Relevant bibliographies by topics / Sporadic amyotrophic lateral sclerosis

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Sporadic amyotrophic lateral sclerosis'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Sporadic amyotrophic lateral sclerosis"

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Saudagar, Ravindranath, and Lajri Garge. "Amyotrophic Lateral Sclerosis: An Overview." Journal of Drug Delivery and Therapeutics 9, no. 3 (2019): 613–16. http://dx.doi.org/10.22270/jddt.v9i3.2874.

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Amyotrophic Lateral Sclerosis is an adult-onset neurodegenerative disease that causes paralysis. Presently it is incurable and rapidly progressive with a survival of 4-5 years from onset. It is degeneration of upper and lower motor neurons which results in weakness and wasting of muscles in arms, legs, trunk and bulbar region. There are two types sporadic and familial amyotrophic lateral sclerosis. The age of onset of people with familial is widely reported as being about 10 years younger than for those with apparently sporadic amyotrophic lateral sclerosis. Epidemiological studies show a worl
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Schymick, J. C., K. Talbot, and B. J. Traynor. "Genetics of sporadic amyotrophic lateral sclerosis." Human Molecular Genetics 16, R2 (2007): R233—R242. http://dx.doi.org/10.1093/hmg/ddm215.

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Swerdlow, Russell H., Janice K. Parks, David S. Cassarino, et al. "Mitochondria in Sporadic Amyotrophic Lateral Sclerosis." Experimental Neurology 153, no. 1 (1998): 135–42. http://dx.doi.org/10.1006/exnr.1998.6866.

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Mackenzie, Ian R. A., Eileen H. Bigio, Paul G. Ince, et al. "Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis withSOD1 mutations." Annals of Neurology 61, no. 5 (2007): 427–34. http://dx.doi.org/10.1002/ana.21147.

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Armon, C., L. T. Kurland, J. R. Daube, and P. C. O'Brien. "Epidemiologic correlates of sporadic amyotrophic lateral sclerosis." Neurology 41, no. 7 (1991): 1077. http://dx.doi.org/10.1212/wnl.41.7.1077.

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Preux, P. M., Ph Couratier, F. Boutros-Toni, et al. "Survival Prediction in Sporadic Amyotrophic Lateral Sclerosis." Neuroepidemiology 15, no. 3 (1996): 153–60. http://dx.doi.org/10.1159/000109902.

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Lai, Shiao-Lin, Yevgeniya Abramzon, Jennifer C. Schymick, et al. "FUS mutations in sporadic amyotrophic lateral sclerosis." Neurobiology of Aging 32, no. 3 (2011): 550.e1–550.e4. http://dx.doi.org/10.1016/j.neurobiolaging.2009.12.020.

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Freischmidt, Axel, Kathrin Müller, Lisa Zondler, et al. "Serum microRNAs in sporadic amyotrophic lateral sclerosis." Neurobiology of Aging 36, no. 9 (2015): 2660.e15–2660.e20. http://dx.doi.org/10.1016/j.neurobiolaging.2015.06.003.

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Andrew, Eisen, and Krieger Charles. "Pathogenic Mechanisms in Sporadic Amyotrophic Lateral Sclerosis." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 20, no. 4 (1993): 286–96. http://dx.doi.org/10.1017/s0317167100048198.

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ABSTRACT:In recognition of the 100th anniversary of Charcot’s death we have reviewed possible pathogenic mechanisms in amyotrophic lateral sclerosis (ALS). Advances in the last 5 years in molecular biology and genetics have identified mutations in the cytosolic dismutase (SODI) gene in some patients with familial ALS raising the possibility that oxidative stress may be involved in the pathogenesis. An excitotoxic pathogenesis has been implicated based on elevated plasma and CSF levels of amino acids and altered contents of amino acids in the nervous system of ALS patients and changes in the nu
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Idrisoglu, H. A. "Sporadic Amyotrophic Lateral Sclerosis (SALS) and environment." Journal of the Neurological Sciences 333 (October 2013): e461. http://dx.doi.org/10.1016/j.jns.2013.07.1642.

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Dissertations / Theses on the topic "Sporadic amyotrophic lateral sclerosis"

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Gros-Louis, François. "Genetics of familial and sporadic amyotrophic lateral sclerosis." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111859.

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Diseases affecting motor neurons, such as amyotrophic lateral sclerosis (Lou Gerhig's disease), hereditary spastic paraplegia and spinal bulbar muscular atrophy (Kennedy's disease) form a heterogeneous group of chronic progressive diseases and are among the most puzzling yet untreatable illnesses. Over the last decade identification of mutations in genes predisposing to these disorders has provided the means to better understand their pathogenesis. The discovery 13 years ago of SOD1 mutations linked to ALS, which account for less than 2% of all cases, had a major impact in the field. However,
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Forsberg, Karin. "Misfolded superoxide dismutase-1 in sporadic and familial Amyotrophic Lateral Sclerosis." Doctoral thesis, Umeå universitet, Patologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-47550.

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Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative syndrome of unknown etiology that most commonly affects people in middle and high age. The hallmark of ALS is a progressive and simultaneous loss of upper and lower motor neurons in the central nervous system that leads to a progressive muscle atrophy, paralysis and death usually by respiratory failure. ALS is not a pure motor neuronal syndrome; it extends beyond the motor system and affects extramotor areas of the brain as well. The majority of the patients suffer from a sporadic ALS disease (SALS) while in at least ten percent
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Morrice, Jessica Rebecca Marie. "Modeling sporadic amyotrophic lateral sclerosis (sals) in zebrafish using environmental stressors." Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/62666.

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Amyotrophic lateral sclerosis (ALS) is characterized by the progressive degeneration of upper and lower motor neurons. The majority of ALS patients are considered of unknown origin, termed sporadic, and are largely assumed to arise from environmental insults. Despite this, research to date has been heavily focused on genetic models of the disease, which represent 10% of ALS cases. Research has made disappointing progress with elucidating disease initiating mechanisms and therapeutic translation in patients. Sporadic ALS (sALS) models may provide substantially more applicable insight into dise
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Kostesky, Trisha Ehren. "A study of potential sporadic amyotrophic lateral sclerosis biomarkers in cerebrospinal fluid." Thesis, University of British Columbia, 2011. http://hdl.handle.net/2429/35690.

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Sporadic Amyotrophic Lateral Sclerosis (sALS) is a debilitating and fatal neurodegenerative disease of unknown etiology. It currently has no biochemical marker to confirm a clinical diagnosis, and this has negative consequences for patients when it comes to initiating early medical intervention and participating in therapeutic trials. Valid biomarkers can be useful for diagnostic and prognostic indications as well as providing insight into disease pathogenesis and identifying targets for therapeutic interventions. Cerebrospinal fluid (CSF) may be a particularly valuable source of biomarkers be
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Simpson, Claire Louise. "A genome-wide, gene density targeted association study in sporadic amyotrophic lateral sclerosis." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.428722.

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BENEDETTI, S. DE. "SPORADIC AMYOTROPHIC LATERAL SCLEROSIS IN PATIENTS WITH COMMON GEOGRAPHICAL ORIGIN: A MULTIDISCIPLINARY STUDY." Doctoral thesis, Università degli Studi di Milano, 2017. http://hdl.handle.net/2434/486489.

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Amyotrophic Lateral Sclerosis (ALS) is a late onset, fatal, neurodegenerative disorder that selectively affects motor neurons. It leads to the degeneration of both upper and lower motor neurons, respectively in the motor cortex and in the brainstem and spinal cord. Different mechanisms have been proposed to explain the pathogenesis of the disease: protein aggregation, oxidative stress, impairment of mitochondrial function, transcription dysfunctions, alterations in the proteasome pathway, inflammation and excitotoxicity. A wide phenotypical variability is described, likely attributable to a c
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Wong, Nelson K. Y. "Expression of nitric oxide synthase in cervical spinal cord in sporadic amyotrophic lateral sclerosis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0007/MQ32522.pdf.

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Vicars, Caitlyn. "Investigating short structural variants within FUS, RAB27B and TARDBP for associations with sporadic amyotrophic lateral sclerosis." Thesis, Vicars, Caitlyn (2022) Investigating short structural variants within FUS, RAB27B and TARDBP for associations with sporadic amyotrophic lateral sclerosis. Honours thesis, Murdoch University, 2022. https://researchrepository.murdoch.edu.au/id/eprint/66200/.

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Over the past decade, 90% of amyotrophic lateral sclerosis (ALS) clinical trials have failed. Furthermore, 90% of sporadic cases have unknown genetic cause. Short structural variants (sSVs) are repetitive genomic regions implicated in complex diseases, with the potential to be utilised for clinical trial enrichment. The purpose of this study was to investigate sporadic ALS (sALS) missing heritability via interrogating novel sSVs, and to determine their potential as genetic biomarkers. This project aimed to develop assays for characterisation and high-throughput genotyping of sSVs within ALS-li
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Ayaki, Takashi. "Immunoreactivity of valosin-containing protein in sporadic amyotrophic lateral sclerosis and in a case of its novel mutant." Kyoto University, 2015. http://hdl.handle.net/2433/200437.

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Jackson, Mandy. "Screening of familial and sporadic amyotrophic lateral sclerosis patients for mutations in CuZn superoxide dismutase (SOD-1) and other candidate genes." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363787.

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Books on the topic "Sporadic amyotrophic lateral sclerosis"

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Brown, Robert H., Michael Swash, and Piera Pasinelli. Amyotrophic Lateral Sclerosis. 2nd ed. CRC Press, 2021. http://dx.doi.org/10.1201/9781003076445.

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Cosi, V., Ann C. Kato, W. Parlette, P. Pinelli, and M. Poloni, eds. Amyotrophic Lateral Sclerosis. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5302-7.

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MD, Brown Robert H., Swash Michael, and Pasinelli Piera, eds. Amyotrophic lateral sclerosis. 2nd ed. Informa Healthcare, 2006.

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1947-, Brown Robert H., Meininger Vincent, and Swash Michael, eds. Amyotrophic lateral sclerosis. Martin Dunitz, 2000.

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National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. Amyotrophic lateral sclerosis. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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1949-, Chad David A., and Pioro Erik P. 1955-, eds. Amyotrophic lateral sclerosis. F.A. Davis, 1998.

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Clifford, Rose F., ed. Amyotrophic lateral sclerosis. Demos, 1990.

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1952-, Brown Robert H., Meininger Vincent, and Swash Michael, eds. Amyotrophic lateral sclerosis. Martin Dunitz, 2000.

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Hiroshi, Mitsumoto, Przedborski Serge, and Gordon Paul H, eds. Amyotrophic lateral sclerosis. Taylor & Francis, 2006.

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Ludwig, Gutmann, and Mitsumoto Hiroshi, eds. Advances in amyotrophic lateral sclerosis. Little, Brown, 1996.

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Book chapters on the topic "Sporadic amyotrophic lateral sclerosis"

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Provinciali, L., A. R. Giovagnoli, S. Battucci, C. Bartocci, and M. Montroni. "Immunological Assessment in Sporadic Amyotrophic Lateral Sclerosis (ALS)." In Advances in Experimental Medicine and Biology. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5302-7_25.

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Bergeron, Catherine, Connie Petrunka, and Luitgard Weyer. "Oxidative Stress Plays a Role in the Pathogenesis of Familial and Sporadic Amyotrophic Lateral Sclerosis." In Neurodegenerative Diseases. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-0209-2_33.

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Mishra, Pooja Shree, Anu Mary Varghese, K. Vijayalakshmi, et al. "Interplay Between Microglia and Astrocytes During Neuroinflammation: Lessons Learnt from In Vitro and In Vivo Models of Sporadic Amyotrophic Lateral Sclerosis." In The Biology of Glial Cells: Recent Advances. Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-8313-8_16.

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Öztürk, Şerefnur. "Amyotrophic Lateral Sclerosis." In Neurological Disorders in Clinical Practice. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23168-6_16.

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Donati, Stéphane Yannis, Didier Demory, and Jean-Michel Arnal. "Amyotrophic Lateral Sclerosis." In Uncommon Diseases in the ICU. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-04576-4_11.

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Rana, Abdul Qayyum, Ali T. Ghouse, and Raghav Govindarajan. "Amyotrophic Lateral Sclerosis." In Neurophysiology in Clinical Practice. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39342-1_17.

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Haase, Georg. "Amyotrophic Lateral Sclerosis." In Neuroprotection. Wiley-VCH Verlag GmbH & Co. KGaA, 2005. http://dx.doi.org/10.1002/3527603867.ch3.

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Tröster, Alexander I. "Amyotrophic Lateral Sclerosis." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_514.

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Lang, Bethan. "Amyotrophic Lateral Sclerosis." In Diagnostic Criteria in Autoimmune Diseases. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_76.

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Turner, Martin R. "Amyotrophic Lateral Sclerosis." In PET and SPECT in Neurology. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54307-4_29.

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Conference papers on the topic "Sporadic amyotrophic lateral sclerosis"

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Tiwari, Shamik, Saikat Gochhait, and Ritam Chatterjee. "Certain Investigations on Classification of Amyotrophic Lateral Sclerosis." In 2024 5th International Conference on Data Analytics for Business and Industry (ICDABI). IEEE, 2024. https://doi.org/10.1109/icdabi63787.2024.10800684.

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Elmakki, Shimaa M., Marwa M. A. Hadhoud, and Vidan F. Ghoneim. "Investigation of Amyotrophic Lateral Sclerosis Using Bioinformatics Tools." In 2025 42nd National Radio Science Conference (NRSC). IEEE, 2025. https://doi.org/10.1109/nrsc65659.2025.11018550.

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Maram, Balajee, and Rohan Raj Maram. "Integrating Explainability into RNNs for Amyotrophic Lateral Sclerosis Diagnosis." In 2025 Fourth International Conference on Smart Technologies, Communication and Robotics (STCR). IEEE, 2025. https://doi.org/10.1109/stcr62650.2025.11020162.

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P, Sri Varshan, Harigaran R, and Vamsi Krishna V. "Amyotrophic lateral sclerosis Disease Detection Through Facial Expression Analysis." In 2025 International Conference on Ambient Intelligence in Health Care (ICAIHC). IEEE, 2025. https://doi.org/10.1109/icaihc64101.2025.10956283.

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Rechichi, Irene, Gianluca Amprimo, Alessandro Cicolin, and Gabriella Olmo. "Predicting Amyotrophic Lateral Sclerosis Progression: an EMG-based Survival Analysis." In 2024 46th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2024. https://doi.org/10.1109/embc53108.2024.10782485.

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Kamasani, Kavya, Deepa Venna, Harshitha Badavathula, and Namratha Ranga. "Ensemble Machine Learning Model for Detection of Amyotrophic Lateral Sclerosis." In 2025 International Conference on Ambient Intelligence in Health Care (ICAIHC). IEEE, 2025. https://doi.org/10.1109/icaihc64101.2025.10956574.

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Pego Roque, Miguel, Andreia S. Martins, Marta Gromicho, et al. "Deep Temporal Consensus Clustering for Patient Stratification in Amyotrophic Lateral Sclerosis." In ESANN 2024. Ciaco - i6doc.com, 2024. http://dx.doi.org/10.14428/esann/2024.es2024-195.

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Bosoni, Pietro, Irene Aiello, Umberto Manera, et al. "Effect of Environmental Personal Exposure on Amyotrophic Lateral Sclerosis Disease Progression." In 2025 IEEE 13th International Conference on Healthcare Informatics (ICHI). IEEE, 2025. https://doi.org/10.1109/ichi64645.2025.00029.

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Wolmer, Paulo Schneider, Thiago Junqueira Ribeiro de Rezende, Fabrício Castro de Borba, and Marcondes Cavalcante França Junior. "Distinct patterns of cerebellar damage in sporadic and ATXN2- related amyotrophic lateral sclerosis." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.511.

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Introduction: Neurodegeneration in amyotrophic lateral sclerosis (ALS) is not confined to the corticospinal tracts and motor neurons. Some studies have lately suggested that the cerebellum is also involved in the disease. Despite that, it is not yet clear whether cerebellar damage is similar in sporadic (sALS) and specific subtypes of familial ALS. This is particularly relevant for ATXN2-related ALS, because this gene is also known to cause cerebellar ataxia (when fully expanded). Objectives: This study aimed to characterize cerebellar damage in vivo in patients with sporadic ALS and ATXN2-ALS
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Stephanie, Taylor, Tyzack Giulia, Mitchell Jamie, Harley Jasmine, and Patani Rickie. "HnRNPM is mislocalised from nucleus to cytoplasm in both sporadic and VCP-related amyotrophic lateral sclerosis." In ABN 2024 annual meeting abstracts. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/jnnp-2024-abn.4.

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Reports on the topic "Sporadic amyotrophic lateral sclerosis"

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Anklesaria, Pervin. Preclinical Development of Therapeutics for Amyotrophic Lateral Sclerosis. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada541412.

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Connor, James R. Apo-Ferritin as a Therapeutic Treatment for Amyotrophic Lateral Sclerosis. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada567828.

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Przedborski, Serge. Neuroprotective Small Molecules for the Treatment of Amyotrophic Lateral Sclerosis. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada567841.

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Connor, James R. Apo-Ferritin as a Therapeutic Treatment for Amyotrophic Lateral Sclerosis. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada598852.

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Grill, Raymond J. Targeted Riluzole Delivery by Antioxidant Nanovectors for Treating Amyotrophic Lateral Sclerosis. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613439.

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Grill, Raymond J. Targeted Riluzole Delivery by Antioxidant Nanovectors for Treating Amyotrophic Lateral Sclerosis. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada598451.

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LoGrasso, Philip, and Serge Przedborski. c-jun-N-Terminal Kinase (JNK) for the Treatment of Amyotrophic Lateral Sclerosis. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada596507.

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Santos, Ana Lúcia Yaeko da Silva, Deyse Mayara Rodrigues Caron, Livia Shirahige, and Abrahão Fontes Baptista. Alterations in Corticomotor Excitability in Amyotrophic Lateral Sclerosis: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.5.0078.

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Review question / Objective: To systematically evaluate the utility of TMS to follow up on ALS patients using neurophysiological metrics and to quantify corticomotor excitability compared to sham controls or other neuromuscular diseases. Condition being studied: Amyotrophic Lateral Sclerosis (ALS) is the third most common neurodegenerative disease (BRUNET et al., 2020). The condition is characterized by progressive muscle atrophy due to upper and lower motor neuron death (GOETZ, 2000).
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Shen, Chen, Xiao-Ti Wu, Chen-Hao Zhang, et al. Traditional Chinese medicine therapy for treating amyotrophic lateral sclerosis: a Bayesian network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.8.0050.

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Wackerman, Brooke L., B. L. Cox, K. L. Grayson, Shari L. Shanklin, and Wilson W. McGriff. Case Series Investigation of Amyotrophic Lateral Sclerosis (ALS) Among Former Kelly Air Force Base Workers. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada437518.

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