Relevant bibliographies by topics / Remnant cholesterol

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Remnant cholesterol'

Author: Grafiati
Published: 23 August 2023
Last updated: 31 July 2025

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Journal articles on the topic "Remnant cholesterol"

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Sultan, F., D. Lagrange, X. Le Liepvre, and S. Griglio. "Chylomicron-remnant uptake by freshly isolated hepatocytes. Effect of heparin and of hepatic triacylglycerol lipase." Biochemical Journal 258, no. 2 (1989): 587–94. http://dx.doi.org/10.1042/bj2580587.

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Chylomicron remnants labelled biologically with [3H]cholesterol were efficiently taken up by freshly isolated hepatocytes during a 3 h incubation in Krebs bicarbonate medium. Their [3H]cholesteryl ester was hydrolysed (74% net hydrolysis), and 0.1 mM-chloroquine could partially inhibit this hydrolysis, provided that hepatocytes were first preincubated for 2 h 30 min at 37 degrees C. This hydrolysis was also measured in preincubated cells with remnants double-labelled (3H and 14C) on their free cholesterol moiety; [3H]cholesterol arising from [3H]cholesteryl ester hydrolysis was recovered in th
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Redgrave, T. G., C. L. Elsegood, J. C. L. Mamo, and M. J. Callow. "Effects of hypothyroidism on the metabolism of lipid emulsion models of triacylglycerol-rich lipoproteins in rats." Biochemical Journal 273, no. 2 (1991): 375–81. http://dx.doi.org/10.1042/bj2730375.

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Methimazole-treated hypothyroid rats were injected intravenously with triacylglycerol/cholesteryl oleate/cholesterol/phospholipid emulsions designed to model the composition of chylomicrons. Compared with controls, hypothyroidism decreased the clearance rates of emulsion cholesteryl oleate. Clearance of emulsion triolein was affected much less and could be accounted for by residual triolein in remnants, suggesting that triacylglycerol lipolysis by lipoprotein lipase was unaffected by hypothyroidism but that clearance of remnants from plasma was decreased. Assays in vitro showed increased activ
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WATTS, G. F., D. C. F. CHAN, P. H. R. BARRETT, I. J. MARTINS, and T. G. REDGRAVE. "Preliminary experience with a new stable isotope breath test for chylomicron remnant metabolism: a study in central obesity." Clinical Science 101, no. 6 (2001): 683–90. http://dx.doi.org/10.1042/cs1010683.

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We aimed to investigate the metabolism of chylomicron remnants in the postabsorptive state employing a new stable isotope breath test in centrally obese men without overt hyperlipidaemia. Groups of 12 centrally obese and 12 non-obese men of similar age and with similar plasma cholesterol and triacylglycerol (triglyceride) levels were studied. The catabolism of chylomicron remnants was measured using an intravenous injection of a remnant-like emulsion containing cholesteryl [13C]oleate. Isotopic enrichment of 13CO2 in breath was determined using isotope-ratio mass spectrometry, and a multi-comp
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Chan, Dick C., Gerald F. Watts, P. Hugh Barrett, John CL Mamo, and Trevor G. Redgrave. "Markers of Triglyceride-rich Lipoprotein Remnant Metabolism in Visceral Obesity." Clinical Chemistry 48, no. 2 (2002): 278–83. http://dx.doi.org/10.1093/clinchem/48.2.278.

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Abstract Background: Triglyceride-rich lipoprotein remnants are atherogenic, and this may be particularly important in visceral obesity. We investigated remnant metabolism in obese men by measuring remnant-like particle-cholesterol (RLP-C), apolipoprotein (apo) B-48, apoC-III, and the clearance of a labeled remnant-like emulsion. Methods: Fasting RLP-C, apoB-48, and apoC-III concentrations were measured in 48 viscerally obese men and 10 lean controls. RLP-C was determined by immunoseparation assay, apoB-48 by sodium dodecyl sulfate-polyacrylamide gel electrophoresis and enhanced chemiluminesce
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Bowler, A., T. G. Redgrave, and J. C. L. Mamo. "Chylomicron-remnant clearance in homozygote and heterozygote Watanabe-heritable-hyperlipidaemic rabbits is defective. Lack of evidence for an independent chylomicron-remnant receptor." Biochemical Journal 276, no. 2 (1991): 381–86. http://dx.doi.org/10.1042/bj2760381.

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Lymph chylomicrons radiolabelled in triacylglycerol and cholesteryl ester were injected into control and Watanabe heritable-hyperlipidaemic (WHHL) rabbits. Clearance of chylomicrons was slower in heterozygote and homozygote WHHL rabbits. Slower remnant clearance in WHHL rabbits was confirmed by monitoring the clearance from plasma of preformed chylomicron remnants. Use of chylomicron-like lipid emulsions injected into control and WHHL rabbits also confirmed the defect in remnant clearance in heterozygote WHHL and homozygote WHHL groups. Clearance from plasma of emulsion triolein was delayed in
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McPherson, Ruth. "Remnant Cholesterol." Journal of the American College of Cardiology 61, no. 4 (2013): 437–39. http://dx.doi.org/10.1016/j.jacc.2012.11.009.

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Langsted, Anne, Anne Marie Reimer Jensen, Anette Varbo, and Børge G. Nordestgaard. "Low High-Density Lipoprotein Cholesterol to Monitor Long-Term Average Increased Triglycerides." Journal of Clinical Endocrinology & Metabolism 105, no. 4 (2019): e1657-e1666. http://dx.doi.org/10.1210/clinem/dgz265.

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Abstract Context Increased triglyceride-rich remnants represent a causal risk factor for ischemic cardiovascular disease. Objective We tested the hypothesis that low high-density lipoprotein (HDL) cholesterol can be used to monitor long-term high triglycerides/remnant cholesterol, just as high hemoglobin A1c (HbA1c) can be used to monitor long-term high glucose levels. Design, Setting, Participants, and Interventions We studied cross-sectionally 108 731 individuals, dynamically 1313 individuals with lipid measurement at 10 repeated visits, short-term 305 individuals during a fat load, and long
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Lambert, M. S., K. M. Botham, and P. A. Mayes. "Variations in composition of dietary fats affect hepatic uptake and metabolism of chylomicron remnants." Biochemical Journal 310, no. 3 (1995): 845–52. http://dx.doi.org/10.1042/bj3100845.

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The hepatic metabolism of [1-14C]oleate- and [1,2-3H]cholesterol-dual-labelled chylomicron remnants derived from olive, corn, palm and fish oil and butter fat was compared by adding each lipoprotein separately to the perfusate of isolated livers from rats fed on a normal diet. Labelled remnants from butter fat and fish oil were removed more rapidly from the perfusate than remnants derived from olive, corn and palm oil. The oxidation of labelled remnant fatty acid from olive oil, fish oil or butter fat was four to seven times greater than that from corn and palm oil. Labelled fatty acid in fish
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Miyauchi, Kazuhito, Norihiko Kayahara, Masato Ishigami, et al. "Development of a Homogeneous Assay to Measure Remnant Lipoprotein Cholesterol." Clinical Chemistry 53, no. 12 (2007): 2128–35. http://dx.doi.org/10.1373/clinchem.2007.092296.

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Abstract Background: Quantification of triglyceride-rich lipoprotein (TRL) remnants is useful for risk assessment of coronary artery disease and the diagnosis of type III hyperlipoproteinemia. Although an immunoseparation procedure for remnant-like particle cholesterol has been evaluated extensively in recent years, available methods for measuring TRL remnants have not achieved wide use in routine laboratory practice, suggesting a need for a homogeneous assay that can measure TRL remnant cholesterol in serum or plasma without pretreatment. Methods: We screened for suitable surfactants that exh
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CHAN, Dick C., Gerald F. WATTS, P. Hugh R. BARRETT, Frans H. O'NEILL, Trevor G. REDGRAVE, and Gilbert R. THOMPSON. "Relationships between cholesterol homoeostasis and triacylglycerol-rich lipoprotein remnant metabolism in the metabolic syndrome." Clinical Science 104, no. 4 (2003): 383–88. http://dx.doi.org/10.1042/cs1040383.

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The dysmetabolic syndrome of insulin resistance and visceral obesity is characterized by elevated plasma concentration of triacylglycerol-rich lipoprotein (TRL) remnants that may be related to increased cardiovascular risk. Perturbed hepato-intestinal cholesterol metabolism may play a contributory role in this abnormality. We therefore investigated the association between plasma markers of cholesterol absorption and synthesis with TRL remnant metabolism in 35 men with the metabolic syndrome (MS). Plasma campesterol:cholesterol and lathosterol:cholesterol ratios were measured as estimates of ch
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Dissertations / Theses on the topic "Remnant cholesterol"

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Wu, Zhiyuan. "Roles of remnant cholesterol in the risk factor, occurrence and in-hospital outcomes of stroke." Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2023. https://ro.ecu.edu.au/theses/2696.

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Background Stroke is the leading cause of death and disability with high incidence and recurrence rates and has caused severe social burden worldwide. Data from epidemiological studies demonstrate that there were more than 101 million stroke cases globally in 2019, with ischemic stroke accounting for 62.4%. Of note, the global lifetime risk of stroke is 24.9%, while the lifetime risk of stroke in China is up to 39.3%. Independent of low-density lipoprotein (LDL) cholesterol, the atherogenic effect of remnant cholesterol is gradually recognized in the residual risk of cardiovascular disease. Ho
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SULTAN, FABRICE. "Lipase hepatique chez le rat : role dans le metabolisme des remnants de chylomicrons et regulation par le cholesterol alimentaire." Paris 7, 1990. http://www.theses.fr/1990PA077097.

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Le role de la lipase hepatique (lh) dans le metabolisme des lipoproteines et plus particulierement dans celui des remnants de chylomicrons (rm) est encore mal compris. La premiere partie de cette etude a ete de rechercher si la lh etait impliquee dans le metabolisme des rm. Les rm marques au cholesterol (#3h) ou au cholesteryl-ether (#1#4c) puis co-incubes avec la lh, sont plus efficacement captes par des hepatocytes que les rm temoins (+50% a 100%). L'utilisation d'un anticorps anti-lh nous a permis de confirmer la specificite de la lh dans ce processus. Des rm traites par de la lh, puis inje
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Warnakula, Samantha. "The role of ezetimibe and simvastatin in modulating intestinal cholesterol transport, chylomicron profile and chylomicron-remnant uptake by the arterial wall in a rodent model of the metabolic syndrome." Master's thesis, 2010. http://hdl.handle.net/10048/1374.

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Intestinally derived chylomicron remnants (CM-r) may contribute to atherogenic dyslipidemia during the Metabolic Syndrome (Mets). However, the combined effects of ezetimibe (EZ) and simvastatin (SV) on post-prandial (PP) dyslipidemia during MetS remains unclear, nor is it known whether the combination has a synergistic anti-atherogenic effect on CM-r arterial retention. The first objective was to delineate the effects of EZ+SV therapy on intestinal cholesterol flux and CM PP metabolism in the JCR:LA-cp rat, a model of MetS. The second objective was to quantify the impact of EZ+SV therapy on ar
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Books on the topic "Remnant cholesterol"

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Kontush. Cholesterol, Lipoproteins, and Cardiovascular Heal Th: Separating the Good , the Bad , and the Remnant. Wiley & Sons, Limited, John, 2024.

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Kontush. Cholesterol, Lipoproteins, and Cardiovascular Heal Th: Separating the Good , the Bad , and the Remnant. Wiley & Sons, Limited, John, 2024.

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Wiklund, Olov, and Jan Borén. Pathogenesis of atherosclerosis: lipid metabolism. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0011.

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Lipids are carried in plasma as microparticles, lipoproteins, composed of a core of hydrophobic lipids and a surface of amphipathic lipids. In addition, the particles carry proteins (i.e. apolipoproteins). The proteins have key functions in the metabolism as receptor ligands, enzymes or activators. Lipoproteins are classified based on density into: chylomicrons, VLDL, IDL, LDL, and HDL. Retention of apoB-containing lipoproteins (LDL, IDL, and VLDL) in the arterial intima is the initiating event in the development of atherosclerosis. Retention is mediated by binding of apoB to structural proteo
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Book chapters on the topic "Remnant cholesterol"

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Nordestgaard, Børge G., and Anette Varbo. "Triglyceride-rich Lipoprotein Cholesterol (Remnant Cholesterol) as a Therapeutic Target for Cardiovascular Disease Risk." In Contemporary Cardiology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56514-5_8.

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Wadström, Benjamin Nilsson, Anders Berg Wulff, Kasper Mønsted Pedersen, and Børge Grønne Nordestgaard. "Triglyceride- and Cholesterol-Rich Remnant Lipoproteins in Risk of Cardiovascular Disease in Diabetes Mellitus." In Contemporary Diabetes. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-26681-2_8.

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Arnold, Kay S., Thomas L. Innerarity, Robert E. Pitas, and Robert W. Mahley. "Lipoprotein - receptor interactions." In Lipoprotein Analysis. Oxford University PressOxford, 1992. http://dx.doi.org/10.1093/oso/9780199631926.003.0006.

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Abstract The interaction of lipoproteins with their receptors is the first step of a homeostatic mechanism that controls plasma cholesterol levels, intracellular cholesterol metabolism, and lipoprotein catabolism. The classic studies of M. S. Brown and J. L. Goldstein (Univ. of Texas, Dallas) have demonstrated the significance of low density lipoprotein (LDL) receptors in supplying cells and tissues with cholesterol and in removing plasma lipoproteins via the liver (1). Other less well-defined lipoprotein receptors are the chylomicron remnant receptor (apolipoprotein E receptor), the high dens
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Cegla, Jaimini, and James Scott. "Lipid disorders." In Oxford Textbook of Medicine, edited by Timothy M. Cox. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0232.

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High blood cholesterol and high blood triglycerides are causal risk factors for atherosclerotic cardiovascular disease, which remains the leading cause of death in the developed world. Lipid and lipoprotein metabolism—cholesterol, triglycerides, and fat-soluble vitamins are transported with specific proteins in the blood as multimeric complexes called lipoproteins. Lipid and lipoprotein metabolism are effected by three principal physiological processes: (1) intestinal absorption of dietary lipid and transport in the blood of dietary lipid and lipids, principally derived from the liver (as trig
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Kritharides, Leonard, and Trevor G. Redgrave. "Lipoprotein influx and efflux in the atherosclerotic intima." In Atherosclerosis. Oxford University PressOxford, 2000. http://dx.doi.org/10.1093/oso/9780198506379.003.0013.

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Abstract Influx and efflux of lipids in the arterial wall are life-long phenomena. Progressive focal accumulations of lipid could even perhaps be regarded as physiological, since aortic fatty streaks are observed in infancy and are present in all individuals by I0 years of age Coronary lesions are detected by the age of 15_(2) These early lesions consist mostly of cholesteryl esters, and undoubtedly originate mainly by influx from plasma lipoproteins_< l Influx is probably continual for low-density lipoprotein (LDL), but may be episodic for post-prandial remnant particles. Net accumulat
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Joner, Michael, Maria Isabel Castellanos, Anna Bulin, and Kristin Steigerwald. "Pathology of stable coronary artery disease." In ESC CardioMed, edited by William Wijns. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0325.

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Coronary artery disease remains the major cause of morbidity and mortality on a global scale. Intimal thickening and fatty streaks represent early adaptive vascular changes, which are often regressive. Pathological intimal thickening represents the earliest progressive atherosclerotic lesion characterized by a focal accumulation of smooth muscle cells and acellular areas, often associated with lipid pools. Fibroatheroma is characterized by a necrotic core and can be split into early and late fibroatheroma, where macrophage apoptosis and defective efferocytosis play important roles in lesion pr
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Joner, Michael, Maria Isabel Castellanos, Anna Bulin, and Kristin Steigerwald. "Pathology of stable coronary artery disease." In ESC CardioMed, edited by William Wijns. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0325_update_001.

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Coronary artery disease remains the major cause of morbidity and mortality on a global scale. Intimal thickening and fatty streaks represent early adaptive vascular changes, which are often regressive. Pathological intimal thickening represents the earliest progressive atherosclerotic lesion characterized by a focal accumulation of smooth muscle cells and acellular areas, often associated with lipid pools. Fibroatheroma is characterized by a necrotic core and can be split into early and late fibroatheroma, where macrophage apoptosis and defective efferocytosis play important roles in lesion pr
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Erbel, Raimund. "Aortic sclerosis: therapy." In ESC CardioMed, edited by Raimund Erbel. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0613.

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Aortic sclerosis is a very common disease and is related to the atherosclerotic process which can start in young adulthood. The loss of compliance of the aortic ‘Windkessel’ is a major drawback of the disease, leading to enhanced blood pressure amplitude—elevated pulse pressure—due to a higher systolic and a lower diastolic blood pressure. The pressure pulse wave augmentation index and the pulse wave velocity increase. Ultrasound is able to visualize only limited aortic segments, whereas computed tomography and magnetic resonance imaging are the best tools to detect and quantify aortic atherom
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Conference papers on the topic "Remnant cholesterol"

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Li, Lifang, Vanessa Chou, Oscar Chou, et al. "The association between time-weighted remnant cholesterol and respiratory-related mortality: A population-based cohort study." In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.pa3289.

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Zhao, J., B. Cai, Y. Zhou, X. Tian, M. Li, and X. Zeng. "OP0214 ASSOCIATIONS OF REMNANT CHOLESTEROL WITH ALL-CAUSE AND CAUSE-SPECIFIC THROMBOTIC RISK IN INDIVIDUALS WITH ANTIPHOSPHOLIPID SYNDROME." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.897.

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Fan, Y. T., Y. W. Zou, T. Wu, et al. "AB0545 ASSOCIATION OF SERUM CONCENTRATIONS OF REMNANT CHOLESTEROL WITH INCIDENT CARDIOVASCULAR DISEASE IN PATIENTS WITH RHEUMATOID ARTHRITIS: A REAL-WORLD DATA FROM 2001 TO 2022." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.4611.

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