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Relevant bibliographies by topics / Porphyromonas gingivalis

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Academic literature on the topic 'Porphyromonas gingivalis'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "Porphyromonas gingivalis"

1

Britos, Maria Rosenda, Cynthya Solange Sin, Silvia Mercedes Ortega, and Olga Miriam Vasek. "Diseño y estandarización de la técnica de PCR para Porphyromonas gingivalis." Revista de la Facultad de Odontología 10, no. 1 (2017): 25. http://dx.doi.org/10.30972/rfo.1012931.

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El objetivo del presente trabajo fue diseñar y estandarizar la técnica de PCR para detección en líquido gingival de Porphyromonas gingivalis, en pacientes con enfermedad periodontal. Material y métodos: Se utilizaron iniciadores específicos para el gen ARNr 16s de Porphyromonas gingivalis. La especificidad de los iniciadores se ensayó utilizando material genético extraído de la cepa de referencia Porphyromonas gingivalis ATCC 33277. Se ajustaron las condiciones de amplificación y concentraciones de la mezcla de reacción. Para validar la técnica se aplicó a diez muestras clínicas de líquido gin

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Griffen, Ann L., Mitzi R. Becker, Sharon R. Lyons, Melvin L. Moeschberger, and Eugene J. Leys. "Prevalence of Porphyromonas gingivalisand Periodontal Health Status." Journal of Clinical Microbiology 36, no. 11 (1998): 3239–42. http://dx.doi.org/10.1128/jcm.36.11.3239-3242.1998.

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Periodontitis is a common, progressive disease that eventually affects the majority of the population. The local destruction of periodontitis is believed to result from a bacterial infection of the gingival sulcus, and several clinical studies have provided evidence to implicate Porphyromonas gingivalis. If P. gingivalis is a periodontal pathogen, it would be expected to be present in most subjects with disease and rarely detected in subjects with good periodontal health. However, in most previous studies, P. gingivalis has not been detected in the majority of subjects with disease, and age-ma

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3

Reyes, Leticia. "Porphyromonas gingivalis." Trends in Microbiology 29, no. 4 (2021): 376–77. http://dx.doi.org/10.1016/j.tim.2021.01.010.

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4

Belibasakis, Georgios, Thomas Thurnheer, and Nagihan Bostanci. "Porphyromonas gingivalis." Virulence 5, no. 4 (2014): 463–64. http://dx.doi.org/10.4161/viru.28930.

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Indah Listiana Kriswandini, Amanda Sofi Nabila Maharani, Alisya Rachel Salsabila, and Tantiana. "Mechanism of inhibiting periodontitis pathogenesis with various variations of inhibitors on Porphyromonas gingivalis: Systematic literature review." World Journal of Advanced Research and Reviews 24, no. 3 (2024): 1172–77. https://doi.org/10.30574/wjarr.2024.24.3.3717.

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Background: Periodontal disease is the most prevalent disease worldwide with a prevalence of 20% to 50%, including periodontitis. Periodontitis involves complex interactions between specific pathogenic bacteria and host immune response. Microbial infections in subgingival biofilms are mainly caused by gram-negative bacteria including Porphyromonas gingivalis. Inhibition of Porphyromonas gingivalis virulence is one way to prevent periodontitis. This inhibition can be done in several ways through peptide inhibitors, quorum sensing inhibitors and natural inhibitors. Objective: This study aims to

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Andrian, E., D. Grenier, and M. Rouabhia. "Porphyromonas gingivalis-Epithelial Cell Interactions in Periodontitis." Journal of Dental Research 85, no. 5 (2006): 392–403. http://dx.doi.org/10.1177/154405910608500502.

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Emerging data on the consequences of the interactions between invasive oral bacteria and host cells have provided new insights into the pathogenesis of periodontal disease. Indeed, modulation of the mucosal epithelial barrier by pathogenic bacteria appears to be a critical step in the initiation and progression of periodontal disease. Periodontopathogens such as Porphyromonas gingivalis have developed different strategies to perturb the structural and functional integrity of the gingival epithelium. P. gingivalis adheres to, invades, and replicates within human epithelial cells. Adhesion of P.

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Nadhifah Salsabila, Nila Kasuma, and Eti Yerizel. "Determinasi Jumlah Bakteri Porphyromonas Gingivalis ATCC 33277 pada Saliva Anak Stunting." e-GiGi 12, no. 1 (2023): 26–31. http://dx.doi.org/10.35790/eg.v12i1.47864.

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Abstract: Nutrition affects the immune system and the development of oral health, including periodontal tissue health. Stunted children experience decreased salivary flow rate causing the growth of microorganisms that cause periodontal disease. Porphyromonas gingivalis is referred to one of the keystone pathogens in the development of periodontal disease. This study aimed to identify the number of Porphyromonas gingivalis ATCC 33277 bacteria in stunted child saliva. This was a descriptive study with a cross sectional design. Prior to collection, subjects were instructed to rinse their mouth fo

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8

Zhou, Yun, Maryta Sztukowska, Qian Wang та ін. "Noncanonical Activation of β-Catenin by Porphyromonas gingivalis". Infection and Immunity 83, № 8 (2015): 3195–203. http://dx.doi.org/10.1128/iai.00302-15.

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Porphyromonas gingivalisis an established pathogen in periodontal disease and an emerging pathogen in serious systemic conditions, including some forms of cancer. We investigated the effect ofP. gingivalison β-catenin signaling, a major pathway in the control of cell proliferation and tumorigenesis. Infection of gingival epithelial cells withP. gingivalisdid not influence the phosphorylation status of β-catenin but resulted in proteolytic processing. The use of mutants deficient in gingipain production, along with gingipain-specific inhibitors, revealed that gingipain proteolytic activity was

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Darveau, Richard P., Carol M. Belton, Robert A. Reife, and Richard J. Lamont. "Local Chemokine Paralysis, a Novel Pathogenic Mechanism for Porphyromonas gingivalis." Infection and Immunity 66, no. 4 (1998): 1660–65. http://dx.doi.org/10.1128/iai.66.4.1660-1665.1998.

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ABSTRACT Periodontitis, which is widespread in the adult population, is a persistent bacterial infection associated with Porphyromonas gingivalis. Gingival epithelial cells are among the first cells encountered by both P. gingivalis and commensal oral bacteria. The chemokine interleukin 8 (IL-8), a potent chemoattractant and activator of polymorphonuclear leukocytes, was secreted by gingival epithelial cells in response to components of the normal oral flora. In contrast, P. gingivalis was found to strongly inhibit IL-8 accumulation from gingival epithelial cells. Inhibition was associated wit

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Indah, Listiana Kriswandini, Sofi Nabila Maharani Amanda, Rachel Salsabila Alisya, and Tantiana. "Mechanism of inhibiting periodontitis pathogenesis with various variations of inhibitors on Porphyromonas gingivalis: Systematic literature review." World Journal of Advanced Research and Reviews 24, no. 3 (2024): 1172–77. https://doi.org/10.5281/zenodo.15187830.

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<strong>Background: </strong>Periodontal disease is the most prevalent disease worldwide with a prevalence of 20% to 50%, including periodontitis. Periodontitis involves complex interactions between specific pathogenic bacteria and host immune response. Microbial infections in subgingival biofilms are mainly caused by gram-negative bacteria including <em>Porphyromonas gingivalis</em>. Inhibition of <em>Porphyromonas gingivalis</em> virulence is one way to prevent periodontitis. This inhibition can be done in several ways through peptide inhibitors, quorum sensing inhibitors

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Dissertations / Theses on the topic "Porphyromonas gingivalis"

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Loomer, Peter Michael. "The direct effects of Porphyromonas gingivalis 2561 on bone formation and mineral resorption in vitro." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ27685.pdf.

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Meuric, Vincent. "Virulence de Porphyromonas gingivalis." Rennes 1, 2008. http://www.theses.fr/2008REN1B197.

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"Porphyromonas gingivalis est une bactérie anaérobie stricte responsable des maladies parodontales. La respiration et la régulation des gènes de virulence restent inconnues. Ce travail explore la chaîne respiratoire et montre l'aérotolérance de la bactérie. Les résultats affirment la capacité de transmission aéroportée et inter individus par des mécanismes activant des gènes de résistance au stress oxydatif tel que "tpx" sous la dépendance d'OsyR régulateur transcriptionnel. Après une analyse de la littérature sur les principaux gènes de virulence, hémagglutinines et gingipaïnes, les études ex

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Hoppe, Tatjana [Verfasser]. "Maligne Transformation gingivaler Epithelzellen durch Porphyromonas gingivalis / Tatjana Hoppe." Bonn : Universitäts- und Landesbibliothek Bonn, 2017. http://d-nb.info/1140525883/34.

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Lobos, Matthei Ignacio Felipe. "Regulación de survivina en células epiteliales gingivales infectadas por Porphyromonas gingivalis." Tesis, Universidad de Chile, 2014. http://www.repositorio.uchile.cl/handle/2250/117229.

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Memoria para optar el título de Bioquímico<br>La periodontitis crónica es una enfermedad de etiología infecciosa, caracterizada por la respuesta inflamatoria e inmune exacerbada por parte del hospedero que produce la destrucción de tejidos de soporte del diente. Actualmente, se postula que para el establecimiento de esta patología se produce una sucesión ecológica desde una comunidad microbiana asociada a salud hacia una asociada a enfermedad periodontal. En esta sucesión es imprescindible la presencia de ciertos patógenos que faciliten el remodelamiento de la microbiota oral y la progresión h

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Bugueño, Valdebenito Isaac Maximiliano. "Caracterización de aislados clínicos de Porphyromonas gingivalis y su efecto en la viabilidad de células epiteliales gingivales." Tesis, Universidad de Chile, 2014. http://www.repositorio.uchile.cl/handle/2250/130110.

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Trabajo de Investigación Requisito para optar al Título de Cirujano Dentista<br>La periodontitis es una enfermedad infecciosa multifactorial, caracterizada por una respuesta inflamatoria exacerbada que produce la destrucción de las estructuras que dan soporte al diente, incluyendo la encía, el ligamento periodontal, el cemento radicular y el hueso alveolar. Para la iniciación de las periodontitis, se requiere la presencia de una comunidad bacteriana subgingival compleja en la cual coexisten diversos géneros y especies microbianas. Dentro de las especies del complejo rojo de Socransky qu

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Maley, Janette. "Genetic studies on Porphyromonas gingivalis W83." Thesis, University of Leicester, 1994. http://hdl.handle.net/2381/35395.

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Until recently molecular biological approaches to the study of putative virulence factors of the oral pathogen Porphyromonas gingivalis were limited. This was due to the absence of a genetic system which could facilitate the production of isogenic mutant derivatives. Among the many components produced, the capsule is believed to be of particular importance in the pathogenicity of P. gingivalis. In order to identify mutants lacking capsular polysaccharide, purified capsular material from P. gingivalis W83 was used to generate capsule-specific antisera. However, these antisera contained antibodi

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Xie, Hua. "Regulation of fimbrillin expression in Porphyromonas gingivalis." Thesis, Connect to this title online; UW restricted, 1998. http://hdl.handle.net/1773/6392.

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Rodrigues, Richelle Soares. "PrevalÃncia de Porphyromonas gingivalis, genÃtipo fima II de Porphyromonas gingivalis e Aggregatibacter actinomycetemcomitans em indivÃduos com periodontite agressiva generalizada." Universidade Federal do CearÃ, 2014. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=11486.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>Porphyromonas gingivalis e Aggregatibacter actinomycetemcomitans sÃo periodontopatÃgenos associados Ã periodontite agressiva. A fÃmbria, uma estrutura relacionada Ã adesÃo e Ã invasÃo de cÃlulas, Ã um dos principais fatores de virulÃncia de P. gingivalis. Baseado na sequÃncia de nucleotÃdeos, seis genÃtipos(fimA) que codificam a fÃmbria principal dessas bactÃrias foram identificados, sendo o fimA II mais comumente relacionado Ã destruiÃÃo periodontal. O objetivo dest

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Rodrigues, Richelle Soares. "Prevalência de Porphyromonas gingivalis, genótipo fima II de Porphyromonas gingivalis e Aggregatibacter actinomycetemcomitans em indivíduos com periodontite agressiva generalizada." reponame:Repositório Institucional da UFC, 2014. http://www.repositorio.ufc.br/handle/riufc/7904.

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RODRIGUES, Richelle Soares. Prevalência de Porphyromonas gingivalis, genótipo fima II de Porphyromonas gingivalis e Aggregatibacter actinomycetemcomitans em indivíduos com periodontite agressiva generalizada. 2014. 44 f. Dissertação (Mestrado em Odontologia) - Universidade Federal do Ceará. Faculdade de Farmácia, Odontologia e Enfermagem, Fortaleza, 2014.<br>Submitted by denise santos (denise.santos@ufc.br) on 2014-04-04T13:13:32Z No. of bitstreams: 1 2014_dis_rsrodrigues.pdf: 909297 bytes, checksum: 47ad16e66062d057016963a13334a1f2 (MD5)<br>Approved for entry into archive by denise santos(den

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Herrera, Bustamante Sergio Humberto. "Variabilidad del LPS de Porphyromonas gingivalis y Porphyromonas endodontalis en dientes con periodontitis apical asintomática." Tesis, Universidad de Chile, 2015. http://repositorio.uchile.cl/handle/2250/137646.

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Trabajo de Investigación Requisito para optar al Título de Cirujano Dentista<br>La Periodontitis Apical Asintomática (PAA) corresponde a una patología periapical de origen infeccioso que se caracteriza por la respuesta negativa a los tests de sensibilidad pulpar del diente afectado y por presentar, al examen radiográfico, un área radiolúcida peri y/o pararadicular. Histológicamente se puede clasificar como quiste radicular inflamatorio (QRI) o granuloma periapical (GP). Las comunidades microbianas asociadas a la PAA se caracterizan por infectar el sistema de canales radiculares (SCR) de

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Books on the topic "Porphyromonas gingivalis"

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N, Shah Haroun, Mayrand D, and Genco Robert J, eds. Biology of the species Porphyromonas gingivalis. CRC Press, 1993.

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Loomer, Peter Michael. The direct effects of Porphyromonas Gingivalis 2561 on bone formation and mineral resorption in vitro. University of Toronto, Faculty of Dentistry], 1997.

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Sun, Frank. Identification of Porphyromonas (Bacteroides) Gingivalis outer membrane proteins that bind to and degrade human matrix proteins. Faculty of Dentistry, University of Toronto, 1992.

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J, Genco Robert, ed. Molecular pathogenesis of periodontaldisease. ASM Press, 1994.

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J, Genco Robert, ed. Molecular pathogenesis of periodontal disease. ASM Press, 1994.

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Papaioannou, W. The Adhesion of Porphyromonas Gingivalis and Periodontitis. Leuven University Press, 1998.

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Reinert, Markus. Antimikrobielle Effekte dentaler Ultraschallscaler auf Actinobacillus actinomycetemcomitans, Porphyromonas gingivalis und Streptococcus mutans in vitro. 2006.

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Beekmann, Johanna. Auswirkungen von Porphyromonas gingivalis: Kulturüberstand auf die Gesamtzellfettsäurezusammensetzung von KB-Zellen und humanen Gingivakeratinozyten. 2005.

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Andreou, Vana. Resveratrol mediated reversal of aryl hydrocarbon and porphyromonas gingivalis lipopolysaccharide induced inhibition of osteogenesis in vitro. 2002.

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Immunization with Porphyromonas Gingivalis Cysteine Protease: Effects on Experimental Gingivitis and Ligature-Induced Periodontitis in Macaca Fascicularis. Storming Media, 1997.

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Book chapters on the topic "Porphyromonas gingivalis"

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Genco, Caroline Attardo, Waltena Simpson, and Teresa Olczak. "Porphyromonas gingivalis." In Iron Transport in Bacteria. ASM Press, 2014. http://dx.doi.org/10.1128/9781555816544.ch21.

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Takeuchi, Hiroki, and Atsuo Amano. "Invasion of Gingival Epithelial Cells by Porphyromonas gingivalis." In Periodontal Pathogens. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_21.

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Yongqing, Tang, Jan Potempa, Robert N. Pike, and Lakshmi C. Wijeyewickrema. "The Lysine-Specific Gingipain of Porphyromonas gingivalis." In Advances in Experimental Medicine and Biology. Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-8414-2_2.

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Arndt, Annette, and Mary Ellen Davey. "Porphyromonas gingivalis: surface polysaccharides as virulence determinants." In Interface Oral Health Science 2009. Springer Japan, 2010. http://dx.doi.org/10.1007/978-4-431-99644-6_111.

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McBride, Barry C., Umadatt Singh, and Angela Joe. "Porphyromonas gingivalis: Gene Cloning of Determinants of Pathogenicity." In Brock/Springer Series in Contemporary Bioscience. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-7087-5_41.

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Amano, Atsuo, Youn-Hee Choi, and Hiroki Takeuchi. "Genotyping of Porphyromonas gingivalis in Relationship to Virulence." In Periodontal Pathogens. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_6.

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Hasegawa, Yoshiaki, Keiji Nagano, Yukitaka Murakami, and Richard J. Lamont. "Purification of Native Mfa1 Fimbriae from Porphyromonas gingivalis." In Periodontal Pathogens. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_8.

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Heidler, Thomas, and Karina Persson. "Crystallization of Recombinant Fimbrial Proteins of Porphyromonas gingivalis." In Periodontal Pathogens. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_9.

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Kadowaki, Tomoko. "Enzymatic Characteristics and Activities of Gingipains from Porphyromonas gingivalis." In Periodontal Pathogens. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_10.

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Yoshida, Yasuo. "Analysis of the Butyrate-Producing Pathway in Porphyromonas gingivalis." In Periodontal Pathogens. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_16.

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Conference papers on the topic "Porphyromonas gingivalis"

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Lee, Seohyun, Hideaki Ota, Hideo Higuchi, Takehiro Yamaguchi, and Ryoma Nakao. "Image Processing Algorithm for Porphyromonas gingivalis Outer Membrane Vesicle Transport in Periodontal Pathogenesis^*." In 2024 IEEE 17th International Conference on Nano/Molecular Medicine & Engineering (NANOMED). IEEE, 2024. https://doi.org/10.1109/nanomed64244.2024.10946030.

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De la Garza-Ramos, M. A., A. Alcázar-Pizaña, M. Garza-Enriquez, et al. "Streptococcus intermedius trigger quorum-sensing genes in Porphyromonas gingivalis." In Proceedings of the III International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2009). WORLD SCIENTIFIC, 2010. http://dx.doi.org/10.1142/9789814322119_0113.

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Sieron, Aleksander, Andrzej Wiczkowski, Mariusz Adamek, Lucja Dyla, Sebastian Mazur, and Beata Wierucka-Mlynarczyk. "Photodynamic destruction of Porphyromonas gingivalis induced by delta-aminolaevulinic acid." In SPIE Proceedings, edited by Leonardo Longo, Alfons G. Hofstetter, Mihail-Lucian Pascu, and Wilhelm R. A. Waidelich. SPIE, 2004. http://dx.doi.org/10.1117/12.584427.

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Mahasneh, Arwa M., Michele Darby, Lynn Tolle, Mounir Laroussi, and Wayne Hynes. "Bactericidal effects of low temperature atmospheric pressure plasma on Porphyromonas gingivalis." In 2010 IEEE 37th International Conference on Plasma Sciences (ICOPS). IEEE, 2010. http://dx.doi.org/10.1109/plasma.2010.5534217.

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Mechid, Farida, Houda Hafirassou, Malika Meddad, et al. "AB0255 CLINICO- BIOLOGICAL PROFILE OF RHEUMATOID ARTHRITIS WITH PERIODONTITIS AND PORPHYROMONAS GINGIVALIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3342.

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Li, Q., M. Chang, K. E. Goggin, et al. "Porphyromonas Gingivalis Mediated T17 Cell Polarization Promotes Lung Cancer Resistance to Immunotherapy." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a6898.

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Andriani, Ika, Ana Medawati, Sri Tasminatun, Citra Lestari, Riefa Ayu Destavia, and Modista Yuarindias. "Effectiveness of clindamycin and doxycycline antibiotics against Porphyromonas gingivalis bacteria causing periodontitis." In THE 2ND NEW DENTAL RESEARCH EXHIBITION AND MEETING (NEW DREAM) 2024. AIP Publishing, 2025. https://doi.org/10.1063/5.0273825.

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Jenning, M., B. Marklein, Z. Konthur, U. Nonhoff, G.-R. Burmester, and K. Skriner. "P043 Infection with citrullinating porphyromonas gingivalis can induce autoimmunity to human ribosomal proteins." In 39th European Workshop for Rheumatology Research, 28 February–2 March 2019, Lyon, France. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2019.35.

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De La Garza-Ramos, M. A., A. L. Luna-Ayala, M. Garza-Enriquez, D. S. Martinez-Carreon, B. Pereyra-Alférez, and R. G. Caffesse. "Streptococcus intermedius can module the expression of some virulence factors in Porphyromonas gingivalis." In MICROBES IN APPLIED RESEARCH - Current Advances and Challenges. WORLD SCIENTIFIC, 2012. http://dx.doi.org/10.1142/9789814405041_0116.

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Wang, Zhi, Xi Wang, and Yiqun Jia. "Abstract 2358: Porphyromonas gingivalis promotes colorectal cancer development by regulating NLRP3 inflammasome signaling." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2358.

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Reports on the topic "Porphyromonas gingivalis"

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จรัสกุลางกูร, อรวรรณ, กนกวรรณ นิสภกุลธร та รังสินี มหานนท์. การตรวจหาเชื้อแบคทีเรียก่อโรคปริทันต์อักเสบเรื้อรังและเป็นโรคเบาหวานชนิดที่ 2 ที่ควบคุมระดับน้ำตาลในเลือดได้ไม่ดี : รายงานผลการวิจัย. จุฬาลงกรณ์มหาวิทยาลัย, 2009. https://doi.org/10.58837/chula.res.2009.12.

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ผู้ป่วยโรคเบาหวานที่ไม่สามารถควบคุมระดับน้ำตาลในเลือดได้ดี มีความเสี่ยงต่อการสูญเสียระดับการยึดเกาะของอวัยวะปริทันต์และการละลายของกระดูกเบ้าฟันมากขึ้น การเปลี่ยนแปลงสมดุลของเชื้อแบคทีเรียใต้เหงือกอาจเป็นสาเหตุในการเพิ่มความรุนแรงของโรคปริทันต์อักเสบในผู้ป่วยกลุ่มนี้ การวิจัยนี้มีวัตถุประสงค์เพื่อหาความชุกของเชื้อแบคทีเรีย Porphyromonas qingivalis, Tannerella forsythia, Treponema denticola และ Prevotella intermedia ในแผ่นคราบจุลินทรีย์ใต้เหงือกของผู้ป่วยโรคเบาหวานชนิดที่ 2 ที่ควบคุมระดับน้ำตาลในเลือดได้ไม่ดีเปรียบเทียบกับผู้ป่วยที่ไม่เป็นโรคเบาหวาน โดยศึกษาในผู้ป่วยโรคปริทันต์อักเสบเรื้อรังทั่ว

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