Relevant bibliographies by topics / Polymorphism of the gene eNOS C786T

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Academic literature on the topic 'Polymorphism of the gene eNOS C786T'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Polymorphism of the gene eNOS C786T"

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Olesya, V. Romaschenko* Eduard A. Snegin Lyudmila R. Zakirova Elena B. Artyushkova Galina A. Batischeva Natalya A. Bystrova Vladimir Y. Provotorov. "GENE POLYMORPHISM OF ENDOTHELIAL NITRIC OXIDE SYNTHASE IN PATIENTS WITH STABLE ANGINA PECTORIS AND ITS SIGNIFICANCE IN THE MANIFESTATION OF GENOPROTECTIVE PROPERTIES OF MELDONIUM." Indo American Journal of Pharmaceutical Sciences 04, no. 10 (2017): 3786–91. https://doi.org/10.5281/zenodo.1020020.

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Introduction: Personalized pharmacotherapy of stable angina pectoris implies an in-depth study of the pharmacogenetic factor when prescribing the recommended drugs in order to increase the effectiveness and safety of their use. The work is devoted to the development of individual approaches to the appointment of meldonium in the complex pharmacotherapy of angina pectoris as a cardiac cytoprotector with endothelioprotective properties, depending on gene polymorphism of endothelial nitric oxide synthase. Research tasks: To study the polymorphism of the endothelial nitric oxide synthase gene eNOS
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Troitskaya, N. I., K. G. Shapovalov, and V. A. Mudrov. "Molecular genetic polymorphisms C786T of the eNOS gene and LYS198ASN of the END1 gene in diabetic foot syndrome." Translational Medicine 9, no. 4 (2022): 13–19. http://dx.doi.org/10.18705/2311-4495-2022-9-4-13-19.

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Background. Diabetic foot syndrome is one of the complications of diabetes mellitus, which could result inthe development of purulent-necrotic lesions of soft tissues or amputation. Nitric oxide and endothelin-1 plays key role in endothelium dysfunction, thus contributing to the pathogenesis. Objective. To evaluate the frequency of genotypes of the C786T polymorphism of the eNOS gene and the LYS198ASN polymorphism of the END1 gene in a group of patients with diabetic foot syndrome and a group of healthy donors living in the Trans-Baikal Territory. Design and methods. In 100 healthy individuals
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Троицкая, Н. И., К. Г. Шаповалов, and В. А. Мудров. "Endothelial fluctuation amplitude of microcirculation in carriers of different polymorphic markers C786T of the eNOS gene and Lys198Asn of the END1 gene with diabetic foot syndrome." Nauchno-prakticheskii zhurnal «Patogenez», no. 2 (May 31, 2022): 38–44. http://dx.doi.org/10.25557/2310-0435.2022.02.38-44.

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Цель работы: изучить амплитуду эндотелиального диапазона колебаний микрокровотока при развитии синдрома диабетической стопы у носителей разных полиморфных маркеров C786T гена eNOS и Lys198Asn гена END1. Материалы и методы: В исследовании приняли участие 198 пациентов с неосложнённым сахарным диабетом и 199 пациентов с синдромом диабетической стопы, у которых оценили распределение генотипов полиморфных маркеров C786T гена eNOS и Lys198Asn гена END1 методом полимеразной цепной реакции. У 30 человек в каждой группе, сопоставимых по распределению полиморфизмов изучаемых генов, исследовали состояни
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ZAKHARYAN, ELENA A., та OLGA YU GRITSKEVICH. "IMPACT OF THE SINGLE-NUCLEOTIDE POLYMORPHISM OF GENES IL1β, EDN1, AND NOS3 GENES ON THE INDIVIDUAL GENETIC PROFILE OF PATIENTS WITH CORONARY HEART DISEASE IN THE REPUBLIC OF CRIMEA". Bulletin of Contemporary Clinical Medicine 16, № 6 (2023): 31–36. http://dx.doi.org/10.20969/vskm.2023.16(6).31-36.

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Abstracts. Introduction. Coronary heart disease prevalence rate grows, which determines the importance of researchers’ inquiries aiming to improve the prevention, diagnosis, and treatment of this pathology. Aim. This study was aimed at identifying the associations of polymorphisms of genes interleukin 1-beta, endothelin-1, and endothelial nitric oxide synthase in patients with coronary heart disease. Materials and Methods. We studied polymorphic markers of endothelin-1 (rs5370), endothelial nitric oxide synthase (rs2070244), and interleukin 1-beta genes (rs1143634 and rs16944) in patients with
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Krotkov, K. O. "GENE POLYMORPHISMS AND CLINICAL, DEMOGRAPHIC, ANTHROPOLOGICAL DATA IN PATIENTS WITH ISCHEMIC HEART DISEASE WHO UNDERWENT CORONARY ARTERY BYPASS GRAFTING UNDER ARTIFICIAL CIRCULATION." Journal of the Grodno State Medical University 22, no. 6 (2025): 544–47. https://doi.org/10.25298/2221-8785-2024-22-6-544-547.

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Objective. To identify the relationship between polymorphisms of the angiotensin type 1 receptor gene (AGT2R1 A1166C) (rs 5186), endothelin-1 gene (LYS198ASN) (rs 5370), endothelial nitric oxide synthase gene NOS3 (C786T) (rs 2070744) and clinical, demographic, anthropological data in patients with coronary heart disease who underwent coronary artery bypass grafting under artificial circulation. Material and methods. All patients (123 people) underwent intraoperative venous blood sampling from a central venous catheter. Then, using the polymerase chain reaction method, there was performed an i
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Krotkov, K. O. "IMPACT OF EXTRACORPOREAL AUTOHEMOMAGNETOTHERAPY ON THE ACID-BASE STATE OF ARTERIAL BLOOD IN THE INTRAOPERATIVE PERIOD DURING MYOCARDIAL REVASCULARIZATION UNDER ARTIFICIAL CIRCULATION." Journal of the Grodno State Medical University 22, no. 5 (2024): 465–69. http://dx.doi.org/10.25298/2221-8785-2024-22-5-465-469.

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Background. To evaluate changes in acid-base balance in patients with coronary heart disease in the intraoperative period during coronary artery bypass grafting under artificial circulation using extracorporeal autohemomagnetotherapy (EAHMT). To consider the presence of a negative effect of this technique on acid-base balance. To study the possible relationship between the polymorphism of the angiotensin-1 receptor gene (AGTR1 A1166C), endothelin-1 (EDN1 Lys 198 Asn), endothelial nitric oxide synthase (NOS3 C786T) and changes in the studied parameters of acid-base balance. Material and methods
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Колоцей, Л. В. "Association of Endothelial NO-Syntase Gene C786T Polymorphism with the Development of Drug-Induced Long QT Interval Syndrome Caused by Class III Antiarrhythmic Drugs." Рецепт, no. 4 (September 13, 2022): 542–55. http://dx.doi.org/10.34883/pi.2022.25.4.020.

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Цель. Установить взаимосвязь полиморфизма С786T гена эндотелиальной синтазы оксида азота (NOS3) с электрокардиографическими показателями реполяризации желудочков, а также риском развития лекарственно-индуцированного синдрома удлиненного интервала QT (СУИ QT) и неустойчивой полиморфной желудочковой тахикардии (ЖТ) на фоне приема антиаритмических препаратов III класса.Материалы и методы. В исследование включено 92 пациента: из них 75 (основная группа) – с ишемической болезнью сердца (ИБС) и/или артериальной гипертензией (АГ) и нарушениями ритма сердца, принимавших антиаритмические препараты III
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Abolhalaj, Milad, Mahsa M. Amoli, and Parvin Amiri. "eNOS Gene Variant in Patients with Coronary Artery Disease." Journal of Biomarkers 2013 (December 4, 2013): 1–6. http://dx.doi.org/10.1155/2013/403783.

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Subject & Aim. Endothelial nitric oxide synthase (eNOS) is one of the most important candidate genes in CAD. A functional polymorphism within eNOS gene is a 27 bp VNTR on its intron 4 which has been shown to be associated with various diseases. In this study we investigated eNOS VNTR polymorphism in addition to eNOS gene expression profile in patients with CAD. Material and Methods. The study comprised patients with angiographically confirmed CAD (CAD+) and individuals with normal coronary as CAD−. eNOS VNTR polymorphism frequencies were determined in both groups. In addition eNOS gene exp
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Nesen, A. O., P. S. Semenovykh, K. O. Savicheva, V. Y. ,. Galchinska, and V. L. Shkapo. "Features of prooxidant/antioxidant balance disorders in patients with diabetic nephropathy — carriers of different genotypes of G894T polymorphism of eNOS gene." Ukrainian Therapeutical Journal, no. 3 (October 7, 2024): 5–11. http://dx.doi.org/10.30978/utj2024-3-5.

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Objective — to determinate the state of prooxidant‑antioxidant balance in patients with type 2 diabetes mellitus with nephropathy — carriers of different genotypes of G894T (rs 1799983) polymorphism of eNOS gene. Materials and methods. 126 patients with diabetic nephropathy (DN) were examined. The control group included 20 healthy individuals. Patients with type 2 diabetes mellitus (DM) were divided into two groups depending on the eNOS rs 1799983 gene polymorphism: group I — patients with DN and G/G genotype eNOS G894T gene (n=80); group II — patients with DN and G/T and T/T genotypes eNOS G8
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Dosenko, Victor E., Vyacheslav Y. Zagoriy, Nikolay V. Haytovich, Olga A. Gordok, and Alexey A. Moibenko. "Allelic polymorphism of endothelial NO-synthase gene and its functional manifestations." Acta Biochimica Polonica 53, no. 2 (2006): 299–302. http://dx.doi.org/10.18388/abp.2006_3342.

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Investigation of the mechanisms of phenotypic realization of allelic polymorphism of the eNOS gene has shown that the level of eNOS mRNA and activity of this enzyme in platelets depends from genotype. We identified a T(-786)-->C polymorphism in the promoter region, a variable number of tandem repeats (4a/4b) in intron 4 and the G(894)-->T polymorphism in exon 7 of the eNOS gene in isolated human platelets. We measured eNOS mRNA in isolated platelets by reverse transcription-PCR and eNOS enzyme activity by fluorimetric detection system FCANOS-1 using diaminofluorescein diacetate (DAF-2A).
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Dissertations / Theses on the topic "Polymorphism of the gene eNOS C786T"

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Barbosa, Andreia Marcelino. "Aterosclerose: Análise do polimorfismo T786C do gene eNOS." Pontifícia Universidade Católica de Goiás, 2017. http://tede2.pucgoias.edu.br:8080/handle/tede/3701.

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Submitted by admin tede (tede@pucgoias.edu.br) on 2017-06-01T14:16:03Z No. of bitstreams: 1 ANDREIA MARCELINO BARBOSA.pdf: 784297 bytes, checksum: e401bab7d446314e9ec1076b99966617 (MD5)<br>Made available in DSpace on 2017-06-01T14:16:04Z (GMT). No. of bitstreams: 1 ANDREIA MARCELINO BARBOSA.pdf: 784297 bytes, checksum: e401bab7d446314e9ec1076b99966617 (MD5) Previous issue date: 2017-03-03<br>Coronary artery disease (CAD) is the most common form of cardiovascular disease. CAD is a multifactorial disease of complex etiology, influenced by genetic and environmental determinants. Atherosclerot
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Almeida, Filho Eduardo Eustáquio de. "ASSOCIAÇÃO DO POLIMORFISMO G894T DO GENE eNOS COM GLAUCOMA PRIMÁRIO DE ÂNGULO ABERTO." Pontifícia Universidade Católica de Goiás, 2017. http://tede2.pucgoias.edu.br:8080/handle/tede/3752.

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Submitted by admin tede (tede@pucgoias.edu.br) on 2017-09-04T12:48:49Z No. of bitstreams: 1 EDUARDO EUSTÁQUIO DE ALMEIDA FILHO.pdf: 1586241 bytes, checksum: ac6c2a57234dbdb8dcad9ea043e60dee (MD5)<br>Made available in DSpace on 2017-09-04T12:48:49Z (GMT). No. of bitstreams: 1 EDUARDO EUSTÁQUIO DE ALMEIDA FILHO.pdf: 1586241 bytes, checksum: ac6c2a57234dbdb8dcad9ea043e60dee (MD5) Previous issue date: 2017-06-12<br>Glaucoma can be defined as a progressive degeneration of the retinal nerve fiber layer that results in death of the ganglion cells, eventually causing blindness. It is the first caus
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Campedelli, Fabio Lemos. "POLIMORFISMO DO GENE eNOS G894T (Glu298Asp) EM PACIENTES SINTOMÁTICOS PARA ATEROSCLEROSE." Pontifícia Universidade Católica de Goiás, 2016. http://tede2.pucgoias.edu.br:8080/handle/tede/3554.

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Submitted by admin tede (tede@pucgoias.edu.br) on 2016-11-30T16:52:02Z No. of bitstreams: 1 FÁBIO LEMOS CAMPEDELLI.pdf: 8948383 bytes, checksum: 229016125b12d4f344bb1c157148ddad (MD5)<br>Made available in DSpace on 2016-11-30T16:52:02Z (GMT). No. of bitstreams: 1 FÁBIO LEMOS CAMPEDELLI.pdf: 8948383 bytes, checksum: 229016125b12d4f344bb1c157148ddad (MD5) Previous issue date: 2016-08-12<br>INTRODUCTION: Atherosclerotic disease (AD) with its cardiovascular complications is responsible for 17.5 million deaths a year, according to the World Health Organization (WHO). There is consensus that ath
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Park, Joon Young. "NFKB1 gene promoter polymorphism and unidirectional laminar shear stress implications for NF-kB activation, eNOS protein expression and endothelial function /." College Park, Md. : University of Maryland, 2006. http://hdl.handle.net/1903/3646.

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Thesis (Ph. D.) -- University of Maryland, College Park, 2006.<br>Thesis research directed by: Kinesiology. Title from t.p. of PDF. Includes bibliographical references. Published by UMI Dissertation Services, Ann Arbor, Mich. Also available in paper.
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Kushnir, O. V., V. P. Prysyazhnyuk, O. I. Voloshyn, et al. "Association of chronic systemic inflammation, liver cirrhosis, cancerogenesis with T894G polymorphism of endothelial nitric oxide synthase gene (eNOS) and vascular injury." Thesis, Abstracts. Inflammation and cancer.-Hamburg.-26-27 Jan., 2012, 2012. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/3766.

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Conference papers on the topic "Polymorphism of the gene eNOS C786T"

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Vlasov, A. P., S. S. Al-Kubaysi, F. A. Ali Fuad, S. T. Al-Anbari, and B. A. Fedotov. "The role of ENOS polymorphism (C774T) in the progression of acute peritonitis and the formation of complications." In VIII Vserossijskaja konferencija s mezhdunarodnym uchastiem «Mediko-fiziologicheskie problemy jekologii cheloveka». Publishing center of Ulyanovsk State University, 2021. http://dx.doi.org/10.34014/mpphe.2021-54-57.

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In order to determine the role of ENOS (C774T) gene polymorphism in the progression of acute peritonitis and the formation of complications, a clinical and biochemical study of 40 patients with acute peritonitis was conducted. As a result of the study, it was proved that the early period of acute peritonitis is characterized by the development of endogenous intoxication, intensification of oxidative phenomena, hypercoagulation of the homeostasis system and inhibition of fibrinolysis, and in patients with acute peritonitis, carriers of the pathological TT genotype of the endothelial nitric oxid
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Duaibel, Al-Karrar Kais Abdul Jaleel, Farqad Abdulelah Ali Al-Janahi, Ahmed Maki Naji, and Haider Ali Alnaji. "Glu298Asp polymorphism in exon 7 of the eNOS gene in foot ulcer of adult patients with type 2 diabetes." In 3RD INTERNATIONAL SCIENTIFIC CONFERENCE OF ALKAFEEL UNIVERSITY (ISCKU 2021). AIP Publishing, 2022. http://dx.doi.org/10.1063/5.0067092.

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