Relevant bibliographies by topics / Soluble CD89

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Soluble CD89'

Author: Grafiati
Published: 22 June 2024
Last updated: 31 July 2025

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Journal articles on the topic "Soluble CD89"

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Launay, Pierre, Béatrice Grossetête, Michelle Arcos-Fajardo та ін. "Fcα Receptor (Cd89) Mediates the Development of Immunoglobulin a (Iga) Nephropathy (Berger's Disease)". Journal of Experimental Medicine 191, № 11 (1999): 1999–2010. http://dx.doi.org/10.1084/jem.191.11.1999.

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The pathogenesis of immunoglobulin A (IgA) nephropathy (IgAN), the most prevalent form of glomerulonephritis worldwide, involves circulating macromolecular IgA1 complexes. However, the molecular mechanism(s) of the disease remain poorly understood. We report here the presence of circulating soluble FcαR (CD89)-IgA complexes in patients with IgAN. Soluble CD89 was identified as a glycoprotein with a 24-kD backbone that corresponds to the expected size of CD89 extracellular domains. To demonstrate their pathogenic role, we generated transgenic (Tg) mice expressing human CD89 on macrophage/monocy
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van Zandbergen, Ger, Ralf Westerhuis, Ngaisah Klar Mohamad, Jan G. J. van de Winkel, Mohamed R. Daha та Cees van Kooten. "Crosslinking of the Human Fc Receptor for IgA (FcαRI/CD89) Triggers FcR γ-Chain-Dependent Shedding of Soluble CD89". Journal of Immunology 163, № 11 (1999): 5806–12. http://dx.doi.org/10.4049/jimmunol.163.11.5806.

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Abstract CD89/FcαRI is a 55- to 75-kDa type I receptor glycoprotein, expressed on myeloid cells, with important immune effector functions. At present, no information is available on the existence of soluble forms of this receptor. We developed an ELISA for the detection of soluble CD89 (sCD89) forms and investigated the regulation of sCD89 production. PMA/ionomycin stimulation of monocytic cell lines (U937, THP-1, and MM6), but not of neutrophils, resulted in release of sCD89. Crosslinking of CD89 either via its ligand IgA or with anti-CD89 mAbs similarly resulted in sCD89 release. Using CD89-
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Esteve Cols, Clara, Freddzia-Amanda Graterol Torres, Bibiana Quirant Sánchez, et al. "Immunological Pattern in IgA Nephropathy." International Journal of Molecular Sciences 21, no. 4 (2020): 1389. http://dx.doi.org/10.3390/ijms21041389.

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The current gold-standard diagnostic technique for IgA nephropathy (IgAN), the leading form of primary glomerulonephritis, is renal biopsy. CD89 (the main IgA receptor) is expressed on the surface of monocytes and plays a role in disease pathogenesis. Immunocomplexes formed by sCD89 (soluble form) and Gd-IgA1 are related to disease prognosis. We hypothesize that reduced CD89 surface expression on monocytes may be a marker of disease severity. We aimed to analyze leukocyte subpopulations in peripheral blood and CD89 surface expression on monocytes in a prospective study of 22 patients and 12 he
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Wu, Haiting, Xiaoyan Wang, Zhe Yang, et al. "Serum Soluble CD89-IgA Complexes Are Elevated in IgA Nephropathy without Immunosuppressant History." Disease Markers 2020 (January 16, 2020): 1–6. http://dx.doi.org/10.1155/2020/8393075.

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Purpose. CD89 (FcαRI), the receptor of IgA, can shed from cells to form complexes with IgA in serum and is supposed to participate in the pathogenesis of IgA nephropathy (IgAN). There are contradictory results on their utility in clinical practice. This study is aimed at investigating whether sCD89-IgA complexes can help in the diagnosis or evaluation of the disease. Methods. A sandwich ELISA was established using anti-CD89 as a capture antibody and HRP-conjugated anti-IgA as a detection antibody. This method was used to measure serum levels of sCD89-IgA complexes in IgAN patients without immu
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Cambier, Alexandra, Patrick J. Gleeson, Lilia Abbad, et al. "Soluble CD89 is a critical factor for mesangial proliferation in childhood IgA nephropathy." Kidney International 101, no. 2 (2022): 274–87. http://dx.doi.org/10.1016/j.kint.2021.09.023.

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Berthelot, Laureline, Thomas Robert, Vincent Vuiblet, et al. "Recurrent IgA nephropathy is predicted by altered glycosylated IgA, autoantibodies and soluble CD89 complexes." Kidney International 88, no. 4 (2015): 815–22. http://dx.doi.org/10.1038/ki.2015.158.

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Vuong, Mai T., Mirjana Hahn-Zoric, Sigrid Lundberg, et al. "Association of soluble CD89 levels with disease progression but not susceptibility in IgA nephropathy." Kidney International 78, no. 12 (2010): 1281–87. http://dx.doi.org/10.1038/ki.2010.314.

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Hahn-Zoric, Mirjana, Mai Vuong, Sigrid Lundberg, et al. "Su.82. Evidence for Genetic Regulation of Fc Alpha Receptor (CD89) Expression: Study of Soluble CD89 in Plasma of IgA Nephropathy Patients and Healthy Controls." Clinical Immunology 127 (January 2008): S151. http://dx.doi.org/10.1016/j.clim.2008.03.433.

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Hahn-Zoric, Mirjana, Neda Tahmasebifar, Cees van Kooten, et al. "Immunoassays for Detection of Soluble Fc Alpha Receptor (CD89) in Plasma of IgA Nephropathy Patients." Clinical Immunology 123 (2007): S54—S55. http://dx.doi.org/10.1016/j.clim.2007.03.335.

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Berthelot, Laureline, Christina Papista, Thiago T. Maciel, et al. "Transglutaminase is essential for IgA nephropathy development acting through IgA receptors." Journal of Experimental Medicine 209, no. 4 (2012): 793–806. http://dx.doi.org/10.1084/jem.20112005.

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IgA nephropathy (IgAN) is a common cause of renal failure worldwide. Treatment is limited because of a complex pathogenesis, including unknown factors favoring IgA1 deposition in the glomerular mesangium. IgA receptor abnormalities are implicated, including circulating IgA–soluble CD89 (sCD89) complexes and overexpression of the mesangial IgA1 receptor, TfR1 (transferrin receptor 1). Herein, we show that although mice expressing both human IgA1 and CD89 displayed circulating and mesangial deposits of IgA1–sCD89 complexes resulting in kidney inflammation, hematuria, and proteinuria, mice expres
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Dissertations / Theses on the topic "Soluble CD89"

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Cambier, Alexandra. "Une étude translationnelle de la néphropathie à IgA de l’enfant : des variants génétiques à la physiopathologie des biomarqueurs et leurs liens de causalité avec les lésions histologiques." Electronic Thesis or Diss., Sorbonne université, 2020. http://www.theses.fr/2020SORUS371.

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La N-IgA est une maladie auto-immune impliquant 3 biomarqueurs, les IgA1 hypogalactosylées (Gd)-lgA1,des auto-anticorps IgG anti-Gd-IgA1 et le récepteur soluble (s) CD89 IgA Fc lié à l'IgA (sCD89-IgA). Le sCD8 en circulation sous forme libre ou complexé aux IgA serait un acteur clé de la prolifération mésangiale suite à une interaction avec le récepteur CD71. Les complexes sCD89-IgA et le sCD89 libre étaient fortement liés à la protéinurie et aux lésions histologiques de prolifération in vivo et in vitro. Ces découvertes révèlent un nouveau rôle du sCD89 comme biomarqueur et comme potentielle
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Staab, Christine [Verfasser], and Lars [Akademischer Betreuer] Nitschke. "Production of Recombinant Human Soluble CD83 in an Eukaryotic System and Generation of Tissue-Specific CD83 Knock-out Mice / Christine Staab. Betreuer: Lars Nitschke." Erlangen : Universitätsbibliothek der Universität Erlangen-Nürnberg, 2011. http://d-nb.info/1015475329/34.

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Sanchez, Vidales Maria Del Mar. "Release of Soluble Interleukin-7 α Receptor (CD127) from CD8+ T-Cells and Human Thymocytes". Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/34631.

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ABSTRACT Background Interleukin-7 (IL-7) is a cytokine crucial for T-cell development and homeostasis. IL-7 is thought to be a limited resource, and its interaction with the IL-7 receptor (IL-7R) has effects on increasing cell survival, proliferation and cytolytic function. Considering the roles of IL-7, it is no surprise that the expression of the IL-7 receptor alpha chain (CD127) is tightly regulated. Despite increased levels of soluble CD127 (sCD127) being detected in a number of disease states and being associated with disease activity, the biological function of sCD127 and its clinical
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Aloufi, Nawaf. "The role of sCD127 in IL-7-Mediated T Cell Homeostasis in Vivo." Thesis, Université d'Ottawa / University of Ottawa, 2020. http://hdl.handle.net/10393/41089.

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Interleukin-7 is an essential cytokine that plays a major role in the development and homeostatic maintenance of T-cells. The presence of soluble forms of various cytokine receptors have been proposed to be involved in the endogenous regulation of cytokine activity. Due to the natural ability of soluble CD127 (sCD127) to bind to IL-7, there is an interest in its potential application as an immunotherapeutic agent in diseases, where IL-7 has been found to be relevant, including HIV infection. In this study, I hypothesize that by administering sCD127 to healthy mice, IL-7 activity should be enha
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Morice, Yoann. "Le virus de l'hépatite C : études de la variabilité inter- et intra-génomique : production sous forme soluble de protéines membranaires impliquées dans l'interaction virus-cellule hôte." Paris 7, 2002. http://www.theses.fr/2002PA077217.

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SADAT, SOWTI COMBADIERE BEHAZINE. "Controle des reponses cytotoxiques par les lymphocytes cd8+cd57+ au cours de l'infection par le vih et apres transplanttion de moelle osseuse / caracterisation d'un facteur soluble inhibiteur (icf)." Paris 7, 1993. http://www.theses.fr/1993PA077357.

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L'infection par le vih induit une reponse cytotoxique specifique d'antigene intense mais incapable de proteger les patients infectes par le vih. Nous avons contribue a l'etude de ce probleme en decrivant une nouvelle propriete des lymphocytes t cd8+ coexprimant le marqueur cd57. En effet, les cellules cd8+cd57+ augmentees chez les patients infectes par le vih et apres transplantation de moelle osseuse, sont spontanement capables d'inhiber la phase effectrice des activites cytotoxiques mediees par les ctl specifiques d'antigenes, les cellules nk et lak. De plus, les cellules cd8+cd57+ exercent
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Kastenmüller, Kathrin [Verfasser], Dirk [Akademischer Betreuer] Busch, Wolfgang [Akademischer Betreuer] Wurst, and Siegfried [Akademischer Betreuer] Scherer. "Generation of CD8+ T cell mediated protective immunity upon vaccination with soluble antigen : involvement of immunmodulatory factors like adjuvant or regulatory lymphocytes / Kathrin Kastenmüller. Gutachter: Wolfgang Wurst ; Siegfried Scherer. Betreuer: Dirk Busch." München : Universitätsbibliothek der TU München, 2006. http://d-nb.info/1058141163/34.

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Dioszeghy, Vincent. "Etude des activités antivirales solubles non cytolytiques des lymphocytes CD8+ au cours de l'infection par SIV : Effet d'un traitement par polychimiothérapie post-exposition, effet d'un traitement immuno-modulateur par l'interleukine 2 et mécanismes de reconstitution lymphocytaire T CD4+." Paris 11, 2006. http://www.theses.fr/2006PA11T012.

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Müssig, Oliver. "Bedeutung des löslichen CD14-Rezeptors in Plasma und Urin als immunologischer Parameter nach Nierentransplantation und sein Verhältnis zu den löslichen Rezeptoren IL2R, CD4 und CD8." Doctoral thesis, 2011. http://hdl.handle.net/11858/00-1735-0000-0006-B1DF-C.

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Berg, Martina [Verfasser]. "Induction of cytotoxic CD8+ T cells by a soluble HLA-derived tumor-associated peptide and regulation of CD8+ T cell activation by CTLA-4 / vorgelegt von Martina Berg." 2005. http://d-nb.info/977277305/34.

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Book chapters on the topic "Soluble CD89"

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Lusso, Paolo. "Suppression of Primate Immunodeficiency Lentiretroviruses by CD8+ T-Cell-Derived Soluble Factors." In AIDS Pathogenesis. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-017-0685-8_8.

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Weiss, C., S. Hohaus, E. Ogniben, M. Dörner, and R. Haas. "Effect of Recombinant Human Granulocyte—Macrophage Colony-Stimulating Factor on Serum Levels of Soluble CD25, CD8, and CD4 in Patients with Hodgkin’s Disease in Sensitive Relapse." In Cytokines in Hemopoiesis, Oncology, and AIDS II. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-48715-6_74.

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Bayhan, Ülkü, and Remziye Banka. "Nobel Molecule Artemisia-Annua and T-CD8+ Receptor Bestfit Interactions." In Matematik ve Fen Bilimleri Üzerine Araştırmalar-II. Özgür Yayınları, 2023. http://dx.doi.org/10.58830/ozgur.pub165.c799.

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Effector T cells, one of the body's defense cells, have an active role in the regulation of immune responses. These T cells have assumed the role of defense between the soluble substances they produce and the ligand interactions on their surface, and the regulators of the immune system. The CD8+ cells surface receptor control intracellular pathogens. CD8+ cells directly mark cells infected with intracellular organisms. In this study, the interactions between Artemisia annua ligand molecule and CD8+ surface protein molecules were investigated. AutoDock 4 –MGLTolls package program was used for t
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"CD8 Antiviral Soluble Factors and Human Immunodeficiency Virus (HIV) Control." In Soluble Factors Mediating Innate Immune Responses to HIV Infection, edited by Nitin K. Saksena, Jing Qin Wu, Katherine Lau, Li Zhou, Maly Soedjono, and Bin Wang. BENTHAM SCIENCE PUBLISHERS, 2012. http://dx.doi.org/10.2174/978160805006211001010001.

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Magnin, Morgane, Philippe Guillaume, George Coukos, Alexandre Harari, and Julien Schmidt. "High-throughput identification of human antigen-specific CD8+ and CD4+ T cells using soluble pMHC multimers." In Methods in Enzymology. Elsevier, 2020. http://dx.doi.org/10.1016/bs.mie.2019.05.019.

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Conference papers on the topic "Soluble CD89"

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Soodaeva, Svetlana K., Nailya Kubysheva, Larisa Postnikova, Viktor Novikov, Tatyana Eliseeva, and Igor A. Klimanov. "The level of soluble CD8 molecules in serum and sputum in COPD patients with an exacerbation." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa3653.

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Asai, Kazuhisa, Nahoko Shiratsuchi-Nakagawa, Gakuya Tamagaki, et al. "Soluble Perforin, A Marker Of CD8+ T Lymphocyte Activation In Epithelial Lining Fluid In COPD Patients." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4545.

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Haile, Samuel, Sonia P. Dalal, Virginia Clements, Koji Tamada, and Suzanne Ostrand-Rosenberg. "Abstract 462: Soluble CD80 restores T-cell activation and overcomes tumor cell programmed death ligand-1-mediated suppression." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-462.

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Horn, Lucas, Virginia Clements, and Suzanne Ostrand-Rosenberg. "Abstract B013: A soluble form of CD80 enhances anti-tumor immunity by inhibiting PDL1 immune suppression and does not suppress via CTLA-4." In Abstracts: CRI-CIMT-EATI-AACR Inaugural International Cancer Immunotherapy Conference: Translating Science into Survival; September 16-19, 2015; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr15-b013.

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Ishikawa, Flavia M., Manuela Llano, Adriana Parra, Bertha Valencia, Alejandra Muñoz та Gustavo Salguero. "947 Soluble hIL-15/IL-15Rα complex enhances human NK and CD8 cytotoxicity and promotes killing of human melanoma in mice bearing a functional human immune system". У SITC 39th Annual Meeting (SITC 2024) Abstracts. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/jitc-2024-sitc2024.0947.

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Long, Tiha M., and Suzanne Ostrand-Rosenberg. "Abstract B017: A soluble form of CD80 inhibits PD-L1 immune suppression and stimulates T cells through CD28-specific pathways indicating potential for increased therapeutic activity over checkpoint inhibition alone." In Abstracts: CRI-CIMT-EATI-AACR Inaugural International Cancer Immunotherapy Conference: Translating Science into Survival; September 16-19, 2015; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr15-b017.

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Zhang, Jinyu, Pablo Larrocha, Payal Dhar, and Jennifer Wu. "Abstract A77: Antibody targeting tumor-derived soluble NKG2D ligand sMIC provides dual costimulation of CD8 T cells and enables sMIC+ tumors to respond to PD1/PD-L1 blockade therapy." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; November 17-20, 2019; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm19-a77.

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