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Relevant bibliographies by topics / Neurotherapeutic effect

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Academic literature on the topic 'Neurotherapeutic effect'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Neurotherapeutic effect"

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Binjhade, Neha, Vinanti Supare, Shailesh Ghaywat, Sagar Trivedi, Kamlesh Wadher, and Milind Umekar. "Agmatine: A potential Neurotherapeutic Agent." Journal of Drug Delivery and Therapeutics 11, no. 4 (2021): 88–92. http://dx.doi.org/10.22270/jddt.v11i4.4855.

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Agmatine, a natural polyamine disregarded almost for over 100 years, was discovered in year 1910. Almost after a decade, several researches on Agmatine indicated its modulatory action at multiple molecular targets such as, nitric oxide synthesis, neurotransmitter systems, and polyamine metabolism unbolt the new avenues for extensive therapeutic applications which includes neurotrauma and neurodegenerative diseases, antidepressant, cognitive disorders. Agmatine exerts its varied biological characteristics and therapeutic potential in diverse arena. Agmatine has been extensively researched for i

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2

Abeer, A. A. Salama, and M. M. Ibrahim Bassant. "Neurotherapeutic effect of allopurinol against brain injury in hyperlipidemic rats." African Journal of Pharmacy and Pharmacology 9, no. 22 (2015): 567–75. http://dx.doi.org/10.5897/ajpp2014.4247.

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3

Lv, Xiu-li, Yi Wang, Bing Hu, Le Kang, Jun Xin, and Guo-Ying Zhang. "Neurotherapeutic effect of mangiferin against hypoxic–ischemic encephalopathy in neonatal rats." African Journal of Traditional, Complementary and Alternative Medicines 13, no. 2 (2016): 229. http://dx.doi.org/10.4314/ajtcam.v13i2.27.

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Lv, Xiu-li, Yi Wang, Bing Hu, Le Kang, Jun Xin, and Guo-Ying Zhang. "NEUROTHERAPEUTIC EFFECT OF MANGIFERIN AGAINST HYPOXIC–ISCHEMIC ENCEPHALOPATHY IN NEONATAL RATS." African Journal of Traditional, Complementary and Alternative Medicines 13, no. 2 (2016): 229–36. http://dx.doi.org/10.21010/ajtcam.v13i2.27.

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Background: Hypoxic–ischemic encephalopathy (HIE) in perinatal condition is highly associated with mortality and several neurological disabilities. The present experiment was blueprinted to ascertain the protective efficacy of mangiferin (MF) against hypoxic–ischemic brain injury in neonatal rats.  Materials and methods: Fourth six neonatal rats (pubs) were arbitrarily separated into four groups as a sham group that received only water (control; n=12), pubs exposed to Hypoxic–ischemic (HI) insult (HI group-II; n=11), pubs receiving 20 (MF 20+HI-III; n=11) or 40 (MF 40+HI-IV; n=12) mg/kg

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Zeng, Xiang, Alexander E. Ropper, Zaid Aljuboori, et al. "Concurrent Oncolysis and Neurolesion Repair by Dual Gene-Engineered hNSCs in an Experimental Model of Intraspinal Cord Glioblastoma." Cells 13, no. 18 (2024): 1522. http://dx.doi.org/10.3390/cells13181522.

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Intramedullary spinal cord glioblastoma (ISCG) is lethal due to lack of effective treatment. We previously established a rat C6-ISCG model and the antitumor effect of F3.CD-TK, an hNSC line expressing CD and TK, via producing cytocidal 5FU and GCV-TP. However, the neurotherapeutic potential of this hNSC approach has remained uninvestigated. Here for the first time, cultured F3.CD-TK cells were found to have a markedly higher oncolytic effect, which was GJIC-dependent, and BDNF expression but less VEGF secretion than F3.CD. In Rowett athymic rats, F3.CD-TK (1.5 × 106 cells/10 µL × 2), injected

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Dr., W.M.D.S. Perera, H.A.R.P. Perera Dr., and Vidhyajini R.V. "Neuro-therapeutic effect of Rauvolfia serpentina (Sarpagandha/ Ekaweriya); A review." Journal of Advances in Experimental Therapeutics and Neurotherapeutics 1, no. 2 (2023): 7–12. https://doi.org/10.5281/zenodo.8085009.

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<em>Sarpagandha </em><em>is a popular</em><em> and rich compendium of herbs of Ayurveda and is botanically identified as </em><em>Rauvolfia serpentina</em><em>. It is extensively used in the management of neurological disorders. It possesses active principles belonging to Alkaloids, Phenols, F</em><em>lavonoids, and Tan</em><em>n</em><em>ins. </em><em>Relevant authentic Ayurveda texts and English language articles from databases PubMed, Research Gate and Google Scholar were searched using appropriate keywords and following the PRISMA model Present review work emphasizes the neurotherapeutic ef

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7

Vázquez Alberdi, Lucia, Gonzalo Rosso, Lucía Velóz, et al. "Curcumin and Ethanol Effects in Trembler-J Schwann Cell Culture." Biomolecules 12, no. 4 (2022): 515. http://dx.doi.org/10.3390/biom12040515.

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Charcot-Marie-Tooth (CMT) syndrome is the most common progressive human motor and sensory peripheral neuropathy. CMT type 1E is a demyelinating neuropathy affecting Schwann cells due to peripheral-myelin-protein-22 (PMP22) mutations, modelized by Trembler-J mice. Curcumin, a natural polyphenol compound obtained from turmeric (Curcuma longa), exhibits dose- and time-varying antitumor, antioxidant and neuroprotective properties, however, the neurotherapeutic actions of curcumin remain elusive. Here, we propose curcumin as a possible natural treatment capable of enhancing cellular detoxification

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BULAMA, Ibrahim, Nasiru SULEİMAN, Lawal BİLBİS, et al. "Antioxidative and Neurotherapeutic Effect of Ascorbic acid on Albino rats Induced with Traumatic Brain Injury." Journal of Cellular Neuroscience and Oxidative Stress 12, no. 1 (2020): 922–36. http://dx.doi.org/10.37212/jcnos.800233.

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9

Arien-Zakay, Hadar, Galit Gincberg, Arnon Nagler, et al. "Neurotherapeutic Effect of Cord Blood Derived CD45+Hematopoietic Cells in Mice after Traumatic Brain Injury." Journal of Neurotrauma 31, no. 16 (2014): 1405–16. http://dx.doi.org/10.1089/neu.2013.3270.

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Ahmad, Mohammad, Homood Alharbi, Abdualrahman Saeed Alshehry, and Ahmed Alsadoun. "Comparative Evaluation of the Ameliorating Effects of Some Natural Products as Neurotherapeutic Interventions for a Neurodegenerative Disorder in Status Epilepticus Model." Annals of Neurodegenerative Disorders 8, no. 1 (2024): 1–9. http://dx.doi.org/10.47739/neurodegenerativedisorders1035.

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Background: The lithium-pilocarpine (Li-Pc) model of status epilepticus (SE) is the most convenient and is frequently used for pathophysiological and management strategies in search of new, safe, and effective therapeutic agents including natural remedies for SE. Oral administration of cinnamon (CIN), Nigella sativa (NS), and curcumin (CUR) can reduce neuroinflammation which is a common feature of neurodegenerative disorders. Although many studies on CUR and NS effects on SE have been undertaken, to our best knowledge no study has been conducted on studying the effect of CIN on SE. Purpose: Th

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Books on the topic "Neurotherapeutic effect"

1

M, Pullan Linda, and Patel Jitendra, eds. Neurotherapeutics: Emerging strategies. Humana Press, 1996.

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Pullan, Linda M. Neurotherapeutics. Springer, 2013.

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Pullan, Linda M., and Jitendra Patel. Neurotherapeutics: Emerging Strategies. Humana Press, 1995.

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Pullan, Linda M., and Jitendra Patel. Neurotherapeutics: Emerging Strategies (Contemporary Neuroscience). Humana Press, 1996.

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Book chapters on the topic "Neurotherapeutic effect"

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Kocyigit, Mine, and Doha Al Marmour. "Neurotherapeutic Properties of Some Medicinal Plants Used in the Treatment of Alzheimer’s Disease, Learning and Memory Impairments." In Brain Health and Medicinal Plants. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359241.11.

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Neurological problems such as Alzheimer’s disease and learning and memory disorders are a growing concern today. In the treatment of such disorders, some herbal remedies used by traditional medicine may be a potential solution. In this review, the neurotherapeutic properties of some medicinal plants used in the treatment of neurological disorders such as Alzheimer’s disease and learning and memory disorders are examined. Herbs such as Ginkgo biloba, Panax ginseng, Morinda citrifolia, and Polygala tenuifolia have been found to have antioxidant, anti-inflammatory, and neuroprotective properties

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Onyeaka, Henry K., Scott R. Beach, Jeff C. Huffman, and Theodore A. Stern. "Side Effects of Psychotropic Medications." In Massachusetts General Hospital Psychopharmacology and Neurotherapeutics. Elsevier, 2025. http://dx.doi.org/10.1016/b978-0-443-11972-9.00013-2.

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Babayeva, Mariana, and Zvi G. Loewy. "Cannabis Compounds: Potential Therapy for Neurological Disease." In Medicinal Plants - Harnessing the Healing Power of Plants for Health [Working Title]. IntechOpen, 2024. http://dx.doi.org/10.5772/intechopen.1005770.

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Identification and development of pharmaceuticals for neurological disorders is associated with several unique challenges. The primary weakness of candidate neurological compounds is the poor penetration efficacy across the blood-brain barrier (BBB). The BBB is the bottleneck in nervous system drug development and is the paramount factor that limits success in neurotherapeutics. Findings suggest cannabinoids might overcome the limiting effects of the BBB and play a key role in improving neurological dysfunctions. This supports the therapeutic potential of cannabidiol for the treatment of ische

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4

Fargo, K. N., T. R. Pak, E. M. Foecking, and K. J. Jones. "Molecular Biology of Androgen Action: Perspectives on Neuroprotective and Neurotherapeutic Effects." In Hormones, Brain and Behavior. Elsevier, 2009. http://dx.doi.org/10.1016/b978-008088783-8.00036-x.

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Sharma, Anuradha, and Gurcharan Kaur. "Tinospora cordifolia in Neurodegeneration: A Strong Antioxidant and Anti-inflammatory Phytotherapeutic Drug Candidate." In Traditional Medicine for Neuronal Health. BENTHAM SCIENCE PUBLISHERS, 2023. http://dx.doi.org/10.2174/9789815040197123010009.

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Tinospora cordifolia is a Rasayana herb of Ayurveda, commonly known as “Heavenly Elixir” or “Amrita”, and one of the most exploited herbs in herbal medicines. T. cordifolia is well reported for its various pharmacological properties, such as anti-diabetic, anti-inflammatory, antipyretic, immunomodulatory, anti-cancer, cardioprotective, neuroprotective, and hepatoprotective activities. The prevalence of neurodegenerative diseases and other neurologic disorders is increasing worldwide. Oxidative stress and neuroinflammation are among the major pathologic mechanisms underlying neurodegenerative d

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