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Relevant bibliographies by topics / PI3K-Akt signaling pathway

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Academic literature on the topic 'PI3K-Akt signaling pathway'

Author: Grafiati

Published: 4 June 2021

Last updated: 27 July 2025

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Journal articles on the topic "PI3K-Akt signaling pathway"

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Urasaki, Yasuyo, Cody Beaumont, Jeffery N. Talbot, David K. Hill, and Thuc T. Le. "Akt3 Regulates the Tissue-Specific Response to Copaiba Essential Oil." International Journal of Molecular Sciences 21, no. 8 (2020): 2851. http://dx.doi.org/10.3390/ijms21082851.

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This study reports a relationship between Akt3 expression and tissue-specific regulation of the pI3K/Akt/mTOR signaling pathway by copaiba essential oil. Akt3, a protein kinase B isoform important for the regulation of neuronal development, exhibited differential expression levels in cells of various origins. In neuronal and microglial cells, where Akt3 is present, copaiba essential oil positively regulated the pI3K/Akt/mTOR signaling pathway. In contrast, in liver cells and T lymphocytes, where Akt3 is absent, copaiba essential oil negatively regulated the pI3K/Akt/mTOR signaling pathway. The

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Huang, Shu-ping, Ze-chao Zhang, Yu Chen, Chang-jie Shang, Min Zhu, and Wei-hong Li. "Review of Chinese medicine intervention in PI3K/AKT pathway to regulate fibrosis." Medicine 104, no. 28 (2025): e42957. https://doi.org/10.1097/md.0000000000042957.

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The phosphatidylinositol-3-kinase (PI3K)/protein kinase B (AKT) signaling pathway plays a crucial role in the regulation of fibrosis, a pathological process characterized by excessive deposition of extracellular matrix components leading to tissue scarring and dysfunction. Traditional Chinese medicine (TCM) has been increasingly recognized for its potential therapeutic effects in fibrosis by targeting various signaling pathways, including the PI3K/AKT pathway. This review aims to summarize the recent advancements in TCM interventions targeting the PI3K/AKT signaling pathway for the regulation

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Liu, Jiali, Pangao Xu, Dekun Liu, et al. "TCM Regulates PI3K/Akt Signal Pathway to Intervene Atherosclerotic Cardiovascular Disease." Evidence-Based Complementary and Alternative Medicine 2021 (December 16, 2021): 1–11. http://dx.doi.org/10.1155/2021/4854755.

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Vascular endothelial injury is the initial stage of atherosclerosis (AS). Stimulating and activating the phosphoinositide 3-kinase (PI3K)/protein kinase B (Akt) signaling pathway can regulate the expression of vascular endothelial cytokines, thus affecting the occurrence and development of AS. In addition, the PI3K/Akt signaling pathway can regulate the polarization and survival of macrophages and the expression of inflammatory factors and platelet function, thus influencing the progression of AS. In recent years, traditional Chinese medicine (TCM) has been widely recognized for its advantages

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Sahu, Rakesh, and Bhaskar Sahu. "Inhibitory potential of Nelumbo nucifera Gaertn extract on the PI3K/AKT/mTOR signaling pathway." Cell Signaling 2, no. 1 (2024): 96–101. http://dx.doi.org/10.46439/signaling.2.037.

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Nelumbo nucifera Gaertn (NNG), commonly known as sacred lotus or Indian lotus, has been extensively used in traditional medicine for its various pharmacological properties. Emerging research has focused on elucidating its potential as a therapeutic agent against cancer and other diseases. The phosphoinositide 3-kinase (PI3K)/protein kinase B (AKT)/mammalian target of rapamycin (mTOR) signaling pathway plays a crucial role in regulating cellular processes such as cell growth, proliferation, and survival, and its dysregulation is implicated in various diseases, including cancer. This review exam

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Meng, Xianwei, Jun Cui, and Guibin He. "Bcl-2 Is Involved in Cardiac Hypertrophy through PI3K-Akt Pathway." BioMed Research International 2021 (March 12, 2021): 1–8. http://dx.doi.org/10.1155/2021/6615502.

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Cardiac hypertrophy (CH) is a common cause of sudden cardiac death and heart failure, resulting in a significant medical burden. The present study is aimed at exploring potential CH-related pathways and the key downstream effectors. The gene expression profile of GSE129090 was obtained from the Gene Expression Omnibus database (GEO), and 1325 differentially expressed genes (DEGs) were identified, including 785 upregulated genes and 540 downregulated genes. Kyoto Encyclopedia of Genes and Genomes (KEGG) and Reactome pathway enrichment analysis of DEGs were then performed. Although there were no

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Zhang, Qingling, Qi Hu, Jian Li, and Zhirui Lin. "WTX beyond WNT signaling pathway." Visualized Cancer Medicine 4 (2023): 2. http://dx.doi.org/10.1051/vcm/2022006.

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Gastric cancer (GC) is a major malignancy in many developing countries with low early detection rate. As a tumour suppressor gene, WTX inhibits PI3K/AKT/mTOR pathway activity by inhibiting PI3K phosphorylation. WTX loss of WTX protein associates with tumor metastasis and poor survival of GC patients. During GC progression, an aberrantly elevated miR-20a-5p expression has been found, which inhibits WTX expression and induces PI3K phosphorylation, thereby activating the PI3K/AKT/mTOR pathway and promoting cellular proliferation and migration. A new mechanism in which miR-20a-5p promotes GC progr

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Lin, Kai, Xinghua Wu, Yuying Qi, Kaiyin Wang, Yunzhu Guan, and Tinghui Hu. "Mechanism of Glucose Transporter Protein 1 Mediating Malignant Behavior in Breast Cancer Through the PI3K/Akt Signaling Pathway." Science of Advanced Materials 15, no. 9 (2023): 1218–23. http://dx.doi.org/10.1166/sam.2023.4533.

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To investigate the mechanism of action of GLUT1 in mediating breast cancer development through the PI3K/Akt signaling pathway. Knockdown of breast cancer cell line MDA-MB-231 GLUT1 was achieved by siRNA with the addition of IGF-1, an activator of the PI3K/AKT signaling pathway. The experimental groupings were NC, shGLUT1, shNC+IGF-1, and shGLUT1+IGF-1. The proliferation, invasion, and migration behaviors of breast cancer cells were observed by MTT, Transwell, and scratch-repair assays; Western blot was used to detect the protein expression levels of p-PI3K, p-AKT, and p-mTOR in the PI3K-AKT si

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Li, Xiao, Xu Feng, Chunkang Chang, Qi He, and Wu Lingyun. "Identification of microRNA-Regulated Pathways through a Integration of Mcrorna-mRNA Microarray and Bioinformatics Analysis in CD34+ Cells of Myelodysplastic Syndromes." Blood 124, no. 21 (2014): 3238. http://dx.doi.org/10.1182/blood.v124.21.3238.3238.

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Abstract Background MicroRNAs (miRNAs) are considered to play a key role in the pathogenesis of myelodysplastic syndromes (MDS). However, the effect of miRNA and targeted mRNA on signal transduction is not fully understood in MDS. Objective The objective of this study is to identify the miRNAs-regulated pathways. Methods Affymetrix GeneChip microRNA and PrimeView Array were used to analyze miRNAs and gene expression profile of CD34+ cells in 12 MDS patients and 6 healthy controls. Comprehensive bioinformatics analysis of the coordinate expression of miRNAs and mRNAs including Difference, Go, P

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Pungsrinont, Thanakorn, Julia Kallenbach, and Aria Baniahmad. "Role of PI3K-AKT-mTOR Pathway as a Pro-Survival Signaling and Resistance-Mediating Mechanism to Therapy of Prostate Cancer." International Journal of Molecular Sciences 22, no. 20 (2021): 11088. http://dx.doi.org/10.3390/ijms222011088.

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Androgen deprivation therapy (ADT) and androgen receptor (AR)-targeted therapy are the gold standard options for treating prostate cancer (PCa). These are initially effective, as localized and the early stage of metastatic disease are androgen- and castration-sensitive. The tumor strongly relies on systemic/circulating androgens for activating AR signaling to stimulate growth and progression. However, after a certain point, the tumor will eventually develop a resistant stage, where ADT and AR antagonists are no longer effective. Mechanistically, it seems that the tumor becomes more aggressive

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Savova, Martina, Liliya Mihaylova, Daniel Tews, Martin Wabitsch, and Milen Georgiev. "Targeting PI3K/AKT signaling pathway in obesity." Targeting PI3K/AKT signaling pathway in obesity 159 (January 11, 2023): 114244. https://doi.org/10.1016/j.biopha.2023.114244.

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Obesity is a disorder with an increasing prevalence, which impairs the life quality of patients and intensifies societal health care costs. The development of safe and innovative prevention strategies and therapeutic approaches is thus of great importance. The complex pathophysiology of obesity involves multiple signaling pathways that influence energy metabolism in different tissues. The phosphatidylinositol 3-kinases (PI3K)/ protein kinase B (AKT) pathway is critical for the metabolic homeostasis and its function in insulin-sensitive tissues is described in the context of health, obesity and

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Dissertations / Theses on the topic "PI3K-Akt signaling pathway"

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MANSOURY, WALAA. "Exploring the role of PI3K/AKT signaling pathway in myxoid liposarcoma." Doctoral thesis, Università degli studi di Brescia, 2021. http://hdl.handle.net/11379/554950.

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Fraser, Sasha. "Development of Dual-Pathway Inhibitors of Raf/MEK/ERK and PI3K/Akt Signaling Pathways." VCU Scholars Compass, 2011. http://scholarscompass.vcu.edu/etd/2619.

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In the present study, we designed a new chemical template that contains an oxindole moiety as potential dual-pathway inhibitors of the Raf/MEK/ERK and PI3K/Akt signaling pathways. The design hypothesis is to evaluate whether the oxindole ring system will approximately orient functional groups in a similar manner to the thiazolidinedione moiety, and thus maintain biological activity as dual-pathway inhibitors of the Raf/MEK/ERK and PI3K/Akt signaling pathways. Furthermore, the oxindole ring will provide the flexibility to allow the introduction of various substituents on the oxindole moiety, th

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Pérez-Tenorio, Gizeh. "Alterations in the PI3K/AKT Signaling Pathway and Response to Adjuvant Treatment in Breast Cancer." Doctoral thesis, Linköpings universitet, Onkologi, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-15043.

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(PI3K)/AKT signaling pathway could be a cause of therapeutic resistance in breast cancer. The PI3K/AKT pathway controls cell proliferation, cell growth and survival, and its members include oncogenes and tumor suppressor genes. Alterations in this pathway are frequent in cancer. In this thesis, we aimed to study the biological significance of some of these alterations in a tumor context as well as their clinical value. PIK3CA gene, encoding the PI3K catalytic subunit, was examined for mutations. The tumor suppressor PTEN, that counteracts PI3Kmediated effects, was studied at the protein level

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Cremer, Thomas John IV. "Mechanisms of Host-Defense Against Intracellular Bacterial Pathogens Through The PI3K/Akt Host Signaling Pathway." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1289571696.

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ULTIMO, Simona. "Inhibition of the PI3K/Akt/mTOR signaling pathway as a therapeutic target for Acute Lymphoblastic Leukemia." Doctoral thesis, Università degli studi di Ferrara, 2018. http://hdl.handle.net/11392/2487845.

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Acute Lymphoblastic Leukemia (ALL) is a malignant disorder characterized by the abnormal clonal proliferation of B-cell progenitors (B-ALL) or immature stage thymocytes (T-ALL). Constitutive activation of the PI3K/Akt/mTOR network is a common feature of B- and T-ALL, influencing cell growth and survival. The PI3K/Akt/mTOR inhibitors are currently being developed for clinical use either as single agents or in combination with conventional chemotherapy for T-ALL patient treatment. In this study it has been investigated the effects of a panel of PI3K/Akt/mTOR inhibitors on healthy human CD4+ T-c

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Wright, Lilyan Yi Tian. "The activity of the helix-loop-helix protein, E47, is regulated by the PI3K/Akt signaling pathway." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3258711.

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Thesis (Ph. D.)--University of California, San Diego, 2007.<br>Title from first page of PDF file (viewed June 4, 2007). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.

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Moraes, Síntique Nunes Schulz. "Análise da expressão da proteína Akt em cultura de células de carcinomas epidermóides de cabeça e pescoço tratadas com curcumina." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/23/23154/tde-08082016-105511/.

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Diversas alterações genéticas estão associadas à patogênese do carcinoma epidermoide (CE), neoplasia maligna mais comum de cabeça e pescoço. Algumas dessas alterações comprometem proteínas pertencentes à via de sinalização do Akt, envolvida em diferentes fenômenos celulares. Este trabalho teve como objetivo estudar a expressão da proteína pAkt em linhagens celulares de carcinomas epidermoides de cabeça e pescoço, de forma a verificar possíveis alterações na transcrição dessa molécula em células de CE tratadas com Curcumina. Foram utilizadas duas linhagens celulares de CE de cabeça e pescoço (F

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Müller, Anja. "Multiple outcomes for PI3K/Akt/mTOR targeting in non-Hodgkin lymphoma." Doctoral thesis, Humboldt-Universität zu Berlin, Lebenswissenschaftliche Fakultät, 2015. http://dx.doi.org/10.18452/17284.

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Wachstumsfaktor bedingte Aktivierung des PI3K/Akt/mTOR Signalweg wirkt positiv auf Vermehrung und Überleben. Konstitutive Aktivierung des Signalweges in NHL ist jedoch an Tumorprogression und Therapieresistenz beteiligt. Am Zelllinienmodell wurden zwei mögliche Therapiestrategien der PI3K/Akt/mTOR Inhibition erprobt, PI3K Inhibition mit BKM120 und horizontale Kombination von Zytostatika mit PI3K/Akt/mTOR Inhibitoren Erstens, BKM120 hat Antitumoraktivität in NHL und induziert Zelltod. Auf molekularer Ebene führt BKM120 vermittelte Dephosphorylierung von CDK1 an Y15 zur Aktivierung des M-Phase

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She, Bin [Verfasser], and Ursula [Akademischer Betreuer] Klingmüller. "Dynamic Modeling of the PI3K/Akt Signal Transduction Pathway to Dissect the Distinct Cell Fate Decisions Triggered by PI3K/Akt Signaling in Hematopoietic System / Bin She ; Betreuer: Ursula Klingmüller." Heidelberg : Universitätsbibliothek Heidelberg, 2011. http://d-nb.info/1179229932/34.

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CANI, Alice. "Targeting the PI3K/Akt/mTOR signaling pathway as a new therapeutic strategy for personalized treatments in acute lymphoblastic leukemia." Doctoral thesis, Università degli studi di Ferrara, 2015. http://hdl.handle.net/11392/2388996.

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Acute lymphoblastic leukemia (ALL) is a hematologic malignancy characterized by the uncontrolled proliferation of lymphoblasts that accumulate in the blood, bone marrow and other organs. It represents 20% of adult acute leukemia and is the most common leukemia in children. The signal transduction pathway mediated by phosphatidylinositol 3-kinase (PI3K)/ Akt/mammalian target of rapamycin (mTOR) plays a key role in the regulation of important events for cells such as proliferation, differentiation and apoptosis, but also for the development of cancer and resistance to chemotherapy. In fact, the

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Books on the topic "PI3K-Akt signaling pathway"

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Z.Y. Luo, Q. Song, X.P. Xiong, et al. The PI3K/Akt/mTOR signaling pathway regulates lipid metabolism mediated by endoplasmic reticulum stress in goose primary hepatocytes. Verlag Eugen Ulmer, 2021. http://dx.doi.org/10.1399/eps.2021.325.

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Book chapters on the topic "PI3K-Akt signaling pathway"

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Thapa, Komal, Heena Khan, Amarjot Kaur Grewal, Thakur Gurjeet Singh, and Rahul Deshmukh. "Regulation of PI3K-Akt Signaling Pathway in Ischemia/Reperfusion Injury." In Ischemic Injury. Apple Academic Press, 2024. http://dx.doi.org/10.1201/9781032680026-27.

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Ertay, Ayse. "Altered PI3K/AKT/mTOR Signaling Pathway and Cancer Stem Cells." In Stem Cell Biology and Regenerative Medicine. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-74842-4_5.

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Saleem, Sadaf, Insha Bashir, Mosin Saleem Khan, and Mohd Syed Ahanger. "Association of PI3K/AKT/mTOR Pathway with Cancer and Its Therapeutic Implications." In Cell Signaling Pathways and Their Therapeutic Implication in Cancers. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-96-2763-9_3.

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Han, Limei, Jingyi Yang, Lei Jing, et al. "Tau-TCHF Inhibits Spleenic Apoptosis via PI3K-Akt Signaling Pathway in Chickens." In Advances in Experimental Medicine and Biology. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8023-5_51.

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Fayard, Elisabeth, Gongda Xue, Arnaud Parcellier, Lana Bozulic, and Brian A. Hemmings. "Protein Kinase B (PKB/Akt), a Key Mediator of the PI3K Signaling Pathway." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/82_2010_58.

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Weeks, Kate L., Bianca C. Bernardo, Jenny Y. Y. Ooi, Natalie L. Patterson, and Julie R. McMullen. "The IGF1-PI3K-Akt Signaling Pathway in Mediating Exercise-Induced Cardiac Hypertrophy and Protection." In Advances in Experimental Medicine and Biology. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-4304-8_12.

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González-Pérez, Pedro Pablo, and Maura Cárdenas-García. "Inspecting the Role of PI3K/AKT Signaling Pathway in Cancer Development Using an In Silico Modeling and Simulation Approach." In Bioinformatics and Biomedical Engineering. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78723-7_7.

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Feldman, Morris E., and Kevan M. Shokat. "New Inhibitors of the PI3K-Akt-mTOR Pathway: Insights into mTOR Signaling from a New Generation of Tor Kinase Domain Inhibitors (TORKinibs)." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/82_2010_64.

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Amornphimoltham, Panomwat, Vyomesh Patel, Alfredo Molinolo, and J. Silvio Gutkind. "Head and Neck Cancer and the PI3K/Akt/mTOR Signaling Network: Novel Molecular Targeted Therapies." In Signaling Pathways in Squamous Cancer. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-7203-3_19.

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Russo, Giulia, Marzio Pennisi, Roberta Boscarino, and Francesco Pappalardo. "Modeling PI3K/PDK1/Akt and MAPK Signaling Pathways Using Continuous Petri Nets." In Intelligent Computing Theories and Application. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-63312-1_15.

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Conference papers on the topic "PI3K-Akt signaling pathway"

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Hasan, A. A., E. V. Kalinina, Yu L. Volodina, et al. "REVERSAL OF CANCER CELL RESISTANCE TOWARDS CISPLATIN BY CURCU-MIN." In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. LLC Institute Information Technologies, 2024. http://dx.doi.org/10.47501/978-5-6044060-4-5.47-50.

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The article discusses the effect of the plant polyphenol curcumin on the resistance of SKOV-3 ovar-ian adenocarcinoma cells to the antitumor drug cisplatin. Data are presented showing that the ac-tion of curcumin causes a “reversal” of drug resistance in tumor cells, which is associated with suppression of the cellular antioxidant system and the PI3K/Akt/mTOR signaling pathway.

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Qi, Yong, Minghong Xie, Li Wei, and Guangjie Hou. "Insulin resistance exacerbates lung inflammation in obese patients via PI3K/Akt signaling pathway." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3343.

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Sobsey, Constance A., Robert Popp, Sahar Ibrahim та ін. "Abstract B21: Protein quantitation assays for Akt, PI3K p110α, and PTEN to assess PI3K pathway activity in tumor tissue". У Abstracts: AACR Special Conference on Targeting PI3K/mTOR Signaling; November 30-December 8, 2018; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.pi3k-mtor18-b21.

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Fransson, Susanne M., Frida Abel, Helena Eriksson, Per Kogner, Tommy Martinsson, and Katarina Ejeskär. "Abstract 2237: Analysis of the PI3K/Akt signaling pathway in neuroblastoma shows stage dependent expression of PI3K isoforms." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2237.

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Dong, Qiongzhu, Xu-Chao Zhu, Chun Dai, et al. "Abstract 2695: Osteopontin regulated epithelial-mesenchymal transition via PI3K/AKT signaling pathway in hepatocellular carcinoma." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2695.

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Bu, Rong, Sarita Prabhakaran, Shaham Beg, et al. "Abstract 4323: ALK overexpression is associated with activation of PI3K/AKT signaling pathway in PTC." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4323.

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Ding, Yan, Jiangang Liu, John N. Calley, et al. "Abstract 2586: PI3K/AKT signaling pathway is transcriptionally elevated in prexasertib-resistant TNBC PDX models." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-2586.

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Tripathi, Satyendra C., Johannes F. Fahrmann, Muge Celiktas, et al. "Abstract 3170: MCAM modulates small cell lung cancer chemoresistance via PI3k/Akt/Sox2 signaling pathway." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3170.

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Chew, Hui Yi, Benedict Panizza, Caroline Cooper, et al. "Abstract B12: Examining EGFR-mediated PI3K/Akt pathway in combination therapy of cetuximab and dynamin inhibition." In Abstracts: AACR Special Conference on Targeting PI3K/mTOR Signaling; November 30-December 8, 2018; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.pi3k-mtor18-b12.

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Thompson, Barry. "Abstract IA29: The Hippo pathway integrates PI3K-Akt signals with mechanical cues to control tissue growth." In Abstracts: AACR Special Conference on the Hippo Pathway: Signaling, Cancer, and Beyond; May 8-11, 2019; San Diego, CA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.hippo19-ia29.

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Reports on the topic "PI3K-Akt signaling pathway"

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Hang, Fei, Rishalaiti Tayier, Ang Li, Yanjie Yuan, and Shunhua Wu. PTEN regulates arsenic-inducedn autophagy in PI3K/AKT/mTOR signaling pathway; A systematic review and meta-analysis of in vivo and in vitro studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.1.0012.

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Chen, Xiaole, Peng Wang, Yunquan Luo, et al. Therapeutic Efficacy Evaluation and Underlying Mechanisms Prediction of Jianpi Liqi Decoction for Hepatocellular Carcinoma. Science Repository, 2021. http://dx.doi.org/10.31487/j.jso.2021.02.04.sup.

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Objective: The aim of this study was to assess the therapeutic effects of Jianpi Liqi decoction (JPLQD) in hepatocellular carcinoma (HCC) and explore its underlying mechanisms. Methods: The characteristics and outcomes of HCC patients with intermediate stage B who underwent sequential conventional transcatheter arterial chemoembolization (cTACE) and radiofrequency ablation (RFA) only or in conjunction with JPLQD were analysed retrospectively. The plasma proteins were screened using label-free quantitative proteomics analysis. The effective mechanisms of JPLQD were predicted through network pha

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