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Relevant bibliographies by topics / Prostaglandins E Hydroquinone Glutathione

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Academic literature on the topic 'Prostaglandins E Hydroquinone Glutathione'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 February 2022

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Journal articles on the topic "Prostaglandins E Hydroquinone Glutathione"

1

ATROSHI, F., J. PARANTAINEN, S. SANKARI, and T. ÖSTERMAN. "Prostaglandins and glutathione peroxidase in bovine mastitis." Research in Veterinary Science 40, no. 3 (1986): 361–66. http://dx.doi.org/10.1016/s0034-5288(18)30551-4.

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Uchida, Koji. "Induction of glutathione S-transferase by prostaglandins." Mechanisms of Ageing and Development 116, no. 2-3 (2000): 135–40. http://dx.doi.org/10.1016/s0047-6374(00)00129-9.

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Xun, Luying, Sara M. Belchik, Randy Xun, et al. "S-Glutathionyl-(chloro)hydroquinone reductases: a novel class of glutathione transferases." Biochemical Journal 428, no. 3 (2010): 419–27. http://dx.doi.org/10.1042/bj20091863.

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Sphingobium chlorophenolicum completely mineralizes PCP (pentachlorophenol). Two GSTs (glutathione transferases), PcpC and PcpF, are involved in the degradation. PcpC uses GSH to reduce TeCH (tetrachloro-p-hydroquinone) to TriCH (trichloro-p-hydroquinone) and then to DiCH (dichloro-p-hydroquinone) during PCP degradation. However, oxidatively damaged PcpC produces GS-TriCH (S-glutathionyl-TriCH) and GS-DiCH (S-glutathionyl-TriCH) conjugates. PcpF converts the conjugates into TriCH and DiCH, re-entering the degradation pathway. PcpF was further characterized in the present study. It catalysed GS

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Green, Abigail R., Robert P. Hayes, Luying Xun, and ChulHee Kang. "Structural Understanding of the Glutathione-dependent Reduction Mechanism of Glutathionyl-Hydroquinone Reductases." Journal of Biological Chemistry 287, no. 43 (2012): 35838–48. http://dx.doi.org/10.1074/jbc.m112.395541.

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Puckett-Vaughn, DeAnna L., Julie A. Stenken, Dennis O. Scott, Susan M. Lunte, and Craig E. Lunte. "Enzymatic formation and electrochemical characterization of multiply substituted glutathione conjugates of hydroquinone." Life Sciences 52, no. 14 (1993): 1239–47. http://dx.doi.org/10.1016/0024-3205(93)90107-e.

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Huang, Yan, Randy Xun, Guanjun Chen, and Luying Xun. "Maintenance Role of a Glutathionyl-Hydroquinone Lyase (PcpF) in Pentachlorophenol Degradation by Sphingobium chlorophenolicum ATCC 39723." Journal of Bacteriology 190, no. 23 (2008): 7595–600. http://dx.doi.org/10.1128/jb.00489-08.

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ABSTRACT Pentachlorophenol (PCP) is a toxic pollutant. Its biodegradation has been extensively studied in Sphingobium chlorophenolicum ATCC 39723. All enzymes required to convert PCP to a common metabolic intermediate before entering the tricarboxylic acid cycle have been characterized. One of the enzymes is tetrachloro-p-hydroquinone (TeCH) reductive dehalogenase (PcpC), which is a glutathione (GSH) S-transferase (GST). PcpC catalyzes the GSH-dependent conversion of TeCH to trichloro-p-hydroquinone (TriCH) and then to dichloro-p-hydroquinone (DiCH) in the PCP degradation pathway. PcpC is susc

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Solecki, Gergely Morten, Isabel Anna Maria Groh, Julia Kajzar, et al. "Genotoxic Properties of Cyclopentenone Prostaglandins and the Onset of Glutathione Depletion." Chemical Research in Toxicology 26, no. 2 (2013): 252–61. http://dx.doi.org/10.1021/tx300435p.

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Low, Lawrence K., Charles E. Lambert, J. Ralph Meeks, Paul A. Naro, and Carl R. Mackerer. "Tissue-Specific Metabolism of Benzene in Zymbal Gland and Other Solid Tumor Target Tissues in Rats." Journal of the American College of Toxicology 14, no. 1 (1995): 40–60. http://dx.doi.org/10.3109/10915819509008680.

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In vitro studies were carried out to investigate whether target organ susceptibility to benzene-induced solid tumor formation is governed by tissue-specific differences in metabolism. The ability of several target and nontarget tissues to deconjugate and conjugate polar metabolites, to metabolize benzene to phenolic metabolites, to carry out peroxidative biotransformations, and to trap tissue glutathione was evaluated. The Zymbal gland, the organ most sensitive to benzene-induced tumorigenicity, showed extensive phenyl- and aryl-sulfatase activity but no phenol sulfoconjugating activity. Simil

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Hayes, John D., Jack U. Flanagan, and Ian R. Jowsey. "GLUTATHIONE TRANSFERASES." Annual Review of Pharmacology and Toxicology 45, no. 1 (2005): 51–88. http://dx.doi.org/10.1146/annurev.pharmtox.45.120403.095857.

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This review describes the three mammalian glutathione transferase (GST) families, namely cytosolic, mitochondrial, and microsomal GST, the latter now designated MAPEG. Besides detoxifying electrophilic xenobiotics, such as chemical carcinogens, environmental pollutants, and antitumor agents, these transferases inactivate endogenous α,β-unsaturated aldehydes, quinones, epoxides, and hydroperoxides formed as secondary metabolites during oxidative stress. These enzymes are also intimately involved in the biosynthesis of leukotrienes, prostaglandins, testosterone, and progesterone, as well as the

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Sunkara, Hari Priya, Krishna Rajesh Kilaru, Adusumilli Pramod Kumar, Hari Babu Ramineni, and Puvvada Rahul Krishna. "A case report on hydroquinone induced exogenous ochronosis." International Journal of Advances in Medicine 7, no. 2 (2020): 337. http://dx.doi.org/10.18203/2349-3933.ijam20200091.

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Exogenous ochronosis is an infrequent skin disorder characterised by bluish-black or grayish brown pigmentation on dermis. It is the most common condition caused due to long term application of Hydroquinone skin preparations for melasma, skin brightening, cholasma, acne induced pigmentation etc. This report refers to a case of 39-year-old female patient who presented to the hospital with chief complaints of progressive formation of dark lesions over face, neck since one and half-year. She had history of usage of hydroquinone (4%) cream for skin brightening for a period of three months. Based o

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Dissertations / Theses on the topic "Prostaglandins E Hydroquinone Glutathione"

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Towndrow, Kelly Marie. "Toxicant-induced prostaglandin E₂ synthesis and prostanoid-mediated cytoprotection /." Digital version accessible at:, 2000. http://wwwlib.umi.com/cr/utexas/main.

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Sawalha, Ansam Feras. "Species susceptibility to nephrotoxicity by hydroquinone and hydroquinone-glutathione conjugates : role of oxidation, specific cytochrome P450 isoforms, and tissue arylation /." Digital version accessible at:, 1998. http://wwwlib.umi.com/cr/utexas/main.

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Kuhlman, Christopher Lee. "Protein Adduct Formation by Reactive Electrophiles: Identifying Mechanistic Links with Benzene-Induced Hematotoxicity." Diss., The University of Arizona, 2013. http://hdl.handle.net/10150/312514.

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The modification of proteins by xenobiotic and endogenous electrophilic species produced in cells undergoing oxidative stress contributes to cellular toxicity and disease processes. Many xenobiotics are themselves reactive electrophiles; however non-reactive compounds may become reactive towards proteins and DNA following metabolism. Identifying actual sites of adduction on target proteins is critical for determining the structural and functional consequences associated with the modification. 1,4-benzoquinone (BQ) is a reactive quinone and environmental toxicant, formed from oxidative metabo

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Medina, Ramos Jonnathan. "ACID-BASE CATALYSIS IN PROTON-COUPLED ELECTRON TRANSFER REACTIONS (PCET): THE EFFECTS OF BRÖNSTED BASES ON THE OXIDATION OF GLUTATHIONE AND HYDROQUINONE." VCU Scholars Compass, 2012. http://scholarscompass.vcu.edu/etd/2918.

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This thesis presents the results and discussion of the investigation of the effects of Brönsted bases on the kinetics and thermodynamics of two proton-coupled electron transfer processes: the mediated oxidation of glutathione and the electrochemical oxidation of hydroquinone. Proton-coupled electron transfer (PCET) is the name given to reactions that involve the transfer of electron(s) accompanied by the exchange of proton(s). PCETs are found in many chemical and biological processes, some of current technological relevance such as the oxygen reduction reaction in fuel cells, which involves th

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Brechbuhl, Heather Michelle. "ATP-cassette binding transporters : modulators of glutathione levels in normal cellular physiology and as a means for therapeutic applications /." Connect to abstract via ProQuest. Full text is not available online, 2008.

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Jia, Zhe Lau Serrine S. Bratton Shawn Brian. "Mechanisms of 11-deoxy-16, 16-dimethyl prostaglandin E₂ mediated cytoprotection." 2004. http://repositories.lib.utexas.edu/bitstream/handle/2152/1335/jiad79486.pdf.

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Jia, Zhe 1976. "Mechanisms of 11-deoxy-16, 16-dimethyl prostaglandin E₂ mediated cytoprotection." Thesis, 2004. http://hdl.handle.net/2152/1335.

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Book chapters on the topic "Prostaglandins E Hydroquinone Glutathione"

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Hammarström, Sven, Lars Örning, Kerstin Bernström, et al. "Glutathione Transferases Catalyzing Leukotriene C Synthesis and Metabolism of Leukotrienes C4 and E4 in Vivo and in Vitro." In Prostaglandins, Leukotrienes, and Lipoxins. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4946-4_7.

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Hill, Barbara A., Herng-Hsiang Lo, Terrence J. Monks, and Serrine S. Lau. "The Role of γ-Glutamyl Transpeptidase in Hydroquinone-Glutathione Conjugate Mediated Nephrotoxicity." In Advances in Experimental Medicine and Biology. Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4684-5877-0_99.

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Atroshi, Faik, Satu Sankari, Aldo Rizzo, Tuomas Westermarck, and Jouko Parantainen. "Prostaglandins, Glutathione Metabolism, and Lipid Peroxidation in Relation to Inflammation in Bovine Mastitis." In Advances in Experimental Medicine and Biology. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5730-8_33.

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Conference papers on the topic "Prostaglandins E Hydroquinone Glutathione"

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Lecomte, M., and J. M. Boeynaems. "COVALENT BINDING OF CYCLOOXYGENASE AND LIPOXYGENASE PRODUCTS TO HUMAN PLATELET PROTEINS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643397.

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Several studies in acellular systems have shown a covalent binding of eicosanoids to proteins (1). We have therefore investigated whether eicosanoids bind covalently to proteins in intact platelets. After incubation of washed human platelets with 14C-arachidonic acid, ethanol precipitation followed by extractions, a small fraction of the radioactivity (0.3%) was tightly bound to the protein pellet. Four criteria suggest the covalent nature of this binding. The radioactivity remained bound after exhaustive extractions with solvents of various polarities, and was not removed by dialysis against

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