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Journal articles on the topic 'Necrotic enteritis'

Author: Grafiati

Published: 4 June 2021

Last updated: 19 February 2022

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1

Wiedosari, Ening, and Y. Sani. "Coccidiosis as A Predisposition Factor for Necrotic Enteritis in Poultry and Their Prevention." Indonesian Bulletin of Animal and Veterinary Sciences 30, no. 3 (September 30, 2020): 139. http://dx.doi.org/10.14334/wartazoa.v30i3.2504.

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<p>Coccidiosis and necrotic enteritis are both affecting the digestive tract of chicken and both are major enteric disease that mainly affect poultry. Among the enteric diseases, necrotic enteritis is a major problem. Coccidiosis is caused by the protozoan of Eimeria sp, and is commonly found as a predisposition factor for necrotic enteritis caused by Clostridum perfringens. Both diseases are commonly shown similar pathological change namely necrosis of epthelial cells in the intestinal mucosa. The diseases have great economic impact in poultry productions, due to the increased mortality

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2

Laishevtsev, A. I., A. V. Kapustin, E. A. Yakimova, A. V. Danilyuk, A. M. Gulyukin, and V. V. Belimenko. "Necrotic enteritis of birds." IOP Conference Series: Earth and Environmental Science 315 (August 23, 2019): 022075. http://dx.doi.org/10.1088/1755-1315/315/2/022075.

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3

Gazdzinski, P., and R. J. Julian. "Necrotic Enteritis in Turkeys." Avian Diseases 36, no. 3 (July 1992): 792. http://dx.doi.org/10.2307/1591787.

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4

GEETHA, M., and K. M. PALANIVEL. "Necrotic enteritis in layer chicken." VETERINARY SCIENCE RESEARCH JOURNAL 8, no. 1 and 2 (October 15, 2017): 73–76. http://dx.doi.org/10.15740/has/vsrj/8.1and2/73-76.

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5

Morris, Winston E., Agustín J. Venzano, Ana Elizondo, Daniel A. Vilte, Elsa C. Mercado, and Mariano E. Fernandez-Miyakawa. "Necrotic Enteritis in Young Calves." Journal of Veterinary Diagnostic Investigation 23, no. 2 (March 2011): 254–59. http://dx.doi.org/10.1177/104063871102300209.

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6

Penny, C., L. Murray, and P. Scott. "Necrotic enteritis in suckled calves." Veterinary Record 128, no. 24 (June 15, 1991): 575. http://dx.doi.org/10.1136/vr.128.24.575-b.

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7

Umar, Sajid, Muhammad Younus, Muhammad Shahzad, Kiran Aqil, Rizwan Qayyum, Aqsa Mushtaq, Muhammad Ali Abdullah Shah, and Muhammad Tanveer Munir. "Role of Wheat Based Diet on the Pathology of Necrotic Enteritis in Turkeys." Scientifica 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/4381067.

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The study was conducted to investigate the effects of wheat based diet on the pathology of necrotic enteritis in turkeys. Turkeys were divided into four groups. Groups A and B were kept as noninoculated and fed normal commercial diet while groups C and D were challenged orally withC. perfringensand fed wheat based diet to promote the development of experimental disease. Infected turkeys showed clinical signs of depression, ruffled feathers, and dark yellowish faeces showing the most prominent disease signs in turkeys of group D with 30% mortality. Similarly, turkeys of group D showed more stri

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8

Zlotowski, Priscila, André Mendes Ribeiro Corrêa, David Emílio Santos Neves de Barcellos, Marisa Ribeiro de Itapema Cardoso, Anílton Cesar Vasconcelos, Luiza Amaral de Castro, and David Driemeier. "Necrotic enterocolitis in pigs naturally infected by porcine circovirus type 2." Ciência Rural 39, no. 6 (June 19, 2009): 1801–7. http://dx.doi.org/10.1590/s0103-84782009005000115.

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Samples of intestine with necrotic enteritis from 63 pigs naturally infected with porcine circovirus type 2 (PCV2) were studied. Colon was the main target of PCV2 associated necrotic enteritis in 60 cases. Immunohistological investigations were carried out to detect the presence of PCV2 in necrotic lesions and to identify the type of cells infected by the virus. Crypt epithelial cells had positive labelling for PCV2 in 17 cases. Depletion of goblet cells occurred in 10 cases. In 24 necrotic enteritis cases, co-infection of PCV2 and Salmonella was identified. An increased rate of apoptosis in t

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9

Cowen, B. S., L. D. Schwartz, R. A. Wilson, and S. I. Ambrus. "Experimentally Induced Necrotic Enteritis in Chickens." Avian Diseases 31, no. 4 (October 1987): 904. http://dx.doi.org/10.2307/1591050.

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10

Droual, R., H. L. Shivaprasad, and R. P. Chin. "Coccidiosis and Necrotic Enteritis in Turkeys." Avian Diseases 38, no. 1 (January 1994): 177. http://dx.doi.org/10.2307/1591854.

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11

Wobeser, G., and D. J. Rainnie. "EPIZOOTIC NECROTIC ENTERITIS IN WILD GEESE." Journal of Wildlife Diseases 23, no. 3 (July 1987): 376–85. http://dx.doi.org/10.7589/0090-3558-23.3.376.

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12

Antonissen, Gunther, Venessa Eeckhaut, Karolien Van Driessche, Lonneke Onrust, Freddy Haesebrouck, Richard Ducatelle, Robert J. Moore, and Filip Van Immerseel. "Microbial shifts associated with necrotic enteritis." Avian Pathology 45, no. 3 (May 3, 2016): 308–12. http://dx.doi.org/10.1080/03079457.2016.1152625.

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13

Shane, S. M., J. E. Gyimah, K. S. Harrington, and T. G. Snider. "Etiology and pathogenesis of necrotic enteritis." Veterinary Research Communications 9, no. 1 (December 1985): 269–87. http://dx.doi.org/10.1007/bf02215151.

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14

Jezdimirovic, Nemanja, Branislav Kureljusic, Vojin Ivetic, Jasna Kureljusic, Dj Cvetojevic, Danka Maslic-Strizak, O. Radanovic, and Milanka Jezdimirovic. "Haemorrhagic-necrotic enteritis in heavy breeds broilers." Veterinarski glasnik 68, no. 3-4 (2014): 265–73. http://dx.doi.org/10.2298/vetgl1404265j.

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The aim of the investigation was to determine the influence of Clostridium perfringens type A on the development of pathomorphological substrate, its intensity and distribution in fifteen weeks old heavy breeds broilers. The investigation was carried out on corpses of 8 hens and 7 roosters of heavy breeds of provenance COBB 500. After the completion of the autopsy, samples of altered parts of jejunum and liver were taken for histopathological examination, and jejunum intestinal contents for bacteriological examination. In all the corpses, in open pleuroperitoneal cavity, even in situ, an alter

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15

Barbara, Angelique J., Hien T. Trinh, Robert D. Glock, and J. Glenn Songer. "Necrotic enteritis-producing strains of Clostridium perfringens displace non-necrotic enteritis strains from the gut of chicks." Veterinary Microbiology 126, no. 4 (January 2008): 377–82. http://dx.doi.org/10.1016/j.vetmic.2007.07.019.

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16

Paiva, Diego, and Audrey McElroy. "Necrotic enteritis: Applications for the poultry industry." Journal of Applied Poultry Research 23, no. 3 (September 2014): 557–66. http://dx.doi.org/10.3382/japr.2013-00925.

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17

Rimoldi, Guillermo, Francisco Uzal, R. P. Chin, Enzo A. Palombo, Milena Awad, Dena Lyras, and H. L. Shivaprasad. "Necrotic Enteritis in Chickens Associated withClostridium sordellii." Avian Diseases 59, no. 3 (September 2015): 447–51. http://dx.doi.org/10.1637/11077-033115-case.1.

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18

Moore, Robert J. "Necrotic enteritis predisposing factors in broiler chickens." Avian Pathology 45, no. 3 (May 3, 2016): 275–81. http://dx.doi.org/10.1080/03079457.2016.1150587.

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19

Dierick, E., O. P. Hirvonen, F. Haesebrouck, R. Ducatelle, F. Van Immerseel, and E. Goossens. "Rapid growth predisposes broilers to necrotic enteritis." Avian Pathology 48, no. 5 (June 9, 2019): 416–22. http://dx.doi.org/10.1080/03079457.2019.1614147.

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20

El-Sergany, Elham, El-Helw Hamed, Hala El-Sawy, Taha Medhat, and Abdalla Yasser. "Preparation of Necrotic Enteritis Vaccine for Turkey." Journal of World's Poultry Research 9, no. 2 (June 25, 2019): 109–16. http://dx.doi.org/10.36380/jwpr.2019.13.

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21

Keyburn, Anthony L, Scott A. Sheedy, Mark E. Ford, Mark M. Williamson, Milena M. Awad, Julian I. Rood, and Robert J. Moore. "Alpha-Toxin of Clostridium perfringens Is Not an Essential Virulence Factor in Necrotic Enteritis in Chickens." Infection and Immunity 74, no. 11 (August 21, 2006): 6496–500. http://dx.doi.org/10.1128/iai.00806-06.

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ABSTRACT The Clostridium perfringens alpha-toxin has previously been implicated as the major virulence factor in necrotic enteritis in chickens, although definitive proof has not been reported. In this study an alpha-toxin mutant was constructed in a virulent chicken isolate and shown to retain full virulence in a chicken disease model. These results demonstrated that alpha-toxin is not an essential virulence factor in the pathogenesis of necrotic enteritis in chickens.

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22

Abubakar, A., H. B. Aliyu, L. Sa idu, and S. G. Usman. "Necrotic enteritis and its management in 13-week old commercial pullets in Katsina, Nigeria." Sokoto Journal of Veterinary Sciences 18, no. 2 (September 9, 2020): 104–7. http://dx.doi.org/10.4314/sokjvs.v18i2.7.

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Necrotic enteritis is rarely reported because it is often misdiagnosed as coccidiosis due to similarity in clinical and pathological features. A field outbreak of necrotic enteritis in a flock of 13 weeks old 4,500 commercial pullets was investigated, the onset of the disease, morbidity and mortality rates were recorded. Post mortem examinations were conducted and gross lesions were documented. Tissues were collected and fixed in 10 % neutral buffered formalin and processed for histopathological examinations. Clinical signs observed were ruffled feathers, weakness, somnolence, loss of weight a

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23

Matsushita, S., and T. Matsumoto. "Spontaneous necrotic enteritis in young RFM/Ms mice." Laboratory Animals 20, no. 2 (April 1, 1986): 114–17. http://dx.doi.org/10.1258/002367786780865124.

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Fatal necrotic enteritis was observed in mice 24-52 days old in the RFM/Ms breeding colony maintained in a clean conventional condition in the National Institute of Radiological Sciences. Gross lesions included hyperaemia, petechiae, erosion and the occasional formation of pseudomembranes in the mucosa of the ileum and caecum. Histologically, there was necrotic enteritis with numerous Gram-positive bacilli-forming spores but no inflammatory cell reaction. Non-type-A Clostridium perfringens was isolated from the intestinal contents. This disease cleared after the addition of chlortetracycline h

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24

Keerqin, C., N. K. Morgan, S. B. Wu, B. Svihus, and M. Choct. "Reintroduction of microflora from necrotic enteritis-resistant chickens reduces gross lesions and improves performance of necrotic enteritis-challenged broilers." Journal of Applied Poultry Research 26, no. 3 (September 2017): 449–57. http://dx.doi.org/10.3382/japr/pfx015.

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25

Asaduzzaman, M., MS Miah, A. Siddika, N. Popy, and MM Hossain. "EXPERIMENTAL PRODUCTION OF NECROTIC ENTERITIS IN BROILER CHICKENS." Bangladesh Journal of Veterinary Medicine 9, no. 1 (July 12, 2012): 33–41. http://dx.doi.org/10.3329/bjvm.v9i1.11207.

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The present study was conducted to isolate and identify the Clostridium perfringens, the etiology of necrotic enteritis (NE) from broiler chickens and experimental production of NE with this isolate. A total of 50 samples were collected from jejunum of necropsied birds for isolation and identification of Cl. perfringens. Out of 50 samples, only 4 isolates of Cl. perfringens were isolated and identified (prevalence 8%). In experimental NE, the birds of group A (orally administered with only 0.1 ml (1x103 sporulated Eimeria acervulina oocysts/bird) showed dullness, ruffled feather, vent feather

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26

Aoyama, Hiroki, Norihiro Yuasa, Eiji Takeuchi, Yasutomo Goto, Hideo Miyake, Hidemasa Nagai, Masaoki Hattori, Kanji Miyata, and Masahiko Fujino. "Repeated Necrotic Ischemic Enteritis with Portal Venous Gas." Japanese Journal of Gastroenterological Surgery 48, no. 1 (2015): 38–45. http://dx.doi.org/10.5833/jjgs.2014.0071.

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27

Broom, L. J. "Necrotic enteritis; current knowledge and diet-related mitigation." World's Poultry Science Journal 73, no. 2 (June 1, 2017): 281–92. http://dx.doi.org/10.1017/s0043933917000058.

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28

Simpson, V., M. Cranwell, C. Johnson, D. Dyson, and J. Gibbens. "Necrotic enteritis of unknown aetiology in suckler calves." Veterinary Record 134, no. 18 (April 30, 1994): 479. http://dx.doi.org/10.1136/vr.134.18.479.

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29

Lovland, Atle, Magne Kaldhusdal, Keith Redhead, Eystein Skjerve, and Atle Lillehaug. "Maternal vaccination against subclinical necrotic enteritis in broilers." Avian Pathology 33, no. 1 (February 2004): 81–90. http://dx.doi.org/10.1080/0379450310001636255.

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30

Knap, I., B. Lund, A. B. Kehlet, C. Hofacre, and G. Mathis. "Bacillus licheniformis Prevents Necrotic Enteritis in Broiler Chickens." Avian Diseases 54, no. 2 (June 2010): 931–35. http://dx.doi.org/10.1637/9106-101509-resnote.1.

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31

Knap, I., B. Lund, A. B. Kehlet, C. Hofacre, and G. Mathis. "Bacillus licheniformis Prevents Necrotic Enteritis in Broiler Chickens." Avian Diseases Digest 5, no. 2 (June 2010): e45-e45. http://dx.doi.org/10.1637/9355-910610-digest.1.

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32

Broussard, C. T., C. L. Hofacre, R. K. Page, and O. J. Fletcher. "Necrotic Enteritis in Cage-Reared Commercial Layer Pullets." Avian Diseases 30, no. 3 (July 1986): 617. http://dx.doi.org/10.2307/1590433.

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33

Rood, Julian I., Anthony L. Keyburn, and Robert J. Moore. "NetB and necrotic enteritis: the hole movable story." Avian Pathology 45, no. 3 (May 3, 2016): 295–301. http://dx.doi.org/10.1080/03079457.2016.1158781.

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34

Milanov, Dubravka, Nevenka Aleksić, Milica Živkov Baloš, Marko Pajić, and Slobodan Knežević. "CURRENTLY UNKNOWN ASPECTS OF POULTRY NECROTIC ENTERITIS PATHOGENESIS." Archives of Veterinary Medicine 12, no. 1 (September 12, 2019): 55–69. http://dx.doi.org/10.46784/e-avm.v12i1.38.

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Necrotic enteritis (NE) or poultry clostridiosis is a disease which poses enormous health problems and makes tremendous economic losses to intensive poultry production worldwide. Despite having been targeted in extensive research for decades, a number of aspects of its pathogenesis remain unknown. For more than 30 years alfa-toxin has been considered to be the main virulence factor of the causative agent, but experimental research using a mutant Clostridium perfringens strain lacking the gene coding for this confirmed that alpha-toxin is not necessary for pathogenesis. Since the 1980s, NetB to

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35

Bansal, Mohit, Tahrir Alenezi, Ying Fu, Ayidh Almansour, Hong Wang, Anamika Gupta, Rohana Liyanage, Danielle B. Graham, Billy M. Hargis, and Xiaolun Sun. "Specific Secondary Bile Acids Control Chicken Necrotic Enteritis." Pathogens 10, no. 8 (August 17, 2021): 1041. http://dx.doi.org/10.3390/pathogens10081041.

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Necrotic enteritis (NE), mainly induced by the pathogens of Clostridium perfringens and coccidia, causes huge economic losses with limited intervention options in the poultry industry. This study investigated the role of specific bile acids on NE development. Day-old broiler chicks were assigned to six groups: noninfected, NE, and NE with four bile diets of 0.32% chicken bile, 0.15% commercial ox bile, 0.15% lithocholic acid (LCA), or 0.15% deoxycholic acid (DCA). The birds were infected with Eimeria maxima at day 18 and C. perfringens at day 23 and 24. The infected birds developed clinical NE

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36

Cravens, R. L., G. R. Goss, F. Chi, E. D. De Boer, S. W. Davis, S. M. Hendrix, J. A. Richardson, and S. L. Johnston. "The effects of necrotic enteritis, aflatoxin B1, and virginiamycin on growth performance, necrotic enteritis lesion scores, and mortality in young broilers." Poultry Science 92, no. 8 (August 2013): 1997–2004. http://dx.doi.org/10.3382/ps.2013-03011.

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37

Yang, Qing, Melanie A. Whitmore, Kelsy Robinson, Wentao Lyu, and Guolong Zhang. "Butyrate, Forskolin, and Lactose Synergistically Enhance Disease Resistance by Inducing the Expression of the Genes Involved in Innate Host Defense and Barrier Function." Antibiotics 10, no. 10 (September 27, 2021): 1175. http://dx.doi.org/10.3390/antibiotics10101175.

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The rising concern of antimicrobial resistance highlights a need for effective alternatives to antibiotics for livestock production. Butyrate, forskolin, and lactose are three natural products known to induce the synthesis of host defense peptides (HDP), which are a critical component of innate immunity. In this study, the synergy among butyrate, forskolin, and lactose in enhancing innate host defense, barrier function, and resistance to necrotic enteritis and coccidiosis was investigated. Our results indicated that the three compounds synergistically augmented the expressions of multiple HDP

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38

., Simon M. Shane. "Re-Emergence of Necrotic Enteritis in the Broiler Industry." International Journal of Poultry Science 4, no. 9 (August 15, 2005): 604–11. http://dx.doi.org/10.3923/ijps.2005.604.611.

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39

Hermans, P. G., and K. L. Morgan. "57. The epidemiology of necrotic enteritis in broiler chickens." Research in Veterinary Science 74 (2003): 19. http://dx.doi.org/10.1016/s0034-5288(03)90056-7.

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40

shanmugasamy, Malmarugan, Boobalan Adhimoolam, and Dorairajan Natarajan. "Necrotic Enteritis in Broiler and Layer Farms in Tamilnadu." International Journal for Agro Veterinary and Medical Sciences 6, no. 4 (2012): 241. http://dx.doi.org/10.5455/ijavms.126.

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41

Kurkure, N. V., S. B. Kale, R. T. Kale, S. P. Awandkar, and N. N. Joat. "Occurrence of necrotic enteritis in a commercial layer flock." Indian Journal of Veterinary Pathology 41, no. 1 (2017): 50. http://dx.doi.org/10.5958/0973-970x.2017.00011.6.

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42

Behera, Debasish, D. C. Pathak, R. K. Sharma, T. N. Upadhyaya, S. Goswami, and S. Tamuly. "Pathology and molecular diagnosis of necrotic enteritis in chicken." Indian Journal of Veterinary Pathology 44, no. 1 (2020): 22. http://dx.doi.org/10.5958/0973-970x.2020.00005.x.

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43

Mcdevitt, R. M., J. D. Brooker, T. Acamovic, and N. H. C. Sparks. "Necrotic enteritis; a continuing challenge for the poultry industry." World's Poultry Science Journal 62, no. 2 (June 1, 2006): 221–47. http://dx.doi.org/10.1079/wps200593.

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44

Asaoka, Yoshiji, Tokuma Yanai, Haruko Hirayama, Yumi Une, Eriko Saito, Hiroki Sakai, Masanobu Goryo, Hideto Fukushi, and Toshiaki Masegi. "Fatal necrotic enteritis associated withClostridium perfringensin wild crows (Corvusmacrorhynchos)." Avian Pathology 33, no. 1 (February 2004): 19–24. http://dx.doi.org/10.1080/03079450310001636228.

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45

Tsiouris, V., I. Georgopoulou, Chr Batzios, N. Pappaioannou, R. Ducatelle, and P. Fortomaris. "Temporary feed restriction partially protects broilers from necrotic enteritis." Avian Pathology 43, no. 2 (February 28, 2014): 139–45. http://dx.doi.org/10.1080/03079457.2014.889278.

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46

Prescott, John F., Joan A. Smyth, Bahram Shojadoost, and Andrew Vince. "Experimental reproduction of necrotic enteritis in chickens: a review." Avian Pathology 45, no. 3 (May 3, 2016): 317–22. http://dx.doi.org/10.1080/03079457.2016.1141345.

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47

Marshall, R. N., R. B. Williams, R. M. La Ragione, J. A. Marshall, and J. Catchpole. "102. Experimental production of necrotic enteritis and its use for studies on the relationships between necrotic enteritis, coccidiosis and anticoccidial vaccination of chickens." Research in Veterinary Science 74 (2003): 34. http://dx.doi.org/10.1016/s0034-5288(03)90101-9.

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48

Wade, Ben, Anthony L. Keyburn, Volker Haring, Mark Ford, Julian I. Rood, and Robert J. Moore. "Two putative zinc metalloproteases contribute to the virulence of Clostridium perfringens strains that cause avian necrotic enteritis." Journal of Veterinary Diagnostic Investigation 32, no. 2 (January 10, 2020): 259–67. http://dx.doi.org/10.1177/1040638719898689.

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Two putative zinc metalloproteases encoded by Clostridium perfringens have been implicated in the pathogenesis of necrotic enteritis, an economically significant poultry disease that is caused by this anaerobic bacterium. These proteases have ~64% amino acid identity and are encoded by the zmpA and zmpB genes. We screened 83 C. perfringens isolates by PCR for the presence of these genes. The first gene, zmpB, is chromosomally located and was present in all screened strains of C. perfringens, regardless of their origin and virulence. The second gene, zmpA, is plasmid-borne and was only found in

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49

Das, Arunava, Yahya Mazumder, Biman Kumar Dutta, Bibek Ranjan Shome, Komal Molla Bujarbarua, and Ashok Kumar. "Clostridium perfringens Type A from Broiler Chicken with Necrotic Enteritis." International Journal of Poultry Science 7, no. 6 (May 15, 2008): 601–9. http://dx.doi.org/10.3923/ijps.2008.601.609.

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50

Kulkarni, R. R., V. R. Parreira, S. Sharif, and J. F. Prescott. "Immunization of Broiler Chickens against Clostridium perfringens-Induced Necrotic Enteritis." Clinical and Vaccine Immunology 14, no. 9 (July 18, 2007): 1070–77. http://dx.doi.org/10.1128/cvi.00162-07.

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ABSTRACT Necrotic enteritis (NE) in broiler chickens is caused by Clostridium perfringens. Currently, no vaccine against NE is available and immunity to NE is not well characterized. Our previous studies showed that immunity to NE followed oral infection by virulent rather than avirulent C. perfringens strains and identified immunogenic secreted proteins apparently uniquely produced by virulent C. perfringens isolates. These proteins were alpha-toxin, glyceraldehyde-3-phosphate dehydrogenase, pyruvate:ferredoxin oxidoreductase (PFOR), fructose 1,6-biphosphate aldolase, and a hypothetical prote

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