Relevant bibliographies by topics / Neural mechanisms autism

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Academic literature on the topic 'Neural mechanisms autism'

Author: Grafiati
Published: 11 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "Neural mechanisms autism"

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Watts, Timothy John. "The Pathogenesis of Autism." Clinical medicine. Pathology 1 (January 2008): CPath.S1143. http://dx.doi.org/10.4137/cpath.s1143.

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Autism is well known as a complex developmental disorder with a seemingly confusing and uncertain pathogenesis. The definitive mechanisms that promote autism are poorly understood and mostly unknown, yet available theories do appear to focus on the disruption of normal cerebral development and its subsequent implications on the functional brain unit. This mini-review aims solely to discuss and evaluate the most prominent current theories regarding the pathogenesis of autism. The main conclusion is that although there is not a clear pathway of mechanisms directed towards a simple pathogenesis a
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Bertone, Armando, Laurent Mottron, Patricia Jelenic, and Jocelyn Faubert. "Motion Perception in Autism: A “Complex” Issue." Journal of Cognitive Neuroscience 15, no. 2 (2003): 218–25. http://dx.doi.org/10.1162/089892903321208150.

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We present the first assessment of motion sensitivity for persons with autism and normal intelligence using motion patterns that require neural processing mechanisms of varying complexity. Compared to matched controls, our results demonstrate that the motion sensitivity of observers with autism is similar to that of nonautistic observers for different types of first-order (luminance-defined) motion stimuli, but significantly decreased for the same types of second-order (texture-defined) stimuli. The latter class of motion stimuli has been demonstrated to require additional neural computation t
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Failla, Michelle D., Estephan J. Moana-Filho, Greg K. Essick, Grace T. Baranek, Baxter P. Rogers, and Carissa J. Cascio. "Initially intact neural responses to pain in autism are diminished during sustained pain." Autism 22, no. 6 (2017): 669–83. http://dx.doi.org/10.1177/1362361317696043.

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Pain assessments typically depend on self-report of the pain experience. Yet, in individuals with autism spectrum disorders, this can be an unreliable due to communication difficulties. Importantly, observations of behavioral hypo- and hyperresponsivity to pain suggest altered pain sensitivity in autism spectrum disorder. Neuroimaging may provide insight into mechanisms underlying pain behaviors. The neural pain signature reliably responds to painful stimulation and is modulated by other outside regions, affecting the pain experience. In this first functional magnetic resonance imaging study o
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Mosner, Maya G., R. Edward McLaurin, Jessica L. Kinard, et al. "Neural Mechanisms of Reward Prediction Error in Autism Spectrum Disorder." Autism Research and Treatment 2019 (July 1, 2019): 1–10. http://dx.doi.org/10.1155/2019/5469191.

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Few studies have explored neural mechanisms of reward learning in ASD despite evidence of behavioral impairments of predictive abilities in ASD. To investigate the neural correlates of reward prediction errors in ASD, 16 adults with ASD and 14 typically developing controls performed a prediction error task during fMRI scanning. Results revealed greater activation in the ASD group in the left paracingulate gyrus during signed prediction errors and the left insula and right frontal pole during thresholded unsigned prediction errors. Findings support atypical neural processing of reward predictio
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Richey, J. Anthony, Cara R. Damiano, Antoinette Sabatino, et al. "Neural Mechanisms of Emotion Regulation in Autism Spectrum Disorder." Journal of Autism and Developmental Disorders 45, no. 11 (2015): 3409–23. http://dx.doi.org/10.1007/s10803-015-2359-z.

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Mazefsky, Carla A., Amanda Collier, Josh Golt, and Greg J. Siegle. "Neural features of sustained emotional information processing in autism spectrum disorder." Autism 24, no. 4 (2020): 941–53. http://dx.doi.org/10.1177/1362361320903137.

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Emotion dysregulation is common in autism spectrum disorder; a better understanding of the underlying neural mechanisms could inform treatment development. The tendency toward repetitive cognition in autism spectrum disorder may also increase susceptibility to perseverate on distressing stimuli, which may then increase emotion dysregulation. Therefore, this study investigated the mechanisms of sustained processing of negative information in brain activity using functional magnetic resonance imaging. We used an event-related task that alternated between emotional processing of personally releva
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Luo, Emanuel, C. Pan, and Xiujun Fan. "Music, Language, and Autism: Neurological Insights for Enhanced Learning." International Journal of Innovative Research in Medical Science 8, no. 09 (2023): 398–408. http://dx.doi.org/10.23958/ijirms/vol08-i09/1743.

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This comprehensive review thoroughly explores the intricate relationship between music and language, encompassing their historical, cognitive, and neural dimensions. It draws evidence from ancient Chinese civilizations dating back to 4500 BC to analyze the coexistence and parallel evolution of music and language for the first time. Comparative studies illuminate the shared and distinctive aspects of pitch, rhythm, and syntax inherent to music and language. The examination extends to the diverse impact of music, including second language acquisition, phonological awareness, pitch processing, me
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Colizzi, Marco, Riccardo Bortoletto, Rosalia Costa, Sagnik Bhattacharyya, and Matteo Balestrieri. "The Autism–Psychosis Continuum Conundrum: Exploring the Role of the Endocannabinoid System." International Journal of Environmental Research and Public Health 19, no. 9 (2022): 5616. http://dx.doi.org/10.3390/ijerph19095616.

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Evidence indicates shared physiopathological mechanisms between autism and psychosis. In this regard, the endocannabinoid system has been suggested to modulate neural circuits during the early stage of neurodevelopment, with implications for both autism and psychosis. Nevertheless, such potential common markers of disease have been investigated in both autism and psychosis spectrum disorders, without considering the conundrum of differentiating the two groups of conditions in terms of diagnosis and treatment. Here, we systematically review all human and animal studies examining the endocannabi
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Schmitz, Nicole, Katya Rubia, Therese van Amelsvoort, Eileen Daly, Anna Smith, and Declan G. M. Murphy. "Neural correlates of reward in autism." British Journal of Psychiatry 192, no. 1 (2008): 19–24. http://dx.doi.org/10.1192/bjp.bp.107.036921.

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BackgroundLack of social interaction, which is characteristically seen in people with autistic-spectrum disorder, may be caused by malfunctioning of the frontostriatal reward systems. However, no reported in vivo brain imaging studies have investigated reward mechanisms in autistic-spectrum disorder.AimsTo investigate functional brain activation during reward feedback in people with autistic-spectrum disorder and control individuals.MethodWe used event-related functional magnetic resonance imaging to examine the neural substrates of monetary reward in individuals with autistic-spectrum disorde
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Larson, Charles R. "2008 Zemlin Award in Speech Sciences Memorial Lecture: The Role of Auditory Feedback for the Control of Voice Fundamental Frequency and Amplitude." Perspectives on Speech Science and Orofacial Disorders 19, no. 1 (2009): 6–17. http://dx.doi.org/10.1044/ssod19.1.6.

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Abstract Previous research has failed to identify precise neural mechanisms involved in auditory feedback regulation of vocalization. The goal of this research project was to improve our understanding of neural mechanisms controlling the voice. Participants were instructed to sustain a vowel or repeat phrases during which perturbations in voice pitch or loudness feedback were presented. Voice signal averaging, neuroimaging, laryngeal electromyography, and cortical event-related potential techniques were used to measure vocal and neural responses to perturbed feedback. Pitch- and loudness-shift
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Dissertations / Theses on the topic "Neural mechanisms autism"

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Lefevre, Arthur. "Neural mechanisms of oxytocin and serotonin interaction in non-human primates and patients with autism." Thesis, Lyon, 2016. http://www.theses.fr/2016LYSE1323/document.

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La neurohormone ocytocine (OT) est de plus en plus étudiée pour son potentiel thérapeutique dans les troubles du comportement social, comme l'autisme, qui sont associés à une dérégulation de plusieurs systèmes de neurotransmission, notamment l'OT et la sérotonine (5-HT). Dans ce cadre, une étape importante afin de développer des médicaments basés sur des mécanismes biologiques est de caractériser les interactions entre l'OT et les autres neurotransmetteurs. La littérature sur les rongeurs montre que la relation entre OT et 5-HT est fortement impliquée dans plusieurs aspects du comportement soc
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Lombardo, Michael. "Cognitive and neural mechanisms underlying self-referential and social cognition in autism and the general population." Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608982.

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Coffman, Marika Cerie. "Common and Distinct Neural Mechanisms of Fear Acquisition and Reversal in comorbid Autism with Social Anxiety and Social Anxiety Disorder uncomplicated by Autism." Diss., Virginia Tech, 2019. http://hdl.handle.net/10919/102409.

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Social Anxiety (SAD) increases in prevalence as children enter adolescence. Adolescents with Autism Spectrum Disorder (ASD) are diagnosed with comorbid SAD at higher rates than these individuals are diagnosed with other clinical disorders, including depression and other anxiety disorders. However, there is little research on whether the presentation and neural underpinning of comorbid SAD within the context of ASD is the same as SAD alone. Individual and diagnostic differences exist in neural and biological mechanisms of fear conditioning. Characterization of whether neural mechanisms of fear
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Peng, Yun, Zhongming Lu, Guohui Li, et al. "The autism associated MET receptor tyrosine kinase engages early neuronal growth mechanism and controls glutamatergic circuits development in the forebrain." NATURE PUBLISHING GROUP, 2016. http://hdl.handle.net/10150/617181.

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The human MET gene imparts a replicated risk for autism spectrum disorder (ASD), and is implicated in the structural and functional integrity of brain. MET encodes a receptor tyrosine kinase, MET, which plays a pleiotropic role in embryogenesis and modifies a large number of neurodevelopmental events. Very little is known, however, on how MET signaling engages distinct cellular events to collectively affect brain development in ASD-relevant disease domains. Here, we show that MET protein expression is dynamically regulated and compartmentalized in developing neurons. MET is heavily expres
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VINCO, Erika. "The neural mechanisms and hemispheric differences supporting learning new facial identities." Doctoral thesis, 2015. http://hdl.handle.net/11562/928534.

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Numerosi studi hanno mostrato una specializzazione emisferica a favore dell'emisfero destro per determinati compiti cognitivi e a favore dell'emisfero sinistro per altri. I volti sono lateralizzati a destra; mentre il materiale verbale a sinistra. Le ipotesi di ricerca consistono nell'indagare se tale specializzazione emisferica è assoluta o relativa. Il primo esperimento consiste in un compito di detezione semplice che compara i tempi di risposta (TR) con la mano destra e la mano sinistra a stimoli facciali e digrammi presentati al centro dello schermo di un computer. Lo scopo dell'esperim
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Liao, Chuan-Ching, and 廖專晶. "Different Neural Mechanisms of Semantic Processing Among Youths with Autism Spectrum Disorder, their Unaffected Siblings and Typically Developing Youths." Thesis, 2019. http://ndltd.ncl.edu.tw/handle/6upj59.

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碩士<br>國立臺灣大學<br>心理學研究所<br>107<br>Autism spectrum disorder (ASD) is characterized as a highly heritable disorder, and aberrant semantic processing is one of common symptoms in individuals with ASD. Unaffected ASD siblings usually have similar genetic backgrounds and early-life environments with individuals with ASD, with a higher risk of developing the same disorder. However, little is known about genetic contributions to neural mechanisms of semantic processing in unaffected ASD siblings. Therefore, this study aimed to adopt an endophenotype approach to investigate the differences in underlyin
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Books on the topic "Neural mechanisms autism"

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Westberg, Lars, and Hasse Walum. Oxytocin and Vasopressin Gene Variation and the Neural Basis of Social Behaviors. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.011.

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Experimental studies in rodents and humans show that the neuropeptides oxytocin and vasopressin are important regulators of behaviors related to social interactions. Evidence for positive effects of oxytocin treatment on symptoms of psychiatric disorders characterized by impaired social functioning has emerged. Numerous studies report associations between various social behaviors, the risk of autism, and polymorphisms inOXTRandAVPR1A. This chapter provides an overview of these genetic association studies. Although many of the published findings are inconclusive and need replication in independ
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Patisaul, Heather B., and Scott M. Belcher. Endocrine Disruptors and Neurobehavioral Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780199935734.003.0006.

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This chapter focuses on the role environmental pollutants are playing in the rapidly rising rate of neurodevelopmental disorders in children. The available EDC data are summarized and analyzed in relation to whether or not evidence supports a role for EDCs as contributing to neural disorders. The distinction between endocrine disruption and neurotoxicity is established by focusing on the differences between toxicants, toxins, and altered endocrine/neuroendocrine effects in organizational alterations of the brain. Evidence from experimental systems demonstrating effects of EDCs on the developin
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Beauchaine, Theodore P., and Sheila E. Crowell, eds. The Oxford Handbook of Emotion Dysregulation. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190689285.001.0001.

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Emotion dysregulation—which is often defined as the inability to modulate strong affective states including impulsivity, anger, fear, sadness, and anxiety—is observed in nearly all psychiatric disorders. These include internalizing disorders such as panic disorder and major depression, externalizing disorders such as conduct disorder and antisocial personality disorder, and various other disorders including schizophrenia, autism, and borderline personality disorder. Among many affected individuals, precursors to emotion dysregulation appear early in development, and often predate the emergence
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Gaitanis, John, Phillip L. Pearl, and Howard Goodkin. The EEG in Degenerative Disorders of the Central Nervous System. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0013.

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Nervous system alterations can occur at any stage of prenatal or postnatal development. Any of these derangements, whether environmental or genetic, will affect electrical transmission, causing electroencephalogram (EEG) alteration and possibly epilepsy. Genetic insults may be multisystemic (for example, neurocutaneous syndromes) or affect only the brain. Gene mutations account for inborn errors of metabolism, channelopathies, brain malformations, and impaired synaptogenesis. Inborn errors of metabolism cause seizures and EEG abnormalities through a variety of mechanisms, including disrupted e
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Friston, Karl J., and Raymond J. Dolan. Computational Psychiatry and the Bayesian Brain. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0072.

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This chapter considers recent advances in computational neuroscience that are especially relevant for psychiatry. We offer a review of computational psychiatry in terms of its ambitions, emerging domains of application, and promises for the future. Our focus is on theoretical formulations of brain function that accommodate subjective beliefs and behavior within formal (computational) frameworks—frameworks that can be grounded in neurophysiology down to the level of synaptic mechanisms. Understanding the nature and principles that underlie functional brain architectures is, we assume, essential
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Book chapters on the topic "Neural mechanisms autism"

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Anagnostou, Evdokia, Deepali Mankad, Joshua Diehl, et al. "Neural Mechanisms in Autism." In Encyclopedia of Autism Spectrum Disorders. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_572.

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Casanova, Manuel. "Neural Mechanisms in Autism." In Encyclopedia of Autism Spectrum Disorders. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_572.

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Ibrahim, Karim, Gregory McCarthy, and Denis G. Sukhodolsky. "Neural Mechanisms of Emotional Dysregulation." In Encyclopedia of Autism Spectrum Disorders. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4614-6435-8_102453-1.

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Ibrahim, Karim, Gregory McCarthy, and Denis G. Sukhodolsky. "Neural Mechanisms of Emotional Dysregulation." In Encyclopedia of Autism Spectrum Disorders. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_102453.

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Folstein, Susan E., Michael Dowd, Raymond Mankoski, and Ovsanna Tadevosyan. "How Might Genetic Mechanisms Operate in Autism?" In Autism: Neural Basis and Treatment Possibilities. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470869380.ch5.

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Arakawa, Hiroyuki, and Yuki Higuchi. "The Usability of Mouse Models to Study the Neural Circuity in Autism Spectrum Disorder: Regulatory Mechanisms of Core Behavioral Symptoms." In Neurobiology of Autism Spectrum Disorders. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-42383-3_6.

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Maltsev, Dmytro. "Efficacy of infliximab in autism spectrum disorders in children associated with genetic deficiency of the folate cycle." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch11.

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The notion of systemic inflammation in autism spectrum disorders in children has been established. A recent meta-analysis of randomized controlled trials published in 2019, which included a systematic review of 25 case-control studies, suggests an association between genetic deficiency of the folate cycle and autism spectrum disorders in children. This evidence is consistent with an earlier meta-analysis of randomized controlled trials from 2013, which included data from 8 studies. The encephalopathy that develops in children with genetic deficiency of the folate cycle and manifests as autism
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Maltsev, Dmytro. "Results of the assessment of immune status in children with ASD: immunodeficiency associated with genetic deficiency of the folate cycle." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch3.

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The results of the last 5 meta-analyses of randomized controlled clinical trials indicate an association between genetically determined deficiency of folate cycle enzyme (GDFC) and autism spectrum disorders (ASD) in children, however, the mechanism of this relationship, as well as the association with additional clinical manifestations of a broad disease phenotype, has not yet been fully explained. Since there are frequent reports of the association of ASD with immunodeficiency diseases and signs of immune dysregulation, and immune-dependent mechanisms of CNS damage in children with ASD have b
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Nakai, Nobuhiro, Eric T. N. Overton, and Toru Takumi. "Optogenetic Approaches to Understand the Neural Circuit Mechanism of Social Deficits Seen in Autism Spectrum Disorders." In Advances in Experimental Medicine and Biology. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-8763-4_36.

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Maltsev, Dmytro. "Efficacy of rituximab in autism spectrum disorders associated with genetic deficiency of the folate cycle with signs of antineuronal autoimmunity." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch12.

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Advances in genetics, molecular biology, and immunology over the past decades have significantly changed our understanding of the etiology and pathogenesis of autism spectrum disorders (ASD) in children. One of the key advances in this direction is the elucidation of the association of genetic deficiency of the folate cycle (GDFC) with ASD, evidence for which is based on the results of at least 5 meta-analyses of randomized controlled clinical trials and a number of additional controlled trials, the data of which have not yet been properly summarized. It has been established that GDFC leads to
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Conference papers on the topic "Neural mechanisms autism"

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Nikolaeva, Elena I. "Genetics and psychophysiology of ADHD and autism." In 2nd International Neuropsychological Summer School named after A. R. Luria “The World After the Pandemic: Challenges and Prospects for Neuroscience”. Ural University Press, 2020. http://dx.doi.org/10.15826/b978-5-7996-3073-7.12.

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The paper discusses the brain mechanisms of autism and attention deficit hyperactivity disorder. It is shown that these disorders are associated with different genetic causes that create certain psychophysiological mechanisms. Nevertheless, their diagnosis is interrelated. Moreover, a child is often first diagnosed with ADHD, and then the diagnosis is changed to “autism spectrum disease”. Among the most common causes of the disease is the behavior of retrotransposons. Retrotransposons (also called transposons via intermediate RNA) are genetic elements that can amplify themselves in the genome.
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Pszeida, Martin, Yannick Lieb, Michael Schneeberger, et al. "Learning Analytics Using Eye Tracking-based Biomarkers on Serious Games for Adults with Autism Spectrum Disorder." In 16th International Conference on Applied Human Factors and Ergonomics (AHFE 2025). AHFE International, 2025. https://doi.org/10.54941/ahfe1006091.

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Social interaction deficits are a core feature of Autism Spectrum Disorder (ASD), often rooted in atypical attentional processing of socially relevant information. A research protocol is proposed to investigate facial emotion processing and attentional switching in ASD to better understand mechanisms of social dysfunction and inform sensor-based learning analytics in serious games. A sample of individuals with ASD as well as neuro-typical (NT) controls will complete a standardized psychological test battery and two computer-based eye-tracking tasks using advanced eye tracking technology as wel
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