Relevant bibliographies by topics / Stem cell quiescence

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Stem cell quiescence'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Stem cell quiescence"

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Otsuki, L., and A. H. Brand. "Cell cycle heterogeneity directs the timing of neural stem cell activation from quiescence." Science 360, no. 6384 (2018): 99–102. http://dx.doi.org/10.1126/science.aan8795.

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Quiescent stem cells in adult tissues can be activated for homeostasis or repair. Neural stem cells (NSCs) in Drosophila are reactivated from quiescence in response to nutrition by the insulin signaling pathway. It is widely accepted that quiescent stem cells are arrested in G0. In this study, however, we demonstrate that quiescent NSCs (qNSCs) are arrested in either G2 or G0. G2-G0 heterogeneity directs NSC behavior: G2 qNSCs reactivate before G0 qNSCs. In addition, we show that the evolutionarily conserved pseudokinase Tribbles (Trbl) induces G2 NSCs to enter quiescence by promoting degradat
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Mohammad, Karamat, Paméla Dakik, Younes Medkour, Darya Mitrofanova, and Vladimir I. Titorenko. "Quiescence Entry, Maintenance, and Exit in Adult Stem Cells." International Journal of Molecular Sciences 20, no. 9 (2019): 2158. http://dx.doi.org/10.3390/ijms20092158.

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Cells of unicellular and multicellular eukaryotes can respond to certain environmental cues by arresting the cell cycle and entering a reversible state of quiescence. Quiescent cells do not divide, but can re-enter the cell cycle and resume proliferation if exposed to some signals from the environment. Quiescent cells in mammals and humans include adult stem cells. These cells exhibit improved stress resistance and enhanced survival ability. In response to certain extrinsic signals, adult stem cells can self-renew by dividing asymmetrically. Such asymmetric divisions not only allow the mainten
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Chen, Danica. "MITOCHONDRIAL METABOLIC CHECKPOINT, STEM CELL AGING AND REJUVENATION." Innovation in Aging 6, Supplement_1 (2022): 287. http://dx.doi.org/10.1093/geroni/igac059.1143.

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Abstract Cell cycle checkpoints are surveillance mechanisms in eukaryotic cells that monitor the condition of the cell, repair cellular damages, and allow the cell to progress through the various phases of the cell cycle when conditions become favorable. Recent advances in stem cell biology highlight a mitochondrial metabolic checkpoint that is essential for stem cells to return to the quiescent state. As quiescent stem cells enter the cell cycle, mitochondrial biogenesis is induced and mitochondrial stress is increased. Mitochondrial unfolded protein response and mitochondrial oxidative stres
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Purnell, B. A. "Translating stem cell quiescence." Science 351, no. 6274 (2016): 677–78. http://dx.doi.org/10.1126/science.351.6274.677-b.

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Liu, Yan, Yasuhiko Miyata, Goro Sashida, et al. "Genetic Uncoupling of the Regulation of Hematopoietic Stem Cell Quiescence and Self-Renewal." Blood 108, no. 11 (2006): 1347. http://dx.doi.org/10.1182/blood.v108.11.1347.1347.

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Abstract It is usually stated that HSCs must choose to either self-renew or to differentiate and lose some of their multi potentiality. Recently, we demonstrated that MEF, an ETS family of transcription factor, played an important role in regulating HSC quiescence, illustrating a third choice for the HSC, namely to make an “active” choice and remain quiescent, without undergoing either self-renewal, or differentiation. MEF null HSCs are more quiescent than normal HSCs. In addition, MEF null mice exhibit greater numbers of hematopoietic stem cells and show resistance to chemotherapy and radiati
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Liu, Yan, Shannon E. Elf, Yasuhiko Miyata, et al. "Regulation of Hematopoietic Stem Cell Quiescence - A Novel Role for p53." Blood 110, no. 11 (2007): 92. http://dx.doi.org/10.1182/blood.v110.11.92.92.

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Abstract Although the p53 tumor suppressor can elicit cell-cycle arrest or apoptosis in hematopoietic cells upon DNA damage, its function during steady-state hematopoiesis is largely unknown. We demonstrated that the Ets transcription factor MEF/ELF4 regulates both HSC proliferation/self-renewal and quiescence, as Mef null mice exhibit greater numbers of hematopoietic stem cells and the Mef null HSCs are more quiescent than normal. As such, the hematopoietic compartment of Mef null mice shows significant resistance to chemotherapy and radiation (Lacorazza et al., Cancer Cell, 2006). In this st
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Ghaffari, Saghi. "Regulation of Hematopoietic Stem Cell Mitochondrial Metabolism." Blood 128, no. 22 (2016): SCI—33—SCI—33. http://dx.doi.org/10.1182/blood.v128.22.sci-33.sci-33.

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Abstract Hematopoietic stem cells (HSCs) like most, if not all, adult stem cells are primarily quiescent but have the potential to become highly active on demand. HSC quiescence is maintained by glycolytic metabolism and low levels of reactive oxygen species (ROS), which indicate that mitochondria are relatively inactive in quiescent HSC. However, HSC cycling - and exit of quiescence state - require a swift metabolic switch from glycolysis to mitochondrial oxidative phosphorylation. To improve our understanding of mechanisms that integrate energy metabolism with HSC homeostasis, my laboratory
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Suda, Toshio. "Purine Metabolism and Hematopoietic Stem and Progenitor Stem Cells Under Stress." Blood 132, Supplement 1 (2018): SCI—19—SCI—19. http://dx.doi.org/10.1182/blood-2018-99-109515.

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Abstract Hematopoietic stem cells (HSCs) play a key role in the lifelong maintenance of hematopoiesis through self-renewal and multi-lineage differentiation. Adult HSCs reside within a specialized microenvironment of the bone marrow (BM), called "niche", in which they are maintained in a quiescent state in cell cycle. Most of HSCs within BM show quiescence under the hypoxic niche. Since the loss of HSC quiescence leads to the exhaustion or aging of stem cells through excess cell division, the regulation of quiescence in HSCs is essential for hematopoietic homeostasis. On the other hand, cellul
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VanHook, Annalisa M. "Inflammation induces stem cell quiescence." Science Signaling 12, no. 605 (2019): eaaz9665. http://dx.doi.org/10.1126/scisignal.aaz9665.

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Sorrentino, Brian P. "Scl and stem cell quiescence." Blood 115, no. 4 (2010): 751–52. http://dx.doi.org/10.1182/blood-2009-11-251264.

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Dissertations / Theses on the topic "Stem cell quiescence"

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Frigola, Tubert David. "Fluctuations, gene circuit architecture and stem cell quiescence." Doctoral thesis, Universitat de Barcelona, 2016. http://hdl.handle.net/10803/395181.

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Biological development is a complex process in which, from a single cell, a whole multicellular organism arises. The formation of this intrincate structures requires a very precise regulation in space and time. This regulation involves a network of proteins and genes that interact. These interactions give rise to nonlinear behaviours, of the same kind that physicists have studied in other systems. Furthermore, cellular processes are often subject to fluctuations, a problem that has been studied by out of equilibrium statistical mechanics. These facts mean that the tools from nonlinear physics
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Althoff, Mark J. "Cell polarity in hematopoietic stem cell quiescence, signaling and fate determination." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1583999632089058.

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Goel, Aviva J. "Niche Regulation of Muscle Stem Cell Quiescence by Classical Cadherins." Thesis, Icahn School of Medicine at Mount Sinai, 2018. http://pqdtopen.proquest.com/#viewpdf?dispub=10743988.

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<p> Many adult stem cells are characterized by prolonged quiescence, promoted by cues from their niche. Upon tissue damage, a coordinated transition to the activated state is necessary for successful repair. Non-physiological breaks in quiescence often lead to stem cell depletion and impaired tissue restoration. Here, I identify cadherin-mediated adhesion and signaling between muscle stem cells (satellite cells; SCs) and their myofiber niche as a mechanism that orchestrates the quiescence-to-activation transition. Conditional removal of N-cadherin and M-cadherin in mice leads to a break in SC
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Wendland, Emily-Marie. "Molecular genetic analysis of germline stem cell quiescence in «Caenorhabditis elegans»." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=96927.

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Cell division must be tightly regulated through both developmental programming and environmental conditions. One of the central foci of cancer research over the last decade has been the analysis of stem cells and their corresponding niche and how these specialized cells contribute to tumourigenesis. The germline stem cells of Caenorhabditis elegans, which are quiescent upon induction of dauer, were used here to study genetic responses in cell cycle regulation to environmental conditions during larval development. Global RNAi screening implicated a number of genes involved inregulating polarity
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Sinclair, Amy. "An investigation into the role of chemokines in haemopoietic stem cell quiescence." Thesis, University of Glasgow, 2015. http://theses.gla.ac.uk/4956/.

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Haemopoietic stem cells (HSC) maintain lifelong haemopoiesis through the monitoring and production of cells from multiple haemopoietic cell lineages. A key property of HSC is their ability to maintain quiescence. Quiescence refers to a state of inactivity in which the cell is not dividing and remains dormant. It is this property of the HSC that is thought to maintain genomic integrity and to allow the HSC to sustain haemopoiesis over the period of a lifetime. However, the regulation of quiescence in this context is not well understood. Numerous studies have aimed to understand the molecular me
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Jones, Kieran Michael. "Quiescence and cell fate regulation are essential for preserving adult stem cell number and function." Thesis, King's College London (University of London), 2014. https://kclpure.kcl.ac.uk/portal/en/theses/quiescence-and-cell-fate-regulation-are-essential-for-preserving-adult-stem-cell-number-and-function(4aed131c-aaf9-4697-94ab-24d4abeb9c6f).html.

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Somatic stem cell populations display a remarkable capacity to self-renew and generate specialised cell types throughout the life of the organism. In my thesis I examined extrinsic and intrinsic factors that regulate stem cell quiescence, a reversible state of growth arrest crucial to the preservation of somatic stem cell number and function in many systems. Skeletal muscle-specific stem cells, known as satellite cells (SCs) are responsible for skeletal muscle regeneration. The ability of skeletal muscle to regenerate declines with age. I identify fibroblast growth factor 2 (FGF2) as a potent
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Labusch, Miriam. "Unraveling the role of the lysosomal protein Prosaposin in neural stem cell quiescence." Electronic Thesis or Diss., Sorbonne université, 2022. http://www.theses.fr/2022SORUS387.

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Les cellules souches neurales (CSN) adultes produisent des neurones dans des régions restreintes du cerveau de la plupart des vertébrés. L'un des éléments clés du maintien des CSN est la quiescence : un état réversible d'arrêt du cycle cellulaire, hétérogène et activement maintenu, qui reste incomplètement compris. Il a été démontré que de nombreuses voies moléculaires, y compris la signalisation Notch, contrôlent la quiescence, mais une attention plus récente a également été accordée aux composants subcellulaires des CSN quiescentes par rapport aux CSN activées. Parmi eux, les lysosomes sont
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Chaouni, Rita. "Characterization of «par-4-»dependent germ line stem cell quiescence in Caenorhabditis elegans." Thesis, McGill University, 2014. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=121257.

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Upon encountering harsh environmental conditions, Caenorhabditis elegans larvae are able to alter their developmental program and enter the dauer diapause, an alternative developmental stage that enables larvae to endure long periods of stress. During this arrested state, the germ line stem cells, which normally divide during reproductive development, halt their proliferation and are consequently rendered quiescent. Previous work has implicated a role for PAR-4/LKB1 in germ line stem cell quiescence. Inactivating mutations in par-4 result in aberrant germ line stem cell proliferation during th
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Yakubovich, Edward. "The Role of Activator E2Fs in Neural Stem Cell Activation and Exit from Quiescence." Thesis, Université d'Ottawa / University of Ottawa, 2019. http://hdl.handle.net/10393/39437.

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Regenerative medicine offers tremendous potential for the treatment of irreversible damage to the brain. Activation of quiescent adult neural stem cells by clinical means to regenerate tissue can improve pathological outcomes of patients afflicted by brain trauma. Control of the cell- cycle is important in activating quiescent neural stem cells for the purpose of enhancing adult neurogenesis. Here, we uncover the role of cell-cycle regulatory transcription factors E2F1 and E2F3 in adult neural stem cell activation and characterize it. We hypothesize that the Retinoblastoma-E2F pathway is cruci
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Noyer, Lucile. "Role of Orai1 in prostate cancer proliferation and cancer stem cell quiescence/activation transition." Thesis, Lille 1, 2019. http://www.theses.fr/2019LIL1S111.

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Le cancer de la prostate (CaP) est le cancer le plus fréquent et le troisième plus mortel chez l’homme en Europe. Les cellules souches cancéreuses (CSC) représentent une sous population de cellules cancéreuses possédant des propriétés de cellules souches qui les rendent résistantes aux thérapies et hautement tumorigènes. Les CSCs sont ainsi associées aux phénomènes de dormance tumorale, puis de rechute suite à leur réactivation. Les mécanismes régulant la transition dormance/prolifération constituent donc une question centrale dans la prise en charge du cancer. L’importance des protéines Orai
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Books on the topic "Stem cell quiescence"

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Paul, Alexander J. Local and Long-range Regulation of Adult Neural Stem Cell Quiescence. [publisher not identified], 2016.

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Lacorazza, H. Daniel. Cellular Quiescence: Methods and Protocols. Springer New York, 2017.

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Lacorazza, H. Daniel. Cellular Quiescence: Methods and Protocols. Springer New York, 2018.

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Book chapters on the topic "Stem cell quiescence"

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Matatall, Katie A., Claudine S. Kadmon, and Katherine Y. King. "Detecting Hematopoietic Stem Cell Proliferation Using BrdU Incorporation." In Cellular Quiescence. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7371-2_7.

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Jalbert, Emilie, and Eric M. Pietras. "Analysis of Murine Hematopoietic Stem Cell Proliferation During Inflammation." In Cellular Quiescence. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7371-2_14.

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Childress, Paul J., Marta B. Alvarez, Brahmananda R. Chitteti, Melissa A. Kacena, and Edward F. Srour. "The Hematopoietic Stem Cell Niche: Cell-Cell Interactions and Quiescence." In Stem Cell Biology and Regenerative Medicine. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-21702-4_1.

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Chicheportiche, Alexandra, Martial Ruat, François D. Boussin, and Mathieu Daynac. "Isolation of Neural Stem and Progenitor Cells from the Adult Brain and Live Imaging of Their Cell Cycle with the FUCCI System." In Cellular Quiescence. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7371-2_5.

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Rocheteau, Pierre, Mathilde Vinet, and Fabrice Chretien. "Dormancy and Quiescence of Skeletal Muscle Stem Cells." In Results and Problems in Cell Differentiation. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-44608-9_10.

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Rojas-Sutterlin, Shanti, and Trang Hoang. "Hematopoietic Stem Cell Quiescence and Long Term Maintenance: Role of SCL/TAL1." In Tumor Dormancy, Quiescence, and Senescence, Volume 1. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-5958-9_8.

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Krtolica, Ana. "Role of Microenvironment in Regulating Stem Cell and Tumor Initiating Cancer Cell Behavior and Its Potential Therapeutic Implications." In Tumor Dormancy, Quiescence, and Senescence, Volume 2. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7726-2_28.

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Yamaguchi, Masahiko, and So-ichiro Fukada. "Regulation of Muscle Stem Cell Quiescent and Undifferentiated State: Roles of Hesr1 and Hesr3 Genes." In Tumor Dormancy, Quiescence, and Senescence, Volume 1. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-5958-9_9.

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Angelozzi, Marco, Charles R. de Charleroy, and Véronique Lefebvre. "EdU-Based Assay of Cell Proliferation and Stem Cell Quiescence in Skeletal Tissue Sections." In Methods in Molecular Biology. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-1028-2_21.

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Arora, Reety, Mohammed Rumman, Nisha Venugopal, Hardik Gala, and Jyotsna Dhawan. "Mimicking Muscle Stem Cell Quiescence in Culture: Methods for Synchronization in Reversible Arrest." In Methods in Molecular Biology. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6771-1_15.

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Conference papers on the topic "Stem cell quiescence"

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White, Andrew C., Joan Khuu, Christine Dang, Kathy Tran, Anqi Liu, and Bill Lowry. "Abstract B46: Adult stem cell quiescence as a tumor suppressor mechanism in squamous tumors." In Abstracts: AACR Special Conference: The Translational Impact of Model Organisms in Cancer; November 5-8, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1557-3125.modorg-b46.

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Wu, Megan YiJun, Angela Celebre, Jeffrey Chan, et al. "Abstract B35: BMP regulation of cancer stem cell quiescence is responsible for chemotherapeutic resistance in glioblastoma." In Abstracts: AACR Special Conference: Advances in Brain Cancer Research; May 27-30, 2015; Washington, DC. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.brain15-b35.

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Amin, Md Ruhul, Yuiko Morita-Fujimura та Shuntaro Ikawa. "Abstract LB-207: ΔNp63α regulates quiescence, stem or progenitor activity of normal and malignant breast cells in a cell type-specific manner". У Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-lb-207.

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Van Dyke, William S., Ozan Akkus, and Eric Nauman. "Murine Osteochondral Stem Cells Express Collagen Type I More Strongly on PDMS Substrates Than on Tissue Culture Plastic." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14272.

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The discovery of the multipotent lineage of mesenchymal stem cells has dawned a new age in tissue engineering, where an autologous cell-seeded scaffold can be implanted into different therapeutic sites. Mesenchymal stem cells have been reported to differentiate into numerous anchorage-dependent cell phenotypes, including neurons, adipocytes, myoblasts, chondrocytes, tenocytes, and osteoblasts. A seminal work detailing that mesenchymal stem cells can be directed towards differentiation of different cell types by substrate stiffness alone [1] has led to numerous studies attempting to understand
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Agudo, Judith, Miriam Merad, and Brian D. Brown. "Abstract A168: Quiescent stem cells evade immune surveillance." In Abstracts: Fourth CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 30 - October 3, 2018; New York, NY. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr18-a168.

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Shiomi, Takayuki, Peter V. N. Bodine, and Jeanine D'Armiento. "SFRP1 Induced Quiescence Of Bronchial Alveolar Stem Cells Prevents Excessive Proliferation Post Injury." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5286.

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Honeth, Gabriella, Rebecca Marlow, Ireneusz Shinomiya, Sara Lombardi, Bharath Buchupalli, and Gabriela Dontu. "Abstract LB-189: A role of Notch2 in quiescence of mammary stem/progenitor cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-lb-189.

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Fuchs, Elaine V. "Abstract IA-15: Balancing quiescence and proliferation in stem cells and its implications for cancer." In Abstracts: First AACR International Conference on Frontiers in Basic Cancer Research--Oct 8–11, 2009; Boston MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.fbcr09-ia-15.

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Wang, Yang, Anne E. Powell, Yina Li, et al. "Abstract 5188: Lrig1, a cell surface negative regulator of ErbB1-4, marks both proliferative and quiescent intestinal stem cells and acts as a tumor suppressor." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-5188.

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Merlo, ME Boggio, M. Mallardo, G. De Conti, et al. "PO-272 Leukemia-associated NPM mutations promote quiescence of hematopoietic stem cells and prevent their functional exhaustion upon oncogene-induced hyper-proliferation." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.303.

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Reports on the topic "Stem cell quiescence"

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DeCastro, Andrew J., Pratima Cherukuri, and James DiRenzo. Regulation of Mammary Stem Cell Quiescence via Post-Translational Modification of DeltaNp63alpha. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada576304.

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DeCastro, Andrew J., Pratima Cherukuri, and James DiRenzo. Regulation of Mammary Stem Cell Quiescence via Post-Translational Modification of DeltaNp63alpha. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada599224.

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Lee, Byeong-Chel. Purinergic Receptors in Quiescence and Localization of Leukemic Stem Cells. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada564468.

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Lee, Byeong-Chel. Purinergic Receptors in Quiescence and Localization of Leukemic Stem Cells. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada581409.

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Lee, Byeong-Chel. Purinergic Receptors in Quiescence and Localization of Leukemic Stem Cells. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada555803.

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