Academic literature on the topic 'Polygenic traits'

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Journal articles on the topic "Polygenic traits"

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BARTON, N. H. "Clines in polygenic traits." Genetical Research 74, no. 3 (1999): 223–36. http://dx.doi.org/10.1017/s001667239900422x.

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This article outlines theoretical models of clines in additive polygenic traits, which are maintained by stabilizing selection towards a spatially varying optimum. Clines in the trait mean can be accurately predicted, given knowledge of the genetic variance. However, predicting the variance is difficult, because it depends on genetic details. Changes in genetic variance arise from changes in allele frequency, and in linkage disequilibria. Allele frequency changes dominate when selection is weak relative to recombination, and when there are a moderate number of loci. With a continuum of alleles
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ZENG, WEN, SUJIT GHOSH, and BAILIAN LI. "A blocking Gibbs sampling method to detect major genes with phenotypic data from a diallel mating." Genetical Research 83, no. 2 (2004): 143–54. http://dx.doi.org/10.1017/s0016672304006718.

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Diallel mating is a frequently used design for estimating the additive and dominance genetic (polygenic) effects involved in quantitative traits observed in the half- and full-sib progenies generated in plant breeding programmes. Gibbs sampling has been used for making statistical inferences for a mixed-inheritance model (MIM) that includes both major genes and polygenes. However, using this approach it has not been possible to incorporate the genetic properties of major genes with the additive and dominance polygenic effects in a diallel mating population. A parent block Gibbs sampling method
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Ma, Xiaoman, Weifeng Guo, Liangrong He, and Xinchuan Cao. "Polygenic Genetic Analysis of Principal Genes for Yield Traits in Land Cotton." Agronomy 14, no. 11 (2024): 2749. http://dx.doi.org/10.3390/agronomy14112749.

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Objective: Yield traits are crucial for cotton breeding. Analyzing the yield traits of terrestrial cotton and exploring their genetic mechanisms through a primary gene + multigene hybrid genetic model provide a theoretical basis for selecting high-quality cotton varieties and identifying associated molecular markers. Methods: Completing the construction of the six populations (P1, P2, F1, F2, B1, B2) using Xinluzhong 37 as the female parent and Xinluzhong 51 as the male parent. Six yield traits were assessed: single boll weight, boll number per plant, lint yield per plant, seed cotton per plan
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Crouch, Daniel J. M., and Walter F. Bodmer. "Polygenic inheritance, GWAS, polygenic risk scores, and the search for functional variants." Proceedings of the National Academy of Sciences 117, no. 32 (2020): 18924–33. http://dx.doi.org/10.1073/pnas.2005634117.

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The reconciliation between Mendelian inheritance of discrete traits and the genetically based correlation between relatives for quantitative traits was Fisher’s infinitesimal model of a large number of genetic variants, each with very small effects, whose causal effects could not be individually identified. The development of genome-wide genetic association studies (GWAS) raised the hope that it would be possible to identify single polymorphic variants with identifiable functional effects on complex traits. It soon became clear that, with larger and larger GWAS on more and more complex traits,
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Luciano, Michelle, Riccardo E. Marioni, Maria Valdés Hernández, et al. "Structural Brain MRI Trait Polygenic Score Prediction of Cognitive Abilities." Twin Research and Human Genetics 18, no. 6 (2015): 738–45. http://dx.doi.org/10.1017/thg.2015.71.

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Structural brain magnetic resonance imaging (MRI) traits share part of their genetic variance with cognitive traits. Here, we use genetic association results from large meta-analytic studies of genome-wide association (GWA) for brain infarcts (BI), white matter hyperintensities, intracranial, hippocampal, and total brain volumes to estimate polygenic scores for these traits in three Scottish samples: Generation Scotland: Scottish Family Health Study (GS:SFHS), and the Lothian Birth Cohorts of 1936 (LBC1936) and 1921 (LBC1921). These five brain MRI trait polygenic scores were then used to: (1)
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Ridenhour, Benjamin J., and Scott L. Nuismer. "POLYGENIC TRAITS AND PARASITE LOCAL ADAPTATION." Evolution 61, no. 2 (2007): 368–76. http://dx.doi.org/10.1111/j.1558-5646.2007.00029.x.

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Wright, Fred A., and Augustine Kong. "Linkage Mapping in Experimental Crosses: The Robustness of Single-Gene Models." Genetics 146, no. 1 (1997): 417–25. http://dx.doi.org/10.1093/genetics/146.1.417.

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The robustness of parametric linkage mapping against model misspecification is considered in experimental breeding designs, with a focus on localization of the gene. By examining the expected LOD across the genome, it is shown that single-gene models are quite robust, even for polygenic traits. However, when the marker map is of low resolution, linked polygenes can give rise to an apparent “ghost” gene, mapped to an incorrect interval. The results apply equally well to quantitative traits or qualitative (categorical) traits. The results are derived for backcross populations, with a discussion
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Jansen, Ritsert C. "Complex plant traits: Time for polygenic analysis." Trends in Plant Science 1, no. 3 (1996): 89–94. http://dx.doi.org/10.1016/s1360-1385(96)80040-9.

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Josephs, Emily B., Jeremy J. Berg, Jeffrey Ross-Ibarra, and Graham Coop. "Detecting Adaptive Differentiation in Structured Populations with Genomic Data and Common Gardens." Genetics 211, no. 3 (2019): 989–1004. http://dx.doi.org/10.1534/genetics.118.301786.

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Adaptation in quantitative traits often occurs through subtle shifts in allele frequencies at many loci—a process called polygenic adaptation. While a number of methods have been developed to detect polygenic adaptation in human populations, we lack clear strategies for doing so in many other systems. In particular, there is an opportunity to develop new methods that leverage datasets with genomic data and common garden trait measurements to systematically detect the quantitative traits important for adaptation. Here, we develop methods that do just this, using principal components of the rela
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Rosenberg, Noah A., Michael D. Edge, Jonathan K. Pritchard, and Marcus W. Feldman. "Interpreting polygenic scores, polygenic adaptation, and human phenotypic differences." Evolution, Medicine, and Public Health 2019, no. 1 (2018): 26–34. http://dx.doi.org/10.1093/emph/eoy036.

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Abstract Recent analyses of polygenic scores have opened new discussions concerning the genetic basis and evolutionary significance of differences among populations in distributions of phenotypes. Here, we highlight limitations in research on polygenic scores, polygenic adaptation and population differences. We show how genetic contributions to traits, as estimated by polygenic scores, combine with environmental contributions so that differences among populations in trait distributions need not reflect corresponding differences in genetic propensity. Under a null model in which phenotypes are
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Dissertations / Theses on the topic "Polygenic traits"

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Lango, Allen Hana. "The role of common genetic variation in model polygenic and monogenic traits." Thesis, University of Exeter, 2010. http://hdl.handle.net/10871/11714.

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The aim of this thesis is to explore the role of common genetic variation, identified through genome-wide association (GWA) studies, in human traits and diseases, using height as a model polygenic trait, type 2 diabetes as a model common polygenic disease, and maturity onset diabetes of the young (MODY) as a model monogenic disease. The wave of the initial GWA studies, such as the Wellcome Trust Case-Control Consortium (WTCCC) study of seven common diseases, substantially increased the number of common variants associated with a range of different multifactorial traits and diseases. The initia
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Rakitsch, Barbara [Verfasser], and Karsten [Akademischer Betreuer] Borgwardt. "Modeling the polygenic architecture of complex traits / Barbara Rakitsch ; Betreuer: Karsten Borgwardt." Tübingen : Universitätsbibliothek Tübingen, 2015. http://d-nb.info/1197057099/34.

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Riffo, Francisco Cubillios. "multi-parent crosses reveal the complex genetic architecture of polygenic traits in yeast." Thesis, University of Nottingham, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.537694.

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Merwood, Andrew. "Exploring the aetiology of ADHD : rater effects, co-occurring traits and polygenic scores." Thesis, King's College London (University of London), 2013. https://kclpure.kcl.ac.uk/portal/en/theses/exploring-the-aetiology-of-adhd(e7a5d527-95e0-4b47-bc20-05dc23e835b2).html.

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The precise aetiology of attention-deficit/hyperactivity disorder (ADHD) and its association with co-occurring traits remains unclear. Accordingly, the overarching aim of this thesis was to address several ambiguities surrounding the causes and correlates of ADHD. The first of these ambiguities concerns rater effects in twin studies. This was addressed by examining parent, teacher and child self-ratings of ADHD symptoms obtained concurrently using population-based twin data. Results revealed significantly lower heritability for self-ratings than for parent or teacher ratings of ADHD symptoms,
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Chan, Ying Leong. "Leveraging genetic association data to investigate the polygenic architecture of human traits and diseases." Thesis, Harvard University, 2014. http://dissertations.umi.com/gsas.harvard:11372.

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Many human traits and diseases have a polygenic architecture, where phenotype is partially determined by variation in many genes. These complex traits or diseases can be highly heritable and genome-wide association studies (GWAS) have been relatively successful in the identification of associated variants. However, these variants typically do not account for most of the heritability and thus, the genetic architecture remains uncertain.
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Miyake, Takeshi. "STATISTICAL GENETIC EVALUATION OF ANIMALS FOR QUANTITATIVE TRAITS UNDER POLYGENIC MODEL AND MIXED INHERITANCE MODEL." Kyoto University, 1999. http://hdl.handle.net/2433/181392.

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Krapohl, Eva Maria Laura. "Multi-variable approaches to polygenic trait prediction." Thesis, King's College London (University of London), 2017. https://kclpure.kcl.ac.uk/portal/en/theses/multivariable-approaches-to-polygenic-trait-prediction(1e69a4a3-f89b-4646-980e-02031d5c23d3).html.

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Robust evidence for the polygenicity and genetic correlations of complex traits across the phenome suggests both the necessity of polygenic instruments and the value of multi-trait prediction models. This thesis used multi-variable approaches in four papers and along two main threads: Multi-variable approaches to trait prediction A primary goal of polygenic scores, which aggregate effects of trait-associated variants discovered in genome-wide association studies (GWAS), is to estimate individual-specific genetic propensities to predict trait variation. This is typically achieved using one poly
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Reavey, Caitlin Teresa. "Analysis of Transcription Activation Distance as a Polygenic Trait in Saccharomyces cerevisiae." Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:11202.

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Much of the eukaryotic transcriptional machinery is conserved from yeast to human. However, the distance over which transcriptional activation can occur differs between Saccharomyces cerevisiae and metazoans. In S. cerevisiae, the upstream activating sequence (UAS) is generally found within 300 base pairs of the transcription start site; when the UAS is moved too far away, activation no longer occurs. In contrast, metazoan enhancers can activate from as far as 100 kilobases from the start site. In past work, our lab identified five genes that, when mutant, allow transcription activation to
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Muiños, Gimeno Margarita. "Analysis of genetic variation in microrna-mediated regulation and the susceptibility to anxiety disorders." Doctoral thesis, Universitat Pompeu Fabra, 2009. http://hdl.handle.net/10803/7192.

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We have investigated genetic variation in microRNA-mediated regulation as a susceptibility factor for anxiety disorders following two different approaches. We first studied two isoforms of the candidate gene NTRK3 by re-sequencing its different 3'UTRs in patients with Panic (PD) and Obsessive Compulsive disorders (OCD) as well as controls. Two rare variants that altered microRNA-mediated regulation were identified in PD. Conversely, association of a common SNP with OCD hoarding subtype was found. Moreover, we have also studied a possible involvement of microRNAs in anxiety disorders. Consequen
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Cantet, Mélissa. "Variation phénotypique de la résistance quantitative à Phytophthora capsici dans la diversité naturelle du piment, et diversité moléculaire et profil d'évolution du QTL majeur Pc5.1." Electronic Thesis or Diss., Montpellier, SupAgro, 2013. http://www.theses.fr/2013NSAM0006.

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L'utilisation de variétés présentant des résistances quantitatives polygéniques est une pratique respectueuse de l'environnement et potentiellement durable pour lutter contre les bioagresseurs. Les résistances quantitatives sont cependant mal connues et encore peu exploitées. Via l'étude de l'interaction Capsicum spp. / Phytophthora capsici, les objectifs sont de (i) caractériser la diversité naturelle de l'hôte pour le phénotype quantitatif de résistance, (ii) décrire la diversité au QTL Pc5.1, déterminant majeur de la résistance, conservé chez les géniteurs et efficace à large spectre, et (i
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Books on the topic "Polygenic traits"

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Luo, Z. W. Polygene location and selection for heterotic traits in 'Drosophila'. University of Birmingham, 1989.

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CHAN, YING LEONG. Leveraging genetic association data to investigate the polygenic architecture of human traits and diseases. 2014.

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Kan, Carol, and Ma-Li Wong. Genetics. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198789284.003.0004.

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An association between type 2 diabetes mellitus (T2DM) and depression has been reported in epidemiological studies. Finding a genetic overlap between T2DM and depression will provide evidence to support a common biological pathway to both disorders. Genetic correlations observed from twin studies indicate that a small magnitude of the variance in liability can be attributed to genetic factors. However, no genetic overlap has been observed between T2DM and depression in genome-wide association studies using both the polygenic score and the linkage disequilibrium score regression approaches. Cla
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Walsh, Bruce, and Michael Lynch. Long-term Response: 1. Deterministic Aspects. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198830870.003.0025.

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In a large population in the absence of new mutation, selection is expected to eventually drive all of the additive-genetic variance in a trait toward zero, resulting in a selection limit. This chapter examines the underlying population-genetics of such a limit, how it is estimated, and reviews the actual nature of limits observed in artificial selection experiments. It also examines the conditions under which a major gene is more important than polygenic response.
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Levinson, Douglas F., and Walter E. Nichols. Genetics of Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0024.

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Major depressive disorder (MDD) is a common and heterogeneous complex trait. Twin heritability is 35%–40%, perhaps higher in severe/recurrent cases. Adverse life events (particularly during childhood) increase risk. Current evidence suggests some overlap in genetic factors among MDD, bipolar disorder, and schizophrenia. Large genome-wide association studies (GWAS) are now proving successful. Polygenic effects of common SNPs are substantial. Findings implicate genes with effects on synaptic development and function, including two obesity-associated genes (NEGR1 and OLFM4), but not previous “can
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Book chapters on the topic "Polygenic traits"

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Pazoki, Raha. "Methods for Polygenic Traits." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7868-7_10.

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Griesbach, R. J. "Microinjection for polygenic traits." In Biotechnology for Solving Agricultural Problems. Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4396-4_8.

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Foulley, J. L., D. Gianola, and S. Im. "Genetic Evaluation for Discrete Polygenic Traits in Animal Breeding." In Advances in Statistical Methods for Genetic Improvement of Livestock. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74487-7_17.

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Utermann, Gerd. "Apolipoproteins, Quantitative Lipoprotein Traits and Multifactorial Hyperlipidaemia." In Ciba Foundation Symposium 130 - Molecular Approaches to Human Polygenic Disease. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470513507.ch5.

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Raben, Timothy G., Louis Lello, Erik Widen, and Stephen D. H. Hsu. "From Genotype to Phenotype: Polygenic Prediction of Complex Human Traits." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2205-6_15.

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Bodmer, Walter F. "The Human Genome Sequence and the Analysis of Multifactorial Traits." In Ciba Foundation Symposium 130 - Molecular Approaches to Human Polygenic Disease. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470513507.ch13.

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Gailing, Oliver, Katharina Birgit Budde, Ludger Leinemann, Markus Müller, and Selina Wilhelmi. "Genetic Connectivity and Local Adaptation of Forest Trees in the Face of Climate Change." In Ecological Connectivity of Forest Ecosystems. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-82206-3_5.

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Abstract The long-term survival of populations depends on genetic variation in traits related to survival and reproductive fitness. The polygenic architecture of traits is thought to facilitate adaptive shifts, but whether tree species will be able to adapt to the currently rapidly changing climatic conditions remains a subject of debate. On the other hand, trees are characterized by considerable phenotypic plasticity that allows them to grow under different or variable environmental conditions caused by global climate change. Phenotypic plasticity may thus help populations survive by “buying
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Chakma, Nidhi, Moutoshi Chakraborty, Salma Bhyan, and Mobashwer Alam. "Molecular breeding for combating salinity stress in sorghum: progress and prospects." In Molecular breeding in wheat, maize and sorghum: strategies for improving abiotic stress tolerance and yield. CABI, 2021. http://dx.doi.org/10.1079/9781789245431.0024.

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Abstract This chapter discusses current progress and prospects of molecular breeding and strategies for developing better saline-tolerant sorghum (Sorghum bicolor) varieties. Most molecular breeding techniques for salt tolerance have been carried out in controlled environments where the plants were not exposed to any variation of the surrounding environment, producing reliable results. Due to the polygenic nature of salt tolerance, the identified quantitative trait loci (QTLs) could be false QTLs. Therefore, QTL validation is important in different plant populations and field conditions. Subse
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Lauwerys, Bernard R., and Edward K. Wakeland. "Analyzing Complex Polygenic Traits." In Stem Cell Therapy for Autoimmune Disease. CRC Press, 2019. http://dx.doi.org/10.1201/9780367813895-22.

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Jones, Byron. "Some Basics, Mendelian Traits, Polygenic Traits, Complex Traits." In Neurobehavioral Genetics. CRC Press, 2006. http://dx.doi.org/10.1201/9781420003567.ch3.

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Conference papers on the topic "Polygenic traits"

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Davidov, Ilan, Roee Levinberg, Guy Attia, Erez Arad, and Ludmila Sidorenko. "Biotechnological Tools in Genetics for Primary Prophylaxis of Essential Arterial Hypertension." In 12th International Conference on Electronics, Communications and Computing. Technical University of Moldova, 2022. http://dx.doi.org/10.52326/ic-ecco.2022/bme.08.

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Hypertension is one of the most spread cardiological diseases, due to recent biotechnological tools it is discovered that arterial hypertension is a polygenic disease. Another possible explanation might be the variety of the studied populations. The current results of genetic analyses of essential hypertension highlight the need for a more differentiated approach to the understanding of complex, polygenetic traits that implements gene-gene- and gene-environment interactions or differentiated functional testing of thoroughly phenotyped cohorts under standardised environmental conditions. The ad
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Gierten, J., T. Fitzgerald, F. Loosli, M. Gorenflo, E. Birney, and J. Wittbrodt. "Genome-Wide Analysis of Polygenic Cardiac Traits in Medaka Inbred Strains." In 52nd Annual Meeting of the German Society for Pediatric Cardiology. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1705539.

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Lutze, Margaret, Curtis R. Brandt, Vivianne C. Smith, Joel Pokorny, and Ron G. Gregg. "Genetic studies of normal color vision." In OSA Annual Meeting. Optica Publishing Group, 1989. http://dx.doi.org/10.1364/oam.1989.fa3.

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We have studied the familial transmissions of Rayleigh match midpoints and photometric matches (667-551 nm) in observers with normal color vision to assess genetic bases for these two color vision traits (Lutze et al., in press). We first employed segregation analysis to evaluate whether each trait was consistent with determination by allelic variation of a single gene, by multiple genes (polygenic), or by environmental factors. We found that each trait was consistent with determination by a single gene. Blood samples were obtained from the majority of family members tested and DNA was isolate
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Kember, Rachel L., Shefali S. Verma, Anurag Verma, et al. "Polygenic risk scores for cardiometabolic traits demonstrate importance of ancestry for predictive precision medicine." In Pacific Symposium on Biocomputing 2024. WORLD SCIENTIFIC, 2023. http://dx.doi.org/10.1142/9789811286421_0046.

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Kember, Rachel L., Shefali S. Verma, Anurag Verma, et al. "Polygenic risk scores for cardiometabolic traits demonstrate importance of ancestry for predictive precision medicine." In Pacific Symposium on Biocomputing 2025. WORLD SCIENTIFIC, 2024. http://dx.doi.org/10.1142/9789819807024_0056.

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"Association between polygenic risk scores for plasma protein N-glycosylation traits and 276 ICD-10 diseases." In Bioinformatics of Genome Regulation and Structure/Systems Biology (BGRS/SB-2022) :. Institute of Cytology and Genetics, the Siberian Branch of the Russian Academy of Sciences, 2022. http://dx.doi.org/10.18699/sbb-2022-266.

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Lutze, Margaret, Nancy J. Cox, Vivianne C. Smith, and Joel Pokorny. "Genetics of Rayleigh matches and photometric matches in normals." In OSA Annual Meeting. Optica Publishing Group, 1988. http://dx.doi.org/10.1364/oam.1988.mz3.

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At the 1988 ARVO meeting we presented Rayleigh match midpoint and photometric match data collected from a population of nonrelated males and from family members. Photometric matches between a 667- and 551-nm stimulus were obtained using heterochromatic modulation photometry (HMP). We employed statistical methods to determine whether the source of variation for each of these traits was due to allelic variation at a single gene locus, variation at multiple loci (polygenic), or environmental factors. Results indicated that variations in Rayleigh match midpoints and HMP photometric matches in obse
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Rahaman, Md Abdur, Amanda Rodrigue, David Glahn, Jessica Turner, and Vince Calhoun. "Shared sets of correlated polygenic risk scores and voxel-wise grey matter across multiple traits identified via bi-clustering." In 2021 43rd Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2021. http://dx.doi.org/10.1109/embc46164.2021.9630825.

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Meyer, K. "361. Accounting for trait-specific genomic and residual polygenic covariances in multivariate single-step genomic evaluation." In World Congress on Genetics Applied to Livestock Production. Wageningen Academic Publishers, 2022. http://dx.doi.org/10.3920/978-90-8686-940-4_361.

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Кочерина, Н. В. "APPLICATION OF MOLECULAR GENETIC TOOLS AND ECOLOGICALGENETIC ALGORITHMS FOR SOLVING PROBLEMS OF INCREASING THE PRODUCTIVITY OF PLANTS." In МАТЕРИАЛЫ II Всероссийской научной конференции с международным участием «ПРИМЕНЕНИЕ СРЕДСТВ ДИСТАНЦИОННОГО ЗОНДИРОВАНИЯ ЗЕМЛИ В СЕЛЬСКОМ ХОЗЯЙСТВЕ» Санкт-Петербург, 26–28 сентября 2018 г. Crossref, 2018. http://dx.doi.org/10.25695/agrophysica.2018.2.18865.

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С точки зрения комплексного междисциплинарного подхода, для повышения продуктивности сельскохозяйственных культур необходимо применение достижений генетико-селекционного характера. В работе приведены примеры эколого-генетических подходов и генетических инструментов маркерной помощи селекции, основанных на статистических методах и подходах. In addition to remote sensing technologies, an integrated interdisciplinary approach wins in solving plant productivity problems. Therefore, the aspects of ecological and genetic achievements in the field of plant breeding, the interaction of selection with
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Reports on the topic "Polygenic traits"

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Perl, Avichai, Bruce I. Reisch, and Ofra Lotan. Transgenic Endochitinase Producing Grapevine for the Improvement of Resistance to Powdery Mildew (Uncinula necator). United States Department of Agriculture, 1994. http://dx.doi.org/10.32747/1994.7568766.bard.

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The original objectives are listed below: 1. Design vectors for constitutive expression of endochitinase from Trichoderma harzianum strain P1. Design vectors with signal peptides to target gene expression. 2. Extend transformation/regeneration technology to other cultivars of importance in the U.S. and Israel. 3. Transform cultivars with the endochitinase constructs developed as part of objective 1. A. Characterize foliar powdery mildew resistance in transgenic plants. Background of the topic Conventional breeding of grapevines is a slow and imprecise process. The long generation cycle, large
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Abbo, Shahal, Hongbin Zhang, Clarice Coyne, Amir Sherman, Dan Shtienberg, and George J. Vandemark. Winter chickpea; towards a new winter pulse for the semiarid Pacific Northwest and wider adaptation in the Mediterranean basin. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7597909.bard.

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Original objectives: [a] Screen an array of chickpea and wild annual Cicer germplasm for winter survival. [b] Genetic analysis of winter hardiness in domesticated x wild chickpea crosses. [c] Genetic analysis of vernalization response in domesticated x wild chickpea crosses. [d] Digital expression analysis of a core selection of breeding and germplasm lines of chickpea that differ in winter hardiness and vernalization. [e] Identification of the genes involved in the chickpea winter hardiness and vernalization and construction of gene network controlling these traits. [f] Assessing the phenotyp
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Sela, Hanan, Eduard Akhunov, and Brian J. Steffenson. Population genomics, linkage disequilibrium and association mapping of stripe rust resistance genes in wild emmer wheat, Triticum turgidum ssp. dicoccoides. United States Department of Agriculture, 2014. http://dx.doi.org/10.32747/2014.7598170.bard.

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The primary goals of this project were: (1) development of a genetically characterized association panel of wild emmer for high resolution analysis of the genetic basis of complex traits; (2) characterization and mapping of genes and QTL for seedling and adult plant resistance to stripe rust in wild emmer populations; (3) characterization of LD patterns along wild emmer chromosomes; (4) elucidation of the multi-locus genetic structure of wild emmer populations and its correlation with geo-climatic variables at the collection sites. Introduction In recent years, Stripe (yellow) rust (Yr) caused
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