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Relevant bibliographies by topics / Proteolyses

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Academic literature on the topic 'Proteolyses'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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Journal articles on the topic "Proteolyses"

1

Rovery, Mireille. "Limited proteolyses in pancreatic chymotrypsinogens and trypsinogens." Biochimie 70, no. 9 (1988): 1131–35. http://dx.doi.org/10.1016/0300-9084(88)90177-0.

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Stalboerger, Paul, Carmelo Panetta, Robert Simari, and Noel Caplice. "Plasmin Proteolysis of Endothelial Cell and Vessel Wall Associated Tissue Factor Pathway Inhibitor." Thrombosis and Haemostasis 86, no. 09 (2001): 923–28. http://dx.doi.org/10.1055/s-0037-1616151.

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SummaryPlasmin is an important protease that mediates clot fibrinolysis and vessel wall extracellular matrix proteolysis. Recently, in vitro studies have suggested that plasmin can cleave and inactivate recombinant TFPI, a major inhibitor of TF-mediated coagulation. We hypothesized that such an interaction may occur in vascular cells expressing TFPI, or in the vessel wall, with implications for thrombolysis. In a series of experiments, we examined the effects of plasmin on cell surface and extracellular matrix (ECM) associated TFPI in endothelial cells (EC) in culture and on EC and smooth musc

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Brandt, K., K. Lundell, and K. Brismar. "P69 Neutrophil-derived azurocidin proteolyses IGF binding protein-1, -2 and -4." Growth Hormone & IGF Research 20 (January 2010): S63. http://dx.doi.org/10.1016/s1096-6374(10)70169-6.

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Yamasaki, S., T. Binz, T. Hayashi, et al. "Botulinum Neurotoxin Type G Proteolyses the Ala81-Ala82 Bond of Rat Synaptobrevin 2." Biochemical and Biophysical Research Communications 200, no. 2 (1994): 829–35. http://dx.doi.org/10.1006/bbrc.1994.1526.

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Marsman, Gerben, Helen von Richthofen, Ingrid Bulder, et al. "Factor VII-Activating Protease Is a Regulator of Histone Cytotoxicity." Blood 126, no. 23 (2015): 1005. http://dx.doi.org/10.1182/blood.v126.23.1005.1005.

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Abstract Background: During sepsis, ischemia-reperfusion injury and sterile inflammation, levels of circulating histones and nucleosomes have been found to correlate with disease severity. Although the pathophysiological effects of circulating nucleosomes are not fully understood, free histones have been described to mediate cytotoxicity and contribute to disease severity. Several plasma proteins have been identified that modulate these cytotoxic effects by neutralizing or degrading histones. We have previously described that the plasma serine protease Factor VII-activating protease (FSAP) is

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Bendayan, P., H. Boccalon, D. Dupouy, and B. Boneu. "Dermatan Sulfate Is a More Potent Inhibitor of Clot-bound Thrombin than Unfractionated and Low Molecular Weight Heparins." Thrombosis and Haemostasis 71, no. 05 (1994): 576–80. http://dx.doi.org/10.1055/s-0038-1642485.

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SummaryClot-bound thrombin proteolyses fibrinogen and amplifies the coagulation cascade at its close vicinity, thereby ensuring the growth of fibrin-rich thrombus. The present study compares the ability of various glycosaminoglycans (GAGs) to inhibit these 2 properties. Unfractionated heparin (UH), 3 low molecular weight heparins (LMWHs) with increasing antifactor Xa/antifactor Ha ratio, the synthetic pentasaccharide (PS), devoid of antifactor Ha activity, and dermatan sulfate (DS), a catalyst of thrombin inhibition by heparin cofactor II, were selected on the basis of their different properti

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Kmiec, E. B., and A. Worcel. "The positive transcription factor of the 5S RNA gene proteolyses during direct exchange between 5S DNA sites." Journal of Cell Biology 103, no. 3 (1986): 673–81. http://dx.doi.org/10.1083/jcb.103.3.673.

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We have examined the association, dissociation, and exchange of the 5S specific transcription factor (TFIIIA) with somatic- and oocyte-type 5S DNA. The factor associates faster with somatic than with oocyte 5S DNA, and the rate of complex formation is accelerated by vector DNA. Once formed, the TFIIIA-5S DNA complex is stable for greater than 4 h in the absence of free 5S DNA, and its dissociation is identical for somatic and for oocyte 5S DNA. In the presence of free 5S DNA, the factor transfers promptly from the complex to the free 5S DNA site. Unexpectedly, the direct exchange of factor bet

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Whelihan, Matthew F., and Kenneth G. Mann. "Plasma Procofactor Activation by Factor Xla." Blood 112, no. 11 (2008): 1024. http://dx.doi.org/10.1182/blood.v112.11.1024.1024.

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Abstract The procofactors FV and FVIII are activated by thrombin, FXa and plasmin. During contact pathway-initiated thrombin generation, FXIa activates FIX thus feeding into the coagulation cascade; however, the procofactors FVIII and FV must be activated to achieve a robust level of thrombin generation. We tested the hypothesis that FXIa can activate FV and FVIII. FV (1uM) was subjected to FXIa (100nM) proteolysis. During the reaction the relative activity and integrity were measured at selected time points using a one stage PT clotting assay and SDS-PAGE. Over the 60 minute time course, FV s

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Trueblood, Cynthia Evans, Victor L. Boyartchuk, Elizabeth A. Picologlou, David Rozema, C. Dale Poulter, and Jasper Rine. "The CaaX Proteases, Afc1p and Rce1p, Have Overlapping but Distinct Substrate Specificities." Molecular and Cellular Biology 20, no. 12 (2000): 4381–92. http://dx.doi.org/10.1128/mcb.20.12.4381-4392.2000.

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ABSTRACT Many proteins that contain a carboxyl-terminal CaaX sequence motif, including Ras and yeast a-factor, undergo a series of sequential posttranslational processing steps. Following the initial prenylation of the cysteine, the three C-terminal amino acids are proteolytically removed, and the newly formed prenylcysteine is carboxymethylated. The specific amino acids that comprise the CaaX sequence influence whether the protein can be prenylated and proteolyzed. In this study, we evaluated processing of a-factor variants with all possible single amino acid substitutions at either the a1, t

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Nishiya, Katsumi, Keiji Nogami, Tomoko Matsumoto, Kenichi Ogiwara, Masahiro Takeyama, and Midori Shima. "Mechanisms of Human Neutrophil Elastase-Catalyzed Inactivation of the Factor VIII(a)." Blood 114, no. 22 (2009): 2125. http://dx.doi.org/10.1182/blood.v114.22.2125.2125.

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Abstract Abstract 2125 Poster Board II-103 Inflammation and coagulation are linked in a variety ways. Since both neutrophil elastase and cathepsin G bind to activated platelets, they can be localized on the platelet membrane providing negatively-charged phospholipid, that is essential for assembly of tenase complex. Although it has been reported that cathepsin G provides some procoagulant effect by activating factor (F)VIII, effect of elastase on FVIII is poorly understood. We now examine the effect of human neutrophil elastase (HNE) on FVIII(a). FVIII activity (used 100 nM) was rapidly decrea

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Dissertations / Theses on the topic "Proteolyses"

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Conseil, Valérie. "Etude des proteolyses en culture et in vitro de la proteine p126 de plasmodium falciparum." Lille 2, 1996. http://www.theses.fr/1996LIL2T007.

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Попадинець, Василь Миронович, Василий Миронович Попадинець, Vasyl Myronovych Popadynets та ін. "Нейтрофільна еластаза як показник стану протеолітичної системи у хворих з трофічними виразками різного генезу". Thesis, Сумський державний університет, 2017. http://essuir.sumdu.edu.ua/handle/123456789/64749.

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Актуальність. Незважаючи на бурхливий розвиток сучасних наукових технологій і накопичений багаторічний досвід у вирішенні багатьох міждисциплінарних проблем, питання профілактики та лікування трофічних виразок залишаються актуальними. Серед основних причин виникнення виразок на нижніх кінцівках провідне місце займають розлади їх кровопостачання. Процеси протеолізу мають безпосереднє відношення до реакцій запалення і деструкції тканина. Недостатньо вивченим залишається стан протеолітичної системи у хворих із захворюваннями судин, а також трофічними виразками різного генезу. Останнім часом з’явл

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El-Daher, Marie-Thérèse. "Huntingtin proteolysis and toxicity." Thesis, Paris 11, 2013. http://www.theses.fr/2013PA11T029/document.

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La maladie de Huntington (MH) est une maladie neurodégénérative héréditaire autosomique dominante. Elle est due à l’expansion anormale de polyglutamine dans la partie N-terminal de la protéine huntingtine (HTT). Une des étapes clés de la pathologie est le clivage de la HTT pleine longueur en fragments N-terminaux plus petits, contenant l’expansion de polyglutamine, et qui sont toxiques pour les neurones. En effet, les clivages de la HTT mutée génère des fragments N-terminaux (N-ter) de tailles comprises entre les acides aminés 1-105 et 1-586 observés dans des extraits de cerveaux de patients M

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Clay, L. "CDC20 function, regulation and proteolysis." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.597750.

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The destruction of mitotic cyclins and other key regulators uses ubiquitin mediated proteolysis controlled via the activation of the ubiquitin ligase the Anaphase Promoting Complex/Cyclosome (APC/C), and its adaptor proteins Cdc20 and Cdh1. The spindle assembly checkpoint coordinates the APC/C with microtubule attachment and sets the timing from NEBD to anaphase. Cdc20 is inactivated by the spindle assembly checkpoint to prevent premature anaphase onset. Once the spindle assembly checkpoint is satisfied, Cdc20 can be released and activate the APC/C. However, cyclin A is degraded independently

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Slee, Adrian. "Regulation of skeletal muscle proteolysis." Thesis, University of Nottingham, 2005. http://eprints.nottingham.ac.uk/13105/.

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Proteolysis is a component of protein turnover, controlled by multiple proteolytic systems. Alterations in system components within skeletal muscle has been associated with hypertrophy, remodelling, atrophy, apoptosis and metabolic dysregulation. Key components may have novel regulatory roles, e. g. calpain-3 and cathepsin-L. Experiments described within this thesis investigated the hypothesis that the gene expression of specific proteolytic system components within skeletal muscle may be co-ordinately regulated and altered during nutritional and pharmacological states known to modify protein

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Haas, Regina. "Proteolyse von Apolipoprotein A-I." [S.l. : s.n.], 1999. http://deposit.ddb.de/cgi-bin/dokserv?idn=956370217.

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Weimann, Dirk. "Proteolyse-induzierte Spaltprodukte des Plasminogens." Diss., lmu, 2002. http://nbn-resolving.de/urn:nbn:de:bvb:19-3608.

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Slimani, Lamia. "Mécanismes impliqués dans l'atrophie et la récupération musculaire après immobilisation chez le rat. : Rôle des altérations de la matrice extracellulaire." Thesis, Clermont-Ferrand 1, 2012. http://www.theses.fr/2012CLF1MM21/document.

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Le muscle squelettique est le réservoir principal d’acides aminés libres de l’organisme. Ainsi, l’atrophie musculaire induite par l’immobilisation peut entraîner un affaiblissement et un allongement des périodes de récupération générant des coûts de santé publique élevés. Une aggravation de l’atrophie caractérise de façon surprenante le muscle tibialis anterior (TA) après le déplâtrage, retardant la récupération. Mon objectif a été de comprendre les mécanismes à l’origine de l’aggravation de l’atrophie du TA pendant les phases précoces de récupération en étudiant i) la structure et le phénotyp

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Ullrich, Oliver. "Mechanismen protektiver und destruktiver Funktionen der Poly(ADP-Ribose)-Polymerase-1 (PARP-1) bei Zell- und Gewebeschädigungen." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2002. http://dx.doi.org/10.18452/13826.

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Während der letzten Dekade neurobiologischer Forschung wurde deutlich, dass inflammatorische Vorgänge in einem Netwerk nicht-neuronaler und neuronale Zellen wesentlich zur Entstehung und zur Progredienz einiger wichtiger neurodegenerativer Erkrankungen beitragen. Therapeutische Ansätze sollten daher auch auf die Protektion initial überlebender Neurone vor dieser sekundären inflammatorischen Schädigung zielen. Ein wesentlicher Bestandteil dieser sekundären Schädigung besteht aus der Migration von Makrophagen und Mikrogliazellen in die Regionen neuronaler Schädigung, wo sie grosse Mengen an toxi

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Andréasson, Claes. "Ligand-activated proteolysis in nutrient signaling /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-075-3/.

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Books on the topic "Proteolyses"

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Chondrogianni, Niki, Elah Pick, and Anna Gioran. Proteostasis and Proteolysis. CRC Press, 2021. http://dx.doi.org/10.1201/9781003048138.

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Antonov, Vladimir K. Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6.

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Keil, Borivoj. Specificity of Proteolysis. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-48380-6.

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Abatangelo, G., L. Donati, and W. Vanscheidt, eds. Proteolysis in Wound Repair. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-61130-8.

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Dougan, David A., ed. Regulated Proteolysis in Microorganisms. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-5940-4.

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1946-, Mellgren Ronald L., and Murachi Takashi 1926-, eds. Intracellular calcium-dependent proteolysis. CRC Press, 1990.

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K, Hopsu-Havu Väinö, Järvinen M, Kirschke Heidrun, and International Conference on Proteolysis and Protein Turnover (11th : 1996 : Turku, Finland), eds. Proteolysis in cell functions. IOS Press, 1997.

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C, Taylor Joseph, and Mittman Charles, eds. Pulmonary emphysema and proteolysis, 1986. Academic Press, 1987.

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Smith, Kate Louise. Tumour associated proteolysis and protein metabolism. Aston University. Department of Pharmaceutical Sciences, 1992.

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Falokun, Christopher D. Aspects of molecular recognition in limited proteolysis. UMIST, 1996.

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Book chapters on the topic "Proteolyses"

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Bond, Judith S., Timothy R. Keiffer, and Qi Sun. "Pericellular Proteolysis." In Extracellular Matrix Degradation. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16861-1_4.

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Antonov, Vladimir K. "Introduction." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_1.

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Antonov, Vladimir K. "Substrates." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_2.

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Antonov, Vladimir K. "Enzymes." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_3.

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Antonov, Vladimir K. "Nonenzymatic Hydrolysis. Models." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_4.

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Antonov, Vladimir K. "Enzyme Hydrolysis. Phenomenology." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_5.

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Antonov, Vladimir K. "Regulation and Effect of External Factors." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_6.

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Antonov, Vladimir K. "Enzyme-Substrate Complexes." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_7.

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Antonov, Vladimir K. "Chemical Transformation of the Substrate." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_8.

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Antonov, Vladimir K. "Specificity and Efficiency. Concepts and Hypotheses." In Chemistry of Proteolysis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-00979-6_9.

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Conference papers on the topic "Proteolyses"

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Berkowitz, S. D., H. Nozaki, K. Titani, T. Murachi, and T. S. Zimmerman. "CALPAIN AND ELASTASE ARE NOT RESPONSIBLE FOR THE VON WILLEBRAND FACTOR FRAGMENTS IN NORMAL PLASMA AND IIA VON WILLEBRAND DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644103.

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Recent evidence suggests that proteolysis plays an important role in some forms of inherited and acquired von Willebrand disease (vWD). Using monoclonal epitope mapping, we have examined the proteolysis of the von Willebrand factor (vWF) subunit with platelet calcium activated neutral protease (CANP) and human leukocyte elastase and found that they are not responsible for the proteolytic cleavage sejen in normal individuals and IIA vWD. Previously we have shown that in vivo proteolysis of vWF is a normal event with a small but consistent proportion of plasma vWF being composed of 189, 176, and

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Vaughan, D. E., and J. Loscalzo. "PLATELET DISAGGREGATION IN PLASMA--A NOVEL EFFECT OF TISSUE PLASMINOGEN ACTIVATOR." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644385.

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Platelet aggregates are thought to play a significant role in many clinically important ischemic vascular events. Recently it has been shown that the platelet surface binds plasminogen and, in so doing, enhances its conversion to plasmin by tissue plasminogen activator (tPA). Since fibrinogen, an alternative substrate for plasmin, serves as the cohesive link among platelets induced to aggregate by a variety of agonists, we hypothesized that the local production of plasmin at the platelet surface may be important in promoting the disaggregation of aggregated platelets. When added to a suspensio

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López-Fernández, M. F., C. López-Berges, R. Martín, A. Pardo, F. J. Romos, and J. Batlle. "ABNORMAL STRUCTURE OF VON WILLEBRAND FACTOR IN MYELOPROLIFERATIVE SYNDROME IS ASSOCIATED WITH EITHER THROMBOTIC OR BLEEDING DIATHESIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644089.

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The multimeric and subunit patterns of plasma von Willebrand factor (vWF) were analyzed in eight patients with myeloproliferative syndrome (MS) in order to investigate the possible existence of heterogeneity in the "in vivo" proteolytic cleavage of the protein, previously observed in this entity. Six patients lacked large vWF multimers, five of them having normal bleeding times (BT) and clinically documented episodes of thrombotic origin, whereas one patient had long BT and bleeding symptoms. Seven patients showed an increase 176 kDa subunit fragment while the 189 kDa polypeptide was increased

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Ghosh, A., R. D. Coakley, D. B. Hill, M. Kesimer, N. E. Alexis, and R. Tarran. "Vaping-Induced Proteolysis Causes Airway Dehydration." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a4060.

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Gao, Jinfeng, Chuhan Meng, and Vivian FT Yuan. "Clinical application of proteolysis targeting chimeras." In International Conference on Modern Medicine and Global Health (ICMMGH 2023), edited by Sheiladevi Sukumaran. SPIE, 2023. http://dx.doi.org/10.1117/12.3000206.

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Bischel, Kristen Moriah, Philip Emmerich, Tonela Qyli, et al. "Abstract 403: Versican proteolysis in endometrial cancer." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-403.

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Antipina, M. I., E. G. Chupakhin, V. V. Kakotkin, M. A. Agapov, and E. V. Semina. "MOLECULAR THERAPY OF NEURODEGENERATIVE PATHOLOGIES USING TARGETED UBIQUITIN-DEPENDENT PROTEOLYSIS OF PROTEIN TARGETS WITH PROTACS (PROTEOLYSIS TARGETING CHIMERAS)." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-287.

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Aggregation of microtubule-associated tau protein (MAPT) due to its hyperphosphorylation is a hallmark of Alzheimer’s disease (AD). Therefore, targeting MAPT is one of the promising approaches for AD therapy. PROTAC (Proteolysis Targeting Chimeras) system is one of the possible therapy options for neurodegenerative diseases, which is used to selectively degrade a target protein in a cell.

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duVERLE, DAVID, ICHIGAKU TAKIGAWA, YASUKO ONO, HIROYUKI SORIMACHI, and HIROSHI MAMITSUKA. "CaMPDB: A RESOURCE FOR CALPAIN AND MODULATORY PROTEOLYSIS." In Proceedings of the 9th Annual International Workshop on Bioinformatics and Systems Biology (IBSB 2009). IMPERIAL COLLEGE PRESS, 2010. http://dx.doi.org/10.1142/9781848165786_0017.

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Roslan, Nur Farhana, Bee Lyn Chew, Hoe-Han Goh, and Nurulhikma Md Isa. "Sequence analysis of PROTEOLYSIS 6 from Solanum lycopersicum." In THE 2017 UKM FST POSTGRADUATE COLLOQUIUM: Proceedings of the University Kebangsaan Malaysia, Faculty of Science and Technology 2017 Postgraduate Colloquium. Author(s), 2018. http://dx.doi.org/10.1063/1.5027977.

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Hou, Li-Xia, Ying-Ying Wang, Yu-Lan Liu, and Xiao-Kun Wang. "Effect of Sonication on Proteolysis of Peanut Protein Isolate." In 2015 International Conference on Medicine and Biopharmaceutical. WORLD SCIENTIFIC, 2016. http://dx.doi.org/10.1142/9789814719810_0147.

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Reports on the topic "Proteolyses"

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Harper, Jeffrey. Identification of Genes Regulated by Proteolysis. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada408062.

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Carlson, Kelsey, Kenneth J. Prusa, Chris A. Fedler, et al. Proteolysis Influences Tenderness of Aged Pork Loins. Iowa State University, 2017. http://dx.doi.org/10.31274/ans_air-180814-328.

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Mudryj, Maria. Calpain-Dependent Proteolysis of the Androgen Receptor. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada517269.

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Strohmaier, Heimo M., and Steven Reed. The Role of Deregulated Cyclin E Proteolysis in Breast Cancer Development. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada409788.

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Drogen, Frank van, Steven Reed, and Heimo M. Strohmaier. The Role of Deregulated Cyclin E Proteolysis in Breast Cancer Development. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada418341.

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Spruck, Charles H. Identification of Substances for Ubiquitin-Dependent Proteolysis During Breast Tumor Progression. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada510763.

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Tzfira, Tzvi, Michael Elbaum, and Sharon Wolf. DNA transfer by Agrobacterium: a cooperative interaction of ssDNA, virulence proteins, and plant host factors. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7695881.bard.

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Abstract:

Agrobacteriumtumefaciensmediates genetic transformation of plants. The possibility of exchanging the natural genes for other DNA has led to Agrobacterium’s emergence as the primary vector for genetic modification of plants. The similarity among eukaryotic mechanisms of nuclear import also suggests use of its active elements as media for non-viral genetic therapy in animals. These considerations motivate the present study of the process that carries DNA of bacterial origin into the host nucleus. The infective pathway of Agrobacterium involves excision of a single-stranded DNA molecule (T-strand

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Srikanth, Appikonda. The Role of Ubiquitin-Mediated Proteolysis of Cyclin D in Breast Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada455151.

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Block, Karen L. The Role of Ubiquitin-Mediated Proteolysis of Cyclin D in Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada416662.

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Khokha, Rama. Molecular Tracking of Proteolysis During Breast Cancer Cell Extravasation: Blockage of Therapeutic Inhibitors. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada411429.

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