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Journal articles on the topic 'Renal tissue'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Nishida, Sho, Tamaki Ishima, Natsuka Kimura, et al. "Metabolomic Profiling of Mice with Tacrolimus-Induced Nephrotoxicity: Carnitine Deficiency in Renal Tissue." Biomedicines 12, no. 3 (2024): 521. http://dx.doi.org/10.3390/biomedicines12030521.

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Tacrolimus (TAC)-induced chronic nephrotoxicity (TAC nephrotoxicity) has a detrimental effect on long-term kidney graft survival. However, the pathogenesis of TAC nephrotoxicity remains largely unknown. We explored it by focusing on metabolic changes in renal tissues. In this study, mice were separated into TAC and control groups (n = 5/group). TAC was administered to the TAC group (1 mg/kg/d for 28 days) subcutaneously. The control group was similarly treated with normal saline. Renal tissue metabolomes were evaluated. Renal fibrosis was observed only in the TAC group. Metabolomic analysis sh

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Yan, Rui, Ye Zhang, Yuxing Yang, et al. "Peroxisome Proliferator-Activated Receptor Gene Knockout Promotes Podocyte Injury in Diabetic Mice." BioMed Research International 2022 (June 30, 2022): 1–8. http://dx.doi.org/10.1155/2022/9018379.

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Objective. To investigate the effects of peroxisome proliferator-activated receptor (PPARγ) expression on renal podocyte in diabetic mice by conditionally knockout mouse PPARγ gene. Methods. Wild-type C57BL mice and PPARγ gene knockout mice were used as research objects to establish the diabetic mouse model, which was divided into normal control group (NC group), normal glucose PPARγ gene knockout group (NK group), diabetic wild-type group (DM group), and diabetic PPARγ gene knockout group (DK group), with 8 mice in each group. After 16 weeks, the mice were sacrificed for renal tissue collecti

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Embarek, Mona. "The Expression pattern of NK cells in systemic lupus erythematosus patients with different disease activities." Egyptian journal of Immunology 31, no. 4 (2024): 157–68. http://dx.doi.org/10.55133/eji.310415.

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Data demonstrated the role of natural killer (NK) cells in systemic lupus erythematosus (SLE). We aimed to determine the immunophenotype and frequency of NK cells and their subsets, CXCR3, CD161 expression in blood and renal tissue of SLE patients with and without lupus nephritis and their relationship with disease activity. The study included 31 SLE patients and 11 controls. Study participants underwent full history and thorough clinical examination. SLE patients underwent routine laboratory investigations. Renal tissue biopsies were taken from patients with lupus nephritis. The frequency of

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Lei, Yu, Hong Wang, Hai-Feng Li, et al. "Diagnostic Significance of Diffusion-Weighted MRI in Renal Cancer." BioMed Research International 2015 (2015): 1–12. http://dx.doi.org/10.1155/2015/172165.

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Background.This study aimed to investigate whether diffusion-weighted imaging (DWI) could contribute to the discrimination between benign and malignant renal cancer.Methods.We searched the PubMed electronic database for eligible studies. STATA 12.0 software was used for statistical analysis. The SMD and 95% CI were calculated.Results.Decreased ADC signal was seen in all renal cancer patients (cancer tissue versus normal tissue: SMD = 1.63 and 95% CI = 0.96~2.29,P<0.001; cancer tissue versus benign tissue: SMD = 2.22 and 95% CI = 1.53~2.90 andP<0.001, resp.). MRI machine type-stratified a

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Miyazaki, M., K. Abe, T. Koji, et al. "Intraglomerular C3 synthesis in human kidney detected by in situ hybridization." Journal of the American Society of Nephrology 7, no. 11 (1996): 2428–33. http://dx.doi.org/10.1681/asn.v7112428.

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Complements 3 and 4 are known to be synthesized in diseased renal tissue and the mRNA of these complements has been demonstrated, using polymerase chain reaction, in renal biopsies from nephritic patients. However, the types of cells producing the complements in intact renal tissue have not been defined. To identify the renal cellular components involved in complement synthesis, we analyzed the expression of C3 mRNA in renal tissues from patients with immune-complex glomerulonephritis by a high-resolution in situ hybridization using digoxigenin-labeled oligonucleotide. Renal tissues from 15 pa

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Pongsittisak, Wanjak, Naphat Wutilertcharoenwong, Tanun Ngamvichchukorn, et al. "The efficacy of blind versus real-time ultrasound-guided percutaneous renal biopsy in developing country." SAGE Open Medicine 7 (January 2019): 205031211984977. http://dx.doi.org/10.1177/2050312119849770.

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Introduction: Renal biopsy is a useful diagnostic procedure. In developing countries, two techniques of renal biopsy, blind percutaneous renal biopsy and real-time ultrasound-guided percutaneous renal biopsy, have been performed. The majority of studies compared these using different types and sizes of biopsy needle. The aim of this study was to compare both techniques in resource constraint country. Method: We reviewed renal biopsy database, between 1 January 2014 to 30 June 2017. The primary outcome was the total number of glomeruli. The other outcomes were tissue adequacy and bleeding compl

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Sasaki, Hidehiko, Motonao Tanaka, Yoshifumi Saijo, et al. "Ultrasonic Tissue Characterization of Renal Cell Carcinoma Tissue." Nephron 74, no. 1 (1996): 125–30. http://dx.doi.org/10.1159/000189291.

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8

Wang, Zijie, Chao Qin, Jing Zhang, et al. "MiR-122 promotes renal cancer cell proliferation by targeting Sprouty2." Tumor Biology 39, no. 2 (2017): 101042831769118. http://dx.doi.org/10.1177/1010428317691184.

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MicroRNAs are short non-coding RNAs, which have been implicated in several biological processes. Aberrant expression of the microRNA miR-122 has frequently been reported in malignant cancers. However, the mechanism underlying the effects of miR-122 in renal cell carcinoma remains unknown. The aim of this study was to determine the biological function of miR-122 in renal cell carcinoma and to identify a novel molecular target regulated by miR-122. We measured the expression levels of Sprouty2 in six renal cell carcinoma tissue samples and adjacent non-tumor tissues by western blot analysis. We

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Yuan, Shan, Matthew Vaughan, and S. Nicholas Agoff. "Left-Sided Splenorenal Fusion With Marked Extramedullary Hematopoiesis and Concurrent Lithium Toxicity." Archives of Pathology & Laboratory Medicine 127, no. 1 (2003): e1-e3. http://dx.doi.org/10.5858/2003-127-e1-lssfwm.

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Abstract Occasionally, heterotopic splenic tissue can occur in the renal fossa secondary to splenosis following splenic trauma or splenectomy. More rarely, it can represent a developmental anomaly secondary to the fusion of splenic and renal tissues. Splenorenal fusion can present as a renal mass, mimicking primary or secondary renal neoplasms on imaging studies, and patients can also present with symptoms of hypersplenism (anemia). We report a case of splenorenal fusion in a 51-year-old woman who initially presented with lithium toxicity, anemia, thrombocytosis, and a large renal mass that mi

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Abbass, Ashwaq Najemaldeen, Salam W. Ahjels, Suhad S. Humadis, Zainab Salam Merzahs, and Samir Abdulhasan Jasims. "Role of Etanercept Against Renal Damage Induced by Cecal Ligation and Puncture in Male Rats." Azerbaijan Pharmaceutical and Pharmacotherapy J 22, no. 2 (2023): 18–21. http://dx.doi.org/10.61336/appj/22-2-06.

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Cecal ligation and puncture (CLP) is one of the most widely utilized models for rat experimental polymicrobial sepsis. In this work, we examine how the anti-oxidant and anti-inflammatory drug Etanercept (ENT), which has been FDA-approved, might lessen the damaging effects of sepsis on renal tissues. In this study, rats were divided into four groups: the control group, which received just CLP; the solvent group, which received DMSO intravenously before CLP; and the treatment group, which received ENT intraperitoneally before CLP, might possibly reduce the risk of sepsis-induced renal tissue dam

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Nagai, Junya, Hiroaki Tanaka, Naoki Nakanishi, Teruo Murakami, and Mikihisa Takano. "Role of megalin in renal handling of aminoglycosides." American Journal of Physiology-Renal Physiology 281, no. 2 (2001): F337—F344. http://dx.doi.org/10.1152/ajprenal.2001.281.2.f337.

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The role of megalin in tissue distribution of aminoglycosides was examined in normal rats and maleate-treated rats that shed megalin from the renal brush-border membrane. In normal rats, amikacin administered intravenously accumulated most abundantly in the renal cortex, followed by the renal medulla. No amikacin was detected in other tissues. Tissue distributions of amikacin were well correlated with megalin levels in each tissue. Bolus administration of gentamicin increased urinary excretion of megalin ligands (vitamin D binding protein and calcium), suggesting the competition between gentam

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Abdelkader, Amany, Julie Ho, Connie P. C. Ow, et al. "Renal oxygenation in acute renal ischemia-reperfusion injury." American Journal of Physiology-Renal Physiology 306, no. 9 (2014): F1026—F1038. http://dx.doi.org/10.1152/ajprenal.00281.2013.

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Tissue hypoxia has been demonstrated, in both the renal cortex and medulla, during the acute phase of reperfusion after ischemia induced by occlusion of the aorta upstream from the kidney. However, there are also recent clinical observations indicating relatively well preserved oxygenation in the nonfunctional transplanted kidney. To test whether severe acute kidney injury can occur in the absence of widespread renal tissue hypoxia, we measured cortical and inner medullary tissue Po2 as well as total renal O2 delivery (Do2) and O2 consumption (V̇o2) during the first 2 h of reperfusion after 60

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13

AGARWAL, ANUPAM, and HARRY S. NICK. "Renal Response to Tissue Injury." Journal of the American Society of Nephrology 11, no. 5 (2000): 965–73. http://dx.doi.org/10.1681/asn.v115965.

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Abstract. Heme oxygenase-1 (HO-1) is a microsomal enzyme involved in the degradation of heme, resulting in the generation of biliverdin, iron, and carbon monoxide. Recent attention has focused on the biologic effects of product(s) of this enzymatic reaction that have important antioxidant, anti-inflammatory, and cytoprotective functions. Induction of HO-1 occurs as an adaptive and beneficial response to a wide variety of oxidant stimuli, including heme, hydrogen peroxide, cytokines, growth factors, heavy metals, nitric oxide, and oxidized LDL. HO-1 has been implicated in several clinically rel

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14

Bulger, Ruth Ellen. "Composition of renal medullary tissue." Kidney International 31, no. 2 (1987): 556–61. http://dx.doi.org/10.1038/ki.1987.35.

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15

Sofras, F., D. Delakas, G. Vlassopoulos, C. Deliveliotis, and C. Dimopoulos. "Renal tissue damage following ESWL." International Urology and Nephrology 22, no. 2 (1990): 125–27. http://dx.doi.org/10.1007/bf02549828.

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16

Reams, G., D. Villarreal, and J. H. Bauer. "Intrarenal metabolism of angiotensin II." American Journal of Physiology-Renal Physiology 258, no. 6 (1990): F1510—F1515. http://dx.doi.org/10.1152/ajprenal.1990.258.6.f1510.

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This study investigated the intrarenal metabolism of circulating angiotensin II. Mean arterial pressure (MAP), renal hemodynamics, and the arterial, renal venous, and renal tissue concentrations of the angiotensin octapeptide [ANG-(1–8)] were examined following the constant intra-arterial infusion of tritiated angiotensin II [( 3H]ANG-(1–8)] in graded doses of 0.5, 2.0, and 2.5 ng.kg-1.min-1 in five uninephrectomized, anesthetized female dogs. The infusion of [3H]ANG-(1–8) had no significant effect on MAP, glomerular filtration rate, renal blood flow, or urine flow rate. The mean degradation r

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Gupta, Pallav, Ramesh Kumar Gupta, and Anil Kumar Bhalla. "Rapid Processing of Renal Allograft Biopsies: Conventional and Microwave." Indian Journal of Transplantation 19, no. 1 (2025): 106–9. https://doi.org/10.4103/ijot.ijot_66_24.

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Renal allograft recipients can experience episodes of graft dysfunction any time after transplantation, and renal biopsies are the gold standard tool in diagnosing the cause of graft dysfunction. Conventional tissue processing protocol followed by most laboratories is time-consuming and causes a delay in diagnosis. Microwave tissue processing generates rapid oscillation of water molecules and uniform heating of tissues, thereby increasing tissue penetration and reducing tissue processing time considerably (1.38 h). We can also perform short program processing protocol in conventional tissue pr

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Luchner, A., M. Jougasaki, E. Friedrich, et al. "Activation of cardiorenal and pulmonary tissue endothelin-1 in experimental heart failure." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 279, no. 3 (2000): R974—R979. http://dx.doi.org/10.1152/ajpregu.2000.279.3.r974.

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Endothelin-1 (ET-1) is a peptide that has been implicated in congestive heart failure (CHF). Although increased concentrations of circulating ET-1 have been repeatedly demonstrated, the activation of local ET-1 in target tissues of CHF remains poorly defined. Our objective was to characterize ET-1 tissue concentrations and gene expression of prepro ET-1 in myocardial, renal, and pulmonary tissue in rapid ventricular pacing-induced canine CHF. Progressive rapid ventricular pacing (38 days) resulted in impaired cardiovascular hemodynamics, increased atrial and left ventricular mass, decreased re

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19

Solodova, RF, MP Tolstykh, TK Isaev, et al. "Instrumental palpation in endoscopic renal surgery: case reports and analysis." Bulletin of Russian State Medical University, no. 5 (December 16, 2018): 91–97. http://dx.doi.org/10.24075/brsmu.2018.069.

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Palpation is one of the classic examination methods in open surgeries. In minimally invasive surgery, intra-operational manual palpation is impossible to use for assessing tactile characteristics of tissues. In Russia, the only available instrument for intra-operational assessment and objective registration of tissue visco-elastic properties is the Medical Tactile Endosurgical Complex (MTEC). The aim of this work was to study the performance of MTEC in renal surgery. The study was performed during nine elective laparoscopic surgeries for clear cell renal carcinoma and simple renal cysts. We ha

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Nanri, Masayuki, Kazuma Udo, Maki Kawasaki, et al. "Microwave tissue coagulator induces renal apoptotic damage to preserved normal renal tissue following partial nephrectomy." Clinical and Experimental Nephrology 13, no. 5 (2009): 424–29. http://dx.doi.org/10.1007/s10157-009-0180-8.

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21

Oertling, Estelle, Cacey Peters, Robert Wood, Jonathan Silberstein, Andrew B. Sholl, and Nadja K. Falk. "Clear cell renal cell carcinoma with heterotopic bone formation: A case report and literature review." Case Reports in Clinical Pathology 5, no. 2 (2019): 16. http://dx.doi.org/10.5430/crcp.v5n2p16.

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The phenomenon of heterotopic bone formation (osseous metaplasia) is defined as an abnormal ossification of non-skeletal tissues and does represent a rare occurrence in the renal cell carcinoma setting. We describe a case of a 40-year old man with bilateral renal cell carcinomas of the histological clear cell subtype, with the right-sided renal cell carcinoma demonstrating heterotopic bone formation, as well as the presence of intratumoral adipose tissue. The etiology of bone formation in a renal cell carcinoma is unclear, but possible explanations include a response to tissue ischemia and the

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Shevchenko, A. N., A. A. Breus, I. V. Neskubina, E. A. Dzhenkova, E. V. Filatova, and D. A. Shvyrev. "EVALUATION OF THE PROGNOSTIC SIGNIFICANCE OF SOME BIOLOGICAL FACTORS IN LOCAL AND GENERALIZED CLEAR CELL RENAL CANCER." South Russian Journal of Cancer 1, no. 1 (2020): 6–22. http://dx.doi.org/10.37748/2687-0533-2020-1-1-1.

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Purpose of the study. To evaluate the prognostic significance of biological factors VEGF-A, sVEGF-R1, VEGF-D, FGF, EGF, EGFR, IGF-1, IGF-2, IGFBP-1, IGFBP-2, somatotropin-releasing factor (GHRH) in kidney tissues (tumour tissue, tissue of the perifocal zone and conditionally intact tissue) in local and generalized clear cell renal cancer using ROC analysis.Materials and methods. Two groups of patients were included in the study. Group 1 comprised 50 patients with local kidney cancer (T1–3N0M0), while group 2 comprised 50 patients with metastatic kidney cancer (T1–4N0M1). 10% cytosolic fraction

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von Klot, Christoph A. J., Natalia Dubrowinskaja, Jörg Hennenlotter, et al. "Rho GDP dissociation inhibitor beta ARHGDIB in renal cell cancer." Journal of Clinical Oncology 33, no. 7_suppl (2015): 474. http://dx.doi.org/10.1200/jco.2015.33.7_suppl.474.

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474 Background: Rho GDP dissociation inhibitor 2 (ARHGDIB) is an important mediator of cellular signaling. The expression of ARHGDIB correlates with tumor growth and metastasis in a variety of non genitourinary cancers, however the role of ARHGDIB in renal cell cancer (RCC) has not yet been evaluated. Methods: Tissue samples from 106 patients undergoing surgery for RCC were obtained. The expression of ARHGDIB mRNA in normal kidney tissue and in corresponding cancer tissue was analyzed by means of quantitative real time PCR. Differences in mRNA expression levels were assessed using paired two-s

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Salama, Samir A., Hany H. Arab, and Ibrahim A. Maghrabi. "Troxerutin down-regulates KIM-1, modulates p38 MAPK signaling, and enhances renal regenerative capacity in a rat model of gentamycin-induced acute kidney injury." Food & Function 9, no. 12 (2018): 6632–42. http://dx.doi.org/10.1039/c8fo01086b.

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Bichlmayer, Eva-Marie, Lina Mahl, Leo Hesse, et al. "A 3D In Vivo Model for Studying Human Renal Cystic Tissue and Mouse Kidney Slices." Cells 11, no. 15 (2022): 2269. http://dx.doi.org/10.3390/cells11152269.

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(1) Background: Autosomal dominant polycystic kidney disease (ADPKD) is a frequent monogenic disorder that leads to progressive renal cyst growth and renal failure. Strategies to inhibit cyst growth in non-human cyst models have often failed in clinical trials. There is a significant need for models that enable studies of human cyst growth and drug trials. (2) Methods: Renal tissue from ADPKD patients who received a nephrectomy as well as adult mouse kidney slices were cultured on a chorioallantoic membrane (CAM) for one week. The cyst volume was monitored by microscopic and CT-based applicati

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Benigni, A., N. Perico, F. Gaspari, et al. "Increased renal endothelin production in rats with reduced renal mass." American Journal of Physiology-Renal Physiology 260, no. 3 (1991): F331—F339. http://dx.doi.org/10.1152/ajprenal.1991.260.3.f331.

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Renal endothelin (ET) production was investigated in rats after renal mass ablation, a model of progressive renal disease characterized by glomerular hemodynamic alterations and capillary thrombosis, and in sham-operated animals. Thrombin stimulation of renal cortical tissue from rats with renal mass reduction, 45 but not 7 days after surgery, generated significantly (P less than 0.01) more ET than tissue from sham-operated animals. Exposure to thrombin of isolated glomeruli from remnant but not sham kidneys also significantly (P less than 0.01) increased ET production compared with unstimulat

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27

Zhang, Dan, Fan Zhang, Meiling Liu, Mingchao Zhang, Jiong Zhang, and Jinquan Wang. "Disease activity prediction and prognosis of anti-GBM nephritis based on T lymphocyte subset ratios." International Journal of Immunopathology and Pharmacology 35 (January 2021): 205873842110393. http://dx.doi.org/10.1177/20587384211039391.

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Introduction Cell-mediated autoimmunity, especially the autoreactivity of T cells, is known to underlie the initiation of anti-glomerular basement membrane disease. However, the T lymphocyte subsets that determine the disease activity, renal fibrosis, and prognosis of anti-GBM disease have not been clearly elucidated. Methods The T lymphocyte subsets (CD4+ and CD8+) were examined on peripheral blood and renal biopsy tissues from 65 patients with biopsy proven anti-GBM disease. Patients were divided into the high ratio group and low ratio group according to the cutoff values in the receiver ope

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Pedersen, Michael, Zsolt Vajda, Hans Stødkilde-Jørgensen, Søren Nielsen, and Jørgen Frøkiær. "Furosemide increases water content in renal tissue." American Journal of Physiology-Renal Physiology 292, no. 5 (2007): F1645—F1651. http://dx.doi.org/10.1152/ajprenal.00060.2006.

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The present study was designed to evaluate the short-term effects of intravenous administration of furosemide on key functions in the kidney cortex and the outer and inner medulla of rats by using magnetic resonance imaging (MRI). Renal tissue water content, renal tissue oxygenation (in relation to the magnetic resonance spin-spin relaxation rate), the apparent diffusion coefficient (ADC) of water, and volume of renal blood flow were measured. Furosemide administration resulted in an increased water content in all regions of the kidney. In parallel with this, we found a significant reduction i

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Yoshida, K., T. Yoneda, K. Fujimoto, Y. Hirao, and N. Konishi. "Pentosidine and Its Deposition in Renal Tissue in Renal Transplantation." Transplantation Proceedings 37, no. 10 (2005): 4266–72. http://dx.doi.org/10.1016/j.transproceed.2005.11.018.

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Lin, Neil Y. C., Kimberly A. Homan, Sanlin S. Robinson, et al. "Renal reabsorption in 3D vascularized proximal tubule models." Proceedings of the National Academy of Sciences 116, no. 12 (2019): 5399–404. http://dx.doi.org/10.1073/pnas.1815208116.

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Three-dimensional renal tissues that emulate the cellular composition, geometry, and function of native kidney tissue would enable fundamental studies of filtration and reabsorption. Here, we have created 3D vascularized proximal tubule models composed of adjacent conduits that are lined with confluent epithelium and endothelium, embedded in a permeable ECM, and independently addressed using a closed-loop perfusion system to investigate renal reabsorption. Our 3D kidney tissue allows for coculture of proximal tubule epithelium and vascular endothelium that exhibits active reabsorption via tubu

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Aeddula, Narothama, Wisit Cheungpasitporn, Charat Thongprayoon, and Samata Pathireddy. "Epicardial Adipose Tissue and Renal Disease." Journal of Clinical Medicine 8, no. 3 (2019): 299. http://dx.doi.org/10.3390/jcm8030299.

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Epicardial adipose tissue (EAT) is derived from splanchnic mesoderm, localized anatomically between the myocardium and pericardial visceral layer, and surrounds the coronary arteries. Being a metabolically active organ, EAT secretes numerous cytokines, which moderate cardiovascular morphology and function. Through its paracrine and vasocrine secretions, EAT may play a prominent role in modulating cardiac function. EAT protects the heart in normal physiological conditions by secreting a variety of adipokines with anti-atherosclerotic properties, and in contrast, secretes inflammatory molecules

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Pavone-Macaluso, M. "Tissue Mast Cells in Renal Diseases." Acta Pathologica Microbiologica Scandinavica 50, no. 4 (2009): 337–46. http://dx.doi.org/10.1111/j.1699-0463.1960.tb01202.x.

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Saito, S., I. Higa, Y. Koyama, et al. "Immature Renal Tissue in Inguinal Canal." Journal of Urology 142, no. 1 (1989): 106–7. http://dx.doi.org/10.1016/s0022-5347(17)38674-3.

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Fernbach, Sandra K., Elizabeth A. Holland, Irwin Benuck, and Stephanie Young. "Hypertension Induced by Occult Renal Tissue." Journal of Urology 138, no. 4 Part 1 (1987): 842–44. http://dx.doi.org/10.1016/s0022-5347(17)43392-1.

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Olweny, Ephrem O., and Jeffrey A. Cadeddu. "Novel methods for renal tissue ablation." Current Opinion in Urology 22, no. 5 (2012): 379–84. http://dx.doi.org/10.1097/mou.0b013e328355ecf5.

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Southwood, Robin L., Bruce A. Mueller, and John B. Copley. "Soft Tissue Calcification in Renal Failure." DICP 24, no. 9 (1990): 855–59. http://dx.doi.org/10.1177/106002809002400913.

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Nauth, B., H. Faust, S. Zelger, and R. Bergner. "Determination of ibandronate in renal tissue." Bone 42 (March 2008): S103. http://dx.doi.org/10.1016/j.bone.2007.12.199.

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Holcomb, George. "Hypertension induced by occult renal tissue." Journal of Pediatric Surgery 23, no. 5 (1988): 500. http://dx.doi.org/10.1016/s0022-3468(88)80513-x.

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Gaskill, Sarah J., Katherine Kagen-Hallett, and Arthur E. Marlin. "Diastematomyelia Associated with Ectopic Renal Tissue." Pediatric Neurosurgery 14, no. 2 (1988): 108–11. http://dx.doi.org/10.1159/000120372.

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Zimmerman, Kurt A., Cheng J. Song, Zhang Li, et al. "Tissue-Resident Macrophages Promote Renal Cystic Disease." Journal of the American Society of Nephrology 30, no. 10 (2019): 1841–56. http://dx.doi.org/10.1681/asn.2018080810.

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BackgroundMutations affecting cilia proteins have an established role in renal cyst formation. In mice, the rate of cystogenesis is influenced by the age at which cilia dysfunction occurs and whether the kidney has been injured. Disruption of cilia function before postnatal day 12–14 results in rapid cyst formation; however, cyst formation is slower when cilia dysfunction is induced after postnatal day 14. Rapid cyst formation can also be induced in conditional adult cilia mutant mice by introducing renal injury. Previous studies indicate that macrophages are involved in cyst formation, howeve

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Mobaraki, Ghalib, Shuai Shi, Dan Liu, et al. "Mapping of Human Polyomavirus in Renal Cell Carcinoma Tissues." International Journal of Molecular Sciences 25, no. 15 (2024): 8213. http://dx.doi.org/10.3390/ijms25158213.

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Worldwide, the incidence of renal cell carcinoma (RCC) is rising, accounting for approximately 2% of all cancer diagnoses and deaths. The etiology of RCC is still obscure. Here, we assessed the presence of HPyVs in paraffin-embedded tissue (FFPE) resected tissue from patients with RCC by using different molecular techniques. Fifty-five FFPE tissues from 11 RCC patients were included in this study. Consensus and HPyV-specific primers were used to screen for HPyVs. Both PCR approaches revealed that HPyV is frequently detected in the tissues of RCC kidney resections. A total of 78% (43/55) of the

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Geoffroy, Karen, Patrick Laplante, Sylvie Clairefond, et al. "High Levels of MFG-E8 Confer a Good Prognosis in Prostate and Renal Cancer Patients." Cancers 14, no. 11 (2022): 2790. http://dx.doi.org/10.3390/cancers14112790.

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Milk fat globule-epidermal growth factor-8 (MFG-E8) is a glycoprotein secreted by different cell types, including apoptotic cells and activated macrophages. MFG-E8 is highly expressed in a variety of cancers and is classically associated with tumor growth and poor patient prognosis through reprogramming of macrophages into the pro-tumoral/pro-angiogenic M2 phenotype. To date, correlations between levels of MFG-E8 and patient survival in prostate and renal cancers remain unclear. Here, we quantified MFG-E8 and CD68/CD206 expression by immunofluorescence staining in tissue microarrays constructe

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Nunes, Priscila Conrado Guerra, Lilimar da Silveira Rioja, Janice Mery Chicarino de Oliveira Coelho, et al. "Renal Injury in DENV-4 Fatal Cases: Viremia, Immune Response and Cytokine Profile." Pathogens 8, no. 4 (2019): 223. http://dx.doi.org/10.3390/pathogens8040223.

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Dengue virus (DENV) infections may result in asymptomatic cases or evolve into a severe disease, which involves multiple organ failure. Renal involvement in dengue can be potentially related to an increased mortality. Aiming to better understand the role of DENV in renal injury observed in human fatal cases, post-mortem investigations were performed in four DENV-4 renal autopsies during dengue epidemics in Brazil. Tissues were submitted to histopathology, immunohistochemistry, viral quantification, and characterization of cytokines and inflammatory mediators. Probably due the high viral load,

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Wang, Furong, Ling Gao, Bendi Gong, et al. "Tissue-specific expression of PPAR mRNAs in diabetic rats and divergent effects of cilostazol." Canadian Journal of Physiology and Pharmacology 86, no. 7 (2008): 465–71. http://dx.doi.org/10.1139/y08-043.

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Cilostazol and ligands of peroxisome proliferator-activated receptors (PPARs) have been effectively used to alleviate diabetic complications, but the common and tissue-specific expression patterns of PPARs in different tissues in diabetic patients and those treated with cilostazol have not been reported. Here, we aimed to assess the effects of diabetes and cilostazol on mRNA expression of PPARα and PPARγ in the aorta, renal cortex, and retina of diabetic rats treated with cilostazol for 8 weeks. PPARα mRNA expression showed uniform downregulation in all these tissues in diabetic rats, and this

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Olgac, Ufuk, and Vartan Kurtcuoglu. "Renal oxygenation: preglomerular vasculature is an unlikely contributor to renal oxygen shunting." American Journal of Physiology-Renal Physiology 308, no. 7 (2015): F671—F688. http://dx.doi.org/10.1152/ajprenal.00551.2014.

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The primary aim of this study was to assess the plausibility of preglomerular arterial-to-venous oxygen shunting in the kidney. To this end, we have developed a segment-wise three-dimensional computational model that takes into account transport processes in arteries, veins, cortical tissue, and capillaries. Our model suggests that the amount of preglomerular oxygen shunting is negligible. Consequently, it is improbable that preglomerular shunting contributes to the hypothesized regulation of renal oxygenation. Cortical tissue oxygenation is more likely determined by the interplay between oxyg

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Forhead, Alison J., Katrina Curtis, Ellen Kaptein, Theo J. Visser, and Abigail L. Fowden. "Developmental Control of Iodothyronine Deiodinases by Cortisol in the Ovine Fetus and Placenta Near Term." Endocrinology 147, no. 12 (2006): 5988–94. http://dx.doi.org/10.1210/en.2006-0712.

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Preterm infants have low serum T4 and T3 levels, which may partly explain the immaturity of their tissues. Deiodinase enzymes are important in determining the bioavailability of thyroid hormones: deiodinases D1 and D2 convert T4 to T3, whereas deiodinase D3 inactivates T3 and produces rT3 from T4. In human and ovine fetuses, plasma T3 rises near term in association with the prepartum cortisol surge. This study investigated the developmental effects of cortisol and T3 on tissue deiodinases and plasma thyroid hormones in fetal sheep during late gestation. Plasma cortisol and T3 concentrations in

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Ow, Connie P. C., Jennifer P. Ngo, Md Mahbub Ullah, et al. "Absence of renal hypoxia in the subacute phase of severe renal ischemia-reperfusion injury." American Journal of Physiology-Renal Physiology 315, no. 5 (2018): F1358—F1369. http://dx.doi.org/10.1152/ajprenal.00249.2018.

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Tissue hypoxia has been proposed as an important event in renal ischemia-reperfusion injury (IRI), particularly during the period of ischemia and in the immediate hours following reperfusion. However, little is known about renal oxygenation during the subacute phase of IRI. We employed four different methods to assess the temporal and spatial changes in tissue oxygenation during the subacute phase (24 h and 5 days after reperfusion) of a severe form of renal IRI in rats. We hypothesized that the kidney is hypoxic 24 h and 5 days after an hour of bilateral renal ischemia, driven by a disturbed

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Li, Ting, Kexin Yang, Yinghao Tong, Shangze Guo, Wei Gao, and Xiangyu Zou. "Targeted Drug Therapy for Senescent Cells Alleviates Unilateral Ureteral Obstruction-Induced Renal Injury in Rats." Pharmaceutics 16, no. 6 (2024): 695. http://dx.doi.org/10.3390/pharmaceutics16060695.

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Hydronephrosis resulting from unilateral ureteral obstruction (UUO) is a common cause of renal injury, often progressing to late-stage renal fibrosis or even potential renal failure. Renal injury and repair processes are accompanied by changes in cellular senescence phenotypes. However, the mechanism is poorly understood. The purpose of this study is to clarify the changes in senescence phenotype at different time points in renal disease caused by UUO and to further investigate whether eliminating senescent cells using the anti-senescence drug ABT263 could attenuate UUO-induced renal disease.

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Fahmy, Nader, Mark Woo, Mona Alameldin, et al. "Endogenous biotin expression in renal and testicular tumors and literature review." Canadian Urological Association Journal 8, no. 7-8 (2014): 268. http://dx.doi.org/10.5489/cuaj.1810.

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Introduction: The aim of this study was to examine endogenous biotin levels in tumour specimens collected from patients with renal and testicular tumours and compare them to the surrounding non-neoplastic surgical margin.Methods: Frozen samples were obtained from the Ontario Tumour Bank. Renal and testicular tumour tissue were included in this study. Normal tissue from the negative surgical margins of each tumour served as a control. Biotin detection in tissue specimens was determined using immunohistochemistry (IHC).Results: Specimens collected from 56 patients (36 men and 20 women) were incl

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Латышев, Виталий Дмитриевич, К. А. Енневайн, А. А. Соловьева, А. Е. Киценко, Н. В. Цветаева та Е. А. Лукина. "Особенности метаболизма железа у пациентов с пароксизмальной ночной гемоглобинурией: результаты одноцентрового амбиспективного клинического исследования". Clinical Oncohematology 18, № 1 (2025): 1–9. https://doi.org/10.21320/2500-2139-2025-18-1-1-9.

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BACKGROUND. Paroxysmal nocturnal hemoglobinuria (PNH) is a rare clonal hematologic disease manifesting among others in hemolytic anemia. Due to predominantly intravascular hemolysis, iron metabolism in PNH patients is characterized by a number of features. Pathogenetic treatment with complement C5-inhibitors modifies the course of hemolysis and results in iron metabolic changes. AIM. To characterize the serum parameters of iron metabolism in PNH patients and to evaluate T2* MRIs of the liver and kidneys. MATERIALS & METHODS. Serum parameters of iron metabolism measured in 97 analyses and 9

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