Relevant bibliographies by topics / Mitochondrial Associated Regulatory Factor (Marf)

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Academic literature on the topic 'Mitochondrial Associated Regulatory Factor (Marf)'

Author: Grafiati
Published: 6 September 2023
Last updated: 31 July 2025

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Journal articles on the topic "Mitochondrial Associated Regulatory Factor (Marf)"

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Debattisti, Valentina, Diana Pendin, Elena Ziviani, Andrea Daga, and Luca Scorrano. "Reduction of endoplasmic reticulum stress attenuates the defects caused by Drosophila mitofusin depletion." Journal of Cell Biology 204, no. 3 (2014): 303–12. http://dx.doi.org/10.1083/jcb.201306121.

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Ablation of the mitochondrial fusion and endoplasmic reticulum (ER)–tethering protein Mfn2 causes ER stress, but whether this is just an epiphenomenon of mitochondrial dysfunction or a contributor to the phenotypes in mitofusin (Mfn)-depleted Drosophila melanogaster is unclear. In this paper, we show that reduction of ER dysfunction ameliorates the functional and developmental defects of flies lacking the single Mfn mitochondrial assembly regulatory factor (Marf). Ubiquitous or neuron- and muscle-specific Marf ablation was lethal, altering mitochondrial and ER morphology and triggering ER stre
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2

Chen, Yutong, Zhixin Liu, Ning An, et al. "Platelet-Derived Mitochondria Attenuate 5-FU-Induced Injury to Bone-Associated Mesenchymal Stem Cells." Stem Cells International 2023 (January 30, 2023): 1–20. http://dx.doi.org/10.1155/2023/7482546.

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Background. Myelosuppression is a common condition during chemotherapy. Bone-associated mesenchymal stem cells (BA-MSCs) play an essential role in the composition of the hematopoietic microenvironment and support hematopoietic activity. However, chemotherapy-induced damage to BA-MSCs is rarely studied. Recent studies have shown that platelets promote the wound-healing capability of MSCs by mitochondrial transfer. Therefore, this study is aimed at investigating the chemotherapy-induced damage to BA-MSCs and the therapeutic effect of platelet-derived mitochondria. Material/Methods. We establishe
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Morciano, Giampaolo, Carlotta Giorgi, Dario Balestra, et al. "Mcl-1 involvement in mitochondrial dynamics is associated with apoptotic cell death." Molecular Biology of the Cell 27, no. 1 (2016): 20–34. http://dx.doi.org/10.1091/mbc.e15-01-0028.

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The B-cell lymphoma-2 (Bcl-2) family proteins are critical regulators of apoptosis and consist of both proapoptotic and antiapoptotic factors. Within this family, the myeloid cell leukemia factor 1 (Mcl-1) protein exists in two forms as the result of alternative splicing. The long variant (Mcl-1L) acts as an antiapoptotic factor, whereas the short isoform (Mcl-1S) displays proapoptotic activity. In this study, using splice-switching antisense oligonucleotides (ASOs), we increased the synthesis of Mcl-1S, which induced a concurrent reduction of Mcl-1L, resulting in increased sensitivity of canc
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Lin, Ruizhu, and Risto Kerkelä. "Regulatory Mechanisms of Mitochondrial Function and Cardiac Aging." International Journal of Molecular Sciences 21, no. 4 (2020): 1359. http://dx.doi.org/10.3390/ijms21041359.

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Aging is a major risk factor for cardiovascular diseases (CVDs), the major cause of death worldwide. Cardiac myocytes, which hold the most abundant mitochondrial population, are terminally differentiated cells with diminished regenerative capacity in the adult. Cardiomyocyte mitochondrial dysfunction is a characteristic feature of the aging heart and one out of the nine features of cellular aging. Aging and cardiac pathologies are also associated with increased senescence in the heart. However, the cause and consequences of cardiac senescence during aging or in cardiac pathologies are mostly u
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Hwang, Keun Young, and Young Bong Choi. "Modulation of Mitochondrial Antiviral Signaling by Human Herpesvirus 8 Interferon Regulatory Factor 1." Journal of Virology 90, no. 1 (2015): 506–20. http://dx.doi.org/10.1128/jvi.01903-15.

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ABSTRACTMitochondrial lipid raft-like microdomains, experimentally also termed mitochondrial detergent-resistant membrane fractions (mDRM), play a role as platforms for recruiting signaling molecules involved in antiviral responses such as apoptosis and innate immunity. Viruses can modulate mitochondrial functions for their own survival and replication. However, viral regulation of the antiviral responses via mDRM remains incompletely understood. Here, we report that human herpesvirus 8 (HHV-8) gene product viral interferon regulatory factor 1 (vIRF-1) is targeted to mDRM during virus replicat
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Laban, Hebatullah, Sophia Siegmund, Maren Zappe, et al. "NFAT5/TonEBP Limits Pulmonary Vascular Resistance in the Hypoxic Lung by Controlling Mitochondrial Reactive Oxygen Species Generation in Arterial Smooth Muscle Cells." Cells 10, no. 12 (2021): 3293. http://dx.doi.org/10.3390/cells10123293.

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Chronic hypoxia increases the resistance of pulmonary arteries by stimulating their contraction and augmenting their coverage by smooth muscle cells (SMCs). While these responses require adjustment of the vascular SMC transcriptome, regulatory elements are not well defined in this context. Here, we explored the functional role of the transcription factor nuclear factor of activated T-cells 5 (NFAT5/TonEBP) in the hypoxic lung. Regulatory functions of NFAT5 were investigated in cultured artery SMCs and lungs from control (Nfat5fl/fl) and SMC-specific Nfat5-deficient (Nfat5(SMC)−/−) mice. Exposu
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Toshima, Takahiro, Mikako Yagi, Yura Do та ін. "Mitochondrial translation failure represses cholesterol gene expression via Pyk2-Gsk3β-Srebp2 axis". Life Science Alliance 7, № 7 (2024): e202302423. http://dx.doi.org/10.26508/lsa.202302423.

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Neurodegenerative diseases and other age-related disorders are closely associated with mitochondrial dysfunction. We previously showed that mice with neuron-specific deficiency of mitochondrial translation exhibit leukoencephalopathy because of demyelination. Reduced cholesterol metabolism has been associated with demyelinating diseases of the brain such as Alzheimer’s disease. However, the molecular mechanisms involved and relevance to the pathogenesis remained unknown. In this study, we show that inhibition of mitochondrial translation significantly reduced expression of the cholesterol synt
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8

D'souza, Donna, Ruanne Y. J. Lai, Michael Shuen, and David A. Hood. "mRNA stability as a function of striated muscle oxidative capacity." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 303, no. 4 (2012): R408—R417. http://dx.doi.org/10.1152/ajpregu.00085.2012.

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A change in mRNA stability alters the abundance of mRNA available for translation and is emerging as a critical pathway influencing gene expression. Variations in the stability of functional and regulatory mitochondrial proteins may contribute to the divergent mitochondrial densities observed in striated muscle. Thus we hypothesized that the stability of mRNAs encoding for regulatory nuclear and mitochondrial transcription factors would be inversely proportional to muscle oxidative capacity and would be facilitated by the activity of RNA binding proteins (RBPs). The stability of mitochondrial
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Wolin, Michael S. "Evidence for novel aspects of Nox4 oxidase regulation of mitochondrial function and peroxide generation in an endothelial cell model of senescence." Biochemical Journal 452, no. 2 (2013): e1-e2. http://dx.doi.org/10.1042/bj20130484.

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Observations by Kozieł et al. reported in this issue of the Biochemical Journal suggest the existence of novel regulatory processes associated with new evidence for increased Nox4 (NAPDH oxidase 4) regulation of mitochondrial function in a cultured endothelial cell aging-induced senescence model. Cellular aging appears to promote a Nox4 interaction with mitochondria that disrupts complex I in the electron transport chain and increases the detection of mitochondrial H2O2. Nox4 appears to maintain a highly interconnected mitochondrial network, which may influence mitochondrial fission and/or fus
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Farge, Géraldine, and Maria Falkenberg. "Organization of DNA in Mammalian Mitochondria." International Journal of Molecular Sciences 20, no. 11 (2019): 2770. http://dx.doi.org/10.3390/ijms20112770.

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As with all organisms that must organize and condense their DNA to fit within the limited volume of a cell or a nucleus, mammalian mitochondrial DNA (mtDNA) is packaged into nucleoprotein structures called nucleoids. In this study, we first introduce the general modes of DNA compaction, especially the role of the nucleoid-associated proteins (NAPs) that structure the bacterial chromosome. We then present the mitochondrial nucleoid and the main factors responsible for packaging of mtDNA: ARS- (autonomously replicating sequence-) binding factor 2 protein (Abf2p) in yeast and mitochondrial transc
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Dissertations / Theses on the topic "Mitochondrial Associated Regulatory Factor (Marf)"

1

Prasanna, Katti. "Investigating the Novel Roles of miR-9a and the Regulators of Mitochondrial Dynamics During the Development and Functioning of Indirect Flight Muscles in Drosophila melanogaster." Thesis, 2016. http://etd.iisc.ac.in/handle/2005/4330.

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The muscular system is a highly complex and important system in the body. Proper muscle physiology is critical for locomotion, digestion, circulation, reproduction as well as for metabolic and immune homeostasis. Defects in muscle development, structure or function result in muscle disorders and diseases. Chapter 1 reviews the important events of muscle development and growth as well as the various processes that are involved in the regulation of the same. The muscle disorders that occur due to the mis -regulation of these processes are discussed. Specifically, the significance of microRNAs in
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