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Relevant bibliographies by topics / Pancreas β-cells regeneration

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Dissertations / Theses
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Academic literature on the topic 'Pancreas β-cells regeneration'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Pancreas β-cells regeneration"

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Pylaev, T. E., I. V. Smyshlyaeva та E. B. Popyhova. "Regeneration of β-cells of the islet apparatus of the pancreas. Literature review". Diabetes mellitus 25, № 4 (2022): 395–404. http://dx.doi.org/10.14341/dm12872.

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Diabetes of both type 1 and type 2 is characterized by a progressive loss of β-cell mass, which contributes to the disruption of glucose homeostasis. The optimal antidiabetic therapy would be simple replacement of lost cells, but at present, many researchers have shown that the pancreas (PZ) of adults has a limited regenerative potential. In this regard, significant efforts of researchers are directed to methods of inducing the proliferation of β-cells, stimulating the formation of β-cells from alternative endogenous sources and/or the generation of β-cells from pluripotent stem cells. Factors

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Beamish, Christine A., Sofia Mehta, Brenda J. Strutt, Subrata Chakrabarti, Manami Hara та David J. Hill. "Decrease in Ins+Glut2LO β-cells with advancing age in mouse and human pancreas". Journal of Endocrinology 233, № 3 (2017): 229–41. http://dx.doi.org/10.1530/joe-16-0475.

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The presence and location of resident pancreatic β-cell progenitors is controversial. A subpopulation of insulin-expressing but glucose transporter-2-low (Ins+Glut2LO) cells may represent multipotent pancreatic progenitors in adult mouse and in human islets, and they are enriched in small, extra-islet β-cell clusters (<5 β cells) in mice. Here, we sought to identify and compare the ontogeny of these cells in mouse and human pancreata throughout life. Mouse pancreata were collected at postnatal days 7, 14, 21, 28, and at 3, 6, 12, and 18 months of age, and in the first 28 days after β-cell m

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Balaji, Shruti, Tiziana Napolitano, Serena Silvano, et al. "Epigenetic Control of Pancreatic Regeneration in Diabetes." Genes 9, no. 9 (2018): 448. http://dx.doi.org/10.3390/genes9090448.

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Both type 1 and type 2 diabetes are conditions that are associated with the loss of insulin-producing β-cells within the pancreas. An active research therefore aims at regenerating these β-cells with the hope that they could restore euglycemia. The approaches classically used consist in mimicking embryonic development, making use of diverse cell sources or converting pre-existing pancreatic cells. Despite impressive progresses and promising successes, it appears that we still need to gain further insight into the molecular mechanisms underlying β-cell development. This becomes even more obviou

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Anggi, Viani, Joni Tandi, and Veronika Veronika. "TOTAL FLAVONOID DAN EFEKTIVITAS EKSTRAK ETANOL BIJI KELOR (Moringa oleifera L) ASAL KOTA PALU SULAWESI TENGAH TERHADAP HISTOPATOLOGI PANKREAS TIKUS PUTIH JANTAN (Rattus norvegicus) YANG DIINDUKSI STREPTOZOTOCIN." Jurnal Ilmiah Manuntung 6, no. 1 (2020): 24. http://dx.doi.org/10.51352/jim.v6i1.294.

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This study aims to determine the content of flavonoid and the effect of ethanol extract of moringa seeds on the regeneration of pancreatic β cells in male white rats streptozotocin induced diabetes. This study method used has total flavonoid equivalent quercetin by spectrophotometry uv-vis and to regeneration of pancreatic β cells in male white rats used 30 test animals,namely male white rats divided into 6 groups, each group consisted of 5 male white rats with details of group I as normal control, Group II as negative control given 0.5% Na-CMC suspension, Group III as positive control given g

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Karampelias, Christos, Bianca Băloiu, Birgit Rathkolb та ін. "Examining the liver–pancreas crosstalk reveals a role for the molybdenum cofactor in β-cell regeneration". Life Science Alliance 7, № 11 (2024): e202402771. http://dx.doi.org/10.26508/lsa.202402771.

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Regeneration of insulin-producing β-cells is an alternative avenue to manage diabetes, and it is crucial to unravel this process in vivo during physiological responses to the lack of β-cells. Here, we aimed to characterize how hepatocytes can contribute to β-cell regeneration, either directly or indirectly via secreted proteins or metabolites, in a zebrafish model of β-cell loss. Using lineage tracing, we show that hepatocytes do not directly convert into β-cells even under extreme β-cell ablation conditions. A transcriptomic analysis of isolated hepatocytes after β-cell ablation displayed alt

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Butler, Alexandra E., Sangeeta Dhawan, Jonathan Hoang та ін. "β-Cell Deficit in Obese Type 2 Diabetes, a Minor Role of β-Cell Dedifferentiation and Degranulation". Journal of Clinical Endocrinology & Metabolism 101, № 2 (2016): 523–32. http://dx.doi.org/10.1210/jc.2015-3566.

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Abstract Context: Type 2 diabetes is characterized by a β-cell deficit and a progressive defect in β-cell function. It has been proposed that the deficit in β-cells may be due to β-cell degranulation and transdifferentiation to other endocrine cell types. Objective: The objective of the study was to establish the potential impact of β-cell dedifferentiation and transdifferentiation on β-cell deficit in type 2 diabetes and to consider the alternative that cells with an incomplete identity may be newly forming rather than dedifferentiated. Design, Setting, and Participants: Pancreata obtained at

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Bonner-Weir, Susan, Akari Inada, Shigeru Yatoh та ін. "Transdifferentiation of pancreatic ductal cells to endocrine β-cells". Biochemical Society Transactions 36, № 3 (2008): 353–56. http://dx.doi.org/10.1042/bst0360353.

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The regenerative process in the pancreas is of particular interest, since diabetes, whether Type 1 or Type 2, results from an inadequate amount of insulin-producing β-cells. Islet neogenesis, or the formation of new islets, seen as budding of hormone-positive cells from the ductal epithelium, has long been considered to be one of the mechanisms of normal islet growth after birth and in regeneration, and suggested the presence of pancreatic stem cells. Results from the rat regeneration model of partial pancreatectomy led us to hypothesize that differentiated pancreatic ductal cells were the pan

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Köhler, Christina U., Marvin Olewinski, Andrea Tannapfel, Wolfgang E. Schmidt, Helga Fritsch та Juris J. Meier. "Cell cycle control of β-cell replication in the prenatal and postnatal human pancreas". American Journal of Physiology-Endocrinology and Metabolism 300, № 1 (2011): E221—E230. http://dx.doi.org/10.1152/ajpendo.00496.2010.

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β-Cell regeneration declines with aging, but the molecular mechanisms controlling β-cell replication in humans are not well understood. We compared the expression of selected cell cycle proteins in prenatal and adult tissue and examined the association of these proteins with β-cell replication. Pancreatic tissue from a total of 20 human fetuses and adults was stained for Ki67, cyclin D3, p16 and p27, and insulin. The β-cellular expression of these cell cycle proteins was determined. The frequency of β-cell replication was lower in adult compared with prenatal β-cells (<0.5 vs. 3.4 ± 0.5%, r

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Azahari, Nuraniza, Nor Azwani Mohd-Shukri та Muhammad Muzaffar Ali Khan Khattak. "Adipocytes containing adipokines causes β-cell regeneration in previously streptozotocin induced hyperglycemic rats model". Advances in Obesity, Weight Management & Control 10, № 2 (2021): 33–38. http://dx.doi.org/10.15406/aowmc.2020.10.00302.

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Introduction:The aim of this study was to evaluate the effect protein containing adiponectin from the adipocytes on the regeneration of pancreatic β-cells in rates previously treated with (STZ). Therefore, adipocytes from different sources were extracted for protein (adipokines) and the effect was evaluated on pancreatic β-cell regeneration. Methods: The protein extracts from trimmed off abdominal adipose tissues of meat sources, namely chicken, beef and lamb, were used in the present study. The fats were removed, and protein were isolated, and hydrolysate were prepared and injected to rats. T

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Movassat, Jamileh, та Bernard Portha. "Early administration of keratinocyte growth factor improves β-cell regeneration in rat with streptozotocin-induced diabetes". Journal of Endocrinology 195, № 2 (2007): 333–40. http://dx.doi.org/10.1677/joe-07-0098.

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The aim of our study was to investigate the ability of keratinocyte growth factor (KGF; palifermin) in regulating β-cell growth in normal newborn rats and in rats with neonatal diabetes. Wistar rats were injected with streptozotocin (STZ) to induce diabetes on the dayof birth. From days 2 to 6 after birth, animals received a daily s.c. injection of KGF (STZ/KGF group) and at the dose of 3 mg/kg body weight or saline solution (STZ groups). A group of non-diabetic Wistar rats was treated either with saline (Wistar group) or with KGF from days 2 to 6 after birth at the dose of 3 mg/kg body weight

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Dissertations / Theses on the topic "Pancreas β-cells regeneration"

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Shamsi, Farnaz. "Pancreatic β-Cell Regeneration in TIF-IA Knockout Mice". Doctoral thesis, 2014. http://hdl.handle.net/11858/00-1735-0000-0023-98E8-9.

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Book chapters on the topic "Pancreas β-cells regeneration"

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Maulis, Matthew, and Roberto Gianani. "β-Cell Regeneration in Human Pancreas." In Advances in Experimental Medicine and Biology. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-5441-0_23.

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Avolio, Fabio, Anja Pfeifer, Monica Courtney, et al. "From Pancreas Morphogenesis to β-Cell Regeneration." In Current Topics in Developmental Biology. Elsevier, 2013. http://dx.doi.org/10.1016/b978-0-12-416021-7.00006-7.

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de Vries, Rick, Adam Stell, Sami Mohammed, et al. "Bioengineering, biomaterials, and β-cell replacement therapy." In Transplantation, Bioengineering, and Regeneration of the Endocrine Pancreas. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-814831-0.00033-6.

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