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Relevant bibliographies by topics / Phosphatidic acid

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Academic literature on the topic 'Phosphatidic acid'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 May 2024

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Journal articles on the topic "Phosphatidic acid"

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ENGLISH, Denis, Margaret MARTIN, Kevin A. HARVEY, Luke P. AKARD, Ruth ALLEN, Theodore S. WIDLANSKI, Joe G. N. GARCIA, and Rafat A. SIDDIQUI. "Characterization and purification of neutrophil ecto-phosphatidic acid phosphohydrolase." Biochemical Journal 324, no. 3 (June 15, 1997): 941–50. http://dx.doi.org/10.1042/bj3240941.

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Phosphatidic acid and its derivatives play potentially important roles as extracellular messengers in biological systems. An ecto-phosphatidic acid phosphohydrolase (ecto-PAPase) has been identified which effectively regulates neutrophil responses to exogenous phosphatidic acid by converting the substrate to diacylglycerol. The present study was undertaken to characterize this ecto-enzyme on intact cells and to isolate the enzyme from solubilized neutrophil extracts. In the absence of detergent, short chain phosphatidic acids were hydrolysed most effectively by neutrophil plasma membrane ecto-

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Kaszkin, M., L. Seidler, R. Kast, and V. Kinzel. "Epidermal-growth-factor-induced production of phosphatidylalcohols by HeLa cells and A431 cells through activation of phospholipase D." Biochemical Journal 287, no. 1 (October 1, 1992): 51–57. http://dx.doi.org/10.1042/bj2870051.

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In response to epidermal growth factor (EGF), HeLa cells and A431 cells rapidly accumulate substantial amounts of phosphatidic acid (up to 0.16 and 0.2 micrograms/10(6) cells respectively), which represents approx. 0.17% of total phospholipid. Phosphatidic acid may be a potential product of diacylglycerol kinase and/or of phospholipase D. To evaluate the contribution of phospholipase D, the phosphatidyl-transfer reaction to a primary alcohol (mostly butan-1-ol; 0.2%) was measured; this reaction is known to be mediated exclusively by phospholipase D in intact cells. In HeLa and in A431 cells pr

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Altin, J. G., and F. L. Bygrave. "Phosphatidic acid and arachidonic acid each interact synergistically with glucagon to stimulate Ca2+ influx in the perfused rat liver." Biochemical Journal 247, no. 3 (November 1, 1987): 613–19. http://dx.doi.org/10.1042/bj2470613.

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The administration of phosphatidic acid to rat livers perfused with media containing either 1.3 mM- or 10 microM-Ca2+ was followed by a stimulation of Ca2+ efflux, O2 uptake and glucose output. The responses elicited by 100 microM-phosphatidic acid were similar to those induced by the alpha-adrenergic agonist phenylephrine. Contrary to suggestions that phosphatidic acid acts like a Ca2+-ionophore, no net influx of Ca2+ was detected until the phosphatidic acid was removed. Sequential infusions of phenylephrine and phosphatidic acid indicate that the two agents release Ca2+ from the same intrace

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Raben, Daniel M., and Casey N. Barber. "Phosphatidic acid and neurotransmission." Advances in Biological Regulation 63 (January 2017): 15–21. http://dx.doi.org/10.1016/j.jbior.2016.09.004.

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Fraser, T., A. Waters, S. Chatrattanakunchai, and K. Stobart. "Does triacylglycerol biosynthesis require diacylglycerol acyltransferase (DAGAT)?" Biochemical Society Transactions 28, no. 6 (December 1, 2000): 698–700. http://dx.doi.org/10.1042/bst0280698.

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Microsomal membrane preparations from the developing seeds of sunflower (Helianthus annuus L.) catalyse the conversion of sn-glycerol-3-phosphate and acyl-CoA to triacylglycerol via phosphatidic acid and diacylglycerol. The formation of diacylglycerol from phosphatidic acid was Mg2+ dependent and in the presence of EDTA phosphatidic acid accumulated. This property was used to generate large quantities of endogenous radioactive phosphatidic acid in the membranes. On addition of Mg2+ the phosphatidic acid was used in triacylglycerol formation. Acyl-CoA had little effect on the label which accumu

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Bodachivska, L. Yu. "Biodegradable surfactants from side streams of the vegetable oils production in technical systems." Voprosy Khimii i Khimicheskoi Tekhnologii, no. 6 (December 2022): 3–11. http://dx.doi.org/10.32434/0321-4095-2022-145-6-3-11.

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This work reports the structure of surfactants synthesized from by-products of the vegetable oil production. These are raw materials that do not compete with food products; they are low-cost phosphatidic sludge that can be used directly for chemical transformation. Fatty acid monoetanolamides derived from side streams of the vegetable oils production do not have residues of the original phosphatides or acylglycerols as determined by spectral methods. There are clearly reflected cross-peaks between the amide group and the adjacent methylene group. This indicates a high conversion of substrate a

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Andringa, Ruben L. H., Marijn Jonker, and Adriaan J. Minnaard. "Synthesis of phosphatidic acids via cobalt(salen) catalyzed epoxide ring-opening with dibenzyl phosphate." Organic & Biomolecular Chemistry 20, no. 11 (2022): 2200–2204. http://dx.doi.org/10.1039/d2ob00168c.

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Sakane, Fumio, Fumi Hoshino, and Chiaki Murakami. "New Era of Diacylglycerol Kinase, Phosphatidic Acid and Phosphatidic Acid-Binding Protein." International Journal of Molecular Sciences 21, no. 18 (September 16, 2020): 6794. http://dx.doi.org/10.3390/ijms21186794.

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Diacylglycerol kinase (DGK) phosphorylates diacylglycerol (DG) to generate phosphatidic acid (PA). Mammalian DGK consists of ten isozymes (α–κ) and governs a wide range of physiological and pathological events, including immune responses, neuronal networking, bipolar disorder, obsessive-compulsive disorder, fragile X syndrome, cancer, and type 2 diabetes. DG and PA comprise diverse molecular species that have different acyl chains at the sn-1 and sn-2 positions. Because the DGK activity is essential for phosphatidylinositol turnover, which exclusively produces 1-stearoyl-2-arachidonoyl-DG, it

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Kolesnikov, Y. S., S. V. Kretynin, V. S. Kravets, and Y. K. Bukhonska. "Phosphatidic acid formation and signaling in plant cells." Ukrainian Biochemical Journal 96, no. 1 (February 23, 2024): 5–21. http://dx.doi.org/10.15407/ubj96.01.005.

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This review conteins updated information on the structure, localization and regulation of phosphatidic acid (PA)-producing enzymes phospholipase D, phosphoinositide-specific and non-specific phospholipases C and diacylglycerol kinases is analyzed. The specific role of PA and PA-producing enzymes in plant stress signaling is discussed.

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Singh, Ram Raj, Priti Prasad, Cynthia Tran, and Ramesh Halder. "A self-glycerophospholipid suppresses immunity and inflammation via recruitment of Ly6C+ myeloid cells." Journal of Immunology 198, no. 1_Supplement (May 1, 2017): 221.19. http://dx.doi.org/10.4049/jimmunol.198.supp.221.19.

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Abstract Membrane lipids function as essential components of biological membranes, as signaling molecules, and as energy storage molecules. Phosphatidic acid is a vital membrane lipid that serves as a precursor for the synthesis of all acylglycerol lipids in the cell. Phosphatidic acid participates in a wide range of cellular processes, including cytoskeletal organization, secretion, endocytosis, and cell proliferation. Here, we examined the effect of phosphatidic acid on myeloid cells and its ability to modulate tumor immunity and inflammatory disease. We found that in vivo administration of

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Dissertations / Theses on the topic "Phosphatidic acid"

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Lee, Sue Chin. "Spatial signalling of phosphatidic acid." Thesis, University of Strathclyde, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.510804.

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Fleming, Ian Neil. "Purification and properties of phosphatidic acid phosphohydrolase." Thesis, University of Newcastle Upon Tyne, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260059.

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Stace, Catherine Lucy. "Functional studies of phosphatidic acid production by phospholipase D1." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612984.

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Jakobson, Katherine. "The role of phosphatidic acid in astrocyte intracellular signalling." Thesis, Open University, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293615.

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Han, Eun Hyang. "Phosphatidic Acid Mediation of Environmentally Induced Adaptive Growth Responses." The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1492678342062212.

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Buckland, Andrew G. "Anionic phospholipids, annexins and the activity of phospholipases A2." Thesis, University of Southampton, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246259.

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Heyward, Catherine Anne. "Investigation of potential phosphatidic acid target proteins in intracellular signalling pathways." Thesis, Birmingham City University, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.479113.

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Williams, David. "Phosphatidic Acid Increases Lean Body Tissue and Strength In Resistance Trained Men." Master's thesis, University of Central Florida, 2012. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/5575.

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Phosphatidic Acid (PA) is a natural phospholipid compound derived from lecithin which is commonly found in egg yolk, grains, fish, soybeans, peanuts and yeast. It has been suggested that PA is involved in several intracellular processes associated with muscle hypertrophy. Specifically, PA has been reported to activate protein synthesis through the mammalian target of rapamycin (mTOR) signaling pathway and thereby may enhance the anabolic effects of resistance training. To our knowledge, no one has examined the effect of PA supplementation in humans while undergoing a progressive resistance t

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Yu, Changhua. "Phospholipase D/phosphatidic acid phosphatase signal transduction pathway in post-infarction congestive heart failure." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq23565.pdf.

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Urs, Aarti N. "Reciprocal binding of sphingosine and phosphatidic acid to steroidogenic factor 1 regulates the transcription of CYP17." Thesis, Available online, Georgia Institute of Technology, 2005, 2005. http://etd.gatech.edu/theses/available/etd-11212005-102620/.

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Thesis (M. S.)--Biology, Georgia Institute of Technology, 2006.<br>Donald Doyle, Committee Member ; Harish Radhakrishna, Committee Member ; Alfred Merrill, Committee Member ; Marion Sewer, Committee Chair Includes bibliographical references.

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Books on the topic "Phosphatidic acid"

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Attiogbevi-Somado, Eklou. The use of phosphate rock in a rice-legume rotation system on acid soil in the humid forest zone of West Africa. Göttingen: Goltze, 2000.

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Yang, Lu Ying *. A comparative study of triacylglycerol biosynthesis via the monocylglyerol and phosphatidic acid pathways in rat small intestine. 1991.

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Book chapters on the topic "Phosphatidic acid"

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Bocckino, Stephen B., and John H. Exton. "Phosphatidic Acid." In Lipid Second Messengers, 75–123. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-1361-6_3.

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Liu, Yu, Yuan Su, and Xuemin Wang. "Phosphatidic Acid-Mediated Signaling." In Advances in Experimental Medicine and Biology, 159–76. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6331-9_9.

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Yin, C. C., B. Z. Lin, and H. Hauser. "Phase Behavior of Phosphatidic Acid." In Progress in Membrane Biotechnology, 30–39. Basel: Birkhäuser Basel, 1991. http://dx.doi.org/10.1007/978-3-0348-7454-0_3.

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Nakamura, Yuki, and Hiroyuki Ohta. "Phosphatidic Acid Phosphatases in Seed Plants." In Lipid Signaling in Plants, 131–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03873-0_9.

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Nakamura, Yuki. "Assaying Plant Phosphatidic Acid Phosphatase Activity." In Methods in Molecular Biology, 233–40. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-401-2_21.

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Ischebeck, Till. "Phosphatidic Acid in Plants: Functional Diversity." In Encyclopedia of Lipidomics, 1–4. Dordrecht: Springer Netherlands, 2017. http://dx.doi.org/10.1007/978-94-007-7864-1_148-1.

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Kooijman, Edgar Eduard, and Christa Testerink. "Phosphatidic Acid: An Electrostatic/Hydrogen-Bond Switch?" In Lipid Signaling in Plants, 203–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03873-0_14.

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Distéfano, Ayelen M., M. Luciana Lanteri, Arjen ten Have, Carlos García-Mata, Lorenzo Lamattina, and Ana M. Laxalt. "Nitric Oxide and Phosphatidic Acid Signaling in Plants." In Lipid Signaling in Plants, 223–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03873-0_15.

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Fleming, Ian N., and Stephen J. Yeaman. "Phosphatidic Acid Phosphohydrolase: Its Role in Cell Signalling." In Signalling Mechanisms — from Transcription Factors to Oxidative Stress, 57–64. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79675-3_6.

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Wang, Xuemin, Yuan Su, Yu Liu, Sang-Chul Kim, and Brian Fanella. "Phosphatidic Acid as Lipid Messenger and Growth Regulators in Plants." In Signaling and Communication in Plants, 69–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-42011-5_4.

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Conference papers on the topic "Phosphatidic acid"

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Sokornova, S. V., A. L. Shavarda, E. A. Gusenkov, D. A. Emelianov, and G. M. Frolova. "Biochemical features of phoma-like fungi." In 2nd International Scientific Conference "Plants and Microbes: the Future of Biotechnology". PLAMIC2020 Organizing committee, 2020. http://dx.doi.org/10.28983/plamic2020.233.

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Biochemical analysis of phoma-like fungi showed significant differences in the phosphatidic acid and glycoceramides levels and close values of phosphatidylcholine / phosphatidylethanolamine, trehalose, arabitol, mannitol, sorbitol levels.

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Cascar, J. M., and J. L. Kavarro. "ARACHIDGIIC ACID METABOLISM II PLATELETS STORED FOR FIVE DAYS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644685.

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Arachidonlc acid (AA) netabolisn has been extensively studied in fresh platelets, but there Is little infomation available for stored platelets . we stored platelets in CLI bags far five days at 22+/-2°C and, on days 0, 3 and 5, six si of platelet concentrate were revved from the container and platelets were labeled with (C-14J-AA. Both incorporation and distribution of radiotracer sere studied in rest and thrcnfcln stivlated platelets.Total uptake of, (C-14)-AA dropped fron day 0 to 5 (p 0.01). Distribution oh day 0 was sinilar to fresh platelets. Incorporation of (C-14)-AA on phosphatidyl in

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Haji, K., S. Sato, H. Ogawa, Y. Isomura, N. Bando, R. Suzue, T. T. Hong Phuong, et al. "The Effect of Novel Autotaxin Inhibitor, 2-carba Cyclic Phosphatidic Acid, in Lung Fibrosis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a2463.

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Setty, B. N. Y., M. Berger, and M. J. Stuart. "13-HYDROXY-9,11-OCTADECADIENOIC ACID (13-HOD) INCREASES PROSTACYCLIN PRODUCTION IN ENDOTHELIAL CELLS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643948.

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Recently, endothelial cells (ECs) have been shown to generate a potent vascular chemorepellant factor. This metabolite, 13-HOD is reported to be the major lipoxygenase product produced in microgram amounts in ECs (JBC 260:16056, 1985). We have studied the effect of 13-HOD on EC arachidonic acid (AA) metabolism, and report modulation of both AA release and conversion to prostacyclin. Using fetal bovine aortic ECs, 13-HOD stimulated prostacyclin production (RIA for 6KPGF1α ) by 40±13% (1SE), and 51±09% at 10 and 30μM (P<0.05; n=5). When the time-course of this effect was evaluated, 13-HOD (30

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Myers, Amanda, Hitham Aldharee, Shimpi Bedi, and Weiwen Long. "Abstract 4308: Phosphatidic acid binds to ERK3 and stimulates phosphorylation of the ERK3 activation loop." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4308.

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Myers, Amanda, Hitham Aldharee, Shimpi Bedi, and Weiwen Long. "Abstract 4308: Phosphatidic acid binds to ERK3 and stimulates phosphorylation of the ERK3 activation loop." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4308.

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Tysnes, O.-B., AJ M. Verhoeven, and H. Holmsen. "Thrombin stimulation of human platelets:Phosphoinositides as the only source of the diacylglycerol moiety in phosphatidic acid." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644505.

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Recent reports on various cell types including platelets have concluded in the heterogenous production of phosphatidic acid (PA) during the stimulus-response coupling. Human platelets were pulse-labelled simultaneously with [32P] P. and [32H]glycerol. Extracts were analyzed for masses and radioactivities of ATP and phosphoinositides. When the cells were stimulated with low concentrations of thrombin, the production of [32P] PA was evident without measurable production of [3H] PA. At higher doses of the agonist,[3H] PA was formed, but distinctly later than [32P] PA.This suggested a heterogenous

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Crouch, Michael F., and Eduardo G. Lapetina. "PHOSPHOLIPASE A2 ACTIVATION BY A MECHANISM SEPARATE TO THAT RESPONSIBLE FOR PHOSPHOLIPASE C STIMULATION IN ALPHA-THROMBIN-STIMULATED HUMAN PLATELETS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644470.

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The ability of cell surface receptor occupation to increase the activity of phospholipase A2 has been thought to be due to the prior activation of phospholipase C and an increase in the intracellular Ca2+ concentration. However, recent evidence from our and other laboratories has suggested that this may not be the case, but rather stimulation of phospholipase A2 may be under the control of separate receptor-activated events. We have investigated this further by comparing the ability of prostacyclin (PGI2) and epinephrine to alter platelet responses to thrombin and examining the resulting phosp

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Pengo, V., M. J. Heine, P. Thiagarajan, and s. s. Shapiro. "A GENERAL MECHANISM FOR LUPUS ANTICOAGULANTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643660.

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Although- a number of observations have implied that lupus anticoagulants have immunologic specificity towards anionic. phospholipids, thereby prolonging phospholipid-dependent coagulation tests, this assumption has been directly demonstrated in only one patient with a monoclonal IgM paraprotein. We have tested the generality of this hypothesis directly by isolating five IgG lupus anticoagulants from patients with lupus-like syndromes and/or thrombosis. IgG lupus anticoagulant fractions were isolated free of other plasma proteins and free of contaminating phospholipid by adsorption to and elut

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Crouch, Michael F., Roger D. Nolan, and Eduardo G. Lapetina. "THROMBIN-INDUCED RELEASE OF INTRA-PLATELET CA2+ STORES IS INHIBITED BY PROSTACYCLIN, BUT GTP- AND IP3-INDUCED RELEASE IS UNAFFECTED." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644515.

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Alpha-thrombin induced the release of internal Ca2+ stores and the influx of Ca2+ in human platelets, as measured by quin-2 fluorescence. This was accompanied by a stimulated formation of inositol phosphates and phosphatidic acid. The Ca2+ responses were inhibited almost totally by pretreatment of cells with prostacyclin (PGI2). Epinephrine was able to restore the influx of Ca2+ from the external medium, but not the alpha-thrombin-induced release of internal Ca2+ stores. This was despite epinephrine restoring phosphatidic acid formation and, at least partially, the generation of inositol trisp

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