Relevant bibliographies by topics / Nicotinamide dehydrogenase subunit 4

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Academic literature on the topic 'Nicotinamide dehydrogenase subunit 4'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Nicotinamide dehydrogenase subunit 4"

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Kumari, Shesh, and Angela Di Cesare. "Nicotinamide dehydrogenase subunit 4 analysis of Xiphinema diversicaudatum and Xiphinema simile (Nematoda: Longidoridae)." European Journal of Plant Pathology 136, no. 4 (2013): 803–10. http://dx.doi.org/10.1007/s10658-013-0208-5.

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Mariotto, Sandra, Mari Rose Oliveira Silva, Cristina Márcia de Menezes Butakka, et al. "Comparative Genetics Using Three mtDNA Markers in Aedes aegypti (Linnaeus) Populations from Municipalities in the State of Mato Grosso, Brazil." Ensino, Saude e Ambiente 15, no. 3 (2023): 503–23. http://dx.doi.org/10.22409/resa2022.v15i3.a54219.

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A methodological strategy for teaching in health and environment is to know the genetics of an important vector of dengue in different populations of mosquitoes. The aim of this study was to investigate the genetic variability of Aedes aegypti through molecular markers of mitochondrial DNA, COI, ND4 and ND5. Mosquitoes were collected using ovitraps to capture them at points located in four municipalities in the state of Mato Grosso. Subsequently, we amplified 169 samples with the best DNA result using primers for mitochondrial DNA (mtDNA): Cytochrome Oxidase Subunit I (COI - F and R), Nicotina
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Dahadhah, Fatina W., Mayyas Saleh Jaweesh, Mazhar Salim Al Zoubi, Manal Issam Abu Alarjah, Mohamad Eid Hammadeh, and Houda Amor. "Mitochondrial nicotinamide adenine dinucleotide hydride dehydrogenase (NADH) subunit 4 (MTND4) polymorphisms and their association with male infertility." Journal of Assisted Reproduction and Genetics 38, no. 8 (2021): 2021–29. http://dx.doi.org/10.1007/s10815-021-02199-w.

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Abstract Purpose The purpose of the present study was to determine the relationship between infertility and the polymorphisms of mitochondrial NADH dehydrogenase subunit 4 (MTND4) by spermatozoa analysis in fertile and subfertile men. Methods Samples were divided into 68 subfertile men (case group) and 44 fertile men (control group). After semen analysis, samples were purified. The whole genome was extracted using a QIAamp DNA Mini Kit and the mitochondrial DNA was amplified by using the REPLI-g Mitochondrial DNA Kit. Polymerase chain reaction (PCR) was used to amplify the MT-ND4 gene. Then, s
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Dunham-Snary, Kimberly J., Danchen Wu, François Potus, et al. "Ndufs2, a Core Subunit of Mitochondrial Complex I, Is Essential for Acute Oxygen-Sensing and Hypoxic Pulmonary Vasoconstriction." Circulation Research 124, no. 12 (2019): 1727–46. http://dx.doi.org/10.1161/circresaha.118.314284.

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Rationale: Hypoxic pulmonary vasoconstriction (HPV) optimizes systemic oxygen delivery by matching ventilation to perfusion. HPV is intrinsic to pulmonary artery smooth muscle cells (PASMCs). Hypoxia dilates systemic arteries, including renal arteries. Hypoxia is sensed by changes in mitochondrial-derived reactive oxygen species, notably hydrogen peroxide (H 2 O 2 ) ([H 2 O 2 ] mito ). Decreases in [H 2 O 2 ] mito elevate pulmonary vascular tone by increasing intracellular calcium ([Ca 2+ ] i ) through reduction-oxidation regulation of ion channels. Although HPV is mimicked by the Complex I in
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Parsons, Richard B., Shylesh Aravindan, Anusha Kadampeswaran, et al. "The expression of nicotinamide N-methyltransferase increases ATP synthesis and protects SH-SY5Y neuroblastoma cells against the toxicity of Complex I inhibitors." Biochemical Journal 436, no. 1 (2011): 145–55. http://dx.doi.org/10.1042/bj20101685.

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NNMT (nicotinamide N-methyltransferase, E.C. 2.1.1.1) catalyses the N-methylation of nicotinamide to 1-methylnicotinamide. NNMT expression is significantly elevated in a number of cancers, and we have previously demonstrated that NNMT expression is significantly increased in the brains of patients who have died of Parkinson's disease. To investigate the cellular effects of NNMT overexpression, we overexpressed NNMT in the SH-SY5Y cell line, a tumour-derived human dopaminergic neuroblastoma cell line with no endogenous expression of NNMT. NNMT expression significantly decreased SH-SY5Y cell dea
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Nishino, Daichi, Taketo Haginouchi, Takeshi Shimogiri, et al. "A Pilot Study: Maternal Undernutrition Programs Energy Metabolism and Alters Metabolic Profile and Morphological Characteristics of Skeletal Muscle in Postnatal Beef Cattle." Metabolites 15, no. 3 (2025): 209. https://doi.org/10.3390/metabo15030209.

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Objectives: This study investigated the long-term effects of maternal undernutrition on overall muscle metabolism, growth performance, and muscle characteristics in postnatal offspring of Wagyu (Japanese Black) cattle. Methods: Wagyu cows were divided into nutrient-adequate (control, CNT; n = 4, 120% of requirements) and nutrient-restricted groups (NR; n = 4; 60% of requirements), and treated from day 35 of gestation until parturition. Diets were delivered on the basis of crude protein requirements, meeting 100% and 80% of dry matter requirements in CNT and NR groups, respectively. All offspri
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Hussain, Shafaat, Abdul Waheed Khan, Alexander Akhmedov, et al. "Hyperglycemia Induces Myocardial Dysfunction via Epigenetic Regulation of JunD." Circulation Research 127, no. 10 (2020): 1261–73. http://dx.doi.org/10.1161/circresaha.120.317132.

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Rationale: Hyperglycemia -induced reactive oxygen species are key mediators of cardiac dysfunction. JunD (Jund proto-oncogene subunit), a member of the AP-1 (activator protein-1) family of transcription factors, is emerging as a major gatekeeper against oxidative stress. However, its contribution to redox state and inflammation in the diabetic heart remains to be elucidated. Objective: The present study investigates the role of JunD in hyperglycemia-induced and reactive oxygen species–driven myocardial dysfunction. Methods and Results: JunD mRNA and protein expression were reduced in the myoca
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Chen, Sixi, Xinzhu Bao, Hongzhi Chen, et al. "Thyroid Cancer-Associated Mitochondrial DNA Mutation G3842A Promotes Tumorigenicity via ROS-Mediated ERK1/2 Activation." Oxidative Medicine and Cellular Longevity 2022 (March 15, 2022): 1–16. http://dx.doi.org/10.1155/2022/9982449.

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Mitochondrial DNA (mtDNA) mutations have been identified in various human cancers, including thyroid cancer. However, the relationship between mtDNA and thyroid cancer remains unclear. Previous studies by others and us strongly suggested that mtDNA mutations in complex I may participate in thyroid cancer processes according to sequencing results of thyroid cancer tissue, although the associated pathogenic processes remain unknown. Here, to investigate whether mtDNA mutations contribute to thyroid cancer, we reanalyzed our sequencing results and characterized thyroid cancer-associated mutations
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Song, Hao, Xin Dang, Yuan-qiu He, Tao Zhang, and Hai-yan Wang. "Selection of housekeeping genes as internal controls for quantitative RT-PCR analysis of the veined rapa whelk (Rapana venosa)." PeerJ 5 (May 31, 2017): e3398. http://dx.doi.org/10.7717/peerj.3398.

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BackgroundThe veined rapa whelkRapana venosais an important commercial shellfish in China and quantitative real-time PCR (qRT-PCR) has become the standard method to study gene expression inR. venosa. For accurate and reliable gene expression results, qRT-PCR assays require housekeeping genes as internal controls, which display highly uniform expression in different tissues or stages of development. However, to date no studies have validated housekeeping genes inR. venosafor use as internal controls for qRT-PCR.MethodsIn this study, we selected the following 13 candidate genes for suitability a
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Cameron, Emilie C., Richard C. Wilkerson, Motoyoshi Mogi, et al. "Molecular Phylogenetics of Aedes japonicus, a Disease Vector That Recently Invaded Western Europe, North America, and the Hawaiian Islands." Journal of Medical Entomology 47, no. 4 (2010): 527–35. http://dx.doi.org/10.1093/jmedent/47.4.527.

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Abstract We used two mitochondrial loci (nicotinamide adenine dinucleotide dehydrogenase subunit 4 and cytochrome oxidase II) and a nuclear locus (28S-D2 spacer) for a total of 1337 bp to evaluate the relationships among the four subspecies of Aedes (Finlaya) japonicus Theobald. Ae. j. japonicus was recently introduced into the United States and has been expanding rapidly. We also included in our analysis a morphologically very closely related species, Aedes (Finlaya) koreicus Edwards, as well as three more distantly related species: Aedes (Finlaya) togoi Theobald, Aedes (Finlaya) hatorii Yama
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Book chapters on the topic "Nicotinamide dehydrogenase subunit 4"

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Kagendo, Dorothy, Eric Muchiri, Peter Gitonga, and Esther Muthoni. "Interlinks between Wildlife and Domestic Cycles of Echinococcus spp. in Kenya." In Managing Wildlife in a Changing World [Working Title]. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.94612.

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Effective conservation and management of wildlife in the current changing world, call for incorporation of infectious zoonotic diseases surveillance systems, among other interventions. One of such diseases is echinococcosis, a zoonotic disease caused by Echinococcus species. This disease exists in two distinct life cycle patterns, the domestic and wildlife cycles. To investigate possible inter-links between these cycles in Kenya, 729 fecal samples from wild carnivores and 406 from domestic dogs (Canis lupus familiaris) collected from Maasai Mara and Samburu National Reserves were analyzed. Tae
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Cox, Timothy M. "Disorders of galactose, pentose, and pyruvate metabolism." In Oxford Textbook of Medicine, edited by Timothy M. Cox. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0229.

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Inborn errors of galactose metabolism—galactose is principally found as free lactose in dairy products. Three inborn errors of galactose metabolism are recognized: (1) galactokinase deficiency (‘galactose diabetes’)—a very rare condition which impairs the assimilation of dietary galactose such that the free sugar and its metabolites appear in plasma and urine; (2) classical galactosaemia (galactose-1-phosphate uridylyltransferase deficiency)—the commonest (1/47 000 births) and most important disorder. High concentrations of galactose in the plasma and tissues lead to aberrant glycosylation of
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