an important cytokine in the implantation process. LIF exerts its biological functions through heterodimerization of its two receptors: LIF receptor (LIFR) and gp130. Point mutations in the LIF gene have been associated with female infertility. The aim of this study was to investigate whether single nucleotide polymorphisms (SNPs) in the genes of LIFR and gp130 could cause reduced fertility in women. To this end, 115 sampl

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18

Matluk, Nicholas N. "Neurotrophin Receptor-Interacting Melanoma-Associated Antigen Protein Activates Nuclear Factor Kappa Beta Repressing Bone Morphogenic Protein Induced Apoptosis Through Macrophage Migration Inhibitory Factor Expression." Fogler Library, University of Maine, 2011. http://www.library.umaine.edu/theses/pdf/MatlukN2011.pdf.

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19

Ziegler, Inna. "Posttranslationale Modifikationen der IL-6-Typ-Zytokin-Rezeptoren gp130 und LIFR und ihr Einfluss auf die Assoziation mit Detergenz-resistenten Membranmikrodomänen (DRM)." [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:100-opus-3124.

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20

Alexandrou, Estella. "The therapeutic effect of LIF in EAE-associated axonal injury." Connect to thesis, 2009. http://repository.unimelb.edu.au/10187/5514.

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Axonal degeneration is a major pathological feature of the central nervous system (CNS) inflammatory demyelinating disease multiple sclerosis (MS). This axonal degeneration has major consequences, as functional axonal regeneration in the CNS is largely absent. Cumulative axonal degeneration is the likely cause of the majority of progressive MS-related disability, and therefore, the need for novel neuroprotective therapies for MS exists. Experimental autoimmune encephalomyelitis (EAE), an animal model of MS pathology, also produces axonal injury. In particular, the optic nerve and spinal cord

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21

McKay, Hart Andrew. "Sensory neuronal protection & improving regeneration after peripheral nerve injury." Doctoral thesis, Umeå universitet, Handkirurgi, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-52.

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Peripheral nerve trauma is a common cause of considerable functional morbidity, and healthcare expenditure. Particularly in the ~15% of injuries unsuitable for primary repair, standard clinical management results in inadequate sensory restitution in the majority of cases, despite the rigorous application of complex microsurgical techniques. This can largely be explained by the failure of surgical management to adequately address the neurobiological hurdles to optimal regeneration. Most significant of these is the extensive sensory neuronal death that follows injury, and which is accompanied by

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22

Escary, Jean-Louis. "Étude fonctionnelle des gènes codant pour la cytokine LIF et pour son récepteur chez la souris." Paris 6, 1994. http://www.theses.fr/1994PA066566.

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Le LIF est un facteur protéique dont l'action biologique est pléiotropique in vitro et in vivo. En effet, en fonction du type de cellules cibles, cette action varie de la survie et/ou la prolifération à la différenciation. En particulier, le LIF induit la survie et la prolifération des cellules souches embryonnaires de souris (les cellules es). La technologie de recombinaison homologue dans ces cellules nous a permis d'obtenir une lignée de souris mutantes pour le LIF, appelées souris LIF-/-, dans laquelle le gène LIF n'est plus fonctionnel. Plusieurs fonctions biologiques différentes sont alt

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23

Humphrey, Peter Saah. "Signal transduction mechanisms for stem cell differentation into cardiomyocytes." Thesis, University of Hertfordshire, 2009. http://hdl.handle.net/2299/3760.

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Cardiovascular diseases are among the leading causes of death worldwide and particularly in the developed World. The search for new therapeutic approaches for improving the functions of the damaged heart is therefore a critical endeavour. Myocardial infarction, which can lead to heart failure, is associated with irreversible loss of functional cardiomyocytes. The loss of cardiomyocytes poses a major difficulty for treating the damaged heart since terminally differentiated cardiomyocytes have very limited regeneration potential. Currently, the only effective treatment for severe heart failure i

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24

Drechsler, Johannes. "Determination of the hypertrophic potential of Oncostatin M on rat cardiac cells and the characterisation of the receptor complexes utilised by rat Oncostatin M." Doctoral thesis, 2012. https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-85215.

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Interleukin-6 (IL-6), oncostatin M (OSM), leukaemia inhibitory factor (LIF) and cardiotrophin-1 (CT-1) are members of the IL-6-type cytokine family that is characterised by sharing the common receptor subunit gp130. While the involvement of these polypeptides in cell differentiation, cell survival, proliferation, apoptosis, inflammation, haematopoiesis, immune response and acute phase reaction has already been demonstrated, the description of their role in development and progression of cardiac hypertrophy is still rather limited. A model has been postulated that declares the transient express

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25

Wiest, Stephanie. "Mutationen im Leukaemia-Inhibitory-Factor (LIF)-Gen bei wiederholtem Implantationsversagen nach extrakorporaler Befruchtung." Doctoral thesis, 2004. https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-12864.

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Das Implantationsversagen stellt die häufigste Ursache für den Misserfolg der menschlichen Reproduktion dar und spielt wahrscheinlich eine bedeutende Rolle bei wiederholt erfolgloser assistierter Reproduktion (IVF/ICSI und ET). Die hormonell eingeleitete Regulation der Implantation beinhaltet eine komplexe Folge von Signalen zwischen Embryo und Endometrium, deren regelrechter Ablauf für die erfolgreiche Einrichtung einer Schwangerschaft entscheidend ist. Eine Fülle von Faktoren wie Zytokine und Wachstumsfaktoren spielen hier eine bedeutende Rolle. Unter diesen Mediatoren ist auch Leukaemia Inh

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26

Wiest, Stephanie [Verfasser]. "Mutationen im Leukaemia-inhibitory-factor-(LIF)-Gen bei wiederholtem Implantationsversagen nach extrakorporaler Befruchtung / vorgelegt von Stephanie Wiest." 2005. http://d-nb.info/975102621/34.

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