Relevant bibliographies by topics / Nicotinic acetylcholine receptors α7

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Nicotinic acetylcholine receptors α7'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Nicotinic acetylcholine receptors α7"

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Lavezzi, Anna. "Toxic Effect of Cigarette Smoke on Brainstem Nicotinic Receptor Expression: Primary Cause of Sudden Unexplained Perinatal Death." Toxics 6, no. 4 (2018): 63. http://dx.doi.org/10.3390/toxics6040063.

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Among the neurotoxicants contained in tobacco smoke, if absorbed during pregnancy, nicotine significantly affects α7-nicotinic acetylcholine receptors, which play essential roles in the development of the brainstem regions receiving cholinergic projections in perinatal life. Immunohistochemical procedures for analysing formalin-fixed and paraffin-embedded brainstem samples from 68 fetuses and early newborns, with smoking and non-smoking mothers, who died of known and unknown causes, were carried out in order to determine if nicotine had activated the α7-nicotinic acetylcholine receptors. High
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Wongtrakool, Cherry, Susanne Roser-Page, Hilda N. Rivera та Jesse Roman. "Nicotine alters lung branching morphogenesis through the α7 nicotinic acetylcholine receptor". American Journal of Physiology-Lung Cellular and Molecular Physiology 293, № 3 (2007): L611—L618. http://dx.doi.org/10.1152/ajplung.00038.2007.

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There is abundant epidemiological data linking prenatal environmental tobacco smoke with childhood asthma and wheezing, but the underlying molecular and physiological mechanisms that occur in utero to explain this link remain unelucidated. Several studies suggest that nicotine, which traverses the placenta, is a causative agent. Therefore, we studied the effects of nicotine on lung branching morphogenesis using embryonic murine lung explants. We found that the expression of α7 nicotinic acetylcholine receptors, which mediate many of the biological effects of nicotine, is highest in pseudogland
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Beckel, Jonathan M., Anthony Kanai, Sun-Ju Lee, William C. de Groat, and Lori A. Birder. "Expression of functional nicotinic acetylcholine receptors in rat urinary bladder epithelial cells." American Journal of Physiology-Renal Physiology 290, no. 1 (2006): F103—F110. http://dx.doi.org/10.1152/ajprenal.00098.2005.

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Although nicotinic acetylcholine receptors in both the central and peripheral nervous systems play a prominent role in the control of urinary bladder function, little is known regarding expression or function of nicotinic receptors in the bladder epithelium, or urothelium. Nicotinic receptors have been described in epithelial cells lining the upper gastrointestinal tract, respiratory tract, and the skin. Thus the present study examined the expression and functionality of nicotinic receptors in the urothelium, as well as the effects of stimulation of nicotinic receptors on the micturition refle
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MANEU, VICTORIA, GUILLERMO GERONA, LAURA FERNÁNDEZ, NICOLÁS CUENCA, and PEDRO LAX. "Evidence of alpha 7 nicotinic acetylcholine receptor expression in retinal pigment epithelial cells." Visual Neuroscience 27, no. 5-6 (2010): 139–47. http://dx.doi.org/10.1017/s0952523810000246.

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AbstractSome evidence suggests that retinal pigment epithelium (RPE) can express nicotinic acetylcholine receptors (nAChRs) as described for other epithelial cells, where nAChRs have been involved in processes such as cell development, cell death, cell migration, and angiogenesis. This study is designed to determine the expression and activity of α7 nAChRs in RPE cells. Reverse transcriptase (RT)-PCR was performed to test the expression of nicotinic α7 subunit in bovine RPE cells. Protein expression was determined by Western blot and by immunocytochemistry. Expression of nicotinic α7 subunits
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Strang, Christianne E., Ye Long, Konstantin E. Gavrikov, Franklin R. Amthor, and Kent T. Keyser. "Nicotinic and muscarinic acetylcholine receptors shape ganglion cell response properties." Journal of Neurophysiology 113, no. 1 (2015): 203–17. http://dx.doi.org/10.1152/jn.00405.2014.

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The purpose of this study was to evaluate the expression patterns of nicotinic and muscarinic ACh receptors (nAChRs and mAChRs, respectively) in relation to one another and to understand their effects on rabbit retinal ganglion cell response properties. Double-label immunohistochemistry revealed labeled inner-retinal cell bodies and complex patterns of nAChR and mAChR expression in the inner plexiform layer. Specifically, the expression patterns of m1, m4, and m5 muscarinic receptors overlapped with those of non-α7 and α7 nicotinic receptors in presumptive amacrine and ganglion cells. There wa
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Hawkins, Brian T., Richard D. Egleton, and Thomas P. Davis. "Modulation of cerebral microvascular permeability by endothelial nicotinic acetylcholine receptors." American Journal of Physiology-Heart and Circulatory Physiology 289, no. 1 (2005): H212—H219. http://dx.doi.org/10.1152/ajpheart.01210.2004.

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Nicotine increases the permeability of the blood-brain barrier in vivo. This implies a possible role for nicotinic acetylcholine receptors in the regulation of cerebral microvascular permeability. Expression of nicotinic acetylcholine receptor subunits in cerebral microvessels was investigated with immunofluorescence microscopy. Positive immunoreactivity was found for receptor subunits α3, α5, α7, and β2, but not subunits α4, β3, or β4. Blood-brain barrier permeability was assessed via in situ brain perfusion with [14C]sucrose. Nicotine increased the rate of sucrose entry into the brain from 0
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Panchal, Jayharsh L. "Dual Signaling Modes of Alpha7 Nicotinic Acetylcholine Receptors (α7 nAChRs)". Annals of Experimental and Molecular Biology 1, № 1 (2018): 1–2. http://dx.doi.org/10.23880/aemb-16000102.

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This themed section of the Annals of Experimental and Molecular Biology is the product of an article that is focusing on metabotropic signaling of an ionotropic channel alpha7 nicotinic ACh receptors (α7 nAChRs). The article talks about how α7 nAChR, being an ion channel, can function in a metabotropic-signaling mode via Gprotein coupling followed by Gαq-PLC-IP3-Ca2+ release
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Christophe, Elodie, Aline Roebuck, Jochen F. Staiger, Daniel J. Lavery, Serge Charpak, and Etienne Audinat. "Two Types of Nicotinic Receptors Mediate an Excitation of Neocortical Layer I Interneurons." Journal of Neurophysiology 88, no. 3 (2002): 1318–27. http://dx.doi.org/10.1152/jn.2002.88.3.1318.

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Nicotinic acetylcholine receptors are widely expressed in the neocortex but their functional roles remain largely unknown. Here we investigated the effect of nicotinic receptor activation on interneurons of layer I, which contains a high density of cholinergic fiber terminals. Ninety-seven of 101 neurons recorded in whole cell configuration in rat acute slices were excited by local pressure application of nicotinic agonists, acetylcholine (500 μM), 1,1-dimethyl-4-phenyl-piperazinium (500 μM) or choline (10 mM). Biocytin labeling confirmed that our sample included different morphological types
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Pinheiro, Nathalia M., Rosana Banzato, Iolanda Tibério та ін. "Acute Lung Injury in Cholinergic-Deficient Mice Supports Anti-Inflammatory Role of α7 Nicotinic Acetylcholine Receptor". International Journal of Molecular Sciences 22, № 14 (2021): 7552. http://dx.doi.org/10.3390/ijms22147552.

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(1) Background: The lung cholinergic pathway is important for controlling pulmonary inflammation in acute lung injury, a condition that is characterized by a sudden onset and intense inflammation. This study investigated changes in the expression levels of nicotinic and muscarinic acetylcholine receptors (nAChR and mAChR) in the lung during acute lung injury. (2) Methods: acute lung injury (ALI) was induced in wild-type and cholinergic-deficient (VAChT-KDHOM) mice using intratracheal lipopolysaccharide (LPS) instillation with or without concurrent treatment with nicotinic ligands. Bronchoalveo
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Montaño-Velázquez, B. B., D. A. Lara-Sánchez, A. Orozco-Sánchez, F. J. García-Vázquez, M. R. Mora-Campos та K. Jáuregui-Renaud. "Sex difference in counts of α4 and α7 nicotinic acetylcholine receptors in the nasal polyps of adults with or without exposure to tobacco smoke". Journal of Laryngology & Otology 132, № 7 (2018): 596–99. http://dx.doi.org/10.1017/s0022215118000373.

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AbstractObjectiveTo assess counts of α4 and α7 nicotinic acetylcholine receptors in nasal polyps of adults with or without long-term exposure to cigarette tobacco smoke.MethodsTwenty-two patients with and 22 patients without exposure to cigarette tobacco smoke participated in the study. After endoscopic polypectomy, the fragments of the nasal polyps were analysed by immunohistochemistry.ResultsCompared to patients with no exposure, patients with exposure showed higher counts of α4 and α7 nicotinic acetylcholine receptors (t-test, p < 0.05). However, in patients with no exposure, multivariat
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Dissertations / Theses on the topic "Nicotinic acetylcholine receptors α7"

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Hammond, Victoria. "α7 nicotinic acetylcholine receptors at the glutamatergic synapse". Thesis, University of Bath, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.633163.

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Nicotinic acetylcholine receptor (nAChR) activation is neuroprotective and nicotine is a cognitive enhancer. Loss of nAChRs, deposition of tau neurofibrillary tangles, cleavage of amyloid precursor protein (APP) and inflammation are well documented in the pathogenesis of Alzheimer’s disease (AD). Sequential cleavage of APP by β- and γ-secretase enzymes generates soluble Aβ peptides, with oligomeric forms of Aβ implicated in both the control of synaptic excitability and dysregulation of synaptic transmission and induction of neuronal death in AD. Aβ production is inhibited by calcium-dependent
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Van, Rensburg Ruan. "Upregulation of neuronal α7 nicotinic acetylcholine receptors and preconditioning". Thesis, Durham University, 2007. http://etheses.dur.ac.uk/2452/.

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The upregulation of alpha 7 nicotinic acetylcholine receptors (α7 nAChRs) are putatively reported to play a role in in vivo cortical spreading depression-elicited neuroprotection. In this study, a reliable in vitro spreading depression model was created for studying this phenomenon. In contradiction to previous studies, it was, however, shown that functional α 7 nAChRs are down-regulated upon chronic depolarisation with KCl, although the activity of this receptor subtype remained essential for the preconditioning mechanism. Evidence was provided for a differential mechanism underlying protecti
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Brown, Jack. "α7 Nicotinic acetylcholine receptor-mediated calcium signalling in neuronal cells". Thesis, University of Bath, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.636524.

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α7 nicotinic acetylcholine receptors (nAChR) are highly permeable to Ca2+ and are clinical targets for Alzheimer’s disease and schizophrenia. The aim of this work was to examine α7 nAChR-mediated Ca2+ signalling in neuronal cells using three different methods, and to evaluate the effects of the desensitizing agonist and prototypical smoking-cessation drug sazetidine-A on α7 nAChRs. Initial studies used 96-well plate assays with SH-SY5Y cells to characterize responses evoked by the α7 nAChR-selective agonist PNU-282987 and positive allosteric modulator PNU-120596. This was complemented by live-
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Jackson, Asti. "Investigating the Modulation and Mechanisms of α7 Nicotinic Acetylcholine Receptors in Nicotine Dependence". VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/4851.

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Tobacco dependence dramatically increases health burdens and financial costs. Limitations of current smoking cessation therapies indicate the need for improved molecular targets. Nicotine, the main addictive component of tobacco, exerts its dependency effects via nicotinic acetylcholine receptors (nAChRs). The homomeric α7 nAChR is one of the most abundant receptors found in the brain and has unique features in comparison to other nAChR subtypes such as high calcium permeability, low probability of channel opening, and a rapid desensitization rate. α7 nAChR agonists reduce nicotine's rewarding
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Udakis, Matthew. "α7 nicotinic acetylcholine receptors : regulation of plasticity and network activity in the prelimbic cortex". Thesis, University of Bath, 2016. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707589.

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Abnormalities in the connectivity and activity of the prefrontal cortex (PFC) is the cause of many of the symptoms of neuropsychiatric disorders such as schizophrenia and Alzheimer’s disease. The PFC relies on a complex regulation of network activity and synaptic plasticity for healthy PFC function. These fundamental processes can be modulated by the neuromodulator acetylcholine, acting at α7 nicotinic acetylcholine receptors (α7 nAChRs), a system also compromised in neuropsychiatric disorders. Despite the evidence that α7 nAChRs are essential for healthy PFC function relatively little is know
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Anderson, Malia L. "The Effects of β-Amyloid on α7 Nicotinic Acetylcholine Receptors Expressed in Xenopus Oocytes". BYU ScholarsArchive, 2011. https://scholarsarchive.byu.edu/etd/2966.

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The exact mechanism and progression of Alzheimer's disease (AD) at present is not fully understood. In patients suffering from AD, damage to the hippocampal region and impairment of learning and memory is present. It is also known that a buildup of β-amyloid plaques occur in AD patients and that β-amyloid interacts with some subtypes of neuronal nicotinic acetylcholine receptors (neuronal nAChRs). These receptors are composed of five subunits. The most prevalent nAChR subunit composition through the brain as a whole is α7. Previous data produced from our lab suggests that α7 nAChRs are also on
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Young, Jared W. "Nicotine induced improvements in cognition : a possible role for the α7 nicotinic acetylcholine receptor". Thesis, University of Edinburgh, 2005. http://hdl.handle.net/1842/27731.

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Assessment of sustained attention in rodents can be performed using the 5-choice serial reaction-time (5-CSR) task; analogous to the continuous performance test used in man. A 5-CSR protocol was established which allowed the demonstration of nicotine-induced improvements in sustained attention in mice. In this task α7 nAChR knockout (KO) mice exhibited impaired acquisition and performance, providing additional evidence that this receptor may be a valid therapeutic target for cognitive enhancement. In order to investigate the role of nAChR manipulation on working memory, the odour span task, a
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Nishio, Takahiro. "Hepatic vagus nerve regulates Kupffer cell activation via α7 nicotinic acetylcholine receptor in nonalcoholic steatohepatitis". Kyoto University, 2017. http://hdl.handle.net/2433/225984.

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Beinat, Corinne. "The design and synthesis of selective α7 nicotinic acetylcholine receptor agonists for the treatment of neurological disease". Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/11635.

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The homomeric α7 nAChR is a ligand gated ion channel found throughout the CNS and PNS and is one of the most commonly expressed nicotinic receptors in the human brain. The α7 nAChR is expressed in particularly high levels in brain regions associated with learning and memory, such as the cerebral cortex and the hippocampus. Experimental evidence supports the involvement of this receptor in schizophrenia and Alzheimer’s disease (AD), modulators of the α7 nAChR have been extensively reviewed for the treatment of the cognitive deficits associated with these pathologies. Multiple α7 nAChR agonists
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Davy, Robert Carlos Barton. "Development of a transient expression system for the α7 neuronal nicotinic acetylcholine receptor in mammalian cells". Thesis, University of Bath, 1995. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295445.

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Books on the topic "Nicotinic acetylcholine receptors α7"

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Thany, Steeve Hervé, ed. Insect Nicotinic Acetylcholine Receptors. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6445-8.

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1944-, Barrantes Francisco J., ed. The nicotinic acetylcholine receptors: Current views and future trends. Springer, 1998.

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Clementi, Francesco, Cecilia Gotti, and Emanuele Sher, eds. Nicotinic Acetylcholine Receptors in the Nervous System. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-74167-8.

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Francesco, Clementi, Gotti Cecilia, Sher Emanuele, and North Atlantic Treaty Organization. Scientific Affairs Division., eds. Nicotinic acetylcholine receptors in the nervous system. Springer-Verlag, 1988.

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Slater, E. Yvonne. The effects of novel alkaloid derivatives on human nicotinic acetylcholine receptors. Oxford Brookes University, 2000.

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Jackson, Charles E. P. The pharmacology of nicotinic acetylcholine receptors in Heliothis virescens and Locusta migratoria neurones in vitro. Oxford Brookes University, 1998.

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A, Nordberg, and International Symposium on Nicotinic Receptors on the CNS - Their Role in Synaptic Transmission (1988 : Uppsala, Sweden), eds. Nicotinic receptors in the CNS: Their role in synaptic transmission. Elsevier, 1989.

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1958-, Parhar Ishwar S., ed. Gonadotropin-releasing hormone: Molecules and receptors. Elsevier, 2002.

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P, Illes, and Zimmermann Herbert 1944-, eds. Nucleotides and their receptors in the nervous system. Elsevier, 1999.

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G, Holstege, Bandler Richard, and Saper C. B, eds. The emotional motor system. Elsevier, 1996.

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Book chapters on the topic "Nicotinic acetylcholine receptors α7"

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Oz, Murat, Georg Petroianu, and Dietrich E. Lorke. "α7-Nicotinic Acetylcholine Receptors: New Therapeutic Avenues in Alzheimer’s Disease." In Nicotinic Acetylcholine Receptor Technologies. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3768-4_9.

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Lorke, Dietrich E., Georg Petroianu, and Murat Oz. "α7-Nicotinic Acetylcholine Receptors and β-Amyloid Peptides in Alzheimer’s Disease." In Nicotinic Acetylcholine Receptor Technologies. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3768-4_10.

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Hajós, Mihály, and Bruce N. Rogers. "α7 Nicotinic Acetylcholine Receptors in the Treatment of Schizophrenia." In Targets and Emerging Therapies for Schizophrenia. John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781118309421.ch12.

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Li, Ming D. "Nicotine Modulates Innate Immune Pathways via α7 Nicotinic Acetylcholine Receptor." In Tobacco Smoking Addiction: Epidemiology, Genetics, Mechanisms, and Treatment. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-7530-8_16.

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Helekar, Santosh A., Lorna Colquhoun, Hong Dang, Danong Chen, Finn Goldner, and Jim Patrick. "A Cyclophilin-Dependent Mechanism for α7 Neuronal Nicotinic Acetylcholine Receptor Maturation." In Effects of Nicotine on Biological Systems II. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7445-8_4.

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Berg, Darwin K., Zhong-wei Zhang, William G. Conroy, et al. "Expression, Function, and Regulation of Neuronal Acetylcholine Receptors Containing the α7 Gene Product." In Effects of Nicotine on Biological Systems II. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7445-8_8.

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Deutsch, Stephen I., and Jessica A. Burket. "An Evolving Therapeutic Rationale for Targeting the α7 Nicotinic Acetylcholine Receptor in Autism Spectrum Disorder." In Behavioral Pharmacology of the Cholinergic System. Springer International Publishing, 2020. http://dx.doi.org/10.1007/7854_2020_136.

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Gooch, Jan W. "Nicotinic Acetylcholine Receptors." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14318.

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Sundermann, Bernd, and Corinna Maul. "Acetylcholine Receptors: 8.1 Nicotinic Acetylcholine Receptors." In Analgesics. Wiley-VCH Verlag GmbH & Co. KGaA, 2005. http://dx.doi.org/10.1002/3527605614.ch8a.

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Marks, Michael J., Sharon R. Grady, Tristan D. McClure-Begley, Heidi C. O’Neill, and Cristian A. Zambrano. "Presynaptic Nicotinic Acetylcholine Receptors: Subtypes and Functions." In Nicotinic Receptors. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1167-7_4.

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Conference papers on the topic "Nicotinic acetylcholine receptors α7"

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Batista, Victor de Sousa, Lourival Rodrigues de Sousa Neto, Roberto Ribeiro Faria, Keli Cristina Barbosa dos Reis та Nailton Monteiro do Nascimento Júnior. "Post-processing of docking results through docking-based comparative intermolecular contacts analysis (dbCICA) of the α4β2 and α7 nicotinic acetylcholine receptors (nAChRs)". У VIII Simpósio de Estrutura Eletrônica e Dinâmica Molecular. Universidade de Brasília, 2020. http://dx.doi.org/10.21826/viiiseedmol2020146.

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The nAChRs are pentameric ligand-gated ionic channels that respond to the endogenous neurotransmitter acetylcholine, with the α4β2 and α7 subtypes being highly expressed in human brain. Those receptors are involved in many neurologic disorders such as Alzheimer’s disease and Schizophrenia, as well as in nicotine addiction. In this context, molecular modelling is a powerful tool for designing novel ligands targeting those receptors. In the present work, we applied dbCICA1 to identify optimal docking conditions for these two receptors. The methodology and results are summarized bellow. Briefly,
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Su, X., C. Zhao та J. Chen. "Vagal-α7 Nicotinic Acetylcholine Receptor Signaling Promotes Influenza Replication". У American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4370.

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Hong, Huixiao, Carmine Leggett та Suguna Sakkiah. "Development of Nicotinic Acetylcholine Receptor nAChR α7 Binding Activity Prediction Model". У BCB '17: 8th ACM International Conference on Bioinformatics, Computational Biology, and Health Informatics. ACM, 2017. http://dx.doi.org/10.1145/3107411.3108191.

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Wongtrakool, C., HN Rivera, S. Roser-Page та J. Roman. "Nicotine Induced Nerve Growth Factor Expression Is through α7 Nicotinic Acetylcholine Receptor Mediated Signals." У American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2429.

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Whiteaker, Paul, Andrew A. George, Sabin J. John та ін. "Analogs of α-conotoxin PnIC are selective for α7β2 over α7-only subtype nicotinic acetylcholine receptors". У ASPET 2024 Annual Meeting Abstract. American Society for Pharmacology and Experimental Therapeutics, 2024. http://dx.doi.org/10.1124/jpet.125.962890.

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McLoughlin, Wesley KX, Christophe Lucatelli, Dan Peters та ін. "P3 α7 nicotinic acetylcholine receptor expression in vascular cells during normoxia and hypoxia". У The Scottish Cardiovascular Forum 2019, Saturday 2nd February 2019, The Centre for Health Science, Old Perth Road, Inverness, Scotland. BMJ Publishing Group Ltd and British Cardiovascular Society, 2019. http://dx.doi.org/10.1136/heartjnl-2019-scf.11.

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Ma, J. D., Y. S. Mou, T. Yan та ін. "SAT0036 Nicotine promotes mmp-3 and rankl secretion through overexpressed nicotinic acetylcholine receptor Α7 in rheumatoid arthritis fibroblast-like synoviocytes". У Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.5115.

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Yilmazer, Selma. "Inhibition of nicotinic acetylcholine receptor nAChR α7 effects expression of BDNF in olfactory bulb organotypic slice cultures". У European Microscopy Congress 2020. Royal Microscopical Society, 2021. http://dx.doi.org/10.22443/rms.emc2020.400.

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Wang, Q., J. Lucas, I. K. Sundar та I. Rahman. "Electronic-Cigarette Induces Dysregulated Repair Response and Extracellular Matrix Remodeling in Mouse Lung Via α7 Nicotinic Acetylcholine Receptor". У American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6240.

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Sobus, Samantha L., Michelle A. Romano та Graham W. Warren. "Abstract 3948: The role of α7 nicotinic acetylcholine receptor activation and pharmacologic tobacco cessation agents on response to radiotherapy". У Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3948.

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Reports on the topic "Nicotinic acetylcholine receptors α7"

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Lindstrom, Jon M. Use of Monoclonal Antibodies to Study the Structure and Function of Nicotinic Acetylcholine Receptors on Electric Organ and Muscle and to Determine the Structure of Nicotinic Acetylcholine Receptors on Neurons. Defense Technical Information Center, 1988. http://dx.doi.org/10.21236/ada198425.

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