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Journal articles on the topic 'Pathogenetic mechanisms'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Makhmudovich, Asadullayev Maksud, Rakhimbayeva Gulnara Sattarovna, Vakhabova Nargiza Maksudovna, and Jangirov Shukhrat Azamatovich. "Hyperhomocysteinemia And Pathogenetic Mechanisms Of Ischemic Stroke." American Journal of Medical Sciences and Pharmaceutical Research 03, no. 02 (2021): 66–76. http://dx.doi.org/10.37547/tajmspr/volume03issue02-10.

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The article is intended to give basic information and the role of homocysteine in the human body. The amino acid homocysteine is a product of methionine demethylation. When the level of homocysteine increases, it damages the tissue structures of the arteries, initiating the release of cytokines, cyclins and other inflammatory mediators. Its accumulation leads to loosening of the arterial walls, the formation of local defects in the endothelium, which, in turn, leads to the deposition of cholesterol and calcium on the vascular wall. Hyperhomocysteinemia as a consequence of impaired homocysteine

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Armanini, Decio, Lorenzo Calò, and Andrea Semplicini. "Pseudohyperaldosteronism: Pathogenetic Mechanisms." Critical Reviews in Clinical Laboratory Sciences 40, no. 3 (2003): 295–335. http://dx.doi.org/10.1080/713609355.

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3

Wollenberg, A., S. Kraft, T. Oppel, and T. Bieber. "Atopic dermatitis: pathogenetic mechanisms." Clinical and Experimental Dermatology 25, no. 7 (2000): 530–34. http://dx.doi.org/10.1046/j.1365-2230.2000.00699.x.

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4

Trubitsyna, I., T. Tarasova, L. Vinokurova, and A. Smirnova. "Pathogenetic mechanisms acute pancreatitis." Pancreatology 13, no. 3 (2013): S56. http://dx.doi.org/10.1016/j.pan.2013.04.190.

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5

Cottier, H., J. Hodler, and R. Kraft. "Oxidative Stress: Pathogenetic Mechanisms." Complementary Medicine Research 2, no. 5 (1995): 233–39. http://dx.doi.org/10.1159/000210155.

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6

Tanashyan, M. M., K. V. Antonova, N. E. Spryshkov, A. А. Panina, O. V. Lagoda, and E. P. Shchukina. "Diabetic Polyneuropathy: from Pathogenesis to Pathogenetic Therapy." Effective Pharmacotherapy 20, no. 8 (2024): 54–62. http://dx.doi.org/10.33978/2307-3586-2024-20-8-54-62.

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The growing worldwide prevalence of prediabetes and diabetes mellitus (DM) has led to an increase in the incidence of related chronic complications, the most common of which is diabetic polyneuropathy (DPN). The development of DPN is caused by chronic hyperglycemia and potentially modifiable cardiovascular risk factors, including elevated triglyceride levels, body mass index, smoking and hypertension. Social determinants of health are also risk factors for DPN. The review presents data on the pathophysiology of DPN with consideration of the causes of the sequence of symptoms. Discussed the mec

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7

Petrovič, Daniel. "Candidate Genes for Proliferative Diabetic Retinopathy." BioMed Research International 2013 (2013): 1–9. http://dx.doi.org/10.1155/2013/540416.

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Several candidate genes have been so far implicated in the pathogenesis of proliferative diabetic retinopathy (PDR) in subjects with type 2 diabetes. Since the principal pathogenetic mechanisms for diabetic retinopathy (DR) and PDR are different, the main pathogenetic mechanism in DR is increased vascular permeability, whereas in PDR the crucial pathogenetic mechanisms are fibrosis and neoangiogenesis. Due to that fact, different candidate genes are expected to be involved in the development of either DR or PDR. None of the candidate genes, however, can be fully and solely responsible for the

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8

Chumakova, G. A., A. V. Ott, N. G. Veselovskaya, O. V. Gritsenko, and N. N. Shenkova. "PATHOGENETIC MECHANISMS OF LEPTIN RESISTANCE." Russian journal of Cardiology, no. 4 (November 27, 2015): 107. http://dx.doi.org/10.15829/1560-4071-2015-4-107-110.

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9

Cooke, T. D. V. "Pathogenetic Mechanisms in Polyarticular Osteoarthritis." Clinics in Rheumatic Diseases 11, no. 2 (1985): 203–38. http://dx.doi.org/10.1016/s0307-742x(21)00540-3.

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10

Schena, Francesco P., and Loreto Gesualdo. "Pathogenetic Mechanisms of Diabetic Nephropathy." Journal of the American Society of Nephrology 16, no. 3 suppl 1 (2005): S30—S33. http://dx.doi.org/10.1681/asn.2004110970.

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11

Shi, Jing. "Pathogenetic mechanisms in gastric cancer." World Journal of Gastroenterology 20, no. 38 (2014): 13804. http://dx.doi.org/10.3748/wjg.v20.i38.13804.

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12

Haussinger, D., and F. Schliess. "Pathogenetic mechanisms of hepatic encephalopathy." Gut 57, no. 8 (2008): 1156–65. http://dx.doi.org/10.1136/gut.2007.122176.

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13

Epstein, Franklin H., and Joseph E. Parrillo. "Pathogenetic Mechanisms of Septic Shock." New England Journal of Medicine 328, no. 20 (1993): 1471–77. http://dx.doi.org/10.1056/nejm199305203282008.

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14

Chierchia, Sergio. "Pathogenetic Mechanisms of Coronary Vasospasm." Acta Medica Scandinavica 211, S660 (2009): 49–56. http://dx.doi.org/10.1111/j.0954-6820.1982.tb00360.x.

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15

Einfeld, S., J. Götz, and R. M. D. Holsinger. "Pathogenetic mechanisms in Down Syndrome." Journal of Intellectual Disability Research 52, no. 10 (2008): 813. http://dx.doi.org/10.1111/j.1365-2788.2008.01119_7.x.

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16

Yarnold, John, and Marie-Catherine Vozenin Brotons. "Pathogenetic mechanisms in radiation fibrosis." Radiotherapy and Oncology 97, no. 1 (2010): 149–61. http://dx.doi.org/10.1016/j.radonc.2010.09.002.

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17

Stefanini, Mario. "Pathogenetic Mechanisms in Thrombocytopenic States." Acta Medica Scandinavica 154, S312 (2009): 500–518. http://dx.doi.org/10.1111/j.0954-6820.1956.tb17050.x.

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18

Di Mario, U. "Pathogenetic mechanisms of diabetic microangiopathy." International Congress Series 1253 (August 2003): 171–82. http://dx.doi.org/10.1016/s0531-5131(03)00145-6.

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19

Moser, Hugo W. "Pathogenetic mechanisms in peroxisomal disorders." Current Opinion in Neurology 9, no. 6 (1996): 473–76. http://dx.doi.org/10.1097/00019052-199612000-00014.

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20

Parnetti, Lucilla, Daniela Mari, Patrizia Mecocci, and Umberto Senin. "Pathogenetic mechanisms in vascular dementia." International Journal of Clinical & Laboratory Research 24, no. 1 (1994): 15–22. http://dx.doi.org/10.1007/bf02592404.

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21

Antoniadis, Antonios P., Yiannis S. Chatzizisis, and George D. Giannoglou. "Pathogenetic mechanisms of coronary ectasia." International Journal of Cardiology 130, no. 3 (2008): 335–43. http://dx.doi.org/10.1016/j.ijcard.2008.05.071.

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22

Kazhzhanova, A. M. Kazhzhanova, and L. A. Yusupova Yusupova. "Pathogenetic mechanisms of alopecia areata." Pharmateca 8_2023 (October 2, 2023): 47–51. http://dx.doi.org/10.18565/pharmateca.2023.8.47-51.

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23

Новиков, Василий Семенович, Игорь Владимирович Дмитриев, and Александр Евдокимович Доросевич. "Pathogenetic Mechanisms and Classification of Shock Types." ВЕСТНИК ОБРАЗОВАНИЯ И РАЗВИТИЯ НАУКИ РОССИЙСКОЙ АКАДЕМИИ ЕСТЕСТВЕННЫХ НАУК, no. 1 (March 15, 2021): 17–29. http://dx.doi.org/10.26163/raen.2021.30.35.002.

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В статье анализируются патогенез шока, основные виды и их классификация. Существенное внимание уделено особенностям видов шока в клинической практике. Представлена характеристика наиболее значимых видов шока. Выделены отдельные виды шока по этиологическому принципу с учетом их патогенетических механизмов. Отмечена важность своевременной диагностики вида шока для патогенетического лечения и предупреждения возможных осложнений. We analyze the pathogenesis of shock, its main types and their classification. Special attention is paid to the features of shock types in clinical practice. A characteri

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24

Belous, S. S., B. A. Vykova, I. S. Anosov, T. L. Aleksandrov, P. I. Chupina, and M. V. Korgunova. "Pathogenetic mechanisms of penetrating Crohn’s disease (review)." Koloproktologia 23, no. 4 (2024): 139–47. http://dx.doi.org/10.33878/2073-7556-2024-23-4-139-147.

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The review evaluates pathogenesis of intestinal fistulas in Crohn’s disease (CD). An idea of the possible contribution of the microbiological and genetic factor is given. The possible effect of matrix metalloproteinases and their tissue inhibitors also assessed. One of the most promising areas which can explain development of intestinal fistulas in CD is epithelial-mesenchymal transition. Further research is required to identify the cause of the complicated CD, which in future will develop correct approaches to pathogenetic therapy.

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25

Signorile, Pietro G., Alfonso Baldi, Rosa Viceconte, and Mariarosaria Boccellino. "The Role of Adenogenesis Factors in the Pathogenesis of Endometriosis." International Journal of Molecular Sciences 26, no. 5 (2025): 2076. https://doi.org/10.3390/ijms26052076.

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Endometriosis is a pathological condition characterized by the presence of the endometrial tissue, outside the uterine cavity. It affects nearly 10% of women of reproductive age and is responsible for infertility, chronic pain, and the weakening of the quality of life. Various pathogenetic mechanisms have been suggested; however, the essential pathogenesis of endometriosis remains insufficiently comprehended. A comprehensive literature search was conducted in databases such as PubMed, Scopus, and Web of Science up to December 2024. Inclusion criteria encompassed studies investigating the patho

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26

MUNASIPOVA, S. E., Z. A. ZALYALOVA, and A. A. TEREKHOVA. "Etiological and pathogenetic mechanisms of forming muscle dystonias." Practical medicine 21, no. 3 (2023): 24–31. http://dx.doi.org/10.32000/2072-1757-2023-3-24-31.

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This article discusses the issues of muscular dystonia pathogenesis that are relevant to modern medicine, as well as the possibility of using the pathogenetic concept as a substrate to supplement the classification criteria. Reviewing the international publications on etiopathophysiology, genetics and classification, the article summarizes the main mechanisms for the occurrence of a pathological process at different levels of the nervous system. Despite the frequent clinical similarity of the symptoms of various forms of muscular dystonia, these symptoms may be the result of dysfunction of com

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27

Dastur, Darab K., Daya K. Manghani, and P. M. Udani. "PATHOLOGY AND PATHOGENETIC MECHANISMS IN NEUROTUBERCULOSIS." Radiologic Clinics of North America 33, no. 4 (1995): 733–52. http://dx.doi.org/10.1016/s0033-8389(22)00616-9.

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28

Salvaggio, John E., and Carol E. O’Neil. "PATHOGENETIC MECHANISMS IN OCCUPATIONAL HYPERSENSITIVITY STATES." Immunology and Allergy Clinics of North America 12, no. 4 (1992): 711–29. http://dx.doi.org/10.1016/s0889-8561(22)00142-4.

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29

Pushkaryova, L. D., and I. V. Zapuskalov. "Pathogenetic mechanisms of Valsalva retinopathy development." Bulletin of Siberian Medicine 10, no. 4 (2011): 99–102. http://dx.doi.org/10.20538/1682-0363-2011-4-99-102.

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The purpose of this review was to analyze existing data in the literature research on some pathogenetic mechanisms of the Valsalva retinopathy development. The main causes of retinal vessel damage of vascular are described. They include arterial pressure and central venous pressure elevation. The latter is transmitted to retinal veins and may cause retinal capillar wall ruptur.

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30

Ostroumova, O. D., E. S. Akimova, and A. I. Kochetkov. "Drug-induced acute pancreatitis: pathogenetic mechanisms." Siberian Medical Review, no. 3 (2021): 35–43. http://dx.doi.org/10.20333/25000136-2021-3-35-43.

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Drug-induced (DI) acute pancreatitis (AP) is a difficultly diagnosed disease amounting to 2% among all AP cases that may lead to development of life-threatening complications. This article considers potentially hazardous medicinal products as well as the most significant variants of the pancreas: structural and functional impairment of the pancreatic duct, direct cytotoxic effects, metabolic disorders, vascular disorders, immune-mediated damage to the pancreas and hypersensitivity. The review was performed based on the publications present in the E-library and PubMed databases dated 1965-2020.

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31

FADEEV, GRIGORIY A., NIKOLAY A. TSIBULKIN, OLGA Y. MIKHOPAROVA, and GALINA V. TUKHVATULLINA. "PATHOGENETIC MECHANISMS OF ALCOHOLIC CARDIOMYOPATHY DEVELOPMENT." Bulletin of Contemporary Clinical Medicine 12, no. 4 (2019): 74–80. http://dx.doi.org/10.20969/vskm.2019.12(4).74-80.

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32

Micci, F., and S. Heim. "Pathogenetic mechanisms in endometrial stromal sarcoma." Cytogenetic and Genome Research 118, no. 2-4 (2007): 190–95. http://dx.doi.org/10.1159/000108300.

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33

Simons, J. P., R. Al-Shawi, S. Ellmerich, et al. "Pathogenetic mechanisms of amyloid A amyloidosis." Proceedings of the National Academy of Sciences 110, no. 40 (2013): 16115–20. http://dx.doi.org/10.1073/pnas.1306621110.

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34

Tosti, Claudia, Serena Pinzauti, Pietro Santulli, Charles Chapron, and Felice Petraglia. "Pathogenetic Mechanisms of Deep Infiltrating Endometriosis." Reproductive Sciences 22, no. 9 (2015): 1053–59. http://dx.doi.org/10.1177/1933719115592713.

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35

Donati, Maria Benedetta, and Anna Falanga. "Pathogenetic Mechanisms of Thrombosis in Malignancy." Acta Haematologica 106, no. 1-2 (2001): 18–24. http://dx.doi.org/10.1159/000046585.

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36

Lopez, Pedro F., Hans E. Grossniklaus, Thomas M. Aaberg, Paul Sternberg, Antonio Capone, and H. Michael Lambert. "Pathogenetic Mechanisms in Anterior Proliferative Vitreoretinopathy." American Journal of Ophthalmology 114, no. 3 (1992): 257–79. http://dx.doi.org/10.1016/s0002-9394(14)71790-8.

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37

Šedý, Jiří, Jaroslav Kuneš, and Josef Zicha. "Pathogenetic Mechanisms of Neurogenic Pulmonary Edema." Journal of Neurotrauma 32, no. 15 (2015): 1135–45. http://dx.doi.org/10.1089/neu.2014.3609.

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38

Oppegaard, Oddvar. "Unravelling pathogenetic mechanisms of epidemic lineages." Virulence 8, no. 7 (2017): 1102–4. http://dx.doi.org/10.1080/21505594.2017.1328344.

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39

Kurbanov, Oybek, Nodir Nurmetov, Zarifboy Ibodullaev, and Dilshoda Abdullaeva. "Pathogenetic mechanisms in epileptic myoclonic hyperkinesia." Parkinsonism & Related Disorders 22 (January 2016): e132. http://dx.doi.org/10.1016/j.parkreldis.2015.10.316.

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40

DIETZ, H. "Pathogenetic Mechanisms in the Marfan Syndrome." American Journal of Hypertension 9, no. 4 (1996): 179A. http://dx.doi.org/10.1016/0895-7061(96)82089-9.

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41

Maseri, Attilio, and Juan Carlos Kaski. "Pathogenetic mechanisms of coronary artery spasm." Journal of the American College of Cardiology 14, no. 3 (1989): 610–12. http://dx.doi.org/10.1016/0735-1097(89)90101-0.

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42

Rohde, Henning. "The pathogenetic mechanisms causing anal fissure." International Journal of Colorectal Disease 18, no. 1 (2003): 95. http://dx.doi.org/10.1007/s00384-002-0438-z.

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43

Kotiuzhynska, S. H., and D. O. Umanskyy. "ATEROSCLEROSIS AND INFLAMMATION: GENERAL PATHOGENETIC MECHANISMS." PRECARPATHIAN BULLETIN OF THE SHEVCHENKO SCIENTIFIC SOCIETY Pulse, no. 6(58) (December 26, 2019): 31–37. http://dx.doi.org/10.21802/10.21802/2304-7437-2019-6(58)-31-37.

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According to modern notions, atherosclerosis is a complex multifactorial process in which the mechanisms of development involved endothelial dysfunction, inflammation, dyslipidemia, disorders of the blood coagulation system, and the like. Polyunsaturated fatty acids are a source of biologically active substances that regulate metabolic processes in the body. It is known that during the development of inflammation, biologically active agents are synthesized and accumulated, which both provide a protective function, and play the role of triggers or lead to the development of additional alteratio

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44

Kotiuzhynska, S. H., and D. O. Umanskyy. "ATEROSCLEROSIS AND INFLAMMATION: GENERAL PATHOGENETIC MECHANISMS." PRECARPATHIAN BULLETIN OF THE SHEVCHENKO SCIENTIFIC SOCIETY Pulse, no. 6(58) (December 26, 2019): 31–37. http://dx.doi.org/10.21802/2304-7437-2019-6(58)-31-37.

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According to modern notions, atherosclerosis is a complex multifactorial process in which the mechanisms of development involved endothelial dysfunction, inflammation, dyslipidemia, disorders of the blood coagulation system, and the like. Polyunsaturated fatty acids are a source of biologically active substances that regulate metabolic processes in the body. It is known that during the development of inflammation, biologically active agents are synthesized and accumulated, which both provide a protective function, and play the role of triggers or lead to the development of additional alteratio

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45

Marin-Valencia, Isaac, Renzo Guerrini, and Joseph G. Gleeson. "Pathogenetic mechanisms of focal cortical dysplasia." Epilepsia 55, no. 7 (2014): 970–78. http://dx.doi.org/10.1111/epi.12650.

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46

Iannaccone, Philip M., and Dante G. Scarpelli. "Exploring Pathogenetic Mechanisms Using Transgenic Animals." Annals of Medicine 25, no. 2 (1993): 131–38. http://dx.doi.org/10.3109/07853899309164155.

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47

PERROS, P., and P. KENDALL-TAYLOR. "Pathogenetic mechanisms in thyroid-associated ophthalmopathy." Journal of Internal Medicine 231, no. 3 (1992): 205–11. http://dx.doi.org/10.1111/j.1365-2796.1992.tb00525.x.

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48

Botirbekov Abdurashid Nazarkulovich. "Modern Pathogenetic Mechanisms of Systemic Scleroderma." Texas Journal of Medical Science 26 (November 20, 2023): 108–12. http://dx.doi.org/10.62480/tjms.2023.vol26.pp108-112.

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Systemic scleroderma is a chronic and often progressive autoimmune inflammation of the connective tissue, characterized by fibrosis of the skin and internal organs, widespread vascular lesions with unknown etiology, and heterogeneous clinical manifestations. Systemic scleroderma is a violation of the synthesis and accumulation of collagen in the skin (scleroderma) and other internal organs, especially in the connective tissue of the lungs, gastrointestinal tract (gastrointestinal tract), heart and kidneys. Over time, patients develop signs of a progressive structural and functional impairment

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49

Voutidou, Styliani, Dimitrios Eleftheriadis, Fotios Drakopanagiotakis, Ilias C. Papanikolaou, and Paschalis Steiropoulos. "Pathogenetic Mechanisms Linking Sarcoidosis to Lymphoma." International Journal of Molecular Sciences 26, no. 2 (2025): 594. https://doi.org/10.3390/ijms26020594.

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Sarcoidosis and lymphoma share immunopathological characteristics that suggest a complex, interconnected relationship. This article examines the multi-faceted mechanisms linking sarcoidosis to lymphoma, a phenomenon called sarcoidosis-lymphoma syndrome (SLS). SLS is hard to diagnose, requiring distinct criteria and imaging to differentiate overlapping features and histological differences. The co-occurrence of these diseases may be explained by genetic predispositions, immune dysregulation, and environmental factors that enhance malignancy risk. In active sarcoidosis, chronic inflammation and

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50

Fedotov, Ilya A., and Dmitriy I. Shustov. "Substance-induced schizophrenia: possible pathogenetic mechanisms." Neurology Bulletin LVII, no. 1 (2025): 46–53. https://doi.org/10.17816/nb641882.

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This brief report presents a narrative literature review describing potential pathogenetic mechanisms underlying the transformation of drug-induced psychoses into chronic psychotic disorders of the schizophrenic spectrum. The central role of hyperdopaminergia in the development of productive psychotic symptoms is discussed, along with the possibility of dopamine D2 receptor sensitization resulting from prolonged use of stimulant drugs. Attention is drawn to the previously described phenomenon of reverse tolerance, the study of which at genetic, molecular, neurotransmitter, and clinical levels

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