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Dissertations / Theses on the topic 'Neurotrophic factotrs'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Saif, Sakina. "AP-1 is required for CMX-8933-induced SOD upregulation and is translocated in response to a human EPN mimetic." Link to electronic thesis, 2004. http://www.wpi.edu/Pubs/ETD/Available/etd-0503104-162858/.

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2

Agerman, Karin. "Specificity of neurotrophins in the nervous system : a genetic approach to determine receptor engagement by neurotrophins /." Stockholm, 2003. http://diss.kib.ki.se/2004/91-7349-730-4/.

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3

Lichtenecker, Petra [Verfasser]. "Modulation der Sekretion des Neurotrophins "Brain-Derived Neurotrophic Factor" durch zyklisches Adenosinmonophosphat / Petra Lichtenecker." Magdeburg : Universitätsbibliothek, 2016. http://d-nb.info/1128726483/34.

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4

Ashcroft, Margaret. "Neurotrophic factors : production and purification of neurotrophic factor receptors using a baculovirus expression system." Thesis, University of Bristol, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260900.

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5

Barkholz, Michael [Verfasser], and Maike [Akademischer Betreuer] Petersen. "Charakterisierungsstudien der biologischen und neurotrophen Eigenschaften des cerebral dopamine neurotrophic factor (CDNF) / Michael Barkholz. Betreuer: Maike Petersen." Marburg : Philipps-Universität Marburg, 2012. http://d-nb.info/1025538609/34.

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6

Linnarsson, Sten. "Neurotrophic factors and neuronal plasticity /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4618-3/.

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7

Treanor, James J. S. "Neurotrophic factors and Alzheimer's disease." Thesis, University of Bristol, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385730.

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8

Martinez, Humberto Jose. "Nerve growth factor actions on the brain /." Access full-text from WCMC, 1989. http://proquest.umi.com/pqdweb?did=744572291&sid=1&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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9

Gunther, Erik Christian. "Molecular mechanisms of brain derived neurotrophic factor secretion and action /." Thesis, Connect to this title online; UW restricted, 2000. http://hdl.handle.net/1773/5086.

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10

Gielen, Alexander W. "Neurotrophic and immune regulatory factors in neuroinflammation /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-122-9/.

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11

Alqudah, Mohammad. "Characterization of the neurotrophic factor Brain-Derived Neurotrophic Factor (BDNF) in intestinal smooth muscle cells." VCU Scholars Compass, 2013. http://scholarscompass.vcu.edu/etd/3095.

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Brain-derived neurotrophic factor (BDNF) belongs to the neurotrophin family of secreted proteins, which include in addition to BDNF, nerve growth factor (NGF) and neurotrophin 3-6 (NT-3-6). BDNF mediates its functions by activating two cell surface receptors, pan-neurotrophin receptor (P75NTR) and tropomyosin-related kinase B (TrkB) and their downstream intracellular cascades. BDNF is best known for its role in neuronal survival, regulation of neuronal differentiation, migration and activity-dependent synaptic plasticity. However, BDNF is widely expressed in non-neuronal tissues as well. The l

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12

Zhu, Shun-Wei. "Brain neurotrophin levels and mouse behavior : relationship to environmental influences /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-843-6/.

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13

Singer, Cherie A. "Neurotrophic and neuroprotective effects of estrogen /." Thesis, Connect to this title online; UW restricted, 1998. http://hdl.handle.net/1773/6301.

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14

Littrell, Ofelia Meagan. "NIGROSTRIATAL DOPAMINE-NEURON FUNCTION FROM NEUROTROPHIC-LIKE PEPTIDE TREATMENT AND NEUROTROPHIC FACTOR DEPLETION." UKnowledge, 2011. http://uknowledge.uky.edu/neurobio_etds/1.

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Trophic factors have shown great promise in their potential to treat neurological disease. In particular, glial cell line-derived neurotrophic factor (GDNF) has been identified as a potent neurotrophic factor for midbrain dopamine (DA) neurons in the substantia nigra (SN), which lose function in Parkinson’s disease (PD). GDNF progressed to phase II clinical trials, which did not meet proposed endpoints. The large size and binding characteristics of GDNF have been suspected to contribute to some of the shortcomings of GDNF related to delivery to target brain regions. Smaller peptides derived fr

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15

Ghazi-Nouri, Seyed Mohammad Sadegh. "Expression of brain derived neurotrophic factor (BDNF), neurotrophin 4 (NT4) and their common receptor, TrkB, by human Müller cells in vitro and in vivo." Thesis, University College London (University of London), 2008. http://discovery.ucl.ac.uk/1445573/.

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Neurotrophins are trophic and mitogenic proteins that play a role in the development, differentiation, connectivity and survival of neurons, acting via their specific receptors in the central and peripheral nervous system, including the retina. The preferred receptor for brain derived neurotrophic factor (BDNF) and neurotrophin 4 (NT4) is TrKB. Muller cells may play an important role in neurotrophin function in the nervous system. It has been suggested that neurotrophins exert their effects on photoreceptors by acting indirectly through activation of Muller cells. The aims of the present work

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16

Zhou, Xiangdong. "Brain-derived Neurotrophic Factor in Autonomic Nervous System: Nicotinic Acetylcholine Receptor Regulation and Potential Trophic Effects." Connect to full-text via OhioLINK ETD Center, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1130160629.

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Thesis (Ph.D.)--Medical University of Ohio, 2005.<br>"In partial fulfillment of the requirements for the degree of Doctor of Philosophy in Medical Sciences." Major advisor: Joseph F. Margiotta. Includes abstract. Document formatted into pages: iii, 226 p. Title from title page of PDF document. Bibliography: pages 80-92,130-139,149-225.

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17

Port, Martha D. "Regulation of expression and function of neurokine receptors /." Thesis, Connect to this title online; UW restricted, 2008. http://hdl.handle.net/1773/6283.

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18

Roeding, Ross L., Marla K. Perna, Elizabeth D. Cummins, Daniel J. Peterson, Matthew I. Palmatier, and Russell W. Brown. "Sex Differences in Adolescent Methylphenidate Sensitization: Effects on Glial Cell-Derived Neurotrophic Factor and Brain-Derived Neurotrophic Factor." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etsu-works/952.

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This study analyzed sex differences in methylphenidate (MPH) sensitization and corresponding changes in glial cell-derived neurotrophic factor (GDNF) and brain-derived neurotprhic factor protein (BDNF) in adolescent male and female rats. After habituation to a locomotor arena, animals were sensitized to MPH (5mg/kg) or saline from postnatal day (P) 33–49, tested every second day. On P50, one group of animals were injected with saline and behavior assessed for conditioned hyperactivity. Brain tissue was harvested on P51 and analyzed for GDNF protein. A second group of animals was also sensitize

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19

Guillin, Olivier. "Le Brain-derived neurotrophic factor contrôle l'expression du récepteur D3 de la dopamine : implications dans la sensibilisation." Paris 6, 2002. http://www.theses.fr/2002PA066169.

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20

Hing, Benjamin. "Investigating differential regulation of BDNF promoter IV activity by upstream polymorphic evolutionary conserved regions : implications for mood disorders and cognitive disfunction." Thesis, University of Aberdeen, 2011. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=185597.

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Major depressive disorder (MDD) and bipolar disorder (BD) are psychiatric diseases that affect behavior and impair cognition. A gene important to these disorders is the brain derived neurotrophic factor (BDNF) which is involved in processes controlling neuroplasticity. Previous studies have suggested that BDNF expression levels have to be finely regulated for normal mental health and cognition. This study therefore aimed to identify cis-regulatory elements that regulate BDNF promoter IV (BP4), which plays a role in mood and cognition, and investigated how polymorphisms in these cis-regulatory

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21

Krzyzanowska, Agnieszka. "Peripheral pain mechanisms and their modulation by neurotrophic factors." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.427819.

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22

Williams, Alan. "Regulation of gene expression and survival by neurotrophic factors." Thesis, University of Bristol, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265495.

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23

Kelly, Gerard. "The effects of neurotrophic factors in facial nerve repair." Thesis, University of Edinburgh, 2002. http://hdl.handle.net/1842/23071.

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The present work established a model for the electrophysiological assessment of the rat facial nerve and used the rat and the sheep as animal models for facial nerve repair. In the rat facial nerve, after transection and repair of the buccal division, there was an increase in the number of regenerated axons with 10 μg of both CNTF and BDNF administered exogenously near the site of nerve repair, over a period of 14 days (p=0.003). There was no increase in the number of fibres with similar administration of GDNF and NT 4/5. The administration of these neurotrophic factors failed to show any othe

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24

Galindo, Ramon Gustavo. "The effect of neurotrophic factors on spiral ganglion neurons." Thesis, University of Iowa, 2012. https://ir.uiowa.edu/etd/3456.

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The current study investigates the survival and neuritogenic effects of various neurotrophic factors on rat spiral ganglion neurons (SGNs) in vitro. In particular, ciliary neurotrophic factor (CNTF), glial derived neurotrophic factor (GDNF), neurotrophin-3 (NT-3) and neurturin (NRTN) were assayed on postnatal day 4-6 SGNs. CNTF and NT-3 produced a robust survival effect while GDNF and NRTN failed to do so. A dose response revealed CNTF to be effective at promoting survival as low as 5ng/ml. In addition, CNTF promoted neurite growth in both depolarizing and non-depolarizing conditions, suggesti

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25

Polacchini, Alessio. "Neurotrophic factors and other humoral mediators in chronic stress." Doctoral thesis, Università degli studi di Trieste, 2015. http://hdl.handle.net/10077/11017.

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2013/2014<br>Lo stress è stato definito come "una risposta specifica del corpo a qualsiasi richiesta non specifica (stressor) su di esso". Quando lo stress, reale o percepito, è duraturo o la risposta non è appropriata (distress), i cambiamenti fisiologici di breve termine lasciano spazio ad alterazioni di lungo termine che possono causare condizioni patologiche, tra cui malattie mentali. Secondo questo modello di "carico allostatico", i mediatori primari come le catecolamine e gli ormoni steroidei, innescano alterazioni multi-sistemiche su mediatori secondari che portano a manifestazioni nega

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26

Goodison, Tavia. "Capacity of Neurotrophic Factors of the GDNF Family in Supporting the Survival of Dopaminergic Neurons in the Central Nervous System of the Aging Mouse." Thesis, Griffith University, 2013. http://hdl.handle.net/10072/367684.

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Dopaminergic neurons of the substantia nigra are vital for proper motor function. The Glial cell line-derived neurotrophic factor (GDNF) family of neurotrophic factors have been shown to promote the survival of dopaminergic neurons both in vitro and in vivo. GDNF and its related factor neurturin have both been trialled as a therapy for Parkinson’s patients for which degeneration of dopaminergic neurons is hallmark pathology. However, GDNF and neurturin are not required for proper development of dopaminergic neurons. Knockout of the GDNF gene in mice causes improper formation of the enteric ner

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27

Gillespie, Lisa N. "Survival and regeneration in the deaf ear : the potential of neurotrophic factors /." Connect to thesis, 2004. http://repository.unimelb.edu.au/10187/1054.

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Spiral ganglion neurons (SGNs) within the cochlea degenerate following the loss of the auditory sensory epithelium, the auditory hair cells. Since these neurons are the target cells of the cochlear implant, which bypasses damaged or lost hair cells to stimulate the SGNs directly, enhanced SGN number and integrity may provide enhanced outcomes for cochlear implant patients. Improved contact between the cochlear implant electrode array and the auditory nerve fibres is also likely to enhance the benefits received by cochlear implant patients. Therefore, the identification of molecules with the

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28

Jones, Elizabeth Ellen. "NEUROTROPHIN EXPRESSION IN SYMPATHETIC NEURONS: INFLUENCES OF EXOGENOUS NGF AND AFFERENT INPUT." Oxford, Ohio : Miami University, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=miami1089903903.

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29

Schindler-Zajzon, Katalin [Verfasser], and Kerstin [Akademischer Betreuer] Krieglstein. "Transforming growth factor-beta/Glial cell derived neurotrophic factor-dependent neuronal differentiation = Transforming Growth Factor-beta/Glial cell Derived Neurotrophic Factor-abhängige neuronale Differenzierung." Freiburg : Universität, 2013. http://d-nb.info/1115490737/34.

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30

Wu, Linyan, and wu0071@flinders edu au. "BRAIN DERIVED NEUROTROPHIC FACTOR TRANSPORT AND PHYSIOLOGICAL SIGNIFICANCE." Flinders University. Medicine, 2007. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20071204.113001.

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Neurotrophins are important signaling molecules in neuronal survival and differentiation. The precursor forms of neurotrophins (proneurotrophins) are the dominant form of gene products in animals, which are cleaved to generate prodomain and mature neurotrophins, and are sorted to constitutive or regulated secretory pathway and released. Brain-derived neurotrophic factor (BDNF) plays a pivotal role in the brain development and in the pathogenesis of neurological diseases. In Huntingtons disease, the defective transport of BDNF in cortical and striatal neurons and the highly expressed polyQ mut

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31

Litzke, Julia Ulrike [Verfasser]. "Veränderungen sowie Korrelation der Neurotrophine nerve growth factor (NGF) und brain-derived neurotrophic factor (BDNF) im präfrontalen Kortex, Hippocampus und Serum von Ratten nach elektrokonvulsiver Behandlung / Julia Ulrike Litzke." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2018. http://d-nb.info/1160515026/34.

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32

Litzke, Julia [Verfasser]. "Veränderungen sowie Korrelation der Neurotrophine nerve growth factor (NGF) und brain-derived neurotrophic factor (BDNF) im präfrontalen Kortex, Hippocampus und Serum von Ratten nach elektrokonvulsiver Behandlung / Julia Ulrike Litzke." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2018. http://d-nb.info/1160515026/34.

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33

Man, David Shu Ki. "Structure and function studies of ciliary neurotrophic factor (CNTF) and its receptor complexes /." View Abstract or Full-Text, 2002. http://library.ust.hk/cgi/db/thesis.pl?BICH%202002%20MAN.

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Thesis (Ph. D.)--Hong Kong University of Science and Technology, 2002.<br>Includes bibliographical references (leaves 155-175). Also available in electronic version. Access restricted to campus users.

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34

Bengtsson, Henrik. "Bone Morphogenetic Protein Receptors in the Nervous System: Neurotrophic Functions with Emphasis on Catecholaminergic Neurons." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5186-1/.

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35

Liu, Ping. "Blood brain-derived neurotrophic factor (BDNF) expression in normal humans and schizophrenic patients." Click to view the E-thesis via HKUTO, 2004. http://sunzi.lib.hku.hk/hkuto/record/B31352121.

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36

Hiltunen, Jukka. "Neurotropic factors in rodent heart : from development to pathophysiology." Helsinki : University of Helsinki, 2001. http://ethesis.helsinki.fi/julkaisut/eri/biote/vk/hiltunen/.

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37

Liu, Ping, and 劉苹. "Blood brain-derived neurotrophic factor (BDNF) expression in normal humans and schizophrenic patients." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B31352121.

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38

Goggi, Julian Luke. "Brain-derived neurotrophic factor and modulation of synaptic output." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.399537.

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39

Owen, Deborah Louise. "The neurotrophic actions of human melanotropin potentiating factor (hMPF)." Thesis, University of Liverpool, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385290.

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40

Dyson, Sean Christopher. "Novel strategies for neurotrophic factor delivery in neurodegenerative disease." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608085.

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41

Korsak, Kris. "The effects of neurotrophic factors on enteric ganglion cells in vitro." Thesis, Open University, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288355.

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42

Boucher, Timothy John. "The GDNF family of neurotrophic factors : effects on adult sensory neurons." Thesis, King's College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.272413.

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43

Bergauer, Lisa [Verfasser], Nicolas [Gutachter] Schlegel, Michael [Gutachter] Scheurlen, and Thomas [Gutachter] Haaf. "Die Bedeutung des neurotrophen Faktors Glial cell line-derived neurotrophic factor (GDNF) für die Integrität der intestinalen Epithelbarriere / Lisa Bergauer ; Gutachter: Nicolas Schlegel, Michael Scheurlen, Thomas Haaf." Würzburg : Universität Würzburg, 2017. http://d-nb.info/1147682062/34.

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44

Palm, Kaia. "Regulation of neuronal gene expression /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980612palm.

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45

Nosrat, Christopher A. "Neurotrophins in the development of the gustatory system and teeth /." Stockholm, 1997. http://diss.kib.ki.se/1997/91-628-2795-2.

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46

Grimsholm, Ola. "Neuropeptides and neurotrophins in arthritis : studies on the human and mouse knee joint." Doctoral thesis, Umeå universitet, Integrativ medicinsk biologi, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1863.

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Neuropeptides, such as substance P (SP) and bombesin/gastrin-releasing peptide (BN/GRP), and neurotrophins are involved in neuro-immunomodulatory processes and have marked trophic, growth-promoting and inflammation-modulating properties. The impact of these modulators in rheumatoid arthritis (RA) is, however, unclear. An involvement of the innervation, including the peptidergic innervation, is frequently proposed as an important factor for arthritic disease. Many patients with RA, but not all, benefit from treatment with anti-TNF medications. The studies presented here aimed to investigate the

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47

Theofilopoulos, Spyridion. "Neurotrophic factors in neurotransmitter phenotype determination and neuroprotection in the developing brain." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405421.

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48

Söderpalm, Annika. "Photoreceptor development and degeneration in retinal organ culture Effects of neurotrophic factors /." [S.l. : s.n.], 1999. http://catalog.hathitrust.org/api/volumes/oclc/44519577.html.

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49

Kang, Jian. "Artemin: from neurotrophic factor to oncogene in human mammary carcinoma." Thesis, University of Auckland, 2011. http://hdl.handle.net/2292/6808.

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Artemin (ARTN), a member of the glial cell derived neurotrophic factor (GDNF) family of ligands (GFLs), plays a pivotal role in the development and maintenance of the sympathetic and sensory nervous systems. A growing body of evidence supports the oncogenic potential of GFLs in the malignancies within and outside the nervous system. I demonstrate herein that ARTN is ubiquitously expressed in human mammary carcinoma cell lines. Forced expression of ARTN in mammary carcinoma cells increases cell survival, enhances anchorage-independent growth and promotes migration and invasion, with resu

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50

Klein, Inna. "Einfluss des Brain-derived neurotrophic factor auf Schizophrenie und Gedächtnisleistungen." Diss., Ludwig-Maximilians-Universität München, 2013. http://nbn-resolving.de/urn:nbn:de:bvb:19-162685.

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Die Schizophrenie ist eine psychiatrische Erkrankung,die phänotypisch in vielgestaltigen Querschnittsbildern auftreten kann.Die Äthiopathogenese der Schizophrenie ist bis heute nicht geklärt.Es wird aber davon ausgegangen, dass sie einem polygenen Erbgang folgt und multifaktorielle Bedingungen zum Ausbruch der Krankheit führen.Ein alternativer Ansatz versucht nun mithilfe von klar definierten Endophänotypen Gene zu identifizieren, die mit neuroanatomischen, neurophysiologischen oder biochemischen Korrelaten einer Erkrankung assoziiert sind.In Zusammenhang mit der Schizophrenie gehören Lern- u

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