Relevant bibliographies by topics / Progenitor Fate

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Progenitor Fate'

Author: Grafiati
Published: 24 May 2025
Last updated: 31 July 2025

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Journal articles on the topic "Progenitor Fate"

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Chen, Lipeng, Qing Sun, Jihao Zhou, et al. "New Insights into Hematopoietic Redefinition and New Paradigms in Hematopoietic Hierarchy Roadmap." Blood 142, Supplement 1 (2023): 4057. http://dx.doi.org/10.1182/blood-2023-173249.

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The definition of hematopoietic progenitor cells (HPCs) has not been standardized. Surface markers are insufficient to capture the entire differentiation stages of progenitor cells and reflect the dynamic differentiation mechanisms underlying hematopoiesis. Deciphering the hematopoiesis mechanism that underlies progenitor cells definition and fate decision are critical to understanding fundamental questions of hematopoietic lineage commitment. Here, we redefined the entire spectrum of adult hematopoietic progenitors, and recovered the critical transcript factors (TFs) in lineages fate decision
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Jagla, T., F. Bellard, Y. Lutz, G. Dretzen, M. Bellard, and K. Jagla. "ladybird determines cell fate decisions during diversification of Drosophila somatic muscles." Development 125, no. 18 (1998): 3699–708. http://dx.doi.org/10.1242/dev.125.18.3699.

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In the mesoderm of Drosophila embryos, a defined number of cells segregate as progenitors of individual body wall muscles. Progenitors and their progeny founder cells display lineage-specific expression of transcription factors but the mechanisms that regulate their unique identities are poorly understood. Here we show that the homeobox genes ladybird early and ladybird late are expressed in only one muscle progenitor and its progeny: the segmental border muscle founder cell and two precursors of adult muscles. The segregation of the ladybird-positive progenitor requires coordinate action of n
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Morrow, Theresa, Mi-Ryoung Song, and Anirvan Ghosh. "Sequential specification of neurons and glia by developmentally regulated extracellular factors." Development 128, no. 18 (2001): 3585–94. http://dx.doi.org/10.1242/dev.128.18.3585.

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Cortical progenitor cells give rise to neurons during embryonic development and to glia after birth. While lineage studies indicate that multipotent progenitor cells are capable of generating both neurons and glia, the role of extracellular signals in regulating the sequential differentiation of these cells is poorly understood. To investigate how factors in the developing cortex might influence cell fate, we developed a cortical slice overlay assay in which cortical progenitor cells are cultured over cortical slices from different developmental stages. We find that embryonic cortical progenit
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Yang, Yun, Hao Wang, Jia He, et al. "A single-cell–resolution fate map of endoderm reveals demarcation of pancreatic progenitors by cell cycle." Proceedings of the National Academy of Sciences 118, no. 25 (2021): e2025793118. http://dx.doi.org/10.1073/pnas.2025793118.

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A progenitor cell could generate a certain type or multiple types of descendant cells during embryonic development. To make all the descendant cell types and developmental trajectories of every single progenitor cell clear remains an ultimate goal in developmental biology. Characterizations of descendant cells produced by each uncommitted progenitor for a full germ layer represent a big step toward the goal. Here, we focus on early foregut endoderm, which generates foregut digestive organs, including the pancreas, liver, foregut, and ductal system, through distinct lineages. Using unbiased sin
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Yuan, Yuan, Yong-hwee Eddie Loh, Xia Han, et al. "Spatiotemporal cellular movement and fate decisions during first pharyngeal arch morphogenesis." Science Advances 6, no. 51 (2020): eabb0119. http://dx.doi.org/10.1126/sciadv.abb0119.

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Cranial neural crest (CNC) cells contribute to different cell types during embryonic development. It is unknown whether postmigratory CNC cells undergo dynamic cellular movement and how the process of cell fate decision occurs within the first pharyngeal arch (FPA). Our investigations demonstrate notable heterogeneity within the CNC cells, refine the patterning domains, and identify progenitor cells within the FPA. These progenitor cells undergo fate bifurcation that separates them into common progenitors and mesenchymal cells, which are characterized by Cdk1 and Spry2/Notch2 expression, respe
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Sánchez-González, Rebeca, María Figueres-Oñate, Ana Cristina Ojalvo-Sanz, and Laura López-Mascaraque. "Cell Progeny in the Olfactory Bulb after Targeting Specific Progenitors with Different UbC-StarTrack Approaches." Genes 11, no. 3 (2020): 305. http://dx.doi.org/10.3390/genes11030305.

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The large phenotypic variation in the olfactory bulb may be related to heterogeneity in the progenitor cells. Accordingly, the progeny of subventricular zone (SVZ) progenitor cells that are destined for the olfactory bulb is of particular interest, specifically as there are many facets of these progenitors and their molecular profiles remain unknown. Using modified StarTrack genetic tracing strategies, specific SVZ progenitor cells were targeted in E12 mice embryos, and the cell fate of these neural progenitors was determined in the adult olfactory bulb. This study defined the distribution and
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Desai, A. R., and S. K. McConnell. "Progressive restriction in fate potential by neural progenitors during cerebral cortical development." Development 127, no. 13 (2000): 2863–72. http://dx.doi.org/10.1242/dev.127.13.2863.

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During early stages of cerebral cortical development, progenitor cells in the ventricular zone are multipotent, producing neurons of many layers over successive cell divisions. The laminar fate of their progeny depends on environmental cues to which the cells respond prior to mitosis. By the end of neurogenesis, however, progenitors are lineally committed to producing upper-layer neurons. Here we assess the laminar fate potential of progenitors at a middle stage of cortical development. The progenitors of layer 4 neurons were first transplanted into older brains in which layer 2/3 was being ge
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Belliveau, M. J., and C. L. Cepko. "Extrinsic and intrinsic factors control the genesis of amacrine and cone cells in the rat retina." Development 126, no. 3 (1999): 555–66. http://dx.doi.org/10.1242/dev.126.3.555.

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The seven major classes of cells of the vertebrate neural retina are generated from a pool of multipotent progenitor cells. Recent studies suggest a model of retinal development in which both the progenitor cells and the environment change over time (Cepko, C. L., Austin, C. P., Yang, X., Alexiades, M. and Ezzeddine, D. (1996). Proc. Natl. Acad. Sci. USA 93, 589–595). We have utilized a reaggregate culture system to test this model. A labeled population of progenitors from the embryonic rat retina were cultured with an excess of postnatal retinal cells and then assayed for their cell fate choi
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Olmos-Carreño, Cindy L., María Figueres-Oñate, Gabriel E. Scicolone, and Laura López-Mascaraque. "Cell Fate of Retinal Progenitor Cells: In Ovo UbC-StarTrack Analysis." International Journal of Molecular Sciences 23, no. 20 (2022): 12388. http://dx.doi.org/10.3390/ijms232012388.

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Clonal cell analysis outlines the ontogenic potential of single progenitor cells, allowing the elucidation of the neural heterogeneity among different cell types and their lineages. In this work, we analyze the potency of retinal stem/progenitor cells through development using the chick embryo as a model. We implemented in ovo the clonal genetic tracing strategy UbC-StarTrack for tracking retinal cell lineages derived from individual progenitors of the ciliary margin at E3.5 (HH21-22). The clonal assignment of the derived-cell progeny was performed in the neural retina at E11.5-12 (HH38) throu
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Soukup, Alexandra, Kirby D. Johnson, Daniel J. Conn, et al. "GATA2-Dependent Developmental and Regenerative Networks." Blood 134, Supplement_1 (2019): 1182. http://dx.doi.org/10.1182/blood-2019-126875.

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Coding and regulatory human GATA2 mutations that deregulate protein expression and/or function cause immunodeficiency that often progresses to MDS/AML (McReynolds et al., 2018). In the mouse, decreased GATA2 expression impairs hematopoietic stem/progenitor cell (HSPC) genesis and function (de Pater et al., 2013; Gao et al., 2013; Tsai et al., 1994). While prior studies demonstrated Gata2 +9.5 and -77 enhancers are essential for HSC emergence (+9.5) and/or progenitor cell fate (+9.5 and -77) (Johnson et al., 2012; Johnson et al., 2015; Mehta et al., 2017) and hematopoietic regeneration (+9.5) (
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Dissertations / Theses on the topic "Progenitor Fate"

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Premarathne, Susitha. "Role of Deubiquitylating Enzyme USP9X in Neural Progenitor Fate Determination." Thesis, Griffith University, 2017. http://hdl.handle.net/10072/367800.

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During brain development, neural progenitors (NPs) need to balance their self-renewal with differentiation, in order to maintain the NP population while establishing the complex tissue architecture of the brain. NP fate is under the close scrutiny of plethora of fate determination factors, which can be divided into two groups based on their site of origin namely, intrinsic and extrinsic fate determinants. To date, a number of intrinsic factors, such as cell polarity and adhesion, and extrinsic factors including Notch, WNT and mTOR signaling pathways have been shown to regulate NP fate specific
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Toeg, Hadi D. "Role of connexin 30 in directing adult neural progenitor cell fate." Thesis, University of Ottawa (Canada), 2009. http://hdl.handle.net/10393/27806.

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This thesis tested the hypothesis that the gap junction protein connexin 30 (Cx30) plays a role in regulating adult neural progenitor cell (NPC) fate. Cx30, previously shown to be expressed by postnatal astrocytes, was localized, for the first time, to adult NPCs specifically to a subset of multipotential nestin+/glial fibrillary acidic protein + (GFAP)+ NPCs in the subventricular zone (SVZ) of adult mice. When bromodeoxyuridine (BrdU) labelling was performed in Cx30 (-/-) mice, the transition of early NPCs to a neuronal lineage was reduced. Increased BrdU cell number in the rostral migratory
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Olabi, Safiah. "The role of β1-integrin in mammary stem and progenitor fate". Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-1integrin-in-mammary-stem-and-progenitor-fate(ce5b50f2-afaf-4cf7-a331-a429475b6cea).html.

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The mammary gland contains a subset of cells with regenerative capacity that is able to generate both luminal and myoepithelial mammary epithelial lineages. Those cells are described as mammary epithelial stem cells. The fate of stem cells is tightly controlled by their microenvironment and adhesion receptors on the stem cells play a vital role in the microenvironment–stem cell communication. They facilitate the interaction of stem cells with the extracellular matrix as well as adjacent cells, and they regulate stem cell homing to their niches, as well as stem cell proliferation, self-renewal,
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Forde, Sinead. "The role of the human sialomucin CD164 in regulating stem/progenitor cell fate." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.442950.

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Melanson-Drapeau, Lysanne. "Connexin32-mediated control of progenitor cell fate in injured and uninjured adult mouse brain." Thesis, University of Ottawa (Canada), 2006. http://hdl.handle.net/10393/29364.

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Gap junction protein expression has been implicated in progenitor cell proliferation, survival, and specification during development. The present study was undertaken to establish the role of the gap junction protein connexin32 in dictating progenitor cell fate in adult mouse brain. In the dentate gyrus of the hippocampus, I localized the connexin32 protein to a subset of NG2 + early oligodendrocyte progenitors. In connexin32-null mutant mice, I found an increase in the total number of proliferating early oligodendrocyte progenitors and demonstrated that the turnover of these cells is constitu
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Roubelakis, Maria G. "The role of SHP-2 and its partners in regulating stem/progenitor cell fate." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400441.

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Lutteropp, Michael. "The emergence and early fate decisions of stem and progenitor cells in the haematopoietic system." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:eef3e876-bde2-4114-8ac2-bf0c87492a55.

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The alternative road map describes the separation of lympho-myeloid and myeloid-megakaryocyte-erythroid (myeloid-Mk-E) lineages as the earliest haematopoietic commitment event. However, a number of aspects of this lineage restriction process remain poorly understood. Herein this work identified a lympho-myeloid restricted progenitor in the embryo, which resembles the adult LMPP, and demonstrated that lymphoid lineage restriction is initiated prior to definitive haematopoiesis, much earlier than previously appreciated. In vivo fate mapping showed that lympho-myeloid progenitors significantly co
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Tanigaki, Kenji. "Notch1 and Notch3 instructively restrict bFGF-responsive multipotent neural progenitor cells to an astroglial fate." Kyoto University, 2001. http://hdl.handle.net/2433/150564.

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Stigloher, Christian. "Specification, maintenance and fate determination of neural progenitor pools in the zebrafish central nervous system." kostenfrrei, 2008. http://mediatum2.ub.tum.de/node?id=652211.

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Horky, Laura L. "Fate of endogenous neural stem/progenitor cells following spinal cord injury in the adult mammal /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2003. http://wwwlib.umi.com/cr/ucsd/fullcit?p3112823.

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Books on the topic "Progenitor Fate"

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Kratchmarov, Radomir. Metabolism regulates cell fate in lymphocytes and progenitor cells. [publisher not identified], 2018.

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Fulcoli, F. Gabriella, and Antonio Baldini. Transcriptional regulation of early cardiovascular development. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0006.

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The two major cardiac cell lineages of the vertebrate heart, the first and second cardiac fields (FHF and SHF), have different developmental ontogeny and thus different transcription programs. Most remarkably, the fate of cardiac progenitors (CPs) of the FHF is restricted to cardiomyocyte differentiation. In contrast, SHF CPs, which are specified independently, are maintained in a multipotent state for a relatively longer developmental time and can differentiate into multiple cell types. The identity of the transcription factors and regulatory elements involved in progenitor cell programming a
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Dettman, Robert, Juan Antonio Guadix, Elena Cano, Rita Carmona, and Ramón Muñoz-Chápuli. The multiple functions of the proepicardial/epicardial cell lineage in heart development. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0020.

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The epicardium is the outer cell layer of the vertebrate heart. In recent years, both the embryonic and adult epicardium have revealed unsuspected peculiarities and functions, which are essential for cardiac development. In this chapter we review the current literature on the epicardium, and describe its evolutionary origin, the mechanisms leading to the induction of its extracardiac progenitor tissue, the proepicardium, and the way in which the proepicardium is transferred to the heart to form the epicardium. We also describe the epicardial epithelial–mesenchymal transition from which mesench
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Lopez-Sanchez, Carmen, Virginio Garcia-Lopez, Gary C. Schoenwolf, and Virginio Garcia-Martinez. From epiblast to mesoderm: elaboration of a fate map for cardiovascular progenitors. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0003.

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The origin and migration of cardiovascular progenitors have been identified using multiple cell fate mapping techniques monitoring marked epiblast cells through time at carefully defined stages of early gastrulation. These studies have revealed that ordered groups of cells from the epiblast move into the anterior region of the primitive streak, and then migrate anterior laterally to define the first heart field in the mesodermal layer. Subsequently, the right and left components of the first heart field fuse into a single straight heart at the embryonic midline. Additional cells derived from t
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Cahill, Thomas J., and Paul R. Riley. Epicardial and coronary vascular development. Edited by Miguel Torres. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0009.

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The coronary circulation is essential for human life. In embryonic development, abnormal formation of the coronary vasculature can cause death in utero or after birth. In adulthood, atherosclerosis of the coronary arteries is the commonest cause of death worldwide. The last decade has witnessed significant strides forward in our understanding of coronary development. Multiple sources of coronary endothelial cells have been identified using genetic tools for fate mapping. The epicardium, the outermost layer of the developing heart, has emerged as both a source of cell progenitors and key signal
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Miquerol, Lucile. Origin and development of the cardiac conduction system. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0015.

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The cardiac conduction system represents the ‘wiring’ of the heart and orchestrates the propagation of the electrical activity to synchronize heartbeats. It is built from specialized cardiomyocytes expressing a subset of ion channels and gap junctions indispensable for their electrophysiological properties. Although representing only a very small volume of the heart, the conduction system plays a crucial role in the appearance of cardiac arrhythmias. The cells forming the conduction system are derived from the same cardiac progenitors as the working cardiomyocytes, and the choice between these
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Anooshahr, Ali. Turkestan and the Rise of Eurasian Empires. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190693565.001.0001.

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It has long been known that the origins of the early modern dynasties of the Ottomans, Safavids, Mughals, Mongols, and Shibanids in the sixteenth century go back to “Turco-Mongol” or “Turcophone” war bands. However, too often has this connection been taken at face value, usually along the lines of ethnolinguistic continuity. The connection between a mythologized “Turkestani” or “Turco-Mongol” origin and these dynasties was not simply and objectively present as fact. Rather, much creative energy was unleashed by courtiers and leaders from Bosnia to Bihar (with Bukhara and Badakhshan along the w
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Aplicación de ciencia tecnología e innovación en el cultivo del café ajustado a las condiciones particulares del Huila: Vol. 2. 2015-2021. Cenicafé, 2021. http://dx.doi.org/10.38141/cenbook-0008.

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En la segunda fase del proyecto (2019-2021) se dio continuidad a tres investigaciones, cuyos resultados están descritos en el presente libro. En esta entrega se describen los logros alcanzados en la caracterizaron morfológica, agronómica y fisiológica, en dos ambientes contrastantes de la caficultura, de 50 accesiones de la Colección Colombiana de Café, que representan la diversidad que está disponible para el desarrollo de variedades de Coffea arabica, y que se denomina Colección Núcleo. Se identificaron accesiones etíopes silvestres que se destacan por su buen comportamiento bajo las condici
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Book chapters on the topic "Progenitor Fate"

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Lilliehook, Christina, and Steven A. Goldman. "Microvascular Influences on Progenitor Cell Mobilization and Fate in the Adult Brain." In Blood-Brain Barriers. Wiley-VCH Verlag GmbH & Co. KGaA, 2007. http://dx.doi.org/10.1002/9783527611225.ch3.

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Lau, Anne-Marie, Yu-Hua Tseng, and Tim J. Schulz. "Adipogenic Fate Commitment of Muscle-Derived Progenitor Cells: Isolation, Culture, and Differentiation." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1453-1_19.

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Herrero, Diego, Susana Cañón, Guillermo Albericio, et al. "Oxidative Stress as a Critical Determinant of Adult Cardiac Progenitor Cell-Fate Decisions." In Modulation of Oxidative Stress in Heart Disease. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8946-7_13.

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Smith, Gilbert H., and Corinne A. Boulanger. "Techniques for the Reprogramming of Exogenous Stem/Progenitor Cell Populations Towards a Mammary Epithelial Cell Fate." In Methods in Molecular Biology. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6475-8_14.

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Babaei, Rohollah, Irem Bayindir-Buchhalter, Irina Meln, and Alexandros Vegiopoulos. "Immuno-Magnetic Isolation and Thermogenic Differentiation of White Adipose Tissue Progenitor Cells." In Thermogenic Fat. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6820-6_5.

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Yoshimura, Kotaro. "Cell-Assisted Lipotransfer for Breast Augmentation: Grafting of Progenitor-Enriched Fat Tissue." In Autologous Fat Transfer. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-00473-5_34.

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Steinbring, Jochen, Antonia Graja, Anne-Marie Jank, and Tim J. Schulz. "Flow Cytometric Isolation and Differentiation of Adipogenic Progenitor Cells into Brown and Brite/Beige Adipocytes." In Thermogenic Fat. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6820-6_4.

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Milner, Laurie A., and Anna Bigas. "Notch in Hematopoiesis: Cell Fate Decisions and Self-Renewal of Progenitors." In Cell Therapy. Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-68506-7_10.

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Mina, Mina. "Strategies for Tracking the Origin and Fate of Odontoblasts and Pulp Cell Progenitors." In The Dental Pulp. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-55160-4_4.

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Thuret, Raphaël, and Nancy Papalopulu. "Following the Fate of Neural Progenitors by Homotopic/Homochronic Grafts in Xenopus Embryos." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-980-8_16.

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Conference papers on the topic "Progenitor Fate"

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Antognazza, Maria Rosa. "Conjugated polymers optically regulate the fate of Endothelial Progenitor Cells." In nanoGe Spring Meeting 2022. Fundació Scito, 2022. http://dx.doi.org/10.29363/nanoge.nsm.2022.245.

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Wang, Tongpeng, Peipei Zhou, and Ruiqi Wang. "Cell commitment motif composed of progenitor-specific TF and fate-decision motif." In 2013 7th International Conference on Systems Biology (ISB). IEEE, 2013. http://dx.doi.org/10.1109/isb.2013.6623811.

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Gore, Kalea, and Deanna M. Thompson. "Effect of flow-stimulated vascular endothelial cell extracellular matrix on neural progenitor cell fate." In 2015 41st Annual Northeast Biomedical Engineering Conference (NEBEC). IEEE, 2015. http://dx.doi.org/10.1109/nebec.2015.7117100.

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Joerger-Messerli, M., M. Spinelli, G. Thomi, et al. "Exosomes derived from Wharton's jelly stem cells switch neural progenitor cell fate to oligodendrogenesis." In 62. Kongress der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe – DGGG'18. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1671425.

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Lee, S. N., J. S. Ahn, S. Kim, J. G. Ha, and J. H. Yoon. "Proprotein Convertase PACE4 Modulates Basal Progenitor Cell Fate in IL-4-Stimulated Human Nasal Epithelial Cells." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2167.

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Lin, T., S. Wang, R. Feng, et al. "Activin-driven fate determination is pivotal for liver progenitor cells to take over hepatocytic function in in ACLF." In 36. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402106.

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Haddad, I., A. Sogge, K. Cook, A. Toth, and W. Zacharias. "Peroxisome Proliferator-Activated Receptor Gamma Function in Progenitor Alveolar Type 2 Cells Is Required for Alveolar Organoid Formation, Cellular Fate Specification, and Morphological Maturation." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a6645.

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Schmit, Travis L., Vijayasaradhi Setaluri, Vladimir S. Spiegelman, and Nihal Ahmad. "Abstract 1064: Numb, a progenitor cell fate determinant and intrinsic inhibitor of Notch signaling, is cell cycle regulated and required for proper mitotic entry and progression in melanoma cells." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1064.

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Okhalnik, A. D., M. S. Gavrish, S. A. Tutukov, and V. S. Tarabykin. "ROLE OF KCNQ3 IN THE FORMATION OF THE CEREBRAL CORTEX AND THE CORPUS CALLOSUM." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-264.

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Abstract:
Identification of genes involved in the control of the cerebral cortex development is an an important task. Together with colleagues, we assessed the contribution of the voltage-gated K+ channel subunit (Kcnq3) to the formation of the corpus callosum, as well as its role in the migration and determination of the cell fate of neuronal progenitors.
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Humeniuk, Rita, Michael Rosu-Myles, and Linda Wolff. "Abstract 1973: Tumor suppressor p15Ink4b determines cell fate of hematopoietic progenitors: Implications for development of human blood disorders." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-1973.

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Reports on the topic "Progenitor Fate"

1

Crowe, David. Progenitor Cell Fate Decisions in Mammary Tumorigenesis. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada542770.

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2

Halevy, Orna, Sandra Velleman, and Shlomo Yahav. Early post-hatch thermal stress effects on broiler muscle development and performance. United States Department of Agriculture, 2013. http://dx.doi.org/10.32747/2013.7597933.bard.

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In broilers, the immediate post-hatch handling period exposes chicks to cold or hot thermal stress, with potentially harmful consequences to product quantity and quality that could threaten poultry meat marketability as a healthy, low-fat food. This lower performance includes adverse effects on muscle growth and damage to muscle structure (e.g., less protein and more fat deposition). A leading candidate for mediating the effects of thermal stress on muscle growth and development is a unique group of skeletal muscle cells known as adult myoblasts (satellite cells). Satellite cells are multipote
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